Relevant bibliographies by topics / Stress response of bacteria

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Stress response of bacteria'

Author: Grafiati
Published: 4 June 2021
Last updated: 2 February 2022

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Journal articles on the topic "Stress response of bacteria"

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Bonilla, Carla Y. "Generally Stressed Out Bacteria: Environmental Stress Response Mechanisms in Gram-Positive Bacteria." Integrative and Comparative Biology 60, no. 1 (February 11, 2020): 126–33. http://dx.doi.org/10.1093/icb/icaa002.

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Abstract The ability to monitor the environment for toxic chemical and physical disturbances is essential for bacteria that live in dynamic environments. The fundamental sensing mechanisms and physiological responses that allow bacteria to thrive are conserved even if the molecular components of these pathways are not. The bacterial general stress response (GSR) represents a conceptual model for how one pathway integrates a wide range of environmental signals, and how a generalized system with broad molecular responses is coordinated to promote survival likely through complementary pathways. Environmental stress signals such as heat, osmotic stress, and pH changes are received by sensor proteins that through a signaling cascade activate the sigma factor, SigB, to regulate over 200 genes. Additionally, the GSR plays an important role in stress priming that increases bacterial fitness to unrelated subsequent stressors such as oxidative compounds. While the GSR response is implicated during oxidative stress, the reason for its activation remains unknown and suggests crosstalk between environmental and oxidative stress sensors and responses to coordinate antioxidant functions. Systems levels studies of cellular responses such as transcriptomes, proteomes, and metabolomes of stressed bacteria and single-cell analysis could shed light into the regulated functions that protect, remediate, and minimize damage during dynamic environments. This perspective will focus on fundamental stress sensing mechanisms and responses in Gram-positive bacterial species to illustrate their commonalities at the molecular and physiological levels; summarize exciting directions; and highlight how system-level approaches can help us understand bacterial physiology.
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Chowdhury, Rukhsana, Gautam K. Sahu, and Jyotirmoy Das. "Stress response in pathogenic bacteria." Journal of Biosciences 21, no. 2 (April 1996): 149–60. http://dx.doi.org/10.1007/bf02703105.

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Poisot, Timothée, Thomas Bell, Esteban Martinez, Claire Gougat-Barbera, and Michael E. Hochberg. "Terminal investment induced by a bacteriophage in a rhizosphere bacterium." F1000Research 1 (October 2, 2012): 21. http://dx.doi.org/10.12688/f1000research.1-21.v1.

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Despite knowledge about microbial responses to abiotic stress, few studies have investigated stress responses to antagonistic species, such as competitors, predators and pathogens. While it is often assumed that interacting populations of bacteria and phage will coevolve resistance and exploitation strategies, an alternative is that individual bacteria tolerate or evade phage predation through inducible responses to phage presence. Using the microbial modelPseudomonas fluorescensSBW25 and its lytic DNA phage SBW25Φ2, we demonstrate the existence of an inducible response in the form of a transient increase in population growth rate, and found that the response was induced by phage binding. This response was accompanied by a decrease in bacterial cell size, which we propose to be an associated cost. We discuss these results in the context of bacterial ecology and phage-bacteria co-evolution.
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Poisot, Timothée, Thomas Bell, Esteban Martinez, Claire Gougat-Barbera, and Michael E. Hochberg. "Terminal investment induced by a bacteriophage in a rhizosphere bacterium." F1000Research 1 (May 20, 2013): 21. http://dx.doi.org/10.12688/f1000research.1-21.v2.

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Despite knowledge about microbial responses to abiotic stress, few studies have investigated stress responses to antagonistic species, such as competitors, predators and pathogens. While it is often assumed that interacting populations of bacteria and phage will coevolve resistance and exploitation strategies, an alternative is that individual bacteria tolerate or evade phage predation through inducible responses to phage presence. Using the microbial modelPseudomonas fluorescensSBW25 and its lytic DNA phage SBW25Φ2, we demonstrate the existence of an inducible response in the form of a transient increase in population growth rate, and found that the response was induced by phage binding. This response was accompanied by a decrease in bacterial cell size, which we propose to be an associated cost. We discuss these results in the context of bacterial ecology and phage-bacteria co-evolution.
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YAMAMOTO, Tomoko. "Stress Response of Pathogenic Bacteria. Are Stress Proteins Virulence Factors?" Nippon Saikingaku Zasshi 51, no. 4 (1996): 1025–36. http://dx.doi.org/10.3412/jsb.51.1025.

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Si, Meiru, Chao Zhao, Brianne Burkinshaw, Bing Zhang, Dawei Wei, Yao Wang, Tao G. Dong, and Xihui Shen. "Manganese scavenging and oxidative stress response mediated by type VI secretion system in Burkholderia thailandensis." Proceedings of the National Academy of Sciences 114, no. 11 (February 27, 2017): E2233—E2242. http://dx.doi.org/10.1073/pnas.1614902114.

