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Relevant bibliographies by topics / Streptococal infections

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Academic literature on the topic 'Streptococal infections'

Author: Grafiati

Published: 4 June 2021

Last updated: 5 February 2022

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Journal articles on the topic "Streptococal infections"

1

Green, K., A. McGreer, B. Schwartz, D. Cann, P. Wilson, and D. E. Low. "Prospective surveillance for nosocomial group a streptococal infections in Ontario: Do single cases warrant investigation?" American Journal of Infection Control 22, no. 2 (April 1994): 110. http://dx.doi.org/10.1016/0196-6553(94)90165-1.

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Santos, Joana Eugénio, Catuxa Rodríguez Magariños, Leticia García Gago, Daniela Astudillo Jarrín, Sonia Pértega, Ana Rodríguez-Carmona, Teresa García Falcón, and Miguel Pérez Fontán. "Long-term trends in the incidence of peritoneal dialysis-related peritonitis disclose an increasing relevance of streptococcal infections: A longitudinal study." PLOS ONE 15, no. 12 (December 21, 2020): e0244283. http://dx.doi.org/10.1371/journal.pone.0244283.

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Background The selective impact of strategies for prevention of PD-related peritonitis (PDrP) may have modified, in the long term, the causal spectrum, clinical presentation and outcomes of these infections. Objectives To compare trends in the incidence of PDrP by different microorganisms during a 30-year period, with a particular focus on streptococcal infections. To analyze the clinical presentation and outcomes of these infections. Secondarily, to investigate how the isolation of different species of streptococci can influence the clinical course of PDrP by this genus of bacteria. Method Following a retrospective, observational design we investigated 1061 PDrP (1990–2019). We used joinpoint regression analysis to explore trends in the incidence of PDrP by different microorganisms, and compared the risk profile (Cox), clinical presentation and outcomes (logistic regression) of these infections. Main results Our data showed a progressive decline in the incidence of PDrP by staphylococci and Gram negative bacteria, while the absolute rates of streptococcal (average annual percent change +1.6%, 95% CI -0.1/+3.2) and polymicrobial (+1.8%, +0.1/+3.5) infections tended to increase, during the same period. Remarkably, streptococci were isolated in 58.6% of polymicrobial infections, and patients who suffered a streptococcal PDrP had a 35.8% chance of presenting at least one other infection by the same genus. The risk profile for streptococcal infections was comparable to that observed for PDrP overall. Streptococcal PDrP were associated with a severe initial inflammatory response, but their clinical course was generally nonaggressive thereafter. We did not observe a differential effect of different groups of streptococci on the clinical presentation or outcome of PDrP. Conclusions Time trends in the incidence of PDrP by different microorganisms have granted streptococci an increasing relevance as causative agents of these infections, during the last three decades. This behaviour suggests that current measures of prevention of PDrP may not be sufficiently effective, in the case of this genus of microorganisms.

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Numberger, Daniela, Ursula Siebert, Marcus Fulde, and Peter Valentin-Weigand. "Streptococcal Infections in Marine Mammals." Microorganisms 9, no. 2 (February 10, 2021): 350. http://dx.doi.org/10.3390/microorganisms9020350.

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Marine mammals are sentinels for the marine ecosystem and threatened by numerous factors including infectious diseases. One of the most frequently isolated bacteria are beta-hemolytic streptococci. However, knowledge on ecology and epidemiology of streptococcal species in marine mammals is very limited. This review summarizes published reports on streptococcal species, which have been detected in marine mammals. Furthermore, we discuss streptococcal transmission between and adaptation to their marine mammalian hosts. We conclude that streptococci colonize and/or infect marine mammals very frequently, but in many cases, streptococci isolated from marine mammals have not been further identified. How these bacteria disseminate and adapt to their specific niches can only be speculated due to the lack of respective research. Considering the relevance of pathogenic streptococci for marine mammals as part of the marine ecosystem, it seems that they have been neglected and should receive scientific interest in the future.

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Liang, Guowei. "Aryl hydrocarbon receptor protects against viridans streptococci infection by activation of immune system through IL-17RA signaling." American Journal of BioMedicine 3, no. 2 (May 19, 2015): 400–410. http://dx.doi.org/10.18081/2333-5106/015-02/400-410.

