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Dissertations / Theses on the topic 'Streptococcus pyogenes'

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Published: 4 June 2021

Last updated: 25 July 2025

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1

Berge, Andreas. "Molecular analysis of Streptococcus pyogenes and its interactions with the human host." Lund : Lund University, 1997. http://catalog.hathitrust.org/api/volumes/oclc/68945123.html.

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2

Thern, Anette. "Interactions between Streptococcus pyogenes and the human immune system with special reference to C4b-binding protein /." Lund : Dept. of Medical Microbiology, Lund University, 1998. http://catalog.hathitrust.org/api/volumes/oclc/39224761.html.

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3

Zhang, Meng. "Proteomic analysis of streptococcus pyogenes." Thesis, Northumbria University, 2007. http://nrl.northumbria.ac.uk/842/.

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Streptococcus pyogenes (group A streptococcus, GAS) is a major human Gram-positive pathogen that causes infections that normally occur in the respiratory tract, the skin, the wound, the lung, the bloodstream and/or muscle tissues and result in millions of deaths every year. To cause such infections, S. pyogenes produces a wide range of virulence factors. The destruction of connective tissue and the hyaluronic acid therein plays an important role in pathogenesis. S. pyogenes was propagated in hyaluronic acid rich growth media in an attempt to create a simple biological system that could reflect

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4

Siou, GeÌrard Paul Serge. "Streptococcus pyogenes interactions with human tonsils." Thesis, University of Newcastle upon Tyne, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.424082.

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5

Desai, Meeta. "Molecular epidemiological typing of Streptococcus pyogenes." Thesis, Open University, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.299021.

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6

Broglia, Laura. "Regulating with ribonucleases in Streptococcus pyogenes." Doctoral thesis, Humboldt-Universität zu Berlin, 2020. http://dx.doi.org/10.18452/21573.

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Bakterien haben eine Vielzahl an Strategien entwickelt, um sich an ständig wechselnde Umweltbedingungen anzupassen, darunter auch post-transkriptionelle regulatorische Mechanismen. Die Genexpression kann hierbei durch gezielten Abbau oder Stabilisierung von RNA durch Ribonukleasen (RNasen) reguliert werden. RNasen weisen je nach Spezies allerdings unterschiedliche Effekte auf Genexpression und bakterielle Physiologie, sowie verschiedene Strategien der Substraterkennung auf. Dies zeigt, dass unser Verständnis des RNA-Abbaus bei weitem nicht vollständig ist. Ziel dieser Arbeit ist es, die Eigen

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7

Becherelli, Marco <1979&gt. "Functional characterization of Streptococcus pyogenes pili." Doctoral thesis, Alma Mater Studiorum - Università di Bologna, 2010. http://amsdottorato.unibo.it/3015/1/Becherelli_Marco_Tesi.pdf.

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Group A Streptococcus is a Gram-positive human pathogen able to colonize both upper respiratory tract and skin. GAS is responsible for several acute diseases and autoimmune sequelae that account for half a million deaths worldwide every year (Cunningham et al., 2000). As other bacteria, GAS infections requires the capacity of the pathogen to adhere to host tissues and to form cell aggregates. The ability to persist in distinct host niches like the throat and the skin and to trigger infections is associated with the expression of different GAS virulence factors. GAS pili has been described as i

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8

Becherelli, Marco <1979&gt. "Functional characterization of Streptococcus pyogenes pili." Doctoral thesis, Alma Mater Studiorum - Università di Bologna, 2010. http://amsdottorato.unibo.it/3015/.

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Group A Streptococcus is a Gram-positive human pathogen able to colonize both upper respiratory tract and skin. GAS is responsible for several acute diseases and autoimmune sequelae that account for half a million deaths worldwide every year (Cunningham et al., 2000). As other bacteria, GAS infections requires the capacity of the pathogen to adhere to host tissues and to form cell aggregates. The ability to persist in distinct host niches like the throat and the skin and to trigger infections is associated with the expression of different GAS virulence factors. GAS pili has been described as i

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9

McNamara, Case W. "Molecular analysis of Streptococcus pyogenes M1 protein." Connect to a 24 p. preview or request complete full text in PDF format. Access restricted to UC campuses, 2006. http://wwwlib.umi.com/cr/ucsd/fullcit?p3230034.

