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Journal articles on the topic 'Streptococcus pyogenes'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2025

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1

Gorb, N. N., E. Yu Smertina, A. V. Merck, V. M. Sorokoletova, and M. V. Lazareva. "Resistance to antimicrobials of the microflora extracted in acute postpartum endometritis in cows." Bulletin of NSAU (Novosibirsk State Agrarian University), no. 2 (July 26, 2023): 163–69. http://dx.doi.org/10.31677/2072-6724-2023-67-2-163-169.

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In the article, the authors presented data on the species structure of the microflora isolated from cows with acute postpartum purulent-catarrhal endometritis. In acute postpartum endometritis, representatives of 8 genera of microorganisms were isolated in washings from the cervical canal. Identification of isolates to the species showed that Streptococcus pyogenes, Escherichia coli and Staphylococcus aureus were most often isolated from sick cows. These microorganisms were more common in associations: Escherichia coli + Streptococcus pyogene; Staphylococcus aureus + Streptococcus pyogenes. An

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2

S Hamim, Saad, Khwam R Hussein, and Younus Younus A Kamel. "Molecular profile of scpA and sdaB virulence genes in Streptococcus pyogenes isolated from pharyngitis." AL-QADISIYAH MEDICAL JOURNAL 13, no. 24 (2018): 67–71. http://dx.doi.org/10.28922/qmj.2017.13.24.67-71.

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Group A Streptococci (GAS) or Streptococcus pyogenes is an important pathogen which causes a wide-ranging of diseases for human. This study was carried out in Ear Nose Throat (ENT) department in Al–Habboby Teaching Hospital, Thi-Qar province, south of Iraq during the period from October 2015 to April 2016. Two hundred and ten swabs were collected from patients infected with pharyngitis. 152 (72.3 %) showed positive growth with S. pyogenes. GAS isolates were subjected to detect two virulence genes (scpA and sdaB) by conventional PCR technique using specific primer pairs and DNA sequencing analy

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3

Masaaki, Minami, Akahori Shunsuke, and Ohta Michio. "Amylase from Streptococcus pyogenes inhibits biofilm formation in Streptococcus salivarius." GSC Advanced Research and Reviews 14, no. 2 (2023): 047–53. https://doi.org/10.5281/zenodo.7928101.

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Biofilms are extracellular constituents composed of polysaccharides and other substances necessary for bacteria to defend themselves against foreign enemies. Amylase of <em>Streptococcus pyogenes</em> is an enzyme that degrades polysaccharides and other substances and produces nutrients for invasion of epithelial cells. <em>S. salivarius</em> is an oral commensal bacterium that shows antibacterial activity against <em>S. pyogenes</em>. We investigated the relationship between <em>S. pyogenes</em> and <em>S. salivarius</em> to determine whether the a

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4

SHEFF, BARBARA. "Streptococcus pyogenes." Nursing 32, no. 1 (2002): 75. http://dx.doi.org/10.1097/00152193-200201000-00068.

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5

Fouch, Sarah. "Streptococcus pyogenes." Biomedical & Life Sciences Collection 2024, no. 10 (2024): e1006450. http://dx.doi.org/10.69645/tcic7956.

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6

Isnaeni, Dewi, Andi Ulfah Magefirah Rasyid, and Rahmawati Rahmawati. "Uji Aktivitas Ekstrak Daun Opo-Opo (Desmodium pulchellum Linn Benth) sebagai Antibakteri terhadap Pertumbuhan Streptococcus viridans dan Streptococcus pyogenes." Jurnal Sains dan Kesehatan 3, no. 2 (2021): 278–89. http://dx.doi.org/10.25026/jsk.v3i2.339.

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Streptococcal tonsillopharyngitis is a type of tonsillopharyngitis caused by group A streptococcal bacterial infection with common symptoms such as sore throat, fever more than 38oC, tonsilar exudate, and cervical adenopathy. Possible complications of streptococcal tonsillopharyngitis include rheumatic fever that occurs at weeks 1-5 after acute respiratory infections by Streptococcus viridans and Streptococcus pyogenes. Treatment needs to be done This research aims to determine the value of Minimum Inhibitory Concentration (MIC) and Minimum Kill Concentration (MKC) of Opo-opo leaf extract (Des

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7

Al-Fariz, Rifqi, Edy Kurniawan, Jumari Ustyawati, and Baiq Isti Hijriani. "Uji Efektivitas Ekstrak Batang Lengkuas (Alpinia Galanga) Terhadap Bakteri Streptococcus Pyogenes Secara In Vitro." JSN : Jurnal Sains Natural 2, no. 4 (2024): 129–33. https://doi.org/10.35746/jsn.v2i4.632.

