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Books on the topic 'Status Epilepticus'

Author: Grafiati

Published: 4 June 2021

Last updated: 20 February 2023

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1

Drislane, Frank W., ed. Status Epilepticus. Totowa, NJ: Humana Press, 2005. http://dx.doi.org/10.1385/1592599451.

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2

Drislane, Frank W., and Peter W. Kaplan MBBS, eds. Status Epilepticus. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-58200-9.

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3

Stefan, Hermann, and Jürgen Bauer. Status epilepticus. Berlin, Heidelberg: Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-76027-3.

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4

Wang, Xuefeng, and Shichuo Li, eds. Refractory Status Epilepticus. Singapore: Springer Singapore, 2017. http://dx.doi.org/10.1007/978-981-10-5125-8.

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5

1951-, Kaplan Peter W., and Drislane Frank, eds. Nonconvulsive status epilepticus. New York: Demos Medical Pub., 2009.

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6

Status epilepticus: Its clinical features and treatment in children and adults. Cambridge: Cambridge University Press, 1994.

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7

Heida, James George. The extended efficacy of ketamine in the neuroprotection of status epilepticus induced neuronal necrosis. Sudbury, Ont: Laurentian University, Behavioural Neuroscience Program, 2001.

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8

Stewart, Lee S. Administration of the N-Methyl-D-Apartate (NMDA) calcium channel antagonist pentamidine isethionate during lithium and pilocarpine induced status epilepticus: Neroprotective efficacy in vivo. Sudbury, Ont: Laurentian University, Behavioural Neuroscience Program, 1997.

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9

A, Beaumanoir, ed. Continuous spikes and waves during slow sleep, electrical status epilepticus during slow sleep: Acquired epileptic aphasia and related conditions : colloquium of the Pierfranco e Luisa Mariani Foundation, Milan, in cooperation with International School of Neurological Sciences, San Servolo, Venice, 15-16 October 1993. London: J. Libbey, 1995.

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10

Seized: Searching for health in the United States. Tamarac, FL: Llumina Press, 2013.

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11

Jill, Robinson. Past forgetting: My memory lost and found. New York: Cliff Street Books, 1999.

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12

Jeff, Welsch, ed. Going the distance: The George Thomas story. Champaign, Ill: Sports Publishing L.L.C., 2002.

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13

Delorenzo. Status Epilepticus. Butterworth-Heinemann Medical, 2007.

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14

Bauer, Jürgen, and Hermann Stefan. Status Epilepticus. Springer London, Limited, 1990.

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15

Bauer, Jürgen, and Hermann Stefan. Status Epilepticus. Springer London, Limited, 2013.

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16

Treiman, David M., and Nancy Y. Walton. Status Epilepticus. Lippincott Williams & Wilkins, 1999.

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17

Wasterlain, Claude, and David Treiman, eds. Status Epilepticus. The MIT Press, 2006. http://dx.doi.org/10.7551/mitpress/6456.001.0001.

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18

Kaplan, Peter W., and Frank W. Drislane. Nonconvulsive Status Epilepticus. Springer Publishing Company, Incorporated, 2009.

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19

Drislane, Frank W., Susan T. Herman, and Peter W. Kaplan. Convulsive Status Epilepticus. Edited by Donald L. Schomer and Fernando H. Lopes da Silva. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190228484.003.0020.

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Generalized convulsive status epilepticus (GCSE) is a serious neurologic illness causing unresponsiveness, major physiologic disturbances, risk of injury and, if prolonged enough, neuronal damage. Causes are many, and the outcome often depends as much on the etiology as on the epileptic seizure itself. Several anti-seizure medications are used in treatment of GCSE, but some cases continue electrographically when clinical convulsions cease (nonconvulsive SE), and EEG is essential in their diagnosis. About 20% of cases become refractory to initial treatment, and the EEG becomes even more crucial in diagnosis and management. This chapter also covers other forms of SE with significant motor manifestations including: focal motor status (including epilepsia partialis continua); myoclonic status, which includes some relatively benign forms as well as some with a very poor prognosis; and clonic and tonic status. It reviews the many different EEG findings in those forms of status, and the use of EEG in their treatment and management, especially in prolonged cases.

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20

Drislane, Frank W., Susan T. Herman, and Peter W. Kaplan. Nonconvulsive Status Epilepticus. Edited by Donald L. Schomer and Fernando H. Lopes da Silva. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190228484.003.0021.

