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1

Kuypers, Friedhelm, and Christian Ucke. "Steh' auf Kreisel!" Physik in unserer Zeit 25, no. 5 (1994): 214–15. http://dx.doi.org/10.1002/piuz.19940250503.

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2

Bester, Dörte. "Steh auf und geh!" Göttinger Predigtmeditationen 63, no. 4 (2008): 417–22. http://dx.doi.org/10.13109/gpre.2008.63.4.417.

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3

Bijak, M., M. Rakos, C. Hofer, et al. "STEH- UND GEHSYSTEM FÜR SPASTISCH GELÄHMTE PARAPLEGIKER." Biomedizinische Technik/Biomedical Engineering 48, s1 (2003): 42–43. http://dx.doi.org/10.1515/bmte.2003.48.s1.42.

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4

Brenner, Helmut. ""Wo i geh und steh": Entstehung, Entwicklung, Varianten." Jahrbuch für Volksliedforschung 42 (1997): 38. http://dx.doi.org/10.2307/848004.

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5

Quentin, B., and J. Wissel. "Remobilisierung nach Schlaganfall." Nervenheilkunde 29, no. 09 (2010): 537–44. http://dx.doi.org/10.1055/s-0038-1628813.

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ZusammenfassungBehandlungen zur Remobilisation nach Schlaganfall haben sich weiterentwickelt. Zur Wiederherstellung der Steh- und Gehfähigkeit sollten so früh wie möglich im multiprofessionellen Team organisierte aktive Behandlungen angeboten werden. Diese haben die frühe Vertikalisierung sowie Kreislauf- und Koordinationstraining und Elemente des aktiven Gehtrainings mit möglichst hoher Schrittwiederholungszahl zum Inhalt. Hierzu können Therapieeinheiten der Lokomotionstherapie auf dem Laufband oder Gangtrainer mit Sturzsicherung in einem Fallschirmgurtsystem und partieller Gewichtsentlastung
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6

Durth, K. Rüdiger. "Prisma – “Steh auf und flieh.” – Die Kirchen und die Flüchtlinge." Homiletische Monatshefte 91, no. 7 (2016): 346–50. http://dx.doi.org/10.13109/homh.2015.91.7.346.

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7

Müller, H., Y. Lampl, and M. Haefele. "Ein TCD-Steh-Test zur klinischen Prüfung der zerebralen Autoregulation." Ultraschall in der Medizin 12, no. 05 (2008): 218–21. http://dx.doi.org/10.1055/s-2007-1005933.

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8

Huber, Martin, and Claudia Pott. "Standpunkte – Gehfähigkeit richtig trainieren." physiopraxis 16, no. 11/12 (2018): 34–39. http://dx.doi.org/10.1055/a-0657-0509.

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Wie schaffe ich es, dass mein Patient gut und schnell gehfähig wird? Trainiere ich dafür nur das Gehen oder auch das Stehen? Die Zusammenhänge zwischen Steh- und Gehfähigkeit sind verdammt komplex, und die Antwort ist nicht mit zwei Sätzen gegeben. Und da die Antworten durchaus unterschiedlich ausfallen, haben wir zwei Experten der Neurorehabilitation gebeten, ihren Standpunkt zu diskutieren.
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9

Hafner, Ottfried. ""Wo i geh und steh..." Erzherzog Johann zwischen Volksmusik und Folklore." Jahrbuch für Volksliedforschung 39 (1994): 20. http://dx.doi.org/10.2307/848625.

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10

Heldman, Marilyn E. "Steh auf und geh nach Süden / Arise and go toward the South." Aethiopica 12 (April 8, 2012): 280–81. http://dx.doi.org/10.15460/aethiopica.12.1.122.

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11

Becker, Ina, Birgit Wallmann-Sperlich, Robert Rupp, and Jens Bucksch. "Interventionen zur Reduzierung sitzenden Verhaltens am Büroarbeitsplatz – eine systematische Literaturanalyse." Das Gesundheitswesen 81, no. 08/09 (2017): 606–14. http://dx.doi.org/10.1055/s-0043-112746.

