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Journal articles on the topic 'Temporal atrophy'

Author: Grafiati

Published: 4 June 2021

Last updated: 17 February 2022

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1

Hayata, Thaís T., Felipe P. G. Bergo, Thiago J. Rezende, et al. "Cortical correlates of affective syndrome in dementia due to Alzheimer’s disease." Arquivos de Neuro-Psiquiatria 73, no. 7 (2015): 553–60. http://dx.doi.org/10.1590/0004-282x20150068.

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Neuropsychiatric symptoms in Alzheimer’s disease (AD) are prevalent, however their relationship with patterns of cortical atrophy is not fully known. Objectives To compare cortical atrophy’s patterns between AD patients and healthy controls; to verify correlations between neuropsychiatric syndromes and cortical atrophy. Method 33 AD patients were examined by Neuropsychiatric Inventory (NPI). Patients and 29 controls underwent a 3T MRI scanning. We considered four NPI syndromes: affective, apathy, hyperactivity and psychosis. Correlations between structural imaging and neuropsychiatric scores w

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2

Cotta Ramusino, Matteo, Daniele Altomare, Ruggero Bacchin, et al. "Medial temporal lobe atrophy and posterior atrophy scales normative values." NeuroImage: Clinical 24 (2019): 101936. http://dx.doi.org/10.1016/j.nicl.2019.101936.

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Martin, Gilles C., Eliane Albuisson, Valentine Brousse, Mariane de Montalembert, Dominique Bremond-Gignac, and Matthieu P. Robert. "Paramacular temporal atrophy in sickle cell disease occurs early in childhood." British Journal of Ophthalmology 103, no. 7 (2018): 906–10. http://dx.doi.org/10.1136/bjophthalmol-2018-312305.

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Background/aimsInitially reported in a few patients with homozygous sickle cell disease (SCD), atrophic areas of the retina temporal from the macula are now known to be present in about 48% of eyes of adult patients with SS-SCD and in 35% of eyes of adult patients with SC-SCD. The aim of this study is to describe this paramacular atrophy in children affected with SCD.MethodsIn this retrospective series, spectral-domain optical coherence tomography images of 81 children with SCD, acquired with specific patterns including one evaluating the retina temporal to the macula, were reviewed, in order

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4

Hermann, Bruce P., Katherine Bayless, Russ Hansen, Joy Parrish, and Michael Seidenberg. "Cerebellar atrophy in temporal lobe epilepsy." Epilepsy & Behavior 7, no. 2 (2005): 279–87. http://dx.doi.org/10.1016/j.yebeh.2005.05.022.

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5

Jack, Clifford R. "Medial temporal lobe atrophy in MCI." Neurobiology of Aging 21 (May 2000): 2. http://dx.doi.org/10.1016/s0197-4580(00)82683-4.

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6

Idone, Francesco, Elisa Bolletta, Andrea Piedimonte, and Ferdinando Paternostro. "Temporal Fossa Atrophy in Aesthetic Medicine." Plastic and Reconstructive Surgery - Global Open 8, no. 10 (2020): e3169. http://dx.doi.org/10.1097/gox.0000000000003169.

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7

Moran, N. F., L. Lemieux, N. D. Kitchen, D. R. Fish, and S. D. Shorvon. "Extrahippocampal temporal lobe atrophy in temporal lobe epilepsy and mesial temporal sclerosis." Brain 124, no. 1 (2001): 167–75. http://dx.doi.org/10.1093/brain/124.1.167.

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8

Ferreira, Fabio Thadeu, Eliane Kobayashi, Iscia Lopes-Cendes, and Fernando Cendes. "Structural Abnormalities are Similar in Familial and Nonfamilial Mesial Temporal Lobe Epilepsy." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 31, no. 3 (2004): 368–72. http://dx.doi.org/10.1017/s0317167100003462.

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Background/Objective:Diffuse temporal lobe abnormalities can be observed on MRI of patients with mesial temporal lobe epilepsy (MTLE). Our objective was to perform qualitative and quantitative analyses of temporal lobe structures in patients with familial MTLE (FMTLE) and nonfamilial MTLE.Methods:Two groups of patients were ascertained: 67 FMTLE patients (14 with refractory seizures) and 30 patients with nonfamilial refractory MTLE. We performed qualitative analyses of MRI (with multiplanar reconstruction) and volumes of hippocampi and anterior temporal lobes in all patients, and in a normal c

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Firth, Nicholas C., Silvia Primativo, Razvan-Valentin Marinescu, et al. "Longitudinal neuroanatomical and cognitive progression of posterior cortical atrophy." Brain 142, no. 7 (2019): 2082–95. http://dx.doi.org/10.1093/brain/awz136.

