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Journal articles on the topic 'Steroid-sensitive nephrotic syndrome'

Author: Grafiati

Published: 2 June 2025

Last updated: 31 July 2025

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1

Nanjundaswamy, H. M., and K. D. Phadke. "Steroid sensitive nephrotic syndrome." Indian Journal of Pediatrics 69, no. 12 (2002): 1059–63. http://dx.doi.org/10.1007/bf02724388.

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2

K., Ramya, Balasubramanian M., and Sharon S. "Urinary Interleukin 18 in Children with Nephrotic Syndrome and Its Role in Steroid Responsiveness." International Journal of Pharmaceutical and Clinical Research 16, no. 3 (2024): 909–14. https://doi.org/10.5281/zenodo.10968918.

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<strong>Introduction: </strong>Nephrotic syndrome includes the clinical manifestation of glomerular diseases associated with heavy proteinuria i.e., nephrotic range (40 mg/m²/hr or >1000 mg/m²/day; spot Up/Uc>2 mg/mg; 3-4+ by urine dipstick); hypoalbuminemia (albumin <3.0 g/dL); and edema. In children, the most common cause of nephrotic syndrome is idiopathic or primary nephrotic syndrome (INS), also called nephrosis. In the kidney, the predominant source of IL-18 is the tubular epithelial cells. In the recent years, the biological and pathological roles of IL-18 have b

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3

Burlaka, Ie A., and I. V. Bagdasarova. "Molecular factors predicting steroid resistance in pediatric nephrotic syndrome." Ukrainian Journal of Nephrology and Dialysis, no. 2(70) (March 21, 2021): 32–37. http://dx.doi.org/10.31450/ukrjnd.2(70).2021.04.

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Objectives: the objective of this paper was to study the levels of cellular hypoxia, apoptosis controlling factors in children with steroid-sensitive and steroid-resistant nephrotic syndrome. Background: patients with steroid-resistant nephrotic syndrome (SRNS) represent a challenging subset of patients with nephrotic syndrome who often fail standard immunosuppression and have a higher likelihood of progressing to end-stage renal disease. The search of the biochemical markers undergoing the steroid-resistance is under urgent need. Methods: an examination of kidney biopsies and bloo

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4

Quader, Mohammed Maruf Ul. "Familial nephrotic syndrome: Does it matter?" Paediatric Nephrology Journal of Bangladesh 8, no. 2 (2023): 69–77. http://dx.doi.org/10.4103/pnjb.pnjb_15_23.

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Abstract Steroid sensitive nephrotic syndrome (SSNS) is the most common form of nephrotic syndrome in childhood. Familial SSNS seems to be clinically homogeneous but reports on genetically informative SSNS families are lacking. Familial SSNS was found to be genetically distinct from familial steroid resistant nephrotic syndrome (SRNS). Several genes have been identified for SRNS. In general, genetic forms of nephrotic syndrome are resistant to common therapeutic approaches. But different cohort of steroid sensitive familial nephrotic syndrome were published. So precise clinical and genetic inf

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5

Palaniyandi, Anitha, and Subramani Palaniyandi. "Comparison of clinical and lab profile between steroid sensitive and steroid resistant nephrotic syndrome at onset of disease and evaluating predictors for developing steroid resistance in nephrotic syndrome." International Journal of Contemporary Pediatrics 7, no. 6 (2020): 1304. http://dx.doi.org/10.18203/2349-3291.ijcp20202138.

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Background: Nephrotic syndrome is a notable chronic disease in children. The objective of this study was to compare the clinical and lab profile between steroid sensitive nephrotic syndrome and steroid resistant nephrotic syndrome at the onset of disease. Certain parameters were tested if they could be significate predictors of developing steroid resistance at the onset of first episode of nephrotic syndrome.Methods: Retrospective observation study done children 1-12 years diagnosed with nephrotic syndrome in Sri Ramachandra Medical College and Hospital, Department of Paediatrics, Chennai. Sam

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Christabel, Eunike Vania. "Steroid-induced diabetic ketoacidosis in a 9-year-old boy with relapsing steroid-sensitive nephrotic syndrome." Paediatria Croatica 65, no. 3 (2021): 147–49. http://dx.doi.org/10.13112/pc.384.