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Zusammenfassung Hintergrund Sitzendes Verhalten wird zunehmend als ein eigenständiger gesundheitlicher Risikofaktor eingeschätzt. Ein wesentliches Handlungsfeld für Interventionen zur Reduzierung sitzenden Verhaltens stellt deshalb der Büroarbeitsplatz dar. Dieses systematische Review untersucht, ob sitz- und/oder bewegungsspezifische Interventionen die Sitzzeiten von Büroangestellten am Arbeitsplatz reduzieren. Methoden Von April bis Mai 2016 wurde in den Datenbanken „PubMed“, „PsycINFO“ und „SPORTDiscus“ eine systematische Literaturrecherche durchgeführt. Insgesamt ließen sich 17 Studien ide
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12

Rödl, R. "Orthopädische Behandlung neuromuskulärer Systemerkrankungen – nicht nur im Kindesalter." Nervenheilkunde 27, no. 04 (2008): 277–83. http://dx.doi.org/10.1055/s-0038-1627251.

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ZusammenfassungDurch die sich entwickelnden Muskelungleichgewichte führen neuromuskuläre Systemerkrankungen zu Deformitäten und Funktionsverlust. Krankengymnastik und Orthesen können den natürlichen Verlauf verlangsamen und Defizite wie den Spitzfuß ausgleichen. Eine Verbesserung oder Beseitigung der Stellungs- und Kraftverluste ist jedoch nicht möglich. Daher ist insbesondere bei der Skolioseentwicklung eine frühzeitige operative Intervention erforderlich, da durch den weiteren Progress die Atemkapazität sich weiter vermindert, was zu Inoperabilität und Verkürzung der Lebenserwartung führt. A
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13

Hasson, M. S., D. Blinder, J. Thorner, and D. D. Jenness. "Mutational activation of the STE5 gene product bypasses the requirement for G protein beta and gamma subunits in the yeast pheromone response pathway." Molecular and Cellular Biology 14, no. 2 (1994): 1054–65. http://dx.doi.org/10.1128/mcb.14.2.1054.

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The STE5 gene encodes an essential element of the pheromone response pathway which is known to act either after the G subunit encoded by the STE4 gene or at the same step. Mutations in STE5, designated STE5Hyp, that partially activate the pathway in the absence of pheromone were isolated. One allele (STE5Hyp-2) was shown to cause a single amino acid substitution near the N terminus of the predicted STE5 protein. Immunoblotting with anti-Ste5 antibodies indicated that the phenotype was not due to an increased level of the mutant STE5 protein. A multicopy episomal plasmid containing a STE5Hyp al
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14

Hasson, M. S., D. Blinder, J. Thorner, and D. D. Jenness. "Mutational activation of the STE5 gene product bypasses the requirement for G protein beta and gamma subunits in the yeast pheromone response pathway." Molecular and Cellular Biology 14, no. 2 (1994): 1054–65. http://dx.doi.org/10.1128/mcb.14.2.1054-1065.1994.

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The STE5 gene encodes an essential element of the pheromone response pathway which is known to act either after the G subunit encoded by the STE4 gene or at the same step. Mutations in STE5, designated STE5Hyp, that partially activate the pathway in the absence of pheromone were isolated. One allele (STE5Hyp-2) was shown to cause a single amino acid substitution near the N terminus of the predicted STE5 protein. Immunoblotting with anti-Ste5 antibodies indicated that the phenotype was not due to an increased level of the mutant STE5 protein. A multicopy episomal plasmid containing a STE5Hyp al
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15

Nakayama, N., Y. Kaziro, K. Arai, and K. Matsumoto. "Role of STE genes in the mating factor signaling pathway mediated by GPA1 in Saccharomyces cerevisiae." Molecular and Cellular Biology 8, no. 9 (1988): 3777–83. http://dx.doi.org/10.1128/mcb.8.9.3777.