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The majority of bacterial infections during neutropenia following high-dose chemotherapy or stem cell transplantation are caused by coagulase-negative staphylococci, a large number are due to viridans streptococci. Despite considerable progress in the understanding of the AhR-mediated regulation of immune responses, the role of AhR in bacterial infections has not been clearly demonstrated. In the study presented here, we sought to determine whether the aryl hydrocarbon receptor (AhR) would protect mice from infection with viridans streptococci. AhR enhances the inflammatory response to viridans streptococci stimuli. Specifically, neutrophil numbers and levels of inflammatory cytokines are often increased in mice treated with viridans streptococci. Furthermore, AhR activation through the IL-17RA is required for protection against viridans streptococcal infection. Taken together, we concluded that AhR plays an important role in optimal innate immunoprotection against microbial infection through the down-regulation of immune response.

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Huang, Allen R., and Dalius J. Briedis. "Group C Streptococcal Endocarditis Presenting as Clinical Meningitis: Report of a Case and Review of the Literature." Canadian Journal of Infectious Diseases 3, no. 5 (1992): 247–52. http://dx.doi.org/10.1155/1992/597941.

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Lancefield group C streptococci are known to be pathogenic in a number of animal species, but cause human disease much less commonly than do streptococci of scrogroups A or B. Reported cases of bacteremic infection, pneumonia or meningitis in humans have been very severe with a grave prognosis. The authors describe a patient who presented with classic clinical and laboratory evidence of bacterial meningitis which proved to be a complication of endocarditis caused by a group C streptococcus. This is the first reported case in which meningitis was the presenting manifestation of group C streptococcal endocarditis and is only the second case in which group C streptococcal meningitis and endocarditis have been associated in the same patient. A total of 13 cases of group C streptococcal meningitis have now been reported in the medical literature. Five of these patients died, and four others recovered only to be left with neurological sequelae. The current case confirms the seriousness of group C streptococcal infections in humans. Such infections are associated with a poor prognosis despite apparently adequate antimicrobial therapy.

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RECCO, R. A., M. M. ZAMAN, H. CORTES, J. COLUCCI, G. POOMKUDY, and E. L. KAPLAN. "Intra-familial transmission of life-threatening group A streptococcal infection." Epidemiology and Infection 129, no. 2 (October 2002): 303–6. http://dx.doi.org/10.1017/s0950268802007343.

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Invasive group A streptococcal (GAS) infections have been of increasing concern worldwide during the past 15 years. Spread of group A streptococci to contacts with resulting invasive infection has been reported in families, in residential nursing homes, and even from patients to health care workers. We report an instance of temporally related life-threatening group A streptococcal infection in a husband and 2 weeks later in his wife. This example further emphasizes the need for careful observation among family members and other close contacts of patients with invasive group A streptococcal infection. Although at present there are no universal recommendations for monitoring or for antibiotic prophylaxis of close contacts of persons with invasive GAS infection, when added to existing literature, this report suggests additional consideration is required.

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Fluckiger, U., K. F. Jones, and V. A. Fischetti. "Immunoglobulins to Group A Streptococcal Surface Molecules Decrease Adherence to and Invasion of Human Pharyngeal Cells." Infection and Immunity 66, no. 3 (March 1, 1998): 974–79. http://dx.doi.org/10.1128/iai.66.3.974-979.1998.

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ABSTRACT The M protein is one of the most important virulence factors of group A streptococci (Streptococcus pyogenes) and may play an important role in the first steps of streptococcal infection. Since acute pharyngitis is a frequently occurring infectious disease caused by these bacteria, we wished to know whether antibodies to the M protein or other surface components inhibit adherence and internalization of streptococci to pharyngeal cells. We investigated the role of whole human secretory immunoglobulin A (sIgA), M6 protein-specific sIgA, and M6 protein-specific serum IgG in the inhibition of streptococcal adherence and internalization to cultured human pharyngeal cells. S. pyogenes D471, which produces a type 6 M protein (M+), and its isogenic M-negative (M−) derivative JRS75 were tested. Purified whole sIgA, M protein-specific sIgA, and sIgA preabsorbed with M protein were able to decrease significantly the adherence of streptococci to pharyngeal cells. Purified IgG against the M6 protein did not diminish the attachment of streptococci to the pharyngeal cells but did reduce internalization. Thus, our data suggest that secretory IgA may play a key role in preventing streptococcal infection at mucosal surfaces by blocking adherence while affinity-purified anti-M protein-specific IgG blocks epitopes responsible for invasion.