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Pyogenes merupakan bakteri patogen yang dapat menyebabkan penyakit mulai dari infeksi ringan hingga infeksi invasif yang mengancam jiwa. Penyakit yang disebabkan oleh bakteri streptococcus pyogens diobati dengan pemberian antibiotik seperti penisilin. Namun penggunaan antibiotik ini dengan dosis 500 mg dapat menimbulkan bakteri resisten akibat perubahan genetik, sehingga perlu dicari alternatif lain selain Streptococcus seperti menggunakan bahan alami yang berasal dari batang lengkuas misalnya. Batang lengkuas sendiri mengandung saponin, flaponoid, fenol yang berfungsi sebagai antibakteri. Tuj

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8

Komala Hadi, Debbie Rose. "Spektrum Klinis Infeksi Streptococcus Grup A pada Anak." Cermin Dunia Kedokteran 50, no. 11 (2023): 627–31. http://dx.doi.org/10.55175/cdk.v50i11.1009.

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Streptococcus Grup A (SGA) atau Streptococcus pyogenes, merupakan bakteri yang sering menyebabkan infeksi saluran napas dan infeksi kulit pada anak. Organisme ini juga dapat menimbulkan infeksi invasif, yaitu sindrom syok toksik streptococcal dan necrotizing fasciitis; serta berbagai komplikasi dan sekuele, di antaranya demam rematik akut, glomerulonefritis akut pasca-Streptococcus, artritis reaktif pasca-Streptococcus dan pediatric autoimmune neuropsychiatric disorders associated with Streptococcus pyogenes (PANDAS). Peningkatan kasus infeksi SGA, baik invasif maupun non-invasif, dengan punca

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9

Barnham, M. "Streptococcal infection in general practice." Epidemiology and Infection 109, no. 2 (1992): 177–80. http://dx.doi.org/10.1017/s0950268800050135.

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The last 30 years have been changes in emphasis in the study of streptococci and streptococcal diseases. Earlier work concentrated mainly on the sources and methods of cross-infection and descriptive epidemiology of Streptococcus pyogenes in its major manifestations of respiratory, cutaneous and invasive infection and in the complications of rheumatic fever (RF), scarlet fever (SF) and post-streptococcal glomerulonephritis (PSGN).

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10

Masaaki Minami, Shunsuke Akahori, and Michio Ohta. "Amylase from Streptococcus pyogenes inhibits biofilm formation in Streptococcus salivarius." GSC Advanced Research and Reviews 14, no. 2 (2023): 047–53. http://dx.doi.org/10.30574/gscarr.2023.14.2.0050.

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Biofilms are extracellular constituents composed of polysaccharides and other substances necessary for bacteria to defend themselves against foreign enemies. Amylase of Streptococcus pyogenes is an enzyme that degrades polysaccharides and other substances and produces nutrients for invasion of epithelial cells. S. salivarius is an oral commensal bacterium that shows antibacterial activity against S. pyogenes. We investigated the relationship between S. pyogenes and S. salivarius to determine whether the amylase of S. pyogenes affects the biofilm of S. salivarius. The amyA gene-deficient strain

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11

Noori, Ahmad Zia, Haji Mohammad Naimi, and Hashmatullah Yousufi. "The rate of asymptomatic throat carriage of Streptococcus pyogenes and its associated risk factors among Kabul University students." International Journal of Innovative Research and Scientific Studies 3, no. 4 (2020): 142–45. http://dx.doi.org/10.53894/ijirss.v3i4.48.

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Streptococcus pyogenes (S. pyogenes) is the main agent of acute pharyngitis and skin infections that may result in the late complications of glomerulonephritis and rheumatic fever. Infection with streptococcus group A is a global health problem, which is most common in children and adults. This study was conducted to investigate the rate of S. pyogenes throat carriers and its main risk factors among healthy students of Kabul university. In the present study pharyngeal swabs of 260, [155 (59.6%) were male and 105 (40.4%) were female] asymptomatic university students aged between 19-30 years, we

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12

Winterhoff, Nora, Ralph Goethe, Petra Gruening, et al. "Identification and Characterization of Two Temperature-Induced Surface-Associated Proteins of Streptococcus suis with High Homologies to Members of the Arginine Deiminase System of Streptococcus pyogenes." Journal of Bacteriology 184, no. 24 (2002): 6768–76. http://dx.doi.org/10.1128/jb.184.24.6768-6776.2002.