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The ste mutants (ste2, ste4, ste5, ste7, ste11, and ste12) are insensitive to mating factors and are, therefore, sterile. Roles of the STE gene products in the GPA1-mediated mating factor signaling pathway were studied by using ste gpa1 double mutants. Mating efficiency of a ste2 mutant defective in the alpha-factor receptor increased 1,000-fold in a gpa1 background, while G1 arrest and aberrant morphology (shmoo) caused by gpa1 were not suppressed by ste2. Furthermore, the steady-state level of the FUS1 transcript, which normally increases in response to mating factors, was also elevated when
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16

Nakayama, N., Y. Kaziro, K. Arai, and K. Matsumoto. "Role of STE genes in the mating factor signaling pathway mediated by GPA1 in Saccharomyces cerevisiae." Molecular and Cellular Biology 8, no. 9 (1988): 3777–83. http://dx.doi.org/10.1128/mcb.8.9.3777-3783.1988.

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The ste mutants (ste2, ste4, ste5, ste7, ste11, and ste12) are insensitive to mating factors and are, therefore, sterile. Roles of the STE gene products in the GPA1-mediated mating factor signaling pathway were studied by using ste gpa1 double mutants. Mating efficiency of a ste2 mutant defective in the alpha-factor receptor increased 1,000-fold in a gpa1 background, while G1 arrest and aberrant morphology (shmoo) caused by gpa1 were not suppressed by ste2. Furthermore, the steady-state level of the FUS1 transcript, which normally increases in response to mating factors, was also elevated when
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17

Singer, Juliette, and Hélène Singer-Delaunay. "“Ich steh auf Berlin”. Entretien avec Blixa Bargeld, chanteur et leader du groupe Einstürzende Neubauten." Ligeia N° 101-104, no. 2 (2010): 146. http://dx.doi.org/10.3917/lige.101.0146.

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18

Adam, Christine, and Helmut Brenner. "Gehundsteh Herzsoweh. Erzherzog-Johann-Liedtraditionen vor, in, neben und nach "Wo i geh und steh"." Jahrbuch für Volksliedforschung 42 (1997): 152. http://dx.doi.org/10.2307/848029.

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19

von Pückler, Kerstin, Carola Tillmanns, Valérie Bornand, Martin Kramer, Martin Schmidt, and Norbert Langen. "Spinales Nephroblastom in ungewöhnlicher Lokalisation bei einem Hund." Tierärztliche Praxis Ausgabe K: Kleintiere / Heimtiere 45, no. 02 (2017): 115–21. http://dx.doi.org/10.15654/tpk-151008.

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ZusammenfassungEin 7 Monate alter, männlicher Otterhund wurde aufgrund einer progressiven Lähmung der Hintergliedmaßen vorgestellt. Die klinischneurologische Untersuchung ergab eine Paraparese mit erhaltener Steh- und Gehfähigkeit sowie einer linksseitig betonten Läsion im Bereich des unteren motorischen Neurons der Hintergliedmaße (L4–S1). Mittels Magnetresonanztomographie wurde eine intradurale extramedulläre Neoplasie auf Höhe des fünften Lendenwirbels diagnostiziert. Bei der chirurgischen Darstellung zeigte sich eine partielle Infiltration des Rückenmarks durch die Neoplasie. Die mit Ausna
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20

Otto, Ilona M., Jonathan F. Donges, Roger Cremades, et al. "Social tipping dynamics for stabilizing Earth’s climate by 2050." Proceedings of the National Academy of Sciences 117, no. 5 (2020): 2354–65. http://dx.doi.org/10.1073/pnas.1900577117.

