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Relevant bibliographies by topics / Tariff on potassium chloride

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Academic literature on the topic 'Tariff on potassium chloride'

Author: Grafiati

Published: 4 June 2021

Last updated: 8 February 2022

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Journal articles on the topic "Tariff on potassium chloride"

1

&NA;. "Potassium chloride." Reactions Weekly &NA;, no. 1095 (April 2006): 23. http://dx.doi.org/10.2165/00128415-200610950-00077.

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&NA;. "Potassium chloride." Reactions Weekly &NA;, no. 1042 (March 2005): 17. http://dx.doi.org/10.2165/00128415-200510420-00048.

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&NA;. "Potassium chloride." Reactions Weekly &NA;, no. 1044-1045 (March 2005): 13. http://dx.doi.org/10.2165/00128415-200510440-00041.

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&NA;. "Potassium chloride." Reactions Weekly &NA;, no. 702 (May 1998): 10. http://dx.doi.org/10.2165/00128415-199807020-00033.

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&NA;. "Potassium chloride." Reactions Weekly &NA;, no. 480 (December 1993): 10. http://dx.doi.org/10.2165/00128415-199304800-00046.

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&NA;. "Potassium chloride." Reactions Weekly &NA;, no. 613 (August 1996): 11. http://dx.doi.org/10.2165/00128415-199606130-00032.

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&NA;. "Potassium chloride." Reactions Weekly &NA;, no. 1196 (April 2008): 32. http://dx.doi.org/10.2165/00128415-200811960-00097.

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&NA;. "Potassium chloride." Reactions Weekly &NA;, no. 1379 (November 2011): 30. http://dx.doi.org/10.2165/00128415-201113790-00114.

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&NA;. "Potassium chloride poisoning." Reactions Weekly &NA;, no. 292 (March 1990): 8. http://dx.doi.org/10.2165/00128415-199002920-00030.

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&NA;. "Potassium chloride abuse." Reactions Weekly &NA;, no. 353 (June 1991): 7. http://dx.doi.org/10.2165/00128415-199103530-00039.

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Dissertations / Theses on the topic "Tariff on potassium chloride"

1

Hettiarachchi, Samanthika Ruvinie. "Exciplex Tuning and Optical Memory Studies for Dicyanoargentate(1) and Dicyanoaurate(1) Ions Doped in Potassium Chloride Crystals Extension to Mixed Metal Gold and Silver Systems." Fogler Library, University of Maine, 2002. http://www.library.umaine.edu/theses/pdf/HettiarachchiSR2002.pdf.

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Tadayyon, Abdolsamad. "Modeling, control and measurement in continuous potassium chloride crystallizers." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape8/PQDD_0030/NQ63928.pdf.

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Imhof, Monica Y. "Salt substitution : the inhibition of potassium chloride bitter aftertaste." Thesis, Oxford Brookes University, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.363568.

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Lee, H. C. "Phospho-dependent modulation of potassium chloride co-transporter KCC2." Thesis, University College London (University of London), 2009. http://discovery.ucl.ac.uk/17998/.

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The neuronal-specific potassium chloride co-transporter 2, KCC2, is a major chloride extruder in brain. The expression of KCC2 during neuronal development is fundamental to the switch of GABAergic response from excitatory to inhibitory. Malfunction of KCC2 can cause impairment of chloride homeostasis in neurons and is implicated in neurological disorders such as epilepsy. To date the role of protein phosphorylation in the regulation of KCC2 remains elusive. In this thesis, direct phosphorylation of KCC2 by PKC and Src tyrosine kinase was shown in vitro and in cultured neurons using the radioactive isotope 32P. Single mutation of serine residue at position 940 in the intracellular domain of KCC2 (Ser940) to alanine (S940A) blocked the phosphorylation of KCC2 under PKC activation. However, tyrosine phosphorylation of KCC2 was shown to not affect Tyr1087, the putative tyrosine kinase phosphorylation site. To better understand phosphorylation of KCC2 at Ser940, a phospho-specific antibody against this residue - namely p-S940 - was developed. Interestingly, agents inhibiting PKC and phosphatases altered signal of p-S940, indicating involvement of PKC, phosphatase-1 (PP1) and phosphatase-2A (PP2A) in the regulation of Ser940 phosphorylation. In an in vitro method using p-S940, it was shown that PP1 and PP2A dephosphorylated KCC2.