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Type VI secretion system (T6SS) is a versatile protein export machinery widely distributed in Gram-negative bacteria. Known to translocate protein substrates to eukaryotic and prokaryotic target cells to cause cellular damage, the T6SS has been primarily recognized as a contact-dependent bacterial weapon for microbe–host and microbial interspecies competition. Here we report contact-independent functions of the T6SS for metal acquisition, bacteria competition, and resistance to oxidative stress. We demonstrate that the T6SS-4 in Burkholderia thailandensis is critical for survival under oxidative stress and is regulated by OxyR, a conserved oxidative stress regulator. The T6SS-4 is important for intracellular accumulation of manganese (Mn2+) under oxidative stress. Next, we identified a T6SS-4–dependent Mn2+-binding effector TseM, and its interacting partner MnoT, a Mn2+-specific TonB-dependent outer membrane transporter. Similar to the T6SS-4 genes, expression of mnoT is regulated by OxyR and is induced under oxidative stress and low Mn2+ conditions. Both TseM and MnoT are required for efficient uptake of Mn2+ across the outer membrane under Mn2+-limited and -oxidative stress conditions. The TseM–MnoT-mediated active Mn2+ transport system is also involved in contact-independent bacteria–bacteria competition and bacterial virulence. This finding provides a perspective for understanding the mechanisms of metal ion uptake and the roles of T6SS in bacteria–bacteria competition.
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Kulp, Adam, and Meta J. Kuehn. "Recognition of β-Strand Motifs by RseB Is Required for σEActivity in Escherichia coli." Journal of Bacteriology 193, no. 22 (September 9, 2011): 6179–86. http://dx.doi.org/10.1128/jb.05657-11.

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Gram-negative bacteria react to misfolded proteins in the envelope through a myriad of different stress response pathways. This cohort of pathways allows the bacteria to specifically respond to different types of damage, and many of these have been discovered to have key roles in the virulence of bacterial pathogens. Misfolded outer membrane proteins (OMPs) are typically recognized by the σEpathway, a highly conserved envelope stress response pathway. We examined the features of misfolded OMPs with respect to their ability to generate envelope stress responses. We determined that the secondary structure, particularly the potential to form β strands, is critical to inducing the σEresponse in an RseB-dependent manner. The sequence of the potential β-strand motif modulates the strength of the σEresponse generated by the constructs. By understanding the details of how such stress response pathways are activated, we can gain a greater understanding of how bacteria survive in harsh environments.
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WESCHE, ALISSA M., JOSHUA B. GURTLER, BRADLEY P. MARKS, and ELLIOT T. RYSER. "Stress, Sublethal Injury, Resuscitation, and Virulence of Bacterial Foodborne Pathogens†." Journal of Food Protection 72, no. 5 (May 1, 2009): 1121–38. http://dx.doi.org/10.4315/0362-028x-72.5.1121.

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Environmental stress and food preservation methods (e.g., heating, chilling, acidity, and alkalinity) are known to induce adaptive responses within the bacterial cell. Microorganisms that survive a given stress often gain resistance to that stress or other stresses via cross-protection. The physiological state of a bacterium is an important consideration when studying its response to food preservation techniques. This article reviews the various definitions of injury and stress, sublethal injury of bacteria, stresses that cause this injury, stress adaptation, cellular repair and response mechanisms, the role of reactive oxygen species in bacterial injury and resuscitation, and the potential for cross-protection and enhanced virulence as a result of various stress conditions.
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Wang, Tianwen, Chen Liang, Mengyuan Zheng, Lu Liu, Yafei An, Hongju Xu, Sa Xiao, and Lei Nie. "Ribosome Hibernation as a Stress Response of Bacteria." Protein & Peptide Letters 27, no. 11 (November 16, 2020): 1082–91. http://dx.doi.org/10.2174/0929866527666200610142118.

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Ribosome is primarily regarded as the committing organelle for the translation process. Besides the expansion of its function from a translational machine for protein synthesis to a regulatory platform for protein quality control, the activity regulation and recycling of ribosome have been deepened significantly. Recent advances have confirmed a novel mechanism in the regulation of ribosome activity when a cell encounters adverse conditions. Due to the binding of certain protein factors onto a ribosome, the structural and functional change of the ribosome inside the cell will take place, thereby leading to the formation of inactive ribosomes (70S monomer or 100S dimer), or ribosome hibernation. By ribosome hibernation, the overall protein synthesis rate of a cell could be slowed down. The resistance to adverse conditions or chemicals of the host cell will be enhanced. In this paper, we discussed the phenomenon, molecular mechanism, and physiological effect of ribosome hibernation when cells are under stresses. And then, we discussed the resuscitation of a hibernating ribosome and the role of ribosome hibernation in the treatment of antimicrobial infection.
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Jordan, Sina, Matthew I. Hutchings, and Thorsten Mascher. "Cell envelope stress response in Gram-positive bacteria." FEMS Microbiology Reviews 32, no. 1 (January 2008): 107–46. http://dx.doi.org/10.1111/j.1574-6976.2007.00091.x.

