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Relevant bibliographies by topics / Superior orbital fissure

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Academic literature on the topic 'Superior orbital fissure'

Author: Grafiati

Published: 4 June 2025

Last updated: 14 July 2025

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Journal articles on the topic "Superior orbital fissure"

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Morard, Marc, Vassily Tcherekayev, and Nicolas de Tribolet. "The Superior Orbital Fissure." Neurosurgery 35, no. 6 (1994): 1087–93. http://dx.doi.org/10.1227/00006123-199412000-00011.

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Morard, Marc, Vassily Tcherekayev, and Nicolas de Tribolet. "The Superior Orbital Fissure." Neurosurgery 35, no. 6 (1994): 1087???1093. http://dx.doi.org/10.1097/00006123-199412000-00011.

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Chen, Chien-Tzung, and Yu-Ray Chen. "Traumatic Superior Orbital Fissure Syndrome: Current Management." Craniomaxillofacial Trauma & Reconstruction 3, no. 1 (2010): 9–16. http://dx.doi.org/10.1055/s-0030-1249369.

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Traumatic superior orbital fissure syndrome is an uncommon complication of craniomaxillofacial trauma with an incidence of less than 1%. The syndrome is characterized by ophthalmoplegia, ptosis, proptosis of eye, dilation and fixation of the pupil, and anesthesia of the upper eyelid and forehead. This article describes a detailed anatomy of the superior orbital fissure as it related to pathophysiology and clinical findings. Etiology and diagnosis are established after detailed physical and radiographic examination. On the basis of our clinical experience in the management of superior orbital fissure syndrome and from the data reported previously in the literature, an algorithm for treatment of traumatic superior orbital fissure syndrome including use of steroid, surgical decompression of superior orbital fissure, and reduction of concomitant facial fracture is presented and its rationale discussed.

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Krupp, Serge, and N. Zachariades. "The superior orbital fissure syndrome." Plastic and Reconstructive Surgery 77, no. 6 (1986): 1016. http://dx.doi.org/10.1097/00006534-198606000-00047.

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McAvoy, C. E., B. Lacey, and A. B. Page. "Traumatic superior orbital fissure syndrome." Eye 18, no. 8 (2004): 844–45. http://dx.doi.org/10.1038/sj.eye.6701320.

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Pendás, S. Llorente, and J. M. Albertos Castro. "Traumatic superior orbital fissure syndrome." Journal of Oral and Maxillofacial Surgery 53, no. 8 (1995): 934–36. http://dx.doi.org/10.1016/0278-2391(95)90285-6.

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Zachariades, Nicholas, Eleftherios Vairaktaris, Demetrius Papavassiliou, Ioannis Papademetriou, Michael Mezitis, and Demetrius Triantafyllou. "The superior orbital fissure syndrome." Journal of Maxillofacial Surgery 13 (1985): 125–28. http://dx.doi.org/10.1016/s0301-0503(85)80031-x.

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Di Somma, Alberto, Norberto Andaluz, Luigi Maria Cavallo, et al. "Endoscopic transorbital superior eyelid approach: anatomical study from a neurosurgical perspective." Journal of Neurosurgery 129, no. 5 (2018): 1203–16. http://dx.doi.org/10.3171/2017.4.jns162749.

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OBJECTIVERecent studies have proposed the superior eyelid endoscopic transorbital approach as a new minimally invasive route to access orbital lesions, mostly in otolaryngology and maxillofacial surgeries. The authors undertook this anatomical study in order to contribute a neurosurgical perspective, exploring the anterior and middle cranial fossa areas through this purely endoscopic transorbital trajectory.METHODSAnatomical dissections were performed in 10 human cadaveric heads (20 sides) using 0° and 30° endoscopes. A step-by-step description of the superior eyelid transorbital endoscopic route and surgically oriented classification are provided.RESULTSThe authors’ cadaveric prosection of this approach defined 3 modular routes that could be combined. Two corridors using bone removal lateral to the superior and inferior orbital fissures exposed the middle and anterior cranial fossa (lateral orbital corridors to the anterior and middle cranial base) to unveil the temporal pole region, lateral wall of the cavernous sinus, middle cranial fossa floor, and frontobasal area (i.e., orbital and recti gyri of the frontal lobe). Combined, these 2 corridors exposed the lateral aspect of the lesser sphenoid wing with the Sylvian region (combined lateral orbital corridor to the anterior and middle cranial fossa, with lesser sphenoid wing removal). The medial corridor, with extension of bone removal medially to the superior and inferior orbital fissure, afforded exposure of the opticocarotid area (medial orbital corridor to the opticocarotid area).CONCLUSIONSAlong with its minimally invasive nature, the superior eyelid transorbital approach allows good visualization and manipulation of anatomical structures mainly located in the anterior and middle cranial fossae (i.e., lateral to the superior and inferior orbital fissures). The visualization and management of the opticocarotid region medial to the superior orbital fissure are more complex. Further studies are needed to prove clinical applications of this relatively novel surgical pathway.