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Systemically administered corticosteroids remain the backbone therapy for nephrotic syndrome up to now. Although 80%-90% of children show remission following steroid treatment and are classifi ed as steroid-sensitive nephrotic syndrome, 90% of initial responders will experience relapse at least once, and about 50%-60% may progress to frequent relapsing nephrotic syndrome and hence become steroid dependent. Hyperglycaemia is one of the best-known side eff ects, although most cases are mild and clinically asymptomatic. Presentation with severe hyperglycaemia is possible but rather uncommon. This

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7

Mbanefo, NR, FN Ogbuka, CA Nwaoha, CL Odimegwu, SN Uwaezuoke, and HU Okafor. "The Pattern of Steroid Sensitivity and Steroid Resistance in Childhood Idiopathic Nephrotic Syndrome: A 5-Year Retrospective Observational Descriptive Study in a South-East Nigerian Tertiary Hospital." Nigerian Journal of Clinical Practice 26, no. 7 (2023): 1017–22. http://dx.doi.org/10.4103/njcp.njcp_1_23.

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ABSTRACT Background/Aim: Nephrotic syndrome is the most common glomerular disease of childhood. Majority of the idiopathic cases frequently respond to steroid therapy and are regarded as steroid-sensitive nephrotic syndrome. Several studies have reported a change in this usual pattern to steroid-resistant nephrotic syndrome in Nigerian children. This study aimed to determine the pattern of steroid sensitivity and steroid resistance in childhood idiopathic nephrotic syndrome seen at a tertiary hospital in Enugu, south-east Nigeria. Materials and Methods: A retrospective study conducted in child

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8

Lestari, Nina, Neti Nurani, and Madarina Julia. "Corticosteroids and obesity in steroid-sensitive and steroid-resistant nephrotic syndrome." Paediatrica Indonesiana 55, no. 4 (2015): 194. http://dx.doi.org/10.14238/pi55.4.2015.194-8.

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Background Children with nephrotic syndrome need high-dose corticosteroids to achieve remission. Studies have estimated a 35-43% risk of obesity in these patients after corticosteroid treatment.Objective To determine the prevalence of obesity in children who received corticosteroids for nephrotic syndrome, and to compare the risk of obesity in children with steroid-sensitive nephrotic syndrome (SSNS) and steroid-resistant nephrotic syndrome (SRNS).Methods We performed a retrospective cohort study in 50 children with SSNS or SRNS who received corticosteroid treatment. Obesity was defined to be

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9

Pardede, Sudung Oloan, Andini Striratnaputri, and Muzal Kadim. "Selenium level in steroid-resistant and steroid-sensitive nephrotic syndrome." Paediatrica Indonesiana 60, no. 6 (2020): 316–20. http://dx.doi.org/10.14238/pi60.6.2020.316-20.

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Background The mechanisms of pathogenesis of steroid-resistant nephrotic syndrome (SRNS) and steroid-sensitive nephrotic syndrome (SSNS) are not well understood. Antioxidants, such as glutathione peroxidase enzyme (GPx) and its cofactor, selenium, are thought to slow the progress of nephrotic syndrome (NS). Objective To compare selenium levels in SRNS and SSNS pediatric patients. Methods This cross-sectional study was conducted in 51 SRNS and 30 SSNS patients, aged 2 to 18 years, who visited the Pediatric Nephrology Outpatient Clinic at Cipto Mangunkusumo Hospital, Jakarta. Subject

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10

Sinha, Aditi, Arvind Bagga, Sushmita Banerjee, et al. "Steroid Sensitive Nephrotic Syndrome: Revised Guidelines." Indian Pediatrics 58, no. 5 (2021): 461–81. http://dx.doi.org/10.1007/s13312-021-2217-3.

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11

Abeyagunawardena, AS, RS Thalgahagoda, AH H. M. Jayaweera, and UI Karunadasa. "Therapies for steroid-sensitive nephrotic syndrome." Asian Journal of Pediatric Nephrology 1, no. 2 (2018): 56. http://dx.doi.org/10.4103/ajpn.ajpn_35_18.