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Lu, Ying. "Effects of sodium chloride salting and substitution with potassium chloride on whey expulsion of cheese." DigitalCommons@USU, 2012. https://digitalcommons.usu.edu/etd/1285.

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The rate and extent of syneresis (whey expulsion) strongly affects cheese composition and quality. During salting, curd syneresis is influenced by the combined effect of both osmotic pressure and protein hydration. Our objective is to examine how cheese composition and whey expulsion are influenced by dry salting curd at various intervals, levels, applications, and potassium chloride (KCl) substitution, or change in calcium or sodium level in test solution (i.e., whey-brine). Four sets of unsalted fresh Cheddar curds were salted with different methods, with at least 3 replicates of each set on separate days. Set A was salted with 30 g/kg NaCl over 3 applications, either 5 or 10 min apart. Set B was salted with 30, 25, and 20 g/kg NaCl over 3 applications 5 min apart. Set C was salted with 20 g/kg NaCl using 1, 2, or 3 applications. Set D received salt consisting of a 2:1 molar ratio of NaCl and KCl over 3 applications 5 min apart. Whey was collected every 5 or 10 min until 30 or 40 min after the start of salting and subsequently pressed for 3 h. Using 10-min intervals delayed whey syneresis but after pressing there was no significant influence on final cheese composition. Decreasing salt levels significantly reduced the amount of whey expelled prior to pressing and resulted in cheeses with higher moisture and slightly lower pH. Adding salt over different applications did not significantly affect cheese composition. Partial substitution with KCl did not affect the amount of whey expelled or cheese moisture composition. Salted milled Cheddar cheese curd was immersed at 22°C for 6 or 18 h in test solution, with the addition of 1, 5, 10, or 20 g/L calcium, and 15 g/L salt. After immersion, curd weight change, moisture, pH, sodium, serum calcium and total calcium levels were measured. When calcium levels in solution increased, curd moisture, pH, and weight gain decreased while serum and total calcium levels increased significantly. Similarly, unsalted milled Cheddar cheese curds were immersed at 22°C for 6 h in test solution with 30, 60, 90, or 120 g/L salt in addition to 6 g/L calcium. The salt level in solution was inversely proportional with weight change, moisture, and salt level of curd.

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Goutierre, Marie. "Contribution of the potassium / chloride cotransporter KCC2 to hippocampal rhythmopathy." Thesis, Sorbonne université, 2018. http://www.theses.fr/2018SORUS600.

