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Relevant bibliographies by topics / T-cell subpopulations

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Journal articles

Academic literature on the topic 'T-cell subpopulations'

Author: Grafiati

Published: 10 March 2023

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Journal articles on the topic "T-cell subpopulations"

1

Tian, Yamin, Seiichiro Kobayashi, Nobuhiro Ohno, et al. "Leukemic T Cells Are Specifically Enriched In a Unique CD3dimCD7low Subpopulation of CD4+ T Cells In Acute-Type Adult T Cell Leukemia." Blood 116, no. 21 (2010): 4144. http://dx.doi.org/10.1182/blood.v116.21.4144.4144.

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Abstract Abstract 4144 [Background] Adult T-cell leukemia (ATL) is a malignant disorder caused by human T-cell leukemia virus type I (HTLV-I). Morphological discrimination of leukemic cells from non-leukemic T cells is often difficult in ATL since ATL cells reveal morphological diversity except for typical “flower cells”. Although a study using CD3 gating in flow cytometry reported that ATL cells were distinguishable as a CD3low population from normal lymphocytes, these cells were not well characterised as ATL cells. Considering that defective expression of CD7 as well as CD3 is common in ATL

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2

Phillips, S. M., D. Walker, S. K. Abdel-Hafez, et al. "The immune response to Schistosoma mansoni infections in inbred rats. VI. Regulation by T cell subpopulations." Journal of Immunology 139, no. 8 (1987): 2781–87. http://dx.doi.org/10.4049/jimmunol.139.8.2781.

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Abstract These studies assess the roles of subpopulations of T lymphocytes in inducing and modulating resistance to Schistosoma mansoni. CDF rats were depleted of RT 7.1+ (anti-Pan-T), W3/25+ (anti-T helper/inducer), or OX8+ (anti-T suppressor) cells by the in vivo administration of monoclonal antibodies (mAb). The development of parasites and immunity to challenge by S. mansoni were compared with results in undepleted normal and congenitally athymic rats. Discrete subpopulations of T lymphocytes were adoptively transferred to ascertain effects upon parasite development and the protective immu

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3

Kanof, Marjorie E. "Purification of T Cell Subpopulations." Current Protocols in Immunology 00, no. 1 (1991): 7.3.1–7.3.5. http://dx.doi.org/10.1002/0471142735.im0703s00.

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4

Kubick, Norwin, Patrick C. Henckell Flournoy, Ana-Maria Enciu, Gina Manda, and Michel-Edwar Mickael. "Drugs Modulating CD4+ T Cells Blood–Brain Barrier Interaction in Alzheimer’s Disease." Pharmaceutics 12, no. 9 (2020): 880. http://dx.doi.org/10.3390/pharmaceutics12090880.

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The effect of Alzheimer’s disease (AD) medications on CD4+ T cells homing has not been thoroughly investigated. CD4+ T cells could both exacerbate and reduce AD symptoms based on their infiltrating subpopulations. Proinflammatory subpopulations such as Th1 and Th17 constitute a major source of proinflammatory cytokines that reduce endothelial integrity and stimulate astrocytes, resulting in the production of amyloid β. Anti-inflammatory subpopulations such as Th2 and Tregs reduce inflammation and regulate the function of Th1 and Th17. Recently, pathogenic Th17 has been shown to have a superior

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5

Leonhardt, U., U. Wagner, M. Werner, and L. Engelmann. "T-cell-subpopulations in septic patients." Critical Care 5, Suppl 1 (2001): P059. http://dx.doi.org/10.1186/cc1127.

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6

Ferrara, Andrea, Marvin M. McMillen, and Garth H. Ballantyne. "T-cell subpopulations and colorectal cancer." Diseases of the Colon & Rectum 33, no. 5 (1990): 367–69. http://dx.doi.org/10.1007/bf02156259.

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7

Abramova, A. V., I. V. Galtseva, E. A. Mikhailova, et al. "Oligoclonality and subpopulation structure of bone marrow T-cells in patients with aplastic anaemia." Russian journal of hematology and transfusiology 65, no. 4 (2020): 417–30. http://dx.doi.org/10.35754/0234-5730-2020-65-4-417-430.

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Introduction. The main pathogenetic mechanism of the development of aplastic anemia (AA) is a violation of the immune regulation of hematopoiesis.Aim: to study of the subpopulation composition of T-cells and the repertoire of the T-cell receptor in AA patients.Patients and Methods. The study included AA patients (n = 40) without prior immunosuppressive therapy in 2018–2020. The T-cell subpopulation structure and T-cell receptor Vβ-family (TCR-Vβ) oligoclonality were studied in samples of bone marrow using ﬂow cytometry.Results. We report characteristic properties of T-cell subpopulations of bo

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8

Hafler, D. A., D. A. Fox, D. Benjamin, and H. L. Weiner. "Antigen reactive memory T cells are defined by Ta1." Journal of Immunology 137, no. 2 (1986): 414–18. http://dx.doi.org/10.4049/jimmunol.137.2.414.

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Abstract Ta1 is a 105,000 dalton protein that is weakly expressed on a small fraction of resting human peripheral blood T cells but strongly expressed in vitro on T cell clones and a substantial proportion of activated T cells. Unlike receptors for growth factors such as IL 2, the Ta1 antigen is present on T cell lines and clones irrespective of cell cycle. The function of Ta1 was investigated after separation of T lymphocytes into Ta1-enriched and Ta1-depleted subpopulations that were obtained from normal human subjects. Although Ta1-enriched T cells constitute only 10 to 15% of the E rosette

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9

SKÖLD, RYTTER, IVARS, and CARDELL. "Characterization of Subpopulations of T-Cell Receptor Intermediate (TCRint) T Cells." Scandinavian Journal of Immunology 49, no. 6 (1999): 611–19. http://dx.doi.org/10.1046/j.1365-3083.1999.00535.x.

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10

Bertrand, F. E., L. G. Billips, G. L. Gartland, H. Kubagawa, and H. W. Schroeder. "The J chain gene is transcribed during B and T lymphopoiesis in humans." Journal of Immunology 156, no. 11 (1996): 4240–44. http://dx.doi.org/10.4049/jimmunol.156.11.4240.

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Abstract In mice and chickens, J chain appears to be expressed only in activated B cells and plasma cells. In humans, studies based mainly on transformed cells suggest that J chain expression may initiate during earlier stages in B lineage differentiation. In the present study, we isolated a series of hematopoietic subpopulations from human fetal and adult tissues by immunofluorescence cell sorting and examined each subpopulation for J chain expression by reverse transcriptase-PCR. In fetal and adult bone marrow, J chain transcripts were detected at all stages of B lineage differentiation, inc

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