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Relevant bibliographies by topics / Stroke/cerebral ischaemia / Dissertations / Theses

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Dissertations / Theses on the topic 'Stroke/cerebral ischaemia'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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1

Bowler, John Vaughan. "Cerebral infarction and '9'9Tc'm HMPAO SPECT." Thesis, King's College London (University of London), 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.260983.

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2

Sibson, Nicola Ruth. "A magnetic resonance imaging study of experimental cerebral ischaemia." Thesis, University of Cambridge, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.360825.

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3

Taylor, Deanna Lesley. "Alterations in interstitial acid-base homeostasis during cerebral ischaemia." Thesis, University College London (University of London), 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.267025.

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4

Greenhalgh, Andrew. "Actions of interleukin-1 receptor antagonist in cerebral ischaemia." Thesis, University of Manchester, 2011. https://www.research.manchester.ac.uk/portal/en/theses/actions-of-interleukin1-receptor-antagonist-in-cerebral-ischaemia(50aacd97-68c1-4f91-90b5-8f8deff5d21d).html.

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Cerebral ischaemia, or stroke, is a leading cause of death and disability worldwide. Ischaemic stroke, as a result of arterial occlusion, and subarachnoid haemorrhage (SAH), as a consequence of arterial rupture in the subarachnoid space, are major subtypes of stroke. Treatment options for both are limited, and many therapeutic strategies have failed. In ischaemic stroke, lack of evidence of brain penetration of treatments has been cited as a major weakness and contributing factor to failed clinical trials. In SAH, animal models do not always mimic key pathophysiological hallmarks of the diseas

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5

Marquardt, Lars. "Large artery disease in patients with cerebral ischaemia : frequency, investigation and management." Thesis, University of Oxford, 2010. http://ora.ox.ac.uk/objects/uuid:70b598c5-97ca-4567-ac32-ed5092972a16.

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Stroke is the third leading cause of death in the developed world and is the leading neurological cause of disability with a massive impact on personal life and society. Large artery atherosclerosis is one of the main causes of ischaemic stroke. However, in several aspects of this condition there is still a significant amount of uncertainty about its prevalence, appropriate investigation and possible treatment. Reliable data on epidemiology are therefore necessary to provide clinicians and researchers with crucial information to guide diagnostic and therapeutic management as well as further re

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6

Calamante, Fernando. "Diffusion and perfusion MRI and applications in cerebral ischaemia." Thesis, University College London (University of London), 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.314345.

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7

Ben-Yoseph, O. "Multinuclear magnetic resonance spectroscopy studies of perturbed cerebral metabolism in vitro." Thesis, University of Cambridge, 1991. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.240078.

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8

Thomas, David Lee. "Magnetic resonance imaging of diffusion and perfusion : techniques and applications to cerebral ischaemia." Thesis, University College London (University of London), 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.391829.

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9

Smith, Stuart Ernest. "Excitatory amino acid antagonists and related agents as potential therapies in focal cerebral ischaemia in the rat." Thesis, King's College London (University of London), 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.281943.

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10

Milidonis, Xenios. "Multicentre evaluation of MRI variability in the quantification of infarct size in experimental focal cerebral ischaemia." Thesis, University of Edinburgh, 2017. http://hdl.handle.net/1842/28877.

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Ischaemic stroke is a leading cause of death and disability in the developed world. Despite that considerable advances in experimental research enabled understanding of the pathophysiology of the disease and identified hundreds of potential neuroprotective drugs for treatment, no such drug has shown efficacy in humans. The failure in the translation from bench to bedside has been partially attributed to the poor quality and rigour of animal studies. Recently, it has been suggested that multicentre animal studies imitating the design of randomised clinical trials could improve the translation o

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11

Young, Christopher Cheng. "The adult neural stem cell niche in ischaemic stroke." Thesis, University of Oxford, 2011. http://ora.ox.ac.uk/objects/uuid:86e6e236-047c-46d8-96e5-449a3f0505a8.

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Ischaemic stroke is a major cause of mortality and chronic disability for which there is no effective treatment. The subventricular zone (SVZ) is an adult neurogenic niche which mediates limited endogenous repair following stroke. To harness this phenomenon for therapy, it is important to understand how the SVZ niche is altered in stroke, and the processes that recruit neural precursors to the site of injury, which becomes a de facto neurogenic niche. Galectin-3 (Gal-3) is a β-galactoside binding protein involved in cellular adhesion, inflammation and tumour metastasis. Gal-3 is specifica

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12

Jeffs, Graham J. "The effect of sodium/calcium exchanger 3 (NCX3) knockout on neuronal survival following global cerebral ischaemia in mice." University of Western Australia. School of Biomedical, Biomolecular and Chemical Sciences, 2007. http://theses.library.uwa.edu.au/adt-WU2008.0063.