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Dissertations / Theses on the topic "Stress response of bacteria"

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Drury, Elliott C. "Stress response and hypothetical genes in Desulfovibrio vulgaris Hildenborough." Diss., Columbia, Mo. : University of Missouri-Columbia, 2008. http://hdl.handle.net/10355/5719.

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Thesis (M.S.)--University of Missouri-Columbia, 2008.
The entire dissertation/thesis text is included in the research.pdf file; the official abstract appears in the short.pdf file (which also appears in the research.pdf); a non-technical general description, or public abstract, appears in the public.pdf file. "December 2008" Includes bibliographical references.
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Alexandre, Ana, and Solange Oliveira. "Heat shock response in bacteria with large genomes: lessons from rhizobia." Bachelor's thesis, Wiley-Blackwell Publishers, 2016. http://hdl.handle.net/10174/19210.

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Rhizobia are important soil bacteria due to their ability to establish nitrogen-fixing symbioses with legume plants. In this dual lifestyle, as free-living bacteria or as plant symbiont, rhizobia are often exposed to different environmental stresses. The present chapter overviews the current knowledge on the heat shock response of rhizobia, highlighting how these large genome bacteria respond to heat from a transcriptional point of view. Response to heat shock in rhizobia involves genome wide changes in the transcriptome that may affect more than 30% of the genome and involve all replicons. In addition to the expected upregulation of genes already known to be involved in stress response (dnaK, groEL, ibpA, clpB), the reports on the heat shock response in rhizobia also showed particular aspects of stress response in these resourceful bacteria. The transcriptional response to heat in rhizobia includes the overexpression of a large number of genes involved in transcription and carbohydrate transport and metabolism. Additional studies are needed in order to better understand the transcriptional regulation of stress response in bacteria with large genomes.
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Atkinson, Deborah Jane. "Stress response and inorganic poly-phosphate in the Bacillus group bacteria." Thesis, University of Bath, 2010. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.538113.

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This thesis concentrates on the Bacillus cereus group of organisms and interactions that they may encounter in their natural environment. Inorganic polyphosphate has been identified as an important factor of stress and survival in B. cereus. One of the aims of this project was to create knock out mutants of certain enzymes involved in polyphosphate metabolism in B. anthracis, the etiological agent of anthrax. Unfortunately, even though B. anthracis is very closely related to B. cereus and despite the application of published methods it was not possible to create these B. anthracis knockout mutants. In order to address the importance of inorganic polyphosphate in B. anthracis, a real time RT‐PCR assay was developed to monitor the mRNA levels of these enzymes when the bacterium is faced with harsh nutrient environments Real time RT‐PCR analysis showed that mRNA levels of the metabolizing enzymes were upregulated in low nutrient conditions but that the profiles of gene expression were varied when grown in a chemically defined media. In addition to abiotic stresses such as low nutrients, B. anthracis is also likely to face biotic stress such as predation by amoeba in the soil. Investigations were performed into the outcome of the interaction of B. cereus group bacteria with a model amoeba, Acanthamoeba polyphaga. Amoebae are bacterial predators but can also be utilised as hosts by bacterial symbionts and pathogens, such as Legionella pneumophila. It was theorised that amoebae may provide a host environment similar to that of the professional macrophages, which B. anthracis encounters in mammalian infection. These investigations confirmed that the B. cereus group bacteria demonstrate a range of interactions with amoeba cells, from surface attachment through to intracellular persistence. These studies went on to show that B. cereus, B. thuringiensis and B. anthracis can all be engulfed by amoebae when challenged in their vegetative form and that spores were able to survive, and apparently germinate. Finally these studies have identified a new developmental stage of the B. cereus group bacteria. When grown in static conditions, especially in the presence of amoeba, the bacterial cells cease to septate and large (often motile) continuous hyphae like filaments form. These filaments can be seen to “weave” together to form large “rope” like macrofibre structures which can even become visible by eye. Previously this macrofibre growth has also been seen in B. subtilis, suggesting it may be common to the whole genus. In the light of these findings we speculate that this group of pathogens have evolved complex behaviours to interact with soil amoeba in order to facilitate survival in harsh environmental conditions.
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Perry, Leslie M. "Regulation of Alternative Sigma Factors During Oxidative and Ph Stresses in the Phototroph Rhodopseudomonas Palustris." Thesis, University of North Texas, 2014. https://digital.library.unt.edu/ark:/67531/metadc700009/.

