Academic literature on the topic 'Systemic lupus erythematosus. Systemic lupus erythematosus'

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Journal articles on the topic "Systemic lupus erythematosus. Systemic lupus erythematosus"

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Chinnusamy, Manokaran, Ram Arvind Viswanathan, Sathiyanarayanan Janakiraman, and Roshna Elayidath. "Drug-Induced Lupus Erythematosus Associated with Proton Pump Inhibitor." Journal of Health and Allied Sciences NU 10, no. 03 (September 8, 2020): 132–34. http://dx.doi.org/10.1055/s-0040-1716601.

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AbstractDrug-induced lupus erythematosus is an autoimmune phenomenon where the drug exposure leads to the development of systemic lupus erythematous like clinical features. Drug-induced lupus erythematosus can be divided into systemic lupus erythematous, subacute cutaneous lupus erythematous, and chronic cutaneous lupus erythematous. Here, we report a case of a 29-year-old female presented with systemic lupus erythematous due to chronic use of proton pump inhibitors, which is considered to be very rare.
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N, Habib. "Review Article: Systemic Lupus Erythematosus." Open Access Journal of Microbiology & Biotechnology 5, no. 1 (2020): 1–4. http://dx.doi.org/10.23880/oajmb-16000158.

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Systemic lupus erythematosus or SLE is a persistent heterogeneous autoimmune disease that affects multisystem of the body. It is distinguished by acute and chronic inflammation of various tissues and even organs of the body principally the skin and joints. Systemic lupus erythematosus is a multisystem disorder and hence, it can affect any tissues, organs and even systems of the body. There are few categories of lupus for instance, lupus dermatitis or cutaneous lupus erythematosus (CLE) that affects the skin and causes malar rash, discoid lupus erythematosus (DLE) as well as systemic lupus erythematosus that causes damage to single or multiple internal organs. The damage is due to the inflammation that is caused by direct antibody reaction to the body tissues as well the deposition of immune complexes. Glucocorticoids, immunosuppressant, and anti- malarial are the combination therapy used to treat SLE besides providing counseling and awareness. Lupus erythematosus in any form particularly systemic lupus erythematosus (SLE) are prevalent in women compared to men with ratio of 6:1. It has the tendency to affect all ages but most frequently attacks women of aged 20 to 45 years old compared to men. On the other hand, if lupus erythematosus causes damage to internal organs either single or multiple, it is known as systemic lupus erythematosus. The damage is due to the inflammation that is caused by direct antibody reaction to the body tissues as well the deposition of immune complexes.
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Garbelini-Lima, Cleide, Gabriela Evangelista de Almeida, Sidharta Quércia Gabdelha, Andrea Cavalcante de Souza, Mara Lúcia Gomes de Souza, and Virginia Vilasboas Figueiras. "Discoid Lupus Erythematosus of the Scalp in a Patient with Systemic Lupus Erythematosus: A Case Report with Complete Hair Regrowth." Journal of the Portuguese Society of Dermatology and Venereology 79, no. 2 (June 26, 2021): 155–58. http://dx.doi.org/10.29021/spdv.79.2.1283.

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Scalp involvement with hair loss is common in systemic lupus erythematosus. Discoid lupus erythematosus may cause scarring alopecia, characterized by well-delimited erythematous plaques with scales, follicular hyperkeratosis and atrophy, which is considered a trichological emergency. Early diagnosis and treatment are necessary in order to prevent permanent hair loss. We describe a 44 years’ old female patient with systemic lupus erythematosus for 4 years, with multiple areas of occipitoparietal alopecia, erythematous plaques, atrophy, scales and some bloody crusts. Trichoscopy, histopathology and direct immunofluorescence led to the diagnosis of discoid lupus erythematosus. After 9 months treatment with thalidomide there was complete hair regrowth.
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Powers, David B. "Systemic Lupus Erythematosus and Discoid Lupus Erythematosus." Oral and Maxillofacial Surgery Clinics of North America 20, no. 4 (November 2008): 651–62. http://dx.doi.org/10.1016/j.coms.2008.07.001.

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Greenberg, Brent, and Margaret Michalska. "Systemic lupus erythematosus." Postgraduate Medicine 106, no. 6 (January 1999): 213–23. http://dx.doi.org/10.3810/pgm.1999.11.779.

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Dall'Era, Maria, and John C. Davis. "Systemic lupus erythematosus." Postgraduate Medicine 114, no. 5 (November 2003): 31–40. http://dx.doi.org/10.3810/pgm.2003.11.1528.