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Safely achieving the goals of the Paris Climate Agreement requires a worldwide transformation to carbon-neutral societies within the next 30 y. Accelerated technological progress and policy implementations are required to deliver emissions reductions at rates sufficiently fast to avoid crossing dangerous tipping points in the Earth’s climate system. Here, we discuss and evaluate the potential of social tipping interventions (STIs) that can activate contagious processes of rapidly spreading technologies, behaviors, social norms, and structural reorganization within their functional domains that
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21

Drebes, Gerald. "Schütz, Monteverdi und die "Vollkommenheit der Musik" : 'Es steh Gott auf' aus den 'Symphoniae sacrae II' (1647)." Schütz-Jahrbuch 14 (August 21, 2017): 25–55. http://dx.doi.org/10.13141/sjb.v1992774.

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22

Jenness, D. D., B. S. Goldman, and L. H. Hartwell. "Saccharomyces cerevisiae mutants unresponsive to alpha-factor pheromone: alpha-factor binding and extragenic suppression." Molecular and Cellular Biology 7, no. 4 (1987): 1311–19. http://dx.doi.org/10.1128/mcb.7.4.1311.

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Mutations in six genes that eliminate responsiveness of Saccharomyces cerevisiae a cells to alpha-factor were examined by assaying the binding of radioactively labeled alpha-factor to determine whether their lack of responsiveness was due to the absence of alpha-factor receptors. The ste2 mutants, known to be defective in the structural gene for the receptor, were found to lack receptors when grown at the restrictive temperature; these mutations probably affect the assembly of active receptors. Mutations in STE12 known to block STE2 mRNA accumulation also resulted in an absence of receptors. M
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23

Jenness, D. D., B. S. Goldman, and L. H. Hartwell. "Saccharomyces cerevisiae mutants unresponsive to alpha-factor pheromone: alpha-factor binding and extragenic suppression." Molecular and Cellular Biology 7, no. 4 (1987): 1311–19. http://dx.doi.org/10.1128/mcb.7.4.1311-1319.1987.

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Mutations in six genes that eliminate responsiveness of Saccharomyces cerevisiae a cells to alpha-factor were examined by assaying the binding of radioactively labeled alpha-factor to determine whether their lack of responsiveness was due to the absence of alpha-factor receptors. The ste2 mutants, known to be defective in the structural gene for the receptor, were found to lack receptors when grown at the restrictive temperature; these mutations probably affect the assembly of active receptors. Mutations in STE12 known to block STE2 mRNA accumulation also resulted in an absence of receptors. M
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24

Schäfer, M., and L. Döderlein. "Rehabilitation von Kindern und Jugendlichen mit angeborenen Gliedmaßendefekten." Kinder- und Jugendmedizin 10, no. 07 (2010): 395–402. http://dx.doi.org/10.1055/s-0038-1630861.

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ZusammenfassungAngeborene Gliedmaßendefekte der oberen und unteren Extremitäten sind selten. Das Spektrum der Erscheinungsformen reicht dabei von einer nur gering störenden Deformität eines Fingers oder einer Zehe bis hin zum vollständigen Fehlen einer oder mehrerer Gliedmaßen.Entsprechend unterschiedlich und individuell ausgerichtet muss auch die Rehabilitation in einem multiprofessionellen Team sein. Das Wachstum neigt dazu, die bereits bestehenden Deformitäten und Einschränkungen oft zusätzlich zu verschlechtern, weshalb operative Rekonstruktionen vor einer technischen Versorgung notwendig
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25

Cross, F. R. "The DAF2-2 mutation, a dominant inhibitor of the STE4 step in the alpha-factor signaling pathway of Saccharomyces cerevisiae MAT alpha cells." Genetics 126, no. 2 (1990): 301–8. http://dx.doi.org/10.1093/genetics/126.2.301.

