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Books on the topic 'Syncope – Diagnostic'

Author: Grafiati

Published: 4 June 2021

Last updated: 16 February 2022

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1

Fits and faints. Mac Keith Press, 1990.

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2

Stephenson, John B. P. Fits and faints. MacKeith Press, 1990.

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3

Benditt, David G. The evaluation and treatment of syncope: A handbook for clinical practice. 2nd ed. Blackwell Pub., 2006.

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4

Disorders of mental status: Dementia, encephalopathy, coma, syncope. Butterworth-Heinemann, 1998.

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5

Katritsis, Demosthenes G., Bernard J. Gersh, and A. John Camm. Syncope. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199685288.003.1517_update_004.

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6

Anitescu, Magdalena, and Chirag Shah. The Vasovagal Reflex and Neuraxial Techniques. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190271787.003.0042.

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Syncope, or the transient loss of consciousness, is one of the leading causes of emergency department visits. Syncope can be neurally mediated, orthostatic, cardiac, or cerebrovascular. Neurally mediated vasovagal syncope is the most frequent form. Diagnostic modalities are tilt- table testing and implantable loop recorders. Therapeutic options usually begin with supportive measures, such as a fluid bolus or changing patient positioning, but complex cases may require vasoactive agents or placement of a pacemaker. In many situations patients who present to the operating room for various surgeri

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7

Delamont, Shane. Syncope. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199688395.003.0029.

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This chapter details the epidemiology, clinical spectrum, and major causes of syncope. There is a conceptual framework of the physiology behind syncope and a discussion about cerebral blood flow. Particular attention is given to understanding neurocardiogenic syncope, which is the commonest cause. It looks at the latest physiological understanding of syncope and the importance of clinical context which enables risk stratification and facilitates diagnosis of the causes and hence management of syncope. National Institute of Clinical Excellence (NICE) guidelines, their effective use are emphasiz

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8

Roberto, García-Civera, ed. Syncope cases. Blackwell Pub., 2006.

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9

Smith, Jacky. Cough. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0016.

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A cough is an explosive forced expiratory manoeuvre, usually against a closed glottis, and gives rise to a characteristic sound. Acute cough is defined as a cough of less than 3 weeks duration, and chronic cough as one of more than 8 weeks duration. Acute cough is the commonest presenting symptom in primary care: by far the most frequent cause is a viral respiratory tract infection. The main effect of coughing is on quality of life and this is particularly prominent in patients with chronic cough, as these frequently develop physical complications such as chest pain, retching and vomiting, hoa

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10

Benditt, David G., Michele Brignole, Jean-Jacques Blanc, Jean-Jacques Blanc, and Richard Sutton. Evaluation and Treatment of Syncope: A Handbook for Clinical Practice. Wiley & Sons, Incorporated, John, 2009.

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11

Benditt, David G., Michele Brignole, Jean-Jacques Blanc, and Richard Sutton. Evaluation and Treatment of Syncope: A Handbook for Clinical Practice. Wiley & Sons, Incorporated, John, 2008.

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12

European Society of Cardiology Staff (Contributor) and David G. Benditt (Editor), eds. The Evaluation and Treatment of Syncope. Blackwell Publishing, Incorporated, 2003.

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13

G, Benditt David, ed. Syncope and transient loss of consciousness: Multidisciplinary management. Blackwell Futura, 2007.

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14

(Editor), David G. Benditt, Michele Brignole (Editor), Antonio Raviele (Editor), and Wouter Wieling (Editor), eds. Syncope and Transient Loss of Consciousness: Multidisciplinary Management. Wiley-Blackwell, 2007.

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15

Raviele, Antonio, David G. Benditt, Michele Brignole, and Wouter Wieling. Syncope and Transient Loss of Consciousness: Multidisciplinary Management. Wiley & Sons, Incorporated, John, 2008.

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16

Cohen, Jeffrey A., Justin J. Mowchun, Victoria H. Lawson, and Nathaniel M. Robbins. A 44-Year-Old Male with Subacute Onset of Syncope. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190491901.003.0032.

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Syncope in a patient with orthostatic hypotension (OH) may indicate autonomic dysfunction. The definition of OH is presented. Clinical features of parasympathetic and sympathetic function are discussed. The differential of acute autonomic dysfunction includes a number of conditions. An autoimmune etiology may occur autoimmune autonomic ganglionopathy. Serologic testing can assist in this diagnosis. If autoimmune immune modulating therapies may be indicated. Autonomic neuropathy may be a paraneoplastuc syndrome. Autonomic testing can also help with documenting autonomic neuropathy as well as th

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17

M, Bertrand, and King Spencer B. 1937-, eds. Acute coronary syndromes: A handbook for clinical practice. Blackwell Futura, 2006.

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18

Schwartz, Peter J., and Lia Crotti. Monogenic and oligogenic cardiovascular diseases: genetics of arrhythmias—catecholaminergic polymorphic ventricular tachycardia. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0152.

