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Dissertations / Theses on the topic 'Synaptic time dependent plasticity'

Author: Grafiati

Published: 2 June 2025

Last updated: 31 July 2025

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1

Enge, Sören, Monika Fleischhauer, Anne Gärtner, et al. "Brain-Derived Neurotrophic Factor (Val66Met) and Serotonin Transporter (5-HTTLPR) Polymorphisms Modulate Plasticity in Inhibitory Control Performance Over Time but Independent of Inhibitory Control Training." Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2017. http://nbn-resolving.de/urn:nbn:de:bsz:14-qucosa-221795.

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Several studies reported training-induced improvements in executive function tasks and also observed transfer to untrained tasks. However, the results are mixed and there is a large interindividual variability within and across studies. Given that training-related performance changes would require modification, growth or differentiation at the cellular and synaptic level in the brain, research on critical moderators of brain plasticity potentially explaining such changes is needed. In the present study, a pre-post-follow-up design (N = 122) and a 3-weeks training of two response inhibition tas

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2

Enge, Sören, Monika Fleischhauer, Anne Gärtner, et al. "Brain-Derived Neurotrophic Factor (Val66Met) and Serotonin Transporter (5-HTTLPR) Polymorphisms Modulate Plasticity in Inhibitory Control Performance Over Time but Independent of Inhibitory Control Training." Frontiers Research Foundation, 2016. https://tud.qucosa.de/id/qucosa%3A30228.

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Several studies reported training-induced improvements in executive function tasks and also observed transfer to untrained tasks. However, the results are mixed and there is a large interindividual variability within and across studies. Given that training-related performance changes would require modification, growth or differentiation at the cellular and synaptic level in the brain, research on critical moderators of brain plasticity potentially explaining such changes is needed. In the present study, a pre-post-follow-up design (N = 122) and a 3-weeks training of two response inhibition tas

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3

Coquinco, Ainsley. "Activity dependent synaptic plasticity and microfluidics." Thesis, University of British Columbia, 2012. http://hdl.handle.net/2429/40663.

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The visual cortex of the brain is one of the fundamental preparations to study critical periods and activity dependent changes in the brain. During development, when sensory input from one eye is prevented, visual acuity and brain connectivity is lost in favour of inputs from the active eye. Because of the brain’s complexity, it is difficult to perform thorough analyses of synaptic mechanisms that exist during development. Therefore, the development of simpler in vitro models would be advantageous. In our studies, we used a 3-compartment microfluidic device and created a new model for dual inp

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4

Billings, Guy. "Memory stability and synaptic plasticity." Thesis, University of Edinburgh, 2009. http://hdl.handle.net/1842/3853.

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Numerous experiments have demonstrated that the activity of neurons can alter the strength of excitatory synapses. This synaptic plasticity is bidirectional and synapses can be strengthened (potentiation) or weakened (depression). Synaptic plasticity offers a mechanism that links the ongoing activity of the brain with persistent physical changes to its structure. For this reason it is widely believed that synaptic plasticity mediates learning and memory. The hypothesis that synapses store memories by modifying their strengths raises an important issue. There should be a balance between the nec

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5

Yeung, Luk Chong. "A mechanistic model of calcium-dependent synaptic plasticity /." View online version; access limited to Brown University users, 2005. http://wwwlib.umi.com/dissertations/fullcit/3174704.

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6

Arnold, F. J. L. "An experimental model of transcription-dependent synaptic plasticity." Thesis, University of Cambridge, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.596164.

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A cell culture system was developed to study synaptic plasticity. Electrical activity across cultured hippocampal networks was monitored using an array of 60 microelectrodes (MEA). These networks demonstrated spontaneous low rate asynchronous electrical activity. the GABA-A receptor antagonist bicuculline (50μM) transformed this activity into synchronous repetitive bursts of action potentials across the network, accompanied by NMDA receptor-dependent sustained global calcium transients. A 15 minute bicuculline exposure induced a long-lasting increase in synaptic efficacy, and a dramatic change

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7

Lee, Bo Young. "Signaling events in activity dependent neuroprotection, neurodegeneration, and synaptic plasticity." Columbus, Ohio : Ohio State University, 2007. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=osu1180458484.

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8

Monzon, Joshua Jen C. "Analog VLSI circuit design of spike-timing-dependent synaptic plasticity." Thesis, Massachusetts Institute of Technology, 2008. http://hdl.handle.net/1721.1/54636.

