Log in

Relevant bibliographies by topics / T-CAD

Contents

Journal articles

Academic literature on the topic 'T-CAD'

Author: Grafiati

Published: 11 September 2023

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the lists of relevant articles, books, theses, conference reports, and other scholarly sources on the topic 'T-CAD.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Journal articles on the topic "T-CAD"

1

Uhm, Tae-Kyoung, Ki-Seung Kim, Yu-Deok Seo, and Sung-Kie Youn. "T-spline Finite Element Method for CAD/CAE Integrated Approach." Transactions of the Korean Society of Mechanical Engineers A 33, no. 2 (2009): 127–34. http://dx.doi.org/10.3795/ksme-a.2009.33.2.127.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

Netto, Jeffrey, Andrej Teren, Ralph Burkhardt, et al. "Biomarkers for Non-Invasive Stratification of Coronary Artery Disease and Prognostic Impact on Long-Term Survival in Patients with Stable Coronary Heart Disease." Nutrients 14, no. 16 (2022): 3433. http://dx.doi.org/10.3390/nu14163433.

Full text

Abstract:

Knowledge about cardiac and inflammatory biomarkers in patients with stable coronary artery disease (CAD) is limited. To address this, we analyzed 3072 patients (36% female) with a median follow-up of 10 years in the Leipzig LIFE Heart Study with suspected CAD with coronary angiography. Selected biomarkers included troponin T (hsTNT), N-terminal pro B-type natriuretic peptide (NT-proBNP), copeptin, C-reactive protein (hsCRP), and interleukin-6 (IL-6). Patients were stratified by CAD severity: CAD0 (no sclerosis), CAD1 (non-obstructive, i.e., stenosis < 50%), and CAD2 (≥one stenosis ≥ 50%).

APA, Harvard, Vancouver, ISO, and other styles

3

Lloyd, Peter, Colin C. McAndrew, Michael J. McLennan, et al. "Technology CAD at AT&T." Microelectronics Journal 26, no. 2-3 (1995): 79–97. http://dx.doi.org/10.1016/0026-2692(95)98915-e.

Full text

APA, Harvard, Vancouver, ISO, and other styles

4

Baltrūnienė, Vaida, Ieva Rinkūnaitė, Julius Bogomolovas, et al. "The Role of Cardiac T-Cadherin in the Indicating Heart Failure Severity of Patients with Non-Ischemic Dilated Cardiomyopathy." Medicina 56, no. 1 (2020): 27. http://dx.doi.org/10.3390/medicina56010027.

Full text

Abstract:

Background and objectives: T-cadherin (T-cad) is one of the adiponectin receptors abundantly expressed in the heart and blood vessels. Experimental studies show that T-cad sequesters adiponectin in cardiovascular tissues and is critical for adiponectin-mediated cardio-protection. However, there are no data connecting cardiac T-cad levels with human chronic heart failure (HF). The aim of this study was to assess whether myocardial T-cad concentration is associated with chronic HF severity and whether the T-cad levels in human heart tissue might predict outcomes in patients with non-ischemic dil

APA, Harvard, Vancouver, ISO, and other styles

5

Fukuda, Shiro, Shunbun Kita, Kazuya Miyashita, et al. "Identification and Clinical Associations of 3 Forms of Circulating T-cadherin in Human Serum." Journal of Clinical Endocrinology & Metabolism 106, no. 5 (2021): 1333–44. http://dx.doi.org/10.1210/clinem/dgab066.

Full text

Abstract:

Abstract Context T-cadherin (T-cad) is a glycosylphosphatidylinositol (GPI)-anchored cadherin that mediates adiponectin to induce exosome biogenesis and secretion, protect cardiovascular tissues, promote muscle regeneration, and stimulate therapeutic heart protection by transplanted mesenchymal stem cells. CDH13, the gene locus of T-cad, affects plasma adiponectin levels most strongly, in addition to affecting cardiovascular disease risk and glucose homeostasis. Recently, it has been suggested that T-cad exists in human serum, although the details are still unclear. Objective To validate the e

APA, Harvard, Vancouver, ISO, and other styles

6

Philippova, Maria, Danila Ivanov, Manjunath B. Joshi, et al. "Identification of Proteins Associating with Glycosylphosphatidylinositol- Anchored T-Cadherin on the Surface of Vascular Endothelial Cells: Role for Grp78/BiP in T-Cadherin-Dependent Cell Survival." Molecular and Cellular Biology 28, no. 12 (2008): 4004–17. http://dx.doi.org/10.1128/mcb.00157-08.

Full text

Abstract:

ABSTRACT There is scant knowledge regarding how cell surface lipid-anchored T-cadherin (T-cad) transmits signals through the plasma membrane to its intracellular targets. This study aimed to identify membrane proteins colocalizing with atypical glycosylphosphatidylinositol (GPI)-anchored T-cad on the surface of endothelial cells and to evaluate their role as signaling adaptors for T-cad. Application of coimmunoprecipitation from endothelial cells expressing c-myc-tagged T-cad and high-performance liquid chromatography revealed putative association of T-cad with the following proteins: glucose-

APA, Harvard, Vancouver, ISO, and other styles

7

Khanna, Roopali, Avinash Bansal, Sudeep Kumar, et al. "Association between Endogenous Sex Hormones and Coronary Artery Disease in Postmenopausal Women." Indian Journal of Cardiovascular Disease in Women - WINCARS 06, no. 03 (2021): 168–73. http://dx.doi.org/10.1055/s-0041-1736250.

Full text

Abstract:

Background Incidence of coronary artery disease (CAD) increases significantly in postmenopausal women, which is assumed to be an imbalance between serum androgen and estrogen levels. However, studies assessing serum sex hormones and CAD are few and have shown conflicting results. Objective To compare serum sex hormone levels and traditional risk factors among postmenopausal women with angiographically proven CAD and without CAD. Method The study included consecutive postmenopausal women undergoing coronary angiography in our institute from May 2016 to June 2017. The clinical and coronary angio

APA, Harvard, Vancouver, ISO, and other styles

8

Labay, V. A., and J. Bornemann. "CAD of T-septum waveguide evanescent-mode filters." IEEE Transactions on Microwave Theory and Techniques 41, no. 4 (1993): 731–33. http://dx.doi.org/10.1109/22.231675.

Full text

APA, Harvard, Vancouver, ISO, and other styles

9

Foster, Jeffrey C. "CAE/CAD at AT&T: An Introduction." AT&T Technical Journal 70, no. 1 (1991): 2–8. http://dx.doi.org/10.1002/j.1538-7305.1991.tb00493.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

10

Abu-Amero, Khaled K., Carol A. Wyngaard, and Nduna Dzimiri. "Prevalence and Role of Methylenetetrahydrofolate Reductase 677 C→T and 1298 A→C Polymorphisms in Coronary Artery Disease in Arabs." Archives of Pathology & Laboratory Medicine 127, no. 10 (2003): 1349–52. http://dx.doi.org/10.5858/2003-127-1349-paromr.

Full text

Abstract:

Abstract Context.—Previous studies reported an association of 677 C→T and 1298 A→C methylenetetrahydrofolate reductase (MTHFR) variants with coronary artery disease (CAD). No previous studies concerning the prevalence of these 2 MTHFR variants or their possible association with CAD in Arabs are currently available in the literature. Objective.—To determine the prevalence of MTHFR variants and their potential relevance to CAD among Arabs. Design.—We used polymerase chain reaction and restriction enzyme digestion to determine the prevalence of these 2 MTHFR polymorphisms in 625 healthy blood don

APA, Harvard, Vancouver, ISO, and other styles

More sources

We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!