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Journal articles on the topic 'Teratogens'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2025

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1

Killeen, Alexis, and Caralina Marin de Evsikova. "Effects of sub-lethal teratogen exposure during larval development on egg laying and egg quality in adult Caenorhabditis elegans." F1000Research 5 (December 28, 2016): 2925. http://dx.doi.org/10.12688/f1000research.8934.1.

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Background: Acute high dose exposure to teratogenic chemicals alters the proper development of an embryo leading to infertility, impaired fecundity, and few viable offspring. However, chronic exposure to sub-toxic doses of teratogens during early development may also have long-term impacts on egg quality and embryo viability. Methods: To test the hypothesis that low dose exposure during early development can impact long-term reproductive health, Caenorhabditis elegans larvae were exposed to 10 teratogens during larval development, and subsequently were examined for the pattern of egg-laying an

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2

Mishal Suhail, Bushra Ameer Saeed Awan, Amna Shamim, and Uswa Zeb. "Maternal Knowledge of Antenatal Care, Teratogens and Patient Satisfaction in Tertiary Care Hospitals of Rawalpindi /Islamabad: a Cross-Sectional Analytical Study." Pakistan Armed Forces Medical Journal 75, no. 1 (2025): 188–92. https://doi.org/10.51253/pafmj.v75i1.12922.

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Objective: To assess maternal knowledge of antenatal care teratogens and to determine patient satisfaction among women visiting tertiary care hospitals of Rawalpindi/Islamabad. Study Design: Cross-sectional analytical study. Place and Duration of Study: Two Tertiary Care Hospitals of Rawalpindi/ Islamabad, Pakistan from Jan to May 2023. Methodology: The study was conducted on pregnant females visiting tertiary care hospitals in Rawalpindi and Islamabad from January to May 2023. Participants aged 18-45 were recruited through a convenience sampling technique. A pre-tested questionnaire comprisin

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3

STEVENSON, AUDREY M. "Teratogens." MCN, The American Journal of Maternal/Child Nursing 23, no. 6 (1998): 333. http://dx.doi.org/10.1097/00005721-199811000-00011.

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4

Kuller, Jeffrey A., and Jerome Yankowitz. "Viral Teratogens." Postgraduate Obstetrics & Gynecology 17, no. 20 (1997): 1. http://dx.doi.org/10.1097/00256406-199717200-00001.

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5

Morgan, Thomas M., Deborah P. Jones, and William O. Cooper. "Renal Teratogens." Clinics in Perinatology 41, no. 3 (2014): 619–32. http://dx.doi.org/10.1016/j.clp.2014.05.010.

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6

Pooh, Ritsuko, Andrew Matthys, and Maria Teresa Ambat. "Psychotropic Medication Use during Pregnancy and Lactation: Role of Ultrasound Assessment." Donald School Journal of Ultrasound in Obstetrics and Gynecology 8, no. 1 (2014): 109–21. http://dx.doi.org/10.5005/jp-journals-10009-1345.

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ABSTRACT The potential teratogenic effects of psychotropic medication use during pregnancy have been scrutinized ever since thalidomide contributed to phocomelia in the 1950's. Up to 35% of the half a million women becoming pregnant each day take psychotropic medication(s). Teratogens alter normal intrauterine fetal growth, anatomic structures, functioning and postnatal development. In this educational review article, we examine whether different classes of psychotropic medications have the potential to be teratogens or have other nonteratogenic effects. Specifically, we examined antidepressan

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7

NAVA-OCAMPO, ALEJANDRO A., and GIDEON KOREN. "Human Teratogens and Evidence-based Teratogen Risk Counseling: The Motherisk Approach." Clinical Obstetrics and Gynecology 50, no. 1 (2007): 123–31. http://dx.doi.org/10.1097/grf.0b013e31802f1880.

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8

Soprano, Dianne Robert, and Kenneth J. Soprano. "Retinoids as Teratogens." Annual Review of Nutrition 15, no. 1 (1995): 111–32. http://dx.doi.org/10.1146/annurev.nu.15.070195.000551.

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9

Schardein, James L. "Naturally Occurring Teratogens." Journal of Toxicology: Toxin Reviews 15, no. 4 (1996): 369–91. http://dx.doi.org/10.3109/15569549609064089.