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Thesis (Ph. D.)--University of California, San Diego, 2006.<br>Title from first page of PDF file (viewed November 17, 2006). Available via ProQuest Digital Dissertations. Vita. Includes bibliographical references.

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10

Dixon, Emma Victoria. "Mechanisms of immunoglobulin deactivation by Streptococcus pyogenes." Thesis, University of Oxford, 2014. http://ora.ox.ac.uk/objects/uuid:ec80e3f9-0c73-4d39-bc68-c39b927365d4.

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The bacteria Streptococcus pyogenes produces a multitude of proteins which interact with and alter the functions of the host immune system. Two such proteins, Endoglycosidase S (EndoS) and Immunoglobulin G-degrading enzyme from S. pyogenes (IdeS) are able to specifically alter the effector functions of immunoglobulin G (IgG). EndoS is a glycoside hydrolase which removes the conserved <i>N</i>-linked glycan from IgG Fc whereas IdeS is a cysteine protease that cleaves the exible protein hinge of IgG. The activity of both proteins results in the reduced ability of IgG to elicit immune responses t

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11

Caswell, Clayton Christopher. "The SCL1 protein of Streptococcus pyogenes a structure-function analysis /." Morgantown, W. Va. : [West Virginia University Libraries], 2008. https://eidr.wvu.edu/etd/documentdata.eTD?documentid=6026.

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Thesis (Ph. D.)--West Virginia University, 2008.<br>Title from document title page. Document formatted into pages; contains xi, 190 p. : ill. (some col.). Includes abstract. Includes bibliographical references.

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12

Darenberg, Jessica. "Streptococcus pyogenes infections and toxic shock syndrome : molecular epidemiology and immunotherapy /." Stockholm, 2006. http://diss.kib.ki.se/2006/91-7140-676-X/.

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13

Meinhart, Anton. "Kristallstrukturanalyse des e, z-Proteinkomplexes [Epsilon, zeta-Proteinkomplexes], kodiert vom Plasmid pSM19035 aus Streptrococcus pyogenes." [S.l.] : [s.n.], 2001. http://www.diss.fu-berlin.de/2001/188/index.html.

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14

Luis, Philippe Renard Vincent. "Le TDR modifie-t-il la pratique des médecins généralistes d'Ile de France ?" Créteil : Université de Paris-Val-de-Marne, 2006. http://doxa.scd.univ-paris12.fr:80/theses/th0240166.pdf.

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15

Russell, Hugh Hayden. "Molecular basis of epithelial internalisation of Streptococcus pyogenes." Thesis, Imperial College London, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.423540.

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16

Le, Rhun Anaïs. "Multifaceted RNA-mediated regulatory mechanisms in Streptococcus pyogenes." Doctoral thesis, Umeå universitet, Molekylär Infektionsmedicin, Sverige (MIMS), 2015. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-111090.

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Bacterial pathogens rely on precise regulation of gene expression to coordinate host infection processes and resist invasion by mobile genetic elements. An interconnected network of protein and RNA regulators dynamically controls the expression of virulence factors using a variety of mechanisms. In this thesis, the role of selected regulators, belonging to the class of small RNAs (sRNAs), is investigated. Streptococcus pyogenes is a pathogen responsible for a wide range of human diseases. Genome-wide screenings have indicated that S. pyogenes encodes numerous sRNAs, yet only a limited number h

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17

Sethman, Chad Robert. "Attachment of Streptococcus pyogenes to Host Epithelial Cells." Miami University / OhioLINK, 2003. http://rave.ohiolink.edu/etdc/view?acc_num=miami1071776892.