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Abstract A dominant mutation (DAF2-2) resulting in resistance to the mating pheromone alpha-factor in Saccharomyces cerevisiae MATa cells was identified and characterized genetically. Whereas wild-type cells induce a high level of the FUS1 mRNA from a low baseline on exposure to alpha-factor, DAF2-2 cells were constitutive producers of an intermediate level of FUS1 RNA; the level was increased only modestly by alpha-factor. FUS1 constitutivity required STE4, STE5 and STE18, but did not require STE2, the alpha-factor receptor gene. DAF2-2 suppressed the alpha-factor supersensitivity of a STE2 C
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26

Fuchs, Susanne. ""So steh' ich denn hier wehrlos gegen dich?": —Figures of Armament and Disarmament in German Drama before and after the French Revolution." Goethe Yearbook 25, no. 1 (2018): 239–66. http://dx.doi.org/10.1353/gyr.2018.0012.

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27

Inouye, Carla, Namrita Dhillon, Tim Durfee, Patricia C. Zambryski, and Jeremy Thorner. "Mutational Analysis of STE5 in the Yeast Saccharomyces cerevisiae: Application of a Differential Interaction Trap Assay for Examining Protein-Protein Interactions." Genetics 147, no. 2 (1997): 479–92. http://dx.doi.org/10.1093/genetics/147.2.479.

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Ste5 is essential for the yeast mating pheromone response pathway and is thought to function as a scaffold that organizes the components of the mitogen-activated protein kinase (MAPK) cascade. A new method was developed to isolate missense mutations in Ste5 that differentially affect the ability of Ste5 to interact with either of two MAPK cascade constituents, the MEKK (Ste11) and the MEK (Ste7). Mutations that affect association with Ste7 or with Ste11 delineate discrete regions of Ste5 that are critical for each interaction. Co-immunoprecipitation analysis, examining the binding in vitro of
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28

Katz, Margaret E., Jill Ferguson, and Steven I. Reed. "Temperature-Sensitive Lethal Pseudorevertants of ste Mutations in Saccharomyces cerevisiae." Genetics 115, no. 4 (1987): 627–36. http://dx.doi.org/10.1093/genetics/115.4.627.

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ABSTRACT A procedure was devised to isolate mutations that could restore conjugational competence to temperature sensitive ste mutants and simultaneously confer temperature-sensitive lethal growth phenotypes. Three such mutations, falling into two complementation groups, were identified on the basis of suppression of ste5 alleles. These same mutations were later shown to be capable of suppressing ste4 and ste7 alleles. Five mutations in a single complementation group were isolated as suppressors of ste2 alleles. None of the mutations described in this study conferred a homogeneous cell cycle a
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29

David, Rachel. "SteC actin rearrangements, step by step." Nature Reviews Microbiology 11, no. 1 (2012): 5. http://dx.doi.org/10.1038/nrmicro2936.

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30

Douay, Luc. "Hematopoietic Stem Cell Protocols: Hematopoietic stem cell analysis ‘step by step’." Trends in Immunology 23, no. 9 (2002): 464. http://dx.doi.org/10.1016/s1471-4906(02)02271-8.

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31

GICQUEL, J., and HS DUA. "Step by step limbal stem cell transplantation techniques." Acta Ophthalmologica 91 (August 2013): 0. http://dx.doi.org/10.1111/j.1755-3768.2013.2631.x.

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32

Sette, Claudio, Carla J. Inouye, Shannon L. Stroschein, Phillip J. Iaquinta, and Jeremy Thorner. "Mutational Analysis Suggests That Activation of the Yeast Pheromone Response Mitogen-activated Protein Kinase Pathway Involves Conformational Changes in the Ste5 Scaffold Protein." Molecular Biology of the Cell 11, no. 11 (2000): 4033–49. http://dx.doi.org/10.1091/mbc.11.11.4033.