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Thesis (M. Eng.)--Massachusetts Institute of Technology, Dept. of Electrical Engineering and Computer Science, 2008.<br>Cataloged from PDF version of thesis.<br>Includes bibliographical references (p. 61-63).<br>Synaptic plasticity is the ability of a synaptic connection to change in strength and is believed to be the basis for learning and memory. Currently, two types of synaptic plasticity exist. First is the spike-timing-dependent-plasticity (STDP), a timing-based protocol that suggests that the efficacy of synaptic connections is modulated by the relative timing between presynaptic and pos

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9

Birtoli, Barbara. "Firing mode dependent synaptic plasticity in rat neocortical pyramidal neurons /." Bern : [s.n.], 2004. http://www.ub.unibe.ch/content/bibliotheken_sammlungen/sondersammlungen/dissen_bestellformular/index_ger.html.

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10

Heidari, Mohajerani Majid. "Activity-dependent synaptic plasticity processes in the immature rat hippocampus." Doctoral thesis, SISSA, 2007. http://hdl.handle.net/20.500.11767/4747.

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11

Prokin, Ilia. "Synaptic plasticity emerging from chemical reactions : Modeling spike-timing dependent plasticity of basal ganglia neurons." Thesis, Lyon, 2016. http://www.theses.fr/2016LYSEI115/document.

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Notre cerveau prend en charge différentes formes d’apprentissage dans ses diverses parties. C’est par exemple le cas des ganglions de la base, un ensemble de noyaux sous-corticaux qui est impliqué dans la sélection de l’action et une forme spécifique de l’apprentissage / mémoire, la mémoire procédurale (mémoire des compétences ou d’expertise). A l’échelle du neurone unique, le support le plus plausible de l’apprentissage et de la mémoire est la plasticité synaptique, le processus par lequel l’efficacité de la communication entre deux neurones change en réponse à un pattern spécifique de condit

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12

Sdrulla, Dan Alexandru. "Adenosine-dependent short- and long-term changes in hippocampal synaptic plasticity /." Connect to full text via ProQuest. IP filtered, 2005.

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Thesis (Ph.D. in Neuroscience) -- University of Colorado, 2005.<br>Typescript. Includes bibliographical references (leaves 96-111). Free to UCDHSC affiliates. Online version available via ProQuest Digital Dissertations;

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13

Appleby, Peter A. "A synaptic and temporal ensemble interpretation of spike-timing-dependent plasticity." Thesis, University of Southampton, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.427459.

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14

Su, Susan C. (Susan Chih-Chieh). "Regulation of synaptic function and plasticity by cyclin-dependent kinase 5." Thesis, Massachusetts Institute of Technology, 2012. http://hdl.handle.net/1721.1/79141.

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Thesis (Ph. D. in Neuroscience)--Massachusetts Institute of Technology, Dept. of Brain and Cognitive Sciences, February 2013.<br>This electronic version was submitted by the student author. The certified thesis is available in the Institute Archives and Special Collections.<br>Cataloged from student-submitted PDF version of thesis. "February 2013." Page 192 blank.<br>Includes bibliographical references.<br>The neuronal serine/threonine kinase cyclin-dependent kinase 5 (Cdk5) is activated by its regulatory subunit, p35, to post-translationally modify substrates through phosphorylation. In this

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15

Graupner, Michael. "Induction and Maintenance of Synaptic Plasticity." Doctoral thesis, Technische Universität Dresden, 2007. https://tud.qucosa.de/id/qucosa%3A23857.

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Synaptic long-term modifications following neuronal activation are believed to be at the origin of learning and long-term memory. Recent experiments suggest that these long-term synaptic changes are all-or-none switch-like events between discrete states of a single synapse. The biochemical network involving calcium/calmodulin-dependent protein kinase II (CaMKII) and its regulating protein signaling cascade has been hypothesized to durably maintain the synaptic state in form of a bistable switch. Furthermore, it has been shown experimentally that CaMKII and associated proteins such as protein k

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16

Schecter, Rachel W. "Structural mechanisms of experience-dependent synaptic plasticity in the mouse visual cortex." Thesis, Massachusetts Institute of Technology, 2016. http://hdl.handle.net/1721.1/106441.

