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Relevant bibliographies by topics / Systemic EBV^+ T cell lymphoproliferative disease of childhood

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Academic literature on the topic 'Systemic EBV^+ T cell lymphoproliferative disease of childhood'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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Journal articles on the topic "Systemic EBV^+ T cell lymphoproliferative disease of childhood"

1

Quintanilla-Martinez, Leticia, Cecilia Ridaura, Florian Nagl, et al. "Hydroa vacciniforme-like lymphoma: a chronic EBV+ lymphoproliferative disorder with risk to develop a systemic lymphoma." Blood 122, no. 18 (2013): 3101–10. http://dx.doi.org/10.1182/blood-2013-05-502203.

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Key Points HVLL is a chronic EBV+ lymphoproliferative disorder of childhood with risk to develop systemic lymphoma. The disease shows favorable response to conservative therapy despite the presence of a T- or NK-cell monoclonal proliferation.

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2

Syrykh, Charlotte, Sarah Péricart, Claire Lamaison, Frédéric Escudié, Pierre Brousset, and Camille Laurent. "Epstein–Barr Virus-Associated T- and NK-Cell Lymphoproliferative Diseases: A Review of Clinical and Pathological Features." Cancers 13, no. 13 (2021): 3315. http://dx.doi.org/10.3390/cancers13133315.

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Epstein–Barr virus (EBV) is a ubiquitous virus detected in up to 95% of the general population. Most people are asymptomatic, while some may develop a wide range of EBV-associated lymphoproliferative disorders (LPD). Among them, EBV-positive T/NK LPD are uncommon diseases defined by the proliferation of T- or NK-cells infected by EBV. The 2017 World Health Organization (WHO) classification recognizes the following entities characterized by different outcomes: chronic active EBV infection of T- or NK-cell types (cutaneous and systemic forms), systemic EBV-positive T-cell lymphoma of childhood,

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3

Hyo, Rie, Yasunobu Abe, Kisato Nosaka, et al. "Proposal of Progressive Adult Onset EBV-Associated Lymphoproliferative Disorder (PAEBV) as a Rapid-Onset Neoplastic Disease." Blood 114, no. 22 (2009): 2920. http://dx.doi.org/10.1182/blood.v114.22.2920.2920.

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Abstract Abstract 2920 Poster Board II-896 [Background] Epstein-Barr virus (EBV) is currently recognized as a pathogen of several known human diseases ranging from transient infection, chronic lymphoproliferative disorder (LPD) to EBV-positive malignant lymphomas. Chronic active EBV infection (CAEBV) is typically an indolent but long-lasting EBV-LPD of childhood. Another well documented EBV-associated LPD is the post-transplant EBV-LPD, which also occurs in adults. Fulminant proliferation of EBV after primary infection or in the setting of CAEBV is an aggressive type of disease, which has rece

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4

Chen, Chien-Chin, Kung-Chao Chang, L. Jeffrey Medeiros, and Julia Yu-Yun Lee. "Hydroa Vacciniforme and Hydroa Vacciniforme-Like Lymphoproliferative Disorder: A Spectrum of Disease Phenotypes Associated with Ultraviolet Irradiation and Chronic Epstein–Barr Virus Infection." International Journal of Molecular Sciences 21, no. 23 (2020): 9314. http://dx.doi.org/10.3390/ijms21239314.

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Hydroa vacciniforme (HV) is a rare form of photosensitivity disorder in children and is frequently associated with Epstein–Barr virus (EBV) infection, whereas HV-like lymphoproliferative disorders (HVLPD) describe a spectrum of EBV-associated T-cell or natural killer (NK)-cell lymphoproliferations with HV-like cutaneous manifestations, including EBV-positive HV, atypical HV, and HV-like lymphoma. Classic HV occurs in childhood with papulovesicules on sun-exposed areas, which is usually induced by sunlight and ultraviolet irradiation, and mostly resolves by early adult life. Unlike classic HV,

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5

Chen, Zihang, Mi Wang, Pujun Guan, et al. "Comparison of Systemic EBV-positive T-Cell and NK-Cell Lymphoproliferative Diseases of Childhood Based on Classification Evolution." American Journal of Surgical Pathology 44, no. 8 (2020): 1061–72. http://dx.doi.org/10.1097/pas.0000000000001495.

