Relevant bibliographies by topics / Subarachnoid Hemorrhage Subarachnoid Hemorrhage Vasospasm

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Subarachnoid Hemorrhage Subarachnoid Hemorrhage Vasospasm'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Subarachnoid Hemorrhage Subarachnoid Hemorrhage Vasospasm"

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Hui, Ferdinand K., Albert J. Schuette, Shaye I. Moskowitz, Rishi Gupta, Alejandro M. Spiotta, Nancy A. Obuchowski, and C. Michael Cawley. "Antithrombotic States and Outcomes in Patients With Angiographically Negative Subarachnoid Hemorrhage." Neurosurgery 68, no. 1 (January 1, 2011): 125–31. http://dx.doi.org/10.1227/neu.0b013e3181fd82b6.

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Abstract BACKGROUND: Antithrombotic states are encountered frequently, either because of medical therapy or by preexistent pathological states, and may affect the severity of hemorrhagic strokes such as angiographically negative subarachnoid hemorrhages. OBJECTIVE: To determine the effects of antithrombotic states on the outcomes of patients with angiographically negative subarachnoid hemorrhage by examining data pooled from 2 institutions. METHODS: This is a retrospective review of patients who experienced angiographically negative subarachnoid hemorrhage at 2 institutions over the past 5 years. The patients were grouped into those with and those without an antithrombotic state at time of hemorrhage and were stratified according to presentation, clinical grades, outcomes, need for cerebrospinal fluid diversion, and development of vasospasm. Computed tomography of the head was assessed for bleed pattern and modified Fisher grade. Patients were excluded if a causative lesion was subsequently discovered. RESULTS: There is a statistically significant association between antithrombotic states and poorer presentation, higher Hunt and Hess score, increased amount of subarachnoid hemorrhage, higher modified Fisher grade, increased incidence of vasospasm, hydrocephalus, and poor outcomes as assessed by modified Rankin scale (P < .001). Patients with an antithrombotic state experience worse outcomes even with adjustment for the amount of hemorrhage as assessed by modified Fisher grade (P < .001). CONCLUSION: Patients in an antithrombotic state presenting with angiographically negative subarachnoid hemorrhage present with inferior clinical scores, diffuse hemorrhage patterns, and worse modified Fisher grades and have worse outcomes.
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Condette-Auliac, S., S. Bracard, R. Anxionnat, E. Schmitt, J. C. Lacour, M. Braun, J. Meloneto, A. Cordebar, L. Yin, and L. Picard. "Vasospasm After Subarachnoid Hemorrhage." Stroke 32, no. 8 (August 2001): 1818–24. http://dx.doi.org/10.1161/01.str.32.8.1818.

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Balanzar, Gerardo Guinto, and Yoshiaki Guinto-Nishimura. "Vasospasm in Subarachnoid Hemorrhage." World Neurosurgery 82, no. 6 (December 2014): e677-e678. http://dx.doi.org/10.1016/j.wneu.2013.09.003.

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Aires, Rogério, Márcio Francisco Lehmann, Renata Simm, Miguel Melgar, Carlos Alexandre Martins Zicarelli, and Paulo Henrique Pires De Aguiar. "Magnesium in subarachnoid hemorrhage." JBNC - JORNAL BRASILEIRO DE NEUROCIRURGIA 21, no. 3 (March 19, 2018): 158–61. http://dx.doi.org/10.22290/jbnc.v21i3.845.

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Cerebral vasospasm is responsible for a great number of deaths and morbidity after acute subarachnoid hemorrhage. Beyond any doubt, it is related to physiopathology of endothelial cell contraction and relaxation. It has been proved as a therapeutic agent when administrated intravenously in patients after subarachnoid hemorrhage. A critical review of literature was accomplished by the authors in order to analyse all actions of vasospasm in SAH. The intravenous administration of magnesium is imperative and should be done immediately after the onset of SAH, as reccomended in the most important guidelines for the treatment of cerebral vasospasm.
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Burns, Shannon K., Kacie J. Brewer, Courtney Jenkins, and Sally Miller. "Aneurysmal Subarachnoid Hemorrhage and Vasospasm." AACN Advanced Critical Care 29, no. 2 (June 15, 2018): 163–74. http://dx.doi.org/10.4037/aacnacc2018491.