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Park, Sung Hwan. "Systemic Lupus Erythematosus." Journal of the Korean Medical Association 52, no. 7 (2009): 645. http://dx.doi.org/10.5124/jkma.2009.52.7.645.

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Kiriakidou, Marianthi. "Systemic Lupus Erythematosus." Annals of Internal Medicine 159, no. 7 (October 1, 2013): ITC4. http://dx.doi.org/10.7326/0003-4819-159-7-201310010-01004.

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Shaikh, Maliha F., Natasha Jordan, and David P. D'Cruz. "Systemic lupus erythematosus." Clinical Medicine 17, no. 1 (February 2017): 78–83. http://dx.doi.org/10.7861/clinmedicine.17-1-78.

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Steinberg, Alfred D. "Systemic Lupus Erythematosus." Annals of Internal Medicine 115, no. 7 (October 1, 1991): 548. http://dx.doi.org/10.7326/0003-4819-115-7-548.

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Dissertations / Theses on the topic "Systemic lupus erythematosus. Systemic lupus erythematosus"

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Teh, Lee-Suan. "Neuropsychiatric systemic lupus erythematosus." Thesis, University of Aberdeen, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.240703.

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Neuropsychiatric (NP) symptoms are relatively common in patients with SLE. The diverse and dramatic clinical presentations, the unclear pathogenesis, the lack of diagnostic test/s and uncertainties about the optimal management are some problems facing a clinician. When serum anti P antibodies were claimed to be highly correlated to lupus psychosis, this needed confirmation. An ELISA for measuring anti P antibodies was developed and validated. The prevalence of anti P antibodies was determined in different patient groups in a large retrospective study. Although anti P antibodies were highly specific for SLE, there was no correlation between the presence of these antibodies and lupus psychosis or other NP symptoms. Two prospective studies were carried out to eliminate any bias in our retrospective study. In one, none of the patients developed psychosis and these antibodies were not found to be specific for lupus depression or anxiety. In the other, anti P antibodies were measured in Malaysian Chinese SLE patients. No correlation was found between these antibodies and NP-SLE but a high prevalence of these antibodies was demonstrated in this group. Genetic studies showed that there was an increase in HLA-Dr2w16X subtype allele in anti P-positive patients but this did not reach significance. The usefulness of measuring antineuronal antibodies in helping to diagnose NP-SLE was examined but these antibodies were not better indicators of NP-SLE. Although the clinical correlations of anti P antibodies remain controversial, anti P antibodies were found to selectively bind to neuroblastoma cell surfaces in vitro but the nature of the surface antigen was not determined. Finally, sera from patients with lupus psychosis were found to significantly influence the response of neuroblastoma cells to agonist-induced stimulation and if confirmed, would offer an explanation for the reversible changes in cell function associated with psychiatric lupus.
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Bernatsky, Sasha. "Malignancy in systemic lupus erythematosus." Thesis, McGill University, 2001. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=32761.

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Objectives. (1) To estimate cancer incidence in systemic lupus erythematosus (SLE) as compared to the general population. (2) To estimate the sensitivity and specificity of methods of cancer ascertainment. (3) To determine the prevalence of malignancy risk factors in SLE. Methods. (1) We determined the incidence of malignancy in the Montreal General Hospital (MGH) lupus cohort, through linkage with the Quebec tumor registry. Standardized incidence ratios (SIRS) were generated, using Quebec population rates. In addition, a meta-analysis was performed by pooling data from eight cohort studies of malignancy in SLE. (2) We administered a postal survey to cohort members to determine risk factors for cancer and self-report of cancer occurrence. For dead or lost-to-follow-up patients, data was abstracted from charts. We calculated the sensitivity and specificity of self-report and chart review for cancer ascertainment, compared to registry linkage results. (3) Using the data collected on self-report and chart review, we compared risk factor prevalence within the MGH cohort to that of the Quebec population. Results. (1) Observed cancers in our cohort were greater than what would be expected; for all cancers, the SIR was 1.8 (95% Confidence Interval 1.2--2.6). The meta-analysis SIR (for all malignancies) was 1.67 (1.42--1.94). Postal survey and chart review methods demonstrated high specificity. Sensitivity was imperfect, but did not greatly effect estimation of the SIR estimate. (2) Our lupus cohort had a distinct profile of risk factors for malignancy compared to the general population; differences included more prevalent nulliparity, obesity, and use of hormone replacement therapy. Conclusions. The risk of malignancy in SLE patients is increased. Risk factor profiles could influence the incidence of certain malignancies in SLE.
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Clarke, Alexander James. "Autophagy in Systemic Lupus Erythematosus." Thesis, King's College London (University of London), 2015. http://kclpure.kcl.ac.uk/portal/en/theses/autophagy-in-systemic-lupus-erythematosus(1e5a4a5e-99f7-456e-bcb4-634a4e3fd986).html.