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ABSTRACT The present study was performed to identify stress-induced putative virulence proteins of Streptococcus suis. For this, protein expression patterns of streptococci grown at 32, 37, and 42°C were compared by one- and two-dimensional gel electrophoresis. Temperature shifts from 32 and 37 to 42°C induced expression of two cell wall-associated proteins with apparent molecular masses of approximately 47 and 53 kDa. Amino-terminal sequence analysis of the two proteins indicated homologies of the 47-kDa protein with an ornithine carbamoyltransferase (OCT) from Streptococcus pyogenes and of t

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13

Courtney, Harry S., David L. Hasty, and James B. Dale. "Serum Opacity Factor (SOF) of Streptococcus pyogenes Evokes Antibodies That Opsonize Homologous and Heterologous SOF-Positive Serotypes of Group A Streptococci." Infection and Immunity 71, no. 9 (2003): 5097–103. http://dx.doi.org/10.1128/iai.71.9.5097-5103.2003.

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ABSTRACT Serum opacity factor (SOF) is a protein expressed by Streptococcus pyogenes that opacifies mammalian serum. SOF is also a virulence factor of S. pyogenes, but it has not been previously shown to elicit a protective immune response. Herein, we report that SOF evokes bactericidal antibodies against S. pyogenes in humans, rabbits, and mice. Rabbit antiserum against purified recombinant SOF2 opsonized SOF-positive M type 2, 4, and 28 S. pyogenes in human blood but had no effect on SOF-negative M type 5 S. pyogenes. Furthermore, affinity-purified human antibodies against SOF2 also opsonize

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14

SITKIEWICZ, IZABELA, and WALERIA HRYNIEWICZ. "Pyogenic Streptococci – Danger of Re-Emerging Pathogens." Polish Journal of Microbiology 59, no. 4 (2010): 219–26. http://dx.doi.org/10.33073/pjm-2010-034.

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Beta-hemolytic, pyogenic streptococci are classified according to type of major surface antigen into A (Streptococcus pyogenes), B (Streptococcus agalactiae), C (multiple species including Streptococcus dysagalactiae) and G (multiple species including Streptococcus canis) Lancefield groups. Group A Streptococcus causes each year hundreds of thousands deaths globally as a result of infections and post-infectional sequelae. An increasing number of severe, invasive infections is caused by selected, specialized pathogenic clones. Within the last 50 years, an increasing number of human infections c

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15

Jalava, Jari, Janne Kataja, Helena Seppälä, and Pentti Huovinen. "In Vitro Activities of the Novel Ketolide Telithromycin (HMR 3647) against Erythromycin-Resistant StreptococcusSpecies." Antimicrobial Agents and Chemotherapy 45, no. 3 (2001): 789–93. http://dx.doi.org/10.1128/aac.45.3.789-793.2001.

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ABSTRACT The in vitro susceptibilities of 184 erythromycin-resistant streptococci to a novel ketolide, telithromycin (HMR 3647), were tested. These clinical isolates included 111 Streptococcus pyogenes, 18 group C streptococcus, 18 group G streptococcus, and 37 Streptococcus pneumoniae strains. The MICs for all but eight S. pyogenes strains were ≤0.5 μg/ml, indicating that telithromycin is active in vitro against erythromycin-resistant Streptococcus strains. All strains for which MICs were ≥1 μg/ml had an erm(B) resistance gene and six strains for which MICs were ≥4 μg/ml had a constitutiveerm

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16

ZERVAS, SOPHIA J., LAWRENCE S. ZEMEL, MARK J. ROMNESS, EDWARD L. KAPLAN, and JUAN C. SALAZAR. "Streptococcus pyogenes pyomyositis." Pediatric Infectious Disease Journal 21, no. 2 (2002): 166–68. http://dx.doi.org/10.1097/00006454-200202000-00017.

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17

Mathur, Purva, N. K. Arora, Arti Kapil, and Bimal Das. "Streptococcus pyogenes meningitis." Indian Journal of Pediatrics 71, no. 5 (2004): 423–26. http://dx.doi.org/10.1007/bf02725632.

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18

Norimatsu, Yuta, and Yuki Ohno. "Streptococcus pyogenes balanoposthitis." IDCases 21 (2020): e00832. http://dx.doi.org/10.1016/j.idcr.2020.e00832.