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18

Tedde, Vittorio. "Zinc uptake in Streptococcus pyogenes: characterisation of adcA." Doctoral thesis, Università degli studi di Padova, 2012. http://hdl.handle.net/11577/3422118.

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Streptococcus pyogenes (also known as Group A Streptococcus, GAS) is a capsulated Gram-positive, human-adapted pathogen. GAS strains express different virulence factors exposed on the bacterial surface or secreted outside the cell. Among the secreted virulence factors, superantigens (SAGs) are certainly the most toxic factors. A recent study demonstrates that, in vitro, the streptococcal superantigen SpeI interacts with AdcA and Lmb, the substrate binding subunits of the two zinc transporters. In particular, AdcA belongs to the high-affinity zinc transporter Adc, involved in adhesion, competen

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19

Smeesters, Pierre. "Epidémiologie, pathogénie et prise en charge des infections à Streptococcus pyogenes touchant les enfants de Bruxelles et de Brasília." Doctoral thesis, Universite Libre de Bruxelles, 2007. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/210620.

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Les Streptocoques Béta-hémolytiques du groupe A (GAS) sont responsables de manifestations cliniques variées et de séquelles non suppuratives comme notamment le rhumatisme articulaire aigu (RAA). Les affections sévères à GAS tuent plus de 500.000 personnes chaque année. Le pouvoir pathogène du GAS est encore mal compris. Il semble être notamment lié à la présence de nombreux gènes codant pour des facteurs de virulence dans le génome du GAS, dont celui codant la protéine emm. La protéine transmembranaire M joue un rôle essentiel dans la virulence du GAS. Le typage moléculaire des GAS se base sur

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20

Cunningham, Cynthia A. "Induction of myosin cross-reactive antibody and cytolytic T cell responses in mice with Streptococcus pyogenes." Morgantown, W. Va. : [West Virginia University Libraries], 2000. http://etd.wvu.edu/templates/showETD.cfm?recnum=1530.

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21

Malerba, Mariangela. "The immunomodulatory role of epidermal hepcidin in infectious condition." Thesis, Sorbonne Paris Cité, 2018. https://wo.app.u-paris.fr/cgi-bin/WebObjects/TheseWeb.woa/wa/show?t=2166&f=13318.

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L'hepcidine, initialement identifiée comme un peptide antimicrobien, s'est révélée être un peptide hormonal clé du métabolisme du fer capable d'inhiber l'absorption intestinale du fer alimentaire et son recyclage par les macrophages. L'expression de l'hepcidine est induite par l’excès de fer mais également en réponse à l' infection/inflammation. L'hepcidine, comme son nom l'indique, est principalement synthétisée par les hépatocytes mais également, à plus faibles niveaux, par d'autres types cellulaires. Notre équipe a démontré que ce peptide hépatique était suffisant pour assurer l'homéostasie

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22

Beck, Isabel L. "Tolérance à la pénicilline G de souches de "Streptococcus pyogenes" isolées au cours d'angines aigue͏̈s de l'enfant." Paris 5, 1993. http://www.theses.fr/1993PA05P199.

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23

Galeas, Pena Trilce Michelle. "Thermoregulation of capsule production of Streptococcus pyogenes strain HSC5 /." Available to subscribers only, 2009. http://proquest.umi.com/pqdweb?did=1967978671&sid=7&Fmt=2&clientId=1509&RQT=309&VName=PQD.

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24

Delgado, Giselle M. "SIAA and Neat2 Heme Binding Proteins from Streptococcus Pyogenes." Digital Archive @ GSU, 2009. http://digitalarchive.gsu.edu/chemistry_theses/24.