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Autophagy has emerged as a critical homeostatic mechanism in lymphocytes, influencing proliferation and differentiation. I sought to explore the role of autophagy in the pathogenesis of human and murine lupus, a disease in which B cells are critical effectors of pathology. Autophagy was assessed using multiple techniques in NZB/W and control mice, and in patients with SLE compared to healthy controls. I evaluated the phenotype of the B cell compartment in Vav-Atg7-/- mice in vivo, and examined human and murine plasmablast formation following inhibition of autophagy. I found activation of autophagy in early developmental stages of B cell development in a lupus mouse model even before disease-onset, and which progressively increased with age. In human disease, again autophagy was activated compared with healthy controls, principally in naïve B cells. B cells isolated from Vav-Atg7-/- mice failed to effectively differentiate into plasma cells following stimulation in vitro. Similarly, human B cells stimulated in the presence of autophagy inhibition did not differentiate into plasmablasts. My data suggest activation of autophagy is a mechanism for survival of autoreactive B cells, and also demonstrate that it is required for plasmablast differentiation, processes that induce significant cellular stress. The implication of autophagy in two major pathogenic pathways in SLE suggests the potential to use inhibition of autophagy as a novel treatment target.
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Peschken, Christine A. "Systemic lupus erythematosus in Manitoba Aboriginals." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape9/PQDD_0019/MQ55086.pdf.

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Svenungsson, Elisabet. "Cardiovascular disease in systemic lupus erythematosus /." Stockholm, 2003. http://diss.kib.ki.se/2003/91-7349-501-8.

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Russell, Andrew Ian. "Genetic predisposition to systemic lupus erythematosus." Thesis, Imperial College London, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.406513.

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Wong, Kee-lam, and 黃基林. "Systemic lupus erythematosus in Hong Kong." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1990. http://hub.hku.hk/bib/B31981410.

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Li, Hei Philip, and 李曦. "Subphenotype stratification in systemic lupus erythematosus." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2012. http://hub.hku.hk/bib/B48334765.

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Subsets of systemic lupus erythematosus (SLE) patients with distinct patterns of disease manifestations and autoantibody production have been reported, but seldom have these two phenomena been analysed together. Cluster analysis was performed on 1928 Chinese SLE patients based on autoantibody profile and the frequencies of various clinical manifestations were compared between each cluster. Separate association analyses between individual autoantibodies and clinical manifestations, as well as between clinical manifestations, were also performed. This study identifies three separate autoantibody clusters each with different clinical manifestations, and proposes that the phenomena of autoantibody clustering and clinical subsets may be inter-related. Patient clusters could also be stratified into a bipolar spectrum. On one end are patients with over-representation of anti-dsDNA and renal disorder; whilst on the other end are two distinct autoantibody clusters (anti-Sm/anti-RNP/aPL and aPL/anti-Ro/anti-La) with overlapping of other non-renal manifestations. Patient stratification could aid disease prediction and subsequent management. These findings may also elucidate disease pathogenesis and guide future study on potential common pathological processes within autoantibody clusters.
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Paediatrics and Adolescent Medicine
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Courtney, Philip. "Leukocyte apoptosis in systemic lupus erythematosus." Thesis, Queen's University Belfast, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.326415.

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Chan, Madelynn Tsu-Li. "Serological biomarkers in systemic lupus erythematosus." Thesis, University of Bath, 2013. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.607470.

