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Journal articles on the topic 'Sulpiridum'

Author: Grafiati

Published: 2 June 2025

Last updated: 31 July 2025

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1

Shtanko, V.A., A.P. Romanchuk, M.Y. Marish, and G.B. Tesluk. "SENSORIMOTOR EFFECTS OF THE SULPIRIDE USE IN THE COMPLEX OF ANTIHYPERTENSIVE THERAPY IN PRIMARY ARTERIAL HYPERTENSION." Journal of Health Sciences 4, no. 16; 2014 (2014): 89–98. https://doi.org/10.5281/zenodo.16116.

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Primary  arterial  hypertension  (PAH)  is one of the  most widespread cardiovascular diseases. High blood pressure affects the structures of target organs, especially neurons in the brain cortex, neuro-muscular synapses and autonomic ganglia. Their damage causes various neurological disorders and dysfunction of sensory function in patients with PAH.   At the University Clinic of Odessa National Medical University (Center of Restorative and Reconstructive Medicine) were examined and  treated 80 patients (men and women in equal numbers

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Usuki, Satoshi, and Yoshihito Ichikawa. "Effects of Hachimijiogan on Dopamine, Serotonin and Vasoactive Intestinal Peptide in Plasma and Hypothalamus in Sulpiride – Induced Hyperprolactinemic Rats." American Journal of Chinese Medicine 19, no. 03n04 (1991): 217–22. http://dx.doi.org/10.1142/s0192415x91000296.

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Effect of Hachimijiogan (HJ) on dopamine (DA), serotonin (5-HT) and vasoactive intestinal peptide (VIP) was examined in plasma and hypothalamic tissue of sulpiride-induced hyperprolactinemic rats (SHR). Similar to bromocriptine (BR), HJ, in combination with sulpiride, suppressed plasma prolactin levels raised by sulpiride alone. Furthermore, HJ, together with sulpiride, increased plasma DA levels decreased by sulpiride alone, while 5-HT in plasma was increased by sulpiride and HJ, individually or in combination. However, plasma VIP levels were under detection limits on some occasions after HJ

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3

Gerlach, Jes, Kirsten Behnke, Jon Heltberg, Ebbe Munk-Andersen, and Henrik Nielsen. "Sulpiride and Haloperidol in Schizophrenia: A Double-blind Cross-over Study of Therapeutic Effect, Side Effects and Plasma Concentrations." British Journal of Psychiatry 147, no. 3 (1985): 283–88. http://dx.doi.org/10.1192/bjp.147.3.283.

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SummaryIn a double-blind cross-over trial, 20 chronic schizophrenic patients were treated with sulpiride and haloperidol in two 12-week periods. The final median dose of sulpiride was 2000 mg/day (range 800–3200) and of haloperidol 12 mg/day (range 6–24). Sulpiride had an antipsychotic effect and therapeutic profile not significantly different from that of haloperidol. In spite of the high doses of sulpiride, extrapyramidal side-effects were seen less frequently during the first four weeks of the sulpiride period than during the corresponding haloperidol period (P < 0.05), whereas autonomic

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4

&NA;. "Sulpiride." Reactions Weekly &NA;, no. 1397 (2012): 28. http://dx.doi.org/10.2165/00128415-201213970-00104.

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&NA;. "Sulpiride." Reactions Weekly &NA;, no. 563 (1995): 10. http://dx.doi.org/10.2165/00128415-199505630-00033.

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6

&NA;. "Sulpiride." Reactions Weekly &NA;, no. 788 (2000): 10. http://dx.doi.org/10.2165/00128415-200007880-00032.

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7

&NA;. "Sulpiride." Reactions Weekly &NA;, no. 854 (2001): 11. http://dx.doi.org/10.2165/00128415-200108540-00028.

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&NA;. "Sulpiride." Reactions Weekly &NA;, no. 1398 (2012): 39. http://dx.doi.org/10.2165/00128415-201213980-00142.

