Academic literature on the topic 'SS sickle cell disease'

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Journal articles on the topic "SS sickle cell disease"

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Parrish, James M., Paul A. Page, David Cohen, et al. "Prebypass Pheresis and Red Blood Cell Exchange in a Patient with Homozygous SS Sickle Cell Disease Undergoing Cardiopulmonary Bypass: A Case Report." Journal of ExtraCorporeal Technology 26, no. 3 (1994): 143–51. http://dx.doi.org/10.1051/ject/1994263143.

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Sickle cell disease was first described by Herrick in 1910. This disease involves an abnormality of the hemoglobin molecule which, under certain conditions, causes the red cell to take on a sickle shape. This abnormal shape and hemoglobin prevent the red cell from performing the normal respiratory functions and interfere with the normal flow through the circulatory system. Individuals demonstrate either a homozygous (dominant) SS or a heterozygous Ss state. Clinical symptoms for patients with SS disease are most often characterized by recurrent painful crises, sequestration of sickled cells, c
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Adhikari, Ram Chandra, T. B. Shrestha, R. B. Shrestha, R. C. Subedi, K. P. Parajuli, and S. Dali. "SICKLE CELL DISEASE - CASE REPORTS." Journal of Nepal Medical Association 42, no. 145 (2003): 36–38. http://dx.doi.org/10.31729/jnma.715.

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ABSTRACTSickle cell diseases are inherited hematological diseases, prevalent in certain parts of the world. We reporttwo cases of sickle cell diseases, first being sickle cell b-thalassaemia and second homozygous sickle celldisease (SS). Our first case was 5 year old boy presenting with hemolytic anaemia & hepatosplenomegalyhaving sickle cell b-thalassaemia disease . Second case was 17 years female presenting with hemolyticanaemia & joint pain having homozygous sickle cell disease.Key Words: Homozygous sickle cell disease, sickle cell b - thalassaemia, hemoglobin electrophoresis.
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Koshy, M., SJ Weiner, ST Miller, et al. "Surgery and anesthesia in sickle cell disease. Cooperative Study of Sickle Cell Diseases." Blood 86, no. 10 (1995): 3676–84. http://dx.doi.org/10.1182/blood.v86.10.3676.bloodjournal86103676.

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From 1978 to 1988, The Cooperative Study of Sickle Cell Disease observed 3,765 patients with a mean follow-up of 5.3 +/- 2.0 years. One thousand seventy-nine surgical procedures were conducted on 717 patients (77% sickle cell anemia [SS], 14% sickle hemoglobin C disease [SC], 5.7% S beta zero thalassemia, 3% S beta zero + thalassemia). Sixty-nine percent had a single procedure, 21% had two procedures, and the remaining 11% had more than two procedures during the study follow- up. The most frequent procedure was abdominal surgery for cholecystectomy or splenectomy (24% of all surgical procedure
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Gill, FM, LA Sleeper, SJ Weiner, et al. "Clinical events in the first decade in a cohort of infants with sickle cell disease. Cooperative Study of Sickle Cell Disease [see comments]." Blood 86, no. 2 (1995): 776–83. http://dx.doi.org/10.1182/blood.v86.2.776.bloodjournal862776.

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Within the Cooperative Study of Sickle Cell Disease, 694 infants with confirmed sickle cell disease were enrolled at less than 6 months of age. Information about the nature and frequency of complications was collected prospectively over a 10-year period. Painful crises and acute chest syndrome were the most common sickle cell-related events in homozygous sickle cell anemia (SS), hemoglobin SC disease (SC), and S beta thalassemia patients (overall incidence in SS patients of 32.4 and 24.5 cases per 100 person-years, respectively). Bacteremia occurred most frequently in SS children under 4 years
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Hlouedjè, Houénoukpo Wilfried, Jacques Ezéchiel Lokonon, Maximin Sènou, et al. "Some markers of inflammation in patients with sickle cell disease at Zou-Collines departmental hospital in Benin." International Journal of Research in Medical Sciences 10, no. 6 (2022): 1219. http://dx.doi.org/10.18203/2320-6012.ijrms20221475.

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Background: Sickle cell disease was a genetic pathology of the red blood cell, which caused systemic functional and tissue damage. The present work aimed to study some biomarkers of inflammation and renal function in sickle cell patients.Methods: The biochemical and inflammatory markers were assayed with a spectrophotometer and the hemogram was performed on a hematology analyzer in SS homozygous sickle cell subjects and controls with AA phenotypes.Results: The male sex (63.49%) and the age group 5-18 years (58.73%) were predominant among the SS subjects of the study population. Blood urea and
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Boisson, Camille, Minke A. E. Rab, Elie Nader, et al. "Effects of Genotypes and Treatment on Oxygenscan Parameters in Sickle Cell Disease." Cells 10, no. 4 (2021): 811. http://dx.doi.org/10.3390/cells10040811.

