Log in

Relevant bibliographies by topics / Stress hematopoiesis

Contents

Journal articles
Dissertations / Theses
Books
Book chapters
Conference papers

Academic literature on the topic 'Stress hematopoiesis'

Author: Grafiati

Published: 24 May 2025

Last updated: 31 July 2025

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the lists of relevant articles, books, theses, conference reports, and other scholarly sources on the topic 'Stress hematopoiesis.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Journal articles on the topic "Stress hematopoiesis"

1

Link, Daniel C. "Clonal Evolution During Stress Hematopoiesis." Blood 130, Suppl_1 (2017): SCI—38—SCI—38. http://dx.doi.org/10.1182/blood.v130.suppl_1.sci-38.sci-38.

Full text

Abstract:

Hematopoietic stem and progenitor cells (HSPCs) acquire somatic mutations with age resulting in a heterogeneous cell population, with each HSPC possessing its own unique set of private mutations. HSPCs that acquire somatic mutations that confer a competitive fitness advantage relative to their normal counterparts may clonally expand. Indeed, several groups have documented the presence of clonal hematopoiesis in healthy individuals. Although originally thought to be limited to older individuals, a recent study using an ultra-sensitive sequencing technique showed that expanded hematopoietic clon

APA, Harvard, Vancouver, ISO, and other styles

2

Chen, Ying-Ying, Yu-Feng Liu, Yong-Dong Liu, Xiao-Hui Deng, and Jie Zhou. "IRF7 suppresses hematopoietic regeneration under stress via CXCR4." Stem Cells 39, no. 2 (2020): 183–95. http://dx.doi.org/10.1002/stem.3308.

Full text

Abstract:

Abstract Hematopoietic stem cells (HSCs) maintain quiescence under steady state; however, they are compelled to proliferate and expand to replenish the blood system under stress. The molecular basis underlying stress hematopoiesis remains to be fully understood. In this study, we reported that IRF7 represents an important regulator of stress hematopoiesis. Interferon regulatory factor 7 (IRF7) was dispensable for normal hematopoiesis, whereas its deficiency significantly enhanced hematopoietic stem and progenitor cells (HSPCs) regeneration and improved long-term repopulation of HSCs under stre

APA, Harvard, Vancouver, ISO, and other styles

3

Watt, Suzanne M., and Maria G. Roubelakis. "Deciphering the Complexities of Adult Human Steady State and Stress-Induced Hematopoiesis: Progress and Challenges." International Journal of Molecular Sciences 26, no. 2 (2025): 671. https://doi.org/10.3390/ijms26020671.

Full text

Abstract:

Human hematopoietic stem cells (HSCs) have traditionally been viewed as self-renewing, multipotent cells with enormous potential in sustaining essential steady state blood and immune cell production throughout life. Indeed, around 86% (1011–1012) of new cells generated daily in a healthy young human adult are of hematopoietic origin. Therapeutically, human HSCs have contributed to over 1.5 million hematopoietic cell transplants (HCTs) globally, making this the most successful regenerative therapy to date. We will commence this review by briefly highlighting selected key achievements (from 1868

APA, Harvard, Vancouver, ISO, and other styles

4

Wu, Jiang, Weiwei Zhang, Qian Ran, et al. "The Differentiation Balance of Bone Marrow Mesenchymal Stem Cells Is Crucial to Hematopoiesis." Stem Cells International 2018 (2018): 1–13. http://dx.doi.org/10.1155/2018/1540148.

Full text

Abstract:

Bone marrow mesenchymal stem cells (BMSCs), the important component and regulator of bone marrow microenvironment, give rise to hematopoietic-supporting stromal cells and form hematopoietic niches for hematopoietic stem cells (HSCs). However, how BMSC differentiation affects hematopoiesis is poorly understood. In this review, we focus on the role of BMSC differentiation in hematopoiesis. We discussed the role of BMSCs and their progeny in hematopoiesis. We also examine the mechanisms that cause differentiation bias of BMSCs in stress conditions including aging, irradiation, and chemotherapy. M

APA, Harvard, Vancouver, ISO, and other styles

5

Tseng, Yu-Jung, Richard Chapple, Tianyuan Hu, et al. "Hematopoietic Hierarchy Under Steady-State and Stress Conditions." Blood 134, Supplement_1 (2019): 1181. http://dx.doi.org/10.1182/blood-2019-125110.