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Aneurysmal subarachnoid hemorrhage is potentially fatal and is associated with poor outcomes in many patients. Advances in neurosurgical and medical management of ruptured aneurysms have improved mortality rates in patients with aneurysmal subarachnoid hemorrhage. Surgical and endovascular interventions, such as external ventricular drain placement, aneurysm clipping, and endovascular coiling, have been developed over the past few decades. Patients with aneurysmal subarachnoid hemorrhage are also at risk for cerebral vasospasm and delayed cerebral ischemia. This article describes the diagnosis and treatment of aneurysmal subarachnoid hemorrhage, vasospasm, and cerebral ischemia. Concurrent medical considerations and ideas for future neuroinflammatory vasospasm research are also discussed.
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Milojevic, T. M., B. V. Baljozovic, M. Lj Rakic, B. D. Nestorovic, M. M. Dostanic, B. D. Milakovic, Z. Kojic, N. R. Repac, and I. S. Cvrkota. "Cerebral vasospasm after subarachnoid hemorrhage." Acta chirurgica Iugoslavica 55, no. 2 (2008): 55–60. http://dx.doi.org/10.2298/aci0802055m.

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Cerebral vasospasm causes permanent neurolological deficit or death occurrence in 13% of clinical cases. Peak frequency is from 8-10th day after SAH. The purpose of this study is factor analysis that may have influence on vasospasm development , as well as predictor determination. The study is prospective and analysis 192 patients treated in Institute of Neurosurgery, Clinical Centre of Serbia, Belgrade. The majority of patients were admitted in hospital in first four days after SAH, and 184 had GCS over 7. Univariate methods of factor analysis were used, and for significance of predictors influence testing multivariante regression analysis was used . Vasospasm occurred in 22,40% of all cases. No relationships have been found between sex, age, previous hypertension, timing of surgery, appearance of hydrocephalus and intracerebral hematoma, hypertermia or mean arterial blood pressure, with occurrence of cerebral vasospasm. Factors with significantly associated with the occurrence of vasospasm were: hearth disease, hypernatriemia, Hct, clinical grade on admission as well as preoperative clinical grade and Fisher CT scan grade. In the first four days after SAH, Fisher scan grade, preoperative clinical grade and Hct, appeared as predictors. After four days, clinical grade on admission and hypernatiemia, showed as predictors.
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Oyama, Katie, and Laura Criddle. "Vasospasm After Aneurysmal Subarachnoid Hemorrhage." Critical Care Nurse 24, no. 5 (October 1, 2004): 58–67. http://dx.doi.org/10.4037/ccn2004.24.5.58.

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Mohindra, Sandeep, Amey Savardekar, and Ankur Kapoor. "Vasospasm after iatrogenic subarachnoid hemorrhage." Neurology India 64, no. 7 (2016): 126. http://dx.doi.org/10.4103/0028-3886.178057.

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Macdonald, R. Loch. "Predicting Vasospasm After Subarachnoid Hemorrhage." World Neurosurgery 75, no. 1 (January 2011): 25–26. http://dx.doi.org/10.1016/j.wneu.2010.07.018.

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Roberts, Debra, and Emad Nourollah-Zadeh. "Cerebral vasospasm after subarachnoid hemorrhage." Neurology 93, no. 5 (July 5, 2019): 192–93. http://dx.doi.org/10.1212/wnl.0000000000007854.

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Dissertations / Theses on the topic "Subarachnoid Hemorrhage Subarachnoid Hemorrhage Vasospasm"

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Ryttlefors, Mats. "Subarachnoid Hemorrhage in the Elderly." Doctoral thesis, Uppsala universitet, Institutionen för neurovetenskap, 2009. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-9504.