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Bagga, Arvind, Aditi Sinha, Sushmita Banerjee, et al. "Steroid sensitive nephrotic syndrome: Revised guidelines." Asian Journal of Pediatric Nephrology 4, no. 2 (2021): 48. http://dx.doi.org/10.4103/ajpn.ajpn_34_21.

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13

Srivastava, R. N., and V. K. Agarwal. "Current Management: Steroid Sensitive Nephrotic Syndrome." Apollo Medicine 2, no. 4 (2005): 342–48. http://dx.doi.org/10.1016/s0976-0016(11)60518-8.

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Abeyagunawardena, A. S. "Treatment of steroid sensitive nephrotic syndrome." Indian Journal of Pediatrics 72, no. 9 (2005): 763–69. http://dx.doi.org/10.1007/bf02734149.

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Srivastava, R. N., and V. K. Agarwal. "Current Management: Steroid Sensitive Nephrotic Syndrome." Apollo Medicine 2, no. 4 (2005): 342–48. http://dx.doi.org/10.1177/0976001620050415.

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16

Akbar, Muhammad, Oke Rina Ramayani, and Gema Nazri Yanni. "Blood Pressure Differences in Nephrotic Syndrome Patients with Steroid Resistant Nephrotic Syndrome Patients and Steroid Sensitive Nephrotic Syndrome." Open Access Macedonian Journal of Medical Sciences 9, T3 (2021): 349–52. http://dx.doi.org/10.3889/oamjms.2021.6315.

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BACKGROUND: Syndrome nephrotic is the most common kidney disease found in pediatric kidney disease, classification based on clinical response to steroids or histopathological characteristics. Increased blood pressure in steroid-resistant nephrotic syndrome (NS) is still a complication to be aware of in cases of NS. AIM: The aim of the study was to determine the differences in systolic and diastolic blood pressure in patients with steroid-sensitive NS and steroid-resistant NS. METHODS: Analytical correctional study in 50 children with NS divided into 25 Steroid Resistant NS (SRNS) groups and 25

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17

Akbar, Muhammad, Oke Rina Ramayani, and Gema Nazri Yanni. "Blood Pressure Differences in Nephrotic Syndrome Patients with Steroid Resistant Nephrotic Syndrome Patients and Steroid Sensitive Nephrotic Syndrome." Open Access Macedonian Journal of Medical Sciences 9, T3 (2021): 349–52. http://dx.doi.org/10.3889/oamjms.2021.6315.

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BACKGROUND: Syndrome nephrotic is the most common kidney disease found in pediatric kidney disease, classification based on clinical response to steroids or histopathological characteristics. Increased blood pressure in steroid-resistant nephrotic syndrome (NS) is still a complication to be aware of in cases of NS. AIM: The aim of the study was to determine the differences in systolic and diastolic blood pressure in patients with steroid-sensitive NS and steroid-resistant NS. METHODS: Analytical correctional study in 50 children with NS divided into 25 Steroid Resistant NS (SRNS) groups and 25

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18

Mühlig, Anne K., Jun Young Lee, Markus J. Kemper, et al. "Levamisole in Children with Idiopathic Nephrotic Syndrome: Clinical Efficacy and Pathophysiological Aspects." Journal of Clinical Medicine 8, no. 6 (2019): 860. http://dx.doi.org/10.3390/jcm8060860.

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Steroid sensitive nephrotic syndrome is one of the most common pediatric glomerular diseases. Unfortunately, it follows a relapsing and remitting course in the majority of cases, with 50% of all cases relapsing once or even more often. Most children with idiopathic nephrotic syndrome respond initially to steroid therapy, nevertheless repeated courses for patients with relapses induce significant steroid toxicity. Patients with frequent relapses or steroid dependency thus require alternative treatment, such as cyclophosphamide, cyclosporine, tacrolimus, mycophenolate mofetil, levamisole, or rit

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19

Nickavar, Azar, Sahar Sadr Moharerpour, and Ehsan Abiry. "Predictive value of platelet indices in children with idiopathic nephrotic syndrome." Immunopathologia Persa 6, no. 1 (2019): e04-e04. http://dx.doi.org/10.15171/ipp.2020.04.

