Academic literature on the topic 'Streptococcus agalactiae mic alterations'

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Journal articles on the topic "Streptococcus agalactiae mic alterations"

1

Haenni, Marisa, Laure Galofaro, Mathilde Ythier, et al. "Penicillin-Binding Protein Gene Alterations in Streptococcus uberis Isolates Presenting Decreased Susceptibility to Penicillin." Antimicrobial Agents and Chemotherapy 54, no. 3 (2010): 1140–45. http://dx.doi.org/10.1128/aac.00915-09.

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ABSTRACT Streptococcus uberis is an environmental pathogen commonly causing bovine mastitis, an infection that is generally treated with penicillin G. No field case of true penicillin-resistant S. uberis (MIC > 16 mg/liter) has been described yet, but isolates presenting decreased susceptibility (MIC of 0.25 to 0.5 mg/liter) to this drug are regularly reported to our laboratory. In this study, we demonstrated that S. uberis can readily develop penicillin resistance in laboratory-evolved mutants. The molecular mechanism of resistance (acquisition of mutations in penicillin-binding protein 1A
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Trivedi, Mahendra Kumar, Alice Branton, Dahryn Trivedi, et al. "Effect of Biofield Energy Treatment on Streptococcus group B: A Postpartum Pathogen." Journal of Microbial & Biochemical Technology 7, no. 5 (2015): 269–73. https://doi.org/10.4172/1948-5948.1000223.

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Streptococcus agalactiae group B (S. agalactiae gr. B) is widespread in nature mainly causes bacterial septicemia and neonatal meningitis. The current study was attempted to investigate the effect of biofield treatment on S. agalactiae gr. B with respect of antimicrobial sensitivity, biochemical reactions and bio typing. S. agalactiae gr. B strain was used in this experiment bearing the American Type Culture Collection (ATCC 12386) number and stored according to the recommended storage protocol. The revived and lyophilized state of ATCC strains of S. agalactiae gr. B were selected for the stud
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Trivedi, Mahendra Kumar, Alice Branton, Dahryn Trivedi, et al. "Effect of Biofield Energy Treatment on Streptococcus group B: A Postpartum Pathogen." Journal of Microbial & Biochemical Technology 7, no. 5 (2015): 269–73. https://doi.org/10.5281/zenodo.167155.

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Streptococcus agalactiae group B (S. agalactiae gr. B) is widespread in nature mainly causes bacterial septicemia and neonatal meningitis. The current study was attempted to investigate the effect of biofield treatment on S. agalactiae gr. B with respect of antimicrobial sensitivity, biochemical reactions and bio typing. S. agalactiae gr. B strain was used in this experiment bearing the American Type Culture Collection (ATCC 12386) number and stored according to the recommended storage protocol. The revived and lyophilized state of ATCC strains of S. agalactiae gr. B were selected for the stud
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4

Oliveira, Pâmella Schramm, Aline Rossato, Larissa da Silva Silveira, et al. "GRAPHENE OXIDE AND REDUCED GRAPHENE OXIDE." International Journal for Innovation Education and Research 9, no. 12 (2021): 142–69. http://dx.doi.org/10.31686/ijier.vol9.iss12.3572.

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To present a possible new alternative for wound treatment, this work evaluated the biological safety and therapeutic efficacy of graphene oxide (GO) and reduced graphene oxide (rGO) nanoparticles (NPs). First, the nanostructures were studied in silico and showed to be able to inhibit the production of some pro-inflammatory cytokines and stimulate the production of the anti-inflammatory cytokine IL-10, especially rGO. The results of the morphological and structural characterization of GO NPs synthesized from the Hummers method and reduced by ascorbic acid, were consistent with the literature, c
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5

Faccone, Diego, Florencia Lalonardi, Sofia Abel, et al. "Multiple-Clones of Streptococcus agalactiae harbouring lnuB gene." Journal of Infection in Developing Countries 4, no. 09 (2010): 580–82. http://dx.doi.org/10.3855/jidc.941.

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Six S. agalactiae displaying an L-phenotype were recovered from recto-vaginal screening culture from 3 hospitals. S. agalactiae isolates showed susceptible MICs to erythromycin, MIC≤0.12mg/L, and azithromycin, MIC≤0.25mg/L, but resistant MICs to clindamycin, MIC=4mg/L, and lincomycin, MIC=64-128mg/L. Isolates were PCR positive for lnuB gene. Four clones were discriminated by ApaI-PFGE (n): A (3), B (1), C (1) and D (1). Here we describe for the first time the polyclonal emergence of S. agalactiae harboring the lnuB gene in Latin-America.
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Han, Long, Wen Zheng, Zhangjiang He, Shengyan Qian, Xiaoya Ma, and Jichuan Kang. "Endophytic fungus Biscogniauxia petrensis produces antibacterial substances." PeerJ 11 (June 7, 2023): e15461. http://dx.doi.org/10.7717/peerj.15461.

