Journal articles: 'Tendinitis'

1

Robinson, Kim. "Tendinitis." Physiotherapy 88, no. 7 (July 2002): 446. http://dx.doi.org/10.1016/s0031-9406(05)61292-8.

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Petsche, Timothy S., and F. Harlan Selesnick. "Popliteus Tendinitis." Physician and Sportsmedicine 30, no. 8 (August 2002): 27–31. http://dx.doi.org/10.3810/psm.2002.08.401.

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3

Olson, WR, and L. Rechkemmer. "Popliteus tendinitis." Journal of the American Podiatric Medical Association 83, no. 9 (September 1, 1993): 537–40. http://dx.doi.org/10.7547/87507315-83-9-537.

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Popliteus tendinitis is a relatively infrequent and often misdiagnosed injury of the posterior aspect of the knee. The clinical significance of this injury is particularly relevant to the serious runner and triathlete, as the symptoms resulting from this injury can be disabling.

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4

Menz, Robert J. "“Texting” tendinitis." Medical Journal of Australia 182, no. 6 (March 2005): 308. http://dx.doi.org/10.5694/j.1326-5377.2005.tb06708.x.

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5

Uhthoff, H. K., and K. Sarkar. "Calcifying tendinitis." Baillière's Clinical Rheumatology 3, no. 3 (December 1989): 567–81. http://dx.doi.org/10.1016/s0950-3579(89)80009-3.

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6

Leach, Robert E., Anthony A. Schepsis, and Hiroaki Takai. "Achilles Tendinitis." Physician and Sportsmedicine 19, no. 8 (August 1991): 87–92. http://dx.doi.org/10.1080/00913847.1991.11702231.

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7

Jafarnia, Kourosh, Gerard T. Gabel, and Bernard F. Morrey. "Triceps tendinitis." Operative Techniques in Sports Medicine 9, no. 4 (October 2001): 217–21. http://dx.doi.org/10.1053/otsm.2001.26782.

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8

Holt, Peter D., and Theodore E. Keats. "Calcific Tendinitis." Contemporary Diagnostic Radiology 19, no. 15 (1996): 1–5. http://dx.doi.org/10.1097/00219246-199619150-00001.

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9

Bertone, Alicia L. "Equine tendinitis." Journal of Equine Veterinary Science 16, no. 1 (January 1996): 16–17. http://dx.doi.org/10.1016/s0737-0806(96)80060-8.

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10

Torstensen, Eric T., Robert C. Bray, and J. Preston Wiley. "Patellar Tendinitis." Clinical Journal of Sport Medicine 4, no. 2 (April 1994): 77–82. http://dx.doi.org/10.1097/00042752-199404000-00002.

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11

Hughes, P. J., and B. Bolton-Maggs. "Calcifying tendinitis." Current Orthopaedics 16, no. 5 (October 2002): 389–94. http://dx.doi.org/10.1054/cuor.2002.0259.

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12

Clain, Michael R., and Donald E. Baxter. "Achilles Tendinitis." Foot & Ankle 13, no. 8 (October 1992): 482–87. http://dx.doi.org/10.1177/107110079201300810.

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Achilles tendinitis is a problem encountered frequently. There are certain anatomical and biomechanical principles that help explain the etiology of this entity. We prefer to separate our thinking into “insertional” and “noninsertional” Achilles tendinitis. This is helpful because it allows nonoperative and operative treatment to be problem specific and systematic.

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13

Fahlgren, Holger. "Retropharyngeal Tendinitis." Cephalalgia 6, no. 3 (September 1986): 169–74. http://dx.doi.org/10.1046/j.1468-2982.1986.0603169.x.

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A rare clinical syndrome, with acute onset of severe pains localized in the back of the neck and aggravated by head movements and swallowing, has been studied in 28 patients. X-ray examinations of the cervical spine and prevertebral soft tissues in straight lateral projection of all patients in the acute phase showed significant soft-tissue swelling anterior to the vertebral bodies of C1–C4. Eighteen patients had an amorphous calcific deposit below the tuberculum anterius atlantis in or near the mid-line. The clinical symptoms were characteristic, with a benign course and freedom from complaints and regression of the radiological changes after 1–2 weeks. The diagnosis was usually made clinically and confirmed after two or more X-ray examinations of the cervical spine and the prevertebral soft tissues. The disease was in all probability caused by acute tendinitis in the longus colli musculus.