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(1) Background: The aim of the present study was to compare oxygen gradient ektacytometry parameters between sickle cell patients of different genotypes (SS, SC, and S/β+) or under different treatments (hydroxyurea or chronic red blood cell exchange). (2) Methods: Oxygen gradient ektacytometry was performed in 167 adults and children at steady state. In addition, five SS patients had oxygenscan measurements at steady state and during an acute complication requiring hospitalization. (3) Results: Red blood cell (RBC) deformability upon deoxygenation (EImin) and in normoxia (EImax) was increased,
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Raghuwanshi, Pranav Kumar, and Somnath Singh Raghuvanshi. "Vascular endothelial dysfunction in sickle cell disease by brachial artery flow mediated dilatation." Asian Journal of Medical Sciences 5, no. 3 (2014): 105–7. http://dx.doi.org/10.3126/ajms.v5i3.9445.

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Objective: The present study was conducted aiming to assess endothelial function in sickle cell disease (SS), sickle cell trait(SA) and compare to endothelial dysfunction between sickle cell anemia (SS), sickle cell trait (SA) cases and control (AA) patients to evaluate correlation of endothelial dysfunction. Methods: The study population comprised of, total 25 cases having sickle cell disease and sickle cell trait and 25 age and sex matched normal control. Endothelial dysfunction as assessed by brachial artery flow mediated dilatation by colour Doppler (non-invasive method)by using Siemens So
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Pranav Kumar Raghuwanshi and Somnath Singh Raghuvanshi. "Vascular endothelial dysfunction in sickle cell disease by brachial artery flow mediated dilatation." Asian Journal of Medical Sciences 5, no. 3 (2014): 105–7. https://doi.org/10.71152/ajms.v5i3.3338.

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Objective: The present study was conducted aiming to assess endothelial function in sickle cell disease (SS), sickle cell trait(SA) and compare to endothelial dysfunction between sickle cell anemia (SS), sickle cell trait (SA) cases and control (AA) patients to evaluate correlation of endothelial dysfunction. Methods: The study population comprised of, total 25 cases having sickle cell disease and sickle cell trait and 25 age and sex matched normal control. Endothelial dysfunction as assessed by brachial artery flow mediated dilatation by colour Doppler (non-invasive method)by using Siemens So
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Quinn, Charles T., Zora R. Rogers, and George R. Buchanan. "Survival of children with sickle cell disease." Blood 103, no. 11 (2004): 4023–27. http://dx.doi.org/10.1182/blood-2003-11-3758.

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Abstract Contemporary survival data are not available for children with sickle cell disease (SCD). The few previous childhood SCD cohort studies do not reflect the benefits of modern therapy. We defined an inception cohort of newborns with sickle cell anemia (SS), sickle-β°-thalassemia (S β°), sickle-hemoglobin C disease (SC), or sickle-β+-thalassemia (Sβ+) who were identified by newborn screening and followed for up to 18 years. The incidence of death and stroke were calculated. Overall survival, SCD-related survival (considering only SCD-related deaths), and strokefree survival were determin
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Nabors, Nina A., and Angela K. Freymuth. "Attention Deficits in Children with Sickle Cell Disease." Perceptual and Motor Skills 95, no. 1 (2002): 57–67. http://dx.doi.org/10.2466/pms.2002.95.1.57.

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Previous research has suggested that children with sickle cell disease may exhibit cognitive deficits even in the absence of direct cerebrovascular involvement (stroke). This study was designed to assess specific attentional deficits in children with sickle cell disease. 12 children with sickle cell disease (Hb SS) with a prior history of stroke, 14 children with sickle cell disease (Hb SS) without evidence of stroke, and 13 similar aged siblings (Hb AA or Hb AS) were compared on measures of attention, intellectual functioning, achievement, and adaptive Functioning, Significant differences wer
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Dissertations / Theses on the topic "SS sickle cell disease"

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Lalanne-Mistrih, Marie-Laure. "Caractérisation des profils lipidiques plasmatiques des patients drépanocytaires guadeloupéens SS et SC à l'état stable. Profil lipidique et hémorhéologique des complications du sous-phénotype hyperhémolytique/vasculopatie et hypervisqueux/vaso occlusif. : Profil lipidique et hémorhéologique des complications du sous-phénotype hyperhémolytique/vasculopathie et hypervisqueux/vaso occlusif." Thesis, Antilles, 2019. http://www.theses.fr/2019ANTI0437.