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Ste5 is essential for pheromone response and binds components of a mitogen-activated protein kinase (MAPK) cascade: Ste11 (MEKK), Ste7 (MEK), and Fus3 (MAPK). Pheromone stimulation releases Gβγ (Ste4-Ste18), which recruits Ste5 and Ste20 (p21-activated kinase) to the plasma membrane, activating the MAPK cascade. A RING-H2 domain in Ste5 (residues 177–229) negatively regulates Ste5 function and mediates its interaction with Gβγ. Ste5(C177A C180A), carrying a mutated RING-H2 domain, cannot complement a ste5Δ mutation, yet supports mating even in ste4Δ ste5Δ cells when artificially dimerized by f
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33

Velardi, Andrea. "Haploidentical Hematopoietic Stem Cell Transplantation: Step-by-Step Progress." Biology of Blood and Marrow Transplantation 21, no. 4 (2015): 579–80. http://dx.doi.org/10.1016/j.bbmt.2015.02.004.

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34

Schrick, Kathrin, Barbara Garvik, and Leland H. Hartwell. "Mating in Saccharomyces cerevisiae: The Role of the Pheromone Signal Transduction Pathway in the Chemotropic Response to Pheromone." Genetics 147, no. 1 (1997): 19–32. http://dx.doi.org/10.1093/genetics/147.1.19.

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Abstract The mating process in yeast has two distinct aspects. One is the induction and activation of proteins required for cell fusion in response to a pheromone signal; the other is chemotropism, i.e., detection of a pheromone gradient and construction of a fusion site available to the signaling cell. To determine whether components of the signal transduction pathway necessary for transcriptional activation also play a role in chemotropism, we examined strains with null mutations in components of the signal transduction pathway for diploid formation, prezygote formation and the chemotropic p
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35

Tompers, Dennie M., and Patricia A. Labosky. "Electroporation of Murine Embryonic Stem Cells: A Step-by-Step Guide." STEM CELLS 22, no. 3 (2004): 243–49. http://dx.doi.org/10.1634/stemcells.22-3-243.

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36

Goodman, Laurie. "States step in for stem cell research." Journal of Clinical Investigation 113, no. 10 (2004): 1376–77. http://dx.doi.org/10.1172/jci21950.

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37

Khalid, Ahmad Khudzairi, Nurul Balqis Nor’rah, Norbaiti Tukiman, and CT Munnirah Niesha Mohd Shafee. "THE ROLE OFISTEM VOLUNTEERISM TOWARDS iV-STEM MODULE THROUGH PRACTICAL SKILLS IN THE STEM SKILL INFORMATION ONLINE." International Journal of Education, Psychology and Counseling 6, no. 40 (2021): 168–79. http://dx.doi.org/10.35631/ijepc.640014.

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In the context of the Covid-19 pandemic that is sweeping the world today, volunteering continues to be an important agenda in the country's development mission. The participation of the community, especially the younger generation in volunteer activities is very important in creating an empathetic community. Therefore, to produce a national group that has a clear and far-sighted goal, it is very appropriate if the youths can continue to engage in volunteer activities. Nowadays, volunteering in the field of STEM education is very rare we hear, especially its implementation among university stud
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38

Whiteway, M., L. Hougan, and D. Y. Thomas. "Overexpression of the STE4 gene leads to mating response in haploid Saccharomyces cerevisiae." Molecular and Cellular Biology 10, no. 1 (1990): 217–22. http://dx.doi.org/10.1128/mcb.10.1.217.

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The STE4 gene of Saccharomyces cerevisiae encodes the beta subunit of the yeast pheromone receptor-coupled G protein. Overexpression of the STE4 protein led to cell cycle arrest of haploid cells. This arrest was like the arrest mediated by mating pheromones in that it led to similar morphological changes in the arrested cells. The arrest occurred in haploid cells of either mating type but not in MATa/MAT alpha diploids, and it was suppressed by defects in genes such as STE12 that are needed for pheromone response. Overexpression of the STE4 gene product also suppressed the sterility of cells d
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Whiteway, M., L. Hougan, and D. Y. Thomas. "Overexpression of the STE4 gene leads to mating response in haploid Saccharomyces cerevisiae." Molecular and Cellular Biology 10, no. 1 (1990): 217–22. http://dx.doi.org/10.1128/mcb.10.1.217-222.1990.