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Subarachnoid hemorrhage (SAH) is a disease with high risk of mortality and morbidity. Elderly patients have an even higher risk of poor outcome. The incidence of SAH increases with age and the elderly constitute a substantial and increasing proportion of the population. Thus, the management of elderly SAH patients is an imminent clinical challenge. Time trends in clinical management and outcome were investigated in 281 SAH patients aged ≥65 years admitted over an 18-year period. The volume of elderly patients, especially patients ≥70 years and patients in worse clinical condition increased over time. The proportion of patients with favorable outcome increased over time, without an increase in severely disabled patients. Technical results and clinical outcome of endovascular aneurysm treatment (EVT) was investigated in 62 elderly SAH patients. EVT can be performed in elderly SAH patients with high technical success, acceptable aneurysm occlusion degree, acceptable procedural complication rate, and fair outcome results. EVT was compared to neurosurgical clipping (NST) in 278 elderly SAH patients in the International Subarachnoid Aneurysm Trial. In good grade elderly SAH patients, EVT should probably be the favored treatment for internal carotid and posterior communicating artery aneurysms, while elderly patients with middle cerebral artery aneurysms appear to benefit from NST. Occurrence of secondary insults and their impact on clinical deterioration were studied in 99 patients with severe SAH. High intracranial pressure increased and high cerebral perfusion pressure decreased the risk of clinical deterioration. Elderly patients had less intracranial hypertension insults and more hypertensive, hypotensive and hypoxemic insults. Good outcome was achieved in 24% of elderly patients with severe SAH, and the proportion of severe disability was similar to that of younger patients. Patient age was not a significant predictor for vasospasm in 413 SAH patients when admission and treatment variables were adjusted for with multiple logistic regression.
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Ekelund, Anders. "Detection and haemodilutive treatment of cerebral arterial vasospasm and delayed ischaemia after aneurysmal subarachnoid haemorrhage." Lund : Lund University, 1999. http://catalog.hathitrust.org/api/volumes/oclc/68945106.html.

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Lee, Hubert. "Clinical Prediction of Symptomatic Vasospasm in Aneurysmal Subarachnoid Hemorrhage." Thesis, Université d'Ottawa / University of Ottawa, 2017. http://hdl.handle.net/10393/35673.

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Objective: This study aims to derive a clinically-applicable decision rule to predict the risk of symptomatic vasospasm, a neurological deficit primarily due to abnormal narrowing of cerebral arteries supplying an attributable territory, in aneurysmal subarachnoid hemorrhage (SAH). Methods: SAH patients presenting from 2002 to 2011 were analyzed using logistic regression and recursive partitioning to identify clinical, radiological, and laboratory features that predict the occurrence of symptomatic vasospasm. Results: The incidence of symptomatic vasospasm was 21.0%. On multivariate logistic regression analysis, significant predictors of symptomatic vasospasm included age 40-59 years, high Modified Fisher Grade (Grades 3 and 4), and anterior circulation aneurysms. Conclusion: Development of symptomatic vasospasm can be reliably predicted using a clinical decision rule created by logistic regression. It exhibits increased accuracy over the Modified Fisher Grade alone and may serve as a useful clinical tool to individualize vasospasm risk once prospectively validated in other neurosurgical centres.
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Alsalman, Abdulkhaliq. "Impact of Statin Therapy on Outcomes in Aneurysmal Subarachnoid Hemorrhage Patients." VCU Scholars Compass, 2009. http://scholarscompass.vcu.edu/etd/1994.

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There is conflicting data on the effects of statins on cerebral vasospasm and clinical outcomes in aneurysmal subarachnoid hemorrhage (aSAH) patients. In this retrospective cohort study, patients were divided into those who received pravastatin (PRAV group) 40mg/d and those who did not (NP group). Data were analyzed using multivariate logistic regression. Eighty-one patients met inclusion criteria. There was a statistically significant decreased in the incidence of vasospasm in the PRAV group; however, this association did not retain significance after adjusting for WFNS, race, elevated WBC, and clipping (59% PRAV vs. 88% NP, p=0.08). There was no statistically significant difference in proportion of severe radiological vasospasm or mortality between groups. However, there was a trend towards a decreased mean length of stay (P=0.06) and a significantly higher proportion of survivors discharged to home in the PRAV group (P<0.0001). In conclusion, there was a trend towards a decrease in the incidence of vasospasm in the aSAH receiving pravastatin, but this trend did not achieve statistical significance after adjusting for potential confounders. Pravastatin was associated with other favorable clinical outcomes.
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Blissitt, Patricia A. "The effect of head of bed elevation on cerebrovascular dynamics in mild or moderate cerebral vasospasm following aneurysmal subarachnoid hemorrhage /." Thesis, Connect to this title online; UW restricted, 2002. http://hdl.handle.net/1773/7309.