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6

Hong, Mineui, Young Hyeh Ko, Keon Hee Yoo, et al. "EBV-Positive T/NK-Cell Lymphoproliferative Disease of Childhood." Korean Journal of Pathology 47, no. 2 (2013): 137. http://dx.doi.org/10.4132/koreanjpathol.2013.47.2.137.

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7

Yasa Benkli, C., A. Marcogliese, M. Elghetany, et al. "Diagnostic Challenges And Clinical Implications For Systemic EBV-Associated T-Cell Lymphoproliferative Disorders Of Childhood." American Journal of Clinical Pathology 154, Supplement_1 (2020): S158. http://dx.doi.org/10.1093/ajcp/aqaa161.345.

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Abstract Introduction/Objective Systemic EBV-associated T-cell lymphoproliferative disorders of childhood (S-EBV-T-LPD) comprise three major forms: EBV-positive hemophagocytic lymphohistiocytosis (EBV-HLH), systemic EBV-positive T- cell lymphoma (S-EBV-TCL), and systemic T-cell chronic active EBV infection (S-T-CAEBV). These disorders are rare in children and young adults in Western countries and are associated with poor outcomes. Frequently patients were treated initially for EBV-HLH and subsequently found to have relapsed/refractory EBV-HLH vs S-EBV-TCL or overt EBV+ TCL, the latter of which

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8

Dutz, Jan P., Loralyn Benoit, Xiaoxia Wang, et al. "Lymphocytic vasculitis in X-linked lymphoproliferative disease." Blood 97, no. 1 (2001): 95–100. http://dx.doi.org/10.1182/blood.v97.1.95.

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Abstract Systemic vasculitis is an uncommon manifestation of X-linked lymphoproliferative disease (XLP), a disorder in which there is a selective immune deficiency to Epstein-Barr virus (EBV). The molecular basis for XLP has recently been ascribed to mutations within SLAM-associated protein (SAP), an SH2 domain–containing protein expressed primarily in T cells. The authors describe a patient who died as a result of chronic systemic vasculitis and fulfilled clinical criteria for the diagnosis of XLP. Sequencing of this patient'sSAP gene uncovered a novel point mutation affecting the SH2 domain.

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9

Zhou, Jiehao, Dehua Wang, and Mehdi Nassiri. "Clonal CD8+ T Lymphocytic Proliferation and Karyotypical Abnormalities in an EBV Associated Hemophagocytic Lymphohistiocytosis." Case Reports in Pathology 2015 (2015): 1–6. http://dx.doi.org/10.1155/2015/513968.

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EBV associated hemophagocytic lymphohistiocytosis and EBV-positive T cell lymphoproliferative disease of childhood share many histologic and clinical features, which sometimes makes it very difficult to render a definitive diagnosis. In this report, we present a 16-year-old male who developed symptoms clinically consistent with EBV associated hematophagocytic lymphohistiocytosis including fulfilling most of HLH diagnostic criteria and responding promptly to HLH targeted therapy. However, histologic and cytogenetics features of this case are very concerning for EBV-positive T cell lymphoprolife

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10

Kimura, Hiroshi, Yoshinori Ito, Shinji Kawabe, et al. "EBV-associated T/NK–cell lymphoproliferative diseases in nonimmunocompromised hosts: prospective analysis of 108 cases." Blood 119, no. 3 (2012): 673–86. http://dx.doi.org/10.1182/blood-2011-10-381921.

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AbstractEBV-associated T/NK–cell lymphoproliferative disease (T/NK-LPD) is defined as a systemic illness characterized by clonal proliferation of EBV-infected T or NK cells. We prospectively enrolled 108 nonimmunocompromised patients with this disease (50 men and 58 women; median onset age, 8 years; age range, 1-50 years) evidenced by expansion of EBV+ T/NK cells in the peripheral blood; these were of the T-cell type in 64 cases and of the NK-cell type in 44, and were clinically categorized into 4 groups: 80 cases of chronic active EBV disease, 15 of EBV-associated hemophagocytic lymphohistioc

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Dissertations / Theses on the topic "Systemic EBV^+ T cell lymphoproliferative disease of childhood"

1

Ito, Yoshinori, Jun-ichi Kawada, and Hiroshi Kimura. "EPSTEIN-BARR VIRUS-ASSOCIATED LYMPHOID MALIGNANCIES : THE EXPANDING SPECTRUM OF HEMATOPOIETIC NEOPLASMS." Nagoya University School of Medicine, 2013. http://hdl.handle.net/2237/18466.

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