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Dans le système nerveux central, la transmission inhibitrice est principalement assurée par le relargage du neurotransmetteur GABA dans la fente synaptique. La fixation du GABA sur les récepteurs GABAA induit en effet un flux entrant d’ions chlorure, résultant en une hyperpolarisation du neurone. Le maintien d’une faible concentration intraneuronale en chlore est donc essentielle à l’action inhibitrice du GABA. Dans les neurones matures, cette fonction est principalement réalisée grâce à l’activité du transporteur potassium – chlore KCC2 qui exporte en permanence les ions chlorures. Dans de nombreuses pathologies neurologiques, telles que l’épilepsie, le syndrome de Rett ou encore les douleurs neuropathiques, on observe une diminution de l’expression de KCC2. Cela conduit à une élévation du niveau de chlore intraneuronal et à une altération de la transmission GABAergique. Cet effet est supposé être à la base de nombre des symptômes observés dans les pathologies citées précédemment. Cependant, KCC2 est également fortement exprimé à proximité des synapses glutamatergiques. Sa présence influence ainsi l’efficacité de la transmission excitatrice et est nécessaire à l’expression de la potentialisation à long terme des synapses. Ces fonctions inattendues de KCC2 aux synapses excitatrice ne reposent pas sur sa fonction de transport de chlore mais plutôt sur ses interactions avec diverses protéines. Ainsi, le transporteur KCC2 possède de multiple fonctions et régule différemment les transmissions excitatrice et inhibitrice. Prédire l’effet de la perte du transporteur sur l’activité globale d’un réseau neuronal est donc compliqué. Durant ma thèse, j’ai caractérisé les effets de la suppression de KCC2 dans les cellules en grains du gyrus denté sur leurs propriétés cellulaires, synaptiques et sur l’activité du réseau hippocampique. De façon inattendue, j’ai montré que la perte de KCC2 ne s’accompagnait pas de modifications majeures de la transmission inhibitrice. En revanche, j’ai mis en évidence un nouveau mécanisme indépendant du transport de chlore par lequel KCC2 contrôle l’excitabilité des neurones et la rythmogénèse hippocampique à travers son interaction avec le canal potassique Task-3. Mes résultats prédisent que les déficits associés à une perte de KCC2 pourraient être en partie expliqués par cet effet sur l’excitabilité. Ils suggèrent également que Task-3 pourrait constituer une nouvelle cible thérapeutique dans le traitement de ces pathologies
In the CNS, synaptic release of GABA neurotransmitter is mainly responsible for fast inhibitory transmission. This is mediated by chloride flow through GABAARs. Hence, tight control of chloride homeostasis is critical for maintenance of the efficacy of GABAergic transmission. In mature neurons, this is primarily achieved by the activity of the potassium – chloride transporter KCC2 which extrudes chloride from the cells. Expression of KCC2 is compromised in numerous neurological disorders including epilepsy, Rett syndrome or neuropathic pain. Subsequent alterations of GABAergic signaling through accumulation of intraneuronal chloride are thought to underlie many of the pathological symptoms observed in these conditions. However, KCC2 is also highly expressed in the vicinity of glutamatergic synapses where it plays a major role in controling the efficacy of glutamatergic transmission and gates long-term potentiation of excitatory synapses. Remarkably, these functions did not depend on chloride transport but rather on KCC2 interaction with several protein partners. Hence, KCC2 can be classified as a moonlightning protein with multiple functions at excitatory and inhibitory synapses. This complicates predictions of the overall effect of its suppression on a neuronal network. During my PhD, I characterized the effects of KCC2 downregulation in dentate granule cells at the cellular, synaptic and network levels. Unexpectedly, lack of KCC2 did not impact steady-state GABAergic transmission. In contrast, my work shed light on a novel critical role of KCC2 in controling neuronal excitability through its interaction with the leak-potassium channel Task-3. This in turn alters hippocampal rhythmogenesis. My results thus described a novel mechanism through which KCC2 influence neuronal activity indepently of its transport function. They predict that deficits associated with KCC2 downregulation may be at least partly explained by regulation of cell excitability and point to Task-3 as a new potential therapeutic target in the treatment of these pathologies

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MacKenzie, Georgina Louise. "Control of membrane excitability by potassium and chloride leak conductances." Thesis, Imperial College London, 2011. http://hdl.handle.net/10044/1/7038.

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The permeability of the neuronal membrane to different ions determines both resting membrane potential (RMP) and input conductance. These parameters determine the cells response to synaptic input. In this thesis I have examined how the molecular properties of potassium and chloride ion channels can influence neuronal excitability in ways that have not previously been considered. For example, two‐pore domain potassium (K2P) channels open at rest to generate a persistent potassium ion efflux. In addition to its accepted role in setting the RMP, I have tested the hypothesis that this conductance is sufficient to repolarise the membrane during an action potential (AP) in the absence of voltage‐dependent potassium channels (Kv). We tested this prediction using heterologous expression of TASK3 or TREK1 K2P channels combined with conductance injection to simulate the presence of a voltage‐gated sodium conductance. These experiments demonstrated that K2P channels are sufficient to support APs during short and prolonged depolarising current pulses. The membranes permeability to chloride ions can also be affected by extrasynaptic GABAA receptors containing the delta subunit (δ‐GABAARs) that produce a tonic conductance due to their high apparent affinity for GABA. The anaesthetics Propofol and THIP are both believed to alter neuronal excitability by enhancing this persistent chloride flux. We have examined how this anaesthetic action is affected by the steady‐state ambient GABA concentrations that are believed to exist in vivo. Surprisingly, the anaesthetic enhancement of δ‐GABAARs is lost at low ambient GABA concentrations. Therefore, I would suggest that the anaesthetic potency of these drugs is affected by the resting ambient GABA concentration in a manner that has not previously been appreciated. In the current Thesis I have examined the molecular and pharmacological properties of two very different ion channel families that both generate a leak conductance, and I will present models that link the behaviour of these ion channels to their ability to modulate neuronal excitability.

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8

Cook, John William. "The effect of foliar applied fertilisers on leaf diseases of cereals." Thesis, Open University, 1998. http://oro.open.ac.uk/57740/.

