Relevant bibliographies by topics / Streptococcus gordonii

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  2. Dissertations / Theses
  3. Books
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  5. Conference papers

Academic literature on the topic 'Streptococcus gordonii'

Author: Grafiati
Published: 4 June 2021
Last updated: 25 July 2025

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Journal articles on the topic "Streptococcus gordonii"

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Brown, Alan E., Jeffrey D. Rogers, Elaine M. Haase, Peter M. Zelasko, and Frank A. Scannapieco. "Prevalence of the Amylase-Binding Protein A Gene (abpA) in Oral Streptococci." Journal of Clinical Microbiology 37, no. 12 (1999): 4081–85. http://dx.doi.org/10.1128/jcm.37.12.4081-4085.1999.

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Salivary amylase binds specifically to a number of oral streptococcal species. This interaction may play an important role in dental plaque formation. Recently, a 585-bp gene was cloned and sequenced from Streptococcus gordonii Challis encoding a 20.5-kDa amylase-binding protein (AbpA). The goal of this study was to determine if related genes are present in other species of oral streptococci. Biotinylated abpA was used in Southern blot analysis to screen genomic DNA from several strains representing eight species of oral streptococci. This probe hybridized with a 4.0-kbHindIII restriction frag
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Kreth, Jens, Yongshu Zhang, and Mark C. Herzberg. "Streptococcal Antagonism in Oral Biofilms: Streptococcus sanguinis and Streptococcus gordonii Interference with Streptococcus mutans." Journal of Bacteriology 190, no. 13 (2008): 4632–40. http://dx.doi.org/10.1128/jb.00276-08.

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ABSTRACT Biofilms are polymicrobial, with diverse bacterial species competing for limited space and nutrients. Under healthy conditions, the different species in biofilms maintain an ecological balance. This balance can be disturbed by environmental factors and interspecies interactions. These perturbations can enable dominant growth of certain species, leading to disease. To model clinically relevant interspecies antagonism, we studied three well-characterized and closely related oral species, Streptococcus gordonii, Streptococcus sanguinis, and cariogenic Streptococcus mutans. S. sanguinis a
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Cheng, X., S. Redanz, P. Treerat, et al. "Magnesium-Dependent Promotion of H2O2 Production Increases Ecological Competitiveness of Oral Commensal Streptococci." Journal of Dental Research 99, no. 7 (2020): 847–54. http://dx.doi.org/10.1177/0022034520912181.

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The pyruvate oxidase (SpxB)–dependent production of H2O2 is widely distributed among oral commensal streptococci. Several studies confirmed the ability of H2O2 to antagonize susceptible oral bacterial species, including caries-associated Streptococcus mutans as well as several periodontal pathobionts. Here we report a potential mechanism to bolster oral commensal streptococcal H2O2 production by magnesium (Mg2+) supplementation. Magnesium is a cofactor for SpxB catalytic activity, and supplementation increases the production of H2O2 in vitro. We demonstrate that Mg2+ affects spxB transcription
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Ashby, Michael T., Jens Kreth, Muthu Soundarajan, and Laure Sita Sivuilu. "Influence of a model human defensive peroxidase system on oral streptococcal antagonism." Microbiology 155, no. 11 (2009): 3691–700. http://dx.doi.org/10.1099/mic.0.031310-0.

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Streptococcus is a dominant genus in the human oral cavity, making up about 20 % of the more than 800 species of bacteria that have been identified, and about 80 % of the early biofilm colonizers. Oral streptococci include both health-compatible (e.g. Streptococcus gordonii and Streptococcus sanguinis) and pathogenic strains (e.g. the cariogenic Streptococcus mutans). Because the streptococci have similar metabolic requirements, they have developed defence strategies that lead to antagonism (also known as bacterial interference). S. mutans expresses bacteriocins that are cytotoxic toward S. go
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Woo, Patrick CY, Jade LL Teng, Kit-wah Leung, et al. "Streptococcus sinensis may react with Lancefield group F antiserum." Journal of Medical Microbiology 53, no. 11 (2004): 1083–88. http://dx.doi.org/10.1099/jmm.0.45745-0.

