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Journal articles on the topic 'Stem tumors'

Author: Grafiati

Published: 28 May 2022

Last updated: 31 July 2025

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1

Lelieveld, Peter, and Jannes H. Mulder. "Three tumor sensitivity tests evaluated with mouse tumors." International Journal of Cell Cloning 5, no. 4 (1987): 335–46. http://dx.doi.org/10.1002/stem.5530050408.

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2

Dunkel, Ira J., and Mark M. Souweidane. "Brain stem tumors." Current Treatment Options in Neurology 7, no. 4 (2005): 315–21. http://dx.doi.org/10.1007/s11940-005-0041-z.

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3

Smith, Richard R. "Brain stem tumors." Seminars in Roentgenology 25, no. 3 (1990): 249–62. http://dx.doi.org/10.1016/0037-198x(90)90054-8.

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4

Dove, Alan. "Stem cells hunt tumors." Nature Biotechnology 18, no. 12 (2000): 1231. http://dx.doi.org/10.1038/82291.

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5

Stephenson, Joan. "Tumors From Stem Cells." JAMA 301, no. 11 (2009): 1118. http://dx.doi.org/10.1001/jama.2009.356.

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6

Hoffman, Harold J. "Dorsally Exophytic Brain Stem Tumors and Midbrain Tumors." Pediatric Neurosurgery 24, no. 5 (1996): 256–62. http://dx.doi.org/10.1159/000121048.

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7

Bertino, J. R., E. Göker, R. Gorlick, W. W. Li, and D. Banerjee. "Resistance Mechanisms to Methotrexate in Tumors." Stem Cells 14, no. 1 (1996): 5–9. http://dx.doi.org/10.1002/stem.140005.

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8

Lichtenauer, Urs D., and Felix Beuschlein. "The tumor stem cell concept—Implications for endocrine tumors?" Molecular and Cellular Endocrinology 300, no. 1-2 (2009): 158–63. http://dx.doi.org/10.1016/j.mce.2008.10.037.

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9

Beltramello, Alberto, Maria Cristina Lombardo, Barbara Masotto, and Albino Bricolog. "Imaging of brain stem tumors." Operative Techniques in Neurosurgery 3, no. 2 (2000): 87–105. http://dx.doi.org/10.1053/oy.2000.6571.

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10

Kaur, Kawaljit, Anna Karolina Kozlowska, Paytsar Topchyan, et al. "Probiotic-Treated Super-Charged NK Cells Efficiently Clear Poorly Differentiated Pancreatic Tumors in Hu-BLT Mice." Cancers 12, no. 1 (2019): 63. http://dx.doi.org/10.3390/cancers12010063.

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Background and Aims: We have previously demonstrated that the stage of differentiation of tumors has profound effect on the function of NK cells, and that stem-like/poorly differentiated tumors were preferentially targeted by the NK cells. Therefore, in this study we determined the role of super-charged NK cells in immune mobilization, lysis, and differentiation of stem-like/undifferentiated tumors implanted in the pancreas of humanized-BLT (hu-BLT) mice fed with or without AJ2 probiotics. The phenotype, growth rate and metastatic potential of pancreatic tumors differentiated by the NK cells (

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11

Bukowski, R. M., D. Mclain, T. Olencki, G. T. Budd, and S. A. Murthy. "Interleukin‐2: Use in solid tumors." Stem Cells 11, no. 1 (1993): 26–32. http://dx.doi.org/10.1002/stem.5530110106.

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12

Mohseny, Alexander B., and Pancras C. W. Hogendoorn. "Concise Review: Mesenchymal Tumors: When Stem Cells Go Mad." STEM CELLS 29, no. 3 (2011): 397–403. http://dx.doi.org/10.1002/stem.596.

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13

Werbowetski-Ogilvie, Tamra E., Ludivine Coudière Morrison, Aline Fiebig-Comyn, and Mickie Bhatia. "In Vivo Generation of Neural Tumors from Neoplastic Pluripotent Stem Cells Models Early Human Pediatric Brain Tumor Formation." STEM CELLS 30, no. 3 (2012): 392–404. http://dx.doi.org/10.1002/stem.1017.

