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Books on the topic 'Systemic circulation'

Author: Grafiati

Published: 3 June 2025

Last updated: 20 July 2025

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1

Norbert, Heisler, and Boutilier R. G. 1953-, eds. Mechanisms of systemic regulation: Respiration and circulation. Springer, 1995.

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2

B, Blauhut, and Lundsgaard-Hansen P, eds. Albumin and the systemic circulation: International Albumin Workshop, Grindelwald, March 5-7, 1986. Karger, 1986.

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3

Lagerkvist, Birgitta. Systemic effects of occupational exposure to arsenic: With special reference to peripheral circulation and nerve function. University of Umeaa, 1989.

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4

Kvietys, Peter R. The gastrointestinal circulation. Morgan & Claypool Life Sciences, 2010.

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5

1953-, Holdrege Craig, and Creeger Katherine, eds. The dynamic heart and circulation. Association of Waldorf Schools of North America, 2002.

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6

Korshunova, Lyudmila, Natal'ya Prodanova, Elena Zacarinnaya, and Tat'yana Bondarenko. Finance, money circulation and credit. INFRA-M Academic Publishing LLC., 2023. http://dx.doi.org/10.12737/1550594.

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The textbook systematically presents educational material that gives a holistic view of the essence, functions and role of finance, the structure of the financial system of the Russian Federation, the functional foundations of financial management and financial control, the principles of the organization of the budget system and the budget process in the Russian Federation, the appointment of state extra-budgetary funds, the principles of the organization of finances of economic entities, as well as reflecting other relevant questions. To self-check the completeness of mastering the educational material, tests of current control are placed at the end of each chapter. Meets the requirements of the federal state educational standards of secondary vocational education of the latest generation. It is intended for students of secondary educational institutions studying in the specialty 38.02.06 "Finance". It can also be used as a textbook for students studying in other economic specialties and areas of training.

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7

R, Kvietys Peter, Barrowman J. A, and Granger D. Neil, eds. Pathophysiology of the splanchnic circulation. CRC Press, 1987.

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8

Perloff, Joseph K. Physical examination of the heart and circulation. 4th ed. People's Medical Pub. House, 2009.

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9

Noordergraaf, Abraham. Blood in Motion. Springer Science+Business Media, LLC, 2011.

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10

Barraclough, Sue. The circulatory system: Why does my heart beat? Heinemann Library, 2008.

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11

Whittemore, Susan. The circulatory system. Chelsea House Publishers, 2004.

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12

Whittemore, Susan. The circulatory system. Chelsea House, 2008.

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13

Stille, Darlene R. The circulatory system. Children's Press, 1997.

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14

R, Pinsky Michael, Dhainaut J. -F, Artigas Antonio, and International Symposium on Applied Physiology of the Peripheral Circulation (1st : 1995 : Barcelona, Spain), eds. The splanchnic circulation: No longer a silent partner. Springer, 1995.

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15

Sandblom, Erik. The venous circulation in teleost fish: Responses to exercise, temperature and hypoxia. Dept. of Zoology/Zoophysiology, Göteborg University, 2007.

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16

B, Garfein Oscar, ed. Current concepts in cardiovascular physiology. Academic Press, 1990.

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17

Mandin, David. Les systèmes d'échanges locaux (SEL): Circulations affectives et économie monétaire. L'Harmattan, 2009.

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18

Tsiaras, Alexander. The Invision guide to lifeblood. Collins, 2008.

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19

Ballard, Carol. What is my pulse? Raintree, 2011.

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20

Jean, Ginsburg, ed. The circulation in the female: From the cradle to the grave. Parthenon Publishing, 1989.

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21

Tsuchiya, Kiichi. Mechanical simulator of the cardiovascular system: Design, development, and its application. Vieweg, 1987.

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22

Heisler, Norbert, G. Boutilier, W. W. Burggren, M. E. Feder, and N. Heisler. Mechanisms of Systemic Regulation: Respiration and Circulation. Springer London, Limited, 2012.

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23

Boutilier, G., W. W. Burggren, M. E. Feder, N. Heisler, and J. W. Hicks. Mechanisms of Systemic Regulation: Respiration and Circulation. Springer London, Limited, 2011.

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24

Mechanisms of Systemic Regulation. Island Press, 1995.

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25

Lundsgaard-Hansen, P. Albumin and the Systemic Circulation (Current Studies in Hematology and Blood Transfusion). S. Karger AG (Switzerland), 1986.

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26

Iwakiri, Yasuko. The Molecules: Abnormal Vasculatures in the Splanchnic and Systemic Circulation in Portal Hypertension.". INTECH Open Access Publisher, 2012.