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Rhodopseudomonas palustris is a metabolically versatile phototrophic α-proteobacterium. The organism experiences a wide range of stresses in its environment and during metabolism. The oxidative an pH stresses of four ECF (extracytoplasmic function) σ-factors are investigated. Three of these, σ0550, σ1813, and σ1819 show responses to light-generated singlet oxygen and respiration-generated superoxide reactive oxygen species (ROS). The EcfG homolog, σ4225, shows a high response to superoxide and acid stress. Two proteins, one containing the EcfG regulatory sequence, and an alternative exported catalase, KatE, are presented to be regulated by σ4225. Transcripts of both genes show similar responses to oxidative stress compared to σ4225, indicating it is the EcfG-like σ-factor homolog and controls the global stress response in R. palustris.
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Zaklikowski, Anna Emilia. "The Effect of Chlorine and Chloramines on the Viability and Activity of Nitrifying Bacteria." Thesis, Virginia Tech, 2006. http://hdl.handle.net/10919/33758.

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Nitrification is a significant concern for drinking water systems employing chloramines for secondary disinfection. Utilities have implemented a range of disinfection strategies that have varying levels of effectiveness in the prevention and control of nitrification events, including optimizing the chlorine-to-ammonia ratio, maintaining chloramine residual throughout the distribution system, controlling pH, and temporal switching to free chlorination. Annual or semi-annual application of free chlorination is practiced by 23% of chloraminating systems on a temporary basis as a preventative measure, even though it has the undesirable consequences of temporarily increasing disinfection byproducts, facilitating coliform detachment, and altering water taste and odor.

Although temporal free chlorination and other nitrification control methods have been widely studied in the field and in pilot-scale systems, very little is known about the stress responses of nitrifying bacteria to different disinfection strategies and the role physiological state plays in the resistance to disinfection. It is well known that many commonly studied bacteria, such as Escherichia coli, are able to better resist disinfection by free chlorine and chloramines under nutrient limitation through regulation of stress response genes that encode for DNA protection and enzymes that mediate reactive oxygen species. We compared the genomes of E. coli and the ammonia-oxidizing bacterium Nitrosomonas europaea, and found that many of the known stress response mechanisms and genes present in E. coli are absent in N. europaea or not controlled by the same mechanisms specific to bacterial growth state. These genetic differences present a general susceptibility of N. europaea to disinfection by chlorine compounds.

Using an experimental approach, we tested the hypothesis that N. europaea does not develop increased resistance to free chlorine and monochloramine during starvation to the same degree as E. coli. In addition, N. europaea cells were challenged with sequential treatments of monochloramine and hypochlorous acid to mimic the disinfectant switch employed by drinking water utilities. Indicators of activity (specific nitrite generation rate) and viability (LIVE/DEAD® BacLight⠢ membrane-integrity based assay) were measured to determine short-term effectiveness of disinfection and recovery of cells over a twelve day monitoring period. The results of disinfectant challenge experiments reinforce the hypothesis, indicating that the response of N. europaea to either disinfectant does not significantly change during the transition from exponential phase to stationary phase. Exponentially growing N. europaea cells showed greater susceptibility to hypochlorous acid and monochloramine than stationary phase E. coli cells, but had increased resistance compared with exponential phase E. coli cells. Following incubation with monochloramine, N. europaea showed increased sensitivity to subsequent treatment with hypochlorous acid. Complete loss of ammonia-oxidation activity was observed in cells immediately following treatment with hypochlorous acid, monochloramine, or a combination of both disinfectants. Replenishing ammonia and nutrients did not invoke recovery of cells, as detected in activity measurements during the twelve day monitoring period. The results provide evidence for the effectiveness of both free chlorine and chloramines in the inhibition of growth and ammonia-oxidation activity in N. europaea. Furthermore, comparison of viability and activity measurements suggest that the membrane integrity-based stain does not serve as a good indicator of activity. These insights into the responses of pure culture nitrifying bacteria to free chlorine and monochloramine could prove useful in designing disinfection strategies effective in the control of nitrification.


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Drews, Oliver. "Differential proteome analysis of selected lactic acid bacteria, stress response and database construction." [S.l. : s.n.], 2005. http://deposit.ddb.de/cgi-bin/dokserv?idn=974284742.

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Mirhabibollahi, B. "Influence of mode of DNA replication on the response of Salmonella typhimurium to physical stress." Thesis, University of Reading, 1988. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.383460.

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Zhu, Zeyu. "Multi-Omics Stress Responses and Adaptive Evolution in Pathogenic Bacteria: From Characterization Towards Diagnostic Prediction." Thesis, Boston College, 2020. http://hdl.handle.net/2345/bc-ir:108912.

