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Books on the topic 'Stress-protective effect'

Author: Grafiati
Published: 4 June 2025
Last updated: 23 June 2025

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1

B, Mekjavic Igor, Banister Eric W, Morrison James B, Simon Fraser University. School of Kinesiology., and International Environmental Ergonomics Conference (2nd : 1986 : Whistler, B.C.), eds. Environmental ergonomics: Sustaining human performance in harsh environments. Taylor & Francis, 1988.

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2

Roychoudhury, Aryadeep, and Durgesh Kumar Tripathi. Protective Chemical Agents in the Amelioration of Plant Abiotic Stress: Biochemical and Molecular Perspectives. Wiley & Sons, Incorporated, John, 2020.

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3

Roychoudhury, Aryadeep, and Durgesh K. Tripathi. Protective Chemical Agents in the Amelioration of Plant Abiotic Stress: Biochemical and Molecular Perspectives. Wiley & Sons, Limited, John, 2020.

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4

Roychoudhury, Aryadeep, and Durgesh Kumar Tripathi. Protective Chemical Agents in the Amelioration of Plant Abiotic Stress: Biochemical and Molecular Perspectives. Wiley & Sons, Incorporated, John, 2020.

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5

Roychoudhury, Aryadeep, and Durgesh Kumar Tripathi. Protective Chemical Agents in the Amelioration of Plant Abiotic Stress: Biochemical and Molecular Perspectives. Wiley & Sons, Limited, John, 2020.

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6

Long, Laura J., and Matthew W. Gallagher. Hope and Posttraumatic Stress Disorder. Edited by Matthew W. Gallagher and Shane J. Lopez. Oxford University Press, 2017. http://dx.doi.org/10.1093/oxfordhb/9780199399314.013.24.

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Traumatic events can have a debilitating effect on mental health, and may lead to the development of posttraumatic stress disorder (PTSD). However, most people can adjust after adversity, and some even experience posttraumatic growth (PTG). Hope theory suggests that hope provides a psychological resource that can help individuals to respond to trauma with resilience. This chapter explores the role of hope as a protective factor preventing the development of PTSD, the relationship between hope and coping in the context of PTSD, and how hope may facilitate PTG. It also discusses how hope may act as a common factor across psychotherapies for the treatment of PTSD. Future research directions include investigating hope as a mechanism of change in psychotherapy for PTSD and the degree to which hope can incrementally predict PTSD and PTG beyond related types of positive thinking.
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7

Hermans, Greet. Effects of Insulin and Glycaemic Management on Neuromuscular Function. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199653461.003.0042.

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Observational studies have indicated an association between stress-induced hyperglycaemia and neuromuscular complications in critically ill patients. This observation is further supported by electrophysiological findings from two randomized controlled trials which suggested that titrating insulin therapy to achieve a normal blood glucose has a beneficial effect on neuromuscular function, associated with a reduced need for prolonged mechanical ventilation. The underlying pathophysiological mechanisms explaining these clinical observations are not well understood. There is no clear evidence that insulin has anabolic effects on muscle nor that it provides mitochondrial protection. It is possible that the effect of insulin therapy reflects a protective action on peripheral nerves; however, this hypothesis awaits confirmation. Overall, the potential benefits on neuromuscular function must be considered in light of large-scale randomized trials which demonstrate equivalent or worse outcome in critically ill patients managed with intensive insulin therapy.
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8

Barton, Allen W., and Gene H. Brody. Parenting as a Buffer That Deters Discrimination and Race-Related Stressors from “Getting Under the Skin”: Theories, Findings, and Future Directions. Edited by Brenda Major, John F. Dovidio, and Bruce G. Link. Oxford University Press, 2017. http://dx.doi.org/10.1093/oxfordhb/9780190243470.013.21.