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Background: Systemic lupus erythematosus (SLE) is a multi-system autoimmune disease characterised by autoantibody production and variable clinical features, ranging from mild to severe disease. Patients with SLE are at increased risk of developing accelerated atherosclerosis. Biomarkers have potential utility in SLE as markers of disease or predictors of future clinical events and mortality. Objective The aim of this thesis was to identify serological biomarkers predictive for erosive arthritis (EA), cardiovascular events (CVEs), mortality and subclinical atherosclerosis in SLE. Methods: In chapters 2 to 4, study subjects were SLE patients from Bath. Anti-cyclic citrullinated peptide antibodies (ACPA) and HLA-DR and -DQ were studied for markers of EA, and anticardiolipin (aCL) and lipoprotein profiles for markers of CVEs and mortality. In chapters 5 and 6, study subjects were women with SLE from Manchester. B-mode ultrasound scans of subjects' carotid arteries were performed at baseline and follow-up time-points to detect atherosclerotic plaque. Baseline IgG and IgM antiphospholipid (aPL) antibodies and CV risk factors were studied for markers of subclinical atherosclerosis. Clinical data collected for all studies included SLE features and auto-antibody profiles. Results: ACPA was identified as a marker of a SLE phenotype with EA - "rhupus". Patients with major erosive arthritis were HLA-DQB1*0302 carriers. Increased aCL GPL levels and total cholesterol : high density lipoprotein-C (TC : HDL-C) ratio were markers for future CVEs, and increased TC : HDL ratio, aCL GPL and lipoprotein(a) concentrations were markers for increased mortality. Lower HDL-C concentrations and anti-annexin A5 (anti-AnxA5) GPL were markers of carotid plaque progression. Conclusion: This thesis identified new markers for EA, subclinical atherosclerosis and future CVE and mortality risk in SLE. Strategies to incorporate these new CV markers into clinical CV risk assessments may assist in distinguishing the subset of SLE patients most at risk of developing accelerated atherosclerosis.
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Books on the topic "Systemic lupus erythematosus. Systemic lupus erythematosus"

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Systemic lupus erythematosus. 5th ed. Amsterdam: Elsevier Academic Press, 2011.

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Eggleton, Paul, and Frank J. Ward, eds. Systemic Lupus Erythematosus. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-0326-9.

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Miescher, Peter A., ed. Systemic Lupus Erythematosus. Berlin, Heidelberg: Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-642-79622-7.

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Smolen, Josef S., and Christoph C. Zielinski. Systemic Lupus Erythematosus. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71642-3.

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Hirohata, Shunsei, ed. Neuropsychiatric Systemic Lupus Erythematosus. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-76496-2.

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A, Sessa, Meroni Mietta, and Battini Graziana, eds. Systemic lupus erythematosus: Renal vasculitis. Basel: Karger, 1992.

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Systemic lupus erythematosus: Methods and protocols. New York: Humana Press, 2014.

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Arnaud, Laurent, and Ronald van Vollenhoven. Advanced Handbook of Systemic Lupus Erythematosus. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-43035-5.

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1917-, Aladjem Henrietta, ed. Understanding lupus. New York: Scribner, 1985.

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Stehlin, Dori. Living with lupus. Rockville, Md: Dept. of Health and Human Services, Public Health Service, Food and Drug Administration, Office of Public Affairs, 1990.

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Book chapters on the topic "Systemic lupus erythematosus. Systemic lupus erythematosus"

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Bermas, Bonnie L. "Systemic Lupus Erythematosus and Pregnancy." In Lupus Erythematosus, 183–96. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4614-1189-5_14.

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Kalman, R. S., and J. L. Wolf. "Gastrointestinal Manifestations of Systemic Lupus Erythematosus." In Lupus Erythematosus, 153–68. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4614-1189-5_12.

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Gayed, Mary, Chee-Seng Yee, Sasha Bernatsky, and Caroline Gordon. "Co-morbidities in Systemic Lupus Erythematosus." In Lupus Erythematosus, 229–49. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4614-1189-5_18.

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Goldberg, Hilary J., and Paul F. Dellaripa. "Pulmonary Manifestations of Systemic Lupus Erythematosus." In Lupus Erythematosus, 115–25. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4614-1189-5_9.

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Bertolaccini, Maria Laura, Graham R. V. Hughes, and Munther A. Khamashta. "Systemic Lupus Erythematosus." In Diagnostic Criteria in Autoimmune Diseases, 3–8. Totowa, NJ: Humana Press, 2008. http://dx.doi.org/10.1007/978-1-60327-285-8_1.

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Fassbender, Hans G. "Systemic Lupus Erythematosus." In Pathology and Pathobiology of Rheumatic Diseases, 251–64. Berlin, Heidelberg: Springer Berlin Heidelberg, 2002. http://dx.doi.org/10.1007/978-3-662-04819-1_9.

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Treadwell, Patricia. "Systemic Lupus Erythematosus." In Atlas of Adolescent Dermatology, 65–68. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-58634-8_15.

