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Journal articles on the topic 'T cell'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2025

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1

Ohshima, Kôichi, Junji Suzumiya, and Masahiro Kikuchi. "T cell rich B cell lymphoma." Journal of the Japan Society of the Reticuloendothelial System 36, no. 5-6 (1996): 391–93. http://dx.doi.org/10.3960/jslrt1961.36.391.

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2

Y, Elshimali. "Chimeric Antigen Receptor T-Cell Therapy (Car T-Cells) in Solid Tumors, Resistance and Success." Bioequivalence & Bioavailability International Journal 6, no. 1 (2022): 1–6. http://dx.doi.org/10.23880/beba-16000163.

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CARs are chimeric synthetic antigen receptors that can be introduced into an immune cell to retarget its cytotoxicity toward a specific tumor antigen. CAR T-cells immunotherapy demonstrated significant success in the management of hematologic malignancies. Nevertheless, limited studies are present regarding its efficacy in solid and refractory tumors. It is well known that the major concerns regarding this technique include the risk of relapse and the resistance of tumor cells, in addition to high expenses and limited affordability. Several factors play a crucial role in improving the efficacy

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3

CPK, Cheung. "T Cells, Endothelial Cell, Metabolism; A Therapeutic Target in Chronic Inflammation." Open Access Journal of Microbiology & Biotechnology 5, no. 2 (2020): 1–6. http://dx.doi.org/10.23880/oajmb-16000163.

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The role of metabolic reprogramming in the coordination of the immune response has gained increasing consideration in recent years. Indeed, it has become clear that changes in the metabolic status of immune cells can alter their functional properties. During inflammation, stimulated immune cells need to generate sufficient energy and biomolecules to support growth, proliferation and effector functions, including migration, cytotoxicity and production of cytokines. Thus, immune cells switch from oxidative phosphorylation to aerobic glycolysis, increasing their glucose uptake. A similar metaboli

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4

Rimpo, Kenji, Yumiko Kagawa, and Tetsushi Yamagami. "T-cell-rich B-cell lymphoma in a dog." Journal of Japan Veterinary Cancer Society 4, no. 1 (2013): 1–5. http://dx.doi.org/10.12951/jvcs.2012-001.

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5

Toner, Keri, Catherine M. Bollard, and Hema Dave. "T-cell therapies for T-cell lymphoma." Cytotherapy 21, no. 9 (2019): 935–42. http://dx.doi.org/10.1016/j.jcyt.2019.04.058.

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6

Fitch, F. W. "T-cell clones and T-cell receptors." Microbiological Reviews 50, no. 1 (1986): 50–69. http://dx.doi.org/10.1128/mmbr.50.1.50-69.1986.

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7

Fitch, F. W. "T-cell clones and T-cell receptors." Microbiological Reviews 50, no. 1 (1986): 50–69. http://dx.doi.org/10.1128/mr.50.1.50-69.1986.

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8

Mohapatra, Manisha, and Yerraguntala Subramanya Sarma. "T-Cell/ Histiocyte-Rich Large B-Cell Lymphoma of Posterior Mediastinum." Annals of Pathology and Laboratory Medicine 6, no. 6 (2019): C63–66. http://dx.doi.org/10.21276/apalm.2389.

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9

Chinnikatti, Shravana kumar, Soumya shravan, H. N. Asikur Rahaman, and Shraavya Shraavya. "New Treatments for Synovial Cell Sarcoma with Genetically Modified T-Cell?" Cancer Research and Cellular Therapeutics 6, no. 3 (2022): 01–02. http://dx.doi.org/10.31579/2640-1053/113.

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Synovial cell sarcoma is rare but very aggressive tumour if not treated early, due to the painless nature of this tumour patients normally come in late and advances stage, can occur in bones, muscle cells, cartilages, ligaments and de-novo from pleuripotent stem cells from asnywhere in the body but most commonly arm, leg, or foot, and near joints such as the wrist or ankle and possibly from any joints in the body, even from soft tissues of lung and abdomen, the other name for this tumour is called malignant synovioma.The 5 year survival after the effective primary treatment is 30-75% and the s

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10

Udhayakumar, Venkatachalam, Bondada Subbarao, Aruna Seth, Mitzi Nagarkatti, and Prakash S. Nagarkatti. "Impaired autoreactive T cell-induced T cell-T cell interaction in aged mice." Cellular Immunology 116, no. 2 (1988): 299–307. http://dx.doi.org/10.1016/0008-8749(88)90232-8.

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11

Ramming, Andreas, Katja Thümmler, Hendrik Schulze-Koops, and Alla Skapenko. "Homotypic T-cell/T-cell interaction induces T-cell activation, proliferation, and differentiation." Human Immunology 70, no. 11 (2009): 873–81. http://dx.doi.org/10.1016/j.humimm.2009.08.003.