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Lancefield group F streptococci have been found almost exclusively as members of the ‘Streptococcus milleri’ group, although they have been reported very occasionally in some other streptococcal species. Among 302 patients with bacteraemia caused by viridans streptococci over a 6-year period, three cases were caused by Streptococcus sinensis (type strain HKU4T, HKU5 and HKU6). All three patients had infective endocarditis complicating their underlying chronic rheumatic heart diseases. Gene sequencing showed no base differences between the 16S rRNA gene sequences of HKU5 and HKU6 and that of HK
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Nobbs, Angela H., Yongshu Zhang, Ali Khammanivong, and Mark C. Herzberg. "Streptococcus gordonii Hsa Environmentally Constrains Competitive Binding by Streptococcus sanguinis to Saliva-Coated Hydroxyapatite." Journal of Bacteriology 189, no. 8 (2007): 3106–14. http://dx.doi.org/10.1128/jb.01535-06.

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ABSTRACT Competition between pioneer colonizing bacteria may determine polymicrobial succession during dental plaque development, but the ecological constraints are poorly understood. For example, more Streptococcus sanguinis than Streptococcus gordonii organisms are consistently isolated from the same intraoral sites, yet S. gordonii fails to be excluded and survives as a species over time. To explain this observation, we hypothesized that S. gordonii could compete with S. sanguinis to adhere to saliva-coated hydroxyapatite (sHA), an in vitro model of the tooth surface. Both species bound sim
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Loimaranta, V., N. S. Jakubovics, J. Hytönen, J. Finne, H. F. Jenkinson, and N. Strömberg. "Fluid- or Surface-Phase Human Salivary Scavenger Protein gp340 Exposes Different Bacterial Recognition Properties." Infection and Immunity 73, no. 4 (2005): 2245–52. http://dx.doi.org/10.1128/iai.73.4.2245-2252.2005.

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ABSTRACT Salivary scavenger receptor cysteine-rich protein gp340 aggregates streptococci and other bacteria as part of the host innate defense system at mucosal surfaces. In this article, we have investigated the properties of fluid-phase gp340 and hydroxylapatite surface-adsorbed gp340 in aggregation and adherence, respectively, of viridans group streptococci (e.g., Streptococcus gordonii and Streptococcus mutans), non-viridans group streptococci (e.g., Streptococcus pyogenes and Streptococcus suis), and oral Actinomyces. Fluid-phase gp340 and surface-phase gp340 bioforms were differentially
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Wan, S. X., J. Tian, Y. Liu, A. Dhall, H. Koo, and G. Hwang. "Cross-Kingdom Cell-to-Cell Interactions in Cariogenic Biofilm Initiation." Journal of Dental Research 100, no. 1 (2020): 74–81. http://dx.doi.org/10.1177/0022034520950286.

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Candida albicans is known to form polymicrobial biofilms with various Streptococcus spp., including mitis and mutans group streptococci. Streptococcus gordonii (mitis group) has been shown to bind avidly to C. albicans hyphae via direct cell-to-cell interaction, while the cariogenic pathogen Streptococcus mutans (mutans group) interacts with the fungal cells via extracellular glucans. However, the biophysical properties of these cross-kingdom interactions at the single-cell level during the early stage of biofilm formation remain understudied. Here, we examined the binding forces between S. mu
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Mallaley, P. P., S. A. Halperin, A. Morris, A. MacMillan, and S. F. Lee. "Expression of a pertussis toxin S1 fragment by inducible promoters in oral Streptococcus and the induction of immune responses during oral colonization in mice." Canadian Journal of Microbiology 52, no. 5 (2006): 436–44. http://dx.doi.org/10.1139/w05-151.

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Previous work aimed at developing a live oral vaccine expressing pertussis toxin S1 fragment on the surface of the bacterium Streptococcus gordonii elicited a lower than expected antibody response, perhaps because of low antigen expression. In this study, in-frame promoter fusions were constructed to investigate whether an increase in antigen production by the streptococcal vaccine strain results in a better antibody response. The promoters tested were (i) the Streptococcus mutans sucrose-inducible fructosyltransferase (ftf) promoter and (ii) the Bacillus subtilis/Escherichia coli chimeric tet
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W S Harty, Derek. "Virulence factors in streptococcal infective endocarditis." Microbiology Australia 26, no. 3 (2005): 114. http://dx.doi.org/10.1071/ma05114.

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Infective endocarditis (IE) is a life threatening, endovascular infection occurring when bacteria enter the blood stream and adhere to heart valves. Mortality rates remain in the range of 11-27%. The most common infecting micro-organisms are now the staphylococci (44%) although streptococci (31%) and particularly the oral streptococci (21%) are still major causative agents. Many different oral streptococci have been isolated from IE cases, the most common being Streptococcus sanguinis, Streptococcus oralis, Streptococcus gordonii, Streptococcus mitis, Streptococcus anginosus group and mutans s
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Dissertations / Theses on the topic "Streptococcus gordonii"

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Christie, Julie. "Fibronectin-interacting proteins in Streptococcus gordonii." Thesis, University of Bristol, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.324360.