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19

Gilmer, Daniel B., Jonathan E. Schmitz, Chad W. Euler, and Vincent A. Fischetti. "Novel Bacteriophage Lysin with Broad Lytic Activity Protects against Mixed Infection by Streptococcus pyogenes and Methicillin-Resistant Staphylococcus aureus." Antimicrobial Agents and Chemotherapy 57, no. 6 (2013): 2743–50. http://dx.doi.org/10.1128/aac.02526-12.

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ABSTRACTMethicillin-resistantStaphylococcus aureus(MRSA) andStreptococcus pyogenes(group A streptococcus [GrAS]) cause serious and sometimes fatal human diseases. They are among the many Gram-positive pathogens for which resistance to leading antibiotics has emerged. As a result, alternative therapies need to be developed to combat these pathogens. We have identified a novel bacteriophage lysin (PlySs2), derived from aStreptococcus suisphage, with broad lytic activity against MRSA, vancomycin-intermediateS. aureus(VISA),Streptococcus suis,Listeria,Staphylococcus simulans,Staphylococcus epiderm

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20

Talanin, NY, WB Shelley, R. Raeder, ED Shelley, and MD Boyle. "Detection of streptococcal class I M protein in psoriasis by confocal immunofluorescent microscopy." Acta Dermato-Venereologica 77, no. 3 (1997): 175–80. http://dx.doi.org/10.2340/0001555577175180.

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Epidemiological evidence implicates Streptococcus pyogenes (group A) infection as a common triggering stimulus for psoriasis. Unequivocal demonstration of streptococcal antigens in psoriatic skin has been difficult due to cross-reactive antigens in both normal human tissue and group A streptococci, which complicate immunohistological analysis. In this study cryostat sections of involved psoriatic skin were stained with monoclonal antibody 111-15504 to group A streptococci. The epitope recognized by this antibody was found to be specific for group A streptococci and is associated with class I M

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21

Sutcliffe, J., A. Tait-Kamradt, and L. Wondrack. "Streptococcus pneumoniae and Streptococcus pyogenes resistant to macrolides but sensitive to clindamycin: a common resistance pattern mediated by an efflux system." Antimicrobial Agents and Chemotherapy 40, no. 8 (1996): 1817–24. http://dx.doi.org/10.1128/aac.40.8.1817.

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Macrolide-resistant Streptococcus pyogenes isolates from Finland, Australia, and the United Kingdom and, more recently, Streptococcus pneumoniae and S. pyogenes strains from the United States were shown to have an unusual resistance pattern to macrolides, lincosamides, and streptogramin B antibiotics. This pattern, referred to as M resistance, consists of susceptibility to clindamycin and streptogramin B antibiotics but resistance to 14- and 15-membered macrolides. An evaluation of the macrolide-lincosamide-streptogramin B resistance phenotypes among our streptococcal strains collected from 19

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22

DELVECCHIO, A., B. J. CURRIE, J. D. McARTHUR, M. J. WALKER, and K. S. SRIPRAKASH. "Streptococcus pyogenes prtFII, but not sfbI, sfbII or fbp54, is represented more frequently among invasive-disease isolates of tropical Australia." Epidemiology and Infection 128, no. 3 (2002): 391–96. http://dx.doi.org/10.1017/s0950268802006787.

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Streptococcus pyogenes (group A streptococcus) strains may express several distinct fibronectin-binding proteins (FBPs) which are considered as major streptococcal adhesins. Of the FBPs, SfbI was shown in vitro to promote internalization of the bacterium into host cells and has been implicated in persistence. In the tropical Northern Territory, where group A streptococcal infection is common, multiple genotypes of the organism were found among isolates from invasive disease cases and no dominant strains were observed. To determine whether any FBPs is associated with invasive disease propensity

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Valentin-Weigand, Peter, Gursharan S. Chhatwal, and Hans Blobel. "Adherence of streptococcal isolates from cattle and horses to their respective host epithelial cells." American Journal of Veterinary Research 49, no. 9 (1988): 1485–88. https://doi.org/10.2460/ajvr.1988.49.09.1485.