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Fujimori, Isao, Izuru Nozawa, Kazuhito Kikushima, Rei Goto, Ken-Ichi Hisamatsu, and Yoshihiko Murakami. "Interaction between Oral Alpha-Streptococci and Group a Streptococci in Patients with Tonsillitis." Annals of Otology, Rhinology & Laryngology 106, no. 7 (July 1997): 571–74. http://dx.doi.org/10.1177/000348949710600708.

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The incidence of oral α-streptococci with inhibitory activity against group A streptococci, as a defense mechanism against bacterial infection in the oral cavity, was investigated in 141 patients with streptococcal tonsillitis. The study population included both children (n = 79) and adults (n = 62). Infection by group A streptococci appeared to be more common in children than in adults, as the detection rates of inhibitory α-streptococci in healthy children (29.7%), as well as pediatric patients with tonsillitis (14.9%), were lower than those in adults (63.0%; p < .01). It is possible to consider oral α-streptococci with inhibitory activity to be among the indications for tonsillectomy in patients with streptococcal tonsillitis, since the detection rate of inhibitory α-streptococci in surgical cases (10.9%) was significantly lower than that in nonsurgical cases (31.1 %; p < .01). The high detection rate of these strains during the postoperative state supported the observation that the incidence of group A streptococcal infection was decreased postoperatively. Accordingly, it is useful to investigate bacterial interference between oral α-streptococci and group A streptococci in patients scheduled for tonsillectomy.

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Higgins, P. M. "Streptococcal pharyngitis in general practice. 1. Some unusual features of the epidemiology." Epidemiology and Infection 109, no. 2 (October 1992): 181–89. http://dx.doi.org/10.1017/s0950268800050147.

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SUMMARYThis report is based on a study of acute infections of the upper respiratory tract in 1965 and detailed records of such infections in 1963 and 1964. A change from illnesses mainly yielding viruses to illnesses mainly yielding group A streptococci was noted around the age of 5 years. A positive culture for group A streptococci in patients over 4 years of age was highly correlated with a complaint of sore throat and with serological evidence of streptococcal infection. A bimodal age distribution curve for pharyngitis associated with a positive culture for group A streptococci was consistently noted. The incidence was highest in children aged 5–9 but a second smaller peak occurred among adults in the 30–39 age group. The evidence suggests that being female increases the risk of acquiring group A streptococci and of experiencing sore throat.

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Liao, Pei-Chih, Yi-Lun Tsai, Yao-Chung Chen, Pei-Chi Wang, Shu-Chu Liu, and Shih-Chu Chen. "Analysis of Streptococcal Infection and Correlation with Climatic Factors in Cultured Tilapia Oreochromis spp. in Taiwan." Applied Sciences 10, no. 11 (June 10, 2020): 4018. http://dx.doi.org/10.3390/app10114018.

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Tilapia (Oreochromis spp.), a prominent warm water food fish, is one of the major fish species grown in the aquaculture industry in south-east Asia. Tilapia can tolerate adverse water quality and other stressors, like diverse salinity and fluctuation of pH value, better than most other commercial aquaculture species. Environmental fluctuations are one of the main factors that affect the outbreak of infectious diseases in cultured tilapia. Cultured tilapia in Taiwan appears to be more susceptible to infections caused by Streptococci during the summer season. The present study emphasizes the Streptococcus spp. infection in tilapia in Taiwan and is the first study on the analysis of the potential impact of climate change on streptococcal infection in cultured tilapia in Asia. The data collected from the treatment and diagnosis system (TDS) of the aquatic animal diseases database from 2006 to 2015 were used to analyze the endemic streptococcal infection and the effect of climatic factors. Based on the results, the factor, average atmospheric pressure, is negatively correlated to streptococcal infection, while the other three, including average temperature, ultraviolet (UV) index, and rainfall, are positively correlated to streptococcal infection. A multivariate logistic regression model with these four factors was also built. When the average temperature is above 27.0 °C, the average atmospheric pressure is lower than 1005.1 hPa, or the UV index is above 7.2, the percentage of cumulated positive farms from all submitted tilapia cases was more than 50%. In addition, within 3 days of rain, rainfall is relevant to the occurrence of Streptococcus in tilapia. Using TDS to alert the occurrence of streptococcal infection in tilapia can be a very useful tool for veterinary aquatic animal inspection stations, and reducing economic losses and labour costs in aquatic agriculture.

