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Books on the topic 'Stomach disorders'

Author: Grafiati

Published: 28 May 2022

Last updated: 29 May 2022

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1

Lacy, Brian E., John K. DiBaise, Mark Pimentel, and Alexander C. Ford, eds. Essential Medical Disorders of the Stomach and Small Intestine. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-01117-8.

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2

Harvey, Diamond. Living without pain: Stomach & digestive disorders, arthritis, fibromyalgia, lupus, chronic fatigue syndrome and more--. Osprey, Florida: VP Nutrition, 2007.

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3

Gershon, Michael D. The second brain: A groundbreaking new understanding of nervous disorders of the stomach and intestine. New York, NY: HarperPerennial, 1999.

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4

Gershon, Michael D. The second brain: The scientific basis of gut instinct and a groundbreaking new understanding of nervous disorders of the stomach and intestine. New York, NY: HarperCollinsPublishers, 1998.

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5

The second brain: The scientific basis of gut instinct and a groundbreaking new understanding of nervous disorders of the stomach and intestine. New York, NY: HarperCollinsPublishers, 1998.

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6

Chai, Jianyuan, ed. Stomach Disorders. InTech, 2018. http://dx.doi.org/10.5772/66029.

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7

Vries, Jan De. Stomach & Bowel Disorders. Mainstream Publishing, 2004.

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8

Vries, Jan De. Stomach and Bowel Disorders (Well Woman). Mainstream Publishing, 2002.

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9

Stomach and Bowel Disorders (By Appointment Only). Mainstream Publishing, 1993.

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10

Stomach and Bowel Disorders (By Appointment Only). Mainstream Publishing, 1993.

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11

Coping With Ulcers, Heartburn, and Stress-Related Stomach Disorders. Rosen Publishing Group, 1999.

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12

Stomach Ulcers and Acidity: Practical Measures to Help You Avoid and Treat These Painful Stomach Disorders (New Self Help). Thorsons Pub, 1990.

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13

DiBaise, John K., Brian E. Lacy, Mark Pimentel, and Alexander C. Ford. Essential Medical Disorders of the Stomach and Small Intestine: A Clinical Casebook. Springer, 2019.

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14

Gershon, Michael. The Second Brain: A Groundbreaking New Understanding of Nervous Disorders of the Stomach and Intestine. Harper Paperbacks, 1999.

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15

Gershon, Michael. The Second Brain: A Groundbreaking New Understanding of Nervous Disorders of the Stomach and Intestine. Harper Paperbacks, 1999.

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16

Natural Stomach Care: Treating and Preventing Digestive Disorders with the Best of Eastern and Western Healing Therapies. Avery, 2003.

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17

Diane, Gibson, ed. The Evaluation and treatment of eating disorders. New York: Haworth Press, 1986.

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18

Camilleri, Michael. Mayo Clinic Illustrated Textbook of Neurogastroenterology. Oxford University Press, 2021. http://dx.doi.org/10.1093/med/9780197512104.001.0001.

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Mayo Clinic Illustrated Textbook of Neurogastroenterology reflects experience covering the spectrum and maturation of the field of gastrointestinal motility— from studies of exotic patterns of myoelectric recordings in research laboratories to a clinical discipline with major advances in the clinical management of thousands of patients seen by gastroenterologists, primary care physicians, surgeons, or neurologists. Development of the specialty has been characterized by advances in imaging, developments of methods to measure diverse motor and sensory functions from the stomach to the anorectum, introduction of genetic testing, and a plethora of novel pharmacologic and interventional therapies that have revolutionized its practice. The book covers the spectrum of neurogastroenterologic disorders: from those that are associated with genetic and molecular disorders, through disturbances of the extrinsic neural control or the enteric neuromuscular apparatus, to dysfunction associated with disorders of the gut-brain axis; it reflects a desire to pass on clinical and mechanistic insights and advances in therapies that are relevant to a diverse spectrum of clinicians or clinicians in training who care for the estimated 40% of patients presenting to gastroenterologists with symptoms suggestive of disorders of stomach, intestinal, colonic, or anorectal function.

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19

Sedlack, Robert E., Conor G. Loftus, Amy S. Oxentenko, and Thomas R. Viggiano. Gastroenterology. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199755691.003.0210.