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The effects of foliar applied urea and potassium chloride on the severity of leaf diseases of cereals were investigated in the laboratory, glasshouse and field between 1992 and 1995. Field studies with urea gave inconsistent results with respect to severity of Erysiphe graminis and consistently increased the leaf area affected by Septaria tritici. However, potassium chloride applied as a foliar spray consistently decreased the leaf area of wheat affected by E. graminis and S. trifid compared with equivalent applications of soil applied fertiliser. Disease control was achieved at early stem extension and after flag leaf emergence but yield responses were not detected. Laboratory investigations were undertaken to determine the mechanism by which foliar applied potassium chloride reduced the leaf area affected by E. graminis. The timing of application, within seven days pre or post inoculation, had no consistent effect on the efficacy of the fertiliser. Investigations using polyethylene glycol as a control showed that the percentage leaf area affected declined linearly as the osmotic potential of the solutions were increased. Light microscopy revealed that the germination of spores in solution and on treated leaves was reduced as the osmotic potential of the solutions were increased. Spores which did germinate developed normally but those on leaves treated with solutions of high osmotic potential rarely formed haustoria. This suggested a second mechanism acting inside the leaf. Multiple regression analysis of experimental data indicated that the inhibition of spore germination was the major response reducing the area of the leaf affected. Although the data were not conclusive it appeared that the increase in leaf water potential, following the foliar application of potassium chloride, was involved in the control of E. graminis.

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Reynolds, Annie 1978. "Over-expression of the potassium-chloride co-transporter KCC2 in developing zebrafish." Thesis, McGill University, 2006. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=98778.

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In embryonic neurons, the intracellular chloride concentration is elevated, making GABA and glycine depolarizing. Later in development, coincident with neuronal maturation, the extruding potassium-chloride co-transporter KCC2 is expressed. It reverses the chloride gradient, rendering it hyperpolarizing. Early depolarization is assumed to play trophic roles during nervous system development. I thus decided to investigate the effects of the depolarizing chloride gradient on development in vivo in the zebrafish embryo. I first determined the temporal pattern of KCC2 expression in zebrafish and found it was absent in the embryo. I then over-expressed wild-type, gain-of-function and loss-of-function variants of human KCC2, using GFP-tagged constructs for detection purposes. Over-expression of functional hKCC2 perturbed the morphology and motor behaviours of the embryos. At the cellular level, KCC2 impaired axonal growth and affected the neuronal populations in the brain, hindbrain and spinal cord. This suggests the depolarizing effects of glycine are critical for neurogenesis.

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Mann, Ruth Louise. "Suppression of Septoria tritici by foliar applied potassium chloride on winter wheat." Thesis, University of Wolverhampton, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.322183.

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The effect of foliar applied potassium chloride on Septoria tritici on winter wheat was quantified and possible modes of action investigated by in vitro, glasshouse, controlled environment and field experiments between 1996 and 1999. In vitro experiments showed that as the concentration of potassium chloride increased, mycelial growth and germination of conidia decreased (EO sos of 1.36M and 0.7M, respectively). One glasshouse and two field experiments showed a significant reduction in the leaf area affected by S. trttict after foliar application of potassium chloride compared to untreated controls. In general, application of potassium chloride reduced the leaf area affected by S. tritici by 20 -.40%. However, a significant yield increase was not observed. Potassium chloride applied to the lower leaves of winter wheat did not confer systemic acquired resistance against S. tritici on the upper leaves. Inhibition of conidial germination, on leaf surfaces by potassium chloride was observed. Similar inhibition was observed when polyethylene glycol, an inert osmoticum, was applied at the same calculated osmotic potential. During field experiments there was no significant difference in the leaf area affected by S. tritici in plots treated with potassium chloride or polyethylene glycol, although both significantly reduced the leaf area affected compared to untreated control plots. Therefore, the principal mode of action of potassium chloride was proposed to be as a result of adverse osmotic conditions caused by the salt on leaf surfaces. However, the addition of a range of adjuvants to potassium chloride did not increase the efficacy of S. tritici control in glasshouse studies. The results from this study show that potassium chloride, when applied to foliage of winter wheat can reduce the leaf area affected by S. tritici and it is proposed that this reduction was by adverse osmotic conditions caused by the salt on leaf surfaces.

