Relevant bibliographies by topics / Syndecans

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Academic literature on the topic 'Syndecans'

Author: Grafiati
Published: 4 June 2021
Last updated: 30 January 2023

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Journal articles on the topic "Syndecans"

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CAREY, David J. "Syndecans: multifunctional cell-surface co-receptors." Biochemical Journal 327, no. 1 (1997): 1–16. http://dx.doi.org/10.1042/bj3270001.

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This review will summarize our current state of knowledge of the structure, biochemical properties and functions of syndecans, a family of transmembrane heparan sulphate proteoglycans. Syndecans bind a variety of extracellular ligands via their covalently attached heparan sulphate chains. Syndecans have been proposed to play a role in a variety of cellular functions, including cell proliferation and cell–matrix and cell–cell adhesion. Syndecan expression is highly regulated and is cell-type- and developmental-stage-specific. The main function of syndecans appears to be to modulate the ligand-d
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Kim, C. W., O. A. Goldberger, R. L. Gallo, and M. Bernfield. "Members of the syndecan family of heparan sulfate proteoglycans are expressed in distinct cell-, tissue-, and development-specific patterns." Molecular Biology of the Cell 5, no. 7 (1994): 797–805. http://dx.doi.org/10.1091/mbc.5.7.797.

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The syndecans are a gene family of four transmembrane heparan sulfate proteoglycans that bind, via their HS chains, diverse components of the cellular microenvironment. To evaluate the expression of the individual syndecans, we prepared cDNA probes to compare mRNA levels in various adult mouse tissues and cultured mouse cells representing various epithelial, fibroblastic, endothelial, and neural cell types and B cells at various stages of differentiation. We also prepared antibody probes to assess whether the extracellular domains of the individual syndecans are shed into the conditioned media
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Mitsou, Ioli, Hinke A. B. Multhaupt, and John R. Couchman. "Proteoglycans, ion channels and cell–matrix adhesion." Biochemical Journal 474, no. 12 (2017): 1965–79. http://dx.doi.org/10.1042/bcj20160747.

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Cell surface proteoglycans comprise a transmembrane or membrane-associated core protein to which one or more glycosaminoglycan chains are covalently attached. They are ubiquitous receptors on nearly all animal cell surfaces. In mammals, the cell surface proteoglycans include the six glypicans, CD44, NG2 (CSPG4), neuropilin-1 and four syndecans. A single syndecan is present in invertebrates such as nematodes and insects. Uniquely, syndecans are receptors for many classes of proteins that can bind to the heparan sulphate chains present on syndecan core proteins. These range from cytokines, chemo
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Betriu, Nausika, Juan Bertran-Mas, Anna Andreeva, and Carlos E. Semino. "Syndecans and Pancreatic Ductal Adenocarcinoma." Biomolecules 11, no. 3 (2021): 349. http://dx.doi.org/10.3390/biom11030349.

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Pancreatic Ductal Adenocarcinoma (PDAC) is a fatal disease with poor prognosis because patients rarely express symptoms in initial stages, which prevents early detection and diagnosis. Syndecans, a subfamily of proteoglycans, are involved in many physiological processes including cell proliferation, adhesion, and migration. Syndecans are physiologically found in many cell types and their interactions with other macromolecules enhance many pathways. In particular, extracellular matrix components, growth factors, and integrins collect the majority of syndecans associations acting as biochemical,
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Hudák, Anett, Annamária Letoha, László Szilák, and Tamás Letoha. "Contribution of Syndecans to the Cellular Entry of SARS-CoV-2." International Journal of Molecular Sciences 22, no. 10 (2021): 5336. http://dx.doi.org/10.3390/ijms22105336.

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The severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) is a novel emerging pathogen causing an unprecedented pandemic in 21st century medicine. Due to the significant health and economic burden of the current SARS-CoV-2 outbreak, there is a huge unmet medical need for novel interventions effectively blocking SARS-CoV-2 infection. Unknown details of SARS-CoV-2 cellular biology hamper the development of potent and highly specific SARS-CoV-2 therapeutics. Angiotensin-converting enzyme-2 (ACE2) has been reported to be the primary receptor for SARS-CoV-2 cellular entry. However, emerging
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De Rossi, Giulia, and James R. Whiteford. "Syndecans in angiogenesis and endothelial cell biology." Biochemical Society Transactions 42, no. 6 (2014): 1643–46. http://dx.doi.org/10.1042/bst20140232.

