Relevant bibliographies by topics / Stress adaptations

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Stress adaptations'

Author: Grafiati
Published: 4 June 2021
Last updated: 15 February 2022

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Journal articles on the topic "Stress adaptations"

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Vartapetian, B. B., and M. B. Jackson. "Plant Adaptations to Anaerobic Stress." Annals of Botany 79, suppl 1 (January 1, 1997): 3–20. http://dx.doi.org/10.1093/oxfordjournals.aob.a010303.

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Mayorga, Edith J., David Renaudeau, Brett C. Ramirez, Jason W. Ross, and Lance H. Baumgard. "Heat stress adaptations in pigs." Animal Frontiers 9, no. 1 (October 30, 2018): 54–61. http://dx.doi.org/10.1093/af/vfy035.

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Paul, Diby. "Osmotic stress adaptations in rhizobacteria." Journal of Basic Microbiology 53, no. 2 (May 14, 2012): 101–10. http://dx.doi.org/10.1002/jobm.201100288.

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Cameron, Heather A., and Timothy J. Schoenfeld. "Behavioral and structural adaptations to stress." Frontiers in Neuroendocrinology 49 (April 2018): 106–13. http://dx.doi.org/10.1016/j.yfrne.2018.02.002.

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Bohnert, Hans J., and Elena Sheveleva. "Plant stress adaptations — making metabolism move." Current Opinion in Plant Biology 1, no. 3 (June 1998): 267–74. http://dx.doi.org/10.1016/s1369-5266(98)80115-5.

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Pierce, Kyle, and Michael H. Stone. "TRAINING ADAPTATIONS: Stress manifestations and training." National Strength & Conditioning Association Journal 8, no. 2 (1986): 26. http://dx.doi.org/10.1519/0744-0049(1986)008<0026:smat>2.3.co;2.

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Barbour, Jayne Alexandra, and Nigel Turner. "Mitochondrial Stress Signaling Promotes Cellular Adaptations." International Journal of Cell Biology 2014 (2014): 1–12. http://dx.doi.org/10.1155/2014/156020.

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Mitochondrial dysfunction has been implicated in the aetiology of many complex diseases, as well as the ageing process. Much of the research on mitochondrial dysfunction has focused on how mitochondrial damage may potentiate pathological phenotypes. The purpose of this review is to draw attention to the less well-studied mechanisms by which the cell adapts to mitochondrial perturbations. This involves communication of stress to the cell and successful induction of quality control responses, which include mitophagy, unfolded protein response, upregulation of antioxidant and DNA repair enzymes, morphological changes, and if all else fails apoptosis. The mitochondrion is an inherently stressful environment and we speculate that dysregulation of stress signaling or an inability to switch on these adaptations during times of mitochondrial stress may underpin mitochondrial dysfunction and hence amount to pathological states over time.
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Prewitt, R. L. "Teaching vascular adaptations to mechanical stress." Advances in Physiology Education 277, no. 6 (December 1999): S211. http://dx.doi.org/10.1152/advances.1999.277.6.s211.

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Blood vessels change their number and structure in attempt to meet tissue demands for blood flow while simultaneously controlling mechanical stresses. A great deal of information is emerging in this field, especially concerning the role of the endothelium and signaling pathways for mechanotransduction. While not delving too deeply into the rapidly changing details, the students can be introduced to this exciting field by describing the structural changes that take place and outlining the major theories that are being investigated. The applications to peripheral vascular disease, myocardial infarctions, hypertension and tumor growth are readily apparent.
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Corkal, D., Harry Diaz, and B. Hurlbert. "Institutional adaptations for long-term adaptations to climate and water stress." IOP Conference Series: Earth and Environmental Science 6, no. 29 (February 1, 2009): 292036. http://dx.doi.org/10.1088/1755-1307/6/29/292036.

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Birkeland, Siri, A. Lovisa S. Gustafsson, Anne K. Brysting, Christian Brochmann, and Michael D. Nowak. "Multiple Genetic Trajectories to Extreme Abiotic Stress Adaptation in Arctic Brassicaceae." Molecular Biology and Evolution 37, no. 7 (March 13, 2020): 2052–68. http://dx.doi.org/10.1093/molbev/msaa068.

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Abstract Extreme environments offer powerful opportunities to study how different organisms have adapted to similar selection pressures at the molecular level. Arctic plants have adapted to some of the coldest and driest biomes on Earth and typically possess suites of similar morphological and physiological adaptations to extremes in light and temperature. Here, we compare patterns of molecular evolution in three Brassicaceae species that have independently colonized the Arctic and present some of the first genetic evidence for plant adaptations to the Arctic environment. By testing for positive selection and identifying convergent substitutions in orthologous gene alignments for a total of 15 Brassicaceae species, we find that positive selection has been acting on different genes, but similar functional pathways in the three Arctic lineages. The positively selected gene sets identified in the three Arctic species showed convergent functional profiles associated with extreme abiotic stress characteristic of the Arctic. However, there was little evidence for independently fixed mutations at the same sites and for positive selection acting on the same genes. The three species appear to have evolved similar suites of adaptations by modifying different components in similar stress response pathways, implying that there could be many genetic trajectories for adaptation to the Arctic environment. By identifying candidate genes and functional pathways potentially involved in Arctic adaptation, our results provide a framework for future studies aimed at testing for the existence of a functional syndrome of Arctic adaptation in the Brassicaceae and perhaps flowering plants in general.
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Dissertations / Theses on the topic "Stress adaptations"

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Smith, Hannah. "Metabolic adaptations to micro-environmental stress in tumour spheroids." Thesis, University of Oxford, 2014. http://ora.ox.ac.uk/objects/uuid:3651d265-ddc0-4258-b3f7-2a0242697d21.