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Les patients drépanocytaires ont un profil lipidique spécifique, à priori non athérogène.En 2009 est découvert qu’un taux d’apolipoprotéine AI et de HDL-cholestérol abaissés et de triglycérides élevés (HTG), constitue un profil lipidique plus souvent associé à une hypertension artérielle pulmonaire. Notre travail de thèse a consisté en l’étude, à l’état stable, des profils lipidiques des cohortes d’adultes drépanocytaires homozygotes SS (HbS/HbS) et hétérozygotes composites SC (HbS/HbC), à l’unité transversale de la Drépanocytose du CHU de la Guadeloupe, comparés ensuite à celui de la populati
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Tozatto, Maio Karina. "Immunogenetics in sickle cell disease." Thesis, Sorbonne Paris Cité, 2019. http://www.theses.fr/2019USPCC093.

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La drépanocytose est l’hémoglobinopathie héréditaire la plus fréquente, causée par un polymorphisme unique d’un nucléotide (SNP) dans le gène de la beta-globine (HBB). Ce SNP détermine la synthèse de l’hémoglobine S, qui polymérise lorsqu’elle est soumise au stress, et ceci change la forme des hématies drépanocytaires en faucille. Les drépanocytes sont moins déformables, plus adhérents à l’endothélium, et plus susceptibles à l’hémolyse. Les complications cliniques de la drépanocytose peuvent être expliquées par l’interaction entre la vaso-occlusion, l’hémolyse et l’activation inflammatoire rés
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Hays, Mary Margaret. "Stem cell transplant for sickle cell disease." Thesis, Boston University, 2013. https://hdl.handle.net/2144/12117.

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Thesis (M.A.)--Boston University<br>Background: Sickle cell disease (SCD) is the most common inherited blood disorder in the United States. As SCD can cause significant morbidity and decrease in life expectancy, further research on curative options is of great interest. Hematopoietic stem cell transplant (HSCT) is the only treatment option offering a chance of cure, but the risks of treatment are not negligible. Because the outcomes of HSCT are best when the procedure is performed at a younger age, understanding what parents know about transplant, their opinion on this option and the risks the
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Kawadler, J. M. "Neuroimaging biomarkers in paediatric sickle cell disease." Thesis, University College London (University of London), 2015. http://discovery.ucl.ac.uk/1464063/.

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Sickle Cell Disease (SCD) is a collection of genetic haemoglobinopathies, the most common and severe being homozygous sickle cell anaemia. In the UK, it has been estimated that 1 in 2000 children are born with SCD. The disease is characterised by chronic anaemia, recurrent pain crises and vascular occlusion. Neurologically, there is a high incidence of stroke in childhood, as well as cognitive dysfunction. Newborn screening programmes and preventative treatments have allowed a much longer lifespan; however recently, neurological research has shifted to characterising subtler aspects of brain d
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Gavlak, J. C. D. "Sleep in children with sickle cell disease." Thesis, University College London (University of London), 2016. http://discovery.ucl.ac.uk/1528622/.

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BACKGROUND: Sickle Cell Anaemia (SCA) or homozygosity for the sickle haemoglobin gene (HbSS) is the most common genetic condition in the UK. A high prevalence of Sleep Disordered Breathing (SDB) in SCA is widely accepted but there is a lack of unselected population based studies. As Polysomnography (PSG) is expensive, screening for SDB using a robust tool, e.g. the Delta 12 (Δ12) index and 3% Oxygen Desaturation Indices (ODI) calculated from home pulse oximetry, should be validated. Elevated Cerebral Blood Flow Velocity (CBFV) is a predictor of stroke in SCA, and may be associated with SDB. ME
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Gardner, Catherine Joanne. "Genotype-phenotype correlation in sickle cell disease." Thesis, King's College London (University of London), 2017. https://kclpure.kcl.ac.uk/portal/en/theses/genotypephenotype-correlation-in-sickle-cell-disease(07a190be-c88a-41f2-8e74-e063d85919a3).html.

