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Dissertations / Theses on the topic 'Stevens Johnson's syndrome'

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Published: 3 June 2025

Last updated: 31 July 2025

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1

Chartier, Claire Patey Olivier. "Infection à Mycoplasma pneumoniae et syndrome de Stevens-Johnson." Créteil : Université de Paris-Val-de-Marne, 2006. http://doxa.scd.univ-paris12.fr:80/theses/th0236637.pdf.

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2

Leclercq, Emmanuel. "Syndrome de stevens-johnson associee a un syndrome de detresse respiratoire de type adulte : un cas pediatrique." Lille 2, 1989. http://www.theses.fr/1989LIL2M392.

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3

Gaultier, Frédérick. "Contribution à l'étude de la nécrolyse épidermique toxique et du syndrome de Stevens-Johnson." Paris 5, 2006. http://www.theses.fr/2006PA05M001.

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La nécrolyse épidermique toxique et le Syndrome de Stevens-Johnson sont des toxidermies médicamenteuses rares d'étiologies inconnues. Au cours de notre étude nous avons cherché à préciser les mécanismes physiopathogéniques impliqués dans la genèse de ces pathologies. L'histologie a montré une atteinte et une désorganisation modérées de la matrice extracellulaire. L'immunohistochimie et les études biochimiques nous ont permis de mettre en évidence un déséquilibre de la balance MMPs/TIMPs en faveur des MMPs avec un schéma d'expression essentiellement épidermique. Enfin, nous proposons un process

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4

Van, Zyl Lourens Marthinus. "Prevalence of chronic ocular complications in Stevens-Johnson Syndrome and toxic epidermal necrolysis." Master's thesis, University of Cape Town, 2013. http://hdl.handle.net/11427/2899.

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Includes abstract.<br>Includes bibliographical references.<br>The main objective of the study is to identify and grade the severity of chronic ocular complications in patients who were treated for SJS and TEN at Groote Schuur Hospital. The secondary objective is to identify patients who need referrals to specialist ophthalmic clinics for treatable ocular complications of SJS and TEN.

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5

Wolkenstein, Pierre. "Prédisposition métabolique et mécanisme de mort cellulaire au cours du syndrome de Stevens-Johnson et de la nécrolyse épidermique toxique." Paris 12, 1996. http://www.theses.fr/1996PA120079.

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Le but de notre travail etait d'etudier les predispositions metaboliques et la mort keratinocytaire au cours de la necrolyse epidermique toxique. Nous avons montre que: 1) la necrolyse epidermique toxique aux sulfamides et aux anticomitiaux etait liee a un deficit hautement specifique de la detoxication des metabolites reactifs du medicament responsable, constitutionnel et genetiquement transmis ; 2) un genotype d'acetylation lente est un facteur de risque de la necrolyse epidermique toxique induite par les sulfamides ; 3) le cytochrome p450 3a4 a un role majeur dans la formation des metabolit

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6

Poletti, Jabbour Jamil, Rospigliosi Andrés Wiegering, Elías Reneé Pereyra, and Barrera Carmen Cecilia Elías. "Carbamazepine and oxcarbazepine: reflections after an oxcarbazepine-induced Stevens-Johnson syndrome/toxic epidermal necrolysis overlap." Springer International Publishing, 2016. http://hdl.handle.net/10757/609483.

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7

AMRAOUI, ABDERRAHIM. "L'erytheme polymorphe majeur ou syndrome de stevens-johnson associe a une infection a mycoplasma pneumoniae : une observation pediatrique." Lille 2, 1993. http://www.theses.fr/1993LIL2M149.

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8

Giron, Sabrina Delverdier Maxence. "L'Érythème polymorphe chez le chien et le chat données bibliographiques récentes /." [S.l.] : [s.n.], 2008. http://oatao.univ-toulouse.fr/1147/1/jan_1147.pdf.

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9

Cooper, Ryan. "Intravenous Immunoglobulin Use in the Treatment of Toxic Epidermal Necrolysis and Stevens-Johnson Syndrome: A 10-year Retrospective Analysis of Patients of a Single Burn Center." Thesis, The University of Arizona, 2014. http://hdl.handle.net/10150/315845.