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SUMMARY Adherence of Streptococcus dysgalactiae isolates from cattle and S equi isolates from horses to their respective host epithelial cells was compared with the adherence of S pyogenes to human epithelial cells. The adherence was quantitatively determined by use of fluorescein-labeled streptococci. All 3 streptococcal species adhered selectively to their respective host cells. The mechanism of adherence was evaluated by binding studies with adhesive plasma protein, fibronectin. Although all 3 streptococcal species bound fibronectin, S dysgalactiae and S equi interacted preferentially with

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Betriu, C., E. Culebras, I. Rodríguez-Avial, M. Gómez, B. A. Sánchez, and J. J. Picazo. "In Vitro Activities of Tigecycline against Erythromycin-Resistant Streptococcus pyogenes and Streptococcus agalactiae: Mechanisms of Macrolide and Tetracycline Resistance." Antimicrobial Agents and Chemotherapy 48, no. 1 (2004): 323–25. http://dx.doi.org/10.1128/aac.48.1.323-325.2004.

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ABSTRACT The activity of tigecycline was tested against erythromycin-resistant streptococci (107 Streptococcus pyogenes and 98 Streptococcus agalactiae strains). The presence of erythromycin and tetracycline resistance genes was determined by PCR. Among S. pyogenes strains the most prevalent gene was mef(A) (91.6%). The erm(B) gene was the most prevalent (65.3%) among S. agalactiae strains. Tigecycline proved to be very active against all the isolates tested (MIC at which 90% of the isolates tested were inhibited, 0.06 μg/ml), including those resistant to tetracycline.

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BUDIANTO, NUGROHO EKO WIRAWAN. "Efektivitas Ekstrak Serbuk Batang Siwak Terhadap Pertumbuhan Bakteri Streptococcus Pyogenes." Hang Tuah Medical Journal 18, no. 1 (2020): 100. http://dx.doi.org/10.30649/htmj.v18i1.468.

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<p>Abstract</p><p><strong>Background</strong> : Siwak (Salvadora Persica) is a plant that is widely used in the Middle East which was used to clean teeth and mouth. Currently there is also toothpaste that contains siwak as its active ingredient. Previous research, Siwak has anti-bacterial power against Streptococcus mutans and Streptococcus faecalis. This study aims to prove whether the extract of siwak stem powder can be used to inhibit the growth of Streptococcus pyogenes bacteria, one of which can cause pharyngitis.</p><p><strong>Method:</s

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Azmi, Dini Aulia, Nurlailah Nurlailah, and Ratih Dewi Dwiyanti. "Ethanol Extract Of Centella Asiatica (L.) Urban Leaves Effectively Inhibit Streptococcus pyogenes and Pseudomonas aeruginosa by Invitro Test." Tropical Health and Medical Research 2, no. 2 (2020): 69–76. http://dx.doi.org/10.35916/thmr.v0i0.23.

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Streptococcus pyogenes and Pseudomonas aeruginosa are some of the causes of infectious diseases. Centella asiatica (L.) Urban has many benefits for humans, including overcoming fever, anti-bacterial, and anti-inflammatory. This study aims to determine the inhibition of Centella asiatica (L.) Urban leaves ethanol extract on the growth of Streptococcus pyogenes and Pseudomonas aeruginosa. This research is the initial stage of the development of herbal medicines to treat Streptococcus pyogenes and Pseudomonas aeruginosa infections. The independent variable was the concentration of ethanol extract

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27

Vogel, Verena, and Barbara Spellerberg. "Bacteriocin Production by Beta-Hemolytic Streptococci." Pathogens 10, no. 7 (2021): 867. http://dx.doi.org/10.3390/pathogens10070867.

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Beta-hemolytic streptococci cause a variety of infectious diseases associated with high morbidity and mortality. A key factor for successful infection is host colonization, which can be difficult in a multispecies environment. Secreting bacteriocins can be beneficial during this process. Bacteriocins are small, ribosomally produced, antimicrobial peptides produced by bacteria to inhibit the growth of other, typically closely related, bacteria. In this systematic review, bacteriocin production and regulation of beta-hemolytic streptococci was surveyed. While Streptococcus pyogenes produces eigh

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Burova, L. A., A. N. Suvorov, and A. A. Totolian. "Streptococcus pyogenes: phenomenon of nonimmune binding of human immunoglobulins and its role in pathology." Medical Immunology (Russia) 24, no. 2 (2022): 217–34. http://dx.doi.org/10.15789/1563-0625-spp-2450.