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12

Kotulová, Jana, (Kufova) Zuzana Chyra, Zhongfei Tao, et al. "Impact of T cell characteristics on CAR-T cell therapy in hematological malignancies." Blood Cancer Journal 14, no. 1 (2024): 1–10. https://doi.org/10.1038/s41408-024-01193-6.

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13

Russi, Abigail E., Margaret E. Walker-Caulfield, Yong Guo, Claudia F. Lucchinetti, and Melissa A. Brown. "Meningeal mast cell-T cell crosstalk regulates T cell encephalitogenicity." Journal of Autoimmunity 73 (September 2016): 100–110. http://dx.doi.org/10.1016/j.jaut.2016.06.015.

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14

Jamal, Iffat. "Hepatosplenic T Cell Lymphoma: A Rare Disease." Cytology & Histology International Journal 4, no. 1 (2020): 1–3. http://dx.doi.org/10.23880/chij-16000118.

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Hepatosplenic T cell lymphoma (HSTCL) is an uncommon neoplasm comprising 5% of peripheral T-cell lymphomas. We report an uncommon case of Peripheral T-cell lymphoma that is characterized by primary extranodal disease with malignant T cell proliferation in spleen, liver and bone marrow. 19 year old male patient presented with fever, weakness and pain abdomen for 2 months. On clinical examination he was pale and had massive hepatosplenomegaly. The diagnosis was quite challenging as thorough clinical, hematological and immunophenotypic correlation was required.

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15

KA, Awadhesh. "Basics of T Cell Development and Activation." Journal of Embryology & Stem Cell Research 2, no. 1 (2018): 1–4. http://dx.doi.org/10.23880/jes-16000103.

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16

Khanolkar, Aaruni, Michael J. Fuller, and Allan J. Zajac. "CD4 T Cell-Dependent CD8 T Cell Maturation." Journal of Immunology 172, no. 5 (2004): 2834–44. http://dx.doi.org/10.4049/jimmunol.172.5.2834.

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17

Boismenu, Richard, and Wendy L. Havran. "T-Cell Development: T-cell lineage commitment revisited." Current Biology 5, no. 8 (1995): 829–31. http://dx.doi.org/10.1016/s0960-9822(95)00164-3.

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18

Lindsey, JW, RH Kerman, and JS Wolinsky. "T cell-T cell activation in multiple sclerosis." Multiple Sclerosis Journal 3, no. 4 (1997): 238–42. http://dx.doi.org/10.1177/135245859700300404.

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Activated T cells are able to stimulate proliferation in resting T cells through an antigen non-specific mechanism. The in vivo usefulness of this T cell-T cell activation is unclear, but it may serve to amplify immune responses. T cell-T cell activation could be involved in the well-documented occurrence of multiple sclerosis (MS) exacerbations following viral infections. Excessive activation via this pathway could also be a factor in the etiology of MS. We tested the hypothesis that excessive T cell-T cell activation occurs in MS patients using in vitro proliferation assays comparing T cells

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19

Lechler, Robert, Jian-Guo Chai, Federica Marelli-Berg, and Giovanna Lombardi. "T–cell anergy and peripheral T–cell tolerance." Philosophical Transactions of the Royal Society of London. Series B: Biological Sciences 356, no. 1409 (2001): 625–37. http://dx.doi.org/10.1098/rstb.2001.0844.

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The discovery that T–cell recognition of antigen can have distinct outcomes has advanced understanding of peripheral T–cell tolerance, and opened up new possibilities in immunotherapy. Anergy is one such outcome, and results from partial T–cell activation. This can arise either due to subtle alteration of the antigen, leading to a lower–affinity cognate interaction, or due to a lack of adequate co–stimulation. The signalling defects in anergic T cells are partially defined, and suggest that T–cell receptor (TCR) proximal, as well as downstream defects negatively regulate the anergic T cell's a

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20

Gill, Ronald G. "T-cell-T-cell collaboration in allograft responses." Current Opinion in Immunology 5, no. 5 (1993): 782–87. http://dx.doi.org/10.1016/0952-7915(93)90137-h.

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21

Testa, Ugo, Patrizia Chiusolo, Elvira Pelosi, Germana Castelli, and Giuseppe Leone. "CAR-T CELL THERAPY FOR T-CELL MALIGNANCIES." Mediterranean Journal of Hematology and Infectious Diseases 16, no. 1 (2024): e2024031. http://dx.doi.org/10.4084/mjhid.2024.031.