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The bacterium Streptococcus pyogenes requires heme, which is taken up via an ABC transporter. An understanding of this pathway may result in new approaches to antibacterial agents. Both SiaA and NEAT2 (NEAr Transporter 2) are proteins involved in heme binding. One of the axial ligands of SiaA, His 229, was purified to study how mutagenesis affects heme binding. UV-visible studies showed a small band at 420 nm with respect to the protein band at 288 nm which probably indicates that heme was lost easily from this mutant. We have also worked to optimize the yield of Shr-NEAT2 by changing differen

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25

Calusni, Ana Lucia Roscani. "Interação entre o Streptococcus pyogenes e a hemoglobina S." [s.n.], 1994. http://repositorio.unicamp.br/jspui/handle/REPOSIP/316745.

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Orientador: Antonio Sergio Ramalho<br>Dissertação (mestrado) - Universidade Estadual de Campinas, Instituto de Biologia<br>Made available in DSpace on 2018-07-19T08:31:44Z (GMT). No. of bitstreams: 1 Calusni_AnaLuciaRoscani_M.pdf: 1715301 bytes, checksum: acd3974eacbc813ea7e1416eeec145b7 (MD5) Previous issue date: 1994<br>Mestrado<br>Genetica<br>Mestre em Ciências Biológicas

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26

LE, BOUGUENEC-BRIVE CHANTAL. "Etude d'un element genetique mobile (tn3701) chez streptococcus pyogenes." Paris 7, 1989. http://www.theses.fr/1989PA077082.

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Mise en evidence chez streptococcus pyogenes d'un element transposable (tn3701) capable de conferer la resistance aux antibiotiques erythromycine, tetracycline et microcycline. Une carte de restriction de la region contenant cet element a ete etablie. Par hybridation moleculaire il a ete possible de reveler une forte homologie entre tn3701 et d'autres elements chromosomiques transferables par conjugaison decrits dans d'autres souches de streptococcus

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27

Weinstein, Kathryn Elizabeth. "Generation of Diversity During the Survival of Streptococcus pyogenes." Diss., Temple University Libraries, 2010. http://cdm16002.contentdm.oclc.org/cdm/ref/collection/p245801coll10/id/105846.

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Microbiology and Immunology<br>Ph.D.<br>Streptococcus pyogenes is a human-specific pathogen that can cause a wide variety of diseases. These diseases range from the relatively mild pharyngitis and impetigo to invasive diseases such as necrotizing fasciitis to post-streptococcal sequelae such as rheumatic heart disease. The bacteria are frequently carried asymptomatically and may cause recurrent disease. Corresponding with their etiologic variation amongst diseases, clinical isolates demonstrate diverse virulence factor expression and random genetic mutations. In these studies, we examine the r

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28

Steinberg, Gregory. "Long-term Stationary Phase Behavior of Streptococcus pyogenes Biofilms." Master's thesis, Temple University Libraries, 2012. http://cdm16002.contentdm.oclc.org/cdm/ref/collection/p245801coll10/id/170151.

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Microbiology and Immunology<br>M.S.<br>Long-term Stationary Phase Behavior of Streptococcus pyogenes Biofilms Department of Microbiology and Immunology Streptococcus pyogenes is the etiological agent of many human diseases ranging from mild superficial skin infections and pharyngitis to life-threatening necrotizing fasciitis. There can be several complications as a result of S. pyogenes infection including post-streptococcal glomerulonephritis and rheumatic fever, which leads to rheumatic heart disease. Despite the significant virulence associated with the pathogen, the bacteria can also persi

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29

Galeas, Trilce Michelle. "Thermoregulation of Capsule Production of Streptococcus pyogenes Strain HSC5." OpenSIUC, 2009. https://opensiuc.lib.siu.edu/theses/122.

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Group A Streptococcus (GAS) is responsible for mild and common infections like tonsillitis and pharyngitis, and more serious invasive disorders like necrotizing fasciitis and glomerulonephritis. The ability to invade tissues is closely linked to the virulence factors expressed by the bacterium. Hyaluronic acid capsule expression is variable among all the strains in S. pyogenes and confers the capacity to evade the immune response. In a previous study, it was found that capsule production in CovR mutants was temperature-regulated, showing no capsule production at 37℃ but increased produ

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30

Wright, Jordan. "Capsule Thermoregulation and Non-Coding RNA in Streptococcus pyogenes." OpenSIUC, 2014. https://opensiuc.lib.siu.edu/theses/1505.