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9

&NA;. "Sulpiride." Reactions Weekly &NA;, no. 1405 (2012): 35. http://dx.doi.org/10.2165/00128415-201214050-00121.

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10

&NA;. "Sulpiride." Reactions Weekly &NA;, no. 374 (1991): 7. http://dx.doi.org/10.2165/00128415-199103740-00039.

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11

&NA;. "Sulpiride." Reactions Weekly &NA;, no. 1202 (2008): 32. http://dx.doi.org/10.2165/00128415-200812020-00100.

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12

&NA;. "Sulpiride." Reactions Weekly &NA;, no. 918 (2002): 11. http://dx.doi.org/10.2165/00128415-200209180-00037.

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13

&NA;. "Sulpiride." Reactions Weekly &NA;, no. 308 (1990): 11. http://dx.doi.org/10.2165/00128415-199003080-00050.

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14

&NA;. "Sulpiride." Reactions Weekly &NA;, no. 310 (1990): 11. http://dx.doi.org/10.2165/00128415-199003100-00057.

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15

Wagstaff, Antona J., Andrew Fitton, and Paul Benfield. "Sulpiride." CNS Drugs 2, no. 4 (1994): 313–33. http://dx.doi.org/10.2165/00023210-199402040-00007.

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16

&NA;. "Sulpiride." Reactions Weekly &NA;, no. 949 (2003): 15. http://dx.doi.org/10.2165/00128415-200309490-00062.

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17

&NA;. "Sulpiride." Reactions Weekly &NA;, no. 490 (1994): 10. http://dx.doi.org/10.2165/00128415-199404900-00053.

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18

&NA;. "Sulpiride." Reactions Weekly &NA;, no. 514 (1994): 9. http://dx.doi.org/10.2165/00128415-199405140-00034.

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19

&NA;. "Sulpiride." Reactions Weekly &NA;, no. 526 (1994): 11. http://dx.doi.org/10.2165/00128415-199405260-00035.

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20

Lee, Hyang-Ae, Ki-Suk Kim, Sang-Joon Park, and Eun-Joo Kim. "Cellular Mechanism of the QT Prolongation Induced by Sulpiride." International Journal of Toxicology 28, no. 3 (2009): 207–12. http://dx.doi.org/10.1177/1091581809337261.

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In this study, the authors investigated the electrophysiological effect of sulpiride on cardiac repolarization using conventional microelectrode recording techniques in isolated canine Purkinje fibers and a whole-cell patch clamp technique in transiently transfected cells with the hERG, KCNQ1/KCNE1, KCNJ2, and SCN5A cDNA and in rat cardiac myocytes for ICa. In studies of action potential duration, 10 μM, 100 μM, 300 μM, and 1 mM sulpiride prolonged action potential duration in a concentration-dependent manner. In studies of cardiac ion channels, sulpiride did not significantly affect INa, ICa,

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21

Hashemi, Marziyeh-Sadat, Manizheh Karami, and Mahdiyeh Jafarpour Fard. "Effect of Sulpiride on Dopaminergic Synapse of Dorsal Hippocampus of Morphine-Treated Rats." Caspian Journal of Neurological Sciences 8, no. 4 (2022): 206–12. http://dx.doi.org/10.32598/cjns.4.31.330.1.

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Background: As previous studies show, several effects of morphine are induced by the dopaminergic system. Sulpiride is a dopamine D2 receptor (DAD2) antagonist widely used in clinics to treat DArelated disorders. DAD2 receptors are abundant at hippocampal cornu ammonis (CA1). Objectives: This study aimed to investigate the possible interaction of morphine and sulpiride on DA synapses in CA1. Materials & Methods: In this study, 48 Wistar rats weighing 220 to 250 g were used. These animals were classified into eight groups (6 rats per group): saline control group (1 mL/kg), morphine group (5

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22

Fedotova, Julia. "Effects of Stimulation and Blockade of Receptor on Depression-Like Behavior in Ovariectomized Female Rats." ISRN Pharmacology 2012 (March 1, 2012): 1–8. http://dx.doi.org/10.5402/2012/305645.