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Syndecans are multifunctional heparan sulfate proteoglycans (HSPGs) with roles in cell adhesion, migration, receptor trafficking and growth-factor interactions and signalling. Studies using syndecan null animals have revealed limited roles for syndecans during development; however, under conditions of challenge or insult, several phenotypes have emerged. Angiogenesis is an important process both in development and in wound healing, but also in pathologies such as cancer and chronic inflammatory conditions. In the present paper, we summarize the main studies elucidating the role of syndecans in
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Hudák, Anett, Katalin Jósvay, Ildikó Domonkos, Annamária Letoha, László Szilák, and Tamás Letoha. "The Interplay of Apoes with Syndecans in Influencing Key Cellular Events of Amyloid Pathology." International Journal of Molecular Sciences 22, no. 13 (2021): 7070. http://dx.doi.org/10.3390/ijms22137070.

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Apolipoprotein E (ApoE) isoforms exert intricate effects on cellular physiology beyond lipid transport and metabolism. ApoEs influence the onset of Alzheimer’s disease (AD) in an isoform-dependent manner: ApoE4 increases AD risk, while ApoE2 decreases it. Previously we demonstrated that syndecans, a transmembrane proteoglycan family with increased expression in AD, trigger the aggregation and modulate the cellular uptake of amyloid beta (Aβ). Utilizing our previously established syndecan-overexpressing cellular assays, we now explore how the interplay of ApoEs with syndecans contributes to key
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Finsen, Alexandra Vanessa, Per Reidar Woldbaek, Jian Li, et al. "Increased syndecan expression following myocardial infarction indicates a role in cardiac remodeling." Physiological Genomics 16, no. 3 (2004): 301–8. http://dx.doi.org/10.1152/physiolgenomics.00144.2002.

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Finsen, Alexandra Vanessa, Per Reidar Woldbaek, Jian Li, Jiaping Wu, Torstein Lyberg, Theis Tonnessen, and Geir Christensen. Increased syndecan expression following myocardial infarction indicates a role in cardiac remodeling. Physiol Genomics 16: 301-308, 2004. First published November 18, 2003; 10.1152/physi-olgenomics. 00144.2002.—The purpose of this study was to identify essential genes involved in myocardial growth and remodeling following myocardial infarction (MI). Left ventricular noninfarcted tissues from six mice subjected to MI under general anesthesia and from six sham-operated mic
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Termini, Christina, Michelle Li, Joyce Kim, Liman Zhao, and John P. Chute. "Syndecan-2 Surface Expression Identifies Hematopoietic Stem Cells with Increased Repopulating Capacity." Blood 132, Supplement 1 (2018): 1273. http://dx.doi.org/10.1182/blood-2018-99-110701.

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Abstract Syndecans are transmembrane glycoproteins, which can regulate cell proliferation, growth, and adhesion through interactions with neighboring proteins within the plasma membrane or at the cytoplasmic interface. Although syndecans have been described to regulate aberrant signaling in hematological malignances, the role of syndecans in regulating normal hematopoietic stem cell (HSC) proliferation, differentiation, and self-renewal is largely unknown. We demonstrate that syndecan-1 and syndecan-3 are expressed on the surface of < 10% of murine hematopoietic stem and progenitor cells, w
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Gopal, Sandeep, Pernille Søgaard, Hinke A. B. Multhaupt, et al. "Transmembrane proteoglycans control stretch-activated channels to set cytosolic calcium levels." Journal of Cell Biology 210, no. 7 (2015): 1199–211. http://dx.doi.org/10.1083/jcb.201501060.

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Transmembrane heparan sulfate proteoglycans regulate multiple aspects of cell behavior, but the molecular basis of their signaling is unresolved. The major family of transmembrane proteoglycans is the syndecans, present in virtually all nucleated cells, but with mostly unknown functions. Here, we show that syndecans regulate transient receptor potential canonical (TRPCs) channels to control cytosolic calcium equilibria and consequent cell behavior. In fibroblasts, ligand interactions with heparan sulfate of syndecan-4 recruit cytoplasmic protein kinase C to target serine714 of TRPC7 with subse
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Dissertations / Theses on the topic "Syndecans"

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Castro, Cruz Monica del Carmen. "The impact of the syndecan-PDZ interactome on endosomal trafficking and extracellular vesicle composition." Thesis, Aix-Marseille, 2018. http://www.theses.fr/2018AIXM0302.

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Les syndécans forment une famille de quatre protéines transmembranaires qui sont substituées par l'héparane sulfate. Grâce à ces chaînes glucidiques extracellulaires, les syndécans contrôlent la signalisation d'une pléthore de facteurs de croissance et de molécules d'adhésion. Une autre caractéristique remarquable des syndécans est la conservation de leur domaine intracellulaire au cours de l'évolution. Ce domaine contient un motif C-terminal qui peut induire une interaction avec les protéines dites «PDZ». Les interactions PDZ sont promiscues et les protéines PDZ contrôlent divers aspects de l
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Alexopoulou, Annika. "The role of syndecans in murine embryonic stem cells and their differentiation into mesodermal cell types." Thesis, Imperial College London, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.438968.