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10

Holmes, Lewis B. "Identifying human teratogens." Reproductive Toxicology 80 (September 2018): 137. http://dx.doi.org/10.1016/j.reprotox.2018.07.018.

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11

Oberst, Richard D. "Viruses as Teratogens." Veterinary Clinics of North America: Food Animal Practice 9, no. 1 (1993): 23–31. http://dx.doi.org/10.1016/s0749-0720(15)30668-x.

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12

Mills, James L., and Duane Alexander. "Teratogens and Litogens." New England Journal of Medicine 315, no. 19 (1986): 1234–36. http://dx.doi.org/10.1056/nejm198611063151925.

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13

THOMSON, ELIZABETH J., and JOSE F. CORDERO. "The New Teratogens." MCN, The American Journal of Maternal/Child Nursing 14, no. 4 (1989): 244???248. http://dx.doi.org/10.1097/00005721-198907000-00006.

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14

Holmes, Lewis B. "Putative new teratogens." Teratology 65, no. 5 (2002): 204–5. http://dx.doi.org/10.1002/tera.10056.

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15

Paredes-Páliz, Karina, Joselyn Armendáriz-Ramos, Anabell Urbina Salazar, and Alberto Renato Inca Torres. "The most common teratogens as factors of mutability: A literature review." Salud, Ciencia y Tecnología 4 (June 30, 2024): 1098. http://dx.doi.org/10.56294/saludcyt20241098.

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Introduction: The teratogenic agents during the embryonic period can result in disorders in organ differentiation. Concerns regarding medication use during pregnancy heightened after the thalidomide case in the 1960s, underscoring the need for a balance between caution and effective treatment, given the lack of clear information on risks. The aim of the research was to understand teratogens as factors that increase mutability and highlight the relevance of preventive strategies to ensure fetal and maternal health. Methods: Literature review using the PubMed database, Scopus and Web of Science

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16

Hurst, Helena S., Richard H. Clothier, and Margaret Pratten. "An Evaluation of a Novel Chick Cardiomyocyte Micromass Culture Assay with Two Teratogens/Embryotoxins Associated with Heart Defects." Alternatives to Laboratory Animals 35, no. 5 (2007): 505–14. http://dx.doi.org/10.1177/026119290703500510.

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This study was aimed at determining whether the chick cardiomyocyte micromass (MM) system could be employed to predict the teratogenicity/embryotoxicity of exogenous chemicals. Two documented teratogens/embryotoxins, sodium valproate (the sodium salt of valproic acid; VPA) and all -trans retinoic acid (tRA), were used in the initial phase of the study. White Leghorn 5-day-old embryo hearts were dissociated to produce a cardiomyocyte suspension in Dulbecco's Modified Eagle's Medium. Cultures were incubated at 37°C in 5% CO2 in air, and observations were made every 24 hours over 5 days, for the

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17

Amigo, Ryan, John Leones Damo, Nestor V. De Leon, Roostvelt Daniel D. Eteroza, and Adonis B. Daliri. "Level of Knowledge of Mothers on the Effects of Teratogens A Basis for Pre-Natal Health Information Program." Jurnal Keperawatan Respati Yogyakarta 10, no. 3 (2023): 175. http://dx.doi.org/10.35842/jkry.v10i3.755.

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The setting will have an effect on the child in several well-documented ways. There are a lot of babies that are born defective as the result of events before and during the course of their pregnancy and such circumstances are classified as teratogens. Teratogens are environmental agents that will disturb the conventional fetal development once the mother is exposed to them, leading to prenatal maldevelopment or death (Kalter, 2010). This study made used a descriptive survey design as a method of research. A descriptive design is a type of quantitative research. The main source of data for thi

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18

Young, D. L., R. A. Schneider, D. Hu, and J. A. Helms. "Genetic and Teratogenic Approaches To Craniofacial Development." Critical Reviews in Oral Biology & Medicine 11, no. 3 (2000): 304–17. http://dx.doi.org/10.1177/10454411000110030201.