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M and M-like proteins represent the main pathogenicity factors of Streptococcus pyogenes, a widely spread and potentially lethal bacterial pathogen. These proteins provide resistance of the microbe to innate and adaptive immune response, due to attraction of specific human proteins to the streptococcal surface. Nonimmune binding of immunoglobulins G (IgG) and A (IgA) via their Fc domains to M and M-like proteins was described over 40 years ago, but its role for the pathogenicity of Streptococcus pyogenes is far from definite resolution. The discovery of this phenomenon should be considered amo

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Burova, L. A., A. N. Suvorov, and A. A. Totolian. "Streptococcus pyogenes: phenomenon of nonimmune binding of human immunoglobulins and its role in pathology." Medical Immunology (Russia) 24, no. 2 (2022): 217–34. http://dx.doi.org/10.15789/1563-0625-spp-2450.

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M and M-like proteins represent the main pathogenicity factors of Streptococcus pyogenes, a widely spread and potentially lethal bacterial pathogen. These proteins provide resistance of the microbe to innate and adaptive immune response, due to attraction of specific human proteins to the streptococcal surface. Nonimmune binding of immunoglobulins G (IgG) and A (IgA) via their Fc domains to M and M-like proteins was described over 40 years ago, but its role for the pathogenicity of Streptococcus pyogenes is far from definite resolution. The discovery of this phenomenon should be considered amo

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Stinson, Murray W., Robert McLaughlin, Suk Ho Choi, Zaira E. Juarez, and John Barnard. "Streptococcal Histone-Like Protein: Primary Structure ofhlpA and Protein Binding to Lipoteichoic Acid and Epithelial Cells." Infection and Immunity 66, no. 1 (1998): 259–65. http://dx.doi.org/10.1128/iai.66.1.259-265.1998.

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ABSTRACT In addition to its role in the nucleoid, the histone-like protein (HlpA) of Streptococcus pyogenes is believed to act as a fortuitous virulence factor in delayed sequelae by binding to heparan sulfate-proteoglycans in the extracellular matrix of target organs and acting as a nidus for in situ immune complex formation. To further characterize this protein, the hlpA genes were cloned from S. pyogenes, S. gordonii, S. mutans, and S. sobrinus, using PCR amplification, and sequenced. The encoded HlpA protein of S. pyogenes has 91 amino acids, a predicted molecular mass of 9,647 Da, an isoe

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Upton, M., J. R. Tagg, P. Wescombe, and H. F. Jenkinson. "Intra- and Interspecies Signaling betweenStreptococcus salivarius and Streptococcus pyogenes Mediated by SalA and SalA1 Lantibiotic Peptides." Journal of Bacteriology 183, no. 13 (2001): 3931–38. http://dx.doi.org/10.1128/jb.183.13.3931-3938.2001.

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ABSTRACT Streptococcus salivarius 20P3 produces a 22-amino-acid residue lantibiotic, designated salivaricin A (SalA), that inhibits the growth of a range of streptococci, including all strains ofStreptococcus pyogenes. Lantibiotic production is associated with the sal genetic locus comprisingsalA, the lantibiotic structural gene; salBCTXgenes encoding peptide modification and export machinery proteins; andsalYKR genes encoding a putative immunity protein and two-component sensor-regulator system. Insertional inactivation ofsalB in S. salivarius 20P3 resulted in abrogation of SalA peptide produ

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Kombarova, S. Yu, A. M. Bichucher, Y. L. Soldatsky, et al. "Detection of meningococcus, pneumococcus, Haemophilus influenzae, and group A streptococcus DNA in pediatric adenoid bioptats." Russian Journal of Infection and Immunity 10, no. 1 (2020): 111–20. http://dx.doi.org/10.15789/2220-7619-dom-1163.

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Meningococcal, pneumococcal, streptococcal A and Haemophilus influenzae infections are manifested in different clinical forms, ranging from bacterial carriage to generalized life-threatening conditions. However, a connection between bacterial carriage and disease development has not been fully explored. A PCR assay was performed with adenoid biopsy samples collected from 112 children after planned adenotomy to detect Neisseria meningitidis, Streptococcus pneumoniae, Streptococcus pyogenes, H. influenzae carriage. A DNA specific to at least one of the four studied microbial species was found in

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MARHAMAH, MARHAMAH, and Indah Wahyuni Putri. "Efektivitas Ekstrak Batang Pisang Kepok (Musa x paradisiaca Linn.) Terhadap Pertumbuhan Bakteri Streptococcus pyogenes." Jurnal Analis Kesehatan 7, no. 1 (2018): 704. http://dx.doi.org/10.26630/jak.v7i1.932.