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In this chapter, the hypothesis is explored that parenting processes buffer children’s physical health from the deleterious effects of discrimination. First, the existing empirical literature is reviewed on the stress-buffering effects of parenting on children’s physical health outcomes, with respect to the stress of general childhood adversity and the specific stressor of discrimination. Next, theoretical approaches are considered that account for the capacity of parenting to buffer contextual stressors, including discrimination, from compromising mental and physical health outcomes. Particular emphasis is given to the putative mechanisms potentially responsible for protective-stabilizing effects. Then, a critique of the extant research and theory on this topic highlights particular qualifications and considerations for the research community. Finally, suggestions are offered for future research directions, including the use of randomized prevention trials to test hypotheses about the protective functions of parenting in preventing discrimination from “getting under the skin.”
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9

Yehuda, Rachel, and Bruce S. McEwen. Biobehavioral Stress Response: Protective And Damaging Effects (Annals of the New York Academy of Sciences, V. 1032). New York Academy of Sciences, 2004.

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10

Jenney, Colin, and Angela Liegey Dougall. Immune System Functioning and Mental Health. Edited by Sara Maltzman. Oxford University Press, 2016. http://dx.doi.org/10.1093/oxfordhb/9780199739134.013.45.

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During the past few decades, research from multiple disciplines has elucidated the profound connections between the immune system and mental health. This chapter provides a review of this literature, placing emphasis on the connections between inflammation and cytokines, and stress, depression, posttraumatic stress disorder, schizophrenia, bipolar disorder, and social support. Additionally, brief overviews of the role of the natural immune system and adaptive immunity, as well as past research investigating stress are included. Further attention is focused on the physical health consequences of immune system dysregulation, ranging from increased susceptibility to infectious diseases, delayed wound healing, autoimmune responses, age-related elevations in proinflammatory cytokines, and decreased effectiveness of protective vaccinations. The chapter concludes with a discussion of the health effects and clinical implications of the relationships discussed throughout, as well as future directions to advance this field of study.
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11

Workplace health and safety sourcebook: Basic consumer health information about workplace health and safety, including the effect of workplace hazards on the lungs, skin, heart, ears, eyes, brain, reproductive organs, musculoskeletal system, and other organs and body parts : along with information about occupational cancer, personal protective equipment, toxic and hazardous chemicals, child labor, stress, and workplace violence. Omnigraphics, 2000.

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12

McKee-Ryan, Frances, and Robyn Maitoza. Job Loss, Unemployment, and Families. Edited by Ute-Christine Klehe and Edwin van Hooft. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780199764921.013.027.

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The detrimental effects of job loss and unemployment are not limited to the unemployed worker but ripple out to affect those closest to him or her. These ripple effects most notably impact the unemployed worker’s family, including a spouse or partner and/or children. In this chapter, we summarize previous research related to the impacts on marital or partner relationships and families and the particular effects of unemployment on children. For couples and families, we explore the financial or economic stressors and strain brought about by job loss; the direct, crossover, and relationship quality effects of stress and reduced mental health among unemployed workers and their spouses; protective resources for coping with job loss, such as social support and family resilience; and the social roles and identity of the unemployed worker. For children, we focus on mental health, child development, and educational/human capital attainment. We then offer suggestions for future research on families facing unemployment.
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13

Protective and Damaging Effects of the Biobehavioral Stress Response: From Molecules to Man (Annals of the New York Academy of Sciences). New York Academy of Sciences, 2004.

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14

Puttler, Leon I., Robert A. Zucker, and Hiram E. Fitzgerald. Developmental Science, Alcohol Use Disorders, and the Risk–Resilience Continuum. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780190676001.003.0001.

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The origins and expression of addiction are best understood within the context of developmental processes and dynamic systems organization and change. For some individuals, these dynamic processes lead to risk cumulative or cascade effects that embody adverse childhood experiences that exacerbate risk; predict early onset of drinking, smoking, or other substance use; and often lead to a substance use disorder (SUD) during the transitions to adolescence and emergent adulthood. In other cases, protective factors within or outside of the individual’s immediate family enable embodiment of normative stress regulatory systems and neural networks that support resilience and prevention of SUDs. A case study is provided to illustrate these processes and principles of the organization of addictive behavior. Finally, a model of risk to resilience captures the flow of development and the extent to which individual-experience relationships contribute to risk and resilience.
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15

Turner, Neil. Mechanisms of progression of chronic kidney disease. Edited by David J. Goldsmith. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0136.