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Akdemir Aktaş, Hilal, Şahin Hanalioğlu, Osman Tunç, and İlkan Tatar. "Revisiting anatomical structures of the superior orbital fissure using with interactive 3D-PDF model." Acta Medica 54, no. 3 (2023): 165–71. http://dx.doi.org/10.32552/2023.actamedica.907.

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The superior orbital fissure is an important cleft that connects the orbit with the middle cranial fossa. The upper border of this fissure is formed by the lesser wing of the sphenoid bone, anterior clinoid process, and optic strut. The lower border is formed by the greater wing of the sphenoid bone. The oculomotor, trochlear, ophthalmic, abducens nerves and orbital veins pass through this small slit. The aim of this study was to review anatomical structures of the superior orbital fissure, through a 3D-PDF model that simplifies the understanding of complex anatomy of this region. According to the literature, any major artery does not pass through it, but it is closely related to the internal carotid artery. There are numerous intracranial-extracranial anastomoses around it. While extracranial branches originate from the maxillary artery, intracranial branches arise from the inferolateral trunk or the ophthalmic artery. Nerves and vascular structures related with this fissure can be damaged due to post-traumatic sphenoid fractures, infectious diseases, aneurysms, carotid-cavernous fistulas, and neoplasms. Surgeries involving the superior orbital fissure are quite complex as there are many important anatomical structures in this region. The radiological anatomy of this fissure in normal and pathological conditions is still an under-studied subject in the literature. There is a need for more detailed studies related to the superior orbital fissure enriching with anatomic models and including pathological conditions. The 3D-PDF model of the superior orbital fissure is an innovative tool to enhance the knowledge of the anatomical structures related with this region. Better understanding of this critical region is necessary to perform safe and successful surgical procedures.

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Shields, J. A., J. Kapustiak, V. Arbizo, J. J. Augsburger, and R. E. Schnitzer. "Orbital Neurilemoma With Extension Through the Superior Orbital Fissure." Archives of Ophthalmology 104, no. 6 (1986): 871–73. http://dx.doi.org/10.1001/archopht.1986.01050180105040.

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Book chapters on the topic "Superior orbital fissure"

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Campbell, Ashley A., and Gary Joseph Lelli. "Superior Orbital Fissure." In Encyclopedia of Ophthalmology. Springer Berlin Heidelberg, 2017. http://dx.doi.org/10.1007/978-3-642-35951-4_1329-1.

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Campbell, Ashley A., and Gary Joseph Lelli. "Superior Orbital Fissure." In Encyclopedia of Ophthalmology. Springer Berlin Heidelberg, 2018. http://dx.doi.org/10.1007/978-3-540-69000-9_1329.

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Okano, Atsushi, Stefan Lieber, and Shunya Hanakita. "Superior Orbital Fissure and Inferior Orbital Fissure." In Orbital Apex and Periorbital Skull Base Diseases. Springer Nature Singapore, 2023. http://dx.doi.org/10.1007/978-981-99-2989-4_3.

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Bernardo, Antonio, Alexander I. Evins, and Sergio Corvino. "Microsurgical Anatomy of the Superior and Inferior Orbital Fissures." In Cranio-Orbital Mass Lesions. Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-35771-8_3.

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Jordan, David, Louise Mawn, and Richard L. Anderson. "Orbital Bones." In Surgical Anatomy of the Ocular Adnexa. Oxford University Press, 2012. http://dx.doi.org/10.1093/oso/9780199744268.003.0009.

