Relevant bibliographies by topics / Stable isotopeS/lipoprotein kinetics/HDL metabolis

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  2. Dissertations / Theses

Academic literature on the topic 'Stable isotopeS/lipoprotein kinetics/HDL metabolis'

Author: Grafiati
Published: 4 June 2021
Last updated: 18 February 2022

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Journal articles on the topic "Stable isotopeS/lipoprotein kinetics/HDL metabolis"

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Whyte, Martin B., Fariba Shojaee-Moradie, Sharaf E. Sharaf, et al. "HDL-apoA-I kinetics in response to 16 wk of exercise training in men with nonalcoholic fatty liver disease." American Journal of Physiology-Endocrinology and Metabolism 318, no. 6 (2020): E839—E847. http://dx.doi.org/10.1152/ajpendo.00019.2020.

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Nonalcoholic fatty liver disease (NAFLD) is characterized by low-circulating concentration of high-density lipoprotein cholesterol (HDL-C) and raised triacylglycerol (TAG). Exercise reduces hepatic fat content, improves insulin resistance and increases clearance of very-low-density lipoprotein-1 (VLDL1). However, the effect of exercise on TAG and HDL-C metabolism is unknown. We randomized male participants to 16 wk of supervised, moderate-intensity aerobic exercise ( n = 15), or conventional lifestyle advice ( n = 12). Apolipoprotein A-I (apoA-I) and VLDL-TAG and apolipoprotein B (apoB) kineti
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CHAN, Dick C., P. Hugh R. BARRETT, and Gerald F. WATTS. "Lipoprotein transport in the metabolic syndrome: methodological aspects of stable isotope kinetic studies." Clinical Science 107, no. 3 (2004): 221–32. http://dx.doi.org/10.1042/cs20040108.

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The metabolic syndrome encapsulates visceral obesity, insulin resistance, diabetes, hypertension and dyslipidaemia. Dyslipidaemia is a cardinal feature of the metabolic syndrome that accelerates the risk of cardiovascular disease. It is usually characterized by high plasma concentrations of triacylglycerol (triglyceride)-rich and apoB (apolipoprotein B)-containing lipoproteins, with depressed concentrations of HDL (high-density lipoprotein). However, lipoprotein metabolism is complex and abnormal plasma concentrations can result from alterations in the rates of production and/or catabolism of
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CHAN, Dick C., P. Hugh R. BARRETT, and Gerald F. WATTS. "Lipoprotein transport in the metabolic syndrome: pathophysiological and interventional studies employing stable isotopy and modelling methods." Clinical Science 107, no. 3 (2004): 233–49. http://dx.doi.org/10.1042/cs20040109.

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The accompanying review in this issue of Clinical Science [Chan, Barrett and Watts (2004) Clin. Sci. 107, 221–232] presented an overview of lipoprotein physiology and the methodologies for stable isotope kinetic studies. The present review focuses on our understanding of the dysregulation and therapeutic regulation of lipoprotein transport in the metabolic syndrome based on the application of stable isotope and modelling methods. Dysregulation of lipoprotein metabolism in metabolic syndrome may be due to a combination of overproduction of VLDL [very-LDL (low-density lipoprotein)]-apo (apolipop
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Brämswig, Susanne, Anja Kerksiek, Thomas Sudhop, Claus Luers, Klaus Von Bergmann, and Heiner K. Berthold. "Carbamazepine increases atherogenic lipoproteins: mechanism of action in male adults." American Journal of Physiology-Heart and Circulatory Physiology 282, no. 2 (2002): H704—H716. http://dx.doi.org/10.1152/ajpheart.00580.2001.

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Treatment with carbamazepine (CBZ) affects cholesterol concentrations, but little is known about the precise nature and underlying mechanisms of changes in lipoprotein metabolism. We investigated prospectively the effects of CBZ on lipid metabolism in normolipemic adults. In 21 healthy males, lipoprotein and noncholesterol sterol concentrations were measured before and during treatment with CBZ for 70 ± 18 days. Thirteen subjects underwent kinetic studies of apolipoprotein-B (ApoB) metabolism with the use of endogenous stable isotope labeling. Lipoprotein kinetic parameters were calculated by
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Chan, Dick C., P. Hugh R. Barrett, Esther M. M. Ooi, Juying Ji, Doris T. Chan, and Gerald F. Watts. "Very Low Density Lipoprotein Metabolism and Plasma Adiponectin as Predictors of High-Density Lipoprotein Apolipoprotein A-I Kinetics in Obese and Nonobese Men." Journal of Clinical Endocrinology & Metabolism 94, no. 3 (2009): 989–97. http://dx.doi.org/10.1210/jc.2008-1457.

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Abstract Context: Hypercatabolism of high-density lipoprotein (HDL) apolipoprotein (apo) A-I results in low plasma apoA-I concentration. The mechanisms regulating apoA-I catabolism may relate to alterations in very low density lipoprotein (VLDL) metabolism and plasma adiponectin and serum amyloid A protein (SAA) concentrations. Objective: We examined the associations between the fractional catabolic rate (FCR) of HDL-apoA-I and VLDL kinetics, plasma adiponectin, and SAA concentrations. Study Design: The kinetics of HDL-apoA-I and VLDL-apoB were measured in 50 obese and 37 nonobese men using st
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Wang, Xuewen, Faidon Magkos, Bruce W. Patterson, Dominic N. Reeds, Janine Kampelman, and Bettina Mittendorfer. "Low-dose dexamethasone administration for 3 weeks favorably affects plasma HDL concentration and composition but does not affect very low-density lipoprotein kinetics." European Journal of Endocrinology 167, no. 2 (2012): 217–23. http://dx.doi.org/10.1530/eje-12-0180.