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Alterations in energy metabolism due to factors including cellular stress from the hostile tumour micro-environment are a emerging cancer hallmark. Distinct hypoxic and quiescent cell populations develop, which are resistant to chemotherapy due to lack of proliferation, drug inactivity in the altered redox status of the cell and enhanced drug biotransformation. The present study characterises the metabolic strategies employed by these distinct populations of cancer cells. The in vitro 3-dimensional tumour spheroid model, which reflects tumour architecture and behaviour, cultured under different micro-environmental conditions was utilized in this study. Metabolic enzyme activity and expression, overall metabolic flux rates for nutrients, metabolomics profiles of specific pathways and tissue status were assessed. Metabolic adaptations consistent with the Warburg effect were observed in fully oxygenated, proliferative tumour spheroids, with glucose being metabolised to produce lactate. Additionally, metabolomics investigations determined glucose was metabolised by the pentose phosphate pathway, demonstrated by high enrichment of glucose-derived carbon in 6-phophogluconate. The extraction of 39.7 ± 7.6 μ moles (mg protein) -1 glutamine from the medium over 24 hours was observed in these spheroids, consistent with glutaminolysis pathway activity. A 2-fold higher rate of glycolytic flux (measured by production of 3h2O from 5-3H-glucose) was measured in hypoxic tumour spheroids, despite reduced levels of glycolytic enzymes being determined. Surprisingly, although lower rates of glycolysis (2.6-fold) were measured in quiescent spheroids, increased glycolytic enzyme activities (HK 1.9 fold, PK 2 fold and LDH 1.8 fold), glucose (1.9 fold over 24 hours) and glutamine uptake (5.5 fold over 12 hours) as well as lactate production (1.8 fold) were measured, relative to their proliferating counterparts. This study demonstrates that metabolic strategies employed by tumour spheroids differ upon exposure to distinct micro-environmental stresses, additionally identifying hexokinase as a potential therapeutic target for the inhibition of glycolysis under all micro-environmental stress conditions analysed.
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Singh, François. "Skeletal muscle toxicity and statins : role of mitochondrial adaptations." Thesis, Strasbourg, 2016. http://www.theses.fr/2016STRAJ050/document.

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Bien que les statines forment la classe d'hypolipidémiants la plus utilisée, une toxicité musculaire a été reportée, pouvant ainsi provoquer l’apparition d’une myopathie. Dans la première partie, nous avons montré chez l’Homme et l’animal que les statines inhibent directement la chaine respiratoire mitochondriale, et induisent la production de radicaux libres dérivés de l’oxygène (RLO), qui active les voies apoptotiques dans les muscles glycolytiques, alors que les muscles oxydatifs ne sont pas atteints. Nous avons ensuite montré in vitro que le stress réducteur peut engendrer une oxydation mitochondriale, pouvant conduire à une activation de la voie de biogenèse mitochondriale. De plus l’augmentation du contenu mitochondrial induite a permis de protéger les cellules contre l’apoptose induite par les statines. Enfin, nous avons montré in vivo que l’induction des voies de biogenèse mitochondriale est nécessaire à la tolérance des statines dans les muscles oxydatifs. En conclusion, le phénotype mitochondrial, tant au niveau quantitatif que qualitatif, semble être un facteur clé dans l’apparition de la myopathie aux statines
Although statins are the most prescribed class of lipid-lowering agents, adverse muscular toxicity has been reported, which can lead to the appearance of a myopathy. In the first part, we showed in Humans and animals that statins inhibit directly the mitochondrial respiratory chain, and induce the production of reactive oxygen species (ROS), that trigger apoptotic pathways in glycolytic skeletal muscles, whereas oxidative muscles are not impaired. We then showed in vitro that reductive stress can provoke mitochondrial oxidation, that could lead to an activation of mitochondrial biogenesis pathways. Moreover, the consequent increase in mitochondrial content enabled to protect cells against statin-induced apoptosis. Finally, we showed in vivo that the induction of mitochondrial biogenesis is necessary for statin tolerance in oxidative skeletal muscles. In conclusion, mitochondrial phenotype, both quantitatively and qualitatively, seems to be a key factor in the appearance of statin myopathy
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Desquiret, Valérie. "Mitochondrie et stress énergetique : voies de signalisation et adaptations cellulaires." Phd thesis, Université d'Angers, 2008. http://tel.archives-ouvertes.fr/tel-00433520.