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The STE4 gene of Saccharomyces cerevisiae encodes the beta subunit of the yeast pheromone receptor-coupled G protein. Overexpression of the STE4 protein led to cell cycle arrest of haploid cells. This arrest was like the arrest mediated by mating pheromones in that it led to similar morphological changes in the arrested cells. The arrest occurred in haploid cells of either mating type but not in MATa/MAT alpha diploids, and it was suppressed by defects in genes such as STE12 that are needed for pheromone response. Overexpression of the STE4 gene product also suppressed the sterility of cells d
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40

Clark, K. L., and G. F. Sprague. "Yeast pheromone response pathway: characterization of a suppressor that restores mating to receptorless mutants." Molecular and Cellular Biology 9, no. 6 (1989): 2682–94. http://dx.doi.org/10.1128/mcb.9.6.2682.

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Saccharomyces cerevisiae haploid cells, alpha and a, mate after being appropriately stimulated by the pheromone secreted by the opposite cell type (a-factor and alpha-factor, respectively). The binding of a pheromone to its receptor is a signal that initiates a series of intracellular changes that lead to the specific physiological alterations required for mating. To identify components of the signal transduction pathway, we sought pseudorevertants that restored mating competence to receptor mutants (MAT alpha ste3::LEU2). The suppressor srm1-1 was isolated as a recessive mutation that conferr
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41

Clark, K. L., and G. F. Sprague. "Yeast pheromone response pathway: characterization of a suppressor that restores mating to receptorless mutants." Molecular and Cellular Biology 9, no. 6 (1989): 2682–94. http://dx.doi.org/10.1128/mcb.9.6.2682-2694.1989.

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Saccharomyces cerevisiae haploid cells, alpha and a, mate after being appropriately stimulated by the pheromone secreted by the opposite cell type (a-factor and alpha-factor, respectively). The binding of a pheromone to its receptor is a signal that initiates a series of intracellular changes that lead to the specific physiological alterations required for mating. To identify components of the signal transduction pathway, we sought pseudorevertants that restored mating competence to receptor mutants (MAT alpha ste3::LEU2). The suppressor srm1-1 was isolated as a recessive mutation that conferr
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42

Dutton, Gail. "Spray-On Stem Cells Can Step Up Healing." Genetic Engineering & Biotechnology News 37, no. 8 (2017): 6–7. http://dx.doi.org/10.1089/gen.37.08.04.

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43

Stojkovic, Miodrag, and Majlinda Lako. "Neural Stem Cells, a Step Closer to Clinic?" STEM CELLS 29, no. 10 (2011): 1477–78. http://dx.doi.org/10.1002/stem.729.

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44

Pera, Martin F. "Stem cell culture, one step at a time." Nature Methods 2, no. 3 (2005): 164–65. http://dx.doi.org/10.1038/nmeth0305-164.

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Boman, Bruce M., and Max S. Wicha. "Cancer Stem Cells: A Step Toward the Cure." Journal of Clinical Oncology 26, no. 17 (2008): 2795–99. http://dx.doi.org/10.1200/jco.2008.17.7436.

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Nagy, Henriett, Tímea Magyaródi, and Beatrix Séllei. "A képességalapú érzelmi intelligencia: új paradigmák a tesztfejlesztésben és pontozásban. Hazai tapasztalatok az érzelemmegértés és érzelemszabályozás szituációs tesztekkel." Magyar Pszichológiai Szemle 70, no. 4 (2015): 827–46. http://dx.doi.org/10.1556/0016.2015.70.4.7.