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Part 1 reviews the major portions of the gastrointestinal system (esophagus, stomach, small intestine, colon, and pancreas), their function (motility, acid production, enzymatic function, and absorption), and various disorders associated with them (dysmotility, ulceration, malabsorption, inflammation, and dysplasia). Symptoms, diagnostic testing, and treatment of common gastrointestinal conditions, such as gastroesophageal reflux disease, peptic ulcer disease, diarrhea, constipation, inflammatory bowel disease, and pancreatitis, are reviewed.

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20

Lyon, Callum C., and Amanda Smith, eds. Abdominal Stomas and Their Skin Disorders. CRC Press, 2009. http://dx.doi.org/10.3109/9780203443996.

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21

C, Lyon Calum, Smith Amanda J. 1965-, and A.-Malik Rafik, eds. Abdominal stomas and their skin disorders: An atlas of diagnosis and management. London: Dunitz, 2001.

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22

Abdominal Stomas And Their Skin Disorders An Atlas Of Diagnosis And Management. Informa Healthcare, 2009.

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23

Smith, Amanda J., and Calum C. Lyon. Abdominal Stomas and Their Skin Disorders: An Atlas of Diagnosis and Management. Informa Healthcare, 2001.

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24

Shaw, Pamela, and David Hilton-Jones. The lower cranial nerves and dysphagia. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569381.003.0429.

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Disorders affecting the lower cranial nerves – V (trigeminal), VII (facial), IX (glossopharyngeal), X (vagus), XI (accessory) and XII (hypoglossal) – are discussed in the first part of this chapter. The clinical neuroanatomy of each nerve is described in detail, as are disorders – often in the form of lesions – for each nerve.Trigeminal nerve function may be affected by supranuclear, nuclear, or peripheral lesions. Because of the wide anatomical distribution of the components of the trigeminal nerve, complete interruption of both the motor and sensory parts is rarely observed in practice. However, partial involvement of the trigeminal nerve, particularly the sensory component, is relatively common, the main symptoms being numbness and pain. Reactivation of herpes zoster in the trigeminal nerve (shingles) can cause pain and a rash. Trigeminal neuralgia and sensory neuropathy are also discussed.Other disorders of the lower cranial nerves include Bell’s palsy, hemifacial spasm and glossopharyngeal neuralgia. Cavernous sinus, Tolosa–Hunt syndrome, jugular foramen syndrome and polyneuritis cranialis are caused by the involvement of more than one lower cranial nerve.Difficulty in swallowing, or dysphagia, is a common neurological problem and the most important consequences include aspiration and malnutrition (Wiles 1991). The process of swallowing is a complex neuromuscular activity, which allows the safe transport of material from the mouth to the stomach for digestion, without compromising the airway. It involves the synergistic action of at least 32 pairs of muscles and depends on the integrity of sensory and motor pathways of several cranial nerves; V, VII, IX, X, and XII. In neurological practice dysphagia is most often seen in association with other, obvious, neurological problems. Apart from in oculopharyngeal muscular dystrophy, it is relatively rare as a sole presenting symptom although occasionally this is seen in motor neurone disease, myasthenia gravis, and inclusion body myositis. Conversely, in general medical practice, there are many mechanical or structural disorders which may have dysphagia as the presenting feature. In some of the disorders, notably motor neurone disease, both upper and lower motor neurone dysfunction may contribute to the dysphagia. Once dysphagia has been identified as a real or potential problem, the patient should undergo expert evaluation by a clinician and a speech therapist, prior to any attempt at feeding. Videofluoroscopy may be required. If there is any doubt it is best to achieve adequate nutrition through the use of a fine-bore nasogastric tube and to periodically reassess swallowing. Anticholinergic drugs may be helpful to reduce problems with excess saliva and drooling that occur in patients with neurological dysphagia, and a portable suction apparatus may be helpful. Difficulty in clearing secretions from the throat may be helped by the administration of a mucolytic agent such as carbocisteine or provision of a cough assist device.

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25

Koch, Kenneth L., and Robert M. Stern, eds. Handbook of Electrogastrography. Oxford University Press, 2003. http://dx.doi.org/10.1093/oso/9780195147889.001.0001.