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La mitochondrie est un centre de régulation métabolique à la fois intégrateur de signaux (visant à ajuster son fonctionnement selon les besoins énergétiques cellulaires) et initiateur de voies rétrogrades (permettant une réponse cellulaire à des changements d'états fonctionneles de la mitochondrie). Ce travail s'intéresse plus particulièrement au métabolisme oxydatif mitochondrial et aux voies de signalisation activées, dans les cellules HepG2, lors de deux situations de stress énergétique : le découplage mitochondrial constitue un signal conduisant les cellules à développer leur métabolisme oxydatif sans modifier la glycolyse (notamment par activation de la transcription de gènes codant pour des protéines mitochondriales). La mitochondrie est également une des cibles du traitement par glucocorticoïdes, ces hormones induisant à la fois des effets à court terme et à long terme. les effets rapides (modification de l'activité des complexes I, II et III de la chaîne respiratoire mitochondriale) sont non génomiques et impliquent la fixation de la dexamethasone sur un récepteur membranaire. Ces effets sont médiés par l'activation calcium-dépendante de la protéine p38MAPK. Les effets à long terme (augmentation de la capacité de la chaîne respiratoire) sont transcriptionnels et nécessitent le recrutement du récepteur intracellulaire classique aux glucocorticoïdes. Les modifications du fonctionnement de la chaîne respiratoire mitochondriale par les glucocorticoïdes sont induites par le recrutement graduel de différents sites de liaison aux glucocorticîdes (membranaire et intracellulaire).
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Tinken, Toni Marie. "Exercise Training, Shear Stress and Arterial Adaptations in Healthy Humans." Thesis, Liverpool John Moores University, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.515355.

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Jacobsen, Anna Linden. "Woody plant adaptations to water stress in arid shrub communities." Diss., Connect to online resource - MSU authorized users, 2008.

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Henshaw, Thomas L. "Morphological adaptations of soybean in response to early season flood stress." [Gainesville, Fla.] : University of Florida, 2005. http://purl.fcla.edu/fcla/etd/UFE0011761.

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Swanson, Andrew K. "Ecophysiological adaptations of northeast Pacific kelp communities to ultraviolet light stress." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape2/PQDD_0022/NQ51923.pdf.

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Xie, Guohao. "Metabolic and endocrine adaptations to heat stress in lactating dairy cows." Diss., Virginia Tech, 2015. http://hdl.handle.net/10919/52903.

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Heat stress (HS), a stress response in homeotherms mainly due to elevated ambient temperature and failure of effective heat dissipation, causes a substantial negative economic impact to livestock industry worldwide. Reduced feed intake, a typical phenomenon observed during HS, was thought to be the primary driver for the milk production loss. However, accumulating evidence indicates that HS influences animal metabolism and endocrine profiles independent of reduced feed intake. Previous studies comparing heat-stressed lactating cows with control group pair-fed (PF) to the intake of HS group but housed in thermoneutral conditions, in order to eliminate the confounding factors result from differentiated feed intakes, showed that HS increased circulating insulin and decreased plasma non-esterified fatty acid (NEFA) in lactating cow, the opposite responses typical of PF cohorts. Therefore, the present studies were performed in order to elucidate the mechanism(s) underlying these counterintuitive changes. In response to a glucose tolerance test (GTT), both HS and PF decreased whole body glucose disposal rate, a sign of insulin resistance. Only PF decreased skeletal muscle insulin sensitivity in terms of reduced protein kinase B (PKB/AKT) phosphorylation, a downstream protein of insulin receptor (IR), while HS group maintained similar intact insulin responsiveness in the liver and skeletal muscle as thermoneutral conditions. There was a global reduction in gene expression of the enzymes related to lipid metabolism in adipose tissue of heat-stressed cows. Similarly, β-adrenergic signaling, a major stimulator of lipid mobilization, was suppressed in terms of NEFA release response during a chronic epinephrine challenge in HS group. After the challenge, phosphorylations of cAMP-response element binding protein (CREB) and hormone sensitive lipase, both located downstream of β-adrenergic receptor, were decreased in HS, but not in thermoneutral conditions, another indicator of impaired adrenergic signaling. In contrast, IR and AKT phosphorylation were increased in HS conditions indicating insulin signaling may be elevated during HS in adipose. Collectively, HS reduces lipid mobilization and appears to favor glucose utilization via alterations of lipid metabolism and hormones signaling pathways. These unique alterations in HS might shed some light on developing counter-HS approaches in the future.
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Chen, Cheng-Ping. "Physiological and biochemical adaptations to cold and heat stress in flesh flies (Diptera:sacrophagidae) /." The Ohio State University, 1990. http://rave.ohiolink.edu/etdc/view?acc_num=osu1487678444256714.

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Hardman, Adrianne E. "Human adaptations to endurance training : their influence on the ability to sustain submaximum exercise." Thesis, Loughborough University, 1994. https://dspace.lboro.ac.uk/2134/14405.