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Thesis: Ph. D. in Neuroscience, Massachusetts Institute of Technology, Department of Brain and Cognitive Sciences, 2016.<br>Vita. Cataloged from PDF version of thesis.<br>Includes bibliographical references (pages 151-171).<br>Changes in the sensory experience of an animal shapes behavior through synaptic plasticity. Modification in the strength of synaptic drive can result from adjustments in the strength of existing synapses, creation of new synapses, or removal of existing ones and involves presynaptic, postsynaptic, and extra-synaptic mechanisms. Ocular dominance (OD) plasticity following

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17

Clopath, Claudia. "Synaptic plasticity across different time scales and its functional implications /." [S.l.] : [s.n.], 2009. http://library.epfl.ch/theses/?nr=4498.

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18

Xu, Pei. "Cdk5 activity is required for BDNF-stimulated neuronal survival and synaptic plasticity /." View abstract or full-text, 2008. http://library.ust.hk/cgi/db/thesis.pl?BICH%202008%20XU.

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19

Graupner, Michael. "Induction and Maintenance of Synaptic Plasticity." Doctoral thesis, Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2008. http://nbn-resolving.de/urn:nbn:de:bsz:14-ds-1221145787153-31869.

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Synaptic long-term modifications following neuronal activation are believed to be at the origin of learning and long-term memory. Recent experiments suggest that these long-term synaptic changes are all-or-none switch-like events between discrete states of a single synapse. The biochemical network involving calcium/calmodulin-dependent protein kinase II (CaMKII) and its regulating protein signaling cascade has been hypothesized to durably maintain the synaptic state in form of a bistable switch. Furthermore, it has been shown experimentally that CaMKII and associated proteins such as protein k

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20

Vogt, Simon Matthew [Verfasser]. "Synaptic plasticity and timing-dependent reinforcement in spiking neural networks / Simon Matthew Vogt." Lübeck : Zentrale Hochschulbibliothek Lübeck, 2017. http://d-nb.info/1142648060/34.

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21

Davies, Kurtis Daniel. "Protein phosphorylation : roles in subcellular localization and synaptic plasticity /." Connect to abstract via ProQuest. Full text is not available online, 2008.

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22

Chandrasekaran, Santosh. "Unbiased, High-Throughput Electron Microscopy Analysis of Experience-Dependent Synaptic Changes." Research Showcase @ CMU, 2015. http://repository.cmu.edu/dissertations/462.

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Neocortical circuits can adapt to changes in sensory input by modifying the strength or number of synapses. These changes have been well-characterized electrophysiologically in primary somatosensory (barrel) cortex of rodents across different ages and with different patterns of whisker stimulation. Previous work from our lab has identified layer-specific critical periods for synaptic potentiation after selective whisker experience (SWE), where all but one row of facial whiskers has been removed. Although whole-cell patch-clamp recording methods enable a mechanistic understanding of how synapti

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23

Waddington, Amelia. "Growing synfire chains with triphasic spike-time-dependent plasticity." Thesis, University of Leeds, 2011. http://etheses.whiterose.ac.uk/1758/.

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How collections of neurons combine into functional networks capable of intricate and accurate information processing is one of the biggest and most interesting challenges in neuroscience today. To approach this challenge, it is necessary to address the problem one structure at a time. In this thesis the focus is the development of synfire chains. Synfire chains are feed-forward neural structures which have long been suggested as a possible mechanism by which precisely timed sequences of neural activity could be generated. Precise spatiotemporal firing patterns are known to occur in the brains

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24

Xiao, Wei. "Class 5 semaphorins mediate synapse elimination and activity-dependent synaptic plasticity in hippocampal neurons." Thesis, University of British Columbia, 2017. http://hdl.handle.net/2429/60340.

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Although autism spectrum disorders (ASDs) have long been known to have a strong heritability, the genetic basis of these disorders has remained largely elusive. Hundreds of genes have been linked to ASDs, but most of them only contribute a small increase in risk. In 2009, a genome-wide association study identified Semaphorin 5A (SEMA5A) as a novel autism susceptibility gene. Sema5A is a member of the Semaphorin family consisting of secreted and membrane-associated proteins characterized by the Sema domain. Although initially identified as axon guidance cues, Semaphorins have been found to play

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25

Albers, Christian [Verfasser], Klaus [Akademischer Betreuer] Pawelzik, and Stefan [Akademischer Betreuer] Bornholdt. "Functional Implications of Synaptic Spike Timing Dependent Plasticity and Anti-Hebbian Membrane Potential Dependent Plasticity / Christian Albers. Gutachter: Klaus Pawelzik ; Stefan Bornholdt. Betreuer: Klaus Pawelzik." Bremen : Staats- und Universitätsbibliothek Bremen, 2015. http://d-nb.info/107560947X/34.