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Books on the topic "Tariff on potassium chloride"

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Potassium chloride from Canada: Determination of the Commission in investigation no. 731-TA-374 (preliminary) under the Tariff Act of 1930, together with the information obtained in the investigation. Washington, DC: U.S. International Trade Commission, 1987.

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United States International Trade Commission. Potassium chloride from the U.S.S.R.: Determination of the Commission in investigation no. 731-TA-187 (final) under the Tariff Act of 1930, together with the information obtained in the investigation. Washington, DC: U.S. International Trade Commission, 1985.

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Kafkafi, U. Potassium and chloride in crops and soils: The role of potassium chloride fertilizer in crop nutrition. Basel, Switzerland: International Potash Institute, 2001.

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Imhof, Monica Y. Salt substitution: The inhibition of potassium chloride bitter aftertaste. Oxford: Oxford Brookes University, 1997.

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1936-, Lehr Jay H., and McEachern Rod J. 1958-, eds. Water softening with potassium chloride: Process, health, and environmental benefits. Hoboken, NJ: Wiley, 2009.

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Shenassa, Reyhaneh. Chloride and potassium control in closed Kraft mill liquor cycles. Ottawa: National Library of Canada, 1995.

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Mann, Ruth Louise. Suppression of Septoria Tritici by foliar applied potassium chloride in winter wheat. Wolverhampton: University of Wolverhampton, 1999.

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Papadimitropoulos, Emmanuel A. Post consolidation behaviour of two crystalline materials, sodium chloride and potassium bromide. Ottawa: National Library of Canada, 1990.

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Schmok, Klaus. Beitrag zur Modellierung des Massenkristallisationsprozesses unter Einschluss der Agglomeration. Leipzig: Deutscher Verlag für Grundstoffindustrie, 1987.

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Johannes, Fabricius. Geochemical investigation of potassium-magnesium chloride mineralization of Zechstein 2 salt, Mors Dome, Denmark: Microthermometry on solid inclusions in quartz crystals. København: I kommission hos C.A. Reitzels forlag, 1987.

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Book chapters on the topic "Tariff on potassium chloride"

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Bährle-Rapp, Marina. "Potassium Chloride." In Springer Lexikon Kosmetik und Körperpflege, 443. Berlin, Heidelberg: Springer Berlin Heidelberg, 2007. http://dx.doi.org/10.1007/978-3-540-71095-0_8276.

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Preuss, Harry G., and Dallas L. Clouatre. "Sodium, Chloride, and Potassium." In Present Knowledge in Nutrition, 475–92. Oxford, UK: Wiley-Blackwell, 2012. http://dx.doi.org/10.1002/9781119946045.ch31.

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Knox, Alan J. "Sodium/Potassium/Chloride Co-Transport." In Airways Smooth Muscle: Peptide Receptors, Ion Channels and Signal Transduction, 217–32. Basel: Birkhäuser Basel, 1995. http://dx.doi.org/10.1007/978-3-0348-7362-8_10.

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Fusch, Christoph, and Frank Jochum. "Water, Sodium, Potassium and Chloride." In Nutritional Care of Preterm Infants, 99–120. Basel: S. KARGER AG, 2014. http://dx.doi.org/10.1159/000358461.

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Hüttner, W. "175 ClK X 1Σ+ Potassium chloride." In Diamagnetic Diatomic Molecules. Part 1, 242–43. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-540-69954-5_177.

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Dharmarajan, Kumar, and Kenneth L. Minaker. "Water, Potassium, Sodium, and Chloride in Nutrition." In Geriatric Gastroenterology, 145–52. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4419-1623-5_16.

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Dharmarajan, Kumar. "Water, Potassium, Sodium, and Chloride in Nutrition." In Geriatric Gastroenterology, 1–16. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-319-90761-1_94-1.

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Dharmarajan, Kumar. "Water, Potassium, Sodium, and Chloride in Nutrition." In Geriatric Gastroenterology, 539–54. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-30192-7_94.

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Parker, Kenneth R., and R. C. von Borstel. "Antimutagenesis in Yeast by Sodium Chloride, Potassium Chloride, and Sodium Saccharin." In Antimutagenesis and Anticarcinogenesis Mechanisms II, 367–71. Boston, MA: Springer US, 1990. http://dx.doi.org/10.1007/978-1-4615-9561-8_34.