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The Handbook of Electrogastrography is the first textbook dedicated to reviewing the physiology of gastric myelectrical activity and the measurement of this electrical activity with electrodes placed on the abdominal surface - the electrogastrogram. The Handbook is divided into three major sections. The first section (Chapters 1-3) focuses on the history of electrogastrography, electrical activity of the interstitial cells of Cajal, the cells from which gastric electrical rhythmicity emanates. The cellular level of gastric electrical rhythmicity provides an understanding of the physiological basis of the electrogastrogram signal. The second major section of the book (Chapters 4-6) incorporates the practical aspects of recording a high quality electrogastrogram and approaches to the analysis of the electrogastrogram using visual inspection and computer techniques. This section focuses on the authors' combined experience of examining EGG recordings for more than sixty years. From this rich research and clinical experience, the clinical application of EGG recordings in an approach to patients with unexplained nausea and vomiting is described. Neuromuscular disorders of the stomach involving gastric dysrhythmias are reviewed. The third major section of the book (Chapters 7-9) comprises many examples of gastric dysrythmias ranging from bradygastrias to tachygastrias and mixed dysrythmias. Current understanding of the mechanisms of gastric dysrhythmias is reviewed. Artifacts in the EGG signal, which may be confused with gastric dysrhythmias, are also presented. The Handbook of Electrogastrography will be a valuable reference for physicians interested in recording gastric electrical activity in clinical practices or in clinical research. Gastroenterologists, internists, psychologists and others with an interest in gastric myoelectrical events will also find extensive and relevant information for recording and interpreting EGGs in the Handbook.

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26

Guts: In “Guts,” a series of events propels Raina, already struggling with the sometimes nerve-racking challenges of navigating fourth grade, into a full-blown anxiety disorder, which begins to take over her life. London, England: Raina Telgemeier, 2019.

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27

White, Maddie. Colorectal and lower gastrointestinal surgery. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199642663.003.0019.

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The lower gastrointestinal tract includes most of the small intestine and all of the large intestine. The patient presenting with an acute abdomen may have a bowel obstruction which could lead to ischaemia and perforation. Thorough assessment and close observation are imperative, because, if surgical intervention is indicated, this needs to be performed promptly. This chapter provides an overview of inflammatory bowel disorders, tumours, hernias, obstructions, the acute abdomen, ischaemic bowel, complications of bowel surgery, and stomas.

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28

Wójcik-Gładysz, Anna. Ghrelin – hormone with many faces. Central regulation and therapy. The Kielanowski Institute of Animal Physiology and Nutrition, Polish Academy of Sciences, 2020. http://dx.doi.org/10.22358/mono_awg_2020.

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Discovered in 1999, ghrelin, is one of the peptides co-creating the hypothalamicgastrointestinal axis, otherwise known as the brain-gut axis. Ghrelin participates in many physiological processes and spectrum of its activity is still being discovered. This 28 amino acid peptide ‒ a product of the ghrl gene, was found in all vertebrates and is synthesized and secreted mainly from enteroendocrine X/A cells located in the gastric mucosa of the stomach. Expression of the ghrelin receptor has been found in many nuclei of the hypothalamus involved in appetite regulation. Therefore it’s presumed that ghrelin is one of the crucial hormones deciphering the energy status required for the maintenance of organism homeostasis. Ghrelin acts as a signal of starvation or energy insufficiency and its level in plasma is reduced after the meal. Neuropeptide Y (NPY) and agouti-related peptide (AgRP; NPY/AgRP) neurons located in the arcuate nucleus (ARC) area are the main target of ghrelin in the hypothalamus. This subpopulation of neurons is indispensable for inducing orexigenic action of ghrelin. Moreover ghrelin acting as a neurohormone, mainly in the hypothalamus area, plays an important role in the regulation of growth and reproduction processes. Indeed, ghrelin action on reproductive processes has been observed in the systemic effects exerted at both hypothalamus-pituitary and gonadal levels. Similarly the GH-releasing ghrelin action was observed both on the hypothalamus level and directly on the somatotrophic cells in the pituitary and this dose-related GH releasing activity was found in in vitro as well as in in vivo experiments. In recent years, numerous studies revealed that ghrelin potentially takes part in the treatment of diseases associated with serious disturbances in the organism energy balance and/or functioning of the gastrointestinal tract. It was underlined that ghrelin may be a hormone with a broad spectrum of therapeutic effect on obesity and anorexia nervosa, as well as may also have protective effect on neurodegenerative diseases, inflammatory disorders or functional changes in the body caused by cancers. In overall, ghrelin treatment has been tested in over 100 preclinical studies with healthy volunteers as well as patients with various types of cancer, eating disorders such as anorexia nervosa and bulimia nervosa. It was observed that ghrelin has an excellent clinical safety profile and emerging side effects occurred only in 3–10% of patients and did not constitute a sufficient premise to discontinue the therapy. In general, it can be concluded that ghrelin may be sufficiently used as a prescription drug.

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