Relevant bibliographies by topics / Stimolo purinergico

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  1. Journal articles
  2. Dissertations / Theses
  3. Book chapters
  4. Conference papers

Academic literature on the topic 'Stimolo purinergico'

Author: Grafiati
Published: 9 March 2023
Last updated: 31 July 2025

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Journal articles on the topic "Stimolo purinergico"

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Shirai, Y., K. Kashiwagi, N. Sakai, and N. Saito. "Phospholipase A(2) and its products are involved in the purinergic receptor-mediated translocation of protein kinase C in CHO-K1 cells." Journal of Cell Science 113, no. 8 (2000): 1335–43. http://dx.doi.org/10.1242/jcs.113.8.1335.

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The signal transduction involved in the purinergic stimuli-induced activation of protein kinase C (PKC) in CHO-K1 cells was investigated. Purinergic stimuli such as adenosine triphosphate and uridine triphosphate induced a transient translocation of PKC epsilon, gamma, and delta from the cytoplasm to the plasma membrane. These translocations were blocked by an inhibitor of phosphatidylinositol-specific phospholipase C (PLC), but not by an inhibitor of phosphatidylcholine-specific PLC. A diacylglycerol (DAG) analogue also induced reversible translocations of PKC gamma, epsilon, and delta from t
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Spek, Annabel, Bingsheng Li, Beata Rutz, et al. "Purinergic smooth muscle contractions in the human prostate: estimation of relevance and characterization of different agonists." Naunyn-Schmiedeberg's Archives of Pharmacology 394, no. 6 (2021): 1113–31. http://dx.doi.org/10.1007/s00210-020-02044-4.

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AbstractNon-adrenergic prostate smooth muscle contractions may account for the limited effectiveness of α1-adrenoceptor antagonists, which are the first-line option for medical treatment of voiding symptoms suggestive of benign prostatic hyperplasia. In non-human prostates, purinergic agonists induce contractions reaching similar magnitudes as α1-adrenergic contractions. However, evidence for the human prostate is highly limited, and pointed to much weaker purinergic contractions. Here, we examined contractions of different purinergic agonists in human prostate tissues. Tissues were obtained f
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Klausen, Polina, Daria Semenova, Daria Kostina, Vladimir Uspenskiy, and Anna Malashicheva. "Purinergic Signaling in Pathologic Osteogenic Differentiation of Aortic Valve Interstitial Cells from Patients with Aortic Valve Calcification." Biomedicines 11, no. 2 (2023): 307. http://dx.doi.org/10.3390/biomedicines11020307.

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Purinergic signaling is associated with a vast spectrum of physiological processes, including cardiovascular system function and, in particular, its pathological calcifications, such as aortic valve stenosis. Aortic valve stenosis (AS) is a degenerative disease for which there is no cure other than surgical replacement of the affected valve. Purinergic signaling is known to be involved in the pathologic osteogenic differentiation of valve interstitial cells (VIC) into osteoblast-like cells, which underlies the pathogenesis of AS. ATP, its metabolites and related nucleotides also act as signali
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Tingler, Anna, Charulekha Packirisamy, and Kristen Engevik. "EPITHELIAL PURINERGIC RECEPTORS ARE DOWNREGULATED IN PATIENTS WITH INFLAMMATORY BOWEL DISEASE." Inflammatory Bowel Diseases 31, Supplement_1 (2025): S46. https://doi.org/10.1093/ibd/izae282.109.

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Abstract BACKGROUND Purines are among the most influential and ubiquitous signaling molecules. In the human body, there are 19 different purinergic receptor subtypes that are classified into P1 and P2 receptors. Several studies have revealed crucial roles for P2 purinergic receptors during inflammatory and infectious diseases experimentally using animal models. However, the distribution of P1 and P2 receptors in the human colon has not been fully mapped. Additionally, there is little information about changes in purinergic receptors in the setting of inflammatory bowel disease (IBD). We hypoth
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Zhang, Yong, and William G. Paterson. "Characterization of the peristaltic reflex in murine distal colon." Canadian Journal of Physiology and Pharmacology 94, no. 2 (2016): 190–98. http://dx.doi.org/10.1139/cjpp-2015-0086.

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Ascending and descending neuromuscular reflexes play an important role in gastrointestinal motility. However, the underlying mechanisms in colon are incompletely understood. Nerve stimulation (NS)- and balloon distention (BD)-mediated reflexes in distal colonic circular smooth muscle (CSM) and longitudinal smooth muscle (LSM) of mice were investigated using conventional intracellular recordings. In the CSM, NS evoked ascending purinergic inhibitory junction potentials (IJPs), whereas BD induced atropine-sensitive ascending depolarization with superimposed action potentials (APs). The ascending
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Guzman-Aranguez, Ana, Xavier Gasull, Yolanda Diebold, and Jesús Pintor. "Purinergic Receptors in Ocular Inflammation." Mediators of Inflammation 2014 (2014): 1–11. http://dx.doi.org/10.1155/2014/320906.

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Inflammation is a complex process that implies the interaction between cells and molecular mediators, which, when not properly “tuned,” can lead to disease. When inflammation affects the eye, it can produce severe disorders affecting the superficial and internal parts of the visual organ. The nucleoside adenosine and nucleotides including adenine mononucleotides like ADP and ATP and dinucleotides such as P1,P4-diadenosine tetraphosphate (Ap4A), and P1,P5-diadenosine pentaphosphate (Ap5A) are present in different ocular locations and therefore they may contribute/modulate inflammatory processes
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Chen, Zhiyong, Chi Zhang, Xiaodan Song, et al. "BzATP Activates Satellite Glial Cells and Increases the Excitability of Dorsal Root Ganglia Neurons In Vivo." Cells 11, no. 15 (2022): 2280. http://dx.doi.org/10.3390/cells11152280.