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Macarthur, Deborah Jane. "Mapping The Proteome Of Streptococcus Gordonii." Thesis, The University of Sydney, 2005. http://hdl.handle.net/2123/5097.

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Streptococcus gordonii is a primary coloniser of the tooth surface where it efficiently ferments carbohydrates at pH levels above 6.0. By not being able to maintain the pH of dental plaque to a level required for enamel dissolution, the dominance of S. gordonii in dental plaque is considered a sign of a healthy oral cavity. However, upon entering the bloodstream and encountering a rise in pH, S. gordonii may become pathogenic, being one of the major causative organisms associated with infective endocarditis. Proteome analyses of S. gordonii grown at steady state in a chemostat allowed t
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Macarthur, Deborah Jane. "Mapping the proteome of Streptococcus gordonii." University of Sydney. Health Science, 2005. http://hdl.handle.net/2123/686.

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Streptococcus gordonii is a primary coloniser of the tooth surface where it efficiently ferments carbohydrates at pH levels above 6.0. By not being able to maintain the pH of dental plaque to a level required for enamel dissolution, the dominance of S. gordonii in dental plaque is considered a sign of a healthy oral cavity. However, upon entering the bloodstream and encountering a rise in pH, S. gordonii may become pathogenic, being one of the major causative organisms associated with infective endocarditis. Proteome analyses of S. gordonii grown at steady state in a chemostat allowed t
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Jack, Alison Alexandra. "Signalling interactions between Streptococcus gordonii and Candida albicans." Thesis, University of Bristol, 2014. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.633446.

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Streptococcus gordonii is an early bacterial coloniser of the oral cavity and influences development of plaque biofilms. Interactions of Candida albicans, an opportunistic fungal pathogen, with S. gordonii are hypothesised to promote fungal carriage and persistence. Evidence suggests that signalling molecules produced by streptococci, including competence stimulating peptide (eSP) and autoinducer 2 (AI -2L affect C. albicans growth. However, less is understood about the molecular basis of these interactions.
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Forsgren, Nina. "Structural studies of the surface adhesin SspB from Streptococcus gordonii." Doctoral thesis, Umeå : Umeå universitet, 2010. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-32910.

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Ilias, Mohammad. "Family II soluble inorganic pyrophosphatases from 'Streptococcus gordonii' and 'Vibrio cholerae'." Thesis, University of Birmingham, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.410862.

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Robinson, Jill Christie. "Impact of L-arginine on Streptococcus gordonii gene expression and biofilm formation." Thesis, University of Newcastle upon Tyne, 2016. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.701159.

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Streptococcus gordonii is an oral commensal bacterium, and an early coloniser of the acquired salivary pellicle that coats tooth surfaces. As such, it is a key organism in the establishment of dental plaque biofilms. The amino acid L-arginine has been previously shown to play a role in biofilm formation in other oral species, and depletion of L-arginine has a significant impact upon S. gordonii growth and gene regulation. Three L-arginine-dependent transcription regulators have been identified in S. gordonii, but it is currently not clear how these co-ordinate to sense and respond to changes i
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O'Connell, Silverman Richard James. "Molecular basis of mixed-species biofilm formation between streptococcus gordonii and candida albicans." Thesis, University of Bristol, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.529889.

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Higashi, Daniela. "Modulação do biofilme de Porphyromonas gingivalis pela associação com Streptococcus gordonii e com Prevotella intermedia." Universidade de São Paulo, 2015. http://www.teses.usp.br/teses/disponiveis/42/42132/tde-10042015-123236/.

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P. gingivalis é um dos principais patógenos da doença periodontal, é encontrado em biofilmes orais com S. gordonii e P. intermedia e em células endoteliais da artéria coronária in vivo. P. gingivalis necessita de ferro em seu metabolismo e pode usar certas proteínas do hospedeiro como fontes deste íon em ambientes limitantes. Assim, este estudo investigou o papel dos genes PGN0741/PG0637 (receptor dependente de TonB) e PGN0531/PG1380 (fvW) de P. gingivalis na formação de biofilme em diferentes concentrações de ferro, em biofilmes mistos com S. gordonii e P. intermedia, e na adesão e invasão de
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Nylander, Åsa, Gunnel Svensäter, Dilani B. Senadheera, Dennis G. Cvitkovitch, Julia R. Davies, and Karina Persson. "Structural and functional analysis of the N-terminal domain of the Streptococcus gordonii adhesin Sgo0707." Umeå universitet, Oral mikrobiologi, 2013. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-71563.