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Catecholaminergic polymorphic ventricular tachycardia (CPVT) is a rare inherited disorder associated with syncope and sudden death manifesting in the young during sympathetic activation. The electrocardiogram is normal and the heart is structurally normal. The diagnosis is usually made with an exercise stress test that shows a typical pattern of onset and offset of adrenergically induced ventricular arrhythmias. Molecular screening of RyR2, the major CPVT gene, is recommended whenever the suspicion of CPVT is high. If a disease-causing mutation is identified, cascade screening allows pre-sympt

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19

Chadwick, David. Seizures, epilepsy, and other episodic disorders in adults. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569381.003.0709.

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Epilepsy, or more correctly a seizure, is most easily defined in physiological terms, being ‘the name for occasional sudden, excessive, rapid, and local discharges of grey matter’ (Jackson 1873). It is more difficult to offer a comprehensive clinical definition of epileptic seizures and epilepsy because of the varied clinical manifestations produced by cerebral neuronal discharge. However, an epileptic seizure can be defined as an intermittent and stereotyped disturbance of consciousness, behaviour, emotion, motor function, or sensation that on clinical grounds is believed to result from corti

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20

Elliott, Perry, and Alexandros Protonotarios. Arrhythmogenic right ventricular cardiomyopathy: management of symptoms and prevention of sudden cardiac death. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0361.

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Patients with arrhythmogenic right ventricular cardiomyopathy (ARVC) have arrhythmia-related symptoms or are identified during screening of an affected family. Heart failure symptoms occur late in the disease’s natural history. As strenuous exercise has been associated with disease acceleration and worsening of ventricular arrhythmias, lifestyle modification with restricted athletic activities is recommended upon disease diagnosis or even identification of mutation carrier status. An episode of an haemodynamically unstable, sustained ventricular tachycardia or ventricular fibrillation as well

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21

Torbicki, Adam, Marcin Kurzyna, and Stavros Konstantinides. Pulmonary embolism. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0066.

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Pulmonary embolism is usually a consequence of deep vein thrombosis, and together the two conditions are known as venous thromboembolism. Non-thromboembolic causes of pulmonary embolism are rare. Pulmonary thromboembolism is a potentially life-threatening disease, if left untreated. This is due to a natural tendency towards early recurrence of pulmonary emboli which may lead to fatal right ventricular failure. In more severe cases, secondary right ventricular failure may result from myocardial ischaemia and injury caused by systemic hypotension and adrenergic overstimulation. Clinical presenta

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22

Torbicki, Adam, Marcin Kurzyna, and Stavros Konstantinides. Pulmonary embolism. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199687039.003.0066_update_001.

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Pulmonary embolism is usually a consequence of deep vein thrombosis, and together the two conditions are known as venous thromboembolism. Non-thromboembolic causes of pulmonary embolism are rare. Pulmonary thromboembolism is a potentially life-threatening disease, if left untreated. This is due to a natural tendency towards early recurrence of pulmonary emboli which may lead to fatal right ventricular failure. In more severe cases, secondary right ventricular failure may result from myocardial ischaemia and injury caused by systemic hypotension and adrenergic overstimulation. Clinical presenta

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23

Torbicki, Adam, Marcin Kurzyna, and Stavros Konstantinides. Pulmonary embolism. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199687039.003.0066_update_002.

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Abstract:

Pulmonary embolism is usually a consequence of deep vein thrombosis, and together the two conditions are known as venous thromboembolism. Non-thromboembolic causes of pulmonary embolism are rare. Pulmonary thromboembolism is a potentially life-threatening disease, if left untreated. This is due to a natural tendency towards early recurrence of pulmonary emboli which may lead to fatal right ventricular failure. In more severe cases, secondary right ventricular failure may result from myocardial ischaemia and injury caused by systemic hypotension and adrenergic overstimulation. Clinical presenta

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24

Torbicki, Adam, Marcin Kurzyna, and Stavros Konstantinides. Pulmonary embolism. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199687039.003.0066_update_003.

Full text

Abstract:

Pulmonary embolism is usually a consequence of deep vein thrombosis, and together the two conditions are known as venous thromboembolism. Non-thromboembolic causes of pulmonary embolism are rare. Pulmonary thromboembolism is a potentially life-threatening disease, if left untreated. This is due to a natural tendency towards early recurrence of pulmonary emboli which may lead to fatal right ventricular failure. In more severe cases, secondary right ventricular failure may result from myocardial ischaemia and injury caused by systemic hypotension and adrenergic overstimulation. Clinical presenta

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25

Hakim, Alan J., and Rodney Grahame. Hypermobility syndromes. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0159.

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Hypermobility-related syndromes constitute a family of heritable disorders of connective tissue (HDCT) that derive from abnormalities affecting genes that encode for the connective tissue matrix proteins such as collagen, fibrillin, and tenascin. They range from such commonplace though poorly recognized conditions such as the joint hypermobility syndrome (JHS) to the better-known, if more rare, eponymous syndromes such as Marfan's syndrome (MFS) and the different types of the Ehlers-Danlos syndrome (EDS). The more common presentations are with skin pathology (bruising, scaring), joint or spina

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