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Books on the topic 'Synthesis of purine nucleotides'

Author: Grafiati
Published: 8 September 2021
Last updated: 1 February 2022

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1

Pharmacology of purine and pyrimidine receptors. San Diego, CA: Elsevier, 2011.

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2

Teshiba, Sadao. Production of nucleotides and nucleosides by fermentation. New York: Gordon and Breach Science Publishers, 1989.

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3

Reiner, Tilman. [Beta]-eliminierbare Schutzgruppen als allgemeines Prinzip in der Oligonucleotidchemie: Ein Beitrag zur automatisierten Synthese von Oligonucleotiden. Konstanz: Hartung-Gorre, 1988.

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4

Schwarz, Michael Walter. Synthese von Oligonukleotiden und Nukleotiden mit terminaler Phosphotriester- und Thiophosphotriesterfunktion mit Hilfe neuer Phosphor (III) Verbindungen nach der Phosphitamidmethode. Konstanz: Hartung-Gorre, 1986.

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5

M, Bezborodov A., ed. Mikrobiologicheskiĭ sintez nukleozidfosfatov. Moskva: "Nauka", 1990.

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6

Ruf, Klaus. Synthese biologisch interessanter Nukleoside und Oligonukleotide. Konstanz: Hartung-Gorre, 1987.

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7

Eastwood, P. R. Synthesis of new purine anti-viral and anti-cancer agents. Manchester: UMIST, 1993.

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8

Forman, Gary. The synthesis of potential anti-hepatitis B virus nucleotides. Birmingham: University of Birmingham, 1994.

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9

M, Poltavchenko G., ed. Rolʹ adenozina v reguli͡a︡t͡s︡ii fiziologicheskikh funkt͡s︡iĭ organizma. Sankt-Peterburg: "Nauka," S.-Peterburgskoe otd-nie, 1991.

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10

Tusa, Girolamo. Synthesis and biological activity of conformationally constrained nucleosides and nucleotides. Ottawa: National Library of Canada, 1998.

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11

Steppan, Heinz. Mehrfache und selektive elektrochemische Schutzgruppenabspaltung in der Oligonucleotidsynthese. Konstanz: Hartung-Gorre, 1985.

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12

Alfred, Jäger. Neue Wege zu Nukleosidmethylphosphonaten, sowie deren Thio- und Selenophosphonatanaloga: Synthese eines Adenylattrimers mit 2', 5'-Methylphosphonatinternukleotidbindung. Konstanz: Hartung-Gorre, 1985.

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13

S, Bruzik K., and Stec W. J, eds. Biophosphates and their analogues: Synthesis, structure, metabolism, and activity : proceedings of the 2nd International Symposium on Phosphorus Chemistry Directed Towards Biology, Łodz, Poland, 8-12 September 1986. Amsterdam: Elsevier, 1987.

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14

Zhao, Xiaoqin S. Purine nucleotide-induced seizures in rat prepiriform cortex. 1994.

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15

Zhao, Xiaoqin S. Purine nucleotide-induced seizures in rat prepiriform cortex. 1994.

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16

Professor, Turner John T., Weisman Gary A, and Fedan Jeffrey S, eds. The P2 nucleotide receptors. Totowa, N.J: Humana Press, 1998.

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17

Nelson, Jeffrey Stephen. Synthesis and oligomerization of y,4-Diamino-2-oxo-1(2H)-pyrimidinepentanoic acid. 1989.

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18

Stebbins, Jeffrey W. Catalysis, regulation and relationship to structure of Escherichia coli aspartate transcarbamoylase: By Jeffrey William Stebbins. 1990.

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19

H, Schlesinger David, ed. Macromolecular sequencing and synthesis: Selected methods and applications. New York: A.R. Liss, 1988.

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20

Egli, Martin, and Piet Herdewijn. Chemistry and Biology of Artificial Nucleic Acids. Wiley & Sons, Limited, John, 2012.

