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Relevant bibliographies by topics / Stroke, mice, recovery, biomarker

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Journal articles
Dissertations / Theses
Book chapters

Academic literature on the topic 'Stroke, mice, recovery, biomarker'

Author: Grafiati

Published: 10 March 2023

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Journal articles on the topic "Stroke, mice, recovery, biomarker"

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Cecchini, Gloria, Alessandro Scaglione, Anna Letizia Allegra Mascaro, et al. "Cortical propagation tracks functional recovery after stroke." PLOS Computational Biology 17, no. 5 (2021): e1008963. http://dx.doi.org/10.1371/journal.pcbi.1008963.

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Stroke is a debilitating condition affecting millions of people worldwide. The development of improved rehabilitation therapies rests on finding biomarkers suitable for tracking functional damage and recovery. To achieve this goal, we perform a spatiotemporal analysis of cortical activity obtained by wide-field calcium images in mice before and after stroke. We compare spontaneous recovery with three different post-stroke rehabilitation paradigms, motor training alone, pharmacological contralesional inactivation and both combined. We identify three novel indicators that are able to track how m

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Zaidi, Syed Kashif, Md Nasrul Hoda, Shams Tabrez, and Mohammad Imran Khan. "Pharmacological Inhibition of Class III Alcohol Dehydrogenase 5: Turning Remote Ischemic Conditioning Effective in a Diabetic Stroke Model." Antioxidants 11, no. 10 (2022): 2051. http://dx.doi.org/10.3390/antiox11102051.

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The restoration of cerebral blood flow (CBF) to achieve brain tissue oxygenation (PbtO2) is the primary treatment for ischemic stroke, a significant cause of adult mortality and disability worldwide. Nitric oxide (NO) and its bioactive s-nitrosylated (SNO) reservoirs, such as s-nitrosoglutathione (GSNO), induce hypoxic vasodilation to enhance CBF during ischemia. The endogenous pool of SNOs/GSNO is enhanced via the activation of endothelial NO synthase (eNOS/NOS3) and by the suppression of class III alcohol dehydrogenase 5 (ADH5), also known as GSNO reductase (GSNOR). Remote ischemic condition

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Zhao, Haiping, Guangwen Li, Sijia Zhang, et al. "Inhibition of histone deacetylase 3 by MiR-494 alleviates neuronal loss and improves neurological recovery in experimental stroke." Journal of Cerebral Blood Flow & Metabolism 39, no. 12 (2019): 2392–405. http://dx.doi.org/10.1177/0271678x19875201.

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HDAC3 is an essential negative regulator of neuronal plasticity and memory formation. Although a chemical inhibitor has been invented, little is known about its endogenous modulators. We explored whether miR-494 affects HDAC3-mediated neuronal injury following acute ischemic stroke. A substantial increase in plasma miR-494 was detected in AIS patients and was positively associated with the mRS at one year after symptom onset. The miR-494 levels were transiently increased in the infarcted brain tissue of mice. In contrast, miR-494 levels were reduced in neurons but increased in the medium after

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Leon, Lisa R., David A. DuBose, and Clifford W. Mason. "Heat stress induces a biphasic thermoregulatory response in mice." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 288, no. 1 (2005): R197—R204. http://dx.doi.org/10.1152/ajpregu.00046.2004.

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Previous animal models of heat stress have been compromised by methodologies, such as restraint and anesthesia, that have confounded our understanding of the core temperature (Tc) responses elicited by heat stress. Using biotelemetry, we developed a heat stress model to examine Tcresponses in conscious, unrestrained C57BL/6J male mice. Before heat stress, mice were acclimated for >4 wk to an ambient temperature (Ta) of 25°C. Mice were exposed to Taof 39.5 ± 0.2°C, in the absence of food and water, until they reached maximum Tcof 42.4 ( n = 11), 42.7 ( n = 12), or 43.0°C ( n = 11), defined a

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Garcia, Christian K., Gerard P. Robinson, Bryce J. Gambino, Michael T. Rua, Orlando Laitano, and Thomas L. Clanton. "The impact of castration on physiological responses to exertional heat stroke in mice." PLOS ONE 17, no. 10 (2022): e0275715. http://dx.doi.org/10.1371/journal.pone.0275715.