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Three major hypotheses attempt to explain progressive kidney disease following diverse diseases and injuries. To varying degrees they can explain the observed risk factors for progression and the ability of interventions to lower risk. The hyperfiltration hypothesis argues that progression is due to stress on residual nephrons leading to injury and damage to remaining glomeruli. The toxicity of proteinuria hypothesis proposes that serum proteins or bound substances are toxic to tubular or tubulointerstitial cells. This sets up cycles of damage which lead to tubulointerstitial scarring. The podocyte loss hypothesis contends that proteinuria is simply a biomarker for damaged or dying podocytes, and that it is further podocyte loss that leads to progressive glomerulosclerosis. Renoprotective strategies might have direct effects on podocytes. Importantly these different hypotheses suggest different therapeutic approaches to protecting the function of damaged kidneys. Differences between repair mechanisms may explain why some injuries lead to recovery and others to progressive disease, and may suggest new targets for protective therapy.
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16

Air Pollution and Cardiovascular Diseases. Exon Publications, 2024. https://doi.org/10.36255/air-pollution-cardiovascular-diseases.

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Air Pollution and Cardiovascular Diseases is a comprehensive guide that explains the connection between air pollution and heart health. The article is organized into clear sections, each providing essential information on the causes, mechanisms, health impacts, and preventive measures associated with cardiovascular diseases resulting from exposure to polluted air. It begins by defining air pollution and identifying the major pollutants, such as particulate matter (PM2.5), nitrogen dioxide (NO₂), and ozone (O₃), that are most harmful to cardiovascular health. The article explains how these pollutants enter the body through inhalation and travel from the lungs into the bloodstream. Once in the bloodstream, pollutants trigger oxidative stress, inflammation, and blood vessel damage, which contribute to the development of heart disease, strokes, high blood pressure, and other cardiovascular conditions. It highlights the groups most at risk, including older adults, people with pre-existing heart conditions, and children. Strategies for reducing exposure, such as air quality monitoring, and use of protective masks are also discussed. The article concludes with a call for continued research to better understand the long-term effects of air pollution on heart health. The information is presented in simple terms to ensure it is accessible and easy to understand for all readers.
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17

Silveira, Larissa de Carvalho, and Renata Meira Véras. Associated factors and more frequent sleep disorders in university students: Integrative review. Ludomedia, 2022. http://dx.doi.org/10.36367/ntqr.13.2022.e695.

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Introduction: Sleep disorders affect quality of life on three levels, short, medium and long term. University students are groups prone to changes in sleep quality, as they live with sleep deprivation, among other factors such as anxiety, stress, excessive use of social networks, cell phones and television. The importance of sleep for undergraduates becomes evident in the face of the consequences that poor sleep quality can cause, in addition to allowing professionals in the areas of education and health to create programs that promote and encourage routines with beneficial consequences for students. Objectives: To review the literature on the most frequent sleep disorders in university students and the factors associated with sleep. Methods: This study is a systematic review. The search was performed in December 2021, in the electronic journals PubMed and CAPES journals. The time frame comprised the period from 2017 to 2021. In the end, 20 articles were selected to compose this review. Results: Regarding the main sleep disorders of undergraduates, the main outcomes found in the studies were insufficient sleep, long latency, nocturnal awakenings, excessive daytime sleepiness, insomnia and delayed sleep-wake phase disorder. Factors that are modifiable and non-modifiable for the occurrence of sleep disorders, as well as protective factors that can mitigate the occurrence of sleep disorders. Conclusions: The number of articles included in the review showed that over the five years the interest in researching the subject has been increasing. Even so, there is still a need for more effort from researchers to understand which factors are associated with reduced hours of sleep and how society and universities contribute to this practice of increasing waking hours and reducing sleep hours.
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