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Fahlgren, H., S. Jansa, and S. Löfstedt. "RETROPHARYNGEAL TENDINITIS." Acta Neurologica Scandinavica 43, S31 (January 29, 2009): 188. http://dx.doi.org/10.1111/j.1600-0404.1967.tb02114.x.

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15

Heckmann, J. G., R. Tröscher-Weber, M. Pawlowski, F. Seifert, C. J. G. Lang, A. Dörfler, and S. Schwab. "Retropharyngeale Tendinitis." Der Nervenarzt 77, no. 8 (August 2006): 952–57. http://dx.doi.org/10.1007/s00115-006-2124-9.

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16

Goldner, J. Leonard, and Leo M. Rozmaryn. "OVERUSE TENDINITIS." Orthopedics 22, no. 3 (March 1999): 288–89. http://dx.doi.org/10.3928/0147-7447-19990301-18.

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17

Scioli, Mark W. "Achilles Tendinitis." Orthopedic Clinics of North America 25, no. 1 (January 1994): 177–82. http://dx.doi.org/10.1016/s0030-5898(20)31876-9.

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18

Bejko, Etleva, Angela Gupta, and Maya Mattar. "Not Every Hot, Tender, Inflamed Joint is Infected (or Gout)!" Journal of Clinical Case Studies Reviews & Reports 2, no. 2 (April 30, 2020): 1–5. http://dx.doi.org/10.47363/jccsr/2020(2)115.

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Calcific tendinitis is a rare condition that classically involves the rotator cuff. Only isolated case reports exist of the disease affecting other anatomical locations. Our case is noteworthy because it describes calcific tendinitis in an uncommon location; the flexor pollicis longus of the thumb. Calcific tendinitis has a prevalence of 3-10% in the general population, and is most commonly seen among women in their 4th to 6th decade of life. Calcific tendinitis is best diagnosed by imaging where plain radiography and ultrasound are both helpful for detecting calcifications. Blood tests are generally not used for making the diagnosis. However, elevated white blood cells and inflammatory markers (e.g. sedimentation rate and C-reactive protein) may be seen in the acute phase of calcific tendinitis, but are usually normal. Although the exact pathophysiology of calcific tendinitis is unknown. Calcific tendinitis condition is often self-limited and interventions (i.e. anti-inflammatory medications, analgesics, glucocorticoid injections, physical therapy) are used with a “try it and see” mindset. Other treatment options (extracorporeal shock wave therapy, therapeutic ultrasound, iontophoresis, platelet rich plasma) have been tried. Acute calcific tendinitis should be on the differential diagnosis for acute musculoskeletal pain—even pain that is not at the rotator cuff. This case demonstrates that pain that is associated with a tender, inflamed joint can be more than either infection or gout. It is important to recognize calcific tendinitis as a potential diagnosis, as this could prevent unnecessary interventions and therapy such as intravenous antibiotics.

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19

Ohtsuka, Yuichiro, Hideaki Chazono, Homare Suzuki, Yusuke Ohkuma, Toshioki Sakurai, Toyoyuki Hanazawa, and Yoshitaka Okamoto. "Eight Cases of Calcific Retropharyngeal Tendinitis/Retropharyngeal Calcific Tendinitis." Nippon Jibiinkoka Gakkai Kaiho 116, no. 11 (2013): 1200–1207. http://dx.doi.org/10.3950/jibiinkoka.116.1200.

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20

Suzuki, Takeshi, and Akiko Okamoto. "Marked Multiple Tendinitis at the Onset of Rheumatoid Arthritis in a Patient with Heterozygous Familial Hypercholesterolemia: Ultrasonographic Observation." Case Reports in Rheumatology 2014 (2014): 1–6. http://dx.doi.org/10.1155/2014/486348.