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The aim of the present study was to explore the hedonic effects of D2 receptor agonist, quinpirole and D2 receptor antagonist, and sulpiride alone or in combination with a low dose of 17β-E2-estradiol (17β-E2) in the adult ovariectomized female rats (OVX). OVX rats of Wistar strain were used in all experiments. Two weeks after surgery rats were chronically treated with vehicle, a low dose of 17β-E2 (5.0 μg/rat), quinpirole (0.1 mg/kg), sulpiride (10.0 mg/kg), quinpirole plus 17β-E2, or sulpiride plus 17β-E2 for 14 days before the forced swimming test. We found that sulpiride significantly decr

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23

Hanew, Kunihiko, Atsushi Utsumi, Akira Sugawara, Yasuyuki Shimizu, and Kaoru Yoshinaga. "Evidence for dopamine-related and TRH-related pituitary TSH and PRL pools in patients with prolactinoma." Acta Endocrinologica 124, no. 5 (1991): 545–52. http://dx.doi.org/10.1530/acta.0.1240545.

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Abstract. The sources of TSH, which was excessively released by sulpiride (dopamine D2 receptor antagonist), were studied in 15 female patients with PRL-secreting adenoma (18-43 years). Sequential 3-day administration of sulpiride (100 mg, im) was given to 12 patients with prolactinoma and 6 normal female subjects (19-24 years). Patients with prolactinoma showed much greater TSH responses than normal subjects on the first day. However, TSH responses to sulpiride disappeared on the 2nd and 3rd day in both groups. In contrast, plasma PRL responses to the 1st sulpiride administration were smaller

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24

Campos, Lucas, Isabele Cyrillo, and Jorge Henna Neto. "Desenvolvimento e avaliação de material alternativo de solução coloide de sulpirida para tratamento de esquizofrenia." Debates em Psiquiatria 14 (November 15, 2024): 1–8. http://dx.doi.org/10.25118/2763-9037.2024.v14.1351.

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Introdução: A sulpirida é um agente antipsicótico de primeira geração usado para o tratamento de esquizofrenia. Sua administração é realizada por via oral, porém está associada a muitos efeitos colaterais. Alguns deles são constipação, sedação, insônia, ganho ponderal, acatisia e parkinsonismo. Por causa desses efeitos, a adesão ao tratamento com a sulpirida torna-se um grande desafio. Um método alternativo para administração de sulpirida seria então através da aplicação de um gel na pele do paciente. Tal gel pode oferecer muitas vantagens sobre as formas tradicionais de administração, como re

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25

Maury, Luc, Jean-Louis Delarbre, Joëlle Rambaud, and Bernard Pauvert. "Caractérisation et spectres de vibration du sulpiride." Canadian Journal of Chemistry 65, no. 7 (1987): 1613–18. http://dx.doi.org/10.1139/v87-270.

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The authors have characterized two forms for sulpiride by infrared spectroscopy and Raman scattering (lattice vibrations). An assignment of the fundamental vibrations of the amide and sulfonamide groups involved in the inter or intramolecular hydrogen bonds has been proposed. Comparison of the sulpiride chlorhydrate spectra with those of sulpiride indicates that the chlorhydrate itself will be in the chelated form.

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26

Pérez, R. L., G. A. Machiavelli, M. I. Romano, and J. A. Burdman. "Prolactin release, oestrogens and proliferation of prolactin-secreting cells in the anterior pituitary gland of adult male rats." Journal of Endocrinology 108, no. 3 (1986): 399–403. http://dx.doi.org/10.1677/joe.0.1080399.