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The paired orbital cavities are formed by the facial bones and serve as sockets for the eyes. The orbital bones and the structures contained within the orbit (connective tissue, fat, nerves, vessels) act to support, protect, and maximize the function of the eye. In form, the orbit is roughly a quadrilateral pyramid with rounded angles and resembles a pear. Its volume in the average individual is 30 cc, of which the eyeball contributes about 7.5 cc (range: 6.9–9.0 cc). There are four surfaces: the roof, floor, lateral wall, and medial wall. The base of the pyramid is the opening onto the face (orbital entrance) and is circumscribed by the orbital margin (or orbital rim). The orbit narrows inward to its termination, the apex. The widest portion of the orbital cavity lies 5 to 10 mm behind the orbital rim. The orbit is made up of seven bones: frontal, sphenoid, zygomatic, malar, palatine, lacrimal, and ethmoid. Superiorly, the orbit is bordered by the anterior cranial fossa and the frontal sinus. Nasally, the ethmoid sinus is separated from the medial orbital wall by the thin lamina papyracea of the ethmoid bone. Inferiorly, the maxillary sinus lies beneath the orbital floor. The lateral orbit is bordered anteriorly by the temporalis fossa, and posteriorly it borders the middle cranial fossa. The lateral and medial walls of each orbit form an angle of approximately 45 ° with each other. The two medial walls diverge somewhat posteriorly but are almost parallel to each other (being about 3 mm farther apart posteriorly than at the orbital margin). The lateral orbital walls of the two orbits form a 90 ° angle with each other. The four walls of each orbit converge posteriorly toward the apex, where the optic canal and superior orbital fissure pass into the middle cranial fossa. The overall dimensions of the orbit, especially its depth, are quite variable. An orbital surgeon cannot rely on precise measurements as a guide to the exact location of the optic canal or superior orbital fissure.

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Thurtell, Matthew J., and Robert L. Tomsak. "Syndromes of the Orbital Apex, Superior Orbital Fissure, and Cavernous Sinus." In Neuro-Ophthalmology, edited by Matthew J. Thurtell and Robert L. Tomsak. Oxford University Press, 2019. http://dx.doi.org/10.1093/med/9780190603953.003.0038.

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Lesions in the orbital apex, superior orbital fissure, and cavernous sinus can give rise to characteristic combinations of cranial nerve palsies. In this chapter, we begin by reviewing the clinical features and common causes of syndromes of the orbital apex, superior orbital fissure, and cavernous sinus. We go on to discuss the pathogenesis and clinical features of rhino-orbital mucormycosis in detail, because it has a grave prognosis if it is not diagnosed and treated in a timely fashion. We then describe the roles and importance of imaging and tissue biopsy for its diagnosis. Lastly, we review the management of rhino-orbital mucormycosis, which includes rapid reversal of predisposing factors (such as diabetic ketoacidosis), immediate initiation of empiric antifungal treatment, and early surgical debridement of infected tissue.

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Thurtell, Matthew J., Robert L. Tomsak, and Robert B. Daroff. "Syndromes of the Orbital Apex, Superior Orbital Fissure, and Cavernous Sinus." In Neuro-Ophthalmology. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780195390841.003.0034.

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Lesions in the orbital apex, superior orbital fissure, and cavernous sinus can give rise to characteristic combinations of cranial nerve palsies. In this chapter, we briefly review the possible causes of these syndromes. We discuss rhino-orbital mucormycosis in detail, because it has a grave prognosis if it is not diagnosed and treated in a timely fashion.

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van Gijn, Daniel R., and Jonathan Dunne. "The cranial nerves." In Oxford Handbook of Head and Neck Anatomy, edited by Susan Standring and Simon Eccles. Oxford University Press, 2022. http://dx.doi.org/10.1093/med/9780198767831.003.0004.

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There are 12 pairs of cranial nerves that are individually named and numbered using Roman numerals. Only some cranial nerves are mixed in function, i.e. they carry both sensory and motor fibres; others are purely sensory or motor and some may also carry pre- or post-ganglionic parasympathetic fibres. They pass through foramina in the base of the skull and are the olfactory (through cribriform plate to the nasal cavity), optic (through the optic foramen to the eye), oculomotor (through the cavernous sinus and superior orbital fissure to supply the eye), trochlear (as per oculomotor), trigeminal (three main branches that pass through the superior orbital fissure, foramen rotundum and foramen ovale, respectively), abducens (as per oculomotor), facial (through stylomastoid foramen to supply muscles of facial expression), vestibulocochlear (through the internal acoustic canal to control balance and hearing), glossopharyngeal, vagus, accessory (all pass through the jugular foramen) and hypoglossal (through the hypoglossal canal to control movements of the tongue) nerves.

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Kikkawa, Don O., and Christine C. Annunziata. "Fractures Involving the Orbit." In Surgery of the Eyelid, Lacrimal System, and Orbit. Oxford University Press, 2011. http://dx.doi.org/10.1093/oso/9780195340211.003.0023.