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Thesis advisor: Tim van Opijnen
Thesis advisor: Welkin Johnson
Pathogenic bacteria can experience various stress factors during an infection including antibiotics and the host immune system. Whether a pathogen will establish an infection largely depends on its survival-success while enduring these stress factors. We reasoned that the ability to predict whether a pathogen will survive under and/or adapt to a stressful condition will provide great diagnostic and prognostic value. However, it is unknown what information is needed to enable such predictions. We hypothesized that under a stressful condition, a bacterium triggers responses that indicate how the stress is experienced in the genome, thereby correctly identifying a stress response holds the key to enabling such predictions. Bacterial stress responses have long been studied by determining how small groups of individual genes or pathways respond to certain environmental triggers. However, the conservation of these genes and the manner in which they respond to a stress can vary widely across species. Thus, this thesis sought to achieve a genome-wide and systems-level understanding of a bacterial stress response with the goal to identify signatures that enable predictions of survival and adaptation outcomes in a pathogen- and stress-independent manner. Here, we first set up a multi-omics framework that maps out a stress response on a genome-wide level using the human respiratory pathogen Streptococcus pneumoniae as a model organism. Under an environmental stress, gene fitness changes are determined by transposon insertion sequencing (Tn-Seq) which represents the phenotypic response. Differential expression is profiled by RNA-Seq which represents as the transcriptional response. Much to our surprise, the phenotypic response and transcriptional response are separated on different genes, meaning that differentially expressed genes are poor indicators of genes that contribute to the fitness of the bacterium. By devising and performing topological network analysis, we show that phenotypic and transcriptional responses are coordinated under evolutionary familiar stress, such as nutrient depletion and host infection, in both Gram-positive and -negative pathogens. However, such coordination is lost under the relatively unfamiliar stress of antibiotic treatment. We reasoned that this could mean that a generalizable stress response signature might exist that indicates the level to which a bacterium is adapted to a stress. By extending stress response profiling to 9 antibiotics and 3 nutrient depletion conditions, we found that such a signature indeed exists and can be captured by the level of transcriptomic disruption, defined by us as transcriptomic entropy. Centered on entropy, we constructed predictive models that perform with high accuracy for both survival outcomes and antibiotic sensitivity across 7 species. To further develop these models with the goal to eventually enable predictions on disease progression, we developed a dual RNA-Seq technique that maps out the transcriptomic responses of both S. pneumoniae and its murine host during lung infection. Preliminary data show that a high entropy is observed in the pathogen’s transcriptome during clearance (a failed infection) compared to a successful/severe infection, while the host transcriptome exhibits a pro-inflammatory and active immune response under the severe infection. Lastly, we characterized evolutionary trajectories that lead to long-term survival success of S. pneumoniae, for instance this means that the bacterium successfully adapts to the presence of an antibiotic and becomes resistant or can grow successfully in the absence of a formerly critical nutrient. These trajectories show that adaptive mutations tend to occur in genes closely related to the adapted stress. Additionally, independent of the stress, adaptation triggers rewiring of transcriptional responses resulting in a change in entropy from high to low. Most importantly, we demonstrate that by combining multi-omics profiles with additional genomic data including gene conservation and expression plasticity, and feeding this into machine learning models, that adaptive evolution can become (at least partially) predictable. Additionally, the genetic diversity in bacterial genomes across different strains and species can indeed influence a bacterium’s adaptation trajectory. In conclusion, this thesis presents a substantial collection of multi-omics stress response profiles of S. pneumoniae and other pathogenic bacteria under various environmental and clinically-relevant stresses. By demonstrating the feasibility of predictions on bacterial survival and adaptive outcomes, this thesis paves the way towards future improvements on infectious disease prognostics and forecasting the emergence of antibiotic resistance
Thesis (PhD) — Boston College, 2020
Submitted to: Boston College. Graduate School of Arts and Sciences
Discipline: Biology
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Hardwick, Steven. "Structural and functional characterisation of partner switching proteins involved in the environmental stress response of gram-positive bacteria." Thesis, University of Newcastle Upon Tyne, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.438402.

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Reiske, Lena [Verfasser], and Volker [Akademischer Betreuer] Stefanski. "Stress hormone-induced immunomodulation and interplay between immune cells and bacteria in response to stress hormones in domestic pigs / Lena Reiske ; Betreuer: Volker Stefanski." Hohenheim : Kommunikations-, Informations- und Medienzentrum der Universität Hohenheim, 2020. http://d-nb.info/1223023249/34.

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Books on the topic "Stress response of bacteria"

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Kidd, Stephen P. Stress response in pathogenic bacteria. Wallingford, Oxfordshire, UK: CABI, 2011.

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Kidd, S. P., ed. Stress response in pathogenic bacteria. Wallingford: CABI, 2011. http://dx.doi.org/10.1079/9781845937607.0093.

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Wong, Hin-chung. Stress response of foodborne microorganisms. Hauppauge, N.Y: Nova Science Publishers, 2011.

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Tsakalidou, Effie, and Konstantinos Papadimitriou, eds. Stress Responses of Lactic Acid Bacteria. Boston, MA: Springer US, 2011. http://dx.doi.org/10.1007/978-0-387-92771-8.

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Konstantinos, Papadimitriou, and SpringerLink (Online service), eds. Stress Responses of Lactic Acid Bacteria. Boston, MA: Springer Science+Business Media, LLC, 2011.