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ABSTRACT Relationships among the release of prolactin, the effect of oestrogens and the proliferation of prolactin-secreting cells were studied under several experimental conditions. Administration of sulpiride or oestradiol released prolactin and stimulated cell proliferation in the anterior pituitary gland of adult male rats. Clomiphene completely abolished the rise in cell proliferation, but did not interfere with the sulpiride-induced release of prolactin. Treatment with oestradiol plus sulpiride significantly increased serum prolactin concentrations and the mitotic index compared with the

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27

Bell, Caroline, Shamina Bhika, Richard Porter, et al. "The response to sulpiride in major depression before and after cognitive behavioural therapy: D2 receptor function." Acta Neuropsychiatrica 20, no. 4 (2008): 199–206. http://dx.doi.org/10.1111/j.1601-5215.2008.00284.x.

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Background:Previous studies have suggested that antidepressant treatment of depression may potentiate dopamine transmission through increased sensitivity of postsynaptic D2 receptors.Method:D2 receptor function was assessed in 24 patients with major depression before and 16 patients after 16 weeks of treatment with cognitive behavioural therapy (CBT) using a challenge with a selective D2 antagonist, sulpiride. Four hundred milligrams of sulpiride was administered orally on two test days and response was measured by the change in prolactin levels and changes in self-rating scale measures of moo

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28

Shiloh, Roni, Zvi Zemishlany, Dov Aizenberg, et al. "Sulpiride augmentation in people with schizophrenia partially responsive to clozapine." British Journal of Psychiatry 171, no. 6 (1997): 569–73. http://dx.doi.org/10.1192/bjp.171.6.569.

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BackgroundWe hypothesised that a combined regimen of clozapine, a relatively weak D2-dopaminergic antagonist, and sulpiride, a selective D2 blocker, would demonstrate a greater antipsychotic efficacy by enhancing the D2 blockade of clozapine.MethodTwenty-eight people with schizophrenia, previously unresponsive to typical antipsychotics and only partially responsive to current treatment with clozapine, received, double-blind, 600 mg/day sulpiride or placebo, in addition to an ongoing clozapine treatment. The clinical status was evaluated before, during, and at the end of 10 weeks of sulpiride a

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29

Vázquez-Carrillo, Dina I., Ana Luisa Ocampo-Ruiz, Arelí Báez-Meza, et al. "Dopamine D2 receptor antagonist counteracts hyperglycemia and insulin resistance in diet-induced obese male mice." PLOS ONE 19, no. 4 (2024): e0301496. http://dx.doi.org/10.1371/journal.pone.0301496.

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Obesity leads to insulin resistance (IR) and type 2 diabetes. In humans, low levels of the hormone prolactin (PRL) correlate with IR, adipose tissue (AT) dysfunction, and increased prevalence of T2D. In obese rats, PRL treatment promotes insulin sensitivity and reduces visceral AT adipocyte hypertrophy. Here, we tested whether elevating PRL levels with the prokinetic and antipsychotic drug sulpiride, an antagonist of dopamine D2 receptors, improves metabolism in high fat diet (HFD)-induced obese male mice. Sulpiride treatment (30 days) reduced hyperglycemia, IR, and the serum and pancreatic le

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30

Lewis, Mark, and Peter W. Howie. "Induction of transient hyperprolactinaemia in neonatal rats by direct or maternal treatment with the dopamine receptor blocker, sulpiride." Acta Endocrinologica 115, no. 3 (1987): 357–64. http://dx.doi.org/10.1530/acta.0.1150357.

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Abstract. Prolactin was measured in the plasma of neonatal rats after iv and ip injection of the dopamine receptor blocking drug sulpiride, and after its ip injection to neonatal rats' nursing mothers. The sulpiride dose PRL response relationship in 10 –25 day old neonatal rats was similar to that found in lactating rats, with a threshold sensitivity around 29 nmol sulpiride/kg body weight and a maximal response at about 2.9 μmol/kg. Absolute levels of PRL in the neonate (both peak and increment-over-basal) were, however, 90% lower than in adults. Treatment of lactating mothers with a maximall

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31

&NA;. "Sulpiride withdrawal." Reactions Weekly &NA;, no. 693 (1998): 14. http://dx.doi.org/10.2165/00128415-199806930-00047.