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14

Liu, Hai-Long, Ya-Nan Wang, and Shi-Yu Feng. "Brain tumors: Cancer stem-like cells interact with tumor microenvironment." World Journal of Stem Cells 12, no. 12 (2020): 1439–54. http://dx.doi.org/10.4252/wjsc.v12.i12.1439.

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15

Kizilova, E. A. "Optimization of teratoma formation assay." Genes & Cells 11, no. 2 (2016): 119–28. http://dx.doi.org/10.23868/gc120606.

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Teratoma formation assay is necessary to estimate in vivo pluripotency of stem cells especially stem cell lines of human origin. Nevertheless convenient, valid and universal “standards” to analyze stem cell derived tumors have not been developed yet. New protocol for monitoring teratoma growth, morphological and histological analyzes of tumor samples is proposed in this paper. This protocol is oriented on review of tumors morphology and histology per se. The list-describer includes 17 obligate and 12 facultative diagnostic sell types and 7 diagnostic cell complexes. The protocol takes into acc

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16

Valet, Matthieu, and Patrick Narbonne. "Formation of benign tumors by stem cell deregulation." PLOS Genetics 18, no. 10 (2022): e1010434. http://dx.doi.org/10.1371/journal.pgen.1010434.

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Within living organisms, stem cells respond to various cues, including to niche signals and growth factors. Niche signals originate from the stem cell’s microenvironment and promote the undifferentiated state by preventing differentiation, allowing for stem cell self-renewal. On the other hand, growth factors promote stem cell growth and proliferation, while their sources comprise of a systemic input reflecting the animal’s nutritional and metabolic status, and a localized, homeostatic feedback signal from the tissue that the stem cells serve. That homeostatic signal prevents unnecessary stem

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17

Dornhoffer, John L., Jan Helms, and Dirk H. Hoehmann. "Presentation and Diagnosis of Small Acoustic Tumors." Otolaryngology–Head and Neck Surgery 111, no. 3P1 (1994): 232–35. http://dx.doi.org/10.1177/01945998941113p111.

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With the recent advent of magnetic resonance imaging and auditory brain stem response, it is now possible to diagnose acoustic tumors while they are still quite small. As a result, it is becoming obvious that the clinical presentation of these smaller lesions can be somewhat variant to what is considered typical for an acoustic neuroma. Likewise, although the sensitivity of auditory brain stem response for larger tumors is believed to be quite good, the sensitivity for smaller tumors has recently been questioned, particularly when the patient is first seen early in the course of the disease wi

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18

Baker, Fraser, and Laura Sanger. "The deensity of clonogenic cless in human solid tumors." International Journal of Cell Cloning 9, no. 2 (1991): 155–65. http://dx.doi.org/10.1002/stem.5530090206.

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19

Nandeesh, B. N., Sharmistha Naskar, Arun H. Shashtri, A. Arivazhagan, and Vani Santosh. "Recurrent Glioblastomas Exhibit Higher Expression of Biomarkers with Stem-like Properties." Journal of Neurosciences in Rural Practice 09, no. 01 (2018): 086–91. http://dx.doi.org/10.4103/jnrp.jnrp_417_17.

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ABSTRACT Background: Despite advances in the treatment of glioblastoma (GBM), the prognosis of patients continues to remain dismal. This unfavorable prognosis is mainly attributed to the tumor's propensity for progression and recurrence, which in turn is due to the highly aggressive nature of the persisting GBM cells that actively egress from the main tumor mass into the surrounding normal brain tissue. Such a recurrent tumor described to have a more malignant potential is highly invasive and resistant to current therapies, probably due to increased stemness and preferential selection of thera

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20

Hazar, Volkan. "STEM CELL TRANSPLANTATION IN BRAIN TUMORS." Hematology, Transfusion and Cell Therapy 44 (October 2022): S11. http://dx.doi.org/10.1016/j.htct.2022.09.1205.