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A Thesis submitted to The University of Arizona College of Medicine - Phoenix in partial fulfillment of the requirements for the Degree of Doctor of Medicine.<br>Stevens - Johnson syndrome and Toxic Epidermal Necrolysis Syndrome are rare, but serious conditions affecting skin and mucous membranes that are primarily treated with supportive care. Other more specific therapies have limited evidence to support the benefit of their use; one such treatment is intravenous immunoglobulin (IVIG). The use of IVIG in the treatment of these syndromes remain controversial due to mixed results demonstrated

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10

Tanno, Luciana Kase. "Estudo de associação de fatores genéticos em indivíduos com reações de hipersensibilidade tardia induzida por anticonvulsivantes aromáticos." Universidade de São Paulo, 2014. http://www.teses.usp.br/teses/disponiveis/5/5146/tde-01122014-113756/.

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Intrdodução: As terapias com anticonvulsivantes de anel aromático (ACA) são freqüentemente associadas a reações adversas. No entanto, reações de hipersensibilidade (RH) não-imediatas (tardias) a estes fármacos são raras, imprevisíveis e geralmente relacionadas à alta morbidade e mortalidade. Foi demonstrado que estas RH aos ACA estão fortemente associadas ao Antígenio de Leucócitos Humanos (HLA)-B*1502 em pacientes chineses e ao HLA-A*3101 em caucasianos. Polimorfismos de genes do metabolismo do Citocromo P450 (CYP)2C9 foram mais associados a estas reações em pacientes orientais. Objetivo: Nos

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11

Chu, Hong-Yi, and 朱紘毅. "Applying Sequential Pattern Approach to Identify Clinic Sequence - An Example of Stevens-Johnson Syndrome." Thesis, 2016. http://ndltd.ncl.edu.tw/handle/z9sev5.

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碩士<br>國立臺灣科技大學<br>工業管理系<br>104<br>Stevens-Johnson syndrome (SJS), a form of toxic epidermal necrolysis, is a lifethreatening skin condition, in which cell death causes the epidermis to separate from the dermis. There are many factors cause SJS, most of SJS cases are caused by using improper drugs. The drugs which cause SJS are used in various medical fields. However, very few physician are familiar with all these drugs, so there are many cases when physician is not aware about the drugs causing SJS. This study uses 1-year data of Taiwan’s NHIRD (National Health Insurance Research Database) to

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12

Liu, Yi-pei, and 劉宜佩. "Developing a Mining Approach to Investigate Physician Prescription Behavior-An Example of Stevens-Johnson Syndrome." Thesis, 2011. http://ndltd.ncl.edu.tw/handle/72297871776297192905.

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碩士<br>國立臺灣科技大學<br>工業管理系<br>99<br>In clinical practice, a severe illness might be the result of an untoward reaction to the pharmacological treatment for another disease. For instance, Stevens-Johnson syndrome (SJS) can be caused by antibiotics; nonsteroidal anti-inflammatory drugs; antiepileptics; antugout agents; and cardiovascular drugs, as well as the drugs for local use in ophthalmology and dermatology. Since there are so many drugs covering various different medical fields, very few physicians are familiar with all the potential drugs that can cause SJS. Hence new cases or even recurrent

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13

Freitas, Daniel Vilaverde. "Síndrome Stevens-Johnson / Necrólise Epidérmica Tóxica - foco na fisiopatologia e tratamento." Master's thesis, 2018. http://hdl.handle.net/10316/82801.

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Trabalho Final do Mestrado Integrado em Medicina apresentado à Faculdade de Medicina<br>Stevens-Johnson syndrome and toxic epidermal necrolysis are part of a spectrum of rare but acute and potentially life-threatening mucocutaneous reactions. The vast majority of cases are caused by medication, with allopurinol, non-steroidal anti-inflammatory drugs, antibiotics and anticonvulsants as the most frequently involved drugs. Generally, the reaction occurs within 7-21 days of the onset of drug administration.The main manifestation of SJS/TEN is a painful erythema with extensive exfoliation of the ep

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14

Han-Yu, Shih. "The study of T cell repertoire in patients with Carbamazepine-induced Stevens-Johnson syndrome (CBZ-SJS)." 2006. http://www.cetd.com.tw/ec/thesisdetail.aspx?etdun=U0001-2301200613060900.