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Chimeric antigen receptor T-cell (CAR-T) therapy has revolutionized the treatment of B-cell lymphoid neoplasia and, in some instances, improved disease outcomes. Thus, six FDA-approved commercial CAR-T cell products that target antigens preferentially expressed on malignant B-cells or plasma cells have been introduced in the therapy of B-cell lymphomas, B-ALLs and multiple myeloma. These therapeutic successes have triggered the application of CAR-T cell therapy to other hematologic tumors, including T-cell malignancies. However, the success of CAR-T cell therapies in T-cell neoplasms was

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22

Mitchell, Emily, and George S. Vassiliou. "T-Cell Cancer after CAR T-Cell Therapy." New England Journal of Medicine 390, no. 22 (2024): 2120–21. http://dx.doi.org/10.1056/nejme2405538.

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23

Yu, Guang, Hongyu Luo, Yulian Wu, and Jiangping Wu. "EphrinB1 Is Essential in T-cell-T-cell Co-operation during T-cell Activation." Journal of Biological Chemistry 279, no. 53 (2004): 55531–39. http://dx.doi.org/10.1074/jbc.m410814200.

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24

Matsumoto, Yosuke, Hisao Nagoshi, Mihoko Yoshida, et al. "Expression of Master Regulators of T-cell, Helper T-cell and Follicular Helper T-cell Differentiation in Angioimmunoblastic T-cell Lymphoma." Internal Medicine 56, no. 21 (2017): 2851–56. http://dx.doi.org/10.2169/internalmedicine.8570-16.

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25

Lamers, Cor H. J., Sabine van Steenbergen-Langeveld, Mandy van Brakel, et al. "T Cell Receptor-Engineered T Cells to Treat Solid Tumors: T Cell Processing Toward Optimal T Cell Fitness." Human Gene Therapy Methods 25, no. 6 (2014): 345–57. http://dx.doi.org/10.1089/hgtb.2014.051.

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26

Manolios, Nicholas, Simon Collier, Jude Taylor, John Pollard, Leonard C. Harrison, and Veronica Bender. "T-cell antigen receptor transmembrane peptides modulate T-cell function and T cell-mediated disease." Nature Medicine 3, no. 1 (1997): 84–88. http://dx.doi.org/10.1038/nm0197-84.

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27

Salzmann, Michael, and Martin F. Bachmann. "The role of T cell receptor dimerization for T cell antagonism and T cell specificity." Molecular Immunology 35, no. 5 (1998): 271–77. http://dx.doi.org/10.1016/s0161-5890(98)00035-2.

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28

Hashimoto, Y., and K. J. Blank. "T cell receptor genes and T cell development in virus-transformed early T cell lines." Journal of Immunology 144, no. 4 (1990): 1518–25. http://dx.doi.org/10.4049/jimmunol.144.4.1518.

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Abstract We have derived T cell lines from mice inoculated with Gross leukemia virus, which appear to represent early T cell developmental stages and to reflect normal T cell development. These cell lines may provide a breakthrough in the study of T cell development as Abelson transformants have done for the study of B cell development. Analysis of the TCR gene expression in these cell lines reveals that the sequence of rearrangement and expression of each TCR gene is not strictly ordered. Expression of RNA for the TCR alpha and -beta genes appears to be coordinated with rearrangement at the a

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29

Liu, Qi, Wenxuan Cai, Wei Zhang, and Yi Li. "Cancer immunotherapy using T-cell receptor engineered T cell." Annals of Blood 5 (March 2020): 5. http://dx.doi.org/10.21037/aob.2020.02.02.

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30

Bruneau, Julie, Danielle Canioni, Amédée Renand, et al. "Regulatory T-Cell Depletion in Angioimmunoblastic T-Cell Lymphoma." American Journal of Pathology 177, no. 2 (2010): 570–74. http://dx.doi.org/10.2353/ajpath.2010.100150.

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31

Rodríguez-Pinilla, Socorro María, Lidia Atienza, Cristina Murillo, et al. "Peripheral T-cell Lymphoma With Follicular T-cell Markers." American Journal of Surgical Pathology 32, no. 12 (2008): 1787–99. http://dx.doi.org/10.1097/pas.0b013e31817f123e.

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32

Davis, Mark M., and Pamela J. Bjorkman. "T-cell antigen receptor genes and T-cell recognition." Nature 334, no. 6181 (1988): 395–402. http://dx.doi.org/10.1038/334395a0.

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33

Davis, Mark M., and Pamela J. Bjorkman. "T-cell antigen receptor genes and T-cell recognition." Nature 335, no. 6192 (1988): 744. http://dx.doi.org/10.1038/335744b0.

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34

Mori, Naoyoshi, Kuniyuki Oka, Yasuhiro Yoda, Tsukasa Abe, and Mizu Kojima. "T-Cell Receptor Expression in the T-Cell Malignancies." American Journal of Clinical Pathology 93, no. 4 (1990): 495–501. http://dx.doi.org/10.1093/ajcp/93.4.495.

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35

DiPersio, John F., Karl Staser, and Matthew Cooper. "Immunotherapy for T-Cell ALL and T-Cell NHL." Clinical Lymphoma Myeloma and Leukemia 20 (September 2020): S56—S58. http://dx.doi.org/10.1016/s2152-2650(20)30462-6.