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26

Mensching, Daniel [Verfasser], and Dietmar [Akademischer Betreuer] Kuhl. "Investigation of activity dependent Arl5b mediated signaling in synaptic plasticity / Daniel Mensching. Betreuer: Dietmar Kuhl." Hamburg : Staats- und Universitätsbibliothek Hamburg, 2016. http://d-nb.info/1103233211/34.

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27

Li, Rui. "Chemical and stimulus-induced NMDA-dependent synaptic plasticity in hippocampus and the possible involved mechanisms /." Göteborg : Dept. of Medical Biophysics, Institute of Neuroscience and Physiology, Göteborg University, 2006. http://hdl.handle.net/2077/740.

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28

Hauser, Florian [Verfasser]. "Formation and stability of spiking cell assemblies with spike-timing-dependent synaptic plasticity / Florian Hauser." Ulm : Universität Ulm. Fakultät für Ingenieurwissenschaften und Informatik, 2012. http://d-nb.info/1028567995/34.

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29

Iglesias, Javier. "Emergence of oriented circuits driven by synaptic pruning associated with spike-timing-dependent plasticity (STDP)." Université Joseph Fourier (Grenoble), 2005. https://tel.archives-ouvertes.fr/tel-00010650.

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L'élagage massif des synapses après une croissance excessive est une phase normale de la maturation du cerveau des mammifères. L'élagage commence peu avant la naissance et est complété avant l'âge de la maturité sexuelle. Les facteurs déclenchants capables d'induire l'élagage des synapses pourraient être liés à des processus dynamiques qui dépendent de la temporalité relative des potentiels d'actions. La plasticité synaptique à modulation temporelle relative STDP correspond à un changement de la force synaptique basé sur l'ordre des décharges pré- et post-synaptiques. La relation entre l'effic

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30

Mensching, Daniel Verfasser], and Dietmar [Akademischer Betreuer] [Kuhl. "Investigation of activity dependent Arl5b mediated signaling in synaptic plasticity / Daniel Mensching. Betreuer: Dietmar Kuhl." Hamburg : Staats- und Universitätsbibliothek Hamburg, 2016. http://d-nb.info/1103233211/34.

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31

Pineda, Victor Viray. "A genetic and pharmacological dissection of synaptic plasticity in the hippocampus /." Thesis, Connect to this title online; UW restricted, 2003. http://hdl.handle.net/1773/6290.

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32

Dylda, Evelyn. "Neuronal circuits of experience-dependent plasticity in the primary visual cortex." Thesis, University of Edinburgh, 2018. http://hdl.handle.net/1842/31234.

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Our ability to learn relies on the potential of neuronal networks to change through experience. The primary visual cortex (V1) has become a popular system for studying how experience shapes cortical neuronal networks. Experience-dependent plasticity in V1 has been extensively studied in young animals, revealing that experiences in early postnatal life substantially shape neuronal activity in the developing cortex. In contrast, less is known about how experiences modify the representation of visual stimuli in the adult brain. In addition, adult experience-dependent plasticity remains largely un

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33

Xing, Xiaomin. "Genetic and functional analysis of synaptic CA²⁺ dynamics in Drosophila." Diss., University of Iowa, 2014. https://ir.uiowa.edu/etd/2168.

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Ca²⁺ influx is one of the critical events that trigger synaptic vesicular release, and the accumulation of residual free Ca²⁺ in synapses is also important for activity-dependent synaptic plasticity. Ca²⁺ imaging with fluorescence indicators (synthetic or genetically encoded) is a powerful approach to monitor Ca²⁺ levels in neurons and synapses. Although accumulating studies in vertebrate systems have been carried out to demonstrate the role of Ca²⁺ in synaptic transmission and plasticity, most of these studies rely on pha

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34

RUBEN, MASSIMO. "Coordination in space and time of excitatory and inhibitory synaptic plasticity at dendritic synapses." Doctoral thesis, Università degli studi di Genova, 2021. http://hdl.handle.net/11567/1044902.

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For years, GABAergic synapses were considered poorly plastic. However, an increasing body of evidence demonstrated that, similarly to excitatory synapses, inhibitory synapses formed by interneurons onto pyramidal cells can be modulated in response to neuronal activity. The spiking output of a neuron is determined by the opposite but concomitant action of excitation and inhibition. In this view, it is crucial to understand how plasticity at excitatory and inhibitory synapses is coordinated. In this thesis, I will describe two different types of plasticity interplay between excitation and inhibi

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35

CARRANO, NICOLO'. "LINKING NMDA RECEPTOR-DEPENDENT PLASTICITY AND NEURONAL ARCHITECTURE: THE ROLE OF RING FINGER PROTEIN 10." Doctoral thesis, Università degli Studi di Milano, 2019. http://hdl.handle.net/2434/694271.