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Cerebral ischaemia is a leading cause of disability and death world-wide. The only effective treatments are thrombolytic therapy (plasminogen activator; tPA) and hypothermia (33?C). However, tPA has limited clinical application due to its short therapeutic time window and its specific application in thrombo-embolic stroke. Moderate hypothermia (33?C) is only being used following cardiac arrest in comatose survivors. Hence more treatments are urgently required. The first step in developing new treatments is the identification and characterisation of a potential therapeutic target. Since brain d

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13

von, Sarnowski Bettina, Ulf Schminke, Ulrike Grittner, Franz Fazekas, Christian Tanislav, Manfred Kaps, Turgut Tatlisumak, et al. "Cervical Artery Dissection in Young Adults in the Stroke in Young Fabry Patients (sifap1) Study." Karger, 2015. https://tud.qucosa.de/id/qucosa%3A70584.

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Background: Patients with carotid artery dissection (CAD) have been reported to have different vascular risk factor profiles and clinical outcomes to those with vertebral artery dissection (VAD). However, there are limited data from recent, large international studies comparing risk factors and clinical features in patients with cervical artery dissection (CeAD) with other TIA or ischemic stroke (IS) patients of similar age and sex. Methods: We analysed demographic, clinical and risk factor profiles in TIA and IS patients ≤ 55 years of age with and without CeAD in the large European, multi-ce

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14

Gubern, Mérida M. Carme. "Nuevos determinantes moleculares en la respuesta a isquemia cerebral." Doctoral thesis, Universitat de Girona, 2015. http://hdl.handle.net/10803/362102.

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Cerebral ischaemia is a potent inducer of gene expression, however, little is known about the mechanisms that regulate this expression. Studies of gene expression and the study of the mechanisms regulating this expression allow identifying new molecules regulated by ischaemia and possibly involved in the physiopathology of stroke. This thesis shows, (1) the importance of validating suitable endogenous controls for the studies of gene expression for each experimental condition; (2)that Gcf2/Lrrfip1 presents as a gene with diverse isoforms that are differentially expressed in response to stroke

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15

Overell, James Richard. "Vascular investigation and management of ischaemic stroke." Thesis, University of Glasgow, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.394761.

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16

Pogoryelova, Oksana. "A study of epigenetics in ischaemic stroke." Thesis, University of Aberdeen, 2013. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=201969.

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Ischaemic stroke rates are expected to rise significantly in the next decades due to an aging population. This increases the demand for new stroke biomarkers for early detection of patients at risk and new targets for treatment. It has been hypothesized that epigenetics may be important in the aetiology of stroke. The study consisted of three types of investigation: analysis of candidate gene polymorphism, candidate gene methylation analysis and epigenome-wide methylation analysis (EWAS) of pooled stroke and control samples. The stroke types studied were large vessel disease (LVD), small vesse

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17

Dawson, Suzanne L. "Blood pressure variability and cerebral autoregulation in acute ischaemic stroke." Thesis, University of Leicester, 2000. http://hdl.handle.net/2381/29609.

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This thesis examines the haemodynamic changes that occur following acute ischaemic stroke with reference to beat-to-beat blood pressure (BP) levels and variability as well as static and dynamic cerebral autoregulation (CA). Elevated 24h BP levels following acute stroke are associated with a poor outcome, but whether shorter durations of recording using beat-to-beat measurements or other BP parameters such as variability have the same prognostic significance is unknown. A single 10 minute non-invasive beat-to-beat BP monitoring period following acute cerebral infarction showed that increasing B

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18

Schulz, Ursula Gabriele Renate. "Risk factors for specific subtypes of ischaemic stroke." Thesis, University of Oxford, 2004. http://ora.ox.ac.uk/objects/uuid:43200f6f-abe0-4b1e-84a2-31df49707b6f.