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21

Ortiz-Sánchez, Elizabeth. "Cancer Stem Cells in Solid Tumors." Current Stem Cell Research & Therapy 14, no. 5 (2019): 374. http://dx.doi.org/10.2174/1574888x1405190520103520.

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22

Aleynik, Alexander, and Pranela Rameshwar. "Stem Cell Therapy for Brain Tumors." International Journal of Translational Science 2015, no. 1 (2015): 67–106. http://dx.doi.org/10.13052/ijts2246-8765.20151005.

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23

Pérez-Castillo, Ana, Diana Aguilar-Morante, José A. Morales-García, and Jorge Dorado. "Cancer stem cells and brain tumors." Clinical and Translational Oncology 10, no. 5 (2008): 262–67. http://dx.doi.org/10.1007/s12094-008-0195-8.

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24

Hermann, Patrick C., Sonu Bhaskar, Michele Cioffi, and Christopher Heeschen. "Cancer stem cells in solid tumors." Seminars in Cancer Biology 20, no. 2 (2010): 77–84. http://dx.doi.org/10.1016/j.semcancer.2010.03.004.

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25

Jan, Michel, Gilles Guy, Yvon Guégan, Jean Marie Brucher, and Dominique Fournier. "Neuroepithelial Tumors of the Brain Stem." Neurosurgery Quarterly 4, no. 4 (1994): 220–47. http://dx.doi.org/10.1097/00013414-199412000-00002.

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26

Er, Ozlem. "Cancer Stem Cells in Solid Tumors." Onkologie 32, no. 10 (2009): 605–9. http://dx.doi.org/10.1159/000232375.

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27

Lasky, Joseph L., and Linda M. Liau. "Targeting Stem Cells in Brain Tumors." Technology in Cancer Research & Treatment 5, no. 3 (2006): 251–60. http://dx.doi.org/10.1177/153303460600500312.

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28

Ailles, Laurie E., and Irving L. Weissman. "Cancer stem cells in solid tumors." Current Opinion in Biotechnology 18, no. 5 (2007): 460–66. http://dx.doi.org/10.1016/j.copbio.2007.10.007.

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29

Witt, H., A. Korshunov, M. Remke, et al. "DNA methylation pattern of brain stem pilocytic astrocytomas in children." Journal of Clinical Oncology 27, no. 15_suppl (2009): 10021. http://dx.doi.org/10.1200/jco.2009.27.15_suppl.10021.

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10021 Background: Pilocytic astrocytoma (WHO grade I) comprises the most frequent brain tumor in childhood. We were recently able to identify BRAF as a centrally important oncogene in these tumors showing duplication or activation in a majority of cases. Although histologically indistinguishable, tumors with brain stem location have a particularly poor prognosis. It is not well established, whether this is due to their close proximity to pivotal anatomic sites or due to distinct biological characteristics. Methods: To identify novel genes involved in astrocytoma pathogenesis, we performed a ge

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30

Bussolati, Benedetta, Alessia Brossa, and Giovanni Camussi. "Resident Stem Cells and Renal Carcinoma." International Journal of Nephrology 2011 (2011): 1–6. http://dx.doi.org/10.4061/2011/286985.

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According to the cancer stem cell hypothesis tumors are maintained by a cancer stem cell population which is able to initiate and maintain tumors. Tumor-initiating stem cells display stem or progenitor cell properties such as self-renewal and capacity to re-establish tumors that recapitulate the tumor of origin. In this paper, we discuss data relative to the presence of cancer stem cells in human renal carcinoma and their possible origin from normal resident stem cells. The cancer stem cells identified in human renal carcinomas are not derived from the normal CD133+progenitors of the kidney, b

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31

Stuckey, Daniel W., Shawn D. Hingtgen, Nihal Karakas, Benjamin E. Rich, and Khalid Shah. "Engineering Toxin-Resistant Therapeutic Stem Cells to Treat Brain Tumors." STEM CELLS 33, no. 2 (2015): 589–600. http://dx.doi.org/10.1002/stem.1874.

