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Books on the topic 'Subarachnoid Hemorrhage Subarachnoid Hemorrhage Vasospasm'

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Published: 4 June 2021
Last updated: 1 February 2022

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1

Mario, Zuccarello, ed. Cerebral vasospasm: Neurovascular events after subarachnoid hemorrhage. Vienna: Springer, 2013.

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2

Zuccarello, Mario, Joseph F. Clark, Gail Pyne-Geithman, Norberto Andaluz, Jed A. Hartings, and Opeolu M. Adeoye, eds. Cerebral Vasospasm: Neurovascular Events After Subarachnoid Hemorrhage. Vienna: Springer Vienna, 2013. http://dx.doi.org/10.1007/978-3-7091-1192-5.

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3

International, Conference on Cerebral Vasospasm (8th 2003 Chicago Ill ). Cerebral vasospasm: Advances in research and treatment. New York: Thieme, 2005.

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4

Martin, Robert D., Warren Boling, Gang Chen, and John H. Zhang, eds. Subarachnoid Hemorrhage. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-04615-6.

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5

Feng, Hua. Early Brain Injury or Cerebral Vasospasm: Volume 2: Clinical Management. Vienna: Springer-Verlag/Wien, 2011.

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6

1941-, McAllister V. L., ed. Subarachnoid haemorrhage. Berlin: Springer-Verlag, 1986.

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7

Subarachnoid hemorrhage: Causes and cures. New York: Oxford University Press, 1998.

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8

Fandino, Javier, Serge Marbacher, Ali-Reza Fathi, Carl Muroi, and Emanuela Keller, eds. Neurovascular Events After Subarachnoid Hemorrhage. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-04981-6.

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9

Rinkel, Gabriel J. E., and Paut Greebe. Subarachnoid Hemorrhage in Clinical Practice. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-17840-0.

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10

Bederson, Joshua B. Subarachnoid hemorrhage: Pathophysiology and management. Park Ridge, Ill: American Association of Neurological Surgeons, 1997.

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11

W, Lindsay Kenneth, and Van Gijn J, eds. Subarachnoid haemorrhage. London: Saunders, 1992.

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12

Kakarieka, A. Traumatic subarachnoid haemorrhage. Berlin: Springer, 1997.

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13

Neuropsychological sequelae of subarachnoid hemorrhage and its treatment. Wien: Springer, 2000.

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14

Hütter, B. O. Neuropsychological sequelae of subarachnoid hemorrhage and its treatment. New York: Springer, 2000.

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15

Hütter, B. O. Neuropsychological Sequelae of Subarachnoid Hemorrhage and its Treatment. Vienna: Springer Vienna, 2000. http://dx.doi.org/10.1007/978-3-7091-6327-6.

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16

A dented image: Journeys of discovery from subarachnoid haemorrhage. Hove, East Sussex: Routledge, 2008.

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17

Laakso, Aki. Surgical management of cerebrovascular disease. Wien: Springer, 2010.

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18

Luca, Regli, Hänggi Daniel, Turowski Bernd, Steiger Hans-Jakob, and SpringerLink (Online service), eds. Trends in Neurovascular Surgery. Vienna: Springer-Verlag/Wien, 2011.

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19

T, Andrews Brian, ed. Neurotrauma: Evidence-based answers to common questions. New York: Thieme, 2005.

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20

1935-, Sakai Shizu, Tomita Yasuhiko, Ōniwa Kunihiko 1957-, and Ōniwa Kunihiko, eds. Jin. Tōkyō: Shūeisha, 2009.

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21

Royal College of Physicians of London. Clinical Effectiveness and Evaluation Unit, ed. National clinical guidelines for stroke. 2nd ed. London: Clinical Effectiveness & Evaluation Unit, Royal College of Physicians, 2004.

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22

Kevin Luk, K. H., and Deepak Sharma. Subarachnoid Hemorrhage. Edited by David E. Traul and Irene P. Osborn. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190850036.003.0024.

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Subarachnoid hemorrhage (SAH) is commonly caused by rupture of an intracranial aneurysm, arteriovenous malformation, or due to trauma. Prompt diagnosis and intervention are required to control intracranial pressure, maintain cerebral perfusion, and prevent rebleeding. Clinical grading of the bleed predicts morbidity and mortality, whereas imaging grading predicts risk of cerebral vasospasm. Hydrocephalus can occur as a result of SAH, which requires treatment with an external ventricular drain. Endovascular and open microsurgical procedures are available for securing the vascular abnormalities. Patients are typically monitored in a neurocritical care unit for up to 21 days post-bleed to monitor for the development of cerebral vasospasm/delayed cerebral ischemia (DCI). Mainstay of treatment for DCI includes induced hypertension, balloon angioplasty, and intraarterial vasodilator therapy. In addition, patient may experience significant derangement in their cardiac, pulmonary, and endocrine systems, requiring inotropic support, mechanical ventilation, or insulin infusion therapy.
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23

Clark, Joseph F., Mario Zuccarello, Gail Pyne-Geithman, Norberto Andaluz, Jed A. Hartings, and Opeolu M. Adeoye. Cerebral Vasospasm: Neurovascular Events After Subarachnoid Hemorrhage. Springer, 2014.