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Winkelmann, J. "Diffusion of oxygen (1); water (2); potassium chloride (3)." In Gases in Gases, Liquids and their Mixtures, 2313–14. Berlin, Heidelberg: Springer Berlin Heidelberg, 2007. http://dx.doi.org/10.1007/978-3-540-49718-9_1799.

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Conference papers on the topic "Tariff on potassium chloride"

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Shkuratov, S. I., J. Baird, V. G. Antipov, E. F. Talantsev, and L. L. Altgilbers. "Conductivity of explosively shocked potassium chloride." In 2009 IEEE Pulsed Power Conference (PPC). IEEE, 2009. http://dx.doi.org/10.1109/ppc.2009.5386297.

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Jaiswal, Ratan Lal, Brijesh Kumar Pandey, and Sachin. "Temperature dependence of elastic properties of potassium chloride (KCl)." In ADVANCES IN BASIC SCIENCE (ICABS 2019). AIP Publishing, 2019. http://dx.doi.org/10.1063/1.5122525.

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Zaretsky, E. "Spatial Evolution of Three-Wave Structure in Shocked Potassium Chloride." In Shock Compression of Condensed Matter - 2001: 12th APS Topical Conference. AIP, 2002. http://dx.doi.org/10.1063/1.1483519.

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Golkov, R. "Impact Response of Single Crystal Potassium Chloride at Elevated Temperatures." In SHOCK COMPRESSION OF CONDENSED MATTER - 2003: Proceedings of the Conference of the American Physical Society Topical Group on Shock Compression of Condensed Matter. AIP, 2004. http://dx.doi.org/10.1063/1.1780343.

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Wen, Xinrong, and Changqing Tu. "Study on Flotation Separation of Mercury (II) by sodium chloride- potassium iodide-octadecyl trimethyl ammonium chloride system." In 2015 International Forum on Energy, Environment Science and Materials. Paris, France: Atlantis Press, 2015. http://dx.doi.org/10.2991/ifeesm-15.2015.19.

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Taouk, JB, B. Taouk, M. Diracca, E. Pieri, S. Gayetty, F. Raymond, C. Nassibian, and S. Salini. "5PSQ-020 Safety evaluation of injectable potassium chloride prescriptions in hospital." In 24th EAHP Congress, 27th–29th March 2019, Barcelona, Spain. British Medical Journal Publishing Group, 2019. http://dx.doi.org/10.1136/ejhpharm-2019-eahpconf.453.

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Charbonneau, F., AC Desbuquois, AM Liebbe, and M. Boisgontier. "5PSQ-130 Intravenous potassium chloride: is the medication use process secure?" In 25th Anniversary EAHP Congress, Hospital Pharmacy 5.0 – the future of patient care, 23–28 March 2021. British Medical Journal Publishing Group, 2021. http://dx.doi.org/10.1136/ejhpharm-2021-eahpconf.249.

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Houchin, L. R., W. E. Foxenberg, and Zhao Jun. "Evaluation of Potassium Carbonate as a Non-Corrosive, Chloride-Free Completion Fluid." In SPE Formation Damage Control Symposium. Society of Petroleum Engineers, 1994. http://dx.doi.org/10.2118/27392-ms.

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ALmeziny, M., M. Alghamdi, H. Al-Basheer, T. Al-Qarni, and R. Al-Otibi. "CP-111 Association between potassium chloride intravenous concentration and severity of pain." In 22nd EAHP Congress 22–24 March 2017 Cannes, France. British Medical Journal Publishing Group, 2017. http://dx.doi.org/10.1136/ejhpharm-2017-000640.110.

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Hosseini, S., J. Taheri-Shakib, E. Kazemzadeh, and M. Rajabi-Kochi. "Using Ultrasonic Waves as an Advanced Technology to Remove Potassium Chloride Scale." In EAGE 2020 Annual Conference & Exhibition Online. European Association of Geoscientists & Engineers, 2020. http://dx.doi.org/10.3997/2214-4609.202011443.

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Reports on the topic "Tariff on potassium chloride"

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Felker, S., P. Hailey, T. Lian, K. Staggs, and G. Gdowski. Alloy 22 Localized Corrosion Susceptibility In Aqueous Solutions Of Chloride And Nitrate Salts Of Sodium And Potassium At 110 - 150?C. Office of Scientific and Technical Information (OSTI), January 2006. http://dx.doi.org/10.2172/893568.

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