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A 59-year-old woman who had been diagnosed with heterozygous familial hypercholesterolemia developed rheumatoid arthritis (RA). She presented with marked tendinitis of the Achilles tendons, patellar tendons, and finger extensor tendons at the onset of RA. Ultrasonographic examination revealed that tendon lesions were predominantly tendinitis rather than paratenonitis, and that the tendinitis was of the noninsertional variety, rather than the insertional variety. Preexisting tendon xanthomas might have contributed to the unusually dominant noninsertional tendinitis of multiple tendons.

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21

Kim, Min-Su, In-Woo Kim, Sanghyeon Lee, and Sang-Jin Shin. "Diagnosis and treatment of calcific tendinitis of the shoulder." Clinics in Shoulder and Elbow 23, no. 4 (December 1, 2020): 203–9. http://dx.doi.org/10.5397/cise.2020.00318.

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Calcific tendinitis is the leading cause of shoulder pain. Among patients with calcific tendinitis, 2.7%–20% are asymptomatic, and 35%–45% of patients whose calcific deposits are inadvertently discovered develop shoulder pain. If symptoms are present, complications such as decreased range of motion of the shoulder joint should be minimized while managing pain. Patients with acute calcific tendinitis respond well to conservative treatment and rarely require surgery. In contrast, patients with chronic calcific tendinitis often do not respond to conservative treatment and do require surgery. Clinical improvement takes time, even after surgical treatment. This review article summarizes the processes related to the diagnosis and treatment of calcific tendinitis with the aim of helping clinicians choose appropriate treatment options for their patients.

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22

Aumann, Emel K., Taner Aksu, Vefa Atansay, Ayhan N. Kara, and Neslihan Aksu. "Relationship of Popliteus Sulcus Depth and Tibiofemoral Rotational Alignment with Popliteus Tendinitis in Professional Folk Dancers Exposed to Turnout Positions: An MRI Analysis." Medical Problems of Performing Artists 34, no. 3 (September 1, 2019): 141–46. http://dx.doi.org/10.21091/mppa.2019.3024.

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AIMS: The popliteus musculotendinous unit plays the major role in range of motion, rotation and stabilization of the knee. Frequent repetition of the rotational strains such as turnout in dancers may be the cause of popliteus tendinitis. In addition, popliteus sulcus depth (PSD) and PSD/lateral condyle width (LCW) ratio are also highly related with popliteal tendinitis in professional folk dancers. In this study, we evaluated the association between clinically diagnosed popliteus tendinitis and PSD with analysis of the PSD/LCW ratio as measured on MRI and tibiofemoral rotational alignment in professional folk dancers. This study was intended to clarify any anatomical liability to popliteal tendinitis in professional folk dancers. Therefore, we looked for the anatomical variances affecting popliteal musculotendinous unit. METHODS: Thirty-two MRI scans from 32 members of a professional folk dance group (mean age 30.2±7.9 yrs, range 18–38) were analyzed retrospectively. Popliteal tendinitis was detected in 5 knees (5 dancers). The relationship of popliteal tendinitis to the tibiofemoral rotational angles (condilary twist angle, posterior codilary angle, posterior tibiofemoral angle), PSD, and PSD/LCW ratio were investigated. RESULTS: The popliteus tendinitis group had statistically significantly higher PSD and PSD/LCW ratio than the group without popliteal tendinitis (p=0.0001). There was no statistically significant difference between the two groups in the tibiofemoral rotational angles. CONCLUSION: In addition to long hours of practice and the turnout position, PSD and PSD/LCW ratio can place the professional folk dancer at increased risk for popliteus tendon injury.

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23

Nozu, Tsukasa, Shima Kumei, Masumi Ohhira, and Toshikatsu Okumura. "Retropharyngeal Calcific Tendinitis." Internal Medicine 54, no. 17 (2015): 2277. http://dx.doi.org/10.2169/internalmedicine.54.4682.

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24

Ashurst, John, and Kevin Weaver. "Longus Colli Tendinitis." Journal of the American Osteopathic Association 114, no. 03 (March 2014): 216. http://dx.doi.org/10.7556/jaoa.2014.040.