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An active synapse-to-nucleus communication is essential for long-term changes in neurons, like the regulation of neuronal plasticity and shaping neuronal morphology. Next to the fast electrochemical signaling, neurons employ a slower mechanism that involves a recently discovered class of proteins, the synaptonuclear messengers. Different studies showed the pivotal role of synaptonuclear messengers in the modulation of synaptic transmission at excitatory synapses. On the other hand, alterations of synaptonuclear messengers’ activity have been correlated to synaptic failure as observed in differ

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36

Leutgeb, Jill K. "Synaptic plasticity in mature cultured hippocampal entorhinal cortex slices: activity-dependent regulation of cAMP response element binding protein." [S.l.] : [s.n.], 2004. http://deposit.ddb.de/cgi-bin/dokserv?idn=972484655.

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37

Kwag, Jeehyun. "Synaptic control of spike timing and spike timing-dependent plasticity during theta frequency oscillation in hippocampal CA1 pyramidal neurons." Thesis, University of Oxford, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.487275.

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Spike timing during oscillation has been suggested to play an important role in hippocampal processing. However, how the hippocampal network and the individual neurons interact to precisely control spike timing when they receive synaptic inputs from two major excitatory input pathways - Schaffer collateral and perforant path - during natural network oscillation is yet unknown. Investigation of spike timing control mechanism would shed light on how the local STDP learning rule could be influenced by different cortical inputs during theta oscillation. Here I used whole-cell path-clamp recording

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38

Schwartz, Neil. "Regulation of the functional development of vision, through an interaction between synaptic changes and experience- dependent regulation of plasticity." Thesis, McGill University, 2010. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=94904.

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Early in development the visual circuitry in the brain is highly plastic. Factors that contribute to this plasticity and its expression are synaptic activity and the proteins expressed in the cell. As the system matures this plasticity is expressed and the system changes such that it becomes better at resolving finer details of the environment. Synaptic input from the retina participate in the initiation, progression, and once established the maintenance of visual function. Thus synaptic drive is key to the normal development of vision. However, the exact relationship between synaptic ac

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39

Kemp, Anne [Verfasser], Denise [Akademischer Betreuer] Manahan-Vaughan, and Ulf [Akademischer Betreuer] Eysel. "Functional role of synaptic plasticity in hippocampus-dependent learning and memory / Anne Kemp. Gutachter: Denise Manahan-Vaughan ; Ulf Eysel." Bochum : Ruhr-Universität Bochum, 2015. http://d-nb.info/1079843345/34.

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40

Song, Fei. "Modelling time-dependent plastic behaviour of geomaterials." Doctoral thesis, Universitat Politècnica de Catalunya, 2021. http://hdl.handle.net/10803/672273.

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Representing the time-dependent plastic behaviour of geomaterials is a critical issue in the correct application of tunnelling design techniques such as the convergence-confinement method or numerical modelling. Furthermore, during underground excavations below the water table, the effect of seepage flow cannot be ignored, and the behaviour of the tunnel must be analysed in a coupled hydro-mechanical framework. The main objective of this thesis is to analyse the response of tunnels excavated in saturated time-dependent plastic rock masses. For this purpose, a time-dependent plastic constituti

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41

Brink, Stephen Isaac. "Learning in silicon: a floating-gate based, biophysically inspired, neuromorphic hardware system with synaptic plasticity." Diss., Georgia Institute of Technology, 2012. http://hdl.handle.net/1853/50143.

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The goal of neuromorphic engineering is to create electronic systems that model the behavior of biological neural systems. Neuromorphic systems can leverage a combination of analog and digital circuit design techniques to enable computational modeling, with orders of magnitude of reduction in size, weight, and power consumption compared to the traditional modeling approach based upon numerical integration. These benefits of neuromorphic modeling have the potential to facilitate neural modeling in resource-constrained research environments. Moreover, they will make it practical to use neural co

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42

Kläschen, Johanne [Verfasser], and Dietmar [Akademischer Betreuer] Kuhl. "The role of Arc/Arg3.1 in protein synthesis dependent and independent forms of synaptic plasticity / Johanne Kläschen. Betreuer: Dietmar Kuhl." Hamburg : Staats- und Universitätsbibliothek Hamburg, 2016. http://d-nb.info/1099594901/34.