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Introduction The capability of male mice to exercise in hot environments without succumbing to exertional heat stroke (EHS) is markedly blunted compared to females. Epidemiological evidence in humans and other mammals also suggests some degree of greater vulnerability to heat stroke in males compared to females. The origins of these differences are unknown, but testosterone has previously been shown to induce faster elevations in core temperature during acute, passive heat exposure. In this study, we tested the hypothesis that loss of testosterone and related sex hormones through castration wo

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Rodriguez-Grande, Beatriz, Matimba Swana, Loan Nguyen, et al. "The Acute-Phase Protein PTX3 is an Essential Mediator of Glial Scar Formation and Resolution of Brain Edema after Ischemic Injury." Journal of Cerebral Blood Flow & Metabolism 34, no. 3 (2013): 480–88. http://dx.doi.org/10.1038/jcbfm.2013.224.

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Acute-phase proteins (APPs) are key effectors of the immune response and are routinely used as biomarkers in cerebrovascular diseases, but their role during brain inflammation remains largely unknown. Elevated circulating levels of the acute-phase protein pentraxin-3 (PTX3) are associated with worse outcome in stroke patients. Here we show that PTX3 is expressed in neurons and glia in response to cerebral ischemia, and that the proinflammatory cytokine interleukin-1 (IL-1) is a key driver of PTX3 expression in the brain after experimental stroke. Gene deletion of PTX3 had no significant effect

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Grosman, Benyamin, Osman S. Shaik, Bryan G. Helwig, Lisa R. Leon, and Francis J. Doyle. "A physiological systems approach to modeling and resetting of mouse thermoregulation under heat stress." Journal of Applied Physiology 111, no. 3 (2011): 938–45. http://dx.doi.org/10.1152/japplphysiol.00519.2010.

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Heat stroke (HS) is a serious civilian and military health issue. Due to the limited amount of experimental data available in humans, this study was conducted on a mouse mathematical model fitted on experimental data collected from mice under HS conditions, with the assumption there is good agreement among mammals. Core temperature (Tc) recovery responses in a mouse model consist of hypothermia and delayed fever during 24 h of recovery that represent potential biomarkers of HS severity. The objective of this study was to develop a simulation model of mouse Tc responses and identify optimal tre

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Vaibhav, Kumar, Molly Braun, Mohammad Badruzzaman Khan, et al. "Remote ischemic post-conditioning promotes hematoma resolution via AMPK-dependent immune regulation." Journal of Experimental Medicine 215, no. 10 (2018): 2636–54. http://dx.doi.org/10.1084/jem.20171905.

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Spontaneous intracerebral hemorrhage (ICH) produces the highest acute mortality and worst outcomes of all stroke subtypes. Hematoma volume is an independent determinant of ICH patient outcomes, making clot resolution a primary goal of clinical management. Herein, remote-limb ischemic post-conditioning (RIC), the repetitive inflation–deflation of a blood pressure cuff on a limb, accelerated hematoma resolution and improved neurological outcomes after ICH in mice. Parabiosis studies revealed RIC accelerated clot resolution via a humoral-mediated mechanism. Whereas RIC increased anti-inflammatory

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Lee, Jungsoo, Heegoo Kim, Jinuk Kim, Won Hyuk Chang, and Yun-Hee Kim. "Multimodal Imaging Biomarker-Based Model Using Stratification Strategies for Predicting Upper Extremity Motor Recovery in Severe Stroke Patients." Neurorehabilitation and Neural Repair 36, no. 3 (2021): 217–26. http://dx.doi.org/10.1177/15459683211070278.