Full text

Abstract:

The maintenance of the hematopoietic system by hematopoietic stem cells (HSCs) is an important topic in both clinical and basic hematology study due to their enormous therapeutic potential. The hematopoietic hierarchy has recently garnered renewed interest following the development of single-cell assays and improved strategies for genetic labeling, resulting in new hierarchical models that challenge the classical view of hematopoiesis. However, the kinetic of hematopoiesis under steady-state and stress condition and the contribution of HSCs toward steady-state hematopoiesis remain controversia

APA, Harvard, Vancouver, ISO, and other styles

6

Karlsson, Stefan. "Stress hematopoiesis requires Erg." Blood 118, no. 9 (2011): 2379–80. http://dx.doi.org/10.1182/blood-2011-07-362699.

Full text

APA, Harvard, Vancouver, ISO, and other styles

7

Suda, Toshio. "Hematopoiesis under the stress." Experimental Hematology 42, no. 8 (2014): S2. http://dx.doi.org/10.1016/j.exphem.2014.07.004.

Full text

APA, Harvard, Vancouver, ISO, and other styles

8

Essers, Marieke. "Inflammation-induced stress hematopoiesis." Experimental Hematology 53 (September 2017): S28. http://dx.doi.org/10.1016/j.exphem.2017.06.017.

Full text

APA, Harvard, Vancouver, ISO, and other styles

9

Landspersky, Theresa, Mehmet Saçma, Jennifer Rivière, et al. "Autophagy in mesenchymal progenitors protects mice against bone marrow failure after severe intermittent stress." Blood 139, no. 5 (2022): 690–703. http://dx.doi.org/10.1182/blood.2021011775.

Full text

Abstract:

Abstract The cellular mechanisms required to ensure homeostasis of the hematopoietic niche and the ability of this niche to support hematopoiesis upon stress remain elusive. We here identify Wnt5a in Osterix+ mesenchymal progenitor and stem cells (MSPCs) as a critical factor for niche-dependent hematopoiesis. Mice lacking Wnt5a in MSPCs suffer from stress-related bone marrow (BM) failure and increased mortality. Niche cells devoid of Wnt5a show defective actin stress fiber orientation due to an elevated activity of the small GTPase CDC42. This results in incorrect positioning of autophagosomes

APA, Harvard, Vancouver, ISO, and other styles

10

Brenet, Fabienne A., Pouneh Kermani, Shahin Rafii та Joseph M. Scandura. "TGFβ Restores Hematopoietic Homeostasis After Chemotherapy." Blood 120, № 21 (2012): 2344. http://dx.doi.org/10.1182/blood.v120.21.2344.2344.

Full text

Abstract:

Abstract Abstract 2344 Background: Hematopoietic stem cells (HSCs) are required for lifelong blood cell production and, to prevent exhaustion, the majority of HSCs are deeply quiescent during steady-state hematopoiesis. Paracrine factors produced by specialized bone marrow niche cells maintain HSC quiescence. Yet, evolution demands a rapid hematopoietic response to stressors such as infection, bleeding or toxin exposure. These triggers set off a remarkable adaptation in hematopoiesis that sacrifices HSPC quiescence, and the protection it affords, to meet an urgent need for new blood cell produ

APA, Harvard, Vancouver, ISO, and other styles

More sources

Dissertations / Theses on the topic "Stress hematopoiesis"

1

Javier, Jose Emmanuel F. "Increased TGF-beta Signaling Drives Different Hematopoietic Disease Outcomes following Stress Hematopoiesis." University of Cincinnati / OhioLINK, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1617109578665394.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

Büchler-Schäff, Marleen [Verfasser], and Ursula [Akademischer Betreuer] Klingmüller. "Investigation into hematopoietic stem cell function in the context of developmental specification and stress hematopoiesis / Marleen Büchler-Schäff ; Betreuer: Ursula Klingmüller." Heidelberg : Universitätsbibliothek Heidelberg, 2021. http://d-nb.info/1236345436/34.

Full text

APA, Harvard, Vancouver, ISO, and other styles

3

Sha, Xiaojin. "Translation initiation factor 4E binding protein 1,2 (4E-BP1,2) in hematopoiesis and stress erythropoiesis." Doctoral thesis, Humboldt-Universität zu Berlin, Mathematisch-Naturwissenschaftliche Fakultät I, 2008. http://dx.doi.org/10.18452/15797.