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Jelen tanulmány két, a MSCEIT (Mayer–Salovey–Caruso Érzelmi Intelligencia Teszt) alternatívájaként megjelenő érzelmi intelligencia (EI) tesztre: az Érzelemmegértés (STEU) és az Érzelemszabályozás (STEM) szituációs tesztekre fókuszál. A STEU azért újszerű megközelítés az EI-mérés területén, mert bevezeti a sztenderdekre épülő pontozás lehetőségét, a STEM pedig azért, mert az SJT (situational judgement test method) megközelítést alkalmazza. Jelen kutatás célja a STEU- és STEM-tesztek magyar változatainak elkészítése, valamint a tesztek megbízhatóságával és validitásával kapcsolatos bizonyítékok
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Vasilyev, A. V., Yu S. Bakhracheva, Kabore Оusman, and Yu O. Zelenskiy. "Valve Cam Design Using Numerical Step-by-Step Method." Vestnik Volgogradskogo gosudarstvennogo universiteta. Serija 10. Innovatcionnaia deiatel’nost’, no. 1 (March 2014): 26–32. http://dx.doi.org/10.15688/jvolsu10.2014.1.4.

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Muslimawati, Nicha, Ketty Suketi, and Anas D. Susila. "Pertumbuhan Stek Batang Pohpohan (Pilea trinervia Wight.) pada Umur Tanaman, Bagian Batang, dan Media Tanam yang Berbeda." Jurnal Hortikultura Indonesia 6, no. 2 (2015): 91. http://dx.doi.org/10.29244/jhi.6.2.91-98.

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<p>ABSTRACT</p><p>Pohpohan (Pilea trinervia Wight.) is one of indigenous vegetables that grows in the mountain areas of West Java. Propagule availability in large quantities and in a short time can be done with cuttings. However, propagation of Pohpohan by cutting has not been developed presently. The objective of the research was to obtain the best plant age, stem part, and growing media for the growth of pohpohan stem cuttings. The research was conducted at the Center of Tropical Horticulture Studies Experimental Field Tajur-Bogor, from January to July 2013. The cuttings we
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Maleri, Seth, Qingyuan Ge, Elizabeth A. Hackett, Yuqi Wang, Henrik G. Dohlman, and Beverly Errede. "Persistent Activation by Constitutive Ste7 Promotes Kss1-Mediated Invasive Growth but Fails To Support Fus3-Dependent Mating in Yeast." Molecular and Cellular Biology 24, no. 20 (2004): 9221–38. http://dx.doi.org/10.1128/mcb.24.20.9221-9238.2004.

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ABSTRACT Mitogen-activated protein kinase kinase kinase-Ste11 (MAPKKK-Ste11), MAPKK-Ste7, and MAPK-Kss1 mediate pheromone-induced mating differentiation and nutrient-responsive invasive growth in Saccharomyces cerevisiae. The mating pathway also requires the scaffold-Ste5 and the additional MAPK-Fus3. One contribution to specificity in this system is thought to come from stimulus-dependent recruitment of the MAPK cascade to upstream activators that are unique to one or the other pathway. To test this premise, we asked if stimulus-independent signaling by constitutive Ste7 would lead to a loss
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Hagen, D. C., G. McCaffrey, and G. F. Sprague. "Pheromone response elements are necessary and sufficient for basal and pheromone-induced transcription of the FUS1 gene of Saccharomyces cerevisiae." Molecular and Cellular Biology 11, no. 6 (1991): 2952–61. http://dx.doi.org/10.1128/mcb.11.6.2952.

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The FUS1 gene of Saccharomyces cerevisiae is transcribed in a and alpha cells, not in a/alpha diploids, and its transcription increases dramatically when haploid cells are exposed to the appropriate mating pheromone. In addition, FUS1 transcription is absolutely dependent on STE4, STE5, STE7, STE11, and STE12, genes thought to encode components of the pheromone response pathway. We now have determined that the pheromone response element (PRE), which occurs in four copies within the FUS1 upstream region, functions as the FUS1 upstream activation sequence (UAS) and is responsible for all known a
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