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36

Moltedo, Bruno, Saskia Hemmers, and Alexander Y. Rudensky. "Regulatory T Cell Ablation Causes Acute T Cell Lymphopenia." PLoS ONE 9, no. 1 (2014): e86762. http://dx.doi.org/10.1371/journal.pone.0086762.

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37

Marelli-Berg, F. M., M. Alvarez-Iglesias, K. Prodromidou, et al. "T cell receptor engagement can influence T cell motility." Transplantation Proceedings 33, no. 1-2 (2001): 312–13. http://dx.doi.org/10.1016/s0041-1345(00)02265-x.

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38

Newrzela, S., N. Al-Ghaili, T. Heinrich, et al. "T-cell receptor diversity prevents T-cell lymphoma development." Leukemia 26, no. 12 (2012): 2499–507. http://dx.doi.org/10.1038/leu.2012.142.

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39

Yanagi, Yusuke. "T-cell receptor and T-cell-resistant virus variants." Current Opinion in Immunology 3, no. 4 (1991): 460–64. http://dx.doi.org/10.1016/0952-7915(91)90003-j.

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40

Mackall, Crystal L., Frances T. Hakim, and Ronald E. Gress. "Restoration of T-cell homeostasis after T-cell depletion." Seminars in Immunology 9, no. 6 (1997): 339–46. http://dx.doi.org/10.1006/smim.1997.0091.

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41

Bajaj, Anubha. "Ripened and Revamped-Adult T Cell Leukaemia/ Lymphoma." Clinical Pathology & Research Journal 7, no. 1 (2023): 1–6. http://dx.doi.org/10.23880/cprj-16000151.

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Adult T cell leukaemia/ lymphoma is an aggressive T cell malignant disorder comprised of significantly pleomorphic, miniature to intermediate, mature CD4+ T cells. Factors such as demographic distribution within HTLV1 endemic zones, hypercalcemia, cutaneous lesions and a leukemic phase contribute to disease emergence. Adult T cell leukaemia/ lymphoma incriminates peripheral blood, lymph nodes, spleen, cutaneous surfaces, pulmonary or hepatic parenchyma, gastrointestinal tract and central nervous system. Bone marrow may demonstrate diffuse, interstitial or sinusoidal pattern of neoplastic occur

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42

Soroosh, Pejman, Shouji Ine, Kazuo Sugamura, and Naoto Ishii. "OX40-OX40 Ligand Interaction through T Cell-T Cell Contact Contributes to CD4 T Cell Longevity." Journal of Immunology 176, no. 10 (2006): 5975–87. http://dx.doi.org/10.4049/jimmunol.176.10.5975.

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43

Penaloza-MacMaster, Pablo, David Masopust, and Rafi Ahmed. "T-cell reconstitution without T-cell immunopathology in two models of T-cell-mediated tissue destruction." Immunology 128, no. 2 (2009): 164–71. http://dx.doi.org/10.1111/j.1365-2567.2009.03080.x.

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44

Wucherpfennig, K. W., P. Hollsberg, J. H. Richardson, D. Benjamin, and D. A. Hafler. "T-cell activation by autologous human T-cell leukemia virus type I-infected T-cell clones." Proceedings of the National Academy of Sciences 89, no. 6 (1992): 2110–14. http://dx.doi.org/10.1073/pnas.89.6.2110.

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45

Ge, Q., D. Palliser, H. N. Eisen, and J. Chen. "Homeostatic T cell proliferation in a T cell-dendritic cell coculture system." Proceedings of the National Academy of Sciences 99, no. 5 (2002): 2983–88. http://dx.doi.org/10.1073/pnas.052714199.

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46

Ehrenstein, M. R., and B. Hahn. "T cell—B cell interactions." Lupus 11, no. 12 (2002): 790–92. http://dx.doi.org/10.1191/0961203302lu319oa.

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47

Mekori, Yoseph A., and Dean D. Metcalfe. "Mast cell–T cell interactions." Journal of Allergy and Clinical Immunology 104, no. 3 (1999): 517–23. http://dx.doi.org/10.1016/s0091-6749(99)70316-7.

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48

Jutila, Mark A. "T cell/endothelial cell interactions." Veterinary Immunology and Immunopathology 54, no. 1-4 (1996): 105–10. http://dx.doi.org/10.1016/s0165-2427(96)05691-7.

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49

Sprent, Jonathan. "T cell–B cell collaboration." Nature Reviews Immunology 17, no. 9 (2017): 532. http://dx.doi.org/10.1038/nri.2017.62.

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50

Mitchison, N. A. "T-cell–B-cell cooperation." Nature Reviews Immunology 4, no. 4 (2004): 308–12. http://dx.doi.org/10.1038/nri1334.

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