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Relevant bibliographies by topics / Teratocarcinoma

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Academic literature on the topic 'Teratocarcinoma'

Author: Grafiati

Published: 4 June 2021

Last updated: 2 March 2023

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Journal articles on the topic "Teratocarcinoma"

1

Singhal, M., and D. Jhavar. "Primary mediastinal giant teratocarcinoma." Indian Journal of Cancer 45, no. 2 (2008): 73. http://dx.doi.org/10.4103/0019-509x.41778.

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Falcone, Richard A., Andrew W. Knott, Janice F. Rafferty, and Brad W. Warner. "Sacrococcygeal teratoma and teratocarcinoma." Seminars in Colon and Rectal Surgery 15, no. 1 (March 2004): 19–25. http://dx.doi.org/10.1053/j.scrs.2004.06.004.

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Shigeta, Hiroyuki, Michiyoshi Taga, Hideya Sakakibara, and Hiroshi Minaguchi. "Epidermal growth factor stimulates the cell growth of the PA-1 teratocarcinoma cell line in an autocrine/paracrine fashion." European Journal of Endocrinology 132, no. 2 (February 1995): 200–205. http://dx.doi.org/10.1530/eje.0.1320200.

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Shigeta H, Taga M, Sakakibara H, Minaguchi H. Epidermal growth factor stimulates the cell growth of the PA-1 teratocarcinoma cell line in an autocrine/paracrine fashion. Eur J Endocrinol 1995;132: 200–5. ISSN 0804–4643 In order to investigate the biological significance of epidermal growth factor (EGF) in the cell function of teratocarcinoma cells, we examined the production, binding and cell proliferative effect of EGF in PA-1 human ovarian teratocarcinoma cell line. The immunoreactivity of EGF in PA-1 cell-conditioned medium was detected by human EGF radioimmunoassay, and prepro-EGF mRNA was demonstrated in PA-1 cells by Northern blot analysis. An [125I] EGF binding study showed the presence of EGF receptor with very high binding affinity and relatively low numbers of binding sites in PA-1 cells. Furthermore, the growth of PA-1 cells was stimulated by EGF and inhibited by anti-EGF monoclonal antibody. These results suggest strongly that EGF plays an important role in controlling the growth of teratocarcinoma cells as an autocrine/paracrine growth factor. Michiyoshi Taga, Department of Obstetrics and Gynecology, Yokohama City University School of Medicine, 3–9 Fukuura, Kanazawa-ku, Yokohama 236, Japan

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Andrews, P. W., M. M. Matin, A. R. Bahrami, I. Damjanov, P. Gokhale, and J. S. Draper. "Embryonic stem (ES) cells and embryonal carcinoma (EC) cells: opposite sides of the same coin." Biochemical Society Transactions 33, no. 6 (October 26, 2005): 1526–30. http://dx.doi.org/10.1042/bst0331526.

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Embryonal carcinoma (EC) cells are the stem cells of teratocarcinomas, and the malignant counterparts of embryonic stem (ES) cells derived from the inner cell mass of blastocyst-stage embryos, whether human or mouse. On prolonged culture in vitro, human ES cells acquire karyotypic changes that are also seen in human EC cells. They also ‘adapt’, proliferating faster and becoming easier to maintain with time in culture. Furthermore, when cells from such an ‘adapted’ culture were inoculated into a SCID (severe combined immunodeficient) mouse, we obtained a teratocarcinoma containing histologically recognizable stem cells, which grew out when the tumour was explanted into culture and exhibited properties of the starting ES cells. In these features, the ‘adapted’ ES cells resembled malignant EC cells. The results suggest that ES cells may develop in culture in ways that mimic changes occurring in EC cells during tumour progression.

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5

Ferreira, L. R., C. E. E. Velano, E. C. Braga, C. C. Paula, H. Martélli-Junior, and J. J. Sauk. "Expression of Sec61alpha in F9 and P19 teratocarcinoma cells after retinoic acid treatment." Brazilian Journal of Biology 63, no. 2 (May 2003): 245–52. http://dx.doi.org/10.1590/s1519-69842003000200009.