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Widespread drug resistance and limited antibiotics challenge the treatment of pathogenic bacteria, which leads to a focus on searching for new antimicrobial lead compounds. We found the endophytic fungus Biscogniauxia petrensis MFLUCC14-0151 from the medicinal plant Dendrobium harveyanum had antibacterial activity for the first time. This work aimed to reveal the capacity of Biscogniauxia petrensis MFLUCC14-0151 against foodborne pathogenic bacteria and identify its bioactive substances. Bioassay-guided isolation led to the discovery of six infrequent active monomers, including (10R)-Xylariter
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7

Syawal, Henni, Rahman Karnila, Angraika Dirta, and Ronal Kurniawan. "Ekstrak Daun Rhizophora sp. Menghambat Pertumbuhan Bakteri Streptococcus agalactiae dan Edwarsiella tarda (RHIZOPHORA SP. LEAF EXTRACT INHIBITS THE GROWTH OF Streptococcus agalactiae AND Edwarsiella tarda)." Jurnal Veteriner 18, no. 4 (2018): 604. http://dx.doi.org/10.19087/jveteriner.2017.18.4.604.

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This study aimed to observe the inhibition capability of Rhizophora sp. leaf extract towards the Streptococcus agalactiae and Edwardsiella tarda bacteria. Rhizophora sp. leaf was extracted using ethanol. Inhibition action of Rhizophora sp. leaf extract towards Streptococcus agalactiae and Edwardsiella tarda was tested on TSA solid media. The concentration of Rhizophora sp. leaf extract used were 1000, 2000, 3000, 4000, 5000, 6000, 7000, 8000, 9000, 10000 ppm, and novobiocin antibiotic was used as a control. Each treatment was conducted three times. MIC (minimum inhibition concentration) was pe
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8

Šukele, Renāte, Ance Bārzdiņa, Rudīte Koka, et al. "Antibacterial Activity of Tanacetum vulgare L. Extracts against Clinical Isolates of Bovine Mastitis." Applied Sciences 13, no. 5 (2023): 3369. http://dx.doi.org/10.3390/app13053369.

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A bovine mastitis is an infectious disease, which is usually treated with antibiotics. Alternatively, herbal medicine has been proposed due to bacterial resistance. The aim of this study was to determine the antibacterial activity of the acetonic and ethanolic extracts of dried flowers and leaves of Tanacetum vulgare L. against bovine mastitis-inducing clinical isolates such as Escherichia coli, Streptococcus agalactiae, Streptococcus uberis, Serratia liquefaciens, Staphylococcus aureus, and reference cultures of S. aureus and E. coli. The extracts of T. vulgare showed partial antibacterial ac
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9

Betriu, C., E. Culebras, I. Rodríguez-Avial, M. Gómez, B. A. Sánchez, and J. J. Picazo. "In Vitro Activities of Tigecycline against Erythromycin-Resistant Streptococcus pyogenes and Streptococcus agalactiae: Mechanisms of Macrolide and Tetracycline Resistance." Antimicrobial Agents and Chemotherapy 48, no. 1 (2004): 323–25. http://dx.doi.org/10.1128/aac.48.1.323-325.2004.

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ABSTRACT The activity of tigecycline was tested against erythromycin-resistant streptococci (107 Streptococcus pyogenes and 98 Streptococcus agalactiae strains). The presence of erythromycin and tetracycline resistance genes was determined by PCR. Among S. pyogenes strains the most prevalent gene was mef(A) (91.6%). The erm(B) gene was the most prevalent (65.3%) among S. agalactiae strains. Tigecycline proved to be very active against all the isolates tested (MIC at which 90% of the isolates tested were inhibited, 0.06 μg/ml), including those resistant to tetracycline.
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10

Achard, Adeline, Corinne Villers, Vianney Pichereau, and Roland Leclercq. "New lnu(C) Gene Conferring Resistance to Lincomycin by Nucleotidylation in Streptococcus agalactiae UCN36." Antimicrobial Agents and Chemotherapy 49, no. 7 (2005): 2716–19. http://dx.doi.org/10.1128/aac.49.7.2716-2719.2005.

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ABSTRACT Streptococcus agalactiae UCN36 was resistant to lincomycin (MIC = 16 μg/ml) but susceptible to clindamycin (MIC = 0.12 μg/ml) and erythromycin (MIC = 0.06 μg/ml). A 4-kb HindIII fragment was cloned from S. agalactiae UCN36 total DNA on plasmid pUC18 and introduced into Escherichia coli AG100A, where it conferred resistance to lincomycin. The sequence analysis of the fragment showed the presence of a 1,724-bp element delineated by imperfect inverted repeats (22 of 25 bp) and inserted in the operon for capsular synthesis of S. agalactiae UCN36. This element carried two open reading fram
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