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Craniofacial malformations are the most common birth defects that occur in humans, with facial clefting representing the majority of these defects. Facial clefts can arise at any stage of development due to perturbations that alter the extracellular matrix as well as affect the patterning, migration, proliferation, and differentiation of cells. In this review, we focus on recent advances in the understanding of the developmental basis for facial clefting through the analysis of the effects of gene disruption experiments and treatments with teratogens in both chickens and mice. Specifically, we

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19

Magdum, Jyoti* Shelake Sardar Patil S. S. Narvekar Namita. "SAFE USE OF MEDICINES IN PREGNANCY." INDO AMERICAN JOURNAL OF PHARMACEUTICAL SCIENCES 05, no. 05 (2018): 4799–806. https://doi.org/10.5281/zenodo.1279512.

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<em>Pregnancy is a special physiological condition which affects the pharmacokinetics of medicines used and certain medications reach to fetal circulation which may cause adverse effects and also teratogenic effects. But complete avoidance of medical treatment during pregnancy is not possible because some women enter pregnancy with some medical problem or some are arises during gestation period. In this review we summarize basic principles of teratology, classification of drugs according to FDA and provide information regarding safe and effective use of medication during pregnancy.</em> <stron

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20

Clowse, Megan EB, Amanda M. Eudy, Jessica Revels, Gillian D. Sanders, and Lisa Criscione-Schreiber. "Rheumatologists’ knowledge of contraception, teratogens, and pregnancy risks." Obstetric Medicine 11, no. 4 (2018): 182–85. http://dx.doi.org/10.1177/1753495x18771266.

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Introduction Rheumatologists are essential partners in planning and managing pregnancies in women with lupus. Whether they know the essentials of contraceptive and medical treatment in pregnancy, however, is unknown. Method Anonymous in-lecture surveys were completed by 270 rheumatologists to assess knowledge of contraceptive effectiveness, emergency contraception, medication teratogenicity, and lupus pregnancy risk assessment. Results Rheumatologists knew the high effectiveness of the intrauterine device, but over-estimated the effectiveness of injectable medroxyprogesterone and condoms. Almo

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21

Blagosklonny, Mikhail V. "Teratogens as Anticancer Drugs." Cell Cycle 4, no. 11 (2005): 1518–21. http://dx.doi.org/10.4161/cc.4.11.2208.

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22

Diav-Citrin, Orna, and Asher Ornoy. "Are SSRIs human teratogens?" Reproductive Toxicology 31, no. 2 (2011): 252. http://dx.doi.org/10.1016/j.reprotox.2010.12.067.

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23

Sepkowitz, Samuel. "TERATOGENS AND PERINATAL EPIDEMIOLOGY." Lancet 331, no. 8594 (1988): 1115. http://dx.doi.org/10.1016/s0140-6736(88)91939-3.

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24

Frías, Jaime L., and Enid Gilbert-Barness. "Human Teratogens: Current Controversies." Advances in Pediatrics 55, no. 1 (2008): 171–211. http://dx.doi.org/10.1016/j.yapd.2008.07.005.

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25

Holmes, Lewis B. "Human teratogens: Update 2010." Birth Defects Research Part A: Clinical and Molecular Teratology 91, no. 1 (2011): 1–7. http://dx.doi.org/10.1002/bdra.20748.

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26

Sulik, K. K., C. S. Cook, and W. S. Webster. "Teratogens and craniofacial malformations: relationships to cell death." Development 103, Supplement (1988): 213–32. http://dx.doi.org/10.1242/dev.103.supplement.213.

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Environmental agents including ethanol, 13-cis retinoic acid (RA, Accutane®), the antimetabolite methotrexate, periods of hypoxia, ionizing radiation or hyperthermic stress, when administered acutely to pregnant experimental animals, induce stage-dependent craniofacial malformations comparable to those in corresponding human teratogen syndromes. Acute treatment regimens have allowed analysis of cell populations initially affected and subsequent dysmorphogenetic sequences as well as speculation relative to mechanisms of teratogenesis. In rodent models, ethanol and RA appear to affect similar ce

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27

Cera, Alessandra, Simona Ceschin, Floriano Del Grosso, Lorenzo Traversetti, and Massimiliano Scalici. "Correlating ecotoxicological early-warning systems to biotic indices to assess riverine teratogenic contamination." Marine and Freshwater Research 71, no. 8 (2020): 1033. http://dx.doi.org/10.1071/mf18471.

