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Relevant bibliographies by topics / Stria Vascularis

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Journal articles
Dissertations / Theses
Book chapters
Conference papers

Academic literature on the topic 'Stria Vascularis'

Author: Grafiati

Published: 4 June 2021

Last updated: 5 February 2022

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Journal articles on the topic "Stria Vascularis"

1

Castaldo, Antonella, and Fred H. Linthicum. "Stria Vascularis Hearing Loss." Otology & Neurotology 27, no. 2 (2006): 285–86. http://dx.doi.org/10.1097/00129492-200602000-00025.

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Labyrinth, Guinea Pig, Mitsuya Suzuki, and Kimitaka Raga. "Effect of Furosemide on Basal Lamina Anionic Sites in." Annals of Otology, Rhinology & Laryngology 110, no. 3 (2001): 283–89. http://dx.doi.org/10.1177/000348940111000315.

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The authors studied the effects of acute furosemide administration on the basal lamina (BL) anionic sites in the stria vascularis, ampullar crista, and endolymphatic sac by using cationic polyethyleneimine (PEI). Furosemide was intravenously administered to albino guinea pigs with normal Preyer's reflexes. After 20 minutes, the bony labyrinth was removed and processed for histologic evaluation. Under a transmission electron microscope, a marked enlargement of the intercellular spaces was observed in the stria vascularis. The PEI distribution decreased significantly on the capillary BL in the s

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Steel, K. P., and C. Barkway. "Another role for melanocytes: their importance for normal stria vascularis development in the mammalian inner ear." Development 107, no. 3 (1989): 453–63. http://dx.doi.org/10.1242/dev.107.3.453.

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The stria vascularis of the mammalian cochlea is composed primarily of three types of cells. Marginal cells line the lumen of the cochlear duct and are of epithelial origin. Basal cells also form a continuous layer and they may be mesodermal or derived from the neural crest. Intermediate cells are melanocyte-like cells, presumably derived from the neural crest, and are scattered between the marginal and basal cell layers. The marginal cells form extensive interdigitations with the basal and intermediate cells in the normal adult stria. The stria also contains a rich supply of blood vessels. We

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Mou, Kewa, Crista L. Adamson, and Robin L. Davis. "Stria vascularis morphogenesis in vitro." Hearing Research 103, no. 1-2 (1997): 47–62. http://dx.doi.org/10.1016/s0378-5955(96)00163-3.

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Peng, Kevin A., and Fred H. Linthicum. "Atrophy of the Stria Vascularis." Otology & Neurotology 37, no. 2 (2016): e9-e11. http://dx.doi.org/10.1097/mao.0000000000000935.

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Carraro, Mattia, Jaina Negandhi, Jafri Kuthubutheen, et al. "Attenuating Cardiac Pulsations within the Cochlea: Structure and Function of Tortuous Vessels Feeding Stria Vascularis." ISRN Otolaryngology 2013 (May 19, 2013): 1–7. http://dx.doi.org/10.1155/2013/941757.

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The mammalian ear has an extraordinary capacity to detect very low-level acoustic signals from the environment. Sound pressures as low as a few μPa (−10 dB SPL) can activate cochlear hair cells. To achieve this sensitivity, biological noise has to be minimized including that generated by cardiovascular pulsation. Generally, cardiac pressure changes are transmitted to most peripheral capillary beds; however, such signals within the stria vascularis of the cochlea would be highly disruptive. Not least, it would result in a constant auditory sensation of heartbeat. We investigate special adaptati

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Chen, Linjun, Lin Wang, Lei Chen, et al. "Transcript Profiles of Stria Vascularis in Models of Waardenburg Syndrome." Neural Plasticity 2020 (August 1, 2020): 1–9. http://dx.doi.org/10.1155/2020/2908182.

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Background. Waardenburg syndrome is an uncommon genetic condition characterized by at least some degree of congenital hearing loss and pigmentation deficiencies. However, the genetic pathway affecting the development of stria vascularis is not fully illustrated. Methods. The transcript profile of stria vascularis of Waardenburg syndrome was studied using Mitf-M mutant pig and mice models. Therefore, GO analysis was performed to identify the differential gene expression caused by Mitf-M mutation. Results. There were 113 genes in tyrosine metabolism, melanin formation, and ion transportations sh

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Watanabe, Kensuke. "Ultrastructural Characteristics of Capillaries Entering and Leaving the Stria Vascularis." Annals of Otology, Rhinology & Laryngology 95, no. 3 (1986): 309–12. http://dx.doi.org/10.1177/000348948609500320.

