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1

Plante, Richard La. Steroid blues. Little, Brown, 1995.

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2

J, Alexander Nancy, and D'Arcangues C, eds. Steroid hormones and uterine bleeding. AAAS Press, 1992.

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3

Rachel, Snow, Hall Peter 1943-, and Symposium on Steroid Contraceptives and Women's Response: Regional Variability in Side-Effects and Steroid Pharmacokinetics (1990 : Exeter, N.H.), eds. Steroid contraceptives and women's response: Regional variability in side-effects and pharmacokinetics. Plenum Press, 1994.

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4

T, Gregoire A., Blye Richard P, United States. Food and Drug Administration. Fertility and Maternal Health Drugs Advisory Committee., and Workshop on Animal Testing Requirements for New Generation Steroidal Contraceptives (1983 : National Institutes of Health), eds. Contraceptive steroids: Pharmacology and safety. Plenum Press, 1986.

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5

Vane, John R. Improved non-steroid anti-flammatory drugs COX-2 enzyme inhibitors: Proceedings of a conference held on October 10-11, 1995, at Regent's College, London. Kluwer Academic Publishers, 1996.

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6

William Harvey Conference (1995 London, England). Improved non-steroid anti-inflammatory drugs: COX-2 enzyme inhibitors : proceedings of a conference held on October 10-11, 1995, at Regent's College, London. Kluwer Academic, 1996.

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7

Yuuki, Inoue, and Watanabe Kouki, eds. Adverse effects of steroids. Nova Science Publishers, 2008.

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8

National Institute on Drug Abuse., ed. Anabolic steroids: A threat to body and mind. National Institute on Drug Abuse, 1991.

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9

1941-, Rainsford K. D., Velo G. P, Università di Verona. Institute of Pharmacology., International Meeting on the Side-effects of Anti-inflammatory Analgesic Drugs (3rd : 1991 : Verona, Italy), and European Workshop on Inflammation (13th : 1991 : Verona, Italy), eds. Side-effects of anti-inflammatory drugs 3. Kluwer Academic Publishers, 1992.

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10

Martin, Bohl, and Duax William L. 1939-, eds. Molecular structure and biological activity of steroids. CRC Press, 1992.

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11

Interdisciplinary Workshop (1st 1996 Tübingen, Germany). Sex steroids and the cardiovascular system: The proceedings of the 1st Interdisciplinary Workshop, Tuebingen, Germany, October 1996. Parthenon Pub., 1998.

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12

1953-, Kurowski M., ed. Adverse reactions to non-steroidal anti-inflammatory drugs: Clinical pharmacoepidemiology. Birkhäuser Verlag, 1992.

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13

Jean, Ginsburg, Lippert T. H, and Mueck A. O, eds. Sex steroids and the cardiovascular system: The proceedings of the 1st Interdisciplinary Workshop, Tubingen, Germany, October 1996. Parthenon Pub., 1997.

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14

T, Borda Ivan, and Koff Raymond S. 1939-, eds. NSAIDs: A profile of adverse effects. Hanley & Belfus, 1992.

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15

D, Catto Graeme R., ed. Drugs and the kidney. Kluwer Academic Publishers, 1990.

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16

Plante, Richard La. Steroid Blues. Little, Brown Book Group Limited, 1996.

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17

Peter, Hall, and Rachel Snow. Steroid Contraceptives and Women's Response: Regional Variability in Side-Effects and Steroid Pharmacokinetics. Springer, 2012.

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18

Snow, Rachel. Steroid Contraceptives and Women's Response: Regional Variability In Side-Effects And Steroid Pharmacokinetics. Springer, 2012.

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19

Steroid Contraceptives and Women's Response: Regional Variability in Side-Effects and Steroid Pharmacokinetics. Island Press, 1994.

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20

Steroid blues. Tom Docherty Associates, 1995.

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21

Steroid blues. Forge, 1995.