Full text

Abstract:

Das Eukaryotische-Initiations faktor-4E Bindungsprotein (4E-BP) ist ein Inhibitor der Translationsinitiation. Nicht-phosphoryliertes 4E-BP bindet an den eukaryotischen Initiationsfaktor 4E (eIF4E). Diese Bindung blockiert die Rekrutierung des Initiationskomplexes eIF4F an die Cap-Struktur des 5´Endes von eukaryotischen zellulären mRNAs, was die Initiation der Translation verhindert. Phosphorylierung von 4E-BP durch die mTOR Kinase führt zur Dissoziation des 4E-BP/eIF4E Komplexes und erhöht die Verfügbarkeit von eIF4E, dies wird mit Zellproliferation assoziiert. Die Aktivität von eIF4E wird nic

APA, Harvard, Vancouver, ISO, and other styles

4

Niederkorn, Madeline R. "TIFAB Links Innate Immune Signaling to the Cellular Stress Response in Myeloid Malignancies." University of Cincinnati / OhioLINK, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1593267529342488.

Full text

APA, Harvard, Vancouver, ISO, and other styles

5

Sha, Xiaojin [Verfasser], Achim [Gutachter] Leutz, Harald [Gutachter] Saumweber, and Wolfgang [Gutachter] Uckert. "Translation initiation factor 4E binding protein 1,2 (4E-BP1,2) in hematopoiesis and stress erythropoiesis / Xiaojin Sha ; Gutachter: Achim Leutz, Harald Saumweber, Wolfgang Uckert." Berlin : Humboldt-Universität zu Berlin, 2008. http://d-nb.info/1208077198/34.

Full text

APA, Harvard, Vancouver, ISO, and other styles

6

Traveset, Martínez Laia 1992. "New insights into transcription that preserve hematopoietic stem cell homeostasis." Doctoral thesis, Universitat Pompeu Fabra, 2020. http://hdl.handle.net/10803/670105.

Full text

Abstract:

Maintenance of steady-state and stress-adapted hematopoiesis depends on the fitness of hematopoietic stem and progenitor cells (HSPCs) in the bone marrow. Hematopoietic stem cells (HSCs) can adapt to stress by expanding their numbers and increasing the output of blood cells. This dynamic and highly complex process needs to be fully regulated in order to maintain a balance between the differentiation of HSCs and the need to keep a constant pool of HSCs. However, the molecular machinery in charge of this tight regulation has yet to be fully characterized. HSCs represent a small proportion inside

APA, Harvard, Vancouver, ISO, and other styles

7

Chiron, Andrada-Silvana. "Investigation into the role of erythropoietin in B cell lymphopoiesis." Electronic Thesis or Diss., Université Paris Cité, 2024. http://www.theses.fr/2024UNIP5262.

Full text

Abstract:

L'érythropoïétine (EPO) est une hormone produite principalement par les reins, dont la fonction essentielle consiste à assurer l'érythropoïèse. Sa concentration systémique augmente en réponse à l'anémie ou à l'hypoxie. En plus de cette production endogène, l'EPO humaine recombinante est couramment utilisée pour traiter l'anémie des patients atteints d'insuffisance rénale chronique ou de cancer. Les effets immunomodulateurs/immunosuppresseurs de l'EPO sur les lymphocytes matures et plus généralement sur les cellules immunitaires ont été bien étudiés. Cependant, son rôle dans la lympho-hématopoï

APA, Harvard, Vancouver, ISO, and other styles

8

McKim, Daniel Boyce. "Neuroimmune and Hematopoietic Regulation of Stress-Induced Anxiety." The Ohio State University, 2017. http://rave.ohiolink.edu/etdc/view?acc_num=osu1492079844476452.

Full text

APA, Harvard, Vancouver, ISO, and other styles

9

Foltz, Ian Nevin. "Regulation of the stress-activated protein kinase pathways in hematopoietic cells." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp02/NQ38887.pdf.

Full text

APA, Harvard, Vancouver, ISO, and other styles

10

OTTOLENGHI, SARA. "ONSET OF ANEMIA IN PULMONARY DISEASES: ROLE OF HYPOXIA, OXIDATIVE STRESS, IRON METABOLISM AND HEMATOPOIETIC REGULATORS." Doctoral thesis, Università degli Studi di Milano, 2021. http://hdl.handle.net/2434/839664.

