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Books on the topic 'Stress response pathways'

Author: Grafiati

Published: 4 June 2021

Last updated: 20 February 2023

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1

Wondrak, Georg T., ed. Skin Stress Response Pathways. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-43157-4.

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Wondrak, Georg T., ed. Stress Response Pathways in Cancer. Dordrecht: Springer Netherlands, 2015. http://dx.doi.org/10.1007/978-94-017-9421-3.

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3

Herman, James P. Limbic Pathways to Stress Control. Edited by Israel Liberzon and Kerry J. Ressler. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190215422.003.0008.

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Appropriate control of the HPA (hypothalamo-pituitary-adrenocortical axis) is required for adaptation to physiological and environmental challenges. Inadequate control is linked to numerous stress-related pathologies, including PTSD, highlighting its importance in linking physiological stress responses with behavioral coping strategies. This chapter highlights neurocircuit mechanisms underlying HPA axis adaptation and pathology. Control of the HPA stress response is mediated by the coordinated activity of numerous limbic brain regions, including the prefrontal cortex, hippocampus, and amygdala

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4

Wondrak, Georg T. Skin Stress Response Pathways: Environmental Factors and Molecular Opportunities. Springer, 2018.

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5

Wondrak, Georg T. Skin Stress Response Pathways: Environmental Factors and Molecular Opportunities. Springer, 2016.

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6

Wondrak, Georg T. Skin Stress Response Pathways: Environmental Factors and Molecular Opportunities. Springer London, Limited, 2016.

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7

Wondrak, Georg T. Stress Response Pathways in Cancer: From Molecular Targets to Novel Therapeutics. Springer, 2016.

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8

Wondrak, Georg T. Stress Response Pathways in Cancer: From Molecular Targets to Novel Therapeutics. Springer, 2014.

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9

Wondrak, Georg T. Stress Response Pathways in Cancer: From Molecular Targets to Novel Therapeutics. Springer, 2014.

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10

Huang, Ruili, and Menghang Xia, eds. Tox21 Challenge to Build Predictive Models of Nuclear Receptor and Stress Response Pathways as Mediated by Exposure to Environmental Toxicants and Drugs. Frontiers Media SA, 2017. http://dx.doi.org/10.3389/978-2-88945-197-5.

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11

Laumbach, Robert, and Michael Gochfeld. Toxicology. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190662677.003.0007.

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This chapter describes the basic principles of toxicology and their application to occupational and environmental health. Topics covered include pathways that toxic substances may take from sources in the environment to molecular targets in the cells of the body where toxic effects occur. These pathways include routes of exposure, absorption into the body, distribution to organs and tissues, metabolism, storage, and excretion. The various types of toxicological endpoints are discussed, along with the concepts of dose-response relationships, threshold doses, and the basis of interindividual dif

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12

Barr, Christina S. Gene-by-Environment Interactions in Primates. Edited by Turhan Canli. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780199753888.013.006.

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Because of their complex social structures, behaviors, and genetic similarities to humans, nonhuman primates are useful for studying how genetic factors influence alcohol consumption. The neurobiological systems that influence addiction vulnerability may do so by acting on alcohol response, reward pathways, behavioral dyscontrol, and vulnerability to stress and anxiety. Rhesus macaques show individual differences in alcohol response and temperament, and such differences are influenced by genetic variants that are similar functionally to those present in humans. Genes in which variation moderat

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13

Heim, Christine, and Charles B. Nemeroff. Neurobiological Pathways Involved in Fear, Stress, and PTSD. Edited by Israel Liberzon and Kerry J. Ressler. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190215422.003.0012.

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The symptoms of post-traumatic stress disorder (PTSD) are believed to reflect an inadequate adaptation of neurobiological systems to exposure to severe stressors. A vast number of studies have revealed multiple alterations in neuroendocrine and neurochemical systems in patients with PTSD. It is now evident that certain neurobiological changes in PTSD actually reflect preexisting vulnerability factors that contribute to maladaptive physiological and behavioral responses to traumatic exposure, as well as altered learning and extinction of fear memories. These results suggest the development of n

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14

Ciano-Oliveira, Caterina Di. Signaling pathways linking osmotic stress to adaptive responses: Roles for Rho family GTPases. 2006.

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15

Rushton, Cynda Hylton. Mapping the Path of Moral Adversity. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190619268.003.0004.

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An alternative path for addressing moral adversity and the resultant moral suffering engages the focal points in a cycle of imperiled integrity in response to moral harms, wrongs, failures, or other forms of moral adversity. Initially moral stress, a neutral state of readiness to respond that will eventually involve an appraisal as positive or negative, may be experienced. Depending on this appraisal and individual capabilities, moral stress may be rebalanced, released, or resolved, engaging our moral resilience to proactively or prospectively respond to moral adversity. Alternatively, when th

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16

Lau, Hi-Po Bobo, and Cecilia Cheng. The Yin-Yang of Stress. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780199348541.003.0020.