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Zong, Fang. "Studies on syndecan-1 in mesenchymal tumors." Stockholm : Department of Laboratory Medicine, Karolinska Institutet, 2010. http://diss.kib.ki.se/2010/978-91-7409-749-8/.

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Rossi, Piva Maria Bethania [Verfasser]. "The impact and underlying molecular mechanisms of cell adhesion molecules integrins and syndecans in the cisplatin chemoresistance of melanoma cells / Maria Bethania Rossi Piva." Bonn : Universitäts- und Landesbibliothek Bonn, 2019. http://d-nb.info/1204479763/34.

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Eustace, Andrew David. "Syndecan 3 and inflammation." Thesis, University of Bristol, 2016. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.720843.

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Carulli, Sonia. "Molecular basis of syndecan-1 mediated cell adhesion to laminin 332." Thesis, Lyon 1, 2011. http://www.theses.fr/2011LYO10134.

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L’interaction du récepteur syndecan-1 de la famille des héparanes sulfates protéoglycanes avec le fragment carboxy-terminal alpha3LG4/5 de la protéine d’adhérence matricielle, la laminine 332, induit une réorganisation du cytosquelette de la cellule conduisant à la formation de filopodes et de microspicules, caractéristiques de la migration cellulaire. Notre laboratoire a mis en évidence que l’adhésion cellulaire syndecan-1 dépendante implique les chaînes d’héparanes sulfates et chondroïtine sulfate. Afin d’identifier le (les) zone(s) impliquée(s) dans l’interaction domaine LG4/5-syndécan-1, u
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Hidding, Heriburg [Verfasser], and Michael J. [Akademischer Betreuer] Raschke. "Die Bedeutung der Proteoglykane Syndecan-1 und Syndecan-4 während der Frakturheilung / Heriburg Hidding ; Betreuer: Michael J. Raschke." Münster : Universitäts- und Landesbibliothek Münster, 2013. http://d-nb.info/1138280402/34.

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Habes, Chahrazed. "Stimulation du signal calcique et de la migration des cellules cancéreuses mammaires par le peptide LL-37 : un mécanisme d’attachement membranaire impliquant les glycosaminoglycanes et les syndécanes." Thesis, Tours, 2019. http://www.theses.fr/2019TOUR3807.

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Le peptide LL-37, peptide antimicrobien de l’immunité innée, est associé au développement tumoral. Sur des lignées cancéreuses mammaires. Il augmente le calcium intracellulaire et la migration. via l’activation de la voie PI3K/AKT et de canaux calciques. L’énantiomère (D)-LL-37 induit des effets similaires, excluant une interaction classique ligand-récepteur. Avec sa structure en hélice α amphipathique et sa charge nette +6, l’hypothèse était que le peptide utilise les charges négatives de glycanes sulfatés ou sialylés pour sa fixation membranaire avant d’induire ses activités. Des lectines vé
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Sadot, Lebouvier Sophie Campone Mario. "Expression du syndecan-1 dans les carcinomes." [S.l.] : [s.n.], 2008. http://castore.univ-nantes.fr/castore/GetOAIRef?idDoc=48311.

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Tkachenko, Eugene. "Syndecan-4 in cell signaling and membrane trafficking." [Maastricht : Maastricht : Universiteit Maastricht] ; University Library, Maastricht University [Host], 2005. http://arno.unimaas.nl/show.cgi?fid=6428.

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Books on the topic "Syndecans"

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Grootjans, Jan Johann. Cytoplasmic interactions of the syndecans. Leuven University Press, 2000.

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Book chapters on the topic "Syndecans"

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Leonova, Elena I., and Oxana V. Galzitskaya. "Role of Syndecans in Lipid Metabolism and Human Diseases." In Advances in Experimental Medicine and Biology. Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-17344-3_10.

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Haubeck, H. D. "Syndecane." In Lexikon der Medizinischen Laboratoriumsdiagnostik. Springer Berlin Heidelberg, 2017. http://dx.doi.org/10.1007/978-3-662-49054-9_2950-1.

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Haubeck, H. D. "Syndecane." In Springer Reference Medizin. Springer Berlin Heidelberg, 2019. http://dx.doi.org/10.1007/978-3-662-48986-4_2950.

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Swain, Niharika, Rashmi Maruti Hosalkar, and Samapika Routray. "Syndecan-1." In Encyclopedia of Signaling Molecules. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-67199-4_102002.