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Introduction: Different biomarkers have been investigated for prognosis of patients with nephrotic syndrome. Qualitative and quantitative changes have been reported in platelets in these patients. Objectives: The aim of this study was to identify platelet abnormalities and their prognostic value of steroid response in children with idiopathic nephrotic syndrome. Patients and Methods: Platelet counts and indices (mean platelet volume [MPV], platelet distribution width [PDW] and platelet larger cell ratio [PLCR]) were evaluated and compared in 122 children with active nephrotic syndrome (64%; st

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20

Warejko, Jillian K., Weizhen Tan, Ankana Daga, et al. "Whole Exome Sequencing of Patients with Steroid-Resistant Nephrotic Syndrome." Clinical Journal of the American Society of Nephrology 13, no. 1 (2017): 53–62. http://dx.doi.org/10.2215/cjn.04120417.

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Background and objectivesSteroid-resistant nephrotic syndrome overwhelmingly progresses to ESRD. More than 30 monogenic genes have been identified to cause steroid-resistant nephrotic syndrome. We previously detected causative mutations using targeted panel sequencing in 30% of patients with steroid-resistant nephrotic syndrome. Panel sequencing has a number of limitations when compared with whole exome sequencing. We employed whole exome sequencing to detect monogenic causes of steroid-resistant nephrotic syndrome in an international cohort of 300 families.Design, setting, participants, &

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Abeyagunawardena, Asiri Abeyagunawardena S. "Childhood nephrotic syndrome: steroid sensitive, steroid resistant and beyond." Sri Lanka Journal of Child Health 38, no. 4 (2009): 115. http://dx.doi.org/10.4038/sljch.v38i4.1389.

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22

Zulfiqar, Zunaira, Fatema Lanewala, Sabeeta Khatri, Irshad Bajeer, Madiha Aziz, and Seema Hashmi. "Steroid induced ocular complications in idiopathic nephrotic syndrome: a cross sectional single center study." Journal of the Pakistan Medical Association 74, no. 2 (2024): 315–19. http://dx.doi.org/10.47391/jpma.10573.

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Objective: To screen children receiving steroids to identify ocular complications and their prevalence. Method: The cross-sectional study was conducted at the Paediatric Nephrology and Ophthalmology departments of the Sindh Institute of Urology and Transplantation, Karachi, from May to October 2022, and comprised patients who received at least 1500mg cumulative steroid dose for a minimum of 3 months. They were screened for steroid-sensitive or steroid-resistant nephrotic syndrome. Ocular examinations, including visual acuity, intraocular pressure, slit-lamp biomicroscopy, lens examination and

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23

B S, Sreelekshmy, Ranjana S R, and Shaiju S Dharan. "Patients with Steroid Sensitive Nephrotic Syndrome: A Case Series." International Journal of Research and Review 11, no. 5 (2024): 15–156. http://dx.doi.org/10.52403/ijrr.20240518.

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Nephrotic syndrome (NS) is one of the most common kidney diseases found in childhood. The prevalence of nephrotic syndrome worldwide is approximately 16 cases per 100,000 children with an incidence of two to seven per 100,000 children. Nephrotic syndrome may affect adults and children of both genders and any race. Nephrotic syndrome in children can be classified into three groups; secondary, congenital, infantile, and idiopathic. In first case, a 3-year-old male patient was presented with complaints of fever, cough for 6 days and swelling all over the body, decreased urine output, oral intake

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Ciuntu, Angela. "The role of cell signaling molecules in the pathogenesis of glomerulonephritis in children." Moldovan Medical Journal 64, no. 2 (2021): 37–41. http://dx.doi.org/10.52418/moldovan-med-j.64-2.21.07.

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Background: Cytokines are functional class of tiny proteins and glycoprotein and fundamentally they are monomers that function as soluble mediators in an autocrine or paracrine manner. Cytokines are produced by a number of cell types, predominantly leukocytes, and their targets implicate both immune and non-immune cells. Material and methods: This study was performed on 75 children with glomerulonephritis (GN), aged from 2 up to 17 years. There were 20 children with steroid-sensitive nephrotic syndrome (SSNS), 15 children with steroid-resistant nephrotic syndrome (SRNS), 20 children with chron

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Sinha, Aditi, Pankaj Hari, Piyush Kumar Sharma, et al. "Disease course in steroid sensitive nephrotic syndrome." Indian Pediatrics 49, no. 11 (2012): 881–87. http://dx.doi.org/10.1007/s13312-012-0220-4.