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Nagaratnam, Nages, Kujan Nagaratnam, and Gary Cheuk. "Systemic Lupus Erythematosus." In Geriatric Diseases, 549–53. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-33434-9_63.

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Laxer, Ronald M., David D. Sherry, and Philip J. Hashkes. "Systemic Lupus Erythematosus." In Pediatric Rheumatology in Clinical Practice, 63–89. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-13099-6_4.

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Sharma, Aman, Shankar Naidu, Manish Rathi, and Ritambhra Nada. "Systemic Lupus Erythematosus." In The Uveitis Atlas, 1–6. New Delhi: Springer India, 2016. http://dx.doi.org/10.1007/978-81-322-2506-5_77-1.

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Conference papers on the topic "Systemic lupus erythematosus. Systemic lupus erythematosus"

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DUARTE, ANNA BEATRIZ GOMES SOUZA, ELISA GUIMARÃES MOTTA, ADIB CHICRE MANSUR, GUILHERME SALLES DE ESCOBAR GONÇAVES, LARISSA BARROS DE OLIVEIRA, and GABRIELLEN VITIELLO. "FEVER IN SYSTEMIC LUPUS ERYTHEMATOSUS." In 36º Congresso Brasileiro de Reumatologia. São Paulo: Editora Blucher, 2019. http://dx.doi.org/10.5151/sbr2019-102.

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Greene, E. R., K. R. Lanphere, J. Sharrar, and C. A. Roldan. "Arterial distensibility in systemic lupus erythematosus." In 2009 Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 2009. http://dx.doi.org/10.1109/iembs.2009.5334459.

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Egeli, Bugra, Asli Ece Soykut, and Serdal Ugurlu. "226 Comorbidity in systemic lupus erythematosus." In 13th International Congress on Systemic Lupus Erythematosus (LUPUS 2019), San Francisco, California, USA, April 5–8, 2019, Abstract Presentations. Lupus Foundation of America, 2019. http://dx.doi.org/10.1136/lupus-2019-lsm.226.

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ZAMBONI, SHERON, JEAN PAULO VERONESE DE SOUZA, CAMILA DE DAVID CRUZ, MARCELO MALTCHIK, PAULO ROBERTO LERIAS ALMEIDA, DEISE MARCELA PIOVESAN, BÁRBARA MENDES DA SILVA, and MARKUS BREDEMEIER. "SEVERE ENTERITIS IN SYSTEMIC LUPUS ERYTHEMATOSUS." In 36º Congresso Brasileiro de Reumatologia. São Paulo: Editora Blucher, 2019. http://dx.doi.org/10.5151/sbr2019-245.

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TANAKA, ELOISE AKEMI, HARYMY BARROS, JULIANA DELFINO, THIAGO ALBERTO FERNANDES GOMES DOS SANTOS, JOSÉ FERNANDO POLANSKI, and THELMA LAROCA SKARE. "HEARING LOSS IN SYSTEMIC LUPUS ERYTHEMATOSUS." In 36º Congresso Brasileiro de Reumatologia. São Paulo: Editora Blucher, 2019. http://dx.doi.org/10.5151/sbr2019-464.

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Barboza, Flavio, Tassia Caroline Beckert Viana, Eduarda Judith Dias Jacome Silva, Raquel Gerep Pereira, Bruna Luiza Oliveira Lima, Thalyne Aparecida Leite de Lima, Maitê Luise Zanette, et al. "Systemic Lupus Erythematosus mimicking Hodgkin's lymphoma." In Congresso Brasileiro de Reumatologia 2020. Sociedade Brasileira de Reumatologia, 2021. http://dx.doi.org/10.47660/cbr.2020.16937.

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Souza da Paz, Adriane, Gustavo Luiz Behrens Pinto, and Mittermayer Barreto Santiago. "COLLAPSING GLOMERULOPATHY IN SYSTEMIC LUPUS ERYTHEMATOSUS." In Congresso Brasileiro de Reumatologia 2020. Sociedade Brasileira de Reumatologia, 2021. http://dx.doi.org/10.47660/cbr.2020.17616.

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dos Santos Cavalcante, Anauá Fernanda, Sabrina Longo, Lucas P. G., Julia Goginski, Aline Neppel, Macleise A. Kemes, Thiago Alberto G. dos Santos, and Thelma L. Skare. "DRY EYE IN SYSTEMIC LUPUS ERYTHEMATOSUS." In Congresso Brasileiro de Reumatologia 2020. Sociedade Brasileira de Reumatologia, 2021. http://dx.doi.org/10.47660/cbr.2020.17297.