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The purpose of the study was to examine the proposition that adaptations of skeletal muscle are more important determinants of the ability to sustain submaximum exercise than maximum oxygen uptake (V0₂ max). The metabolic response to exercise was compared in 2 groups of runners with differing V0₂ max values. Trained female runners, despite their lower V0₂ max values, showed lower respiratory exchange ratio values (R) and higher post-exercise concentrations of plasma fat metabolites than active male subjects. This was so when subjects ran at a common absolute speed and when each individual ran at a speed equivalent to 60% V0₂ max. Six weeks of training on the cycle ergometer resulted in a 16% increase in V0₂ max but a 250% increase in endurance performance, i.e. time to exhaustion at 80% V0₂ max. During standard, submaximum exercise oxygen uptake, ventilation, heart rate, R values and blood lactate concentration were lower post-training than pre-training. The concentrations of plasma fat metabolites during and after exercises were not influenced by training. Training one leg and not the other resulted in 20% and 10% increase in V0₂ max but 340% and 150% increases in endurance for the trained leg (TL) and the untrained leg (UTL) respectively. The increases in both variables were significantly greater for the TL than for the UTL. Changes in submaximum V0₂, ventilation, R values and blood lactate concentration were mainly restricted to exercise with the TL but the reduction in heart rate was similar for each leg. Plasma FFA, glycerol and citrate concentrations during exercise were unchanged for either leg. The role of citrate in the training-induced interaction of fat and carbohydrate metabolism was investigated. Artificial elevation of plasma citrate concentrations by ingestion of citrate did not decrease blood lactate concentrations during exercise. After short-term fasting citrate accumulated in the myocardium but not in skeletal muscles in the rat. Plasma citrate concentration was a poor predictor of muscle citrate concentration in these circumstances. When subjects were exhausted after single-leg exercise at 80% V0₂ max muscle glycogen concentration decreased by 70% and muscle lactate concentration was increased 10-fold. Training halved the rate of glycogen depletion, decreased muscle lactate concentration and resulted in a decrease in R values only during exercise with the TL. The trend towards an increase in muscle citrate concentration was restricted to trained muscle. The experiments showed that absolute values of V0₂ max could be dissociated from the metabolic characteristics of endurance training and that V0₂ max is an insensitive indicator of training improvements in submaximum endurance. Peripheral adaptations are the dominant influence on the ability to sustain submaximum exercise but training only one leg can cause some systemic change which influences both V0₂ max and endurance for the UTL. The mechanism of the training-induced interaction of fat and carbohydrate metabolism remains unclear.
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Books on the topic "Stress adaptations"

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Azooz, M. M. Legumes under environmental stress: Yield, improvement and adaptations. Hoboken, NJ: John Wiley & Sons Inc., 2015.

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Zev, Harel, and Kahana Eva, eds. Holocaust survivors and immigrants: Late life adaptations. New York, NY: Springer, 2005.

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Depression, stress, and adaptations in the elderly: Psychological assessment and intervention. Rockville, Md: Aspen Publishers, 1986.

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Ahmad, Parvaiz, and M. N. V. Prasad, eds. Environmental Adaptations and Stress Tolerance of Plants in the Era of Climate Change. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4614-0815-4.

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Prasad, M. N. V., and Parvaiz Ahmad. Environmental adaptations and stress tolerance of plants in the era of climate change. New York: Springer New York, 2012.

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Randall, Denise Justus. Psychological adaptations in life & work: Subject analysis index with reference bibliography. Washington, D.C: ABBE Publishers Association, 1987.

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Timiras, Paola. Stress, adaptation, longévité. Paris: Economica, 2004.

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Endrőczi, Elemér. Stress and adaptation. Budapest: Akadémiai Kiadó, 1991.

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Environmental stress and behavioural adaptation. London: Croom Helm, 1985.

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Wilson, John P., Zev Harel, and Boaz Kahana, eds. Human Adaptation to Extreme Stress. Boston, MA: Springer US, 1988. http://dx.doi.org/10.1007/978-1-4899-0786-8.

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Book chapters on the topic "Stress adaptations"

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Treherne, J. E. "Neuronal Adaptations to Osmotic Stress." In Proceedings in Life Sciences, 376–88. Berlin, Heidelberg: Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-70613-4_32.

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Faseela, Parammal, Asari Ki Sinisha, Thuruthummel Thomas Dhanya Thomas, and Jos Thomas Puthur. "Oxidative Stress and Its Management in Plants During Abiotic Stress." In Metabolic Adaptations in Plants During Abiotic Stress, 111–26. Boca Raton, FL : CRC Press, Taylor & Francis Group, 2018.: CRC Press, 2018. http://dx.doi.org/10.1201/b22206-10.

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Miczek, Klaus A., Michael L. Thompson, and Walter Tornatzky. "Short and Long Term Physiological and Neurochemical Adaptations to Social Conflict." In Psychobiology of Stress, 15–30. Dordrecht: Springer Netherlands, 1990. http://dx.doi.org/10.1007/978-94-009-1990-7_2.

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Pradhan, Chinmay, and Monalisa Mohanty. "Submergence Stress: Responses and adaptations in crop plants." In Molecular Stress Physiology of Plants, 331–57. India: Springer India, 2013. http://dx.doi.org/10.1007/978-81-322-0807-5_14.

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Blokhina, Olga, and Kurt V. Fagerstedt. "Oxygen Deprivation, Metabolic Adaptations and Oxidative Stress." In Waterlogging Signalling and Tolerance in Plants, 119–47. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-10305-6_7.

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SINGH, S. S., PANKAJ KUMAR, and ASHWANI K. RA. "Ultraviolet radiation stress: molecular and physiological adaptations in trees." In Abiotic stress tolerance in plants, 91–110. Dordrecht: Springer Netherlands, 2006. http://dx.doi.org/10.1007/1-4020-4389-9_6.

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Limami, Anis M. "Adaptations of Nitrogen Metabolism to Oxygen Deprivation in Plants." In Low-Oxygen Stress in Plants, 209–21. Vienna: Springer Vienna, 2013. http://dx.doi.org/10.1007/978-3-7091-1254-0_11.