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The commensal Streptococcus gordonii expresses numerous surface adhesins with which it interacts with other microorganisms, host cells and salivary proteins to initiate dental plaque formation. However, this Gram-positive bacterium can also spread to non-oral sites such as the heart valves and cause infective endocarditis. One of its surface adhesins, Sgo0707, is a large protein composed of a non-repetitive N-terminal region followed by several C-terminal repeat domains and a cell wall sorting motif. Here we present the crystal structure of the Sgo0707 N-terminal domains, refined to 2.1 Å reso
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Books on the topic "Streptococcus gordonii"

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Obinwa, Ngozika Kanayo. Identification and cloning of Streptococcus gordonii LGR2 and Streptococcus crista CR311 genes encoding their coaggregations with other oral bacteria. University of Manchester, 1996.

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Whittaker, Catherine Jill. Identification and cloning of Streptococcus gordonii LGR2 genes encoding adhesins for saliva-coated hydroxyapatite (SHA). University of Manchester, 1993.

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Shirkhodaee, Mohammad Masoud. Isolation of a DNA probe for the identification of the tooth surface adhesin gene(s) of Streptococcus gordonii LGR2. University of Manchester, 1994.

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Vidyasanker, Radhika. The rpsL gene and streptomycin resistance in Streptococcus gordonii and Streptococcus pyogenes. 1999.

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Book chapters on the topic "Streptococcus gordonii"

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Vickerman, M. M., and D. B. Clewell. "Regulation of Streptococcus gordonii Glucosyltransferase." In Streptococci and the Host. Springer US, 1997. http://dx.doi.org/10.1007/978-1-4899-1825-3_154.

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Shiroza, Teruaki, and Howard Kuramitsu. "Secretion of heterologous proteins by genetically engineered Streptococcus gordonii." In Methods for studying the genetics, molecular biology, physiology, and pathogenesis of the streptococci. Springer Netherlands, 1998. http://dx.doi.org/10.1007/978-94-017-2258-2_15.

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Pozzi, Gianni, Marco R. Oggioni, and Donata Medaglini. "Recombinant Streptococcus gordonii as a Live Vehicle for Vaccine Antigens." In Gram-Positive Bacteria. Springer Berlin Heidelberg, 1997. http://dx.doi.org/10.1007/978-3-662-07548-7_3.

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Andersen, Roxanna N., R. Dwayne Lunsford, and Paul E. Kolenbrander. "Determination of the Transcript Size and Start Site of the Putative sca Operon of Streptococcus gordonii ATCC 51656 (Formerly Strain PK488)." In Streptococci and the Host. Springer US, 1997. http://dx.doi.org/10.1007/978-1-4899-1825-3_153.

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Obladen, Michael. "Systemic infection." In Oxford Textbook of the Newborn, edited by Michael Obladen. Oxford University Press, 2021. http://dx.doi.org/10.1093/med/9780198854807.003.0049.

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In antiquity, transmission of disease was attributed to the miasma or contagion theory. In the Middle Ages, living in proximity to domestic animals and flies, the scarce use of soap, and absent sewage augmented the exposure to bacteria. In the early 19th century, Gordon, Holmes, and Semmelweis understood that maternal childbed fever—closely related to neonatal sepsis—was transferred by the physician’s hands to the mother during delivery. Before bacteria were discovered in the mid-19th century, septic infections in the newborn were perceived as different disorders: erysipelas, Buhl’s disease, W
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Conference papers on the topic "Streptococcus gordonii"

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Bath, A. S., and J. Kaur. "A Rare Cause of Empyema: Streptococcus Gordonii." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a3727.

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Soekanto, Sri Angky, Saly Salim Alatas, Rima Ristanti, Ferry P. Gultom, and Muhamad Sahlan. "Efficacy of propolis fluoride in inhibiting the formation of Streptoccocus mutans, Streptococcus gordonii, and Streptococcus sanguinis biofilm." In SECOND INTERNATIONAL CONFERENCE OF MATHEMATICS (SICME2019). Author(s), 2019. http://dx.doi.org/10.1063/1.5096712.

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Withers, Nathan J., Arjun Senthil, Marek Osinski, et al. "Effects of iron-oxide nanoparticles on compound biofilms of streptococcus gordonii and fusobacterium nucleatum." In Colloidal Nanoparticles for Biomedical Applications XIII, edited by Xing-Jie Liang, Wolfgang J. Parak, and Marek Osiński. SPIE, 2018. http://dx.doi.org/10.1117/12.2299280.

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