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Swain, Niharika, Rashmi Maruti Hosalkar, and Samapika Routray. "Syndecan-1." In Encyclopedia of Signaling Molecules. Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4614-6438-9_102002-1.

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Handra-Luca, Adriana. "Syndecan-1 in the Tumor Microenvironment." In Advances in Experimental Medicine and Biology. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-48457-6_3.

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Ibrahim, Sherif Abdelaziz, Hebatallah Hassan, Rolland Reinbold, Nancy Adriana Espinoza-Sanchez, Burkhard Greve, and Martin Götte. "Role of Syndecan-1 in Cancer Stem Cells." In Proteoglycans in Stem Cells. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-73453-4_12.

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Oh, Eok-Soo, and John R. Couchman. "Syndecan-2 Biology and Its Role in Colorectal Carcinoma." In The Extracellular Matrix and the Tumor Microenvironment. Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-99708-3_4.

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Jones, F. K., O. Kehoe, A. Daroszewska, R. J. van’t Hof, and A. Pisconti. "Syndecan-3: A Signaling Conductor in the Musculoskeletal System." In Proteoglycans in Stem Cells. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-73453-4_7.

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Dias-Teixeira, Karina, Xavier Horton, Robert McKown, Jeffrey Romano, and Gordon W. Laurie. "The Lacritin-Syndecan-1-Heparanase Axis in Dry Eye Disease." In Advances in Experimental Medicine and Biology. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-34521-1_31.

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Conference papers on the topic "Syndecans"

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Rodriguez-Jimenez, NA, EG Cardona-Muñoz, JI Gamez-Nava, et al. "THU0161 Syndecans are correlated with high titres of antibodies against citrullinated proteins antigens (ACPAS) in sera from active rheumatoid arthritis." In Annual European Congress of Rheumatology, 14–17 June, 2017. BMJ Publishing Group Ltd and European League Against Rheumatism, 2017. http://dx.doi.org/10.1136/annrheumdis-2017-eular.4965.

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Nadler, Samuel, Qing Lang Li, Peter Chen, and William Parks. "Syndecan-1 Shedding And Neutrophil Activation." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a1398.

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Pap, T. "SP0135 Myostatin, sclerostin, syndecan and more." In Annual European Congress of Rheumatology, 14–17 June, 2017. BMJ Publishing Group Ltd and European League Against Rheumatism, 2017. http://dx.doi.org/10.1136/annrheumdis-2017-eular.7261.

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Cheng, Chao-Min, Robert L. Steward, Danny L. Wang, and Philip R. LeDuc. "Effects of Mechanical Strain on Syndecan-4, Focal Adhesion Complexes, and Site-Specific FAK Phosphorylation." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192360.

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Cellular mechanics involves the ability of cells to sense and respond to external forces. The type of deformations that cells undergo such as stretching, compression or shearing depends on physiological conditions such as how muscle deforms during movement. Using a custom fabricated stretching device, we focused on investigating how statically applied mechanical stretch affects syndecan-4, focal adhesion complexes and phosphorylated focal adhesion kinase (p-FAK). These results have potential implications in a variety of fields including biophysics, mechanotransduction, and cellular structure.
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Tanino, Y., X. Wang, Y. Inokoshi, et al. "Syndecan-4 as a Potential Biomarker for Sarcoidosis." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a2267.

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Inokoshi, Yayoi. "The Role Of Syndecan-1 In ARDS/ALI." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a1707.

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Parimon, T., Y. Huang, D. Narayanan, et al. "Syndecan 1 Regulates Epithelial Cell Senescence Mediating Through Macropinocytosis." In American Thoracic Society 2022 International Conference, May 13-18, 2022 - San Francisco, CA. American Thoracic Society, 2022. http://dx.doi.org/10.1164/ajrccm-conference.2022.205.1_meetingabstracts.a3897.

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Hughes, J. Margaret, Najwa Khalil, Karunasri Vanapalli, et al. "Airway Smooth Muscle Cells From People With Asthma Shed Syndecan-4." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a2089.

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Fröhling, Mareike, Dominik Bettenworth, Sonja Dufentester, et al. "08.23 Syndecan-4 exerts a protective function in experimental intestinal inflammation." In 37th European Workshop for Rheumatology Research 2–4 March 2017 Athens, Greece. BMJ Publishing Group Ltd and European League Against Rheumatism, 2017. http://dx.doi.org/10.1136/annrheumdis-2016-211055.23.

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Schlesinger, Saundra, Catherine Buell, William Parks, and Peter Chen. "Lung Cancer Loss Of Syndecan-1 Promotes A More Invasive Phenotype." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a6359.

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