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25

Casado Burgos, Enrique, Guillermina Viñas Ponce, Ricardo Lauzurica Valdemoros, and Alejandro Olivé Marqués. "Tendinitis por levofloxacino." Medicina Clínica 114, no. 8 (January 2000): 319. http://dx.doi.org/10.1016/s0025-7753(00)71280-2.

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26

Clancy, William G., and Steven V. Hagan. "Tendinitis In Golf." Clinics in Sports Medicine 15, no. 1 (January 1996): 27–35. http://dx.doi.org/10.1016/s0278-5919(20)30156-3.

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27

Cunningham, Marilyn E. "Bursitis and Tendinitis." Orthopaedic Nursing 13, no. 5 (September 1994): 13–16. http://dx.doi.org/10.1097/00006416-199409000-00004.

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28

Kirsch, Jacobo, and Ivan Garza. "Longus Colli Tendinitis." Headache: The Journal of Head and Face Pain 49, no. 5 (May 2009): 753–55. http://dx.doi.org/10.1111/j.1526-4610.2009.01413.x.

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29

Popp, James E., Joseph S. Yu, and Christopher C. Kaeding. "Recalcitrant Patellar Tendinitis." American Journal of Sports Medicine 25, no. 2 (March 1997): 218–22. http://dx.doi.org/10.1177/036354659702500214.

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30

Tanaka, Shiro, Cheryl Fairfield Estill, and Stephen C. Shannon. "Blueberry Rakers' Tendinitis." New England Journal of Medicine 331, no. 8 (August 25, 1994): 552. http://dx.doi.org/10.1056/nejm199408253310819.

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31

BLOME, STEVEN A. "Retropharyngeal Calcific Tendinitis." Australasian Radiology 31, no. 2 (May 1987): 142–43. http://dx.doi.org/10.1111/j.1440-1673.1987.tb01800.x.

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32

Oniel, Marie-Eileen. "Rotator Cuff Tendinitis." Journal of the American Academy of Nurse Practitioners 6, no. 7 (July 1994): 339–40. http://dx.doi.org/10.1111/j.1745-7599.1994.tb00964.x.

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33

Cheng, Alex, and Philip E. Zapanta. "Prevertebral Calcific Tendinitis." Otolaryngology–Head and Neck Surgery 145, no. 2_suppl (August 2011): P155. http://dx.doi.org/10.1177/0194599811415823a68.

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34

Wiper, Jonathan D., and Anselmo Garrido. "Acute Calcific Tendinitis." New England Journal of Medicine 359, no. 23 (December 4, 2008): 2477. http://dx.doi.org/10.1056/nejmicm063524.

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35

Gültuna, Selcan, Seyfettin Köklü, Mehmet Arhan, Fatih Aydn, Pnar Mesci, and Oğuz Üsküdar. "Ciprofloxacin Induced Tendinitis." JCR: Journal of Clinical Rheumatology 15, no. 4 (June 2009): 201–2. http://dx.doi.org/10.1097/rhu.0b013e3181a7b0d4.

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36

Grubić Kezele, Tanja, Ariana Fužinac-Smojver, Luka Đudarić, Tamara Kauzlarić-Živković, and Jan Nemrava. "Učinkovitost liječenja bola terapijom udarnog vala u plantarnom fascitisu, kalcificirajućem tendinitisu ramena i lateralnom epikondilitisu lakta." Medicina Fluminensis 56, no. 2 (June 1, 2020): 157–65. http://dx.doi.org/10.21860/medflum2020_237303.