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32

Kavari, Sanam L., and Khalid Shah. "Engineered stem cells targeting multiple cell surface receptors in tumors." STEM CELLS 38, no. 1 (2019): 34–44. http://dx.doi.org/10.1002/stem.3069.

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33

Bapat, Sharmila A. "Human ovarian cancer stem cells." REPRODUCTION 140, no. 1 (2010): 33–41. http://dx.doi.org/10.1530/rep-09-0389.

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The isolation and identification of stem-like cells in solid tumors or cancer stem cells (CSCs) have been exciting developments of the last decade, although these rare populations had been earlier identified in leukemia. CSC biology necessitates a detailed delineation of normal stem cell functioning and maintenance of homeostasis within the organ. Ovarian CSC biology has unfortunately not benefited from a pre-established knowledge of stem cell lineage demarcation and functioning in the normal organ. In the absence of such information, some of the classical parameters such as long-term culture-

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34

Czerwinska, Patrycja, and Andrzej Adam Mackiewicz. "Mining Transcriptomic Data to Uncover the Association between CBX Family Members and Cancer Stemness." International Journal of Molecular Sciences 23, no. 21 (2022): 13083. http://dx.doi.org/10.3390/ijms232113083.

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Genetic and epigenetic changes might facilitate the acquisition of stem cell-like phenotypes of tumors, resulting in worse patients outcome. Although the role of chromobox (CBX) domain proteins, a family of epigenetic factors that recognize specific histone marks, in the pathogenesis of several tumor types is well documented, little is known about their association with cancer stemness. Here, we have characterized the relationship between the CBX family members’ expression and cancer stemness in liver, lung, pancreatic, and uterine tumors using publicly available TCGA and GEO databases and har

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35

Mačingová, Zuzana, and Stanislav Filip. "Cancer Stem Cells – New Approach to Cancerogenensis and Treatment." Acta Medica (Hradec Kralove, Czech Republic) 51, no. 3 (2008): 139–44. http://dx.doi.org/10.14712/18059694.2017.15.

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Recently, there is an increasing evidence supporting the theory of cancer stem cells not only in leukemia but also in solid cancer. To date, the existence of cancer stem cells has been proven in acute and chronic myeloid leukemia, in breast cancer, in brain tumors, in lung cancer and gastrointestinal tumors. This review is focusing on the recent discovery of stem cells in leukemia, human brain tumors and breast cancer. A small population of cells in the tumor (less than 1 %) shows the potential to give rise to the tumor and its growth. These cells have a substantial characteristic of stem cell

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36

Wallace, Lisa, Anthony Gromovsky, James Hale, et al. "STEM-26. ALTERED LIPID METABOLISM MARKS GLIOBLASTOMA STEM AND NON-STEM CELLS IN SEPARATE TUMOR NICHES." Neuro-Oncology 21, Supplement_6 (2019): vi239. http://dx.doi.org/10.1093/neuonc/noz175.999.

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Abstract Clinical glioblastoma is marked by a strikingly heterogeneous mix of cell types, cellular metabolisms, and cellular microenvironments spread in different spatial locations throughout a tumor. We have created 3-dimensional organoid models that partially mimic the transition zone between nutrient-rich cellular tumor regions and nutrient-poor psuedopallisading and perinecrotic tumor zones. We found a dramatic disparity in lipid droplet presence between these regions with high lipid accumulation in the hypoxic organoid cores of a wide spectrum of patient derived specimens. This is accompa

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37

Bryukhovetskiy, I. S., AБ S. Bryukhovetskiy, and Y. S. Khotimchenko. "Pharmacogenetic and bioengineering approaches to the treatment of glial tumors of the brain." Genes & Cells 9, no. 3 (2014): 140–46. http://dx.doi.org/10.23868/gc120356.