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Streptococcus pyogenes is a re-emerging pathogen that produces superficial and life threatening invasive diseases. One important virulence factor in S. pyogenes is hyaluronic acid capsule which has been shown to increase expression at sub-body temperatures in certain strains. This study showed that thermoregulation is common in invasive clinical isolates. Regulation was shown to occur independent of the CovRS two-component regulator in a post transcription manner and before protein level regulation. The endoribonuclease, CvfA, was also confirmed to be required for capsule thermoregulation.

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31

Johansson, Söderberg Jenny. "The streptococcal IgG degrading enzyme IdeS : studies on host-pathogen interactions." Doctoral thesis, Umeå universitet, Institutionen för molekylärbiologi (Medicinska fakulteten), 2012. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-53706.

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The important human pathogen Streptococcus pyogenes causes both mild infections such as pharyngitis and impetigo but also severe life threatening invasive infections. Specific antibodies (IgG) recognize pathogens and are important mediators for pathogen clearance by the immune defence. S.ipyogenes expresses a highly effective and specific IgG endopeptidase called IdeS (immunoglobulin degrading enzyme of S.ipyogenes). IdeS rescues bacteria from opsonising IgG by cleavage of IgG generating two fragments F(ab´)2 and ½Fc. Moreover, IdeS block ROS production by neutrophils. In this thesis I have s

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32

Libkind, Marianna. "SiaA, a heme protein." unrestricted, 2006. http://etd.gsu.edu/theses/available/etd-02192007-092252/.

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Thesis (honors)--Georgia State University, 2006.<br>Title from title screen. Under the direction of Dabney White Dixon. Electronic text (46 p. : ill. (some col.)) : digital, PDF file. Description based on contents viewed Aug. 1, 2007. Includes bibliographical references (p 45-46).

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33

Fontaine, Michael Christopher. "Allel-replacement mutagenesis of group A streptococci." Thesis, University of Newcastle Upon Tyne, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.285338.

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34

Parks, Thomas Edward. "Host genetic susceptibility to group A streptococcal disease." Thesis, University of Cambridge, 2016. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.709471.

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35

Abe, Lucienne M. "Adhesion and internalization of group A streptococcus isolates found in Hawaii." Thesis, University of Hawaii at Manoa, 2003. http://proquest.umi.com/pqdweb?index=0&did=764803591&SrchMode=2&sid=1&Fmt=2&VInst=PROD&VType=PQD&RQT=309&VName=PQD&TS=1233167604&clientId=23440.

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36

Vindebro, Reine. "Studies on secreted cysteine proteases of Streptococcus pyogenes : IdeS and SpeB." Doctoral thesis, Umeå universitet, Institutionen för molekylärbiologi (Medicinska fakulteten), 2014. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-88223.

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The pathogen Streptococcus pyogenes is a significant cause of human morbidity and mortality. Most of the work in this thesis is focused on streptococcal virulence factor IdeS, but the thesis also features work on SpeB, another streptococcal virulence factor. Both IdeS and SpeB are secreted cysteine proteases and both have previously been shown to degrade human IgG. IgG is the only known substrate for IdeS while SpeB is a more promiscuous protease with a larger number of identified substrates. A significant part of the data presented in this thesis is the result of designing and optimizing meth

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37

Almengor, Audry C. "Transcriptional regulation of the MGA virulence regulon in Streptococcus pyogenes." Access to abstract only; dissertation is embargoed until after 12/19/2006, 2005. http://www4.utsouthwestern.edu/library/ETD/etdDetails.cfm?etdID=115.

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38

Anderton, Stephen M. "A study of murine T lymphocyte responses to Streptococcus pyogenes." Thesis, University of Newcastle Upon Tyne, 1990. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.287332.