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Dissertations / Theses on the topic "Streptococal infections"

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Westling, Katarina. "Viridans group streptococci septicaemia and endocarditis : molecular diagnostics, antibiotic susceptibility and clinical aspects /." Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-364-7/.

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Unnikrishnan, Meera. "Streptococcal superantigens in Group A streptococcal infections." Thesis, Imperial College London, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.248185.

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Flock, Margareta. "Development of a vaccine against strangles /." Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-500-3/.

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Smith, Jennifer Marie. "Characterization of host-bacteria interactions contributing to group B streptococcus colonization." Huntington, WV : [Marshall University Libraries], 2002. http://www.marshall.edu/etd/descript.asp?ref=64.

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Wright, Lynda J. "Identification and characterisation of components expressed by gram-positive bacterial pathogens during human infection." Thesis, University of Sheffield, 2008. http://etheses.whiterose.ac.uk/10312/.

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Gram-positive pathogens are responsible for a wide range of global diseases, including nosocomial infections. The increasing incidence of antibiotic-resistant strains warrants the development of novel therapeutic strategies to combat these organisms.

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Ihendyane, Nahla. "Pathogenesis and immunotherapy of streptococcal septicemia and shock /." Stockholm, 2003. http://diss.kib.ki.se/2003/91-7349-599-9/.

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Darenberg, Jessica. "Streptococcus pyogenes infections and toxic shock syndrome : molecular epidemiology and immunotherapy /." Stockholm, 2006. http://diss.kib.ki.se/2006/91-7140-676-X/.

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Leung, Chin-pang, and 梁展鵬. "Characterization of group a streptococcus in Hong Kong." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1999. http://hub.hku.hk/bib/B3196963X.

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Stofile, P. Z. "Prevalence of Group B streptococcus and staphylococcus aureus colonization in the anogenital tract of pregnant women in the Eastern Cape Province, South Africa." Thesis, University of Fort Hare, 2017. http://hdl.handle.net/10353/5983.