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The article analyzes the existing ideas about the specific mechanisms of therapeutic resistance of glial tumors of the brain, systematized the main trends in modern chemotherapy glial tumors, an attempt to justify the new bioengineering approaches to the creation of personalized cell preparations for therapy of glial tumors based on molecular - biological characteristics of tumor stem cells. It is shown that the main tool of the therapeutic effects can be own stem cells of cancer patients and tumor stem cell proteome can be considered as the primary target cell therapy.

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38

Arcuri, Leonardo Javier, Andreza Alice Feitosa Ribeiro, and Décio Lerner. "HSCT FOR SOLID TUMORS." JBMTCT 2, no. 1 (2021): 131–37. http://dx.doi.org/10.46765/2675-374x.2021v4n1p131-137.

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39

Stroink, Ann R., Harold J. Hoffman, E. Bruce Hendrick, and Robin P. Humphreys. "Diagnosis and management of pediatric brain-stem gliomas." Journal of Neurosurgery 65, no. 6 (1986): 745–50. http://dx.doi.org/10.3171/jns.1986.65.6.0745.

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✓ The authors reviewed the cases of 49 children, ranging in age from 9 months to 15 years, who were diagnosed by computerized tomography (CT) as having brain-stem glioma. Four distinct groups of brain-stem gliomas were identified based on CT scan characteristics: Group I included isodense contrast-enhancing tumors that were dorsally exophytic into the fourth ventricle; Group II(a) included hypodense nonenhancing intrinsic tumors of the brain stem; Group II(b) included intrinsic tumors of the brain stem with hyperdense exophytic components extending ventrally and laterally into the cerebellopon

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40

Dr., Arpita Mukherjee. "Cancer Stem Cells (CSCs) and its various biomarkers for treatment of human cancers: Review article." World Journal of Advanced Research and Reviews 21, no. 3 (2024): 2582–90. https://doi.org/10.5281/zenodo.14182089.

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Traditional cancer treatments often fail to target specific cells, leading to therapy resistance and recurrence. Stem cell biology offers new potential for cancer treatment, including self-renewal, migration, differentiation, and modulatory effects. This review discusses stem cell use in cancer treatment, clinical applications, risks, and future directions for improving cancer outcomes. The kind of treatment chosen is determined by the kind, stage, and goal of the cancer. The first line of treatment for the direct removal of solid tumors that are localized is surgery. Radiotherapy damages the

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41

Donangelo, Ines, Song-Guang Ren, Tamar Eigler, Clive Svendsen, and Shlomo Melmed. "Sca1+ murine pituitary adenoma cells show tumor-growth advantage." Endocrine-Related Cancer 21, no. 2 (2014): 203–16. http://dx.doi.org/10.1530/erc-13-0229.

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The role of tumor stem cells in benign tumors such as pituitary adenomas remains unclear. In this study, we investigated whether the cells within pituitary adenomas that spontaneously develop in Rb+/− mice are hierarchically distributed with a subset being responsible for tumor growth. Cells derived directly from such tumors grew as spheres in serum-free culture medium supplemented with epidermal growth factor and basic fibroblast growth factor. Some cells within growing pituitary tumor spheres (PTS) expressed common stem cell markers (Sca1, Sox2, Nestin, and CD133), but were devoid of hormone

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42

Afify, Chen, Yan, et al. "Method to Convert Stem Cells into Cancer Stem Cells." Methods and Protocols 2, no. 3 (2019): 71. http://dx.doi.org/10.3390/mps2030071.

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The cancer stem cell (CSC) hypothesis suggests that tumors are sustained exclusively by a small population of the cells with stem cell properties. CSCs have been identified in most tumors and are responsible for the initiation, recurrence, and resistance of different cancers. In vitro CSC models will be of great help in revisiting the mechanism of cancer development, as well as the tumor microenvironment and the heterogeneity of cancer and metastasis. Our group recently described the generation of CSCs from induced pluripotent stem cells (iPSCs), which were reprogrammed from normal cells, and/

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43

Jandial, Rahul, Hoisang U, Michael L. Levy, and Evan Y. Snyder. "Brain tumor stem cells and the tumor microenvironment." Neurosurgical Focus 24, no. 3-4 (2008): E27. http://dx.doi.org/10.3171/foc/2008/24/3-4/e26.