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Storz, Gisela, and Regine Hengge, eds. Bacterial Stress Responses. Washington, DC, USA: ASM Press, 2010. http://dx.doi.org/10.1128/9781555816841.

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Zuber, Peter. Function and Control of the Spx-Family of Proteins Within the Bacterial Stress Response. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-6925-4.

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Keyse, Stephen M. Stress Response. New Jersey: Humana Press, 2000. http://dx.doi.org/10.1385/1592590543.

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Bacteria in agrobiology: Stress management. Heidelberg: Springer, 2012.

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Maheshwari, Dinesh K., ed. Bacteria in Agrobiology: Stress Management. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-23465-1.

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Book chapters on the topic "Stress response of bacteria"

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Hengge, Regine. "The General Stress Response in Gram-Negative Bacteria." In Bacterial Stress Responses, 251–89. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555816841.ch15.

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Balaban, Nathalie Q. "Persister Bacteria." In Bacterial Stress Responses, 375–82. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555816841.ch22.

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Makarova, Kira S., and Michael J. Daly. "Comparative Genomics of Stress Response Systems in Deinococcus Bacteria." In Bacterial Stress Responses, 445–57. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555816841.ch27.

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Price, Chester W. "General Stress Response in Bacillus subtilis and Related Gram-Positive Bacteria." In Bacterial Stress Responses, 301–18. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555816841.ch17.

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Spector, Michael P., and John W. Foster. "Starvation-Stress Response (SSR) of Salmonella typhimurium." In Starvation in Bacteria, 201–24. Boston, MA: Springer US, 1993. http://dx.doi.org/10.1007/978-1-4899-2439-1_9.

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Sharma, Anket, Vinod Kumar, Neha Handa, Shagun Bali, Ravdeep Kaur, Kanika Khanna, Ashwani Kumar Thukral, and Renu Bhardwaj. "Potential of Endophytic Bacteria in Heavy Metal and Pesticide Detoxification." In Plant Microbiome: Stress Response, 307–36. Singapore: Springer Singapore, 2018. http://dx.doi.org/10.1007/978-981-10-5514-0_14.

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Matilla, Miguel A., and Tino Krell. "Plant Growth Promotion and Biocontrol Mediated by Plant-Associated Bacteria." In Plant Microbiome: Stress Response, 45–80. Singapore: Springer Singapore, 2018. http://dx.doi.org/10.1007/978-981-10-5514-0_3.

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Bouveret, Emmanuelle, and Aurélia Battesti. "The Stringent Response." In Bacterial Stress Responses, 229–50. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555816841.ch14.

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Kiley, Patricia J., and Timothy J. Donohue. "Global Responses of Bacteria to Oxygen Deprivation." In Bacterial Stress Responses, 175–89. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555816841.ch11.

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Lovett, Susan T. "The DNA Damage Response." In Bacterial Stress Responses, 205–28. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555816841.ch13.

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Conference papers on the topic "Stress response of bacteria"

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Ragoonanan, Vishard, Jason Malsam, Daniel R. Bond, and Alptekin Aksan. "Desiccation of Geobacter Sulfurreducens: Membrane Response to Osmotic Stress." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192461.

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Geobacter sulfurreducens is a δ-proteobacterium capable of electron transfer to extracellular compounds and surfaces [1] that has possible technological applications in microbial-based sensors, bioremediation of contaminated environments and harvesting electricity from waste organic matter [2]. Further development of many of these applications requires stabilization and preservation of bacteria as thin films on surfaces. One method of encapsulating bacteria in thin films is through the use of porous latex coatings [2]. However, during the formation of these films, the bacteria are exposed to osmotic stresses.
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Kniggendorf, Ann-Kathrin, Regina Nogueira, and Bernhard Roth. "Oxygen Stress Response of Nitrifying Bacteria monitored with Raman Spectroscopy In Vivo." In CLEO: Applications and Technology. Washington, D.C.: OSA, 2021. http://dx.doi.org/10.1364/cleo_at.2021.am4p.4.

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Zhou, Lang, Jinzi Deng, Jinzi Deng, Reinaldo E. Alcalde, Reinaldo E. Alcalde, Robert A. Sanford, Robert A. Sanford, et al. "BACTERIAL RESPONSE TO MICROFLUIDIC STRESS GRADIENTS." In 50th Annual GSA North-Central Section Meeting. Geological Society of America, 2016. http://dx.doi.org/10.1130/abs/2016nc-275646.

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Feoktistova, A. V., M. D. Timergalin, T. V. Rameev, and S. P. Chetverikov. "The role of auxin-producing bacteria in the formation of a growth response in wheat plants under herbicidal stress." In 2nd International Scientific Conference "Plants and Microbes: the Future of Biotechnology". PLAMIC2020 Organizing committee, 2020. http://dx.doi.org/10.28983/plamic2020.073.