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Neonatal sickness and death is increasingly becoming a public health problem worldwide. The colonization of Group B Streptococcus and Staphylococcus in the rectovaginal area is among the sources of infections in neonates which can result in illness and mortality. The over exposure of humans to antibiotics is the possible cause of resistance in bacteria. These resistant strains can be passed onto offspring, leading to resistant infections and increasing the morbidity of neonates because of treatment failures. Many people, including healthcare personnel are not aware of the effect of these bacteria, and informing clinics and hospitals can help create awareness and monitoring the levels of resistance among bacteria can assist in preventing the transference of the bacteria. In this study we investigated the prevalence of group B Streptococcus (GBS) and Staphylococcus aureus in the anogenital tract of pregnant women in the Eastern Cape Province, South Africa. A total of 49 isolates from 25 (30.5 percent) pregnant women colonized with GBS were isolated from vaginal and rectal swabs of 82 pregnant women at 25-37 gestation who participated in this study. These isolates were obtained using standard microbiological methods and confirmed by polymerase chain reaction (PCR) technique aimed at the ScpB gene. The isolates were further screened for the presence of 9 serogroups (Ia, Ib, II, III, IV, V, VI, VII, VII) and serogroups Ib 2 (4.8 percent), II 20 (40.8 percent) and IV 5 (10.2 percent) and 22 non-typable (44.9 percent) were identified. Susceptibility profiling of the isolates to 12 antibiotics (tetracycline, clindamycin, erythromycin, gentamycin, naladixic acid, norfloxacin, chloramphenicol, cefuroxime, cefotaxime, imipenem, penicillin and vancomycin) was tested in vitro by the standardized disc diffusion method. All the confirmed GBS isolates (49) were resistant to erythromycin, tetracycline and clindamycin. A higher percentage of the isolates were resistant to gentamycin 44 (90 percent), nalidixic acid 41 (84 percent), penicillin 41 (84 percent), chloramphenicol 38 (78 percent), cefuroxime 36 (74 percent), imipenem 36 (74 percent), cefotaxime 35 (71 percent), norfloxacin 32 (65 percent) and vancomycin 31 (78 percent). Multiple antimicrobial resistance patterns ranged from 9‒11 and indices ranged from 0.7‒0.9, respectively. Among the antimicrobial resistance determinants examined, genes encoding for resistance to erythromycin ermB 25 (51 percent), tetracycline tetM 32 (65 percent) and penicillin bla-Z 4 (8 percent) only were identified. On the other hand, screening for S. aureus yielded a total of 7 isolates from 4 study participants as confirmed by PCR based on staphylococcal, nuc gene. The isolates were further screened for the presence of six virulence genes (Hla, Hlb, LUKM, LUKED, PVL, Eta and Etb) and antibiotic susceptibility pattern by the disc diffusion method using 12 (penicillin, vancomycin, tetracycline, rifampicin, imipenem, gentamycin, chloramphenicol, norfloxacin, oxacillin, erythromycin and sulfamethoxazole-trimethoprim) antibiotics that are adopted in the treatment of infections caused by the organism. PVL 6 (85.7 percent) and eta 1 (14.3 percent) were the two virulence genes detected. The following percentages of antibiotics resistance among the isolates were observed; penicillin G 7 (100 percent), clindamycin 7 (100 percent), vancomycin 5 (100 percent), rifampicin 5 (71 percent), oxacillin 5 (71 percent), erythromycin 5 (71 percent) gentamycin 3 (43 percent), norfloxacin 3 (43 percent), sulfamethoxazole-trimethoprim 3 (43 percent), chloramphenicol 2 (29 percent), imipenem 1 (14 percent). Multiple antimicrobial resistance patterns ranged from 7‒8 and indices ranged from 0.6‒0.7, respectively. Genetic profiling of the resistance genes identified erythromycin ermB 5(71.4 percent), tetracycline tetM 5(71.4 percent) and penicillin bla-Z 1(14.3 percent) only. The findings from the study have revealed GBS and S. aureus colonization of pregnant women in the Eastern Cape Province, and these have great public health implications especially for the neonates who are mostly likely to be infected during birth. The unidentifiable multidrug resistant serogroups of GBS as well as resistant S. aureus limit the choice of drugs in the management of infections caused by these pathogens more so if transmitted to infants. Therefore asymptomatic pregnant women needed to be properly educated about the bacteria as well as the precautions that need to be taken.

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Eriksson, Björn K. G. "Invasive group A streptococcal infection : host and pathogen interactions /." Stockholm, 1999. http://diss.kib.ki.se/1999/91-628-3609-9/.

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Books on the topic "Streptococal infections"

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Lancefield International Symposium on streptococci and Streptococcal Diseases (9th 1984 Yamanakako-mura, Japan). Recent advances in streptococci and streptococcal diseases: Proceedings of the IXth Lancefield International Symposium on Streptococci and Streptococcal Diseases held in September 1984. Bracknell, Berkshire: Reedbooks, 1985.

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Lancefield, International Symposium on Streptococci and Streptococcal Diseases (11th 1990 Siena Italy). New perspectives on streptococci and streptococcal infections: Proceedings of the XI Lancefield International Symposium on Streptococci and Streptococcal Diseases, Siena, September 10-14, 1990. Stuttgart: G. Fischer, 1992.

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Illinois. Department of Public Health. Streptococcal pharyngitis: (strep throat). Springfield, Ill.]: Illinois Dept. of Public Health, 1991.

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Smith, Tara C. Streptococcus (group A). Edited by Alcamo I. Edward and Heymann David L. Philadelphia: Chelsea House Publishers, 2005.

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Verani, Jennifer R. Prevention of perinatal group B streptococcal disease: Revised guidelines from CDC, 2010. Atlanta, GA: Dept. of Health and Human Services, Centers for Disease Control and Prevention, 2010.

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Lancefield, International Symposium on Streptococci and Streptococcal Diseases (16th 2005 Palm Cove Australia). Streptococci: New insights into an old enemy. Amsterdam: Elsevier, 2006.

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Parker, James N., and Philip M. Parker. The official patient's sourcebook on group A streptococcus infection. Edited by Icon Group International Inc and NetLibrary Inc. San Diego, Calif: Icon Health Publications, 2002.