Full text

Abstract:

BACKGROUND: In patients affected by severe hypoxia, such as those with Acute Respiratory Distress Syndrome (ARDS), an acute condition characterized by sudden onset of severe hypoxemia and a severe manifestation of COVID19, anemia is a common complication. It remains to be established weather iron metabolism, especially hepcidin, and oxidative stress are involved in negatively interfering with physiological hematopoietic compensatory mechanisms to hypoxia. AIMS: - To investigate etiology and progression of anemia in ARDS patients, through biomarkers of inflammation, iron metabolism and oxidat

APA, Harvard, Vancouver, ISO, and other styles

More sources

Books on the topic "Stress hematopoiesis"

1

Rechcigl, Miloslav. Handbook of Nutritional Requirements in a Functional Context: Volume II, Hematopoiesis, Metabolic Function, and Resistance to Physical Stress. Taylor & Francis Group, 2018.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

2

Rechcigl, Miloslav. Handbook of Nutritional Requirements in a Functional Context: Volume II, Hematopoiesis, Metabolic Function, and Resistance to Physical Stress. Taylor & Francis Group, 2018.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

3

Rechcigl, Miloslav. Handbook of Nutritional Requirements in a Functional Context: Volume II, Hematopoiesis, Metabolic Function, and Resistance to Physical Stress. Taylor & Francis Group, 2018.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

4

Rechcigl, Miloslav. Handbook of Nutritional Requirements in a Functional Context: Volume II, Hematopoiesis, Metabolic Function, and Resistance to Physical Stress. Taylor & Francis Group, 2018.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

Book chapters on the topic "Stress hematopoiesis"

1

Granjo, Elisa, Alice Santos-Silva, Irene Rebelo, et al. "Hematological and Biochemical Parameters in Hereditary Spherocytosis Under Oxidative Stress." In Molecular Biology of Hematopoiesis 6. Springer US, 1999. http://dx.doi.org/10.1007/978-1-4615-4797-6_41.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

Shibahara, Shigeki, Kazuhisa Takeda, Shoji Okinaga, Miki Yoshizawa, Kazuhiro Takahashi, and Hiroyoshi Fujita. "Transcriptional Control of the Human Heme Oxygenase-1 Gene by Stress." In Molecular Biology of Hematopoiesis 5. Springer US, 1996. http://dx.doi.org/10.1007/978-1-4613-0391-6_53.

Full text

APA, Harvard, Vancouver, ISO, and other styles

3

Granjo, Elisa, Ilídia Moreira, Alice Santos-Silva, et al. "Lymphocyte Populations in Hereditary Spherocytosis Pre and Post Splenectomy and Under Oxidative Stress." In Molecular Biology of Hematopoiesis 6. Springer US, 1999. http://dx.doi.org/10.1007/978-1-4615-4797-6_42.

Full text

APA, Harvard, Vancouver, ISO, and other styles

4

Law, Sujata, and Sayantan Ghosh. "Therapeutic Modalities Regarding ROS in Leukemia and Hematopoietic Stem/Progenitor Cell Perspective." In Handbook of Oxidative Stress in Cancer: Therapeutic Aspects. Springer Nature Singapore, 2022. http://dx.doi.org/10.1007/978-981-16-5422-0_91.

Full text

APA, Harvard, Vancouver, ISO, and other styles

5

Law, Sujata, and Sayantan Ghosh. "Therapeutic Modalities Regarding ROS in Leukemia and Hematopoietic Stem/Progenitor Cell Perspective." In Handbook of Oxidative Stress in Cancer: Therapeutic Aspects. Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-16-1247-3_91-1.

Full text

APA, Harvard, Vancouver, ISO, and other styles

6

Goris, H., B. Bungart, M. Loeffler, B. Dontje, and W. Nijhof. "Mobilization of Stem Cells and Progenitors After Thiamphenicol-Induced Hematopoietic Stress." In Peripheral Blood Stem Cell Autografts. Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-75717-4_5.

Full text

APA, Harvard, Vancouver, ISO, and other styles

7

Maestroni, Georges J. M. "Neuroendocrine Regulation of Hematopoiesis." In Psychoneuroimmunology, Stress, and Infection. CRC Press, 2020. http://dx.doi.org/10.1201/9780367812522-5.

Full text

APA, Harvard, Vancouver, ISO, and other styles

8

Bunaciu, Rodica, and rew Yen. "Retinoic Acid Signaling in Hematopoiesis and Immune Functions, and Options for Chemoprevention." In Inflammation, Oxidative Stress, and Cancer. CRC Press, 2013. http://dx.doi.org/10.1201/b15323-14.

Full text

APA, Harvard, Vancouver, ISO, and other styles

9

Razavi, Arezousadat, and Xiang-Jiao Yang. "Lysine Acetyltransferase 6A and Its Paralog: From Biological Functions to Neurodevelopmental Disorders, Cancer and New Therapeutics." In Understanding Developmental Disorders [Working Title]. IntechOpen, 2025. https://doi.org/10.5772/intechopen.1009241.