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Uvod: Terapija udarnim valom (TUV) (od engl. shockwave therapy) je neinvazivna metoda temeljena na mehaničkim pulsovima koji se u obliku vala šire kroz ljudsko tijelo te izazivaju mikroskopske intersticijalne i ekstracelularne biološke učinke među kojima je i regeneracija tkiva. Cilj: Cilj ovog retrospektivnog istraživanja bio je ispitati učinkovitost i razlike u liječenju bola TUV-om u pacijenata s dijagnozama plantarni fascitis, kalcificirajući tendinitis ramena i lateralni epikondilitis lakta nakon upotrebe tri i pet uzastopnih tretmana. Ispitanici i metode: U radu su korišteni podatci pacijenata iz baze podataka Zavoda za fizikalnu i rehabilitacijsku medicinu Kliničkog bolničkog centra u Rijeci, a pri tome su u skladu s Helsinškom deklaracijom ostali anonimni: dob, spol, mjerenja intenziteta bola dobivena uz pomoć vizualno analogne skale (od engl. Visual Analogue Scale, VAS). Ispitanici su podijeljeni u dvije grupe za svaku dijagnozu (TUV3 i TUV5). Ispitivanje je sadržavalo ukupno 148 pacijenata: plantarni fascitis N = 50, kalcificirajući tendinitis ramena N = 50 i lateralni epikondilitis lakta N = 48. Rezultati: Primjena terapije udarnim valom kod pacijenata s dijagnozama plantarni fascitis, kalcificirajući tendinitis ramena i lateralni epikondilits lakta, s ciljem smanjenja intenziteta bola, pokazala se uspješnom. Dobivenim rezultatima utvrđeno je da primjena pet TUV tretmana dovodi do boljih rezultata smanjenja bola u sve tri dijagnoze negoli samo tri primijenjena tretmana (p < 0,001; p < 0,001; p < 0,001). Nadalje je utvrđeno da se nakon tri primijenjena tretmana postigao bolji učinak smanjenja bola kod plantarnog fascitisa negoli kod kalcificirajućeg tendinitisa ramena i lateralnog epikondilitisa lakta, dok nakon pet primijenjena tretmana nije bilo razlike u smanjenju bola među dijagnozama. Zaključak: Ova studija preporučuje primjenu pet tretmana kod sve tri dijagnoze kako bi pacijenti imali bolji učinak smanjenja bola.

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37

Wako, Masanori, Jiro Ichikawa, Kensuke Koyama, Yoshihiro Takayama, and Hirotaka Haro. "Calcific Tendinitis of the Supraspinatus Tendon in an Infant." Case Reports in Orthopedics 2020 (July 3, 2020): 1–3. http://dx.doi.org/10.1155/2020/9842489.

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Calcific tendinitis of the supraspinatus tendon in adults is common, but it is extremely rare in children. This report presents an unusual case of a 2-year-old boy with calcific tendinitis of the supraspinatus tendon. A mother brought her 2-year-old son to our hospital with a fever and severe left shoulder pain. Examination revealed a temperature of 38.6°C accompanied by a swollen shoulder with extreme pain and restricted movement. The radiographs of his left shoulder showed a large radio-opacity in the subacrominal region, and magnetic resonance imaging showed an elongated T1 and T2 hypointense signal above the supraspinatus tendon. Although these images were suggestive of calcific tendinitis of the supraspinatus tendon, we performed an open biopsy and resection in order to differentiate between a suspected diagnosis of calcific tendinitis, which is incredibly rare within pediatric patients, and infection or a soft tissue tumor. Finally, calcific tendinitis of the supraspinatus tendon was diagnosed by pathologic experiment and successfully treated, with complete resolution of pain and movement. Because only four other pediatric cases of calcific tendinitis of the supraspinatus tendon have ever been reported, there is a lack of information on the diagnostic process, management, and treatment of such a condition in young patients. Calcific tendinitis of the supraspinatus tendon still should be considered when encountering cases with typical findings even if the patient is a child.

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38

Siegal, Daniel S., Jim S. Wu, Joel S. Newman, Jose L. del Cura, and Mary G. Hochman. "Calcific Tendinitis: A Pictorial Review." Canadian Association of Radiologists Journal 60, no. 5 (December 2009): 263–72. http://dx.doi.org/10.1016/j.carj.2009.06.008.

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Calcific tendinitis is caused by the pathologic deposition of calcium hydroxyapatite crystals in tendons and is a common cause of joint pain. The disease typically affects the shoulder and hip, with characteristic imaging findings; however, any joint can be involved. Occasionally, calcific tendinitis can mimic aggressive disorders, such as infection and neoplasm, especially on magnetic resonance imaging. Radiologists should be familiar with the imaging findings to distinguish calcific tendinitis from more aggressive processes. Image-guided percutaneous needle aspiration and steroid injection of calcific tendinitis are useful techniques performed by the radiologist for the treatment of symptomatic cases. Familiarity with these procedures and their imaging appearance is an important aspect in the management of this common disease.