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Sickle cell disease (SCD) has a complex pathophysiology initiated by the polymerisation of deoxy-sickle-haemoglobin. The single nucleotide change underpinning SCD does not account for the vast range and severity of SCD complications. This clinical heterogeneity is only partly explained by the genetic variability of fetal haemoglobin gene levels and co-inheritance of α- thalassemia. Although environmental factors also contribute to the clinical complexity of SCD, further genetic modifiers of SCD severity exist but are yet to be determined. Genetic association studies have been boosted recently
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Mackey, Michelle Noble. "Understanding Parents' Disease-Managing Strategies for Children With Sickle Cell Disease." ScholarWorks, 2019. https://scholarworks.waldenu.edu/dissertations/6610.

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Sickle cell disease (SCD) is one of the most difficult and stressful chronic diseases for parents of afflicted children to manage. Managing SCD can be traumatic for parents particularly if they have no specific coping strategies for managing the disease or ensuring the child visits the doctor as scheduled. The use of certain coping strategies may affect the parents' and patients' perceptions of the illness and influence their decisions regarding treatment, which can have a lasting impact on their lives. Effective parental strategies such as positive thinking can aid in disease management, but
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Edmond, A. M. "The spleen in sickle cell disease in childhood." Thesis, University of Cambridge, 1988. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.598759.

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Singhal, Atul. "Growth and metabolism in homozygous sickle cell disease." Thesis, University College London (University of London), 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.288009.

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Indik, Julia H., Vineet Nair, Ruslan Rafikov, et al. "Associations of Prolonged QTc in Sickle Cell Disease." PUBLIC LIBRARY SCIENCE, 2016. http://hdl.handle.net/10150/622120.

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Sudden death is a leading cause of mortality in sickle cell disease, implicating ventricular tachyarrhythmias. Prolonged QTc on an electrocardiogram (ECG), commonly seen with myocardial ischemia, is a known risk for polymorphic ventricular tachycardia (VT). We hypothesized that prolonged QTc is associated with mortality in sickle cell disease. ECG were analyzed from a cohort of 224 sickle patients (University of Illinois at Chicago, UIC) along with available laboratory, and echocardiographic findings, and from another cohort of 38 patients (University of Chicago, UC) for which cardiac MRI and
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Books on the topic "SS sickle cell disease"

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Peak, Lizabeth. Sickle cell disease. Lucent Books, 2008.

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Peak, Lizabeth. Sickle cell disease. Lucent Books, 2008.

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Serjeant, Graham R. Sickle cell disease. 2nd ed. Oxford University Press, 1992.

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F, Whitten Charles, Bertles John F. 1925-, National Association for Sickle Cell Disease (U.S.), and New York Academy of Sciences., eds. Sickle cell disease. New York Academy of Sciences, 1989.

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Eboh, Winifred Oluchukwu. Sickle cell disease. Birmingham Sickle Cell & Thalassaemia Centre, 1993.

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Jones, Phill. Sickle cell disease. Chelsea House, 2008.

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Samuel, Charache, and Johnson Cage S, eds. Sickle cell disease. W.B. Saunders, 1996.

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McCormick, Marie, Henrietta Awo Osei-Anto, and Rose Marie Martinez, eds. Addressing Sickle Cell Disease. National Academies Press, 2020. http://dx.doi.org/10.17226/25632.

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P, Rodgers Griffin, ed. Sickle cell disease and thalassaemia. Baillière Tindall, 1998.

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author, Chilman-Blair Kim, and DeLoache Shawn author, eds. Medikidz explain sickle cell disease. Medikidz Ltd., 2013.

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Book chapters on the topic "SS sickle cell disease"

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Theisler, Charles. "Sickle Cell Anemia/Hemoglobin SS Disease." In Adjuvant Medical Care. CRC Press, 2022. http://dx.doi.org/10.1201/b22898-310.

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Mallouh, Ahmad A. "Sickle Cell Disease." In Textbook of Clinical Pediatrics. Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-02202-9_324.

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Kashani, John, Richard D. Shih, Thomas H. Cogbill, et al. "Sickle Cell Disease." In Encyclopedia of Intensive Care Medicine. Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-00418-6_342.

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Fischer, Matt, Harsh Sachdeva, and Alaa Abd-Elsayed. "Sickle Cell Disease." In Pain. Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-99124-5_274.

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Roth, Elliot J. "Sickle-Cell Disease." In Encyclopedia of Clinical Neuropsychology. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-57111-9_2197.

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Chaudhury, Sonali, and Shalini Shenoy. "Sickle cell disease." In Clinical Manual of Blood and Bone Marrow Transplantation. John Wiley & Sons, Ltd, 2017. http://dx.doi.org/10.1002/9781119095491.ch27.