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Background. Various prognostic biomarkers for upper extremity (UE) motor recovery after stroke have been reported. However, most have relatively low predictive accuracy in severe stroke patients. Objective. This study suggests an imaging biomarker-based model for effectively predicting UE recovery in severe stroke patients. Methods. Of 104 ischemic stroke patients screened, 42 with severe motor impairment were included. All patients underwent structural, diffusion, and functional magnetic resonance imaging at 2 weeks and underwent motor function assessments at 2 weeks and 3 months after stroke

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Malkki, Hemi. "Gut microbiota influence stroke recovery in mice." Nature Reviews Neurology 12, no. 5 (2016): 252. http://dx.doi.org/10.1038/nrneurol.2016.52.

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Dissertations / Theses on the topic "Stroke, mice, recovery, biomarker"

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Gower, Annette. "The Efficacy of Specific Activation of D1-class Dopamine Receptors to Enhance Motor Recovery in Mice Following Cortical Photothrombotic Stroke." Thesis, Université d'Ottawa / University of Ottawa, 2018. http://hdl.handle.net/10393/37619.

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Stroke is a widespread condition, which often leaves survivors with lasting deficits in motor function, however, physical rehabilitation is the only treatment available after the acute period. A large body of preclinical literature suggests dopamine-augmenting drugs, could enhance motor recovery following stroke. Unfortunately, mixed clinical results have prevented the implementation of such treatments, possibly due to the wide variety of G protein-coupled receptors these drugs can activate. Using a mouse photothrombosis stroke model and a battery of motor and sensorimotor behavioural tests, t

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Hazime, Mahmoud. "Etude de l’effet de l’octadécaneuropeptide sur l’activité du cortex cérébral : intérêt pour la récupération fonctionnelle post-ischémique The gliopeptide ODN, a ligand for the benzodiazepine site of GABAA receptors, boosts functional recovery after stroke Prolonged deficit of gamma oscillations in the peri-infarct cortex of mice after stroke Bi-directional effect of the endozepine ODN on neuronal activity in vivo." Thesis, Normandie, 2019. http://www.theses.fr/2019NORMR127.

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L’ischémie cérébrale demeure un problème de santé publique majeur pour lequel on manque de solution thérapeutique permettant de favoriser la récupération des fonctions perdues. L’équipe de recherche dans laquelle cette thèse fut menée a mis en évidence que l’octadécaneuropeptide (ODN), un ligand endogène du récepteur GABAA, permet d’améliorer la récupération sensori-motrice après une ischémie cérébrale chez la souris. L’objectif de cette thèse était de caractériser les effets de l’ODN sur l’activité neuronale in vivo afin de mieux comprendre par quel mécanisme ce peptide agit sur la réparation

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Busti, Irene. "Identification of prognostic and therapeutic tools for cortical stroke treatment in mice." Doctoral thesis, 2020. http://hdl.handle.net/2158/1190551.

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Stroke is one of the main causes of disability worldwide. Around 70-80% of patients survive the ischemic attack, however around three fourths of them do not recover completely. Currently, there are only few effective therapies to treat this pathology in the early phases. Great is therefore the need for (i) reliable biomarkers predictive of spontaneous recovery and (ii) novel approaches to stimulate the recovery of lost functions. Given this background, the aim of this thesis was to develop novel prognostic and therapeutic tools in preclinical mouse models of stroke.

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Book chapters on the topic "Stroke, mice, recovery, biomarker"

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Syeara, Nausheen, Sounak Bagchi, Abdullah Al Shoyaib, Serob T. Karamyan, Faisal F. Alamri, and Vardan T. Karamyan. "The Finer Aspects of Grid-Walking and Cylinder Tests for Experimental Stroke Recovery Studies in Mice." In Methods in Molecular Biology. Springer US, 2023. http://dx.doi.org/10.1007/978-1-0716-2926-0_23.

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