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Orbital and periorbital injury can occur with localized trauma to the eye or in the setting of multiple trauma associated with injury to other vital organs. A reported 16% of major trauma patients have ocular or orbital injury, and 55% of patients with facial injury have associated ocular or orbital injury. In general, the amount of ocular, soft tissue, and bony damage is related to the amount, duration, and direction of force applied to the orbit and face. Nevertheless, orbital injury is common and can be a subtle finding in the context of other facial or life-threatening injuries. Geometrically, the bony orbit most closely resembles a four-sided pyramid consisting of an apex, a base, and four sides: roof, floor, medial wall, and lateral wall. The absence of the orbital floor posteriorly and the inclination of the lateral wall toward the medial wall changes the geometric shape from a four-sided pyramid to a three-sided pyramid at the orbital apex. The bony margin circumscribes the orbital entrance and provides anterior support for the thin bones of the interior walls of the orbit. Rounding of the orbital walls blends demarcation of the superior, medial, inferior, and lateral walls. The entrance measures 40 mm horizontally and 32 mm vertically. The widest portion of the orbital margin lies about 1 cm behind the anterior orbital rim. In adults, the depth from orbital rim to apex varies from 40 to 45 mm. Safe subperiosteal dissection may be accomplished along the lateral wall and orbital floor for 22 mm and along the medial wall and orbital roof for 30 mm. The volume of the orbit is approximately 30 cc. The triangular floor of the orbit serves as the roof of the maxillary sinus. Several areas of thin bone create weak points in the orbital floor that are susceptible to fracture. The thinnest portion is medial to the infraorbital groove and canal, particularly posteriorly, where the medial wall has no bony support. In the posterior aspect of the floor, the infraorbital fissure extends as the infraorbital canal.

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Wong, Agnes. "Nuclear and Infranuclear Ocular Motor Disorders." In Eye Movement Disorders. Oxford University Press, 2008. http://dx.doi.org/10.1093/oso/9780195324266.003.0021.

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Binocular diplopia is usually caused by strabismus, whereas monocular diplopia is usually caused by ocular diseases. Incomitant diplopia is usually caused by an acquired strabismus resulting from abnormal innervation or mechanical restriction. The oculomotor (third) nerve: ■ Innervates the medial rectus, superior rectus, inferior rectus, inferior oblique, and levator palpebrae muscles ■ Carries parasympathetic fibers to the iris sphincter and the ciliary body. ■ Common causes of third nerve palsy: Adults: aneurysms, vascular disease (including ischemia, diabetes, hypertension, and inflammatory arteritis), trauma, migraine Children: birth trauma, accidental trauma, neonatal hypoxia, migraine The third nerve originates from the oculomotor nucleus complex, which lies at the ventral border of the periaqueductal gray matter in the midbrain. The nerve fascicle passes ventrally through the medial longitudinal fasciculus, the tegmentum, the red nucleus, and the substantia nigra, and finally emerges from the cerebral peduncle to form the oculomotor nerve trunk, which lies between the superior cerebellar and posterior cerebral arteries. The nerve then passes through the subarachnoid space, running beneath the free edge of the tentorium. It continues lateral to the posterior communicating artery and below the temporal lobe uncus, where it runs over the petroclinoid ligament. It pierces the dura mater at the top of the clivus to enter the cavernous sinus. Within the cavernous sinus, the nerve runs along the lateral wall of the sinus together with the trochlear nerve and the ophthalmic (V1) and maxillary (V2) divisions of the trigeminal nerve. As it leaves the cavernous sinus, it divides into the superior and inferior divisions, which pass through the superior orbital fissure, and enters the orbit within the annulus of Zinn. Within the orbit, the smaller superior division runs lateral to the optic nerve and ophthalmic artery and supplies the superior rectus and levator palpebrae muscles. The larger inferior division supplies the medial rectus, inferior rectus, and inferior oblique muscles, as well as the iris sphincter and ciliary body.

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Conference papers on the topic "Superior orbital fissure"

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Kürüm, Ahmet Furkan, Hazan Başak, Süha Beton, Ayhan Cömert, and Cem Meço. "Endonasal endoscopic anatomy of superior orbital fissure: Anatomic study." In 95th Annual Meeting German Society of Oto-Rhino-Laryngology, Head and Neck Surgery e. V., Bonn. Georg Thieme Verlag KG, 2024. http://dx.doi.org/10.1055/s-0044-1785109.