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39

Turner, Claire Elizabeth. "SpyCEP : The Interleukin 8 Cleaving Streptococcus pyogenes Cell Envelope Proteinase." Thesis, Imperial College London, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.501216.

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40

Abou, El Enien Ibrahim. "Caractérisation des propriétés biologiques du facteur d'opacité de Streptococcus pyogenes." Lyon 1, 1991. http://www.theses.fr/1991LYO1T067.

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41

Zarate, Bonilla Lina Johana. "Characterization of Chromosomally Encoded Toxin-Antitoxin Systems in Streptococcus pyogenes." Doctoral thesis, Humboldt-Universität zu Berlin, 2019. http://dx.doi.org/10.18452/20547.

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Streptococcus pyogenes ist ein humanpathogenes Bakterium, welches verschiedene Gewebe besiedeln kann und dadurch unterschiedliche Krankheiten verursacht. Die enorme Anpassungsfähigkeit des Bakteriums beruht auf dessen Fähigkeit, verschiedene, vom Wirt induzierte Stresskonditionen zu ertragen. Genetische Faktoren, die in diesem Zusammenhang eine Rolle spielen, sind Toxin-Antitoxin (TA) Systeme. Typ II TA Systeme kodieren für zwei Proteine, ein Toxin und ein Antitoxin, die einen stabilen TA Komplex bilden. Verschlechtern sich die Wachstumsbedingungen, kann das Antitoxin proteolytisch abgebaut we

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42

Tesorero, Melendez Rafael Angel. "Experimental validation for computationally predicted small RNAs of Streptococcus pyogenes." OpenSIUC, 2011. https://opensiuc.lib.siu.edu/theses/769.

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The human pathogen Streptococcus pyogenes (Group A Streptococcus or GAS) are a versatile Gram-positive cocci that havw shown complex modes of regulation of its different virulence factors. Discoveries of a few small non-coding RNAs (sRNAs) in S. pyogenes and their influence on the expression of virulence factors revealed an important role of sRNAs on S. pyogenes virulence. The genome-wide analysis of bacterial genomes for the discovery of sRNAs through computational methods has become an effective way to discover new sRNAs. In this study we provided a computational scheme where three different

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43

Swe, Pearl M., and n/a. "Mode of action of dysgalacticin and mechanism of its producer cell immunity." University of Otago. Department of Microbiology & Immunology, 2008. http://adt.otago.ac.nz./public/adt-NZDU20081119.111402.

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Dysgalacticin is a large, 21.5 kDa bacteriocin that belongs to subgroup B of the class III bacteriocins. It is ribosomally produced by Streptococcus dysgalactiae subsp. equisimilis strain W2580 and exerts inhibitory activity mainly against the medically important pathogen Streptococcus pyogenes by a "non-lytic" mechanism. Despite numerous studies of the mechanisms of action of a wide variety of bacteriocins and of the basis of their producer strain self-immunity, relatively little is known about the "non-lytic" class of bacteriocins. The structural gene encoding for dysgalacticin (dysA) was kn

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44

Sylla, Maguette Dème. "Contribution à l'étude de l'action opsonisante des immunoglobulines sur les streptocoques du groupe A." Lyon 1, 1987. http://www.theses.fr/1987LYO1T126.

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45

Rivera, Martínez M. Alba. "Estudi epidemiològic d'infeccions invasives i no invasives produïdes per Streptococcus pyogenes." Doctoral thesis, Universitat Autònoma de Barcelona, 2008. http://hdl.handle.net/10803/3920.