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32

&NA;. "Sulpiride overdose." Reactions Weekly &NA;, no. 1152 (2007): 24. http://dx.doi.org/10.2165/00128415-200711520-00076.

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33

&NA;. "Sulpiride overdose." Reactions Weekly &NA;, no. 1407 (2012): 36. http://dx.doi.org/10.2165/00128415-201214070-00127.

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34

&NA;. "Sulpiride overdose." Reactions Weekly &NA;, no. 1283 (2010): 84. http://dx.doi.org/10.2165/00128415-201012830-00267.

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35

&NA;. "Risperidone/sulpiride." Reactions Weekly &NA;, no. 1327 (2010): 32. http://dx.doi.org/10.2165/00128415-201013270-00111.

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36

&NA;. "Dipyrone/sulpiride." Reactions Weekly &NA;, no. 1426 (2012): 20. http://dx.doi.org/10.2165/00128415-201214260-00067.

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37

Liu, Jifeng, Weidong Cao, Haibo Qiu, Xiuhua Sun, Xiurong Yang, and Erkang Wang. "Determination of Sulpiride by Capillary Electrophoresis with End-Column Electrogenerated Chemiluminescence Detection." Clinical Chemistry 48, no. 7 (2002): 1049–58. http://dx.doi.org/10.1093/clinchem/48.7.1049.

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Abstract Background: Capillary electrophoresis (CE) with tris(2,2′-bipyridyl)ruthenium(II) [Ru(bpy)32+]-electrogenerated chemiluminescence (ECL) detection is a promising method for clinical analysis. In this study, a method combining CE with Ru(bpy)32+ ECL (CE-ECL) detection that can be applied to amine-containing clinical species was developed, and the performance of CE-ECL as a quantitative method for determination of sulpiride in human plasma or urine was evaluated. Methods: Sulpiride was separated by capillary zone electrophoresis in uncoated fused-silica capillaries [50 cm × 25 μm (i.d.)]

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38

Bailey, Victoria N., Jennifer L. Sones, Caroline M. Camp, and Erin L. Oberhaus. "72 Endocrine and ovarian responses to combined estradiol benzoate-sulpiride in anestrous mares treated with kisspeptin." Journal of Animal Science 98, Supplement_2 (2020): 36. http://dx.doi.org/10.1093/jas/skz397.082.

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Abstract The objective of this study was to determine if incorporation of kisspeptin 10 (Kp10) into an estradiol benzoate (EB)-sulpiride treatment would result in greater endocrine responses, and a greater number of mares ovulating within 28 days of treatment compared to EB-sulpiride alone. Eighteen anestrous mares were blocked by horse type (light horse and pony crosses), body condition, and age, then randomly assigned to treatment or control. On day 0, all mares received 50 mg EB. On day 1, mini osmotic pumps containing saline (n = 9) or Kp10 (50 mg/hour; n = 9) were inserted subcutaneously

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39

Larisa, Alagić-Džambić, Dacić Minela, Bašić Kenan, et al. "Spectrophotometric Determination of Sulpiride in Pharmaceutical Products." Pharmaceutical and Chemical Journal 7, no. 4 (2020): 75–79. https://doi.org/10.5281/zenodo.13955707.

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A simple spectrophotometric method was developed and validated for determination of Sulpiride in pharmaceutical products. The method was based using two different solvent: 0.1 mol/L sodium hydroxide and 0.1 mol/L hydrochloric acid. The absorbance maximum was at 291 nm. The correlation coefficient were r=0.9994 (in 0.1 mol/L sodium hydroxide as solvent) and r=1.0000 (in 0.1 mol/L hydrochloric acid as solvent). The method was validated and results obtained for the assay of three different brands of Sulpiride capsules, hard. The proposed method was successfully applied to the spectrophotometric d

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40

Ayoub, Abdallah Mohamed, Mahmoud Mokhtar Ibrahim, Marwa Helmy Abdallah, and Mahmoud A. Mahdy. "INTRANASAL MICROEMULGEL AS SURROGATE CARRIER TO ENHANCE LOW ORAL BIOAVAILABILITY OF SULPIRIDE." International Journal of Pharmacy and Pharmaceutical Sciences 8, no. 10 (2016): 188. http://dx.doi.org/10.22159/ijpps.2016v8i10.13776.