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27

Heisler, Norbert, ed. Mechanisms Of Systemic Regulation: RESPIRATION AND CIRCULATION (DISCONTINUED (Advances in Comparative and Environmental Physiology)). SPRINGER-VERLAG, 1995.

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28

Atkinson, D. E., E. Bourke, Norbert Heisler, W. H. Dantzler, and D. H. Evans. Mechanisms of Systemic Regulation: Acid--Base Regulation, Ion-Transfer and Metabolism. Springer London, Limited, 2012.

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29

Hagendorff, Andreas. Cardiac involvement in systemic diseases. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199599639.003.0020.

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Systemic diseases are generally an interdisciplinary challenge in clinical practice. Systemic diseases are able to induce tissue damage in different organs with ongoing duration of the illness. The heart and the circulation are important targets in systemic diseases. The cardiac involvement in systemic diseases normally introduces a chronic process of alterations in cardiac tissue, which causes cardiac failure in the end stage of the diseases or causes dangerous and life-threatening problems by induced acute cardiac events, such as myocardial infarction due to coronary thrombosis. Thus, diagnostic methods—especially imaging techniques—are required, which can be used for screening as well as for the detection of early stages of the diseases. Two-dimensional echocardiography is the predominant diagnostic technique in cardiology for the detection of injuries in cardiac tissue—e.g. the myocardium, endocardium, and the pericardium—due to the overall availability of the non-invasive procedure.The quality of the echocardiography and the success rate of detecting cardiac pathologies in patients with primary non-cardiac problems depend on the competence and expertise of the investigator. Especially in this scenario clinical knowledge about the influence of the systemic disease on cardiac anatomy and physiology is essential for central diagnostic problem. Therefore the primary echocardiography in these patients should be performed by an experienced clinician or investigator. It is possible to detect changes of cardiac morphology and function at different stages of systemic diseases as well as complications of the systemic diseases by echocardiography.The different parts of this chapter will show proposals for qualified transthoracic echocardiography focusing on cardiac structures which are mainly involved in different systemic diseases.

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30

Heisler, Norbert. Mechanisms of Systemic Regulation: Acid - Base Regulation, Ion-Transfer And Metabolism ... and Environmental Physiology ). Springer, 2012.

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31

van der Vlag, Johan, and Jo H. M. Berden. The patient with systemic lupus erythematosus. Edited by Giuseppe Remuzzi. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0161.

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Systemic lupus erythematosus (SLE) is a systemic autoimmune disease with various clinical manifestations. The hallmark of SLE is the presence of antibodies against nuclear constituents, such as double-stranded (ds)DNA, histones, and nucleosomes. Local deposition of antinuclear antibodies in complex with nuclear autoantigens induces serious inflammatory conditions that can affect several tissues and organs, including the kidney.The levels of antinucleosome and anti-dsDNA antibodies seem to correlate with glomerulonephritis and these autoantibodies can often be detected years before the patient is diagnosed with SLE. Apoptotic debris is present in the extracellular matrix and circulation of patients with SLE due to an aberrant process of apoptosis and/or insufficient clearance of apoptotic cells and apoptotic debris. The non-cleared apoptotic debris in patients with SLE may lead to activation of both the innate (myeloid and plasmacytoid dendritic cells) and adaptive (T and B cells) immune system. In addition to the activation by apoptotic debris and immune complexes, the immune system in SLE may be deregulated at the level of (a) presentation of self-peptides by antigen-presenting cells, (b) selection processes for both B and T cells, and (c) regulatory processes of B- and T-cell responses. Lupus nephritis may be classified in different classes based on histological findings in renal biopsies. The chromatin-containing immune complexes deposit in the capillary filter, most likely due to the interaction of chromatin with the polysaccharide heparan sulphate. A decreased renal expression of the endonuclease DNaseI further contributes to the glomerular persistence of chromatin and the development of glomerulonephritis.Current treatment of lupus nephritis is not specific and aims to reduce the inflammatory response with general immunosuppressive therapies. However, research has revealed novel potential therapeutic candidates at the level of dendritic cells, B cells, and T cells.

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32

Beattie, R. Mark, Anil Dhawan, and John W.L. Puntis. Bacterial, fungal, and parasitic infections of the liver. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569862.003.0059.

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Bacterial sepsis 428Spirochaetal infections 431Rickettsial infections 432Fungal infections 432Parasitic infections 434Granulomatous hepatitis 437Infectious agents can affect the liver either via direct invasion or by release of toxins. The liver's dual blood supply renders it uniquely susceptible to infection, receiving blood from the intestinal tract via the hepatic portal system, and from the systemic circulation via the hepatic artery. Because of this unique perfusion, the liver is frequently exposed to systemic or intestinal infections or the mediators of toxaemia. The biliary tree provides a further conduit for gut bacteria or parasites to access the liver parenchyma....