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Streptococcus pyogenes és un patogen humà responsable d'un ampli ventall d'infeccions que varien des d'infeccions superficials com faringitis i impetigen a formes sistèmiques greus com fascitis necrosant (FN) i síndrome del xoc tòxic estreptocòccic (SSTS). El ressorgiment i persistència de formes invasives greus descrit des de mitjans dels anys 1980 ha motivat una intensa recerca sobre els aspectes epidemiològics, microbiològics i clínics d'aquestes infeccions. <br/>S'ha realitzat un estudi retrospectiu de base hospitalària que inclou 126 soques de S. pyogenes (27 procedents d'infeccions invas

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46

Whatmore, Adrian Mark. "Sequence analysis of the emm-like gene family of Streptococcus pyogenes." Thesis, University of Newcastle Upon Tyne, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.357642.

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47

Alam, Faraz Mainul. "Modelling nasopharyngeal colonisation by Streptococcus pyogenes : bioluminescence and other longitudinal techniques." Thesis, Imperial College London, 2013. http://hdl.handle.net/10044/1/14463.

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Streptococcus pyogenes causes an estimated 616 million pharyngitis cases per year and a variety of invasive diseases such as necrotising fasciitis and toxic shock syndrome. The human nasopharynx is the major reservoir for all S. pyogenes infection, including severe invasive disease. A combination of biophotonic imaging (BPI) and direct nasal sampling techniques were used to longitudinally measure the in vivo carriage of S. pyogenes, looking at the effects of virulence factor expression on carriage and transmission and to enable vaccine evaluation. Direct nasal sampling demonstrated that the tw

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48

Power, Daniel Aaron, and n/a. "Non-culture based studies of the human upper respiratory tract microbiota and preliminary considerations of the influence of bacteriocin producing commensal and pathogenic oral streptococci." University of Otago. Department of Microbiology & Immunology, 2007. http://adt.otago.ac.nz./public/adt-NZDU20070620.160726.

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The upper respiratory tract (URT) of humans is complex and interconnected region and comprises several major ecosystems including the oral cavity, oropharynx, nasal cavity, sinuses, nasopharynx and middle ear. Most of the anatomical locations within the URT are colonised with a normal bacterial microbiota, within which are often organisms having the potential to cause disease. The diseases of the URT are both varied and frequent in their occurrence, and conditions such as otitis media, rhinosinusitis and pharyngitis are sources of morbidity and mortality in adults and children in both developi

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49

Amicis, Karine Marafigo De. "Análise in vitro da capacidade de cobertura da vacina em desenvolvimento contra Streptococcus pyogenes." Universidade de São Paulo, 2013. http://www.teses.usp.br/teses/disponiveis/5/5146/tde-02082013-144608/.

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O Streptococcus pyogenes (Grupo A de Lancefield) é uma bactéria Gram positiva e beta-hemolítica, responsável por infecções, tais como Faringite, Sepse, Fasciíte Necrotizante e Síndrome do Choque Tóxico Estreptocócico. Indivíduos suscetíveis podem desenvolver sequela não supurativa auto-imune pós-estreptocócica, como a Febre Reumática, Doença Reumática Cardíaca e a Glomerulonefrite Aguda. A proteína M é o principal antígeno bacteriano. Consiste em aproximadamente 450 resíduos de aminoácidos dispostos em quatro regiões (A, B, C e D), contendo alguns blocos de repetições. As regiões C e D são con

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Noschang, Juliana. "Variabilidade genética de isolados de Streptococcus pyogenes por meio de marcadores RAPD." reponame:Repositório Institucional da UFPR, 2013. http://hdl.handle.net/1884/29830.

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Abstract:

Os estreptococos B-hemoliticos do grupo A (Streptococcus pyogenes) causam varias doencas humanas e estao associados com febre reumatica e glomerulonefrite pos-estreptococica. A sorotipagem dos antigenos M e T tem sido a maneira mais comum de tipagem destes patogenos, entretanto, para alguns isolados a tipagem por este metodo nao e satisfatoria, pois cerca de 15 a 40% dos isolados nao sao tipaveis. Alem disso, estes metodos de sorotipagem nao sao eficientes para diferenciacao clonal em estudos epidemiologicos. Marcadores moleculares sao ferramentas importantes capazes de diferenciar sorotipos d

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