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24

Hasan, David. The Natural History of Cerebral Aneurysms. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0109.

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Subarachnoid hemorrhage (SAH) secondary to rupture of cerebral aneurysms represents a relatively small fraction of strokes (5%) but morbidity and mortality associated with aneurysm rupture remain very high despite advances in the treatment of aneurysmal SAH. Cerebral vasospasm (CV) is the leading cause of delayed morbidity and mortality following aneurysmal subarachnoid hemorrhage, as well as delayed neurological dysfunction 1 to 2 weeks after rupture. Endothelial dysfunction is one of the primary contributing factors to CV following aneurysmal SAH, and this is associated with alterations in intracellular adhesion molecule-1 (ICAM-1), matrix metalloproteinases (MM), and the blood-brain barrier[p63].
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25

Chong, Ji Y., and Michael P. Lerario. Worst Headache of Her Life. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190495541.003.0035.

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Aneurysmal subarachnoid hemorrhage typically causes sudden severe headache. Diagnosis is made by CT scan in most cases, but lumbar puncture may be needed if CT findings are normal and suspicion is high. Rapid evaluation and treatment are important because of the high morbidity and mortality associated with rebleeding and vasospasm.
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26

Osborn, Irene P., and Jocelin Jones Molina. Cerebral Aneurysm Clipping. Edited by David E. Traul and Irene P. Osborn. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190850036.003.0005.

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Subarachnoid hemorrhage (SAH) is usually caused by the rupture of an intracranial aneurysm. Craniotomy and surgical management has been the traditional treatment for decades until the development and evolution of endovascular techniques. Operative clipping of cerebral aneurysms is performed less frequently, but the procedure is still required for aneurysms that are not amenable to endovascular coiling. Some centers do not have the skilled personnel to perform endovascular techniques, and craniotomy is therefore necessary to treat the aneurysm and prevent the problem of rebleeding and avoid vasospasm. This discussion will address specifically the perioperative management of surgical clipping for intracranial aneurysms.
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27

Wijdicks, Eelco F. M., and Sarah L. Clark. Vasopressors and Inotropes. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190684747.003.0012.

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Vasopressors and inotropes are used in the neurosciences intensive care unit to treat hypotension and to augment blood pressure. Hypotension can be attributed to abnormal cardiac output, abnormal intravascular volume or abnormal systemic vascular resistance. Vasopressors are needed to manage hemodynamic augmentation in patients with severe cerebral vasospasm and aneurysmal subarachnoid hemorrhage, in patients with critical carotid or basilar artery stenosis producing marginal blood flow, or when patients are maintained in drug-induced comas. The main incentive is to maintain adequate perfusion pressure to the brain and vital organs, particularly the kidneys. This chapter provides the essentials of management of these complex drugs and how to avoid unintended side effects.
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28

Aneurysmal Subarachnoid Hemorrhage. W.B. Saunders Company, 2010.

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29

Subarachnoid Hemorrhage in Clinical Practice. Springer, 2015.

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30

Management Of Aneurysmal Subarachnoid Hemorrhage. E.M.I.S. MEDICAL PUBLISHING, 1993.

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31

Wood, Cristina. Stroke/Subarachnoid Hemorrhage and Pregnancy. Edited by Matthew D. McEvoy and Cory M. Furse. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190226459.003.0055.

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Death from neurological causes is a massive issue in maternal mortality, and the rate of deaths from neurological causes has remained essentially unchanged for the past thirty years. Two of the most common neurological causes of death are stroke and intracranial hemorrhage. These medical emergencies can be more challenging to diagnose and treat in the parturient. This is largely due to the physiologic changes that occur during pregnancy and in the postpartum period, but fetal considerations are also a factor. This chapter focuses on the pathophysiology, assessment, and management of stroke and subarachnoid hemorrhage in pregnancy. A thorough understanding of each of these components is necessary to ensure appropriate and timely care delivery for optimal outcome in the setting of these neurologic emergencies.
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32

Bederson, Joshua B. Subarachnoid Hemorrhage: Pathphysiology and Management. Edited by Joshua B. Bederson. American Association of Neurological Surgeons, 1996.

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33

1936-, Yanagihara Takehiko, Piepgras David G. 1940-, and Atkinson, John L. D., 1954-, eds. Subarachnoid hemorrhage: Medical and surgical management. New York: M. Dekker, 1998.

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34

Martin, Robert D., Gang Chen, John H. Zhang, and Warren Boling. Subarachnoid Hemorrhage: Neurological Care and Protection. Springer, 2019.