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Abstract Posterior cortical atrophy is a clinico-radiological syndrome characterized by progressive decline in visual processing and atrophy of posterior brain regions. With the majority of cases attributable to Alzheimer’s disease and recent evidence for genetic risk factors specifically related to posterior cortical atrophy, the syndrome can provide important insights into selective vulnerability and phenotypic diversity. The present study describes the first major longitudinal investigation of posterior cortical atrophy disease progression. Three hundred and sixty-one individuals (117 poste

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10

Elliott, CA, C. Yasuda, L. Concha, et al. "Progressive contralateral hippocampal atrophy following Temporal Lobe Epilepsy Surgery (TLS)." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 42, S1 (2015): S26—S27. http://dx.doi.org/10.1017/cjn.2015.130.

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Background: Temporal Lobe Epilepsy is associated with bilateral gray (GM) and white matter (WM) loss. After surgical treatment progressive bilateral temporal and extra-temporal WM change occur, however, less is known regarding post-operative GM change. We set out to measure contralateral hippocampal volume (CHV) following TLS. Methods: 1.5T-3D-1mm-isotropic-MPRAGE scans in 26 TLE patients and 3 controls in two groups: longitudinal (n=10)(imaged POD1,2,3,6,60,120 and >360d) and single post-operative scan (n=16). Manual volumetry protocols. Results: We find significant CHV atrophy at delayed

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11

KOUTINAS (Α.Φ. ΚΟΥΤΙΝΑΣ), A. F., C. D. VAMVAKIDIS (Χ. Δ. ΒΑΜΒΑΚΙΔΗΣ), Z. POLIZOPOULOU (Ζ. ΠΟΛΥΖΟΠΟΥΛΟΥ), S. LEKKAS (Σ. ΛΕΚΚΑΣ), and G. GEORGIADIS (Γ. ΓΕΩΡΓΙΑΔΗΣ). "Chronic or atrophie masticatory myositis in the dog. A report of two spontaneous cases." Journal of the Hellenic Veterinary Medical Society 50, no. 1 (2018): 53. http://dx.doi.org/10.12681/jhvms.15698.

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Two young adult dogs (case 1: German wirehaired pointer male, case 2: mongrel female) were admitted to our Clinic because of a progressive, bilateral and symmetrical atrophy of the masticatory muscles, that was most obvious in the temporal muscles. No signs of mandibular dysfunction were detected in both animals. The clinical diagnosis of chronic or atrophic myositis was confirmed by the histopathology of temporal muscle biopsies, which revealed the typical of the disease lesions, such as myofiber degeneration, regeneration and atrophy, interstitial or perivascular cellular infiltrates, chiefl

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12

Shibuya, Katsuhiko, Hideki Nagatomo, Kiyoshi Iwabuchi, Masayuki Inoue, Saburo Yagishita, and Yoji Itoh. "Asymmetrical temporal lobe atrophy with massive neuronal inclusions in multiple system atrophy." Journal of the Neurological Sciences 179, no. 1-2 (2000): 50–58. http://dx.doi.org/10.1016/s0022-510x(00)00364-6.

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13

Düzel, E., K. Schiltz, T. Solbach, et al. "Hippocampal atrophy in temporal lobe epilepsy is correlated with limbic systems atrophy." Journal of Neurology 253, no. 3 (2005): 294–300. http://dx.doi.org/10.1007/s00415-005-0981-y.

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14

Watson, Rosie, John T. O'Brien, Robert Barber, and Andrew M. Blamire. "Patterns of gray matter atrophy in dementia with Lewy bodies: a voxel-based morphometry study." International Psychogeriatrics 24, no. 4 (2011): 532–40. http://dx.doi.org/10.1017/s1041610211002171.