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Pyne, Gail Jean. "Vascular smooth muscle oxidative metabolism and function during vasospasm after subarachnoid haemorrhage." Thesis, University of Oxford, 1999. http://ora.ox.ac.uk/objects/uuid:a6b78de5-1f66-4225-992e-e490cf68d0e8.

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Aims: The purpose of the research presented in this thesis is to elucidate the mechanism of the stimulation of oxidative metabolism and contractile function that occurs in vascular smooth muscle during cerebral vasospasm (CV) after subarachnoid haemorrhage (SAH). The biochemical mechanisms leading to CV were investigated using an in vitro model of CV developed for this research. CSF (cerebrospinal fluid) from SAH patients at risk of vasospasm which stimulated oxygen consumption (CSF S ) was used to model vasospasm. The hypothesis is CSFS contains a substance which stimulates tension generation over that of CSFN ,(non-stimulatory cerebrospinal fluid) and also inhibits the myosin light chain phosphatase. Methods: The porcine carotid artery was used as a model for the human basilar artery. The rate of oxygen consumption (JO2) was measured in response to CSFS and tension generation was also examined. Various agents were used to treat or pretreat the tissue such as magnesium and andalpha;1-adrenergic receptor agonists. Their effects on the CSFS-induced stimulation were measured to study the mechanism of vasospasm. A myosin light chain phosphatase (MLCP) assay was developed to study the mechanisms leading to CV. Results and conclusion: Addition of CSFS to the porcine carotid artery is a reliable and reproducible in vitro model of CV. Using this model, it was found that Mg++ loading and andalpha;1-adrenergic receptor agonists attenuated the vasospasm, but a non-specific endothelin antagonist had no effect. Acute addition of 12mM Mg++ relaxed the tissue from a CSFS induced contraction significantly and rendered the contraction rinsible. Okadaic acid (InM), a phosphatase inhibitor, had very similar effects to CSFS because it stimulated JO2 and slowed relaxation after a stretch. There was also significant inhibition of phosphatase caused by the CSFS. Vasospasm appears to be caused by a combination of a contractile stimulus, and inhibition of MLCP activity.
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Vora, Yashail Y. "The role of transcranial Doppler ultrasonography in the diagnosis of cerebral vasospasm following aneursymal subarachnoid hemorrhage." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk2/tape17/PQDD_0002/MQ34429.pdf.

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Trandafir, Cristina Corina. "Study on participation of arginine vasopressin and leukotrienes in development of cerebral vasospasm after subarachnoid hemorrhage." Kyoto University, 2006. http://hdl.handle.net/2433/144056.

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Kyoto University (京都大学)
0048
新制・課程博士
博士(人間・環境学)
甲第12400号
人博第318号
新制||人||79(附属図書館)
17||D||177(吉田南総合図書館)
24236
UT51-2006-J392
京都大学大学院人間・環境学研究科相関環境学専攻
(主査)教授 五十棲 泰人, 助教授 倉橋 和義, 教授 津田 謹輔
学位規則第4条第1項該当
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Paschoal, Eric Homero Albuquerque. "Biomarcadores genéticos na hemorragia subaracnoidea aneurismática em pacientes da Amazônia." Universidade de São Paulo, 2017. http://www.teses.usp.br/teses/disponiveis/5/5138/tde-10112017-125704/.