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Voulgari, PV, P. Katsimbri, Y. Alamanos, and AA Drosos. "FRI0115 Systemic lupus erythematosus in men." In Annual European Congress of Rheumatology, Annals of the rheumatic diseases ARD July 2001. BMJ Publishing Group Ltd and European League Against Rheumatism, 2001. http://dx.doi.org/10.1136/annrheumdis-2001.150.

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Moszkorzova, L., C. Dostal, J. Marek, Z. Lacinova, and L. Musilova. "FRI0137 Prolactin in systemic lupus erythematosus." In Annual European Congress of Rheumatology, Annals of the rheumatic diseases ARD July 2001. BMJ Publishing Group Ltd and European League Against Rheumatism, 2001. http://dx.doi.org/10.1136/annrheumdis-2001.172.

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Reports on the topic "Systemic lupus erythematosus. Systemic lupus erythematosus"

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Harley, John. Genome-Wide Association Study in African-Americans with Systemic Lupus Erythematosus. Fort Belvoir, VA: Defense Technical Information Center, September 2013. http://dx.doi.org/10.21236/ada592038.

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Harley, John. Genome-Wide Association Study in African-Americans With Systemic Lupus Erythematosus. Fort Belvoir, VA: Defense Technical Information Center, September 2011. http://dx.doi.org/10.21236/ada554417.

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Harley, John. Genome-Wide Association Study in African-Americans with Systemic Lupus Erythematosus. Fort Belvoir, VA: Defense Technical Information Center, September 2012. http://dx.doi.org/10.21236/ada574794.

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Gontar, I. P., O. A. Rusanova, O. I. Emelyanova, S. S. Khortieva, and E. G. Cherkesova. AUTOIMMUNITY ISSUES IN PATIENTS WITH SYSTEMIC SCLERODERMA AND SYSTEMIC LUPUS ERYTHEMATOSUS WITH PREDOMINANT PULMONARY INVOLVEMENT. "PLANET", 2019. http://dx.doi.org/10.18411/978-5-907192-54-6-2019-xxxvi-73-81.

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Pfeifer, Maria. Self-help Support Groups: Choices in Participation Among Women Facing Systemic Lupus Erythematosus (SLE). Portland State University Library, January 2000. http://dx.doi.org/10.15760/etd.6685.

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Gontar, I. P., O. A. Rusanova, O. I. Emelyanova, and O. V. Paramonova. CLINICLA FEATURES OF SYSTEMIC LUPUS ERYTHEMATOSUS ASSOCIATED WITH CHANGES IN ANTIBODIES TO THYROXIN AND TRIIODOTHYRONINE. Планета, 2018. http://dx.doi.org/10.18411/978-5-907109-24-7-2018-xxxv-96-100.

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Luo, Shuaihantian, Ying Zhou, and Guiying Zhang. Association between complement 4 copy number variation and systemic lupus erythematosus: a protocol for systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, August 2020. http://dx.doi.org/10.37766/inplasy2020.8.0076.

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Aleksandrov, A. V., I. Y. Alekhina, V. A. Aleksandrov, and N. V. Aleksandrova. The role of antibodies to xanthine oxidase in the development of immunopathological reactions in the rheumatoid arthritis and systemic lupus erythematosus. ООО ИМА-Пресс, 2018. http://dx.doi.org/10.18411/1995-4484-2018-56-3(2)-11.

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Yuan, Qihang. Circulating Leptin Level, Soluble Leptin Receptor Level and Their Gene Polymorphism in Patients with Systemic Lupus Erythematosus: A Systematic Review and Meta-analysis. INPLASY - International Platform of Registered Systematic Review Protocols, April 2020. http://dx.doi.org/10.37766/inplasy2020.4.0137.

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Aleksandrova, N. V., E. V. Benedickaya, I. Yu Alekhina, and A. V. Aleksandrov. ROLE OF ANTIBODIES TO PURINE NUCLEOSIDE PHOSPHORYLASE IN THE DIAGNOSIS OF INFECTIOUS DISEASES IN PATIENTS WITH RHEUMATOID ARTHRITIS AND SYSTEMIC LUPUS ERYTHEMATOSUS. Планета, 2018. http://dx.doi.org/10.18411/978-5-907109-24-7-2018-xxxv-23-27.

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