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Karp, Alana M., and Rasheed A. Gbadegesin. "Genetics of childhood steroid-sensitive nephrotic syndrome." Pediatric Nephrology 32, no. 9 (2016): 1481–88. http://dx.doi.org/10.1007/s00467-016-3456-8.

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Mahamithawa, U. D. "The treatment of steroid sensitive nephrotic syndrome." Sri Lanka Journal of Child Health 31, no. 1 (2009): 18. http://dx.doi.org/10.4038/sljch.v31i1.800.

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Mahamithawa, U. D. "The treatment of steroid sensitive nephrotic syndrome." Sri Lanka Journal of Child Health 31, no. 2 (2009): 60. http://dx.doi.org/10.4038/sljch.v31i2.782.

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Deshpande, Pankaj V., and Mark Griffiths. "Pulmonary thrombosis in steroid-sensitive nephrotic syndrome." Pediatric Nephrology 20, no. 5 (2005): 665–69. http://dx.doi.org/10.1007/s00467-004-1741-4.

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Motoyama, Osamu, Hidenori Sugawara, Michihiro Hatano, Tomoo Fujisawa, and Kikuo Iitaka. "Steroid-sensitive nephrotic syndrome in two families." Clinical and Experimental Nephrology 13, no. 2 (2009): 170–73. http://dx.doi.org/10.1007/s10157-008-0117-7.

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Hoque, SM Shamsul, Md Ashraful Islam, Taslima Akter, Ranjit Ranjan Roy, and Md Habibur Rahman. "Comparison of Lipid Profile in Different Types of Steroid Sensitive Idiopathic Relapsing Nephrotic Syndrome in Children during Active Disease and Remission." Medicine Today 34, no. 2 (2022): 111–16. http://dx.doi.org/10.3329/medtoday.v34i2.61424.

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Introduction: Nephrotic syndrome is a disease of relapse and remission. Relapse rate is more than 80%. Hyperlipidemia and hypoalbuminemia are important characteristic of nephrotic syndrome. Hyperlipidemia persist even after remission of disease in frequent relapse nephrotic syndrome possibly due to frequent attack of disease and frequent use of steroid. Hyperlipidemia causes premature atherosclerosis, progressive renal injury leading to chronic renal failure, cardiac complications (myocardial infarction, hypertension), cerebrovascular disease and frequent relapse of nephrotic syndrome. Objecti

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Guaragna, Mara Sanches, Anna Cristina G. B. Lutaif, Andréa T. Maciel-Guerra, Vera M. S. Belangero, Gil Guerra-Júnior, and Maricilda P. De Mello. "NPHS2Mutations: A Closer Look to Latin American Countries." BioMed Research International 2017 (2017): 1–6. http://dx.doi.org/10.1155/2017/7518789.

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Nephrotic syndrome is one of the most common kidney pathologies in childhood, being characterized by proteinuria, edema, and hypoalbuminemia. In clinical practice, it is divided into two categories based on the response to steroid therapy: steroid-sensitive and steroid resistant. Inherited impairments of proteins located in the glomerular filtration barrier have been identified as important causes of nephrotic syndrome, with one of these being podocin, coded byNPHS2gene.NPHS2mutations are the most frequent genetic cause of steroid resistant nephrotic syndrome. The aim of this review is to upda

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Ladapo, Taiwo Augustina, Christopher Imokhuede Esezobor, and Foluso Ebun Lesi. "High Steroid Sensitivity among Children with Nephrotic Syndrome in Southwestern Nigeria." International Journal of Nephrology 2014 (2014): 1–6. http://dx.doi.org/10.1155/2014/350640.