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22

(Editor), Rachel Snow, and Peter Hall (Editor), eds. Steroid Contraceptives and Women's Response:: Regional Variability in Side-Effects and Steroid Pharmacokinetics (Reproductive Biology). Springer, 1994.

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23

Gregoire, A. T. Contraceptive Steroids: Pharmacology and Safety. Springer, 2013.

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24

Gregoire, A. T. Contraceptive Steroids: Pharmacology and Safety. Springer London, Limited, 2013.

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25

Contraceptive Steroids : Pharmacology and Safety (Reproductive Biology). Springer, 1986.

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26

Niaudet, Patrick, and Alain Meyrier. Minimal change disease. Edited by Neil Turner. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0056_update_001.

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Minimal change disease is characteristically responsive to high-dose corticosteroids. As this is the most common cause of nephrotic syndrome in children, and responses are usually prompt, response to 60 mg/m2/day of oral prednisolone (max. 80 mg) is often used as a diagnostic test. Adults respond more slowly and have a wider differential diagnosis, and often a high risk of side effects, so therapy is not recommended without confirmation by renal biopsy. Then first-line treatment is again prednisolone or prednisone, at 1 mg/kg/day (max. 60 mg). KDIGO and other treatment protocols recommend 6 we
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27

Sauvage, Alexandre, and Maxime Levy. Dexamethasone: Therapeutic Uses, Mechanism of Action and Potential Side Effects. Nova Science Publishers, Incorporated, 2013.

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28

gym. Steroids : Use, Side Effects, and Ways to Reduce Bad Effects,: How to Reduce Harmful Effects and Avoid Complications, How Do Steroids Make Users Feel, Myths about Steroids, Facts about Steroids. Independently Published, 2020.

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29

Side-Effects of Anti-Inflammatory Drugs 3. Springer, 2011.

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30

Rainsford, K. D., and G. P. Velo. Side-Effects of Anti-Inflammatory Drugs 3. Springer Netherlands, 2012.

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31

Rainsford, K. D., and G. P. Velo. Side-Effects of Anti-Inflammatory Drugs 3. Springer, 2012.

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32

(Editor), T. H. Lippert, and A. O. Mueck (Editor), eds. Sex Steroids and the Cardiovascular System: The Proceedings of the 1st Interdisciplinary Workshop, Tuebingen, Germany. Informa Healthcare, 1997.

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33

Bohl, Martin. Molecular Structure and Biological Activity of Steroids. Taylor & Francis Group, 2018.

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34

Bohl, Martin. Molecular Structure and Biological Activity of Steroids. Taylor & Francis Group, 2018.

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35

Bohl, Martin. Molecular Structure and Biological Activity of Steroids. Taylor & Francis Group, 2018.

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36

Bohl, Martin. Molecular Structure and Biological Activity of Steroids. Taylor & Francis Group, 2018.

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37

Molecular Structure and Biological Activity of Steroids. Taylor & Francis Group, 2017.

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38

McClenahan, Maureen F., and William Beckman. Pain Management Techniques. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190217518.003.0011.

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This chapter provides a broad review of various interventional pain management procedures with a focus on indications, anatomy, and complications. Specific techniques reviewed include transforaminal epidural steroid injection, lumbar sympathetic block, stellate ganglion block, cervical and lumbar radiofrequency ablation, gasserian ganglion block, sacroiliac joint injection, celiac plexus block, lateral femoral cutaneous nerve block, ilioinguinal block, lumbar medial branch block, obturator nerve block, ankle block, occipital nerve block, superior hypogastric plexus block, spinal cord stimulati
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39

Keh, Didier. Steroids in critical illness. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0054.