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Zohdy, Youssef M., Ali Alawieh, Matthew Agam, et al. "Superior Orbital Fissure Narrowing and Tumor-Associated Pain in Spheno-orbital Meningiomas." In 33rd Annual Meeting North American Skull Base Society. Georg Thieme Verlag KG, 2024. http://dx.doi.org/10.1055/s-0044-1779886.

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Li, Lifeng, Nyall R. London, Daniel M. Prevedello, and Ricardo L. Carrau. "Expanded Exposure and Detailed Anatomic Analysis of the Superior Orbital Fissure and the Associated Neurovascular Structures." In 30th Annual Meeting North American Skull Base Society. Georg Thieme Verlag KG, 2020. http://dx.doi.org/10.1055/s-0040-1702620.

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Ruella, Mauro E., Krishnan Ravindran, Guilherme Gago, et al. "Surgical Anatomy of the Superior Orbital Fissure and Implications in Open and Endoscopic Skull Base Surgery." In 33rd Annual Meeting North American Skull Base Society. Georg Thieme Verlag KG, 2024. http://dx.doi.org/10.1055/s-0044-1780022.

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Rennert, Robert C., Michael T. Bounajem, Karol P. Budohoski, Michael Karsy, and William T. Couldwell. "Modified Lateral Orbitotomy Approach to Lesions of the Superior Orbital Fissure, Cavernous Sinus, and Mesial Temporal Lobe." In 31st Annual Meeting North American Skull Base Society. Georg Thieme Verlag KG, 2022. http://dx.doi.org/10.1055/s-0042-1744005.

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Barile, João Paulo, Amanda Freitas Alves, Fernanda Maria Gonçalves de Sousa Moura, Sonia Maria Cesar de Azevedo Silva, and Roberta Arb Saba Rodrigues Pinto. "Tolosa Hunt syndrome, case report." In XIV Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2023. http://dx.doi.org/10.5327/1516-3180.141s1.317.

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Introduction: Tolosa Hunt syndrome (THS) is a rare condition, incidence of 1/1.000.000 case per year, characterized by unilateral painful ophthalmoparesis caused by idiopathic inflammation in the cavernous sinus. The oculomotor nerve is most commonly involved (80%), followed by abducens nerve (70%), ophthalmic branch of trigeminal nerve (30%), trochlear nerve (29%). Case presentation: Male, 77 years old, admitted with an acute moderate-intensity orbitofrontal headache on the left, envolving with palpebral ptosis of the left eye. Neurological examination: complete palpebral ptosis on the left and ophthalmoplegia of the entire ipsilateral extrinsic ocular musculature. A complete investigation was carried out: metabolic, rheumatological, serological tests without significant alterations and study of the cerebrospinal fluid with mild hyperproteinorachia, without pleocytosis. Magnetic resonance imaging (MRI) of the skull showed thickening of the cavernous sinus on the left, with contrast enhancement; Angio-MRI of the Skull and Neck without alterations. Therefore, THS was diagnosed and treatment with Methylprednisolone 1 g for five days, with complete improvement of headache and partial improvement of ophthalmoparesis. The patient was discharged with 60 mg of prednisone orally with instructions for gradual weaning off, return to the neurology outpatient clinic. Discussion: THS diagnosis is based on the International Classification of Headache Disorders: unilateral periorbital headache; granulomatous inflammation of the cavernous sinus, superior orbital fissure or orbit on cranial MRI; paralysis of one or more of the oculomotor nerves; the headache must precede the ophthalmoparesis by up to two weeks or appear concomitantly. The exclusion of secondary causes is essential. Treatment of choice is cortico steroids, improvement of headache in the first days, and of ophthalmoparesis in 2–8 weeks. Conclusion: Unilateral headache with ipsilateral ophthalmoparesis should raise the suspicion of THS.

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Mortazavi, Armin, Ehsan Dowlati, Daniel R. Felbaum, and Samir Sur. "Combined Extradural/Intradural Trans-Sylvian Approach for Superior Orbital Fissure and Optic Nerve Decompression, Cavernous Sinus Biopsy, and Mesial Temporal Lesion Resection: An Unusual Case of a Tolosa-Hunt Variant with Intraparenchymal Extension." In 31st Annual Meeting North American Skull Base Society. Georg Thieme Verlag KG, 2022. http://dx.doi.org/10.1055/s-0042-1743864.

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