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Recent reports from both Caucasian and black populations suggest changes in steroid responsiveness of childhood nephrotic syndrome. This study was therefore undertaken to determine the features and steroid sensitivity pattern of a cohort of black children with nephrotic syndrome. Records of children managed for nephrotic syndrome from January 2008 to April 2013 were reviewed. Details including age, response to treatment, and renal histology were analysed. There were 108 children (median age: 5.9 years, peak: 1-2 years), 90.2% of whom had idiopathic nephrotic syndrome. Steroid sensitivity was 8

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Morozov, S. L., V. V. Dlin, V. A. Obukhova, et al. "The importance of determining the concentration of myco- phenolic acid in children with steroid-dependent nephrotic syndrome." Rossiyskiy Vestnik Perinatologii i Pediatrii (Russian Bulletin of Perinatology and Pediatrics) 69, no. 4 (2024): 63–69. http://dx.doi.org/10.21508/1027-4065-2024-69-4-63-69.

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Despite the significant effectiveness of steroid therapy, specialists face serious difficulties in managing patients with steroid-dependent nephrotic syndrome, which requires the prescription of immunosuppressive therapy. Among the available adjuvant drugs, mycophenolate mofetil stands out as a preferred option due to its safety profile, good tolerability, and high efficacy. Currently, only a few studies are presented in the literature on the effectiveness of the use of mycophenolic acid in children with nephrotic syndrome, which is explained by the complexity of the pharmacokinetics of the dr

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Roy, Ranjit Ranjan, Md Rafiqul Islam, Abdul Matin, et al. "Relationship of Childhood Idiopathic Nephrotic Syndrome with Asthma, Hypertension, Complement C₃, Urinalysis." Bangladesh Journal of Child Health 35, no. 1 (2012): 11–15. http://dx.doi.org/10.3329/bjch.v35i1.10367.

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A prospective observational study of 43 children with idiopathic nephrotic syndrome (INS) were selected randomly out of 480 children admitted with the disease at Bangabandhu Sheikh Mujib Medical University, Dhaka, Bangladesh from January 2003 to January 2005. Aim of this study was to correlate the difference in frequency of atopic attack, hypertension, complement c3 level and urinalysis in different types of Idiopathic nephrotic syndrome. Result: Among 43 children with idiopathic nephrotic syndrome (INS), 24 children were steroid sensitive nephrotic syndrome (SSNS) and 19 children having stero

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Islam, Md Ashraful, Taslima Akter, and Iftekhar Ul Hoque Khan. "Seroprotection of Hepatitis B in Children with Steroid Sensitive Nephrotic Syndrome." Medicine Today 34, no. 2 (2022): 99–105. http://dx.doi.org/10.3329/medtoday.v34i2.61422.

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Introduction: Children with Steroid sensitive nephrotic syndrome is associated with lower seroconversion of hepatitis B vaccine due to prolonged period of proteinuria, prolonged period of steroid therapy Objective: The primary aims of this study was to assess the seroconversion rate in children with steroid sensitive nephrotic syndrome (SSNS) who were previously vaccinated against hepatitis B and to compare with the titre of vaccinated healthy children. The secondary aims was to study the association of antiHBs titre in different clinical types of diseases and comparison of persistant antibody

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Sharmim, Shanjida, Golam Muin Uddin, Afroza Begum, Syed Saimul Huque, and Tahmina Jesmin. "Early Recovery of Acute Kidney Injury in a 14-Year-Old Boy with Steroid-Dependent Nephrotic Syndrome with MesPGN and Crescentic Glomerulonephritis." Paediatric Nephrology Journal of Bangladesh 9, no. 1 (2024): 48–51. http://dx.doi.org/10.4103/pnjb.pnjb_17_23.

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Acute kidney injury (AKI) is one of the common complications of steroid-sensitive nephrotic syndrome (SSNS) in children. Therefore, many factors are associated with to development of AKI in SSNS like hypovolemia, infection, use of multiple drugs, and histopathological pattern of the disease process itself. Here, we are reporting a 14-and-half-year-old boy of steroid-dependent nephrotic syndrome and histopathologically mesangioproliferative glomerulonephritis with crescentic glomerulonephritis who developed AKI, which was rapidly progressive glomerulonephritis like presentation 11 years after t

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Kumar, R., J. Prakash, A. Krishna, and KK Singh. "Bardet-Biedl syndrome presenting with steroid sensitive nephrotic syndrome." Indian Journal of Nephrology 25, no. 5 (2015): 300. http://dx.doi.org/10.4103/0971-4065.151765.