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We constructed a new ecotoxicological tool to test for the presence of teratogens in rivers by exploiting the regenerative capabilities of Hydra vulgaris because using living organisms is considered good practice in environmental monitoring for detecting detrimental pressures. Outputs on the Hydra assay were integrated with two biotic indices based on benthic invertebrates and macrophytes to evaluate whether the assay acts as a tool for detecting generalised freshwater conditions or as a biological test model revealing only the effects of dissolved teratogens. Several hypostomae of H. vulgaris

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28

Brent, Robert L. "Environmental Causes of Human Congenital Malformations: The Pediatrician’s Role in Dealing With These Complex Clinical Problems Caused by a Multiplicity of Environmental and Genetic Factors." Pediatrics 113, Supplement_3 (2004): 957–68. http://dx.doi.org/10.1542/peds.113.s3.957.

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There have been amazing advances in embryology, teratology, reproductive biology, genetics, and epidemiology in the past 50 years that have provided scientists and clinicians with a better perspective on the causes of congenital malformations. We still cannot provide the families of children with malformations a definitive diagnosis and cause in every instance. The purpose of this article is to inform pediatricians about environmental drugs, chemicals, and physical agents that have been documented to produce congenital malformations and reproductive effects and to indicate that the multitude o

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29

Pekar, Olga, Nataly Molotski, Shoshana Savion, Amos Fein, Vladimir Toder та Arkady Torchinsky. "p53 regulates cyclophosphamide teratogenesis by controlling caspases 3, 8, 9 activation and NF-κB DNA binding". Reproduction 134, № 2 (2007): 379–88. http://dx.doi.org/10.1530/rep-07-0086.

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The tumor suppressor protein p53 regulates the sensitivity of embryos to such human teratogens as ionizing radiation, diabetes, and cytostatics. Yet, the molecular mechanisms whereby it fulfills this function remain undefined. We used p53 heterozygous (p53+/−) female mice mated with p53+/− males and then exposed to cyclophosphamide (CP) to test whether caspases 3, 8, and 9 and the transcription factor nuclear factor (NF)-κB may serve as p53 targets. Mice were exposed to CP on day 12 of pregnancy and killed on days 15 and 18 of pregnancy to evaluate CP-induced teratogenic effect. The brain and

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30

Schuler, R. L., M. A. Radike, B. D. Hardin, and R. W. Niemeier. "Pattern of Response of Intact Drosophila to Known Teratogens." Journal of the American College of Toxicology 4, no. 4 (1985): 291–303. http://dx.doi.org/10.3109/10915818509078681.

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Drosophila, as the test organism, was used to assess 17 chemicals. The teratogenic potential of 15 of these chemicals is well established from animal studies or human epidemiology. The test involves examination of adult flies following treatment during larval stages of development. Flies are examined for abnormal external morphology. The incidence of abnormalities in treated and control populations is compared using the Chi-square test. All 17 chemicals were active to varying degrees in the test system. Most chemicals produced a unique response yielding individual patterns of abnormalities. Th

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31

Chambers, Christina, and Sura Alwan. "Identifying Human Teratogens: An Update." Journal of Pediatric Genetics 04, no. 02 (2015): 039–41. http://dx.doi.org/10.1055/s-0035-1556745.

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32

Ferguson, Lynnette R., and Judith H. Ford. "Overlap between mutagens and teratogens." Mutation Research/Fundamental and Molecular Mechanisms of Mutagenesis 396, no. 1-2 (1997): 1–8. http://dx.doi.org/10.1016/s0027-5107(97)00172-3.

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33

Bishop, Jack B., Kristine L. Witt, and Richard A. Sloane. "Genetic toxicities of human teratogens." Mutation Research/Fundamental and Molecular Mechanisms of Mutagenesis 396, no. 1-2 (1997): 9–43. http://dx.doi.org/10.1016/s0027-5107(97)00173-5.

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34

Faustman, E. M. "Short-term tests for teratogens." Mutation Research/Genetic Toxicology 205, no. 1-4 (1988): 355–84. http://dx.doi.org/10.1016/0165-1218(88)90027-4.