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The paper presents the results of the effect of treatment with bacteria on the growth and hormonal balance of wheat plants with simultaneous exposure to the herbicide Chistalan. It is shown that herbicide stress is leveled by bacteria.
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Marthe, Logan, Catalina Stoica, Laura Floentina Chiriac, Toma Galaon, and Mihai Nita-Lazar. "STUDY OF BACTERIA RESISTANCE MECHANISMS IN RESPONSE TO A STRESS INDUCED BY PHARMACEUTICALS COMPOUNDS." In International Symposium "The Environment and the Industry". National Research and Development Institute for Industrial Ecology, 2017. http://dx.doi.org/10.21698/simi.2017.0038.

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Zheng, Zhouyuan, Parth Bansal, and Yumeng Li. "Numerical Study on Antibacterial Effects of Bio-Inspired Nanostructured Surface." In ASME 2020 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2020. http://dx.doi.org/10.1115/imece2020-23594.

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Abstract Natural bactericidal surfaces are found on the wings of cicada and dragonfly that compose of nanopatterns such as nanopillar arrays. Experimental studies have unveiled that the nanopillars can penetrate the bacterial walls or stretch them, resulting in the cell death. This offers an attractive “chemical-free” and wide-spectrum strategy to fight against bacteria-related infections and fouling, especially for implant-associated infections (IAIs). However, what is the fundamental mechanism and key factors governing the bactericidal performance of the nanostructured surface is the critical research questions need to be answered to realize its full potential. In this work, we developed mechanical single cell model of bacteria based on finite element analysis (FEA) to simulate the interactions between different strains of bacteria and the nanostructured surface. The nanostructured surface contains nanopillar arrays, which are made of polymer materials. Different strains of bacteria are simulated by adopting the corresponding geometry and material properties from experimental values. The mechanical responses of the bacteria cell on the nanopillar arrays with various configurations are studied based on estimated stress and strain distributions within the cell.
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Buzi, Gentian, and Mustafa Khammash. "Detection of loading effects in bacterial stress response using biochemical stochasticity." In 2014 IEEE 53rd Annual Conference on Decision and Control (CDC). IEEE, 2014. http://dx.doi.org/10.1109/cdc.2014.7040237.

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Kamnev, A. A., and A. V. Tugarova. "Intracellular transformations in bacteria as a response to external factors: molecular spectroscopic characterization." In 2nd International Scientific Conference "Plants and Microbes: the Future of Biotechnology". PLAMIC2020 Organizing committee, 2020. http://dx.doi.org/10.28983/plamic2020.110.

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Even-Tzur, Nurit, Uri Zaretsky, Michael Wolf, and David Elad. "Respose of Cultured Nasal Epithelial Cells to Wall Shear Stress." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176374.

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The nasal cavity lining is rich with mucus secreting goblet cells. Nasal defense is based on the mucociliary clearance mechanism, in which the secreted mucus layer traps inhaled particles and is constantly driven towards the nasopharynx for removal of the particles from the body. The mucus layer is also important for the exchange of temperature and water vapor with the inspired air. Airway goblet cells discharge mucus in response to a wide variety of biological stimuli, including cytokines, bacterial products, proteinases, oxidants, irritant gases, and inflammatory mediators [1], as well as biophysical changes, such as osmolarity alterations [2].
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Thomas, Wendy E., Evgeni V. Sokurenko, and Viola Vogel. "How Bacteria Bind More Strongly Under Mechanical Force: The Catch-Bond FimH." In ASME 2003 International Mechanical Engineering Congress and Exposition. ASMEDC, 2003. http://dx.doi.org/10.1115/imece2003-43680.

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We study a protein that responds to mechanical force in most striking manner. We demonstrate that Escherichia coli bacteria need shear stress to bind to certain tissues and model surfaces; they bind strongest precisely when the body tries to wash them off. We have determined that the protein responsible for this behavior is FimH, a ubiquitous adhesion protein in intestinal bacteria that mediates adhesion to host cells via the carbohydrate mannose. Although mechanical force noramlly decreases bond lifetimes, we have shown that the bond betweeen FimH and simple mono-mannose receptors is s “catch-bond” that lasts longer under shear stress. In contrast, structural variations in either FimH or the receptor cause a stronger mode of adhesion in static conditions with little or no activation under force. We derive a structural for how mechanical force switches FimH to a strong binding mode by using steered molecular dynamics simulations, and validate the predictions with subsequent site-directed mutagenesis. The physiological consequences as well as the engineering principles suggested by the structural model will be discussed.
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Reports on the topic "Stress response of bacteria"

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Zhou, Jizhong, and Zhili He. Deduction and Analysis of the Interacting Stress Response Pathways of Metal/Radionuclide-reducing Bacteria. Office of Scientific and Technical Information (OSTI), February 2010. http://dx.doi.org/10.2172/1098145.

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Chesbro, W. Stress responses of subsurface bacteria. Final report, June 1, 1995--February 1, 1998. Office of Scientific and Technical Information (OSTI), July 1998. http://dx.doi.org/10.2172/631202.