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21

H, Blöcker, Frank R, Fritz Hans-Joachim, Stiftung Volkswagenwerk, Fonds der Chemischen Industrie (Germany), and Germany (West). Bundesministerium für Forschung und Technologie., eds. Chemical synthesis in molecular biology: Biological macromolecules with natural and modified monomer units. Weinheim, Federal Republic of Germany: VCH, 1987.

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22

Frank, R., and H. Blocker. Chemical Synthesis in Molecular Biology: Biological Macromolecules With Natural and Modified Monomer Units (Gbf Monographs, Vol 8). Vch Pub, 1987.

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23

1953-, Jacobson Kenneth Alan, Daly John W, and Manganiello V, eds. Purines in cellular signaling: Targets for new drugs. New York: Springer-Verlag, 1990.

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24

Daly, John W., and Kenneth A. Jacobson. Purines in Cellular Signaling: Targets for New Drugs. Springer, 1990.

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25

Jacobson, Kenneth A. Purines in Cellular Signaling: Targets for New Drugs. Springer, 2011.

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26

Dalbeth, Nicola. Pathophysiology of gout. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199668847.003.0039.

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Abstract:
The clinical features of gout occur in response to monosodium urate (MSU) crystals. Gout should be considered a chronic disease of MSU crystal deposition. A number of pathophysiological checkpoints are required for development of gout. First, elevated urate concentrations are required: urate overproduction and underexcretion contribute to total urate balance. Overproduction occurs due to alterations in the purine synthesis and degradation pathways. Renal underexcretion is an important cause of elevated serum urate concentrations (hyperuricaemia), and occurs through alterations in the urate transporters within the renal tubule (collectively known as the urate transportasome). Gut underexcretion (extrarenal urate underexcretion) also contributes to development of hyperuricaemia. The next checkpoint is MSU crystal formation. In some individuals with evidence of MSU crystal deposition, symptomatic gout develops. The acute inflammatory response to MSU crystals represents a self-limiting sterile acute auto-inflammatory response which is mediated by the innate immune system activation. Interleukin 1 beta is the key cytokine that contributes to the acute inflammatory response to MSU crystals. In some patients, advanced gout may occur with structural joint damage. Joint damage in gout is mediated both by direct effects of MSU crystals on joint tissue and by indirect effects of joint inflammation. In addition to their central role in pathogenesis of gout, MSU crystals have a physiological role, particularly as an adjuvant or ‘danger signal’ in immune surveillance.
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27

Byrne, John H., ed. The Oxford Handbook of Invertebrate Neurobiology. Oxford University Press, 2017. http://dx.doi.org/10.1093/oxfordhb/9780190456757.001.0001.

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Abstract:
Invertebrates have proven to be extremely useful models for gaining insights into the neural and molecular mechanisms of sensory processing, motor control, and higher functions, such as feeding behavior, learning and memory, navigation, and social behavior. Their enormous contribution to neuroscience is due, in part, to the relative simplicity of invertebrate nervous systems and, in part, to the large cells found in some invertebrates, like mollusks. Because of the organizms’ cell size, individual neurons can be surgically removed and assayed for expression of membrane channels, levels of second messengers, protein phosphorylation, and RNA and protein synthesis. Moreover, peptides and nucleotides can be injected into individual neurons. Other invertebrate systems such as Drosophila and Caenorhabditis elegans are ideal models for genetic approaches to the exploration of neuronal function and the neuronal bases of behavior. The Oxford Handbook of Invertebrate Neurobiology reviews neurobiological phenomena, including motor pattern generation, mechanisms of synaptic transmission, and learning and memory, as well as circadian rhythms, development, regeneration, and reproduction. Species-specific behaviors are covered in chapters on the control of swimming in annelids, crustacea, and mollusks; locomotion in hexapods; and camouflage in cephalopods. A unique feature of the handbook is the coverage of social behavior and intentionality in invertebrates. These developments are contextualized in a chapter summarizing past contributions of invertebrate research as well as areas for future studies that will continue to advance the field.
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