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Hemorragia subaracnoidea aneurismática (HSAa) é considerada causa importante de morte e de sequelas neurológicas. A taxa de mortalidade desta doença pode alcançar 50% nos primeiros dois meses após sangramento de aneurisma encefálico. Apesar dos avanços científicos da modernidade, o resultado do tratamento da HSAa não mudou nos últimos anos. O presente estudo avaliou o papel de 14 biomarcadores genéticos, incluindo o polimorfismo (SNP) do gene eNOS, em pacientes da Amazônia com HSAa, para verificar as alterações alélicas associadas ao risco de vasoespasmo encefálico e déficit neurológico tardio. Avaliou-se a ancestralidade desta amostra de pacientes em que se utilizou 48 marcadores para identificar possível etnia associada à predisposição ao VE. Investigou-se 14 biomarcadores genéticos no tocante à resposta inflamatória encefálica na HSAa. Foram avaliados 265 doentes que foram divididos em dois grupos: grupo I (pacientes com vasoespasmo encefálico) e grupo 2 (pacientes sem vasoespasmo). A média das idades foi 51 anos, havia 224 mulheres (84%) e 124 pacientes (46,79%) apresentaram vasoespasmo encefálico (VE). A maior incidência de VE ocorreu na idade entre 50 e 59 anos. Tabagismo e hipertensão arterial sistêmica foram os fatores de risco mais associados à VE. Aneurismas encefálicos de tamanho pequeno e médio predominaram nesta casuística. As escalas amarela e vermelha do VASOGRADE associaram-se ao risco de VE (p < 0,001). Não houve variação na distribuição ancestral entre os grupos estudados e o que ocorre na população brasileira saudável na região Amazônica. O gene da eNOS com seus respectivos polimorfismos T-786C e 27VNTR4 correlacionaram-se com VE. Outros marcadores observados foram TP53, CASP8, ACE2, IL4 e XRCC1. O gene TP53 (modelo recessivo alelo 1) mostrou-se ser um fator protetor de VE, enquanto que genes com mutações INDEL CASP8 (modelo recessivo alelo 2) e o XRCC1 (modelo recessivo alelo 1) mostraram tendência ao desenvolvimento de VE com risco 2 vezes maior e 1,4 vezes maior que o grupo II (p < 0,001). Conclui-se que SNPs da eNOS se correlacionam com desenvolvimento de VE sintomático pós-HSAa. Este estudo também mostrou o papel dos marcadores inflamatórios na HSAa, o que auxiliaria na condução da terapia clínica.
Aneurysmal subarachnoid hemorrhage (aSAH) is a leading cause of premature death and neurological disability. It is considered as a devastating condition that accounts to 50% of mortality during the first two months after a hemorrhagic event. Despite foremost advances in the clinical management of post-aSAH patients, the rates of mortality and morbidity have not changed in recent years. This study appraised the role of 14 genetic biomarkers, including the eNOS polymorphism (SNP) between Amazon\'s patients with aSAH, as means to document how variant alleles are related to a higher disposition to cerebral vasospasm (CV) and delayed cerebral ischemia (DCI). 265 patients were evaluated and then divided into two clusters: Group I (with symptomatic CV) and group II (presenting no symptomatic CV). The median ages of patients were 51.61 years of age, 224 (84.52%) were women and 124 patients (46.97%) had symptoms of cerebral vasospasm (CV). Tobacco smoking and systemic arterial hypertension are the risk factors most associated to CV. In the course of this research, most aneurysms found were small and medium-sized. The score VASOGRADE yellow and VASOGRADE red presented a high risk of CV (p < 0.001). We established a panel of 48 ancestry informative markers for estimating which ethnicity could present a predisposition to CV. There was no variation in the ancestral distribution between study groups and healthy brazilian folk over the Amazon region. The eNOS gene with its polymorphisms T-786C and 27 VNTR4 were correlated to CV. Other markers were accomplished: TP53, CASP8, ACE2, IL4, and XRCC1. The TP53 gene (recessive genetic model allele 1) supporting evidence of the protective role to CV. Whilst other genes with INDEL mutation like as CASP8 (recessive model allele 2) and the XRCC1 (recessive model allele 1) indicated a propensity to spread out CV with odds 2-fold higher, and 1.414 times greater than group II (p < 0.001). It follows that eNOS SNPs correlate to a positive association with a syntomatic CV post-aSAH. Also, this study showed up the role of inflammatory markers at aSAH to a further educated therapeutic choice for a better clinical response
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Wallmark, Svante. "Life after Subarachnoid Hemorrhage." Doctoral thesis, Uppsala universitet, Neurokirurgi, 2016. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-307949.