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ABSTRACTBackground: Dementia with Lewy bodies (DLB) is a common form of dementia characterized by visual hallucinations, cognitive fluctuation and parkinsonism. We aimed to compare the patterns of gray matter atrophy in DLB with those in Alzheimer's disease (AD) and normal aging, and to investigate the relationship between atrophy and cognitive measures.Methods: We used voxel-based morphometry (VBM) to investigate gray matter (GM) loss in DLB (n = 35; mean age = 78.4; MMSE = 20.3), AD (n = 36; mean age = 78.3; MMSE = 19.5) and similar aged controls (n = 35; mean age = 76.7; MMSE = 29.1). T1 we

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15

Zhang, Xiuming, Elizabeth C. Mormino, Nanbo Sun, Reisa A. Sperling, Mert R. Sabuncu, and B. T. Thomas Yeo. "Bayesian model reveals latent atrophy factors with dissociable cognitive trajectories in Alzheimer’s disease." Proceedings of the National Academy of Sciences 113, no. 42 (2016): E6535—E6544. http://dx.doi.org/10.1073/pnas.1611073113.

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We used a data-driven Bayesian model to automatically identify distinct latent factors of overlapping atrophy patterns from voxelwise structural MRIs of late-onset Alzheimer’s disease (AD) dementia patients. Our approach estimated the extent to which multiple distinct atrophy patterns were expressed within each participant rather than assuming that each participant expressed a single atrophy factor. The model revealed a temporal atrophy factor (medial temporal cortex, hippocampus, and amygdala), a subcortical atrophy factor (striatum, thalamus, and cerebellum), and a cortical atrophy factor (f

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16

Choi, D. J., B. Y. Cho, and K. J. Lee. "Reconstruction of temporal muscle atrophy with silicon." International Journal of Oral and Maxillofacial Surgery 26 (January 1997): 89. http://dx.doi.org/10.1016/s0901-5027(97)81114-9.

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17

King, David, Susan S. Spencer, Alain Bouthillier, et al. "Medial temporal lobe epilepsy without hippocampal atrophy." Journal of Epilepsy 9, no. 4 (1996): 291–97. http://dx.doi.org/10.1016/s0896-6974(96)00039-4.

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18

Shedlack, Karen J., Eun-Kyu Lee, Rodney A. Radtke, et al. "Ipsilateral subcortical atrophy associated with temporal lobectomy." Psychiatry Research 54, no. 3 (1994): 295–304. http://dx.doi.org/10.1016/0165-1781(94)90023-x.

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19

Pasquier, F., Michèle Hamon, Florence Lebert, Bruno Jacob, Jean-Pierre Pruvo, and Henri Petit. "Medial temporal lobe atrophy in memory disorders." Journal of Neurology 244, no. 3 (1997): 175–81. http://dx.doi.org/10.1007/s004150050069.

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20

Bonilha, L. "Medial temporal lobe atrophy in patients with refractory temporal lobe epilepsy." Journal of Neurology, Neurosurgery & Psychiatry 74, no. 12 (2003): 1627–30. http://dx.doi.org/10.1136/jnnp.74.12.1627.

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21

Bernasconi, N., J. Natsume, and A. Bernasconi. "Progression in temporal lobe epilepsy: Differential atrophy in mesial temporal structures." Neurology 65, no. 2 (2005): 223–28. http://dx.doi.org/10.1212/01.wnl.0000169066.46912.fa.

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22

Granadillo, Elias, Pongsatorn Paholpak, Mario F. Mendez, and Edmond Teng. "Visual Ratings of Medial Temporal Lobe Atrophy Correlate with CSF Tau Indices in Clinical Variants of Early-Onset Alzheimer Disease." Dementia and Geriatric Cognitive Disorders 44, no. 1-2 (2017): 45–54. http://dx.doi.org/10.1159/000477718.

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Background/Aims: Prior studies of late-onset Alzheimer disease (AD) have reported that cerebrospinal fluid (CSF) tau levels correlate with hippocampal/medial temporal lobe atrophy. These findings suggest that CSF tau indices in AD may reflect tau-related neurodegeneration in the medial temporal lobe. However, it remains uncertain whether elevated CSF tau levels in the clinically heterogeneous subtypes of early-onset AD (EOAD; amnestic, posterior cortical atrophy [PCA], and logopenic progressive aphasia [LPA]) are attributable to similar underlying mechanisms. Methods: We identified 41 EOAD pat

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23

Young, Alexandra L., Martina Bocchetta, Lucy L. Russell, et al. "Characterizing the Clinical Features and Atrophy Patterns of MAPT-Related Frontotemporal Dementia With Disease Progression Modeling." Neurology 97, no. 9 (2021): e941-e952. http://dx.doi.org/10.1212/wnl.0000000000012410.