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15

Shih, Han-Yu, and 史涵宇. "The study of T cell repertoire in patients with Carbamazepine-induced Stevens-Johnson syndrome (CBZ-SJS)." Thesis, 2006. http://ndltd.ncl.edu.tw/handle/71313853353789974805.

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碩士<br>國立臺灣大學<br>微生物學研究所<br>94<br>Stevens-Johnson Syndrome (SJS) is a severe, life-threatening cutaneous adverse reaction, most often caused by medication. Growing evidence indicates that CD8+ cytotoxic T lymphocytes, which can be activated by specific antigen-MHC complex expressed on the surface of antigen-presenting cells (APC), are involved in the pathogenesis of SJS. In our previous studies, we found that SJS induced by carbamazepine (CBZ), a commonly prescribed anticonvulsant, is strongly associated with HLA-B*1502 allele. Because specific T cell receptor (TCR) recognition of drug-bound pe

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16

Wei, Chun-Yu, and 魏淳郁. "Pathological role of HLA-B*1502 in carbamazepine-induced Stevens-Johnson syndrome and toxic epidermal necrolysis." Thesis, 2012. http://ndltd.ncl.edu.tw/handle/53019387631640777046.

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博士<br>國立陽明大學<br>生化暨分子生物研究所<br>100<br>Increasing studies revealed that HLA alleles are the major genetic determinants of drug hypersensitivity; however, the underlying molecular mechanism remains unclear. Here, we adopt the HLA-B*1502 genetic predisposition to carbamazepine (CBZ)-induced Stevens-Johnson syndrome (SJS)/toxic epidermal necrolysis (TEN) as a model to study the pathological role of HLA in delayed-type drug hypersensitivity. We in vitro expanded CBZ-specific cytotoxic T lymphocytes (CTLs) from CBZ-SJS/TEN patients and analyzed the interaction between HLA-B and CBZ/analogs by CTLs re

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17

Chih-Wen, Ou Yang. "Pathogenesis of carbamazepine-induced Stevens-Johnson Syndrome : Identification of the drug-modified peptides bind to the HLA-B*1502." 2005. http://www.cetd.com.tw/ec/thesisdetail.aspx?etdun=U0001-2607200514580000.

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18

Yang, Chih-Wen Ou, and 歐陽志玟. "Pathogenesis of carbamazepine-induced Stevens-Johnson Syndrome :Identification of the drug-modified peptides bind to the HLA-B*1502." Thesis, 2005. http://ndltd.ncl.edu.tw/handle/76086018110533190277.

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碩士<br>國立臺灣大學<br>微生物學研究所<br>94<br>Stevens-Johnson syndrome (SJS) and toxic epidermal necrolysis (TEN) are related life-threatening cutaneous adverse reactions most often caused by medication. Carbamazepine (CBZ), a commonly prescribed anticonvulsant, is the number one culprit drug associated with SJS in Taiwan, accounts for 25% of all drug-induced SJS. Susceptibility to drug induced idiosyncratic reactions is thought to be genetically determined and immune-mediated. Previous studies suggest that the pathogenesis of the severe cutaneous adverse drug reactions involves MHC-restricted presentation

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19

Liang, Hsin-Lin, and 梁忻琳. "Correlation between Stevens-Johnson Syndrome belonged to Serious Adverse Drug Events and Drug- Drug Interaction in case of death in Taiwan." Thesis, 2009. http://ndltd.ncl.edu.tw/handle/69400147324332897020.