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<p>Batang pisang memiliki kandungan antibakteri yaitu tanin, flavonoid, alkaloid, steroid. Berdasarkan penelitian yang dilakukan Marhamah (2014) tentang resistensi bakteri Gram positif salah satunya yaitu <em>Streptococcus. sp</em> didapatkan hasil 100% <em>Streptococcus sp</em> resisten terhadap antibiotik gentamisin, amoksilin, dan cefadroxile. <em>Streptococcus pyogenes</em> merupakan bakteri Gram positif penyebab utama faringitis, kasus faringitis di dunia sebesar 616 juta/ tahun, di Indonesia terjadi pada anak-anak sebesar 18%. Penelitian ini bert

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Ramadhanie, Cynthia Dwi, Sri Purwaningsih, and Eko Budi Koendhori. "Effectivity of Cacao Rind Ethanol Extract in Inhibiting Streptococcus Pyogenes Growth In Vitro." JUXTA: Jurnal Ilmiah Mahasiswa Kedokteran Universitas Airlangga 11, no. 1 (2020): 6. http://dx.doi.org/10.20473/juxta.v11i12020.6-8.

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Introduction: Infectious disease is still a common cause of illness and death in developing countries, such as Indonesia. One of the bacteria that causes infectious disease is Streptococcus pyogenes. Cacao fruit is a large commodity in Indonesia and has benefit for human. Cacao’s rind is known to contain several active compounds such as flavonoid and alkaloid that have antibacterial effect that can inhibit Streptococcus pyogenes growth. This research aims to evaluate the Minimum Bactericidal Concentration (MBC) of cacao rind ethanol extract in inhibiting Streptococcus pyogenes growth in vitro.

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Al-Bermani, Oruba K., Hawraa Al-Kaim, and Ali-Malik Saad. "Pathogenesis of Streptococcus pyogenes and immune response." Al-Kufa University Journal for Biology 12, no. 3 (2020): 34–38. http://dx.doi.org/10.36320/ajb/v12.i3.11796.

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Streptococcus pyogenes is a Gram-positive beta-hemolytic bacteria, also known as group A streptococci, that causes a range of infections. The most common presentation is acute pharyngitis. GAS can be subdivided into >100 serotypes by the M-protein antigen that is located on the cell surface and by fimbriae (hairlike fuzz) that project from the outer edge of the cell. Classically, typing of the surface M protein relied upon available polyclonal antisera. GAS produce and release into the surrounding medium a large number of biologically active extracellular products. Some of these are toxic f

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Sawai, Jun, Tadao Hasegawa, Takuya Kamimura, et al. "Growth Phase-Dependent Effect of Clindamycin on Production of Exoproteins by Streptococcus pyogenes." Antimicrobial Agents and Chemotherapy 51, no. 2 (2006): 461–67. http://dx.doi.org/10.1128/aac.00539-06.

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ABSTRACT The administration of high-dose clindamycin plus benzylpenicillin has been recommended for the treatment of streptococcal toxic shock-like syndrome caused by Streptococcus pyogenes, and clindamycin has been found to be more effective than beta-lactams in retrospective analyses of human cases. Although therapeutic doses of clindamycin have also been shown to be effective against experimental infections and clindamycin has great efficacy against the production of bacterial exoproteins, we recently reported that the level of production of some exoproteins was unchanged or even increased

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Lazarevic, Gordana, Suzana Laban, Milica Jovanovic, and Biljana Potkonjak. "Erythromycin-resistant Streptococcus pyogenes." Srpski arhiv za celokupno lekarstvo 132, suppl. 1 (2004): 42–44. http://dx.doi.org/10.2298/sarh04s1042l.

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Streptococcus pyogenes is the most prevalent cause of tonsillopharyngitis in children. The drug of choice for infections caused by this microorganism is penicillin. The problem of treating such infections arises when erythromycin-resistant strains occur. The aim of the study was to determine the prevalence of Streptococcus pyogenes resistant to erythromycin. The organism was recovered from the pharynx of children hospitalized or treated on outpatient basis at the University Children?s Hospital in Belgrade. Streptococcus pyogenes was identified on blood agar, using bacitracin disc, and confirme

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Datoo, Asma Haji, Kyle Hill, and Josebelo Chong. "738: STREPTOCOCCUS PYOGENES ENDOCARDITIS." Critical Care Medicine 50, no. 1 (2021): 363. http://dx.doi.org/10.1097/01.ccm.0000809276.92529.b8.