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<p><strong>Objective: </strong>The purpose of this study is to evaluate microemulsion based gel (MBG) of sulpiride "a poorly water soluble antipsychotic with low oral bioavailability."</p><p><strong>Methods: </strong>Gelling polymers such as sodium carboxymethylcellulose (CMC-Na), hydroxyl propyl methyl cellulose (HPMC K4m), carbopol 940 and Na alginate were evaluated for their potential to gel sulpiride microemulsions (MEs) without affecting the MEs structure. Also, sulpiride solution (SS) and conventional gel (without ME) were prepared and compared w

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41

Yang, Zeng-Jin, Michel Torbey, Xiaoling Li, et al. "Dopamine Receptor Modulation of Hypoxic—Ischemic Neuronal Injury in Striatum of Newborn Piglets." Journal of Cerebral Blood Flow & Metabolism 27, no. 7 (2007): 1339–51. http://dx.doi.org/10.1038/sj.jcbfm.9600440.

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Dopamine receptors regulate glutamatergic neurotransmission and Na+,K+-ATPase via protein kinase A (PKA) and dopamine- and cAMP-regulated phosphoprotein of 32 kDa (DARPP-32)-dependent signaling. Consequently, dopamine receptor activation may modulate neonatal hypoxic–ischemic (H–I) neuronal damage in the selectively vulnerable putamen enriched with dopaminergic receptors. Piglets subjected to two durations of hypoxia followed by asphyxic cardiac arrest were treated with a D1-like (SCH23390) or D2-like (sulpiride) receptor antagonist. At 4 days of recovery from less severe H–I, the remaining vi

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42

Burchinsky, Serhii. "Anxety Syndrome in Neurologic Practice: Possibilities and Strategies of Pharmacotherapy." Family Medicine, no. 4 (December 30, 2016): 107–10. http://dx.doi.org/10.30841/2307-5112.4.2016.248554.

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In the present paper a modern approaches to pharmacotherapy of anxiety syndrome in neurological practice have been looked. An advantages and disadvantages of main groups of drugs, using with this purpose have been analyzed. A main attention paid to possibilities of drugs representing “small neuroleptics” and its “golden standard” sulpiride (Eglonil) as an instrument of complex correction of anxiety, phobic, depressive and psychoautonomic symptoms of neurological pathology. A questions of safety during therapy of sulpiride (Eglonil) as one of the main choice criteria of this drug have been look

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43

Abd, Ali Aziz, Oday Alawi Al-Juhaishi, and Qusai Saleh Jumma. "Effects of Sulpiride on the Reproductive System of Male Rats after Puberty." World's Veterinary Journal 15, no. 1 (2025): 42–48. https://doi.org/10.54203/scil.2025.wvj5.

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Sulpiride is an antipsychotic drug commonly used in humans to mitigate the effects of stress by selectively targeting central dopaminergic receptors. During male rat puberty, neurotransmitter systems, including the dopaminergic system, undergo significant development, playing a crucial role in the release of gonadal hormones and the regulation of reproductive function. The present study aimed to investigate the effects of sulpiride on reproduction parameters in adult male rats. This study used 30 adult male rats with an average body weight of 250-300g and an average age of 90-95 days. The rats

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44

Agbaba, Danica, Tatjana Miljkovic, Valentina Marinkovic, Dobrila Zivanov-Stakic, and Sote Vladimirov. "Quantitative Analysis of Sulpiride and Impurities of 2-Aminomethyl-1-Ethylpyrrolidine and Methyl-5-Sulphamoyl-2-Methoxybenzoate in Pharmaceuticals by High-Performance Thin-Layer Chromatography and Scanning Densitometry." Journal of AOAC INTERNATIONAL 82, no. 4 (1999): 825–29. http://dx.doi.org/10.1093/jaoac/82.4.825.