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Nascent procollagen peptides and other secretory proteins are transported across the endoplasmic reticulum (RE) membrane through a protein-conducting channel called the translocon. Sec61alpha, a multispanning membrane translocon protein, has been implicated as essential for translocation of polypeptides chains into the cisterns of the ER. However, it is not known whether Sec61alpha is ubiquitously expressed in collagen producing teratocarcinoma cells. Furthermore, the production, expression, and utilization of Sec61alpha may depend on the cell differentiation stage. Stem cells from many cultured teratocarcinoma cell lines such as F9 and P19 cells are capable of differentiation in response to low retinoic acid concentrations. This differentiation of the tumorigenic stem cells results in tumorigenicity loss. For this study, mouse F9 and P19 teratocarcinoma cells were grown in culture medium treated with or without retinoic acid. Expression of Sec61alpha was determined by reverse trancriptase polimerase chain reaction (RT-PCR). In untreated conditions, F9 cells expressed undetected Sec61alpha amounts. It was also demonstrated that Sec61alpha expression is stimulated in F9 cells after retinoic acid treatment for 72 hours. No changes were found in Sec61alpha expression in P19 cells after retinoic acid treatment. These data indicate that the expression of Sec61alpha is enhanced with retinoic acid induced differentiation of F9 teratocarcinoma cells.

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Dyban, Pavel A. "Effect of the anticancer drug methotrexate on the ascites form of a teratocarcinoma and survival of mice." Reviews on Clinical Pharmacology and Drug Therapy 16, no. 3 (December 15, 2018): 32–35. http://dx.doi.org/10.17816/rcf16332-35.

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The dose-dependent effect of a methotrexate is established as on an ascite form of a teratocarcinoma CBA9H6, and mice-recipients, however various doses of the entered methotrexate (6; 4 and 2 mkg/g body weights of an animal), at repeated introduction at an interval of 48 and 24 hours, don't destroy all population of embrioid bodies. So, at 3-fold and even 10-fold daily injections in a dose of 2 mkg/g of body weight in an abdominal cavity of mice 1,6 ± 0,2% and 0,038 ± 0,01% embrioid bodies (ascite form of a teratocarcinoma ) remain, respectively, at survival of mice 93,0 ± 8,5% and 14,0 ± 3,0%. The morphological analysis of a mode of a differentiation of embrioid bodies retransplantated from experimental animals to intact has shown earlier that the methotrexate hasn't had effect on histoblastic potentialities of stem cells of a teratocarcinoma CBA9H6.

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7

Niwa, O. "Suppression of the hypomethylated Moloney leukemia virus genome in undifferentiated teratocarcinoma cells and inefficiency of transformation by a bacterial gene under control of the long terminal repeat." Molecular and Cellular Biology 5, no. 9 (September 1985): 2325–31. http://dx.doi.org/10.1128/mcb.5.9.2325-2331.1985.

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The Moloney leukemia virus (M-MuLV) genome was introduced into undifferentiated teratocarcinoma cells by transfection of a plasmid with the virus genome linked to pSV2neo, which carries a bacterial drug resistance gene, neo, or by cotransfection with pSV2neo. In the resulting cells, the M-MuLV genome remained hypomethylated, but its expression was suppressed in cells in an undifferentiated state. The pattern of DNA methylation of the viral genome remained unchanged when the cells were induced to differentiate into epithelial tissues. However, spontaneous M-MuLV expression was detected with differentiation of the cells. To determine to what extent the viral long terminal repeat (LTR) was responsible for this suppression in undifferentiated cells, I constructed plasmids in which neo was placed under the control of the promoter sequence of the dihydrofolate reductase gene or the M-MuLV LTR, and compared the biological activities of the plasmids in Ltk- cells and in undifferentiated teratocarcinoma cells. In Ltk- cells, these plasmids were highly efficient in making the cells resistant to selection by G418. However, in undifferentiated teratocarcinoma cells, the M-MuLV LTR promoted neo gene expression at only 10% of the expected efficiency, as compared with the expression of the neo gene under the control of the simian virus to or dihydrofolate reductase promoter. Thus, the mechanisms of gene regulation are not the same in undifferentiated and differentiated teratocarcinoma cells.

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Niwa, O. "Suppression of the hypomethylated Moloney leukemia virus genome in undifferentiated teratocarcinoma cells and inefficiency of transformation by a bacterial gene under control of the long terminal repeat." Molecular and Cellular Biology 5, no. 9 (September 1985): 2325–31. http://dx.doi.org/10.1128/mcb.5.9.2325.