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Glassberg, Jeffrey, and Michael R. DeBaun. "Sickle Cell Disease." In Respiratory Medicine. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-43447-6_11.

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Orellana, Juan, and Alan H. Friedman. "Sickle Cell Disease." In Clinico-Pathological Atlas of Congenital Fundus Disorders. Springer New York, 1993. http://dx.doi.org/10.1007/978-1-4613-9320-7_19.

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Roth, Elliot J. "Sickle-Cell Disease." In Encyclopedia of Clinical Neuropsychology. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-56782-2_2197-2.

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Hood, Anna M., Emily A. McTate, Naomi E. Joffe, and Lori E. Crosby. "Sickle Cell Disease." In Clinical Handbook of Psychological Consultation in Pediatric Medical Settings. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-35598-2_26.

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Conference papers on the topic "SS sickle cell disease"

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Liang, Jiebin, Yunhyuk Chang, Jinhyeok Kim, Zhiyuan Li, John Canevari, and Sasan Haghani. "Sickle Cell Disease Patient/Provider Match Tool." In 2024 IEEE International Humanitarian Technologies Conference (IHTC). IEEE, 2024. https://doi.org/10.1109/ihtc61819.2024.10855129.

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Highland, Robert E., Cindy X. Chen, David A. Miller, Chao-Chieh Lin, Jen-Tsan A. Chi, and Adam Wax. "Examining sickle cell disease using high-throughput holographic cytometry." In Quantitative Phase Imaging XI, edited by YongKeun Park and Yang Liu. SPIE, 2025. https://doi.org/10.1117/12.3044015.

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Dhivya, R., Nivetha K, Rakshana A, Sudha B, and Vivetha Harini S. "Sickle Cell Disease Detection for Pregnant Women using ResNet." In 2025 7th International Conference on Intelligent Sustainable Systems (ICISS). IEEE, 2025. https://doi.org/10.1109/iciss63372.2025.11076312.

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Wu, Runxin, Mohammed Shihab Kabir, George A. Truskey, and Amanda Randles. "Investigating the impact of sickle cell disease on red blood cell transport in complex capillary networks." In 2024 46th Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC). IEEE, 2024. https://doi.org/10.1109/embc53108.2024.10781578.

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Krupp, N., and A. O. Delecaris. "Pulmonary Embolus and Golden Bronchoalveolar Lavage Fluid: A Rare and Late Presentation of Sickle Cell SS Disease." In American Thoracic Society 2022 International Conference, May 13-18, 2022 - San Francisco, CA. American Thoracic Society, 2022. http://dx.doi.org/10.1164/ajrccm-conference.2022.205.1_meetingabstracts.a1711.

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Glas-Greenwalt, P., J. Palascak, R. Gruppo, D. Stroop, and V. Pollak. "DEFECTIVE FIBRINOLYSIS IN SICKLE CELL DISEASE." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644838.

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Vasocclusive crises (VOC) cause significant morbidity and mortality in sickle cell disease (SCD). Although sickling is thought to be the predominant factor in VOC, investigators have examined the possible role of the hemostatic mechanism in the process. The data are, however, inconsistent. We studied, functionally with the fibrin plate method, the fibrinolytic system in 36 adults in the steady state and in 8 children, 7 of whom suffered from painful crises. Values in 240 normal blood donors were: tissue-type plasminogen activator activity (t-PA); 3-25 activator units/ml, corresponding to 0.04
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Hegemann, I., and J. Sangalli-Baruffaldi. "Evaluation of thrombin generation in patients with sickle cell disease in stable disease and sickle cell crises." In 65th Annual Meeting of the Society of Thrombosis and Haemostasis Research. Georg Thieme Verlag KG, 2021. http://dx.doi.org/10.1055/s-0041-1728172.

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Dautzenberg, M. D., F. Monge, A. M. Fischer, R. Girot, and P. Cornu. "COAGULATION AND FIBRINOLYSIS IN SICKLE CELL DISEASE." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643056.