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39

Jungwirth-Weinberger, Anna, Christian Gerber, Glenn Boyce, Thorsten Jentzsch, Simon Roner, and Dominik C. Meyer. "Restriction of Passive Glenohumeral Abduction Combined With Normal Passive External Rotation Is a Diagnostic Feature of Calcific Tendinitis." Orthopaedic Journal of Sports Medicine 6, no. 2 (February 1, 2018): 232596711775290. http://dx.doi.org/10.1177/2325967117752907.

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Background: Passive glenohumeral range of motion may be characteristically limited to specific shoulder pathologies. While pain associated with loss of range of passive external glenohumeral rotation is recognized as a salient feature in adhesive capsulitis, restriction of glenohumeral range of motion in calcific tendinitis of the supraspinatus tendon has never been studied. Hypothesis: On the basis of clinical observation, we hypothesized that calcific tendinitis of the supraspinatus tendon is associated with loss of passive glenohumeral abduction without loss of external rotation. Study Design: Cohort study; Level of evidence, 3. Methods: Ranges of passive glenohumeral rotation and abduction, which are measured with a standardized protocol in our institution, were retrospectively reviewed and compared for patients diagnosed with either adhesive capsulitis or calcific tendinitis of the supraspinatus tendon. A total of 57 patients met the inclusion criteria for the calcific tendinitis, and 77 met the inclusion criteria for the adhesive capsulitis group. Results: When compared with the contralateral, unaffected shoulder, glenohumeral abduction in the calcific tendinitis group was restricted by a median of 10° (interquartile range [IQR], –20° to –5°) as opposed to glenohumeral external rotation, which was not restricted at all (median, 0°; IQR, 0° to 0°). The adhesive capsulitis group showed a median restriction of glenohumeral abduction of 40° (IQR, –50° to –30°) and a median restriction of passive glenohumeral external rotation of 40° (IQR, –60° to –30°). Conclusion: Calcific tendinitis of the supraspinatus does not typically cause loss of external rotation but is frequently associated with mild isolated restriction of abduction. This finding can be used to clinically differentiate adhesive capsulitis from calcific tendinitis.

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40

S.K, Tmumen,, Anwar M. Abdalmula, and Fahima A. Alnagar. "Hematological And Biochemical Blood Changes In Chronic Tendinitis Thoroughbred Race Horses." International Journal of Scientific Research and Management 8, no. 08 (August 26, 2020): 12–19. http://dx.doi.org/10.18535/ijsrm/v8i08.vs01.

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Tendon injury is the most important veterinary reason for wastage of thoroughbred racehorses. Clinical diagnosis of tendon injuries is confirmed by a combination of clinical, ultrasonographic or post mortem examination of the injured limb. In addition, measurement of hematological and biochemical blood parameters are an important tool that aid health assessment and decision-making in diagnosis, treatment and follow-up of the injured tendon. Therefore, the lack of information or misinterpretation of these parameters may affect the accuracy of disease diagnosis and then lead to poor treatment. The present study was conducted to evaluate the levels of some hematological and biochemical parameter in the blood of thoroughbred horses affected by chronic tendinitis and compared with normal horses. Blood samples were collected from 15 healthy thoroughbred horses (8 stallions and 7 mares) and 21 tendinitis thoroughbred horses (11 stallions and 10 mares); and the levels of 18 blood parameters were determined. The tendinitis horses had higher number of erythrocytes and thrombocytes, higher values of packed cell volume (PCV) and mean corpuscular volume (MCV); lower enzyme activity of creatine kinase (CK), lower values of lactic acid (LA), icteric index and mean corpuscular hemoglobin concentration (MCHC) and lower numbers of band neutrophils than the normal horses. The chronic tendinitis mares had higher number of thrombocytes and lower values of lactate dehydrogenase (LDH) enzyme activity, lactic acid, plasma proteins, MCHC and lower numbers of white blood cells (WBC) than the normal mares. The chronic tendinitis stallions had higher levels of lactic acid, plasma proteins, MCV, and higher numbers of erythrocytes and thrombocytes,; and lower values of icteric jaundice, MCHC, band neutrophils than the normal horses. No significant differences were reported when tendinitis mares were compared with tendinitis males. However, normal mares showed higher levels of plasma proteins than normal stallions. The results obtained by this study can be used as useful index to diagnose and treat chronic tendinitis in horses.