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The Moloney leukemia virus (M-MuLV) genome was introduced into undifferentiated teratocarcinoma cells by transfection of a plasmid with the virus genome linked to pSV2neo, which carries a bacterial drug resistance gene, neo, or by cotransfection with pSV2neo. In the resulting cells, the M-MuLV genome remained hypomethylated, but its expression was suppressed in cells in an undifferentiated state. The pattern of DNA methylation of the viral genome remained unchanged when the cells were induced to differentiate into epithelial tissues. However, spontaneous M-MuLV expression was detected with differentiation of the cells. To determine to what extent the viral long terminal repeat (LTR) was responsible for this suppression in undifferentiated cells, I constructed plasmids in which neo was placed under the control of the promoter sequence of the dihydrofolate reductase gene or the M-MuLV LTR, and compared the biological activities of the plasmids in Ltk- cells and in undifferentiated teratocarcinoma cells. In Ltk- cells, these plasmids were highly efficient in making the cells resistant to selection by G418. However, in undifferentiated teratocarcinoma cells, the M-MuLV LTR promoted neo gene expression at only 10% of the expected efficiency, as compared with the expression of the neo gene under the control of the simian virus to or dihydrofolate reductase promoter. Thus, the mechanisms of gene regulation are not the same in undifferentiated and differentiated teratocarcinoma cells.

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9

Blackshear, P., J. Mahler, L. M. Bennett, K. A. McAllister, D. Forsythe, and B. J. Davis. "Extragonadal Teratocarcinoma in Chimeric Mice." Veterinary Pathology 36, no. 5 (September 1999): 457–60. http://dx.doi.org/10.1354/vp.36-5-457.

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Acharya, Utkarsh, Stephen Strobel, Linda Pepe, and Ranko Miocinovic. "Teratocarcinoma Presenting as Testicular Torsion." Journal of Histotechnology 31, no. 4 (December 2008): 175–77. http://dx.doi.org/10.1179/his.2008.31.4.175.

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Dissertations / Theses on the topic "Teratocarcinoma"

1

Mason, I. J. "Gene expression during mouse development and teratocarcinoma differentiation." Thesis, Imperial College London, 1987. http://hdl.handle.net/10044/1/47226.

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Bonner, Allison E. "Organ development and tumorigenesis a molecular link /." Columbus, Ohio : Ohio State University, 2003. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=osu1073936508.

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Thesis (Ph.D.)--Ohio State University, 2003.
Title from first page of PDF file. Document formatted into pages; contains xviii, 183 p.; also includes graphics (some col.). Includes abstract and vita. Co-advisors: , Ming You and Christoph Plass, Dept. of Medical Microbiology and Immunology. Includes bibliographical references (p. 172-183).

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Smith, Janet. "The developmental genetics of mouse teratocarcinoma and embryonal cells." Thesis, University of Edinburgh, 1985. http://hdl.handle.net/1842/20199.

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Roach, Sherry L. "The molecular analysis of the differentiation of human testicular teratocarcinoma." Thesis, University of Oxford, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.239291.

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McGowan-Jordan, I. Jean. "Suppression of tumorigenicity of the PA-1 human teratocarcinoma cell line." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk2/tape16/PQDD_0011/NQ28358.pdf.

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6

Su, Dan. "Microarray screening and identification of RARgamma regulated genes in F9 teratocarcinoma stem cells /." Access full-text from WCMC:, 2007. http://proquest.umi.com/pqdweb?did=1428838881&sid=1&Fmt=2&clientId=8424&RQT=309&VName=PQD.

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7

Gorbea, Carlos M. "Glycolipids in mouse F9 teratocarcinoma cells : some changes associated with retinoic acid-induced differentiation /." Thesis, This resource online, 1991. http://scholar.lib.vt.edu/theses/available/etd-08142009-040425/.

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8

Gillespie, Robert Francis. "Transcriptional regulation of retinoic acid : responsive genes in F9 wild type and F9 retinoic acid receptor deficient teratocarcinoma stem cells /." Access full-text from WCMC:, 2007. http://proquest.umi.com/pqdweb?did=1432771291&sid=2&Fmt=2&clientId=8424&RQT=309&VName=PQD.

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9

Bonner, Allison E. "Organ development and tumorigenesis: a molecular link." The Ohio State University, 2004. http://rave.ohiolink.edu/etdc/view?acc_num=osu1073936508.

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Podrygajlo, Grzegorz. "Differentiation of human teratocarcinoma cell line into motor neurons: investigation of cellular phenotype in vitro and in transplantation studies." Hannover Bibliothek der Tierärztlichen Hochschule Hannover, 2009. http://d-nb.info/1000125572/34.

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Books on the topic "Teratocarcinoma"

1

J, Robertson E., ed. Teratocarcinomas and embryonic stem cells: A practical approach. Oxford: IRL Press, 1987.