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35

Gijn, J. Van, Kenneth W. Lindsay, and M. Vermeulen. Subarachnoid Hemorrhage (Major Problems in Neurology). W.B. Saunders Company, 1993.

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36

Lee, Christoph I. Rule Out Subarachnoid Hemorrhage for Headache. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190223700.003.0003.

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This chapter, found in the headache section of the book, provides a succinct synopsis of a key study examining the use of computed tomography (CT) to rule out a head bleed or subarachnoid hemorrhage among patients with acute headaches. This summary outlines the study methodology and design, major results, limitations and criticisms, related studies and additional information, and clinical implications. Researchers reported that the criteria had high sensitivity and high negative predictive value for identifying subarachnoid hemorrhage among patients presenting to the emergency department with acute nontraumatic headache that reached maximal intensity within 1 hour and with normal neurologic examinations. In addition to outlining the most salient features of the study, a clinical vignette and imaging example are included in order to provide relevant clinical context.
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37

Yanagihara. Subarachnoid Hemorrhage (Neurological Disease and Therapy). Informa Healthcare, 1997.

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38

British Brain & Spine Foundation., ed. Sub-arachnoid haemorrhage: A guide for patients and carers. 2nd ed. London: British Brain and Spine Foundation, 1998.

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39

Gray, Linda R. Subarachnoid Hemorrhage: Epidemiology, Management and Long-Term Health Effects. Nova Science Publishers, Incorporated, 2015.

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40

Brain Abscess and Meningitis : Subarachnoid Hemorrhage: Timing Problems. Springer, 2011.

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41

Lo, Benjamin W. Y., Aurora W. M. Lo, Adrian Upton, Benedict Beng-Teck Taw, and Hidetoshi Kasuya. Pathophysiology of Brain-Body Interactions in Aneurysmal Subarachnoid Hemorrhage. Nova Science Publishers, Incorporated, 2017.

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42

Kim, Patricia. Phenytoin free-fraction determination in patients with subarachnoid hemorrhage. 1998.

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43

Testai, Fernando, and J. Marc Simard, eds. Neuroinflammation as a Target for Intervention in Subarachnoid Hemorrhage. Frontiers Media SA, 2020. http://dx.doi.org/10.3389/978-2-88963-303-6.

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44

(Editor), I. A. Langmoen, Tryggve Lundar (Editor), Rune Aaslid (Editor), and Hans-J. Reulen (Editor), eds. Neurosurgical Management of Aneurysmal Subarachnoid Haemorrhage (Acta Neurochirurgica Supplementum). Springer, 1999.

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45

Fandino, Javier, Serge Marbacher, Ali-Reza Fathi, Carl Muroi, and Emanuela Keller. Neurovascular Events After Subarachnoid Hemorrhage: Towards Experimental and Clinical Standardisation. Springer, 2016.

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46

Fandino, Javier, Serge Marbacher, Ali-Reza Fathi, Carl Muroi, and Emanuela Keller. Neurovascular Events After Subarachnoid Hemorrhage: Towards Experimental and Clinical Standardisation. Springer, 2014.

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47

Fandino, Javier, Serge Marbacher, and Ali-Reza Fathi. Neurovascular Events After Subarachnoid Hemorrhage: Towards Experimental and Clinical Standardisation. Springer, 2014.

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48

Sharma, Deepak, and Julia Metzner. Nontraumatic Intracranial Hemorrhage. Edited by Matthew D. McEvoy and Cory M. Furse. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190226459.003.0062.

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Nontraumatic subarachnoid, intraventricular, or intraparenchymal hemorrhage can be caused by either rupture of an aneurysm or arteriovenous malformation or by coagulopathy, hypertension, or vasculitis. Pituitary apoplexy results from spontaneous hemorrhage or infarction into a pituitary tumor. Additionally, anesthesiologists must be prepared to manage intraoperative bleeding during craniotomies. Successful management of nontraumatic intracranial hemorrhage requires (1) careful preoperative evaluation and preparation considering extracranial manifestations of intracranial bleeding; (2) administration of balanced anesthesia to facilitate surgical exposure and neurophysiological monitoring; (3) maintenance of cerebral perfusion by preserving circulating volume, judicious use of blood product transfusion and vasopressors, and avoidance of excessive hyperventilation; and, when possible, (4) providing timely emergence from anesthesia to allow neurological assessment. Close communication between the surgical and anesthesia teams is critical for optimizing the potential for good patient outcomes.
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49

1960-, Forsting M., and Wanke I, eds. Intracranial vascular malformations and aneurysms: From diagnostic work-up to endovascular therapy. 2nd ed. [Berlin]: Springer, 2008.

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50

Experimental and Clinical Treatment of Subarachnoid Hemorrhage after Rupture of Saccular Intracranial Aneurysms. MDPI, 2020. http://dx.doi.org/10.3390/books978-3-03943-155-7.

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