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Background and ObjectiveMutations in the MAPT gene cause frontotemporal dementia (FTD). Most previous studies investigating the neuroanatomical signature of MAPT mutations have grouped all different mutations together and shown an association with focal atrophy of the temporal lobe. The variability in atrophy patterns between each particular MAPT mutation is less well-characterized. We aimed to investigate whether there were distinct groups of MAPT mutation carriers based on their neuroanatomical signature.MethodsWe applied Subtype and Stage Inference (SuStaIn), an unsupervised machine learnin

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24

Black, Sandra E., Scott D. Moffat, David C. Yu, Jayson Parker, Peter Stanchev, and Michael Bronskill. "Callosal Atrophy Correlates with Temporal Lobe Volume and Mental Status in Alzheimer's Disease." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 27, no. 3 (2000): 204–9. http://dx.doi.org/10.1017/s0317167100000846.

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Background:Recent studies have reported significant atrophy of the corpus callosum (CC) in Alzheimer's Disease (AD). However, it is currently unknown whether CC atrophy is associated with specific cortical volume changes in AD. Moreover, possible atrophy in extra-callosal commissures has not been examined to date. The purpose of the present study was to quantify atrophy in two cerebral commissures [the CC and the anterior commissure (AC)], to correlate this measure with cognitive status, and to relate commissural size to independent measures of temporal lobe volume in AD patients.Methods:A sam

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25

Andermann, F. "Why study mesial temporal atrophy in patients with intractable temporal lobe epilepsy?" Journal of Neurology, Neurosurgery & Psychiatry 74, no. 12 (2003): 1606–7. http://dx.doi.org/10.1136/jnnp.74.12.1606.

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26

Cho, Hyun, Jee-Hyun Kwon, and Hyun-Jin Seo. "Medial temporal lobe atrophy in vascular dementia: Visual temporal lobe rating scale." Archives of Gerontology and Geriatrics 48, no. 3 (2009): 415–18. http://dx.doi.org/10.1016/j.archger.2008.03.014.

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27

Oosterman, Joukje M., Saskia Oosterveld, Marcel G. Olde Rikkert, Jurgen A. Claassen, and Roy P. C. Kessels. "Medial temporal lobe atrophy relates to executive dysfunction in Alzheimer's disease." International Psychogeriatrics 24, no. 9 (2012): 1474–82. http://dx.doi.org/10.1017/s1041610212000506.

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ABSTRACTBackground: White matter hyperintensities (WMH) have frequently been associated with lower executive function performance. Little is known, however, about the effects of hippocampal atrophy on executive control in Alzheimer's disease (AD). The present study focused on the association of hippocampal atrophy with executive function in AD patients and examined whether a threshold effect is present, indicating that a certain amount of brain damage must be present before cognitive function becomes impaired. Finally, we examined the combined effect of hippocampal atrophy and WMH on cognitive

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Perrotta, Armando, Monica Bolla, Mariano Serrao, et al. "Enhanced temporal pain processing in multiple system atrophy." Neuroscience Letters 555 (October 2013): 203–8. http://dx.doi.org/10.1016/j.neulet.2013.09.035.

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29

Specht, U., T. May, R. Schulz, et al. "Cerebellar atrophy and prognosis after temporal lobe resection." Journal of Neurology, Neurosurgery & Psychiatry 62, no. 5 (1997): 501–6. http://dx.doi.org/10.1136/jnnp.62.5.501.

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30

Kapur, N. "Focal retrograde amnesia following bilateral temporal lobe atrophy." Neurocase 2, no. 1 (1996): 13f—19. http://dx.doi.org/10.1093/neucas/2.1.13-f.

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31

Chan, D., V. Anderson, Y. Pijnenburg, et al. "The clinical profile of right temporal lobe atrophy." Brain 132, no. 5 (2009): 1287–98. http://dx.doi.org/10.1093/brain/awp037.

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32

King, David, Susan S. Spencer, Gregory McCarthy, Marie Luby, and Dennis D. Spencer. "Bilateral Hippocampal Atrophy in Medial Temporal Lobe Epilepsy." Epilepsia 36, no. 9 (1995): 905–10. http://dx.doi.org/10.1111/j.1528-1157.1995.tb01634.x.

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33

Koch, Giacomo, Alessandro Stefani, Marta Panella, Angela Giordano, Orazio Schillaci, and Girolama A. Marfia. "Posterior cortical atrophy with unilateral occipito-temporal degeneration." Journal of Neurology 251, no. 12 (2004): 1530–31. http://dx.doi.org/10.1007/s00415-004-0573-2.