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Sickled erythocytes appear to be primarily responsible for occlusion of microvasculature in patients with homozygous sickle cell disease (SCD), but it is unknown whether the activation of the coagulation pathway is also contributory to these vaso-occlusive crisis and other complications as leg ulcers, aseptic necrosis of bone, strokes. Thus, we studied coagulation and fibrinolysis parameters in 12 patients (ages 2 to 26 years with SCD, in steady-state, far from thrombotic events which occurred in 3 of them) to determine if it would be possible to detect a high-risk group for thrombosis. We wer
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Shah, A., S. Patel, R. Kaiser, J. Gorga, D. A. Buchnea, and R. Wadgaonkar. "The Effect of Sickle Cell Disease and Sickle Cell Trait on COVID-19 Infection Outcomes." In American Thoracic Society 2023 International Conference, May 19-24, 2023 - Washington, DC. American Thoracic Society, 2023. http://dx.doi.org/10.1164/ajrccm-conference.2023.207.1_meetingabstracts.a4902.

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Hosseini, Poorya, Sabia Z. Abidi, Gregory J. Kato, Ming Dao, Zahid Yaqoob, and Peter T. C. So. "Biophysical markers of Sickle Cell Disease at Individual Cell Level." In Cancer Imaging and Therapy. OSA, 2016. http://dx.doi.org/10.1364/cancer.2016.jtu3a.44.

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Reports on the topic "SS sickle cell disease"

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Santos Garcia, João Batista, and Ana Laura Schwartzmann Bruno. Pain in Sickle Cell Disease and Acute Varicella Zoster Pain. World Federation of Societies of Anaesthesiologists, 2024. https://doi.org/10.28923/atotw.537.

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This tutorial addresses the etiology and management of pain associated with sickle cell disease and acute pain related to varicella zoster. Both are severe pain conditions with a significant impact on individuals' functionality and the potential to cause substantial suffering.
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Paul, Satashree. Turning Back the Sickle Cell Disease: A New Drug into Play. Science Repository OÜ, 2021. http://dx.doi.org/10.31487/sr.blog.38.

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Researchers at the Fulcrum Therapeutics developed a bioavailable drug candidate called FTX 6058 – (a novel small molecular fetal haemoglobin inducer for sickle cell disease) that can restore the body’s ability to produce fetal haemoglobin
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Krishnamurti, Lakshmanan, Deepika Darbari, Victor Gordeuk, et al. Can Personalized Encouragement Help People With Sickle Cell Disease Take Hydroxyurea Therapy Regularly? Patient-Centered Outcomes Research Institute® (PCORI), 2020. http://dx.doi.org/10.25302/05.2020.ce.13046859em.

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Yu, Winifred W., Tanvi Reddy, and Melissa Barnhart. Dental Care for People With Sickle Cell Disease: A Rapid Response Literature Review. Agency for Healthcare Research and Quality (AHRQ), 2024. http://dx.doi.org/10.23970/ahrqepcrapid_dental_sicklecell.

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Lanzkron, Sophie, Jane Little, Joshua Field, et al. Comparing Pain Management for Sickle Cell Disease Crises in Emergency Rooms and Infusion Centers. Patient-Centered Outcomes Research Institute (PCORI), 2020. http://dx.doi.org/10.25302/08.2020.ihs.140311888.

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Krishnamurti, Lakshmanan, Diana Ross, Nitya Bakshi, Cynthia Brown Sinha, and Geoerge Loewenstein. An Online Decision Aid to Help Patients and Caregivers Decide on Treatments for Sickle Cell Disease. Patient-Centered Outcomes Research Institute® (PCORI), 2020. http://dx.doi.org/10.25302/1.2020.ce.12114318em.

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Marques, Carla, Larissa Lopes, Rita Lucena, and Abrahão Baptista. Brain morphofunctional changes associated with pain in children, adolescents and young adults with sickle cell disease. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.12.0022.

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Crosby, Lori, Constance Mara, Yolanda Johnson, Rogelle Hackworth, and Charles Quinn. Comparing Ways to Help Parents of Children with Sickle Cell Disease Decide on Treatment -- The ENGAGE HU Study. Patient-Centered Outcomes Research Institute (PCORI), 2024. http://dx.doi.org/10.25302/02.2024.cdr.160936055.

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Amponsah, Isaac, Denzel Opoku-Kwabi, Francis Ackah Armah, John Nii Addotey, Bernard Kofi Turkson, and Emmanuel Quaye Kontoh. A systematic review of validated medicinal plants and their compounds as agents for the management of sickle cell disease. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2024. http://dx.doi.org/10.37766/inplasy2024.4.0121.

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Desai, Payal, Raymona Lawrence, Myra Robinson, et al. Comparing the Effects of Peer Mentoring on Care Transitions in Emerging Adults with Sickle Cell Disease: The ST3P-UP Study. Patient-Centered Outcomes Research Institute (PCORI), 2024. http://dx.doi.org/10.25302/11.2024.mcsc.160835861.

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