Full text

Abstract:

Post-translational modifications of histones, such as lysine acetylation and methylation, significantly influence chromatin structure in the eukaryotic nucleus. Lysine acetyltransferase 6A (KAT6A) and its paralog, KAT6B, participate in a variety of cellular processes crucial for normal development by influencing cell cycle progression, cell differentiation, signal transduction and responses to cellular stress. Studies using knockout mouse models have revealed the important effects of KAT6A and KAT6B on development, with the absence of the former resulting in embryonic lethality and the loss of

APA, Harvard, Vancouver, ISO, and other styles

10

"Hematopoietic Stem Cell Aging and Oxidative Stress." In Stem Cells: From Basic Research to Therapy, Volume 1. CRC Press, 2014. http://dx.doi.org/10.1201/b16487-9.

Full text

APA, Harvard, Vancouver, ISO, and other styles

Conference papers on the topic "Stress hematopoiesis"

1

Nguyen, S. T., C. Graf, S. Gur-Cohen, et al. "Endothelial protein C receptor signaling regulates myeloid-biased hematopoiesis under stress and in aging." In GTH Congress 2023 – 67th Annual Meeting of the Society of Thrombosis and Haemostasis Research – The patient as a benchmark. Georg Thieme Verlag, 2023. http://dx.doi.org/10.1055/s-0042-1760607.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

Elfeky, R., RM Shah, MNM Unni, et al. "132 Improved survival and outcome of HLA-mismatched donor hematopoietic stem cell transplantation in children with primary immunodeficiencies using new graft manipulation strategies in the UK." In Great Ormond Street Hospital Conference 2018: Continuous Care. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2018. http://dx.doi.org/10.1136/goshabs.132.

Full text

APA, Harvard, Vancouver, ISO, and other styles

3

Brenet, Fabienne, and Joseph M. Scandura. "Abstract 2548: P57Kip2 restrains the stress response of hematopoietic stem cells and its absence leads to chemotherapy resistance." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-2548.

Full text

APA, Harvard, Vancouver, ISO, and other styles

4

Leigh, Nicholas D., Kathleen M. Kokolus, Jason W.-L. Eng, et al. "Abstract B43: The degree of adrenergic stress signaling regulates the severity of graft versus host disease following allogeneic hematopoietic cell transplantation." In Abstracts: AACR Special Conference: Tumor Immunology and Immunotherapy: A New Chapter; December 1-4, 2014; Orlando, FL. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/2326-6074.tumimm14-b43.

Full text

APA, Harvard, Vancouver, ISO, and other styles

5

Zhou, Robin, Andrew Hughes, Jane L. Liesveld, and Michael R. King. "Nanoparticle-Coated Microtubes for the Manipulation of Cancer Cells." In ASME 2010 8th International Conference on Nanochannels, Microchannels, and Minichannels collocated with 3rd Joint US-European Fluids Engineering Summer Meeting. ASMEDC, 2010. http://dx.doi.org/10.1115/fedsm-icnmm2010-30168.

Full text

Abstract:

The development of novel methods for the isolation of primary stem and progenitor cells is important for the treatment of blood cancers, tissue engineering, and basic research in the biomedical sciences. Our lab has previously shown that microtubes coated with P-selectin protein can be used to capture and enrich hematopoietic stem and progenitor cells from a mixture of cells perfused through the tube at physiologically-relevant shear stresses[1][2], and that using a surface coating of colloidal silica nanoparticles (12 nm diameter, 30% by weight SiO2) increased cell capture and decreased rolli

APA, Harvard, Vancouver, ISO, and other styles

6

Charles, Nichola, Jane L. Liesveld, and Michael R. King. "Geometry Optimization of a Flow-Based Device to Maximize Selectin-Mediated Hematopoietic Stem Cell Enrichment." In ASME 2008 6th International Conference on Nanochannels, Microchannels, and Minichannels. ASMEDC, 2008. http://dx.doi.org/10.1115/icnmm2008-62228.

Full text

Abstract:

Rare cell enrichment techniques must selectively capture and successfully retain cells that exist at < 5% in a suspension. We developed a device to capture hematopoietic stem and progenitor cells (HSPCs) from adult bone marrow using immobilized adhesion molecules called selectins in the presence of a flow field. While we continue to optimize the immobilized protein surface and improve the selectivity of the device for HSPCs, it appears to be at the expense of cell recovery. To address this issue, we used experimental and computational methods to identify the hydrodynamic factors that contri

APA, Harvard, Vancouver, ISO, and other styles

We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!