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34

Cascino, Gregory D. "Temporal Lobe Epilepsy: More than Hippocampal Pathology." Epilepsy Currents 5, no. 5 (2005): 187–89. http://dx.doi.org/10.1111/j.1535-7511.2005.00059.x.

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Voxel-based Morphometry of the Thalamus in Patients with Refractory Medial Temporal Lobe Epilepsy Bonilha L, Rorden C, Castellano G, Cendes F, Li LM Neuroimage 2005;25:1016–1021 Previous research has suggested that patients with refractory medial temporal lobe epilepsy (MTLE) show gray matter atrophy both within the temporal lobes and in the thalamus. However, these studies have not distinguished between different nuclei within the thalamus. We examined whether thalamic atrophy correlates with the nuclei's connections to other regions in the limbic system. T1-weighted MRI scans were obtained f

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Swardfager, Walter, Hugo Cogo-Moreira, Mario Masellis, et al. "The effect of white matter hyperintensities on verbal memory." Neurology 90, no. 8 (2018): e673-e682. http://dx.doi.org/10.1212/wnl.0000000000004983.

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ObjectiveTo determine the relationship between white matter hyperintensities (WMH) presumed to indicate disease of the cerebral small vessels, temporal lobe atrophy, and verbal memory deficits in Alzheimer disease (AD) and other dementias.MethodsWe recruited groups of participants with and without AD, including strata with extensive WMH and minimal WMH, into a cross-sectional proof-of-principle study (n = 118). A consecutive case series from a memory clinic was used as an independent validation sample (n = 702; Sunnybrook Dementia Study; NCT01800214). We assessed WMH volume and left temporal l

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Burns, Alastair, Robin Jacoby, and Raymond Levy. "Psychiatric Phenomena in Alzheimer's Disease. IV: Disorders of Behaviour." British Journal of Psychiatry 157, no. 1 (1990): 86–94. http://dx.doi.org/10.1192/bjp.157.1.86.

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Out of a sample of 178 patients with AD, aggression was present in 20%, wandering in 19%, binge-eating in 10%, hyperorality in 6%, urinary incontinence in 48%, and sexual disinhibition in 7%. Behavioural abnormalities were greater in those with more severe dementia. Temporal-lobe atrophy correlated with aggression, and widening of the third ventricle with hyperorality. Features of the Kluver–Bucy syndrome were commonly seen, but the full syndrome occurred in only one subject. Patients with at least one feature of the Kluver–Bucy syndrome had greater temporal-lobe atropy than those without any

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37

Bonilha, Leonardo, Eliane Kobayashi, João Paulo V. Mattos, Donizete C. Honorato, Li M. Li, and Fernando Cendes. "Value of extent of hippocampal resection in the surgical treatment of temporal lobe epilepsy." Arquivos de Neuro-Psiquiatria 62, no. 1 (2004): 15–20. http://dx.doi.org/10.1590/s0004-282x2004000100003.

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OBJECTIVE: Unilateral hippocampal atrophy is indicator of good surgical prognosis in patients with temporal lobe epilepsy (TLE). Some patients however do not become seizure free after surgery. We assessed if the extent of hippocampal and amygdala resection is associated with outcome. METHODS: Thirty patients with TLE with unilateral or clearly asymmetric hippocampal atrophy who underwent surgical treatment were evaluated concerning preoperative clinical variables and interictal EEG abnormalities. Amygdala and hippocampal resection was evaluated by post-operative MRI. We compared seizure free v

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38

Caciagli, Lorenzo, Andrea Bernasconi, Samuel Wiebe, Matthias J. Koepp, Neda Bernasconi, and Boris C. Bernhardt. "A meta-analysis on progressive atrophy in intractable temporal lobe epilepsy." Neurology 89, no. 5 (2017): 506–16. http://dx.doi.org/10.1212/wnl.0000000000004176.

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Objective:It remains unclear whether drug-resistant temporal lobe epilepsy (TLE) is associated with cumulative brain damage, with no expert consensus and no quantitative syntheses of the available evidence.Methods:We conducted a systematic review and meta-analysis of MRI studies on progressive atrophy, searching PubMed and Ovid MEDLINE databases for cross-sectional and longitudinal quantitative MRI studies on drug-resistant TLE.Results:We screened 2,976 records and assessed eligibility of 248 full-text articles. Forty-two articles met the inclusion criteria for quantitative evaluation. We obse

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39

HARVEY, G. T., J. HUGHES, I. G. McKEITH, et al. "Magnetic resonance imaging differences between dementia with Lewy bodies and Alzheimer's disease: a pilot study." Psychological Medicine 29, no. 1 (1999): 181–87. http://dx.doi.org/10.1017/s0033291798007806.