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The benefit of prolonged application of moderate-dose corticosteroids in systemic inflammatory diseases remains controversial. In critical illness, the endogenous cortisol effect may become insufficient due to adrenal dysfunction and corticosteroid resistance to counterbalance an exaggerated and protracted inflammatory response, which has been termed ‘critical illness-related corticosteroid insufficiency’ (CIRCI). There is evidence that moderate-dose hydrocortisone (200–300 mg/day) significantly fastens shock reversal in patients with septic shock, but may improve survival probably only in pat
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40

Bloom, Chloe, and Seamus Donnelly. Pulmonary sarcoidosis. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199657742.003.0019.

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This case of a young female with suspected pulmonary sarcoidosis demonstrates the difficulties in confirming the diagnosis and subsequently identifying the appropriate treatment. Current guidelines were developed in the 1990s, and there has been little change in the diagnostic pathway since then. However, there are new clinical tools to help differentiate from the common differential diagnosis of tuberculosis. The patient’s management can be complex, with a host of clinical parameters that can be potentially used to assess each patient’s disease activity, severity, and prognosis, and the decis
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41

Kurowski, M. Adverse Reactions to Non-Steroidal Anti-Inflammatory Drugs: Clinical Pharmacoepidemiology. Birkhauser Verlag, 2013.

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42

Kurowski, M. Adverse Reactions to Non-Steroidal Anti-Inflammatory Drugs: Clinical Pharmacoepidemiology. Birkhäuser, 2013.

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43

Wenham, Claire Y. J., and Philip G. Conaghan. Osteoarthritis—management. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0140.

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Osteoarthritis (OA) is a common condition which often causes pain and functional limitation, significantly impacting on a person's quality of life. A comprehensive assessment of the impact of OA should be performed before selecting therapies and treatment goals. Current recommended therapies include a combination of pharmacological and non-pharmacological therapies, which should be considered for all people with OA, regardless of anatomical site of involvement. Non-pharmacological treatments include education, muscle strengthening and aerobic exercises, weight loss if appropriate, splints and
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44

Kurowski, M. Adverse Reactions to Non-Steroidal Anti-Inflammatory Drugs: Clinical Pharmacoepidemiology (Advances in Pharmacological Sciences). Birkhauser, 1992.

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45

Kurowski, M. Adverse Reactions to Non-Steroidal Anti-Inflammatory Drugs: Clinical Pharmacoepidemiology (Agents and Actions Supplements). Birkhauser, 1992.

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46

Side-Effects of Anti-Inflammatory Drugs: Volume 3 (Inflammation and Drug Therapy Series). Springer, 1992.

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47

Borda, Ivan T., and Raymond S., M.D. Koff. Nsaids: A Profile of Adverse Effects. Lippincott Williams & Wilkins, 1992.

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48

Armstrong, Sarah L., and Gary M. Stocks. Postoperative analgesia after caesarean delivery. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198713333.003.0024.

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Caesarean delivery (CD) is one of the most common operations in the world and providing effective pain relief is important not only for humanitarian reasons but also to speed up recovery and reduce postoperative complications. An understanding of the anatomy and physiology of pain transmission after CD has led to a multimodal approach to analgesia. This involves combining analgesics which work by different mechanisms resulting in an additive effect whilst at the same time reducing side effects. In contemporary practice, most CDs are carried out under neuraxial anaesthesia and neuraxial techniq
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49

Borda, Ivan T. NSAIDs. Hanley & Belfus, 1992.

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50

Sjøgren, Per, Frank Elsner, and Stein Kaasa. Non-opioid analgesics. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199656097.003.0096.

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Non-opioid analgesics encompass the non-steroidal anti-inflammatory drugs (NSAIDs) and paracetamol (acetaminophen). The NSAIDs include acetylsalicylic acid (ASA, aspirin), dipyrone (metamizole), and numerous other drugs in diverse classes. The NSAIDs have potent anti-inflammatory, analgesic and antipyretic activity, and are among the most widely used drugs worldwide. In palliative medicine, they represent the first step of the World Health Organization’s analgesic ladder used for mild pain and they are an important supplement to opioids and adjuvant drugs at higher steps of the ladder. The dis
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