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✓ Recent advances in stem cell research and developmental neurobiology have uncovered new perspectives from which to investigate various forms of cancer. Specifically, the hypothesis that tumors consist of a subpopulation of malignant cells similar to stem cells is of great interest to scientists and clinicians and has been dubbed the “cancer stem cell hypothesis.” The region in which this assertion is most relevant is within the brain. Cancer stem cells have been isolated from brain tumors that exhibit characteristics of differentiation and proliferation normally seen only in neural stem cell

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44

Huang, Binjie, Xin Yan, and Yumin Li. "Cancer Stem Cell for Tumor Therapy." Cancers 13, no. 19 (2021): 4814. http://dx.doi.org/10.3390/cancers13194814.

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Tumors pose a significant threat to human health. Although many methods, such as operations, chemotherapy and radiotherapy, have been proposed to eliminate tumor cells, the results are unsatisfactory. Targeting therapy has shown potential due to its specificity and efficiency. Meanwhile, it has been revealed that cancer stem cells (CSCs) play a crucial role in the genesis, development, metastasis and recurrence of tumors. Thus, it is feasible to inhibit tumors and improve prognosis via targeting CSCs. In this review, we provide a comprehensive understanding of the biological characteristics of

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45

Balaji, Sekaran, Radhakrishnan Santhi, Usha Kim, Veerappan Muthukkaruppan, Chidambaranathan G. Priya, and Ayyasamy Vanniarajan. "Cancer Stem Cells with Overexpression of Neuronal Markers Enhance Chemoresistance and Invasion in Retinoblastoma." Current Cancer Drug Targets 20, no. 9 (2020): 710–19. http://dx.doi.org/10.2174/1568009620666200504112711.

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Background: Retinoblastoma is a sight and life-threatening embryonal tumor in children. Though chemotherapy is the main mode of therapy, evolving resistance remains a major obstacle in treatment success. The presence of cancer stem cells (CSC) is frequently reported to be responsible for chemoresistance in multiple tumors. Objective: Our study aims to identify the molecular factors that facilitate the chemoresistance through cancer stem cells in retinoblastoma. Methods: We developed etoposide and carboplatin resistant retinoblastoma (Y79) cell lines by stepwise drug increment treatment, valida

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46

Friedman, Gregory K., and G. Yancey Gillespie. "Cancer Stem Cells and Pediatric Solid Tumors." Cancers 3, no. 1 (2011): 298–318. http://dx.doi.org/10.3390/cancers3010298.

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47

Wang, Xi. "Cancer Stem Cell: The Seed of Tumors?" American Chinese Journal of Medicine and Science 2, no. 1 (2009): 1. http://dx.doi.org/10.7156/v2i1p001.

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48

Teslenko, D., O. Palamar, A. Huk, R. Aksyonov, and D. Okonskyi. "Intraventricular and brain stem tumors. Endoscopic possibilities." Brain and Spine 1 (2021): 100505. http://dx.doi.org/10.1016/j.bas.2021.100505.

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49

Lasky III, Joseph, Meeryo Choe, and Ichiro Nakano. "Cancer Stem Cells in Pediatric Brain Tumors." Current Stem Cell Research & Therapy 4, no. 4 (2009): 298–305. http://dx.doi.org/10.2174/157488809789649278.

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50

Pierfelice, T. J., K. C. Schreck, C. G. Eberhart, and N. Gaiano. "Notch, Neural Stem Cells, and Brain Tumors." Cold Spring Harbor Symposia on Quantitative Biology 73 (January 1, 2008): 367–75. http://dx.doi.org/10.1101/sqb.2008.73.013.

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