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33

Waberski, Andrew T., and Nina Deutsch. Transposition of the Great Arteries. Edited by Kirk Lalwani, Ira Todd Cohen, Ellen Y. Choi, and Vidya T. Raman. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190685157.003.0010.

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Transposition of the great arteries is a congenital cardiac abnormality that presents in the neonatal period, most commonly as cyanosis. While variations in anatomic features exist, dextro-transposition of the great arteries, the most common form, results in 2 separate circulatory systems in parallel, such that the right ventricle pumps deoxygenated blood to the systemic circulation, and the left ventricle sends oxygenated blood back to the pulmonary circulation. To ensure survival, early diagnosis and intervention to allow for adequate mixing of blood is necessary. The arterial switch operation is the definitive treatment, usually undertaken in the first few days of life. Known complications of surgery include ischemia, bleeding, hemodynamic compromise, and arrhythmias. Anesthetic management must take these factors into account.

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34

McKenzie, Ian. Single Ventricle Physiology. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199764495.003.0031.

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Congenital cardiac abnormalities in which there is functionally only a single ventricle are a heterogeneous group of conditions. These include patients with marked hypoplasia of one ventricle, usually with hypoplasia or atresia of the inflow of the ventricle, such as in hypoplastic left heart syndrome or conditions where surgical separation of the flow to each ventricle is not possible, such as double-inlet left ventricle. The most common pathway for palliating these conditions will be to use cavopulmonary connections to provide lung blood flow direct from systemic venous return (reliant on systemic venous pressure). The single ventricle pumps to the systemic arterial circulation. Many of these patients will be long-term survivors and present with acute surgical conditions unrelated to their cardiac condition. The safe anesthesia management of patients with single ventricle physiology and cavopulmonary connections involves assessing their cardiovascular reserve and understanding the effects of hypovolemia, anesthesia, positive-pressure ventilation, and the procedure itself on their circulation.

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35

Thorne, Sara, and Paul Clift, eds. Influence of pulmonary blood flow on management and outcome. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199228188.003.0005.

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Introduction 50Pulmonary vascular development in early life 50Cyanotic heart disease and pulmonary blood flow 52Delivery of systemic venous blood to the alveolar capillary membrane to allow release of waste CO2 and uptake of O2 depends on the integrity of the pulmonary circulation. Too little blood flow to the lungs and the patient is hypoxic; too much and the lungs become oedematous....

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36

O’Brien, Alastair. Pathophysiology, diagnosis, and assessment of acute or chronic hepatic failure. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0199.

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Cirrhosis is an increasing problem and prognosis following intensive care unit admission is poor. Acute on chronic liver failure (ACLF) is a separate entity to cirrhosis with organ failure at the core of this syndrome. Infection and the associated systemic inflammatory responses are the most important precipitants of ACLF. Clinical assessment should follow the standard airway breathing circulation disability exposure approach to the critically-ill patient.

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37

Nolan, Jerry P., and Michael J. A. Parr. Management after resuscitation from cardiac arrest. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0066.

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Systemic ischaemia during cardiac arrest and the reperfusion response after return of spontaneous circulation (ROSC) cause the post-cardiac arrest syndrome (PCAS). The severity and duration of this syndrome is determined by the cause and duration of cardiac arrest, quality of resuscitation, and interventions after ROSC. Four key clinical components are recognized—post-cardiac arrest brain injury, myocardial dysfunction, other organ ischaemia/reperfusion (e.g. liver, kidney), and potential persistence of the precipitating pathology causing the cardiac arrest. The interventions applied after ROSC impact significantly on the quality of survival. All components of the PCAS need to be addressed if outcome is to be optimized; treatment should start immediately after ROSC. An ‘ABCDE’ (Airway, Breathing, Circulation, Disability, Exposure) systems approach is used to identify and treat physiological abnormalities and organ injury. All survivors of out-of-hospital cardiac arrest should be considered for urgent coronary angiography unless the cause of cardiac arrest is clearly non-cardiac or continued treatment is considered futile. Targeted temperature management (mild hypothermia and avoidance of hyperthermia) should be considered for those patients who remain comatose after ROSC. If targeted temperature management has been used, early prognostication on outcome is unreliable and should be delayed until 3 days after return to normothermia; it should not rely on just one modality.

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38

Thorne, Sara, and Sarah Bowater. Transposition complexes. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198759959.003.0013.