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Kodandapani, Yerroju, Ajay Mohan Varahala, and Rajesh Kumar Songa. "Role of urinary nitrites in predicting steroid responsiveness of nephrotic syndrome: a study conducted in tertiary care center." International Journal of Contemporary Pediatrics 7, no. 4 (2020): 918. http://dx.doi.org/10.18203/2349-3291.ijcp20201155.

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Background: Nephrotic Syndrome (NS) is a common chronic disorder, characterized by alterations of selective permeability at the glomerular capillary wall, resulting in its inability to restrict the urinary loss of protein. Urinary nitrite excretion serves as a useful investigation in differentiating between steroid responsive and steroid resistant nephrotic syndrome. The aim of the study was to assess the relation between urinary nitrite levels and steroid responsiveness in nephrotic syndrome in children.Methods: 76 children were enrolled in the study suffering with nephrotic syndrome of which

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Kayes, Mohammad Imrul. "Recent Update in The Management of Childhood Nephrotic Syndrome." Journal of Bangladesh College of Physicians and Surgeons 34, no. 1 (2016): 26–32. http://dx.doi.org/10.3329/jbcps.v34i1.29119.

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Nephrotic Syndrome (NS) is a common renal disease seen in children. Children who go into complete remission following treatment with corticosteroids are classified as having steroid sensitive NS. In developed countries over 80% of children with idiopathic NS have steroid sensitive disease. The exact pathogenesis of this condition remains elusive. Podocyte injury and proteinuria are the two main issues in the pathogenesis. Recent studies suggest release of cytokines by T-cells as well as a strong contribution of Bcell immunity. Genetic studies have reported human leucocyte antigen (HLA) class I

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Tenbrock, K., J. Müller-Berghaus, A. Fuchshuber, D. Michalk, and U. Querfeld. "Levamisole treatment in steroid-sensitive and steroid-resistant nephrotic syndrome." Pediatric Nephrology 12, no. 6 (1998): 459–62. http://dx.doi.org/10.1007/s004670050487.

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Alwan, Ahmed H., and Nawal M. Al-Shammaa. "Evaluation of the Level of Electrolytes in Children with Steroid Sensitive or Steroid Resistant Nephrotic Syndrome." Journal of the Faculty of Medicine Baghdad 66, no. 3 (2024): 276–82. http://dx.doi.org/10.32007/jfacmedbaghdad.6632270.

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Background: Childhood idiopathic nephrotic syndrome is one of the most common conditions pediatric nephrologists encounter globally. Nephrotic syndrome is characterized by proteinuria, hyperlipidemia, and edema. The degree and duration of proteinuria have an impact on serum electrolyte levels. However, local data is limited. Objectives: To assess and compare the degree of electrolyte imbalance and its relationship to kidney function indicators during relapse and remission in children with idiopathic nephrotic syndrome. Methods: In this case-control study, blood samples were collected from 80 I

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Siraeva, T. A., G. R. Sharafiev, R. F. Gatyatullin, E. A. Shtrangar, D. O. Chirikina, and A. V. Aksenov. "Case of steroid-sensitive nephrotic syndrome in a child with orphan disease – leucinosis." Nephrology (Saint-Petersburg) 25, no. 3 (2021): 91–95. http://dx.doi.org/10.36485/1561-6274-2021-25-3-91-96.

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BACKGROUND. The article presents the case of a clinical and laboratory complex of a recurrent hormone-sensitive variant of nephrotic syndrome in a patient with leucinosis (maple syrup disease). THE AIM: to study the clinical laboratory features of nephrotic syndrome in a child with orphan disease – leucinosis. A PATIENT. A 4-year-old child born in a consanguineous marriage, who had previously been diagnosed with a rare genetic disease leucinosis, was examined. The manifestation of leucinosis began from the 8th day of birth. Based on the results of a molecular genetic study the diagnosis was pi

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Savenkova, N. D., and D. D. Batrakov. "The hereditary nephrotic syndrome in children: features of clinical phenotype and genotype, pathogenesis, renal prognosis of isolated and syndromic forms." Rossiyskiy Vestnik Perinatologii i Pediatrii (Russian Bulletin of Perinatology and Pediatrics) 68, no. 2 (2023): 13–21. http://dx.doi.org/10.21508/1027-4065-2023-68-2-13-21.