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Hilary, Babcock, ed. Streptococcus (group A). 2nd ed. New York, NY: Chelsea House, 2010.

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Rheumatic fever and streptococcal infection: Unraveling the mysteries of a dread disease. Boston: Francis A. Countway Library of Medicine, 1997.

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Landau, Elaine. Strep throat. New York: Marshall Cavendish Benchmark, 2010.

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Book chapters on the topic "Streptococal infections"

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Ryan, Raymond W. "Streptococcal Infections: Alpha-Hemolytic Streptococci." In Laboratory Diagnosis of Infectious Diseases, 483–89. New York, NY: Springer New York, 1988. http://dx.doi.org/10.1007/978-1-4612-3898-0_50.

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Gray, Barry M. "Streptococcal Infections." In Bacterial Infections of Humans, 673–711. Boston, MA: Springer US, 1998. http://dx.doi.org/10.1007/978-1-4615-5327-4_35.

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Gray, Barry M., and Dennis L. Stevens. "Streptococcal Infections." In Bacterial Infections of Humans, 743–82. Boston, MA: Springer US, 2009. http://dx.doi.org/10.1007/978-0-387-09843-2_35.

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Faro, Sebastian. "Streptococcal Infections." In Principles of Medical Therapy in Pregnancy, 441–46. Boston, MA: Springer US, 1985. http://dx.doi.org/10.1007/978-1-4613-2415-7_52.

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Gray, Barry M. "Streptococcal Infections." In Bacterial Infections of Humans, 639–73. Boston, MA: Springer US, 1991. http://dx.doi.org/10.1007/978-1-4757-1211-7_31.

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Mustafa, Mahmoud M. "Streptococcal Infections." In Textbook of Clinical Pediatrics, 1045–50. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-02202-9_93.

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McGee, Lesley, and Bernard Beall. "Streptococci." In Molecular Typing in Bacterial Infections, 109–26. Totowa, NJ: Humana Press, 2012. http://dx.doi.org/10.1007/978-1-62703-185-1_8.

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Cavaillon, Jean-Marc, Heide Müller-Alouf, and Joseph E. Alouf. "Cytokines in Streptococcal Infections." In Streptococci and the Host, 869–79. Boston, MA: Springer US, 1997. http://dx.doi.org/10.1007/978-1-4899-1825-3_206.

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Gerber, Michael A. "Group A Streptococcal Infections." In Laboratory Diagnosis of Infectious Diseases, 294–301. New York, NY: Springer New York, 1988. http://dx.doi.org/10.1007/978-1-4612-3898-0_30.

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Good, M. F., K. S. Sriprakash, and D. J. Kemp. "Vaccine Control Strategies against Group A Streptococcal Infections." In Streptococcal Pharyngitis, 202–14. Basel: KARGER, 2003. http://dx.doi.org/10.1159/000076207.

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Conference papers on the topic "Streptococal infections"

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Regaieg, Chiraz, Manel Charfi, Nour Houda Ben Ayed, Amel Ben Hmed, Ridha Regaieg, Nedia Hmida, Adnen Hamammi, Afef Ben Thabet, and Abdellatif Gargouri. "P453 Neonatal streptococcal B infections." In Faculty of Paediatrics of the Royal College of Physicians of Ireland, 9th Europaediatrics Congress, 13–15 June, Dublin, Ireland 2019. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2019. http://dx.doi.org/10.1136/archdischild-2019-epa.789.

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Tsaprouni, Triantafyllia, Vasiliki Melikoki, and Evridiki Vouloumanou. "P396 Kawasaki disease and group a streptococcal infection: a case report." In Faculty of Paediatrics of the Royal College of Physicians of Ireland, 9th Europaediatrics Congress, 13–15 June, Dublin, Ireland 2019. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2019. http://dx.doi.org/10.1136/archdischild-2019-epa.742.

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Li, F., and H. Zeng. "AB0560 Study on streptococcal infection relationship with henoch-schonlein purpura in children." In Annual European Congress of Rheumatology, 14–17 June, 2017. BMJ Publishing Group Ltd and European League Against Rheumatism, 2017. http://dx.doi.org/10.1136/annrheumdis-2017-eular.2136.