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Kläschen, Johanne Verfasser], and Dietmar [Akademischer Betreuer] [Kuhl. "The role of Arc/Arg3.1 in protein synthesis dependent and independent forms of synaptic plasticity / Johanne Kläschen. Betreuer: Dietmar Kuhl." Hamburg : Staats- und Universitätsbibliothek Hamburg, 2016. http://d-nb.info/1099594901/34.

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Reijak, Viviane [Verfasser], Denise [Akademischer Betreuer] Manahan-Vaughan, and Katrin [Akademischer Betreuer] Marcus. "Investigation of strategies to ameliorate beta-amyloid mediated deficits in hippocampus-dependent synaptic plasticity / Viviane Reijak. Gutachter: Denise Manahan-Vaughan ; Katrin Marcus." Bochum : Ruhr-Universität Bochum, 2016. http://d-nb.info/1095884417/34.

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Nielsen, Mark David. "Regulation of two subfamilies of adenylyl cyclase by Gi-coupled receptors : a possible role during cAMP-dependent synaptic plasticity in the Hippocampus /." Thesis, Connect to this title online; UW restricted, 1997. http://hdl.handle.net/1773/6247.

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46

Lilly, Scott Matthew. "Protein Kinase A Alterations Following Chronic Flurazepam Treatment: Implications for Inhibitory and Excitatory Synaptic Plasticity in Rat Hippocampal CA1." Connect to full-text via OhioLINK ETD Center, 2006. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=mco1145293063.

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Thesis (Ph.D.)--Medical University of Ohio, 2006.<br>"In partial fulfillment of the requirements for the degree of Doctor of Philosophy in Medical Sciences." Major advisor: Elizabeth I. Tietz. Includes abstract. Document formatted into pages: iv, 234 p. Title from title page of PDF document. Bibliography: pages 86-95,126-135,167-174,190-232.

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47

Mohammad, Sameh. "Long-term depression in the rat hippocampus as a memory model : Interrogating the role of protein synthesis in NMDA- and mGluR-dependent synaptic plasticity." Thesis, Högskolan i Skövde, Institutionen för vård och natur, 2010. http://urn.kb.se/resolve?urn=urn:nbn:se:his:diva-4715.

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Long-term potentiation (LTP) and depression (LTD) are important forms of activity-dependent synaptic plasticity believed to play a role in memory at the cellular level. It has previously been described that synthesis of new proteins is needed to maintain LTP longer than a few hours. Other reports argue that sufficient proteins for stable LTP are already available. The present study aims to examine the role of protein synthesis in LTD, the presumed mirror mechanism of LTP. Experiments were carried out in hippocampal slices from young (12-45 days) and old (12-18 weeks) Sprague-Dawley rats. Extra

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48

Du, Nan, Mahdi Kiani, Christian G. Mayr, et al. "Single pairing spike-timing dependent plasticity in BiFeO3 memristors with a time window of 25ms to 125µs." Universitätsbibliothek Chemnitz, 2015. http://nbn-resolving.de/urn:nbn:de:bsz:ch1-qucosa-171458.

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Memristive devices are popular among neuromorphic engineers for their ability to emulate forms of spike-driven synaptic plasticity by applying specific voltage and current waveforms at their two terminals. In this paper, we investigate spike-timing dependent plasticity (STDP) with a single pairing of one presynaptic voltage spike and one postsynaptic voltage spike in a BiFeO3 memristive device. In most memristive materials the learning window is primarily a function of the material characteristics and not of the applied waveform. In contrast, we show that the analog resistive switching of the

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49

Carnell, Andrew Robert. "Aspects of learning within networks of spiking neurons." Thesis, University of Bath, 2008. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.512264.

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Spiking neural networks have, in recent years, become a popular tool for investigating the properties and computational performance of large massively connected networks of neurons. Equally as interesting is the investigation of the potential computational power of individual spiking neurons. An overview is provided of current and relevant research into the Liquid Sate Machine, biologically inspired artificial STDP learning mechanisms and the investigation of aspects of the computational power of artificial, recurrent networks of spiking neurons. First, it is shown that, using simple structure

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50

James, Andy Robert. "The use of pH-sensitive flourescent proteins to monitor real-time AMPA receptor trafficking : a potential tool for the visualisation of synaptic plasticity in the cerebellum." Thesis, University College London (University of London), 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.428011.

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