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The review of the literature presents the clinical phenotype and genotype pathogenesis, renal prognosis of isolated and extra-renal manifestation form of hereditary nephrotic syndrome in children. The clinical and genetic features of hereditary steroid-sensitive and steroid-resistant nephrotic syndrome in children caused by mutations of genes encoding the main components of the slit diaphragm, glomerular basement membrane, structural and functional proteins of the podocyte are highlighted. Literature data demonstrate an unfavorable renal prognosis in children with hereditary steroid-resistant

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Lagodych, Ye, D. Ivanov, L. Vakulenko, and O. Lytvynova. "Clinical case of steroid-dependent nephrotic syndrome in a child." KIDNEYS 12, no. 2 (2023): 119–22. http://dx.doi.org/10.22141/2307-1257.12.2.2023.407.

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Nephrotic syndrome (NS) is a common glomerular pathology encountered in pediatric practice. The main clinical signs are massive proteinuria, hypoalbuminemia, hyperlipidemia, and edema. Among all cases of NS, 75 % of children have a histological variant of glomerular lesions in the form of minimal change disease that is sensitive to hormone therapy, but easily leads to relapse and steroid dependence. These children often need to extend the time of taking hormonal drugs or add other immunosuppressants, which can have significant toxicity. Available immunosuppressant treatment options include cy

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Bagga, Arvind. "Therapies for Glomerular Diseases in Children." Annals of the National Academy of Medical Sciences (India) 54, no. 01 (2018): 043–53. http://dx.doi.org/10.1055/s-0040-1712821.

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AbstractNephrotic syndrome is an important chronic disease of childhood, with a steroid sensitive course in most patients. Research on pathogenesis has emphasized the importance of T-lymphocyte dysregulation and vascular permeability factors that alter podocyte function and glomerular permselectivity. Mutations in genes that encode important podocyte proteins and therapeutic targets within podocytes have been identified. A hypothesis unifying available evidence on pathogenesis is yet to be proposed. An important proportion of patients have difficult disease course, characterized by frequent re

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Sadr Moharrerpour, Sahar, Azar Nickavar, Simin Sadeghi Bojd, Sara Makhtomi, and Hossein Ghorbani. "Serum interleukin-18 in children with steroid sensitive nephrotic syndrome." Journal of Renal Injury Prevention 8, no. 4 (2019): 289–91. http://dx.doi.org/10.15171/jrip.2019.53.

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Introduction: Idiopathic nephrotic syndrome has been considered a cell-mediated immunity disorder with T cell dysfunction. Objectives: The purpose of this study was to investigate immunologic dysfunction in children with steroid-sensitive nephrotic syndrome (SSNS) by evaluating serum interleukin-18 (IL-18). Patients and Methods: A total of 30 children with SSNS (n=20 males, n=10 females), aged 1–11 years were enrolled in this study. All of them were treated with steroids at the time of study. Serum IL-18 was compared during both active and remission phases of nephrotic syndrome. In addition, c

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Hodson, Elisabeth M., and Stephen I. Alexander. "Evaluation and management of steroid-sensitive nephrotic syndrome." Current Opinion in Pediatrics 20, no. 2 (2008): 145–50. http://dx.doi.org/10.1097/mop.0b013e3282f4307a.

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Mohan, KR, and M. Kanitkar. "Growth in Children with Steroid Sensitive Nephrotic Syndrome." Medical Journal Armed Forces India 65, no. 1 (2009): 4–6. http://dx.doi.org/10.1016/s0377-1237(09)80043-9.

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Weisz, Wanja, Markus J. Kemper, Jochen Weil, and Dirk E. Müller-Wiefel. "Asymptomatic intracardiac thrombus in steroid-sensitive nephrotic syndrome." Pediatric Nephrology 17, no. 4 (2002): 287–89. http://dx.doi.org/10.1007/s00467-002-0831-4.

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