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Aneurysmal subarachnoid hemorrhage (SAH) is a devastating disease with mean age of 59 years. SAH accounts for 5% of all stroke and more than one quarter of potential life years lost through stroke. With the advanced neurosurgical methods of today two thirds of the patients survive. We know, however, that various cognitive, psychiatric and physical impairments are common that affect quality of life, social life, and the ability to work in the aftermath of SAH. The overall aim constituting this PhD dissertation is to better understand some of the challenges often faced by those surviving SAH. Two SAH patient cohorts have been studied. The first followed 96 consecutively included patients during the first year after ictus. Spasticity and cognitive impairment was assessed after 6 months and the Swedish stroke register follow-up form was used to investigate family support and the use of medical and social services. Return to work was assessed at 12 months. The second cohort assessed attention deficits using the test of variables of attention (T.O.V.A.) at 7 months after ictus in 19 patients with moderate to good recovery. Spasticity was just as common in our SAH patients as after other stroke, though it was rarely treated pharmacologically. By assessing cognitive impairment at 6 months after ictus using the Montreal cognitive assessment, 68% of the patients could be correctly predicted as having returned/not returned to work at 12 months. Seventeen percent of the patients had not had a follow-up appointment 6 months after ictus. These patients were older, more often living alone, had a lower quality of life, more depressive symptoms and more cognitive impairment compared to those having had a follow-up appointment. Twenty percent had had a follow-up in primary care. Seventy-eight percent of those with moderate to severe disability were living in their own accommodations. Fifty-eight percent of the patients had attention deficits. Challenges after SAH were common and often dealt with in the home environment of the patients. The results of this thesis highlight the importance of assisting the patients and their relatives in their struggle back to life after SAH.
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Books on the topic "Subarachnoid Hemorrhage Subarachnoid Hemorrhage Vasospasm"

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Mario, Zuccarello, ed. Cerebral vasospasm: Neurovascular events after subarachnoid hemorrhage. Vienna: Springer, 2013.

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Zuccarello, Mario, Joseph F. Clark, Gail Pyne-Geithman, Norberto Andaluz, Jed A. Hartings, and Opeolu M. Adeoye, eds. Cerebral Vasospasm: Neurovascular Events After Subarachnoid Hemorrhage. Vienna: Springer Vienna, 2013. http://dx.doi.org/10.1007/978-3-7091-1192-5.

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International, Conference on Cerebral Vasospasm (8th 2003 Chicago Ill ). Cerebral vasospasm: Advances in research and treatment. New York: Thieme, 2005.

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Martin, Robert D., Warren Boling, Gang Chen, and John H. Zhang, eds. Subarachnoid Hemorrhage. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-04615-6.

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Feng, Hua. Early Brain Injury or Cerebral Vasospasm: Volume 2: Clinical Management. Vienna: Springer-Verlag/Wien, 2011.

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1941-, McAllister V. L., ed. Subarachnoid haemorrhage. Berlin: Springer-Verlag, 1986.

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Subarachnoid hemorrhage: Causes and cures. New York: Oxford University Press, 1998.

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Fandino, Javier, Serge Marbacher, Ali-Reza Fathi, Carl Muroi, and Emanuela Keller, eds. Neurovascular Events After Subarachnoid Hemorrhage. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-04981-6.

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Rinkel, Gabriel J. E., and Paut Greebe. Subarachnoid Hemorrhage in Clinical Practice. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-17840-0.

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Bederson, Joshua B. Subarachnoid hemorrhage: Pathophysiology and management. Park Ridge, Ill: American Association of Neurological Surgeons, 1997.

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Book chapters on the topic "Subarachnoid Hemorrhage Subarachnoid Hemorrhage Vasospasm"

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Sekhon, Mypinder, Oliver Ayling, and Peter Gooderham. "Vasospasm After Subarachnoid Hemorrhage." In Echography and Doppler of the Brain, 183–91. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-48202-2_16.

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Danura, Hiroki, Bawarjan Schatlo, Serge Marbacher, Hassen Kerkeni, Michael Diepers, Luca Remonda, Ali-Reza Fathi, and Javier Fandino. "Acute Angiographic Vasospasm and the Incidence of Delayed Cerebral Vasospasm: Preliminary Results." In Neurovascular Events After Subarachnoid Hemorrhage, 187–90. Cham: Springer International Publishing, 2014. http://dx.doi.org/10.1007/978-3-319-04981-6_32.

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Laher, Ismail, and John A. Bevan. "Acute Vasospasm and Subarachnoid Hemorrhage." In The Human Brain Circulation, 389–97. Totowa, NJ: Humana Press, 1994. http://dx.doi.org/10.1007/978-1-4612-0303-2_29.

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Vieira, Eduardo, and Hildo Azevedo-Filho. "Vasospasm Following Aneurysmal Subarachnoid Hemorrhage." In Neurovascular Surgery, 249–54. Singapore: Springer Singapore, 2018. http://dx.doi.org/10.1007/978-981-10-8950-3_29.