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2

Embryonal stem cells: Introducing planned changes into the animal germline. Chur, Switzerland: Harwood Academic, 1992.

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3

1942-, Harris Stephen E., and Mansson Per-Erik, eds. Cellular factors in development and differentiation: Embryos, teratocarcinomas, and differentiated tissues : proceedings of the Third International Symposium on Cellular Endocrinology, held at Lake Placid, New York, August 30-September 2, 1987. New York: Liss, 1988.

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4

Robertson, E. J. Teratocarcinomas and Embryonic Stem Cells: A Practical Approach (Practical Approach Series). Oxford University Press, USA, 1986.

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5

Robertson, E. J. Teratocarcinomas and Embryonic Stem Cells: A Practical Approach (The Practical approach series). Oxford University Press, USA, 1986.

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6

Cellular factors in development and differentiation: Embryos, teratocarcinomas, and differentiated tissues : Proceedings of the Third International Symposium ... in clinical and biological research). Liss, 1988.

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Book chapters on the topic "Teratocarcinoma"

1

Schindler, Joel, and Michael Kelly. "Retinoids, Polyamines and Teratocarcinoma Differentiation." In Vitamins and Cancer, 19–33. Totowa, NJ: Humana Press, 1986. http://dx.doi.org/10.1007/978-1-4612-5006-7_2.

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2

Datta, Prasun K. "Murine Teratocarcinoma-Derived Neuronal Cultures." In Neuronal Cell Culture, 35–44. Totowa, NJ: Humana Press, 2013. http://dx.doi.org/10.1007/978-1-62703-640-5_4.

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3

Datta, Prasun K. "Murine Teratocarcinoma-Derived Neuronal Cultures." In Neuronal Cell Culture, 39–49. New York, NY: Springer US, 2021. http://dx.doi.org/10.1007/978-1-0716-1437-2_4.

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4

Andrews, Peter W., and Ivan Damjanov. "Immunochemistry of Human Teratocarcinoma Stem Cells." In Monoclonal Antibodies in Cancer, 339–64. Totowa, NJ: Humana Press, 1985. http://dx.doi.org/10.1007/978-1-4612-5176-7_15.

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5

Takeichi, Masatoshi. "Molecular Basis for Teratocarcinoma Cell-Cell Adhesion." In The Cellular Basis of Morphogenesis, 373–88. Boston, MA: Springer US, 1986. http://dx.doi.org/10.1007/978-1-4613-2141-5_9.

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6

Cole, Gregory M., and Paola S. Timiras. "Aging-Related Pathology in Human Neuroblastoma and Teratocarcinoma Cell Lines." In Model Systems of Development and Aging of the Nervous System, 453–73. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4613-2037-1_30.

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7

Okada, T. S., Y. Takahashi, and H. Kondoh. "Expression of the Exogenous Chicken δ-Crystallin Genes Incorporated in Mouse Teratocarcinoma Cells." In Coordinated Regulation of Gene Expression, 183–91. Boston, MA: Springer US, 1986. http://dx.doi.org/10.1007/978-1-4613-2245-0_16.

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8

Rusciano, Dario, Patrizia Lorenzoni, and Max M. Burger. "Liver or lung colonization by F9 teratocarcinoma cells follows specific interactions with the target organ." In Experientia Supplementum, 272–76. Basel: Birkhäuser Basel, 1992. http://dx.doi.org/10.1007/978-3-0348-7001-6_43.

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9

Hohjoh, Hirohiko. "MicroRNA Expression During Neuronal Differentiation of Human Teratocarcinoma NTera2D1 and Mouse Embryonic Carcinoma P19 Cells." In MicroRNA Protocols, 257–69. Totowa, NJ: Humana Press, 2012. http://dx.doi.org/10.1007/978-1-62703-083-0_20.

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10

Barry Pierce, G., and Ralph E. Parchment. "Progression in Teratocarcinomas." In Boundaries between Promotion and Progression during Carcinogenesis, 71–81. Boston, MA: Springer US, 1991. http://dx.doi.org/10.1007/978-1-4684-5994-4_7.

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Conference papers on the topic "Teratocarcinoma"

1

Rasoul-Pelińska, K., M. Czech, M. Bobiński, J. Kotarski, A. Czekierdowski, and W. Bednarek. "EP460 Uncommon case of fallopian tubes teratocarcinomas in 36-year-old female." In ESGO Annual Meeting Abstracts. BMJ Publishing Group Ltd, 2019. http://dx.doi.org/10.1136/ijgc-2019-esgo.519.

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