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The clinical presentation and encephalographic (EEG) findings of nonconvulsive status epilepticus (NCSE) can be complicated, making diagnosis difficult. There are generalized (e.g., absence status) and focal (e.g., aphasic status, complex partial status) forms. Some patients are responsive but have cognitive or other neurologic deficits; others are less responsive or even comatose. Increasingly, the diagnosis of NCSE is considered in intensive care unit patients. Here, without clinical signs of seizures such as convulsions, EEG is critical in diagnosis, but there is uncertainty about which EEG patterns represent seizures and which clinical situations and EEG patterns warrant aggressive treatment. Antiseizure medications are tailored to the NCSE type and the clinical condition. Treatment is often easier for NCSE, and the outcome better, than for convulsive SE, but this is not always true for critically ill patients with NCSE in the ICU, for whom continuous EEG monitoring is often crucial for diagnosis and management.

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21

PILBERY. Standby Cpd: Status Epilepticus. Class Publishing, 2012.

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22

Treiman. Status Epilepticus: 100 Maxims. Hodder Education Group, 1996.

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23

Aggarwala, Neha, and Pirjo H. Manninen. Seizures and Status Epilepticus. Edited by Matthew D. McEvoy and Cory M. Furse. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190226459.003.0065.

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Seizures are common with 5%–10% of the general population experiencing a seizure in their lifetime and 1%–3% having a seizure disorder. Patients undergoing neurosurgical procedures are at an increased risk of perioperative seizures, including seizures occurring during surgery. To prevent and minimize perioperative seizures, the preoperative optimization of patients with seizures is crucial, including administration of regular antiepileptic agents. Status epilepticus is a true neurological emergency, with early and aggressive treatment being imperative for successful management. This chapter reviews the diagnosis, risk factors for seizures, and the initial treatment of an acute perioperative seizure as well as the management of status epilepticus.

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24

Taneja, Pravin. Seizures and Status Epilepticus. Edited by Matthew D. McEvoy and Cory M. Furse. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190226459.003.0082.

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Epilepsy is one of the most prevalent chronic neurological disorders both in the U.S. and worldwide, effecting ~1% of the world population. As a result, patients with a history of epilepsy frequently present for surgery. It is important to be aware of how to best characterize the severity of the disease, and treat seizures should they be encountered. In this chapter, we review the epidemiology and pathophysiology of epilepsy. We also discuss the most severe and life threatening form of the condition, status epilepticus. In this emergency situation it is critical that seizures be terminated, and supportive care given. We delineate initial treatment steps and medications, in addition to providing recommendations for refractory cases.

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25

Status epilepticus: Mechanisms and management. Cambridge, MA: MIT Press, 2006.

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26

Drislane, Frank W., and Peter W. Kaplan MBBS. Status Epilepticus: A Clinical Perspective. Springer, 2018.

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27

Drislane, Frank W., and Peter W. Kaplan MBBS. Status Epilepticus: A Clinical Perspective. Springer, 2017.

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28

Drislane, Frank W. Status Epilepticus: A Clinical Perspective. Humana Press Inc.,U.S., 2005.

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29

Nabbout, Rima. Ketogenic Diet in Status Epilepticus. Edited by Eric H. Kossoff. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190497996.003.0009.

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Refractory status epilepticus (RSE) is associated with a significant risk of death or neurological morbidity. The ketogenic diet (KD) is a dietary therapy that succeeds in controlling seizures in otherwise RSE in children and adult patients. Inflammatory etiologies might be a particular target, but KD has reported efficacy in other etiologies. KD is well tolerated, and the effect is seen within days. Randomized and controlled studies are lacking in this area, and studies are needed to prove the efficacy of KD in RSE and to identify specific indications. This would help to increase its use and to implement it in intensive care units to resolve severe epileptic conditions quickly.

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30

Wasterlain, Claude, and David Treiman. Status Epilepticus: Mechanisms and Management. MIT Press, 2006.

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31

Frank, Drislane, ed. Status epilepticus: A clinical perspective. Totowa, N.J: Humana Press, 2005.

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32

(Editor), Claude G. Wasterlain, and David M. Treiman (Editor), eds. Status Epilepticus: Mechanisms and Management. The MIT Press, 2006.

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33

Frank, Drislane, ed. Status epilepticus: A clinical perspective. Totowa, N.J: Humana Press, 2005.

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34

G, Wasterlain Claude, and Treiman David M, eds. Status epilepticus: Mechanisms and management. Cambridge, Mass: MIT Press, 2006.

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35

Drislane, Frank W. Status Epilepticus: A Clinical Perspective. Humana, 2010.

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36

Walker, Matthew C. Convulsive and non-convulsive status epilepticus. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199688395.003.0030.