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Jha, Shweta. "Proteomics of Salinity Stress: Opportunities and Challenges." In Metabolic Adaptations in Plants During Abiotic Stress, 285–92. Boca Raton, FL : CRC Press, Taylor & Francis Group, 2018.: CRC Press, 2018. http://dx.doi.org/10.1201/b22206-25.

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Nair, Sudha K., Pervez Haider Zaidi, Madhumal Thayil Vinayan, and Gajanan Saykhedkar. "Physiological and molecular mechanisms underlying excess moisture stress tolerance in maize: molecular breeding opportunities to increase yield potential." In Molecular breeding in wheat, maize and sorghum: strategies for improving abiotic stress tolerance and yield, 295–317. Wallingford: CABI, 2021. http://dx.doi.org/10.1079/9781789245431.0017.

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Abstract Understanding the impact of excess moisture (EM) on maize plants at various growth stages, and studying the phenological, physiological and molecular responses of tolerant maize genotypes towards adaptation to EM stress, could help define ways in which this trait could be improved through targeted breeding. Thus, this chapter discusses the (i) impact of EM stress on maize plants, (ii) phenological adaptations and physiological mechanisms leading to EM stress tolerance in maize, and (iii) molecular signature of EM stress tolerance. Genetic studies on EM stress tolerance in maize are presented, and the application of molecular mreeding for EM tolerance in maize is described.
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Fatnassi, Nadia, Ralph Horres, Natasa Cerekovic, Angelo Santino, and Palmiro Polronieri. "Differences in Adaptation to Water Stress in Stress Sensitive and Resistant Varieties of Kabuli and Desi Type Chickpea." In Metabolic Adaptations in Plants During Abiotic Stress, 403–12. Boca Raton, FL : CRC Press, Taylor & Francis Group, 2018.: CRC Press, 2018. http://dx.doi.org/10.1201/b22206-34.

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Conference papers on the topic "Stress adaptations"

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Dai, Tao, Aaron Novickis, Eriko Katsuta, Kazuaki Takabe, Scott I. Abrams, and Subhamoy Dasgupta. "Abstract PO022: Metabolic and transcriptional adaptations under hypoxic stress promote breast cancer progression." In Abstracts: AACR Virtual Special Conference: The Evolving Tumor Microenvironment in Cancer Progression: Mechanisms and Emerging Therapeutic Opportunities; in association with the Tumor Microenvironment (TME) Working Group; January 11-12, 2021. American Association for Cancer Research, 2021. http://dx.doi.org/10.1158/1538-7445.tme21-po022.

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Humphrey, J. D. "Constrained Mixture Models of Arterial Homeostasis and Adaptation." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-204922.

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Phenomenological models of the mechanical behavior of the arterial wall continue to play important roles in vascular mechanics. Indeed, such models revealed the importance of residual stresses in homogenizing the transmural distribution of stress in normalcy [1], which in turn led to one of the most important hypotheses in vascular mechanobiology — the existence of a mechanical homeostasis [2]. Nevertheless, classical models are not able to exploit the growing information on the different mechanical properties and rates and extents of turnover of different structurally significant constituents within the arterial wall. To address this need, we have proposed a structurally-motivated, materially nonuniform model of the arterial wall based on a theory of constrained mixtures [cf. 3]. Key features of this model include the ability to prescribe individual stored energy functions for different structurally significant constituents that are constrained to move together within the overall wall while being allowed to possess individual evolving natural (stress-free) configurations, and the ability to prescribe separate stress-mediated constitutive relations for constituent production and removal. We have shown that this constrained mixture approach can capture many salient features of arterial adaptations (e.g., evolving changes in geometry, overall material behavior, and collagen to elastin ratios) to both altered mechanical loading (e.g., altered blood flow and pressure as well as axial stretch) and disease progression (e.g., enlargement of intracranial aneurysms and the development and resolution of cerebral vasospasm). We submit that, in contrast to models built on the assumption of kinematic growth, this constrained mixture approach can incorporate increasingly detailed biological information on cell and matrix turnover and can thereby begin to generate and test novel hypotheses on mechanisms of arterial homeostasis and adaptation. We will show, for example, that the constrained mixture model suggests a possible mechanism for the origin of residual stresses and axial prestretches, the importance of cellular deposition of new extracellular matrix proteins within stressed states, the complementary roles of vasoactivity and matrix remodeling, and stress mediation of matrix turnover [4,5]. We note, in particular, the importance of residual stress and axial prestress in establishing arterial homeostasis and thus targets for subsequent adaptations [5,6].
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Humphrey, J. D. "Importance of Axial Stress in Arterial Growth and Remodeling." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192070.