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Ischaemic stroke is a complex disorder with many different aetiologies, but previous studies of stroke often did not differentiate aetiological subtypes of ischaemic stroke. However, different stroke subtypes may have different risk factors, and to target preventive treatments more effectively, we need to understand these associations. I studied the association of established vascular risk factors with different aetiological stroke subtypes in population-based cohorts of stroke patients. I studied Diffusion Weighted Magnetic Resonance Imaging (DWI) in patients with subacute minor stroke and TI

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19

Arroja, Mariana Moreira. "Cerebral damage following ischaemic stroke : the role of Angiotensin-(1-7)." Thesis, University of Glasgow, 2018. http://theses.gla.ac.uk/9010/.

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The renin angiotensin system (RAS), a homeostatic system involved in blood pressure and volume control, is implicated in the pathology of several risk factors for ischaemic stroke. Mounting evidence now suggests that the RAS may play a role in the pathophysiology of ischaemic stroke. This is thought to be due to an imbalance between the classical RAS axis, Angiotensin converting enzyme/Angiotensin II/Angiotensin II receptor type I (ACE/Ang II/AT1R), and the counter-regulatory RAS axis, Angiotensin converting enzyme 2/Angiotensin-(1-7)/Mas receptor [ACE2/Ang-(1-7)/MasR]. The counter- regulatory

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20

Shrewsbury-Gee, Joanne. "An investigation of compounds of potential value in experimental cerebral ischaemia." Thesis, University of Manchester, 1988. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.329110.

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21

Hassan, Ahmad. "The genetics of ischaemic stroke and the cerebral small vessel disease subtype." Thesis, St George's, University of London, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.407965.

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22

Hamilton-Bruce, Monica Anne. "Conventional and topographic electroencephalography and somatosensory evoked potential studies in ischaemic stroke." Adelaide, 1998. http://web4.library.adelaide.edu.au/theses/09PH/09phh222.pdf.

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Copies of author's previously published articles inserted. Bibliography: leaves I-LXIV. Assesses the diagnostic and prognostic value of early electroencephalography (EEG) and somatosensory evoked potential studies in cortical and non-cortical ischaemic stroke. Both conventional and topographic/quantitative studies were performed. A parallel study was carried out on healthy volunteers to provide an effective control. Equipment and quantitative EEG (qEEG) variability was also assessed.

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23

Salomão, Macedo Salinet Angela. "The natural history of the cerebral blood flow regulation after acute ischaemic stroke." Thesis, University of Leicester, 2014. http://hdl.handle.net/2381/28578.

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Acute stroke is known to lead to impairment of cerebral blood flow (CBF) regulation, but its natural history and techniques for its comprehensive assessment have not been previously reported. Noninvasive measurements of blood pressure (BP), end-tidal CO2 and CBF velocity (CBFv, using transcranial Doppler ultrasound) during active, passive and motor imagery paradigms were performed in healthy older controls (n=27) and in stroke patients (n=27). Two innovative analytical techniques were firstly used in stroke studies: subcomponent analysis and multivariate dynamic modeling. In controls, signific

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24

Aspey, Benjamin Steven. "Studies of an ischaemic stroke model (middle cerebral artery occlusion in the rat) and modifications to improve its consistency." Thesis, University College London (University of London), 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.392357.

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25

Msayib, Yunus. "Quantifying impaired metabolism following acute ischaemic stroke using chemical exchange saturation transfer magnetic resonance imaging." Thesis, University of Oxford, 2017. http://ora.ox.ac.uk/objects/uuid:a98323ce-5998-436d-bca4-09df549cf191.

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In ischaemic stroke a disruption of cerebral blood flow leads to impaired metabolism and the formation of an ischaemic penumbra in which tissue at risk of infarction is sought for clinical intervention. In stroke trials, therapeutic intervention has largely been based on perfusion-weighted measures, but these have not been shown to be good predictors of tissue outcome. The aim of this thesis was to develop analysis techniques for magnetic resonance imaging (MRI) of chemical exchange saturation transfer (CEST) in order to quantify metabolic signals associated with tissue fate in patients with a

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26

Turner, Renée Jade. "Characterising the role of substance P in acute ischaemic stroke." 2007. http://hdl.handle.net/2440/56839.

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More than 15 million people worldwide will suffer a stroke each year two thirds will die or be left permanently disabled. Accordingly, stroke represents an enormous financial burden on the community, due to the cost of hospitalisation, treatment and rehabilitation of stroke patients. Despite the significance of this public health problem, a safe and widely applicable stroke therapeutic remains elusive. Cerebral oedema is widely recognised as a common and often fatal complication of stroke that is associated with worsened outcome. However, the exact mechanisms of oedema formation remain unclear

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