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This chapter describes the definition, epidemiology, classification, diagnosis, and treatment of status epilepticus, concentrating on the roles that electroencephalography (EEG) plays. The term status epilepticus now encompasses a range of conditions from continuous convulsive seizures to clinically subtle non-convulsive seizures, which may manifest as changes in behaviour or personality. EEG is critical for the diagnosis of non-convulsive status epilepticus. Furthermore, the progression of convulsive status epilepticus is to an electromechanical dissociation in which continuous electrical seizure activity may have no or minimal clinical manifestations. In the later stages of status epilepticus, EEG is necessary to monitor treatment, but is confounded by the interpretation of periodic EEG patterns, which represent a continuum from interictal through to ictal activity. Post-status epilepticus EEG patterns have prognostic value: periodic epileptiform discharges, burst suppression patterns (off anaesthesia) and repetitive seizure activity are indicative of a poor long-term prognosis.

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37

Wang, Xuefeng, and Shichuo Li. Refractory Status Epilepticus: Diagnosis and Treatment. Springer, 2017.

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38

Radhakrishnan, Ashalatha. Status Epilepticus: Practical Guidelines in Management. Jaypee Brothers Medical Publishers, 2017.

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39

Wang, Xuefeng, and Shichuo Li. Refractory Status Epilepticus: Diagnosis and Treatment. Springer, 2017.

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40

Wang, Xuefeng, and Shichuo Li. Refractory Status Epilepticus: Diagnosis and Treatment. Springer, 2018.

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41

Drislane, Frank W. Status Epilepticus: A Clinical Perspective (Current Clinical Neurology). Humana Press, 2005.

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42

Kirmani, Batool F., Ashok K. Shetty, and Lee A. Shapiro, eds. New Directions in the Management of Status Epilepticus. Frontiers Media SA, 2019. http://dx.doi.org/10.3389/978-2-88945-727-4.

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43

Bauer, Jürgen, and Marianne Neumann. Notfälle in der Epileptologie: Status epilepticus, pharmakogene und morbogene Zwischenfälle. Steinkopff-Verlag Darmstadt, 2006.

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44

Shorvon, Simon. Status Epilepticus: Its Clinical Features and Treatment in Children and Adults. Cambridge University Press, 2011.

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45

Status Epilepticus: Its Clinical Features and Treatment in Children and Adults. Cambridge University Press, 2006.

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46

Shorvon, Simon. Status Epilepticus: Its Clinical Features and Treatment in Children and Adults. Cambridge University Press, 2009.

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47

Howard, Dr Robin, Dr Thomasin C. Andrews, Dr Robin Howard, Dr Paul Holmes, Dr Robin Howard, Professor Michael Koutroumanidis, Professor Michael Koutroumanidis, and Dr Robin Howard. Neurological diseases and emergencies. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199565979.003.0007.

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Chapter 7 discusses neurological diseases and emergencies, including headache, transient loss of consciousness, states of impaired consciousness, the dementias, gait and disturbances of speech, stroke, neuro-ophthalmology, epilepsies and epileptic states, status epilepticus in adults, infections of the nervous system, demyelinating diseases, neuromuscular disease, movement disorders (disorders of the extrapyramidal system), neuro-oncology, cranial nerve disorders, spinal cord lesions, and toxic and nutritional disease.

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48

Wijdicks, Eelco F. M., and Sarah L. Clark. Antiepileptic Drugs. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190684747.003.0006.

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Patients are often admitted to the neurosciences intensive care unit specifically for the treatment of a series of seizures or focal status epilepticus. Far more urgent is convulsive status epilepticus, which requires multiple intravenously administered antiepileptic drugs. The choices are different when the patient is in status epilepticus and refractoriness is mostly due to pharmacologic resistance. Use of an algorithm for decision making is common in the management of refractory status epilepticus. Antiepileptic drugs often interact with other antiepileptic drugs. This chapter discusses the use of antiepileptic drugs available to treat seizures, recurrent seizures, status epilepticus, and refractory status epilepticus.

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49

Nageshwaran, Sathiji, Heather C. Wilson, Anthony Dickenson, and David Ledingham. Epilepsy. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199664368.003.0003.

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This chapter on epilepsy discusses the major classification of epileptic disorders, the management and evidence base for treatment of chronic epilepsy (focal-onset and generalized seizures), the use of antiepileptic drugs in young women, withdrawal of antiepileptic drugs, and the management of status epilepticus.

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50

Tassinari, Carlo Alberto, and Guido Rubboli. Encephalopathy Related to Status Epilepticus During Slow Sleep: Linking Epilepsy, Sleep Disruption and Cognitive Impairment. Editions John Libbey Eurotext, 2019.

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