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41

Suh, Dong-Hwan, Jong-Hun Ji, and Chang-Yeon Kim. "Isolated calcific tendinitis at the posterosuperior labrum: a rare case study." Clinics in Shoulder and Elbow 23, no. 4 (December 1, 2020): 194–97. http://dx.doi.org/10.5397/cise.2020.00297.

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Calcific tendinitis of the shoulder joint, also known as chemical furuncle of the shoulder, causes intense shoulder pain and usually occurs within 1–2 cm from the insertion of the rotator cuff. We experienced a rare case of calcific tendinitis in the posterosuperior labrum of the shoulder joint in a 39-year-old male patient who presented with severe pain and weakness in the right shoulder. Radiographs and magnetic resonance imaging (MRI) findings showed calcific tendinitis in the posterosuperior labrum of the shoulder joint. A 1-week attempt at conservative treatment failed, so the calcified deposit in the posterosuperior labrum was arthroscopically removed. The patient’s symptoms were completely relieved, and satisfactory clinical outcomes were achieved. Postoperative follow-up X-ray and MRI showed no recurrence of calcific tendinitis.

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42

Knopp, William D., Matthew E. Bohm, and James C. McCoy. "Hypothyroidism Presenting as Tendinitis." Physician and Sportsmedicine 25, no. 1 (January 1997): 47–55. http://dx.doi.org/10.3810/psm.1997.01.1094.

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43

Khan, Karim M., Jill L. Cook, Jack E. Taunton, and Fiona Bonar. "Overuse Tendinosis, Not Tendinitis." Physician and Sportsmedicine 28, no. 5 (May 2000): 38–48. http://dx.doi.org/10.3810/psm.2000.05.890.

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44

Gregorace, Joseph G. "Recognizing Shoulder-Restraint Tendinitis." Physician and Sportsmedicine 28, no. 5 (May 2000): 37. http://dx.doi.org/10.3810/psm.2000.05.918.

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45

Sanghvi, Darshana A., Bhavin G. Jankharia, Nilendu C. Purandare, and Murali Sundaram. "Acute Calcific Retropharyngeal Tendinitis." Orthopedics 29, no. 7 (July 1, 2006): 561, 650–651. http://dx.doi.org/10.3928/01477447-20060701-17.

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46

Johnston, Christopher A. M., J. Preston Wiley, David M. Lindsay, and David A. Wiseman. "Iliopsoas Bursitis and Tendinitis." Sports Medicine 25, no. 4 (1998): 271–83. http://dx.doi.org/10.2165/00007256-199825040-00005.

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Sevier, Thomas L., Julie K. Wilson, Robert H. Helfst, and Sue A. Stover. "Tendinitis: A Critical Review." Critical Reviews in Physical and Rehabilitation Medicine 12, no. 2 (2000): 119–30. http://dx.doi.org/10.1615/critrevphysrehabilmed.v12.i2.30.

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48

&NA;. "Fluoroquinolone use provokes tendinitis." Reactions Weekly &NA;, no. 1122 (October 2006): 5. http://dx.doi.org/10.2165/00128415-200611220-00014.

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49

Christopher Patton, W., and George M. McCluskey. "BICEPS TENDINITIS AND SUBLUXATION." Clinics in Sports Medicine 20, no. 3 (July 2001): 505–29. http://dx.doi.org/10.1016/s0278-5919(05)70266-0.

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50

Greenfield, Garry, and William D. Stanish. "Tendinitis and tendon ruptures." Operative Techniques in Sports Medicine 2, no. 1 (January 1994): 9–17. http://dx.doi.org/10.1016/s1060-1872(10)80004-8.

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Journal articles on the topic 'Tendinitis'

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