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Capillaries entering and leaving the stria vascularis were surrounded by layers of basal cells and fibrocytes. The entering capillaries were surrounded by one or two thin basal cells, while the leaving capillaries were surrounded by four or five thicker and interdigitated basal cell layers. Moreover, the layers surrounding the leaving capillaries persisted further into the spiral ligament. Two kinds of filaments were observed in the basal cells, one thin and the other thick. Capillaries were observed to leak horseradish peroxidase before they entered and after they left the stria vascularis. A

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Marcus, Daniel C., Tao Wu, Philine Wangemann, and Paulo Kofuji. "KCNJ10 (Kir4.1) potassium channel knockout abolishes endocochlear potential." American Journal of Physiology-Cell Physiology 282, no. 2 (2002): C403—C407. http://dx.doi.org/10.1152/ajpcell.00312.2001.

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Stria vascularis of the cochlea generates the endocochlear potential and secretes K+. K+ is the main charge carrier and the endocochlear potential the main driving force for the sensory transduction that leads to hearing. Stria vascularis consists of two barriers, marginal cells that secrete potassium and basal cells that are coupled via gap junctions to intermediate cells. Mice lacking the KCNJ10 (Kir4.1) K+ channel in strial intermediate cells did not generate an endocochlear potential. Endolymph volume and K+ concentration ([K+]) were reduced. These studies establish that the KCNJ10 K+ chan

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Pillutla, Sree Vani Poorna, Charanjeet Kaur, Tony George Jacob, Daya Nand Bhardwaj, and Tara Sankar Roy. "Morphology of adult human stria vascularis." Journal of the Anatomical Society of India 66 (August 2017): S116—S117. http://dx.doi.org/10.1016/j.jasi.2017.08.371.

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Dissertations / Theses on the topic "Stria Vascularis"

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Soulter, Mark. "The postnatal maturation of the organ of Corti and stria vascularis in the gerbil." Thesis, University College London (University of London), 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.244162.

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Jin, Zhe. "Characterization of cochlear degeneration in the inner ear of the German waltzing guinea pig : a morphological, cellular, and molecular study /." Stockholm, 2006. http://diss.kib.ki.se/2006/91-7140-971-8/.

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DiSalvo, Maribeth. "Effects of Cardiovascular Health on Hearing Levels Among Musicians." Miami University / OhioLINK, 2003. http://rave.ohiolink.edu/etdc/view?acc_num=miami1051043965.

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Abou-Khalil, Rana. "Interactions entre les cellules satellites et les cellules vasculaires au sein du muscle strié squelettique : implications dans la myogénèse et la quiescence." Thesis, Paris Est, 2009. http://www.theses.fr/2009PEST0038.

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Dans le muscle squelettique adulte, les cellules souches du muscle, nommées les cellules satellites, résident sous la lame basale des fibres musculaires à l’état quiescent jusqu’à ce qu’un dommage musculaire induise leur activation. Après une phase d’activation, les cellules satellites sont capables de proliférer et de se différencier afin de répondre aux besoins des myonucléi au cours de la régénération musculaire. Les cellules satellites, ou au moins une sous-population, sont actuellement considérées comme la principale population de cellules souches du muscle. Des cellules stromales ont été

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Latroche, Claire. "Organization and function of the vascular network and its interactions with satellite cells in normal and pathological muscle." Thesis, Sorbonne Paris Cité, 2015. http://www.theses.fr/2015USPCB121/document.

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Le muscle strié squelettique est un tissu richement vascularisé. Cependant, les vaisseaux ne sont pas seulement des pourvoyeurs d'oxygène et de nutriments, mais participent activement à l'homéostasie tissulaire en interagissant avec les cellules souches musculaires. La Dystrophie Musculaire de Duchenne (DMD) est une pathologie associée à un dommage musculaire, des cycles de nécrose/régénération, un remodelage tissulaire et une plasticité vasculaire. Nous avons en effet, démontré par des études in vivo un défaut d'organisation du réseau vasculaire en 3D grâce au croisement entre une souris mdx

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Bertoldi, Didier. "Apport méthodologique aux mesures de la perfusion et du métabolisme énergétique par RMN in vivo chez le petit animal et applications à l'exploration fonctionnelle du muscle strié squelettique squelettique." Paris 6, 2006. http://www.theses.fr/2006PA066521.

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Book chapters on the topic "Stria Vascularis"

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Kangelaris, Gerald T., and Lawrence R. Lustig. "Stria Vascularis." In Encyclopedia of Otolaryngology, Head and Neck Surgery. Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-23499-6_200091.

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Gitter, A. H., I. Melichar, and M. Ptok. "Kultivierung lebender Marginalzellen der Stria vascularis." In Teil II: Sitzungsbericht. Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-84592-5_333.

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Ernst, A., H. J. Mest, and P. Braquet. "Lipidmediatoren beeinflussen Ionentransportvorgänge in der Stria vascularis des Meerschweinchens." In Teil II: Sitzungsbericht. Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-84592-5_20.

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Krstić, R. V. "Gefäßhaltiges sezernierendes Oberflächenepithel. Einziges Beispiel: Epithel der Stria vascularis des Innenohres." In Die Gewebe des Menschen und der Säugetiere. Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-61380-7_26.