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Harrigan, Mark R., David W. Newell, and Andrei V. Alexandrov. "Cerebral Vasospasm after Subarachnoid Hemorrhage." In Cerebrovascular Ultrasound in Stroke Prevention and Treatment, 207–13. Oxford, UK: Wiley-Blackwell, 2011. http://dx.doi.org/10.1002/9781444327373.ch12.

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Sun, Qing, and Gang Chen. "Cerebral Vasospasm and Subarachnoid Hemorrhage." In Recent Progress in the Management of Cerebrovascular Diseases, 15–19. Singapore: Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-16-3387-4_3.

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Sozen, Takumi, Reiko Tsuchiyama, Yu Hasegawa, Hidenori Suzuki, Vikram Jadhav, Shigeru Nishizawa, and John H. Zhang. "Advances in Experimental Subarachnoid Hemorrhage." In Early Brain Injury or Cerebral Vasospasm, 15–21. Vienna: Springer Vienna, 2011. http://dx.doi.org/10.1007/978-3-7091-0353-1_3.

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Wu, Bihua, Xiaoming Wang, and John H. Zhang. "Cardiac Damage After Subarachnoid Hemorrhage." In Early Brain Injury or Cerebral Vasospasm, 215–18. Vienna: Springer Vienna, 2011. http://dx.doi.org/10.1007/978-3-7091-0353-1_37.

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Sakowitz, O. W., A. S. Sarrafzadeh, G. Benndorf, W. R. Lanksch, and A. W. Unterberg. "On-line Microdialysis Following Aneurysmal Subarachnoid Hemorrhage." In Cerebral Vasospasm, 141–44. Vienna: Springer Vienna, 2001. http://dx.doi.org/10.1007/978-3-7091-6232-3_30.

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Macdonald, R. Loch. "Vasospasm: My First 25 Years—What Worked? What Didn’t? What Next?" In Neurovascular Events After Subarachnoid Hemorrhage, 1–10. Cham: Springer International Publishing, 2014. http://dx.doi.org/10.1007/978-3-319-04981-6_1.

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Conference papers on the topic "Subarachnoid Hemorrhage Subarachnoid Hemorrhage Vasospasm"

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Al-Mufti, F., N. Manning, M. Crimmins, K. Amuluru, N. Majmundar, M. El-Ghanem, V. Patel, et al. "E-174 Severity of vasospasm associated with development of collaterals following aneurysmal subarachnoid hemorrhage." In SNIS 15TH ANNUAL MEETING, July 23–26, 2018, Hilton San Francisco Union Square San Francisco, CA. BMA House, Tavistock Square, London, WC1H 9JR: BMJ Publishing Group Ltd., 2018. http://dx.doi.org/10.1136/neurintsurg-2018-snis.250.

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Walker, M., A. Mohammed, C. Kelly, S. Levy, M. Erdoes, C. Johnston, and M. Levitt. "E-072 The relationship between cerebral vasospasm and herpesvirus reactivation after aneurysmal subarachnoid hemorrhage." In SNIS 18TH ANNUAL MEETING. BMA House, Tavistock Square, London, WC1H 9JR: BMJ Publishing Group Ltd., 2021. http://dx.doi.org/10.1136/neurintsurg-2021-snis.167.

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Khatibi, K., V. Szeder, G. Korbakis, M. Buitrago Blanco, S. Tateshima, R. Jahan, G. Duckwiler, and P. Vespa. "O-032 Role of bedside multi-modality monitoring in detection of cerebral vasospasm following subarachnoid hemorrhage." In SNIS 14TH, Annual Meeting, July 24–27, 2017, The Broadmoor, Colorado Springs, CO. BMA House, Tavistock Square, London, WC1H 9JR: BMJ Publishing Group Ltd., 2017. http://dx.doi.org/10.1136/neurintsurg-2017-snis.32.

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Vulcu, S., F. Wagner, A. Santos, R. Reitmeir, N. Söll, A. Raabe, J. Beck, and W. Z'Graggen. "The Value of Repetitive CT Perfusion for Detection of Cerebral Vasospasm-related Hypoperfusion after Aneurysmal Subarachnoid Hemorrhage." In Joint Annual Meeting 2017: Swiss Society of Neurosurgery, Swiss Society of Neuroradiology. Georg Thieme Verlag KG, 2017. http://dx.doi.org/10.1055/s-0037-1603832.