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Since the mid-1970s, we have continued to understand better the fundamental importance of mechanotransduction in vascular biology. For example, beginning with Rosen and colleagues in 1974, we discovered that endothelial cells alter their production of vasoactive molecules in response to changes in flow-induced wall shear stress; beginning with Glagov and colleagues in 1976, we discovered that vascular smooth muscle cells alter their production of extracellular matrix proteins in response to changes in cyclic stretching comparable to that induced by pulsatile pressures [1]. Indeed, such findings are not surprising given the well know arterial adaptations that occur in response to sustained changes in blood flow or pressure. The caliber of an artery tends to increase (or decrease) in response to sustained increases (decreases) in blood flow and the thickness of the arterial wall tends to increase (or decrease) in response to sustained increases (decreases) in blood pressure. In both cases, it appears that the associated wall shear or intramural circumferential stresses are returned toward normal following the change in hemodynamics.
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Gupta-Vallur, Piyushi, Yeon Soo Kim, Dong-Hui Shin, LP Madhubhani Hemachandra, Rebecca Phaeton, Karthikeyan Mythreye, and Nadine Hempel. "Abstract GMM-029: MITOCHONDRIAL STRESS RESPONSE ADAPTATIONS ARE REQUIRED FOR OVARIAN CANCER ANCHORAGE-INDEPENDENT SURVIVAL AND METASTASIS." In Abstracts: 12th Biennial Ovarian Cancer Research Symposium; September 13-15, 2018; Seattle, Washington. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1557-3265.ovcasymp18-gmm-029.

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Rennie, Monique Y., Michael Danilchik, Kent L. Thornburg, and Sandra Rugonyi. "Hemodynamic Forces Regulate Collagen Deposition in the Embryonic Chicken Outflow Tract." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14299.

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Alterations in blood flow at early embryonic stages can lead to detrimental remodeling and heart defects, but these structural adaptations are not well understood. We hypothesize that deposition of collagens will be increased as shear stress is increased — leading to a stiffer wall. To test this hypothesis a suture (OTB) was tightened around the outflow tract (OFT) of stage HH18 chick embryos for 24 hours to reduce cross sectional area of the lumen. Sham and OTB embryos were immunostained for collagen I, III, VI and XIV, imaged with confocal microscopy, and staining was quantified by grayscale analysis. Changes in fibril collagens I and III were not observed, however deposition of collagens VI and XIV increased in a degree-of-constriction dependent manner. The observed increase in collagen VI and XIV deposition suggests they play a key role in structural adaptation to increased hemodynamic pressure.
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Franek, Friedrich, Andreas Pauschitz, Vladislav E. Lazarev, Georg Vorlaufer, Thomas Dick, and Robin Jisa. "Complex Micromodel Analysis of Wearing Contact Interfaces." In World Tribology Congress III. ASMEDC, 2005. http://dx.doi.org/10.1115/wtc2005-63540.

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The behavior of tribological systems with hybrid lubrication using solid lubricants on graphite basis and/or liquid lubricant can be optimized. This can be achieved by a detailed knowledge of the transfer mechanisms induced by the tribological stress and consequential material and surface design adaptations. Our first approach is a finite element model containing a representative model of the real asperity geometry and distribution. With this model we can estimate the temperature and pressure distribution in the contact zone. The model is based on an assembly of volumes which limit the surfaces taking part in heat exchange and load transfer. Each surface has a set of equations for heat exchange and mechanical loading in the contact. Similarly, transport mechanisms concerning the liquid lubricant, solid lubricant particles and the dissemination of wear particles are considered.
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Lenkov, Sergey, and Nadezhda Rubtsova. "Involvement into Cyber-Socialisation as a New Factor of Psychological Well-Being." In The Public/Private in Modern Civilization, the 22nd Russian Scientific-Practical Conference (with international participation) (Yekaterinburg, April 16-17, 2020). Liberal Arts University – University for Humanities, Yekaterinburg, 2020. http://dx.doi.org/10.35853/ufh-public/private-2020-37.

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The study is underpinned by a new author’s approach to understanding the involvement into cyber-socialisation as an integral psychological construct consisting of two relatively autonomous components: positive and negative involvement. The aim of this study was to identify relationships between youth engagement in cybersocialisation and the heterogeneous measures of a psychological well-being. For measurements the authors used the author’s ‘Questionnaire of involvement in cybersocialisation’, as well as Russian-language adaptations of the ‘Scale of psychological well-being’ C. D. Ryff, ‘Life satisfaction scale’ by E. Diener et al., and ‘Perceptible stress scale’ by S. Cohen & G. M. Williamson. The sample consisted of 268 persons aged 17 to 30 years, including 143 males (53.4 %) and 125 females (46.6 %), 131 employees in various organisations (48.9 %) and 137 full-time university and college students (51.1 %). Using an analysis of variance and regression, constructive engagement in cybersocialisation was found to increase indicators of psychological well-being and life satisfaction, and to decrease indicators of perceived stress, while destructive engagement in cybersocialisation had the opposite, negative impact on many of these indicators, and a significantly greater impact than constructive engagement. The findings confirm the ambivalent nature of cybersocialisation of contemporary youth and determine the prospects for a more detailed study of the structure and consequences of cybersocialisation processes in the developing information society of modern civilisation.
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Etter, Karen E., and M. Keith Sharp. "Modeling of Orthostatic Intolerance During Lower Body Negative Pressure." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19053.