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Transposition complexes refer to hearts in which there is a reversal in the relationship between the ventricles and great arteries, i.e. there is ventriculoarterial discordance. Thus, the right ventricle gives rise to the aorta and supports the systemic circulation, whilst the left ventricle becomes the subpulmonary ventricle. There are two types of transposition: complete transposition of the great arteries (TGA) and congenitally corrected TGA. This chapter discusses complete TGA, including interarterial repair (Mustard or Senning operation), arterial switch operation, and Rastelli operation. It also covers congenitally corrected transposition of the great arteries (ccTGA), including atrioventricular (AV) and ventriculoarterial (VA) discordance.

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39

Field, John. Therapeutic strategies in managing cardiac arrest. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0064.

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Emergency and critical care specialists are important interdisciplinary physicians who often impact on the long-term survival of patients sustaining cardiac arrest, as well as immediate outcomes. These specialists are often at the crossroads of survival for patients achieving return of spontaneous circulation, and it is important to appreciate that out-of-hospital and in-hospital cardiac arrest patients represent different pathophysiological subgroups with respect to aetiology and pathophysiology. Important time-dependent triage and therapy are crucial, and efforts to identify and treat pathophysiological triggers share priority with the initiation of hypothermia protocols and other targeted interventions, such as coronary angiography and percutaneous coronary intervention. Updated basic life support (BLS) and advanced life support (ACLS) protocols emphasize the importance of high quality chest compressions as central to achieving return of spontaneous circulation and emphasize that airway interventions should not detract from this objective. No specific ACLS intervention including intubation, vasopressor therapy or use of anti-arrhythmic agents has been found to improve outcome. The goal of both BLS and ACLS protocols is the achievement of return of spontaneous circulation, the prevention of re-arrest and the initiation of immediate post-resuscitation interventions associated with improved outcome. These include targeted temperature management (induced hypothermia) and coronary angiography for appropriate patients and ‘bundled’ critical care for all recognizing that the post-arrest state is a systemic inflammatory condition requiring multidisciplinary care beyond hypothermia and cardiovascular support.

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40

Gilmore, Joseph P., and Irving H. Zucker. Reflex Control of the Circulation. Taylor & Francis Group, 2020.

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41

Gilmore, Joseph P., and Irving H. Zucker. Reflex Control of the Circulation. Taylor & Francis Group, 2020.

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42

Gilmore, Joseph P., and Irving H. Zucker. Reflex Control of the Circulation. Taylor & Francis Group, 2020.

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43

Gilmore, Joseph P., and Irving H. Zucker. Reflex Control of the Circulation. Taylor & Francis Group, 2020.

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44

Diseases of the visceral circulation. Arnold, 2002.

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45

(Editor), George Geroulakos, and Kenneth J. Cherry (Editor), eds. Diseases of the Visceral Circulation. A Hodder Arnold Publication, 2002.

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46

Furst, Branko. Heart and Circulation: An Integrative Model. Springer International Publishing AG, 2020.

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47

Furst, Branko. Heart and Circulation: An Integrative Model. Springer, 2013.

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48

Fishman, Alfred P., and Dickinson W. Richards. Circulation of the Blood: Men and Ideas. Springer London, Limited, 2013.

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49

Balhara, Kamna S., Basem F. Khishfe, and Jamil D. Bayram. Sepsis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199976805.003.0004.

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Sepsis is a clinical syndrome characterized by systemic inflammation in the presence of infection. The source of infection may be occult. One must be aware of the epidemiology, presenting features and complications, diagnostic considerations and tests, and the organisms involved. Bacteria (gram positive and negative) are most commonly associated with sepsis, although fungi, viruses, and parasites can cause sepsis. Infections in the lungs, urinary tract, abdomen, skin, brain, and other areas can cause bacteremia and lead to sepsis. Treatment includes airway, breathing, and circulation (ABCs) management; aggressive fluid resuscitation; early administration of broad-spectrum antibiotics; and early goal-directed therapy and severe sepsis resuscitation bundle. Diagnosis can be challenging in pediatric and geriatric populations.

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50

Kahn, S. Lowell. The Anchor and Scaffold Techniques for Precise Coil Embolization. Edited by S. Lowell Kahn, Bulent Arslan, and Abdulrahman Masrani. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199986071.003.0065.

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Deploying coils accurately in high-flow vessels can be challenging. Particularly with larger vessels, obtaining the stability to prevent downstream coil migration is of paramount importance. This is particularly true in the embolization of pulmonary arteriovenous malformations, in which distal coil migration to the systemic circulation can have disastrous consequences. Although detachable coils/balloons, hydrocoils, and plugs mitigate some of this risk, these devices are costly and not always readily available. Furthermore, there are times when a large plug may not be deliverable to the intended target, necessitating use of coils instead. This chapter describes two separate techniques to secure coils at their site of deployment—the anchor technique and the scaffold technique.

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