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McMurray, J. J., D. M. Fraser, and O. Brogan. "Fatal Streptococcus Pyogenes Pneumonia." Journal of the Royal Society of Medicine 80, no. 8 (1987): 525–26. http://dx.doi.org/10.1177/014107688708000821.

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Kahn, Fredrik, and Magnus Rasmussen. "Penicillin-resistant Streptococcus pyogenes?" FEMS Microbiology Letters 326, no. 1 (2011): 1. http://dx.doi.org/10.1111/j.1574-6968.2011.02447.x.

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Toma, Tudor. "Streptococcus pyogenes genome exposed." Genome Biology 2 (2001): spotlight—20010419–01. http://dx.doi.org/10.1186/gb-spotlight-20010419-01.

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Turner, Claire, and Shiranee Sriskandan. "Streptococcus pyogenes under pressure." Nature Medicine 13, no. 8 (2007): 909–10. http://dx.doi.org/10.1038/nm0807-909.

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Barnham, Michael, and Stig E. Holm. "Serious Streptococcus pyogenes disease." Clinical Microbiology and Infection 3, no. 2 (1997): 250–60. http://dx.doi.org/10.1111/j.1469-0691.1997.tb00606.x.

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MANALO, ROSARIO, HARIS MIRZA, and STEVEN OPAL. "Streptococcus pyogenes Tuboovarian Abscess." Sexually Transmitted Diseases 29, no. 10 (2002): 606–7. http://dx.doi.org/10.1097/00007435-200210000-00007.

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Seal, D. V. "Dysentery and Streptococcus pyogenes." Journal of Infection 17, no. 2 (1988): 185–86. http://dx.doi.org/10.1016/s0163-4453(88)91991-3.

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Thomas, P. S., E. Wilkins, and M. Hickey. "Streptococcus pyogenes and dysentery." Journal of Infection 16, no. 2 (1988): 200–201. http://dx.doi.org/10.1016/s0163-4453(88)94172-2.

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Rahman, M. "Dysentery and Streptococcus pyogenes." Journal of Infection 18, no. 1 (1989): 91–92. http://dx.doi.org/10.1016/s0163-4453(89)93792-4.

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PHILLIPS, G., D. PARRATT, GILLIAN V. ORANGE, I. HARPER, H. McEWAN, and N. YOUNG. "Erythromycin-resistant Streptococcus pyogenes." Journal of Antimicrobial Chemotherapy 25, no. 4 (1990): 723–24. http://dx.doi.org/10.1093/jac/25.4.723.

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Sujhithra, A., S. Jayanthi, M. Chokkalingam, D. Danis Vijay, R. Vidhya, and Sanjay Andrew Rajaratnam. "Streptococcal Pharyngitis and Rheumatic Fever." Journal of Pure and Applied Microbiology 16, no. 1 (2022): 55–62. http://dx.doi.org/10.22207/jpam.16.1.58.

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Streptococcus pyogenes (Group A Streptococcus) causes a variety of diseases, from benign self-limiting infections of the skin or throat to lethal infections of soft tissue accompanied by multi-organ failure. GAS is one of significant species among Gram-positive pathogens which is responsible for several suppurative infections and non-suppurative sequelae. They also cause pharyngitis, streptococcal toxic shock syndrome (STSS), necrotizing fasciitis and other diseases. Currently, global burden of RF / RHD is undervalued. In 2010, RF and RHD were estimated as 15.6 million cases and deaths around

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Loimaranta, V., N. S. Jakubovics, J. Hytönen, J. Finne, H. F. Jenkinson, and N. Strömberg. "Fluid- or Surface-Phase Human Salivary Scavenger Protein gp340 Exposes Different Bacterial Recognition Properties." Infection and Immunity 73, no. 4 (2005): 2245–52. http://dx.doi.org/10.1128/iai.73.4.2245-2252.2005.

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ABSTRACT Salivary scavenger receptor cysteine-rich protein gp340 aggregates streptococci and other bacteria as part of the host innate defense system at mucosal surfaces. In this article, we have investigated the properties of fluid-phase gp340 and hydroxylapatite surface-adsorbed gp340 in aggregation and adherence, respectively, of viridans group streptococci (e.g., Streptococcus gordonii and Streptococcus mutans), non-viridans group streptococci (e.g., Streptococcus pyogenes and Streptococcus suis), and oral Actinomyces. Fluid-phase gp340 and surface-phase gp340 bioforms were differentially

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