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Background. Temporal lobe atrophy on magnetic resonance imaging (MRI) has been suggested as a specific diagnostic marker for Alzheimer's disease (AD). No previous comparison with dementia with Lewy bodies (DLB) has been reported.Method. T1-weighted MRI scans were performed on 11 subjects with AD (nine with NINCDS/ADRDA probable AD and two with neuropathologically proven AD) and nine subjects with DLB (four with probable DLB diagnosed by clinical criteria and five with neuropathologically proven DLB). Groups were matched for age, duration of illness and cognitive test score. Two raters, blind t

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40

O'Brien, J. T., P. Desmond, D. Ames, I. Schweitzer, V. Tuckwell, and B. Tress. "The differentiation of depression from dementia by temporal lobe magnetic resonance imaging." Psychological Medicine 24, no. 3 (1994): 633–40. http://dx.doi.org/10.1017/s0033291700027781.

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SynopsisTemporal lobe Magnetic Resonance Imaging (MRI) was performed in 43 patients with NINCDS/ADRDA Alzheimer's disease (AD) (33 probable, 7 possible, 3 definite) and 32 subjects with DSM-III-R Major Depression (DEP) matched for age, sex and level of education. Hippocampus (anterior and posterior, right and left), amygdala, entorhinal cortex, para-hippocampal gyrus and cerebral cortex were rated for atrophy on a 4-point scale. Good discrimination between groups could be achieved using a cut-off of 2 or more on anterior hippocampal atrophy rating (sensitivity 93%; specificity 84%; 89% cases c

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Baldwin, Bob, and Hans Förstl. "‘Pick's Disease’–101 Years on Still There, But in Need of Reform." British Journal of Psychiatry 163, no. 1 (1993): 100–104. http://dx.doi.org/10.1192/bjp.163.1.100.

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On 23 April 1892, Arnold Pick reported the case of AH, who died aged 71 years following a two-year history of progressive ‘feeble-mindedness', outbursts of rage, fits and, in the later stages, severe aphasia (Pick, 1892). The post-mortem showed cerebral atrophy, particularly affecting the left temporal lobe. Pick went on to describe further cases of circumscribed atrophy affecting the temporal lobe (Pick, 1901, 1904), and parietal and frontal lobes (Pick, 1906). Although he believed the focal pathology represented a local emphasis of ‘senile cortical atrophy’, he wanted to show that a localise

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42

Boccardi, M. "The MRI pattern of frontal and temporal brain atrophy in fronto-temporal dementia." Neurobiology of Aging 24, no. 1 (2003): 95–103. http://dx.doi.org/10.1016/s0197-4580(02)00045-3.

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ANDRADE JR., FRANCISCO CARLOS DE, FRANCISCO CARLOS DE ANDRADE, CELSO MACHADO DE ARAUJO FILHO, and JOSÉ CARCAGNOLO FILHO. "Dysfunction of the temporalis muscle after pterional craniotomy for intracranial aneurysms: comparative, prospective and randomized study of one flap versus two flaps dieresis." Arquivos de Neuro-Psiquiatria 56, no. 2 (1998): 200–205. http://dx.doi.org/10.1590/s0004-282x1998000200006.

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Patients with intracranial aneurysm(s) of the carotid artery territory, treated with pterional craniotomy, were prospectively and randomly addressed to one layer flap (n=36) or myocutaneous (MC) versus two layers' dieresis (n=32) or interfascial (IF). The study protocol included the patient's sex, age, area of craniotomy, time of flap dieresis and synthesis, time of bone dieresis and synthesis, the intracranial time, including dura mater dieresis and synthesis and time of flap retraction. Before and after surgery, the patients were evaluated with examination specially oriented to V and VII cra

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Sarbu, N., P. Toledano, A. Calvo, et al. "Advanced MRI techniques: biomarkers in neuropsychiatric lupus." Lupus 26, no. 5 (2017): 510–16. http://dx.doi.org/10.1177/0961203316674820.