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Dialectical thinking refers to the (primarily East Asian) tendency to tolerate contradiction, expect change, and perceive interconnections. Drawing upon a process-oriented approach to coping, in this chapter, two pathways through which dialectical thinking may influence East Asians’ ways of coping are proposed. First, dialectical (and holistic) thinking may enable East Asians to attribute events to both situational and dispositional factors. This breadth in attribution may lead to the perception of less personal control, as well as a weaker association between perceived personal control and co

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17

Goligorsky, Michael S., Julien Maizel, Radovan Vasko, May M. Rabadi, and Brian B. Ratliff. Pathophysiology of acute kidney injury. Edited by Norbert Lameire. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0221.

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In the intricate maze of proposed mechanisms, modifiers, modulators, and sensitizers for acute kidney injury (AKI) and diverse causes inducing it, this chapter focuses on several common and undisputable strands which do exist.Structurally, the loss of the brush border, desquamation of tubular epithelial cells, and obstruction of the tubular lumen are commonly observed, albeit to various degrees. These morphologic hallmarks of AKI are accompanied by functional defects, most consistently reflected in the decreased glomerular filtration rate and variable degree of reduction in renal blood flow, a

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18

Miu, Andrei C., Judith R. Homberg, and Klaus-Peter Lesch, eds. Genes, brain, and emotions. Oxford University Press, 2019. http://dx.doi.org/10.1093/oso/9780198793014.001.0001.

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With the advent of methods from behavioral genetics, molecular biology, and cognitive neuroscience, affective science has recently started to approach genetic influences on emotion, and the underlying intermediate neural mechanisms through which genes and experience shape emotion. The aim of this volume is to offer a comprehensive account of current research in the genetics of emotion, written by leading researchers, with extensive sections focused on methods, intermediate phenotypes, and clinical and translational work. Major methodological approaches are reviewed in the first section, includ

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19

Carpenter, Gregory, and Meenal Patil. Gender Differences in Pain. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190217518.003.0005.

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Epidemiologic and clinical findings demonstrate that women are at increased risk for chronic pain, experience greater pain-related distress, and show heightened sensitivity for pain compared to men. There are differences in analgesic responses to pain and to both opioid and non-opioid medications as well as for endogenous analgesic processes. Many stress-related disorders, such as fibromyalgia and chronic pain, are more prevalent in women. Studies of experimentally induced pain show that women exhibit greater pain sensitivity, enhanced pain facilitation, and reduced pain inhibition compared to

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20

Major, Brenda, John F. Dovidio, Bruce G. Link, and Sarah K. Calabrese. Stigma and Its Implications for Health: Introduction and Overview. Edited by Brenda Major, John F. Dovidio, and Bruce G. Link. Oxford University Press, 2017. http://dx.doi.org/10.1093/oxfordhb/9780190243470.013.1.

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There is growing recognition that stigma plays an important role in producing health disparities between members of socially advantaged and disadvantaged (marginalized) groups. This chapter defines stigma, describes differences among stigmatized marks, and discusses the functions that stigma may serve for individuals, groups, and societies. It also provides a conceptual model of the pathways by which stigma relates to health. This model posits that socially conferred marks that are devalued in society are the basis for four key stigma processes: enacted stigma, felt stigma, internalized stigma

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21

Neuberg, Steven L., and Andreana C. Kenrick. Discriminating Ecologies: A Life History Approach to Stigma and Health. Edited by Brenda Major, John F. Dovidio, and Bruce G. Link. Oxford University Press, 2017. http://dx.doi.org/10.1093/oxfordhb/9780190243470.013.5.

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How does being discriminated against affect one’s health, and through what mechanisms? Most research has focused on two causal pathways, highlighting how discrimination increases psychological stress and exposure to neighborhood hazards. This chapter advances an alternative, complementary set of mechanisms through which stigma and discrimination may shape health. Grounded in evolutionary biology’s life history theory, the framework holds that discrimination alters aspects of the physical and social ecologies in which people live (e.g., sex ratio, unpredictable extrinsic causes of mortality). T

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22

Major, Brenda, John F. Dovidio, and Bruce G. Link, eds. The Oxford Handbook of Stigma, Discrimination, and Health. Oxford University Press, 2017. http://dx.doi.org/10.1093/oxfordhb/9780190243470.001.0001.

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Throughout the world, groups that are socially disadvantaged have poorer health compared to groups that are more advantaged. This book examines the role that stigma and discrimination play in creating and sustaining these group health disparities. Stigma is a social construction in which people who are distinguished by a “mark” are viewed as deviant, socially excluded, and devalued. Stigma and the discrimination it engenders negatively affect health through multiple mechanisms operating at several different levels of influence. Collectively, these shape both the orientations of people toward m

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23

Nielsen, David A., Dmitri Proudnikov, and Mary Jeanne Kreek. The Genetics of Impulsivity. Edited by Jon E. Grant and Marc N. Potenza. Oxford University Press, 2012. http://dx.doi.org/10.1093/oxfordhb/9780195389715.013.0080.

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Impulsivity is a complex trait that varies across healthy individuals, although when excessive, it is generally regarded as dysfunctional. Impulsive behavior may lead to initiation of drug addiction that interferes with inhibitory controls, which may in turn result in facilitation of the individual’s impulsive acts. Although environmental factors play a considerable role in impulsive behavior, a body of evidence collected in twin studies suggests that about 45% of the variance in impulsivity is accounted for by genetic factors. Genetic variants studied in association with impulsivity include t

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