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Wong, Kwong-Kwok. Genetic Analysis of Stress Responses in Soil Bacteria for Enhanced Bioremediation of Mixed Contaminants. Office of Scientific and Technical Information (OSTI), June 2000. http://dx.doi.org/10.2172/827355.

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Wong, Kwong-Kwok. Genetic Analysis of Stress Responses in Soil Bacteria for Enhanced Bioremediation of Mixed Contaminants. Office of Scientific and Technical Information (OSTI), December 2000. http://dx.doi.org/10.2172/827357.

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Timberlake, Sonia, Marcin Joachimiak, Dominique Joyner, Romy Chakraborty, Jason Baumohl, Paramvir Dehal, Adam Arkin, Terry Hazen, and Eric Alm. Conservation of Modules but not Phenotype in Bacterial Response to Environmental Stress. Office of Scientific and Technical Information (OSTI), May 2010. http://dx.doi.org/10.2172/985921.

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Wong, K. K. Genetic analysis of stress responses in soil bacteria for enhanced bioremediation of mixed contaminants. 1997 annual progress report. Office of Scientific and Technical Information (OSTI), June 1997. http://dx.doi.org/10.2172/13695.

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Wong, K. K. Genetic analysis of stress responses in soil bacteria for enhanced bioremediation of mixed contaminants. 1998 annual progress report. Office of Scientific and Technical Information (OSTI), June 1998. http://dx.doi.org/10.2172/13696.

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Hutchinson, M. L., J. E. L. Corry, and R. H. Madden. A review of the impact of food processing on antimicrobial-resistant bacteria in secondary processed meats and meat products. Food Standards Agency, October 2020. http://dx.doi.org/10.46756/sci.fsa.bxn990.

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For meat and meat products, secondary processes are those that relate to the downstream of the primary chilling of carcasses. Secondary processes include maturation chilling, deboning, portioning, mincing and other operations such as thermal processing (cooking) that create fresh meat, meat preparations and ready-to-eat meat products. This review systematically identified and summarised information relating to antimicrobial resistance (AMR) during the manufacture of secondary processed meatand meat products (SPMMP). Systematic searching of eight literature databases was undertaken and the resultantpapers were appraised for relevance to AMR and SPMMP. Consideration was made that the appraisal scores, undertaken by different reviewers, were consistent. Appraisal reduced the 11,000 initially identified documents to 74, which indicated that literature relating to AMR and SPMMP was not plentiful. A wide range of laboratory methods and breakpoint values (i.e. the concentration of antimicrobial used to assess sensitivity, tolerance or resistance) were used for the isolation of AMR bacteria.The identified papers provided evidence that AMR bacteria could be routinely isolated from SPMMP. There was no evidence that either confirmed or refuted that genetic materials capable of increasing AMR in non-AMR bacteria were present unprotected (i.e. outside of a cell or a capsid) in SPMMP. Statistical analyses were not straightforward because different authors used different laboratory methodologies.However, analyses using antibiotic organised into broadly-related groups indicated that Enterobacteriaceaeresistant to third generation cephalosporins might be an area of upcoming concern in SPMMP. The effective treatment of patients infected with Enterobacteriaceaeresistant to cephalosporins are a known clinical issue. No AMR associations with geography were observed and most of the publications identified tended to be from Europe and the far east.AMR Listeria monocytogenes and lactic acid bacteria could be tolerant to cleaning and disinfection in secondary processing environments. The basis of the tolerance could be genetic (e.g. efflux pumps) or environmental (e.g. biofilm growth). Persistent, plant resident, AMR L. monocytogenes were shown by one study to be the source of final product contamination. 4 AMR genes can be present in bacterial cultures used for the manufacture of fermented SPMMP. Furthermore, there was broad evidence that AMR loci could be transferred during meat fermentation, with refrigeration temperatures curtailing transfer rates. Given the potential for AMR transfer, it may be prudent to advise food business operators (FBOs) to use fermentation starter cultures that are AMR-free or not contained within easily mobilisable genetic elements. Thermal processing was seen to be the only secondary processing stage that served as a critical control point for numbers of AMR bacteria. There were significant linkages between some AMR genes in Salmonella. Quaternary ammonium compound (QAC) resistance genes were associated with copper, tetracycline and sulphonamide resistance by virtue of co-location on the same plasmid. No evidence was found that either supported or refuted that there was any association between AMR genes and genes that encoded an altered stress response or enhanced the survival of AMR bacteria exposed to harmful environmental conditions.
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Sward, Jeffrey. Using a Microfluidic Approach to Analyze Bacteria Growth and Antibiotic Response. Office of Scientific and Technical Information (OSTI), July 2014. http://dx.doi.org/10.2172/1148303.

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Yu, David. The Replication Stress Response in Pancreatic Cancer. Fort Belvoir, VA: Defense Technical Information Center, October 2013. http://dx.doi.org/10.21236/ada599228.

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