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The purinergic system plays an important role in pain transmission. Recent studies have suggested that activation of P2-purinergic receptors (P2Rs) may be involved in neuron-satellite glial cell (SGC) interactions in the dorsal root ganglia (DRG), but the details remain unclear. In DRG, P2X7R is selectively expressed in SGCs, which closely surround neurons, and is highly sensitive to 3’-O-(4-Benzoyl) benzoyl-ATP (BzATP). Using calcium imaging in intact mice to survey a large number of DRG neurons and SGCs, we examined how intra-ganglionic purinergic signaling initiated by BzATP affects neurona
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Muñoz, Manuel F., Theanne N. Griffith, and Jorge E. Contreras. "Mechanisms of ATP release in pain: role of pannexin and connexin channels." Purinergic Signalling 17, no. 4 (2021): 549–61. http://dx.doi.org/10.1007/s11302-021-09822-6.

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AbstractPain is a physiological response to bodily damage and serves as a warning of potential threat. Pain can also transform from an acute response to noxious stimuli to a chronic condition with notable emotional and psychological components that requires treatment. Indeed, the management of chronic pain is currently an important unmet societal need. Several reports have implicated the release of the neurotransmitter adenosine triphosphate (ATP) and subsequent activation of purinergic receptors in distinct pain etiologies. Purinergic receptors are broadly expressed in peripheral neurons and
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Scarpellino, Giorgia, Tullio Genova, Daniele Avanzato, et al. "Purinergic Calcium Signals in Tumor-Derived Endothelium." Cancers 11, no. 6 (2019): 766. http://dx.doi.org/10.3390/cancers11060766.

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Tumor microenvironment is particularly enriched with extracellular ATP (eATP), but conflicting evidence has been provided on its functional effects on tumor growth and vascular remodeling. We have previously shown that high eATP concentrations exert a strong anti-migratory, antiangiogenic and normalizing activity on human tumor-derived endothelial cells (TECs). Since both metabotropic and ionotropic purinergic receptors trigger cytosolic calcium increase ([Ca2+]c), the present work investigated the properties of [Ca2+]c events elicited by high eATP in TECs and their role in anti-migratory acti
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Scarpellino, Giorgia, Tullio Genova, Elisa Quarta, et al. "P2X Purinergic Receptors Are Multisensory Detectors for Micro-Environmental Stimuli That Control Migration of Tumoral Endothelium." Cancers 14, no. 11 (2022): 2743. http://dx.doi.org/10.3390/cancers14112743.

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The tumoral microenvironment often displays peculiar features, including accumulation of extracellular ATP, hypoxia, low pH-acidosis, as well as an imbalance in zinc (Zn2+) and calcium (Ca2+). We previously reported the ability of some purinergic agonists to exert an anti-migratory activity on tumor-derived human endothelial cells (TEC) only when applied at a high concentration. They also trigger calcium signals associated with release from intracellular stores and calcium entry from the external medium. Here, we provide evidence that high concentrations of BzATP (100 µM), a potent agonist of
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Dissertations / Theses on the topic "Stimolo purinergico"

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FORCAIA, GRETA. "Multifunctional Liposomes modulate Purinergic Receptor-induced Calcium Wave in Cerebral Microvascular Endothelial Cells and Astrocytes." Doctoral thesis, Università degli Studi di Milano-Bicocca, 2020. http://hdl.handle.net/10281/261943.

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I nostri precedenti studi dimostrano che liposomi multifunzionalizzati con il monomero dell’ApoE e con acido fosfatidico (mApoE-PA-LIP) riducono l’accumulo di Aβ nel cervello migliorando il declino cognitivo in modelli murini di malattia di Alzheimer (AD) (Balducci et al., 2014). In riferimento ai nostri precedenti risultati, abbiamo studiato l’interazione di liposomi funzionalizzati con un peptide derivante dall’ApoE (mApoE) e acido fosfatidico (PA), con le cellule che costituiscono l’unità neurovascolare. In particolar modo, abbiamo valutato la loro attività in cellule di microcircolo cereb
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Book chapters on the topic "Stimolo purinergico"

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Engel, Tobias, and Nicholas Dale. "Purinergic Signaling in Epilepsy." In Jasper's Basic Mechanisms of the Epilepsies, 5th ed., edited by Michael A. Rogawski. Oxford University PressNew York, 2024. http://dx.doi.org/10.1093/med/9780197549469.003.0073.

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Abstract Despite the availability of numerous antiseizure drugs (ASDs), treatment of epilepsy remains a clinical challenge, with over 30% of patients not responding to any pharmacological intervention. In addition, ASDs are mainly symptomatic without significantly impacting on disease progression. Thus, there is a strong focus on the identification of drug targets with a novel mechanism of action that are independent on GABAergic and glutamatergic neurotransmission and that possess a disease-modifying potential. That extracellular released purines such as adenosine triphosphate (ATP) and adeno
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Conference papers on the topic "Stimolo purinergico"

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Vanhoutte, Paul M. "PLATELETS, ENDOTHELIUM AND VASOSPASM." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643722.

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The endothelium can secrete both relaxing and contracting substances. One of the most powerful stimuli to the release of the former are thrombin and aggregating platelets. This contributes to the protective role of the endothelium against inappropriate intraluminal platelet aggregation and coagulation in blood vessels with an intact intima. Thrombin-induced, endothelium-dependent relaxations have been obtained in isolated arteries of different species, including humans. Endothelium-dependent relaxations can be evoked by autologous platelets in isolated blood vessels of the dog, pig and rat; th
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Journal articles
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