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Postflight orthostatic intolerance (POI) afflicts a significant fraction of male astronauts and nearly all female astronauts when they first stand on Earth after spaceflight. Symptoms include dizziness and fainting, which can impact their abilities to perform critical tasks during the post-landing period. On the Moon or Mars, poor performance or accidents resulting from POI may have potentially catastrophic consequences due to the more hazardous conditions and lack of medical facilities. In addition, the long duration flights necessary to reach Mars may elicit adaptations that increase the risk of POI. Of the many factors that may influence POI, one of the few that may explain the gradual decline in arterial blood pressure experienced by many subjects during stand tests is the loss of blood volume by capillary filtration. Previous simulations suggest that elevated capillary filtration rates distinguish nonfinishers from finishers of stand tests [1, 2]. In this investigation, further computer modeling was undertaken to compare modeled orthostatic response to that of volunteers during graded orthostatic stress (GOS) involving head up tilt (HUT) and lower body negative pressure (LBNP), which reliably produces presyncope and is a candidate training countermeasure for preventing POI.
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Torkamaan, Helma, and Jürgen Ziegler. "Rating-based Preference Elicitation for Recommendation of Stress Intervention." In UMAP '19: 27th Conference on User Modeling, Adaptation and Personalization. New York, NY, USA: ACM, 2019. http://dx.doi.org/10.1145/3320435.3324990.

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10

Saeed, Aaqib, Tanir Ozcelebi, Johan Lukkien, Jan B. F. van Erp, and Stojan Trajanovski. "Model Adaptation and Personalization for Physiological Stress Detection." In 2018 IEEE 5th International Conference on Data Science and Advanced Analytics (DSAA). IEEE, 2018. http://dx.doi.org/10.1109/dsaa.2018.00031.

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Reports on the topic "Stress adaptations"

1

Wyndham, Amber, Emile Elias, Joel R. Brown, Michael A. Wilson, and Albert Rango. Drought Vulnerability Assessment to Inform Grazing Practices on Rangelands in Southeast Arizona and Southwest New Mexico’s Major Land Resource Area 41. United States. Department of Agriculture. Southwest Climate Hub, August 2018. http://dx.doi.org/10.32747/2018.6818230.ch.

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Increased climate variability, including more frequent and intense drought, is projected for the southwestern region of the United States. Increased temperatures and reduced precipitation lower soil water availability, resulting in decreased plant productivity and altered species composition, which may affect forage quality and quantity. Reduced forage quality and increased heat stress attributable to warmer temperatures could lead to decreased livestock performance in this system, which is extensively used for livestock grazing. Mitigating the effects of increasing drought is critical to social and ecological stability in the region. Reduced stocking rates and/or a change in livestock breeds and/or grazing practices are general recommendations that could be implemented to cope with increased climatic stress. Ecological Sites (ESs) and their associated state-and-transition models (STMs) are tools to help land managers implement and evaluate responses to disturbances. The projected change in climate will vary depending upon geographic location. Vulnerability assessments and adaptation strategies are necessary at the local level to inform local management decisions and help to ameliorate the effects of climate change on rangelands. The USDA Southwest Climate Hub and the Natural Resources Conservation Service (NRCS) worked together to produce this drought vulnerability assessment at the Major Land Resource Area (MLRA) level: it is based on ESs/STMs that will help landowners and government agencies to identify and develop adaptation options for drought on rangelands. The assessment illustrates how site-specific information can be used to help minimize the effects of drought on rangelands and to support informed decision-making for selecting management adaptations within MLRA 41.
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2

Wyndham, Amber, Emile Elias, Joel R. Brown, Michael A. Wilson, and Albert Rango. Drought Vulnerability Assessment to Inform Grazing Practices on Rangelands of Southeastern Colorado’s Major Land Resource Area 69. United States. Department of Agriculture. Southwest Climate Hub, January 2018. http://dx.doi.org/10.32747/2018.6876399.ch.

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Increased climate variability, including more frequent and intense drought, is projected for the southwestern region of the United States. Increased temperatures and reduced precipitation lower soil water availability resulting in decreased plant productivity and altering species composition which may affect forage quality and quantity. Reduced forage quality and increased heat stress attributable to warmer temperatures could lead to decreased livestock performance in this system, which is extensively used for livestock grazing. Mitigating the effects of increasing drought is critical to social and ecological stability in the region. Reduced stocking rates, change in livestock breeds and/or grazing practices are general recommendations that could be implemented to cope with increased climatic stress. Ecological Sites (ESs) and their associated state and transition models (STMs) are tools to help land managers implement and evaluate responses to disturbances. The projected change in climate will vary depending on geographic location. Vulnerability assessments and adaptation strategies are needed at the local level to inform local management decisions and help ameliorate the effects of climate change on rangelands. The USDA Southwest Climate Hub and Natural Resources Conservation Service (NRCS) worked together to produce this drought vulnerability assessment at the Major Land Resource Area (MLRA) level based on ESs/STMs that will help landowners and government agencies identify and develop adaptation options for drought on rangelands. The assessment illustrates how site-specific information can be used to help minimize the effects of drought on rangelands and support informed decision-making for selecting management adaptations within MLRA 69.
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3

Wyndham, Amber, Emile Elias, Joel Brown, Michael Wilson, and Albert Rango Rango. Drought Vulnerability Assessment to Inform Grazing Practices on Rangelands in Southeast Arizona and Southwest New Mexico’s Major Land Resource Area 41. USDA Southwest Climate Hub, August 2018. http://dx.doi.org/10.32747/2018.6947060.ch.