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ObjectiveSubclinical hypercortisolemia often occurs in subjects with features of the metabolic syndrome, and it has been suggested that it may be, at least in part, responsible for the development of these metabolic abnormalities. However, the metabolic effects of glucocorticoid administration to mimic subclinical glucocorticoid excess have not been evaluated.MethodsWe used stable isotope-labeled tracer methods in conjunction with magnetic resonance techniques to measure the effect of glucocorticoid excess within the physiological range (∼0.7 mg dexamethasone/day for 3 weeks) on glucose and fr
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Ng, Theodore W. K., Dick C. Chan, P. Hugh R. Barrett, and Gerald F. Watts. "Effect of weight loss on HDL-apoA-II kinetics in the metabolic syndrome." Clinical Science 118, no. 1 (2009): 79–85. http://dx.doi.org/10.1042/cs20090110.

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Reduced HDL (high-density lipoprotein) concentration in the MetS (metabolic syndrome) is associated with increased risk of cardiovascular disease and is related to defects in HDL-apoA-II (apolipoprotein A-II) kinetics. Dietary restriction is the most commonly used weight loss strategy. In the present study, we examined the effect of weight loss on HDL-apoA-II kinetics in men with the MetS at the start and end of a 16-week intervention trial of a hypocaloric low-fat diet (n=20) compared with a weight maintenance diet (n=15), using a stable isotope technique and compartmental modelling. The low-
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Magkos, Faidon, Bruce W. Patterson, and Bettina Mittendorfer. "No effect of menstrual cycle phase on basal very-low-density lipoprotein triglyceride and apolipoprotein B-100 kinetics." American Journal of Physiology-Endocrinology and Metabolism 291, no. 6 (2006): E1243—E1249. http://dx.doi.org/10.1152/ajpendo.00246.2006.

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Dyslipidemia, manifested by increased plasma triglyceride (TG), increased total and LDL-cholesterol concentrations and decreased HDL-cholesterol concentration, is an important risk factor for cardiovascular disease. Premenopausal women have a less atherogenic plasma lipid profile and a lower risk of cardiovascular disease than men, but this female advantage disappears after menopause. This suggests that female sex steroids affect lipoprotein metabolism. The impact of variations in the availability of ovarian hormones during the menstrual cycle on lipoprotein metabolism is not known. We therefo
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Lee, Anita Y. H., Nathan A. Yates, Marina Ichetovkin, et al. "Measurement of Fractional Synthetic Rates of Multiple Protein Analytes by Triple Quadrupole Mass Spectrometry." Clinical Chemistry 58, no. 3 (2012): 619–27. http://dx.doi.org/10.1373/clinchem.2011.172429.

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Abstract BACKGROUND Current approaches to measure protein turnover that use stable isotope-labeled tracers via GC-MS are limited to a small number of relatively abundant proteins. We developed a multiplexed liquid chromatography–selected reaction monitoring mass spectrometry (LC-SRM) assay to measure protein turnover and compared the fractional synthetic rates (FSRs) for 2 proteins, VLDL apolipoprotein B100 (VLDL apoB100) and HDL apoA-I, measured by both methods. We applied this technique to other proteins for which kinetics are not readily measured with GC-MS. METHODS Subjects were given a pr
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Telford, Dawn E., Amy C. Burke, Brian G. Sutherland, et al. "Abstract 394: Ldl-cholesterol, ApoB100 Kinetics and Atherosclerosis in Ldlr-deficient Yucatan Minipigs: A New Model for Familial Hypercholesterolemia." Arteriosclerosis, Thrombosis, and Vascular Biology 37, suppl_1 (2017). http://dx.doi.org/10.1161/atvb.37.suppl_1.394.

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Lack of animal models with human-like lipoprotein metabolism and pathology has hampered translational research in atherosclerosis. Recently, a model of familial hypercholesterolemia was developed in Yucatan miniature pigs, in which the LDL receptor (LDLR) was deleted through gene targeting of exon 4. The objective of the present study was to determine the plasma lipoprotein response to a high fat diet and the kinetics of apolipoprotein (apo) B metabolism in LDLR-deficient miniature pigs. LDLR+/+ (n=5), LDLR+/- pigs (n=6) and LDLR-/- pigs (n=5) were fed a diet containing 34% kcal from fat and 0
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Dissertations / Theses on the topic "Stable isotopeS/lipoprotein kinetics/HDL metabolis"

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Uliaque, Cugat Katia. "Implementation of stable isotopes lipoprotein kinetic studies: effects on HDL metabolism of a Mediterranean type diet rich in MUFAs from virgin olive oil." Doctoral thesis, Universitat Rovira i Virgili, 2007. http://hdl.handle.net/10803/8658.

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The anti-atherogenic effects ascribed to a Mediterranean-type diet rich in monounsaturated<br/>fatty acids (MUFAs) from virgin olive oil are due, partly, to an increase in, or maintenance of,<br/>plasma concentrations of high density lipoprotein (HDL) cholesterol. However, the underlying<br/>mechanisms that may explain these concentrations are not well characterised, to-date.<br/>Apolipoprotein (apo) A-I (apoA-I) is the major HDL apo and its kinetic parameters, such as<br/>production rate and catabolic rate, reflect the kinetics of the HDL particle. Our working<br/>hypothesis is as follows: a
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