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PAPKE, AFONSO, BRUNA DE LIMA PORTO, MICHELINE SULZBACHER BATISTA, ANA LAURA FISCHER KUNZLER, MAURICIO SIMONI CANDATEN, VICTORIA SILVEIRA DE CARVALHO, and SANDRA HELENA MACHADO. "CUTANEOUS POLYARTERITIS NODOSA IN A 6-YEAR-OLD CHILD WITH DOCUMENTED STREPTOCOCCAL INFECTION: CASE REPORT." In 36º Congresso Brasileiro de Reumatologia. São Paulo: Editora Blucher, 2019. http://dx.doi.org/10.5151/sbr2019-070.

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Buiting, A. G. M., J. Thomson, J. J. Emeis, H. Mattie, E. J. P. Brommer, and R. Van Furth. "EFFECT OF TISSUE-TYPE PLASMINOGEN ACTIVATOR (t-PA) ON BACTERIAL ENDOCARDITIS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643742.

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In bacterial endocarditis the causing microorganisms are located in a fibrin-platelet matrix, making them less accessible to host-defence mechanisms and antibiotic therapy. Trombolytic treatment could break down the fibrin of these endocardial vegetations and thus eliminate the focus of infection. This approach was studied in vitroand in vivo using recombinant t-PA (rt-PA,Wellcome Biotech) having a fibrinolytic activity comparable with melanoma t-PA. The following results were obtained.1.Incubation of Streptococcus sanguis infected plasma clots in the presence of t-PA resulted in lysis of the clots as evidenced by a significant decrease in the weight of the clots and by an increase in the number of streptococci in the medium.No effect of t-PA was found on the antimicrobial action of penicillin G (PenG)onthe streptococci in the non-lysed part ofthe clots.2. Vegetations isolated from the heart of rabbits with a S. sanguis endocarditis,incubated in medium with t-PA were also lysed. This resulted in a rise in the mediumof both the number of bacteria and the concentration of fibrin degradation products.3.Treatment of rabbits with a S. sanguis endocarditis using a combination of rt-PA, given as bolus injections at one hour intervals (4x0.5 or 4x1 mg per kg/day), andPenG decreased the weight of the endocardial vegetations significantly compared toa control group treated with PenGonly (43.3 and 81.8 mg respectively). No additional decrease in weight was obtained with repeated administration of t-PA at daily intervals up to 2 days. The significant decrease of the number of streptococci per gram of vegetation as a result of the treatmentwith PenG was not influenced by t-PA. Treatment with 4x1 mg t-PA per kg caused a small decrease (about 30%) in the plasma concentration of plasminogen, fibrinogen and aα2~antiplasmin.However, no bleeding complications were observed.In conclusion rt-PA can influence the treatment of bacterial endocarditis in rabbits by decreasing the size of the vegetations but not by influencing antimicrobial action on the bacteria in the non-lysed part of the vegetation.

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Bashir Hamidu, R., D. Cheney-Peters, and C. McGrath. "More Than Strep Throat: A Case of Explosive Pleuritis--Rapidly Progressive Pleural Effusions Secondary to a Group A Streptococcal Infection." In American Thoracic Society 2021 International Conference, May 14-19, 2021 - San Diego, CA. American Thoracic Society, 2021. http://dx.doi.org/10.1164/ajrccm-conference.2021.203.1_meetingabstracts.a3971.

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Reports on the topic "Streptococal infections"

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Evidence Update for Clinicians: Narrow- versus Broad-Spectrum Antibiotics for Common Infections in Children. Patient-Centered Outcomes Research Institute (PCORI), October 2018. http://dx.doi.org/10.25302/eu5.2018.10.

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Comparing Narrow- vs. Broad-Spectrum Antibiotics for Common Infections in Children. The choice of antibiotic to treat acute bacterial upper respiratory tract infections in children can affect both symptom resolution and the risk of side effects such as diarrhea and vomiting. The findings of a PCORI-funded study published in JAMA can help clinicians treating children for acute respiratory tract infections (ARTIs)—including acute otitis media, Group A streptococcal pharyngitis, and acute sinusitis—make decisions with parents about the medicine that is best for the child. The study, led by Jeffrey Gerber, a pediatrician and researcher at the Children’s Hospital of Philadelphia, included 30,086 children ages 6 months to 12 years taking narrow- and broad-spectrum antibiotics to treat ARTIs.

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