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Increased climate variability, including more frequent and intense drought, is projected for the southwestern region of the United States. Increased temperatures and reduced precipitation lower soil water availability, resulting in decreased plant productivity and altered species composition, which may affect forage quality and quantity. Reduced forage quality and increased heat stress attributable to warmer temperatures could lead to decreased livestock performance in this system, which is extensively used for livestock grazing. Mitigating the effects of increasing drought is critical to social and ecological stability in the region. Reduced stocking rates and/or a change in livestock breeds and/or grazing practices are general recommendations that could be implemented to cope with increased climatic stress. Ecological Sites (ESs) and their associated state-and-transition models (STMs) are tools to help land managers implement and evaluate responses to disturbances. The projected change in climate will vary depending upon geographic location. Vulnerability assessments and adaptation strategies are necessary at the local level to inform local management decisions and help to ameliorate the effects of climate change on rangelands. The USDA Southwest Climate Hub and the Natural Resources Conservation Service (NRCS) worked together to produce this drought vulnerability assessment at the Major Land Resource Area (MLRA) level: it is based on ESs/STMs that will help landowners and government agencies to identify and develop adaptation options for drought on rangelands. The assessment illustrates how site-specific information can be used to help minimize the effects of drought on rangelands and to support informed decision-making for selecting management adaptations within MLRA 41.
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4

Wyndham, Amber, Emile Elias, Joel Brown, Michael Wilson, and Albert Rango. Drought Vulnerability Assessment to Inform Grazing Practices on Rangelands of Southeastern Colorado’s Major Land Resource Area 69. USDA Southwest Climate Hub, July 2018. http://dx.doi.org/10.32747/2018.6947062.ch.

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Increased climate variability, including more frequent and intense drought, is projected for the southwestern region of the United States. Increased temperatures and reduced precipitation lower soil water availability, resulting in decreased plant productivity and altering species composition, which may affect forage quality and quantity. Reduced forage quality and increased heat stress attributable to warmer temperatures could lead to decreased livestock performance in this system, which is extensively used for livestock grazing. Mitigating the effects of increasing drought is critical to social and ecological stability in the region. Reduced stocking rates, change in livestock breeds and/or grazing practices are general recommendations that could be implemented to cope with increased climatic stress. Ecological Sites and their associated state–and-transition models (STMs) are tools to help land managers implement and evaluate responses to disturbances. The projected change in climate will vary depending upon geographic location. Vulnerability assessments and adaptation strategies are needed at the local level to inform local management decisions and help ameliorate the effects of climate change on rangelands. The United States Department of Agriculture (USDA) Southwest Climate Hub and Natural Resources Conservation Service (NRCS) worked together to produce this drought vulnerability assessment at the Major Land Resource Area (MLRA) level, based on ecological sites and state-and-transition models that will help landowners and government agencies to identify and develop adaptation options for drought on rangelands. The assessment illustrates how site-specific information can be used to help minimize the effects of drought on rangelands and support informed decision-making for the selection of management adaptations within MLRA 69.
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5

Wyndham, Amber, Emile Elias, Joel Brown, Michael Wilson, and Albert Rango. Drought Vulnerability Assessment to Inform Grazing Practices on Rangelands of Southeastern Colorado’s Major Land Resource Area 69. USDA Southwest Climate Hub, March 2018. http://dx.doi.org/10.32747/2018.6965584.ch.

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Abstract:
Increased climate variability, including more frequent and intense drought, is projected for the southwestern region of the United States. Increased temperatures and reduced precipitation lower soil water availability resulting in decreased plant productivity and altering species composition which may affect forage quality and quantity. Reduced forage quality and increased heat stress attributable to warmer temperatures could lead to decreased livestock performance in this system, which is extensively used for livestock grazing. Mitigating the effects of increasing drought is critical to social and ecological stability in the region. Reduced stocking rates, change in livestock breeds and/or grazing practices are general recommendations that could be implemented to cope with increased climatic stress. Ecological Sites (ESs) and their associated state and transition models (STMs) are tools to help land managers implement and evaluate responses to disturbances. The projected change in climate will vary depending on geographic location. Vulnerability assessments and adaptation strategies are needed at the local level to inform local management decisions and help ameliorate the effects of climate change on rangelands. The USDA Southwest Climate Hub and Natural Resources Conservation Service (NRCS) worked together to produce this drought vulnerability assessment at the Major Land Resource Area (MLRA) level based on ESs/STMs that will help landowners and government agencies identify and develop adaptation options for drought on rangelands. The assessment illustrates how site-specific information can be used to help minimize the effects of drought on rangelands and support informed decision-making for selecting management adaptations within MLRA 69.
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6

Zhou, Aifen, Kristina Hillesland, Zhili He, Marcin Joachimiak, Grant Zane, Paramvir Dehal, Adam Arkin, et al. Genetic Adaptation to Salt Stress in Experimental Evolution of Desulfovibrio vulgaris Hildenborough. Office of Scientific and Technical Information (OSTI), May 2010. http://dx.doi.org/10.2172/985929.

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7

Bestler, C., and R. Stewart, eds. Stream Control Transmission Protocol (SCTP) Direct Data Placement (DDP) Adaptation. RFC Editor, October 2007. http://dx.doi.org/10.17487/rfc5043.

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8

Maston, V. A. Adaptation of a commercially available 200 kW natural gas fuel cell power plant for operation on a hydrogen rich gas stream. Office of Scientific and Technical Information (OSTI), December 1997. http://dx.doi.org/10.2172/670180.

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