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碩士<br>高雄醫學大學<br>藥學研究所碩士在職專班<br>98<br>Background/Aim: Combination of some drugs would cause interactions between drugs, and this kind of interactions could lead to side effect. So far, there were less related reports about SJS in combination of drugs. Therefore, we study the relationship between drug combination and mortal of SJS that to prevent SIS which lead to death by retrospective study. Methods: This longitude and retrospective study uses mortality cases that diagnoses as ICD-9-CM 695.1 from NHI from 1999 to 2008. Statistical comparisons of the results were made using analysis of variance

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20

Leisinger, Silvia [Verfasser]. "Schwere arzneimittelinduzierte Hautreaktionen : Stevens-Johnson-Syndrom (SJS) und toxisch epidermale Nekrolyse (TEN) - und deren ophthalmologische Folgeerscheinungen / vorgelegt von Silvia Leisinger." 2009. http://d-nb.info/996037330/34.

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21

Agustianty, Sheila, and 李雪樂. "Developing a Data Mining Approach to Investigate Association between Physician Prescription and Patient Outcome – A Study on Rehospitalization in Stevens-Johnson Syndrome." Thesis, 2012. http://ndltd.ncl.edu.tw/handle/62483452313655168744.

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碩士<br>國立臺灣科技大學<br>工業管理系<br>100<br>Complications of drug therapy are the most common type of adverse events in hospitalized patients. The example includes Stevens-Johnson syndrome (SJS), a life-threatening skin reactions to medications. Since drugs are the important causes of SJS, the first and most important step in treating SJS is to discontinue any medication that may be causing it. But the potential drugs that may lead SJS spread extensively in various medical fields and very few physicians are familiar with all these drugs. There is a case when physician is not aware about the drugs causin

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22

Chang, Chih-Chien, and 張之倩. "Applying Time Interval Sequential Pattern Mining Approach to Investigate the Patients’ Medication Sequence and Length of Stays Before Suffering Steven Johnson Syndrome." Thesis, 2017. http://ndltd.ncl.edu.tw/handle/4q6839.

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碩士<br>國立臺灣科技大學<br>工業管理系<br>105<br>With plenty of convenient medical resources, it is easy for patients to see a doctor in several hospitals with the same disease or let patients get duplicated medication or interactions between multiple drugs. On the other hand, in the investigation of Taiwan Drug relief system, it indicated that Stevens Johnson Syndrome (shortly as SJS in the following paper) was the main case for application. Therefore, the aim of this study is to use the time interval sequential pattern to explore the patients’ medication sequence and length of stays before suffering Steven

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23

Ko, Tai-Ming, and 柯泰名. "The pathogenic role of T cells in severe adverse drug reactions: A molecular and functional analysis of T cell receptor repertoire in patients with Carbamazepine-induced Stevens-Johnson Syndrome." Thesis, 2011. http://ndltd.ncl.edu.tw/handle/02255200972657449506.

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博士<br>國立臺灣大學<br>微生物學研究所<br>100<br>Drug hypersensitivity remains a major clinical problem. Stevens-Johnson syndrome (SJS) and the related disease toxic epidermal necrolysis (TEN) are life-threatening drug hypersensitivities with robust immune responses to drugs. Despite the strong genetic association between HLA-B*1502 and carbamazepine (CBZ)-induced SJS/TEN, it is not known whether particular T-cell receptor (TCR) repertoires participate in the recognition of small drug-peptide-HLA complexes in T cell-mediated drug hypersensitivity. Using the strong HLA predisposition in CBZ-SJS as a model, we

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24

Oliveira, Ana Rita de Jesus. "Caracterização de Reações de Hipersensibilidade a Medicamentos." Master's thesis, 2017. http://hdl.handle.net/10316/83651.

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Relatório de Estágio do Mestrado Integrado em Ciências Farmacêuticas apresentado à Faculdade de Farmácia<br>A base da intervenção médica é a utilização de medicamentos que estão muitas vezes associados a complicações inerentes ao seu uso, sendo uma das principais causas da ocorrência de eventos adversos nos cuidados de saúde.A definição de RAM é vaga. Deve esclarecer-se que não são resultantes apenas da “utilização autorizada de um medicamento em doses normais, mas também dos erros terapêuticos e das utilizações fora dos termos da autorização de introdução no mercado, incluindo a utilização in

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