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Matthias, R., and O. Michel. "Einfluß der Beschallung auf die Lipidperoxidation in der Stria vascularis von Meerschweinchen." In Teil II: Sitzungsbericht. Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-84592-5_21.

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Krstić, Radivoj V. "Vascularized Secretory Surface Epithelium. Sole Example: Epithelium of Stria Vascularis of Inner Ear." In General Histology of the Mammal. Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-70420-8_26.

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Galić, M., W. Giebel, and C. Brunner. "Quantitative Morphometrie zur Degeneration der Stria vascularis und des Ligamentum spirale bei Verschluß der Kochleagefäße." In Teil II: Sitzungsbericht. Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-83931-3_233.

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Koch, T. "Medikamentöse Hemmung der G-Protein assoziierten Adenylatzyklase in der Stria vascularis — ein Erklärungsmodell für die ototoxische Innenohrschwerhörigkeit?" In Teil II: Sitzungsbericht. Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-84310-5_116.

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Stamper, Robert L. "Hypotony Maculopathy." In Complications of Glaucoma Surgery. Oxford University Press, 2013. http://dx.doi.org/10.1093/oso/9780195382365.003.0037.

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Hypotony is often defined as intraocular pressure (IOP) less than 6 mm Hg. It has been reported to occur after glaucoma filtering surgery in up to 42% of cases and is usually associated with overfiltration or wound leaks. Hypotony requiring revision, however, occurs in about 4% of filtering procedures. Hypotony can follow any IOP-lowering procedure or even “simple” cataract surgery. The advent of guarded filtering surgery has reduced the rate of hypotony significantly compared to full-thickness filtering surgery. Unfortunately in the quest to increase success rates by using adjunctive antifibrotic agents, such as mitomycin-C (MMC) or 5-fluorouracil (5-FU), that prevent fibrotic wound healing, the incidence has increased again. Higher doses of and longer exposure times to MMC are associated with a greater risk of hypotony. Most cases of hypotony are transient and self-limited to a few days or weeks after surgery. Transient hypotony does not seem to have any deleterious effect on long-term visual acuity. However, persistent hypotony may result in structural changes that can become permanent. Hypotony maculopathy is one such condition manifesting from persistent hypotony that can result in permanent vision loss. Hypotony maculopathy occurs in up to 10% of filtering operations with MMC or 5-FU and in about 10% of eyes with chronic hypotony. Maculopathy associated with hypotony was first described by Dellaporta. Some years later, Gass, using fluorescein angiography, better characterized the condition. In hypotony maculopathy, the sclera and choroid develop folds in the posterior pole, which can cause significant visual disturbances. The condition is recognized by characteristic striae or folds in the macular area that do not leak or stain with fluorescein. The posterior sclera appears partially collapsed, causing the folds. The axial length of the eye may be shortened after both filtering and tube shunt surgery and more so in patients with hypotony. The loss of vision is usually gradual after the hypotony has persisted for at least a month or more. Indocyanine green angiography has revealed some vascular abnormalities including vessel tortuosity and filling defects.

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Conference papers on the topic "Stria Vascularis"

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Kindel, G., J. Fareed, and U. Cornelli. "EFFECTS OF DEFIBROTIDE TREATMENT ON THE PLASMA AND SERUM INDUCED CONTRACTION OF RABBIT AORTIC STRIP IN RELATION TO THE PATHOPHYSIOLOGY OF MYOCARDIAL INFARCTION." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643147.

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Several clinical and experimental studies have demonstrated the efficacy of a polydeoxyribonucleotide (Defibrotide) in myocardial ischemic disorders and peripheral arterial diseases. In attempts to investigate the mechanisms of action of this agent, we used a modified rabbit model of hemodynamics and an isolated rabbit aortic strip preparation. Intravenous bolus administration of Defibrotide at 5-50 mg/kg did not produce any changes in the mean arterial blood pressure up to 3 hours. Defibrotide treated rabbits also resisted human serum-stasis and stasis alone induced venous and arterial thromb

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Kindel, G., and J. Fareed. "MODULATORY EFFECT OF SERINE PROTEASES AND RELATED ENZYMES ON ISOLATED SMOOTH MUSCLE PREPARATIONS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644602.

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Thrombin and related proteases produce varying pharmacologic responses in animal models. To more specifically study the in vivo actions of thrombin and related proteases, we have used isolated tissue preparations of the rabbit aortic strip (RAS), isolated guinea pig ileum (GPI) and isolated rat uterus (RU). Standard tissue-agonist regimens include epinephrine, thromboxane B2 with RAS; bradykinin, acetylcholine, histamine and serotonin with GPI; and acetylcholine, bradykinin and angiotensin with RU. The smooth muscle modulant action of numerous proteinases were screened in these regimens by bra

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