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Abstract A simple and reliable thin-layer chromatographic method for determining sulpiride and impurities of 2-aminomethyl-1-ethylpyrrolidine and methyl-5-sulphamoyl-2-methoxybenzoate was developed and validated. A methylene chloride–methanol–ammonia solution (25%; 18 + 2.8 + 0.4, v/v) solvent system is used for separation and quantitative evaluation of chromatograms. The chromatographic plate is first scanned at 240 nm to locate chromatographic zones corresponding to sulpiride and methyl-5-sulphamoyl-2-methoxybenzoate. Then 2-aminomethyl-1-ethylpyrrolidine is derivatized in situ with ninhydri

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45

Abu-Jayyab, Abdulrahim, and Ahmed Mahgoub. "Role of Na+/K+-stimulated adenosine triphosphatase in the action of dopaminergic-D2 receptors of the liver in rats." Bioscience Reports 7, no. 11 (1987): 839–42. http://dx.doi.org/10.1007/bf01119474.

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The dopamine receptor agonist, bromocriptine, in a dose of 10 mg/kg i.p. for 14 days, in rats caused a significant increase in liver Na+/K+-ATPase activity, whereas sulpiride, a dopamine receptor antagonist, in a dose of 10 mg/kg, i.p. for 14 days, in rats, caused a significant decrease in liver Na+/K+-ATPase activity. Injection of bromocriptine and sulpiride simultaneously in a group of rats, under the same conditions and using the same doses caused a complete block of both stimulatory activity of bromocriptine and inhibitory activity of sulpiride on liver Na+/K+-ATPase activity. It is sugges

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46

Burchinsky, Serhii. "Anxiety Syndrome in Neurologic Practice: Possibilities and Strategies of Pharmacotherapy." Family Medicine, no. 4 (October 5, 2016): 107–10. https://doi.org/10.30841/2307-5112.4.2016.248554.

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In the present paper a modern approaches to pharmacotherapy of anxiety syndrome in neurological practice have been looked. An advantages and disadvantages of main groups of drugs, using with this purpose have been analyzed. A main attention paid to possibilities of drugs representing “small neuroleptics” and its “golden standard” sulpiride (Eglonil) as an instrument of complex correction of anxiety, phobic, depressive and psychoautonomic symptoms of neurological pathology. A questions of safety during therapy of sulpiride (Eglonil) as one of the main choice criteria of

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47

Abdallah, Marwa H., Mona M. Shahien, Alia Alshammari, et al. "The Exploitation of Sodium Deoxycholate-Stabilized Nano-Vesicular Gel for Ameliorating the Antipsychotic Efficiency of Sulpiride." Gels 10, no. 4 (2024): 239. http://dx.doi.org/10.3390/gels10040239.

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The present study explored the effectiveness of bile-salt-based nano-vesicular carriers (bilosomes) for delivering anti-psychotic medication, Sulpiride (Su), via the skin. A response surface methodology (RSM), using a 33 Box–Behnken design (BBD) in particular, was employed to develop and optimize drug-loaded bilosomal vesicles. The optimized bilosomes were assessed based on their vesicle size, entrapment efficiency (% EE), and the amount of Sulpiride released. The Sulpiride-loaded bilosomal gel was generated by incorporating the optimized Su-BLs into a hydroxypropyl methylcellulose polymer. Th

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&NA;. "Citalopram/sulpiride overdose." Reactions Weekly &NA;, no. 1136 (2007): 10–11. http://dx.doi.org/10.2165/00128415-200711360-00025.

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&NA;. "Cibenzoline/flecainide/sulpiride." Reactions Weekly &NA;, no. 825 (2000): 6. http://dx.doi.org/10.2165/00128415-200008250-00015.

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&NA;. "Doxepin/sulpiride overdose." Reactions Weekly &NA;, no. 1406 (2012): 19. http://dx.doi.org/10.2165/00128415-201214060-00062.

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