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Kwon, H., Y. Lee, and H. Koh. "P-015 Long-term results of angioplasty using stent-retrievers for cerebral vasospasm in subarachnoid hemorrhage patients." In SNIS 14TH, Annual Meeting, July 24–27, 2017, The Broadmoor, Colorado Springs, CO. BMA House, Tavistock Square, London, WC1H 9JR: BMJ Publishing Group Ltd., 2017. http://dx.doi.org/10.1136/neurintsurg-2017-snis.52.

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Palaniswamy, Sangeetha, Venkatapura Ramesh, Rita Christopher, and Dhananjaya Bhat. "A0022 Effect of Remote Ischemic Preconditioning on Cerebral Vasospasm and Biomarkers of Cerebral Ischemia in Aneurysmal Subarachnoid Hemorrhage." In 20th Annual Conference of Indian Society of Neuroanaesthesiology and Critical Care (ISNACC). Thieme Medical and Scientific Publishers Private Ltd., 2019. http://dx.doi.org/10.1055/s-0039-1684129.

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Daou, B., S. Khalsa, S. Anand, C. Williamson, K. Rajajee, K. Sheehan, and A. Pandey. "E-152 Subarachnoid hemorrhage quantitative volume analysis: blood volume predicts cerebral vasospasm, delayed cerebral infarction and clinical outcome." In SNIS 17TH ANNUAL MEETING. BMA House, Tavistock Square, London, WC1H 9JR: BMJ Publishing Group Ltd., 2020. http://dx.doi.org/10.1136/neurintsurg-2020-snis.184.

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Kamath, Sriganesh. "Effect of Intra-arterial Nimodipine on Cerebral Oxygen Saturation and Cardiac Indices in Patients with Cerebral Vasospasm after Aneurysmal Subarachnoid Hemorrhage." In 19th Annual Conference of the Indian Society of Neuroanaesthesiology and Critical Care (ISNACC). Thieme Medical and Scientific Publishers Private Limited, 2018. http://dx.doi.org/10.1055/s-0038-1636373.

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Gupta, R., K. Woodward, D. Fiorella, H. Woo, D. Liebeskind, D. Frei, A. Siddiqui, R. DeLeacy, R. Hanel, and A. Maud. "P-044 Primary results of the vesalio neva VS for the treatment of symptomatic cerebral vasospasm following aneurysm subarachnoid hemorrhage (VITAL) study." In SNIS 18TH ANNUAL MEETING. BMA House, Tavistock Square, London, WC1H 9JR: BMJ Publishing Group Ltd., 2021. http://dx.doi.org/10.1136/neurintsurg-2021-snis.80.

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Catapano, J., V. Srinivasan, K. Rumalla, M. Labib, C. Ngueyen, T. Cole, J. Baranoski, et al. "O-007 Length of hospital stay in aneurysmal subarachnoid hemorrhage patients without vasospasm on angiography: potential for a fast-track discharge cohort." In SNIS 18TH ANNUAL MEETING. BMA House, Tavistock Square, London, WC1H 9JR: BMJ Publishing Group Ltd., 2021. http://dx.doi.org/10.1136/neurintsurg-2021-snis.7.

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Reports on the topic "Subarachnoid Hemorrhage Subarachnoid Hemorrhage Vasospasm"

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Sherman, Paul M. The Worst Headache of Life: Evaluation of Nontraumatic Subarachnoid Hemorrhage. Fort Belvoir, VA: Defense Technical Information Center, September 2005. http://dx.doi.org/10.21236/ada437539.

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Liu, Jiangfeng, Jinli He, Xun Chen, Yanjin Feng, Chen Wang, Mohamed Arab Awil, Yan Wang, Yi Tian, and Deren Hou. Cilostazol for Aneurysmal Subarachnoid Hemorrhage: an Update Systematic Review and Meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, October 2020. http://dx.doi.org/10.37766/inplasy2020.10.0110.

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Liu, Tao, Yongchang Guan, Lina Guo, Jinghua Du, and Ting-zhong Wang. Medium dosage of pravastatin significantly improves short-term outcome in patients with aneurysmal subarachnoid hemorrhage. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, November 2020. http://dx.doi.org/10.37766/inplasy2020.11.0044.

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