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Objectives The objective of this study was to determine whether advanced MRI could provide biomarkers for diagnosis and prognosis in neuropsychiatric systemic lupus erythematosus (NPSLE). Methods Our prospective study included 28 systemic lupus erythematosus (SLE) patients with primary central NPSLE, 22 patients without NPSLE and 20 healthy controls. We used visual scales to evaluate atrophy and white matter hyperintensities, voxel-based morphometry and Freesurfer to measure brain volume, plus diffusion-tensor imaging (DTI) to assess white matter (WM) and gray matter (GM) damage. We compared t

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Brito-Marques, Paulo Roberto de, Roberto Vieira de Mello, and Luciano Montenegro. "Classic Pick's disease type with ubiquitin-positive and tau-negative inclusions: case report." Arquivos de Neuro-Psiquiatria 59, no. 1 (2001): 128–33. http://dx.doi.org/10.1590/s0004-282x2001000100028.

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We report on a patient presenting Pick's disease similar to the one reported by Pick in 1892, with ubiquitin-positive and tau-negative inclusions. His diagnosis was made on the basis of clinical (language disturbance and behavioural disorders), neuropsychological (progressive aphasia of the expression type and late mutism), neuroimaging with magnetic resonance (bilateral frontal and temporal lobes atrophy) and brain single photon emission computed tomography (frontal and temporal lobes hypoperfusion) studies. Macroscopic examination showed atrophy on the frontal and temporal lobes. The left hi

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Elliott, CA, D. Gross, B. Wheatley, C. Beaulieu, and T. Sankar. "C.03 Progressive contralateral hippocampal atrophy following surgery for medically refractory temporal lobe epilepsy." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 43, S2 (2016): S11—S12. http://dx.doi.org/10.1017/cjn.2016.69.

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Background: It remains difficult to predict which patients will experience ongoing seizures or neuropsychological deficits following Temporal Lobe Epilepsy (TLE) surgery. MRI allows measurement of brain structures, such as the contralateral (non-resected) hippocampus (cHC) after TLE surgery. Preliminary evidence suggests that the cHC atrophies following surgery, however, the time course of this atrophy, relation to cognitive deficits and seizure outcome remains unclear. Methods: T1-weighted MR imaging and hippocampal volumetry in 26 TLE patients pre- and post-TLE surgery (and 12 controls) as:

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Podea, D. M. "Mild cognitive impairment - Correlations of imagistic and clinical data." European Psychiatry 26, S2 (2011): 949. http://dx.doi.org/10.1016/s0924-9338(11)72654-2.

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Mild cognitive impairment is the clinical state between normal cognition and dementia in elderly people which does not interfere notably with activities of daily life; subjects perform more poorly on a variety of cognitive, functional and behavioral measures than normal persons of the same age.The study comprises 141 subjects between 60 and 92, diagnosed with mild cognitive impairment with Mini Mintal State Evaluation (MMSE), clock drawing test (CDT) and computer tomography.MMSE score between 28-21 points is considered mild cognitive impairment.The computer tomography imaging showed specific t

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Ulugut Erkoyun, Hulya, Colin Groot, Ronja Heilbron, et al. "A clinical-radiological framework of the right temporal variant of frontotemporal dementia." Brain 143, no. 9 (2020): 2831–43. http://dx.doi.org/10.1093/brain/awaa225.

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Abstract The concept of the right temporal variant of frontotemporal dementia (rtvFTD) is still equivocal. The syndrome accompanying predominant right anterior temporal atrophy has previously been described as memory loss, prosopagnosia, getting lost and behavioural changes. Accurate detection is challenging, as the clinical syndrome might be confused with either behavioural variant FTD (bvFTD) or Alzheimer’s disease. Furthermore, based on neuroimaging features, the syndrome has been considered a right-sided variant of semantic variant primary progressive aphasia (svPPA). Therefore, we aimed t

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SEIDENBERG, MICHAEL, BRUCE HERMANN, DALIN PULSIPHER, et al. "Thalamic atrophy and cognition in unilateral temporal lobe epilepsy." Journal of the International Neuropsychological Society 14, no. 03 (2008): 384–93. http://dx.doi.org/10.1017/s1355617708080399.

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Pettigrew, Corinne, Anja Soldan, Kelly Sloane, et al. "Progressive medial temporal lobe atrophy during preclinical Alzheimer's disease." NeuroImage: Clinical 16 (2017): 439–46. http://dx.doi.org/10.1016/j.nicl.2017.08.022.

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