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35

Lynch, Tara A., and David E. Abel. "Teratogens and Congenital Heart Disease." Journal of Diagnostic Medical Sonography 31, no. 5 (2015): 301–5. http://dx.doi.org/10.1177/8756479315598524.

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36

Eadie, Mervyn J. "Antiepileptic drugs as human teratogens." Expert Opinion on Drug Safety 7, no. 2 (2008): 195–209. http://dx.doi.org/10.1517/14740338.7.2.195.

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37

Shepard, Thomas H. "Annual commentary on human teratogens." Teratology 66, no. 6 (2002): 275–77. http://dx.doi.org/10.1002/tera.10111.

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38

Rasmussen, Sonja A. "Identifying teratogens: Challenges and opportunities." Neurotoxicology and Teratology 109 (May 2025): 107453. https://doi.org/10.1016/j.ntt.2025.107453.

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39

Khoury, Muin J., José E. Becerra, José F. Cordero, and J. David Erickson. "Clinical-Epidemiologic Assessment of Patterns of Birth Defects Associated With Human Teratogens: Application to Diabetic Embryopathy." Pediatrics 84, no. 4 (1989): 658–65. http://dx.doi.org/10.1542/peds.84.4.658.

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The concepts of sensitivity, specificity, and predictive value can be used to assess patterns of birth defects associated with human teratogens. Although sensitivity of any single defect is generally low for many known teratogens, the presence of specific defect combinations is usually predictive of the teratogen. To evaluate the patterns of birth defects associated with diabetic embryopathy, a sensitivity-specificity analysis was performed on 4929 infants with major defects ascertained by the population-based Metropolitan Atlanta Congenital Defects Program between 1968 and 1980. By reviewing

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40

Renyoet, Agustinus, and Raynard C. Sanito. "Uji Teratogen Ekstrak Akar Jarong (Stachytarpheta jamaicensis (L.) Vahl.) terhadap Sistem Reproduksi Mencit (Mus musculus L.) Betina." JURNAL BIOLOGI PAPUA 3, no. 1 (2018): 32–38. http://dx.doi.org/10.31957/jbp.546.

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This study aim to determine the teratogens effect of jarong (Stachytarpheta jamaicensis (L.) Vahl) roots extract on the reproductive system of mice (Mus musculus L.) female Swiss Webster organogenesis during pregnancy.The method used in this study is the method of extraction and examination of vaginal smears.. The experiment was conducted in August-December 2010, at the Laboratory of Zoology, Department of Biology Cenderawasih University, Jayapura. The Experiment was carried out by dividing the 20 female mice into 4 groups. One group is a control group and with no treatmen and the other 3 grou

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41

Goldberg, Joel S. "Monitoring Maternal Beta Carotene and Retinol Consumption May Decrease the Incidence of Neurodevelopmental Disorders in Offspring." Clinical Medicine Insights: Reproductive Health 6 (December 19, 2011): CMRH.S8372. http://dx.doi.org/10.4137/cmrh.s8372.

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Retinoic acids (13-cis and 13-trans) are known teratogens, and their precursor is retinol, a form of vitamin A. In 1995, Rothman et al demonstrated an association between excessive vitamin A, > 10,000 IU/day, during the first trimester of pregnancy and teratogenic effects, particularly in the central nervous system. However, vitamin A deficiency has long been known to be deleterious to the mother and fetus. Therefore, there may be a narrow therapeutic ratio for vitamin A during pregnancy that has not previously been fully appreciated. Neurodevelopmental disorders may not be apparent by macr

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42

Silva, Natália Rodrigues da, Wanderson Alves Ribeiro, Carleone Vieira dos Santos Neto, et al. "Congenital anomalies in newborns of mothers exposed to teratogenic agents during pregnancy." Concilium 23, no. 9 (2023): 185–96. http://dx.doi.org/10.53660/clm-1314-23k41.

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Congenital modifications are characterized by an interaction between chemical, physical agents or pathogenic organisms, with the embryo in formation, with or without hereditary influence, being conceptualized mainly by changes in cell growth and multiplication, which are called teratogenic agents. . The study aimed to search the literature for the relationship between the main teratogenic agents and fetal congenital anomalies. This is a systematic review of the literature, in which a search was carried out in the following databases: LILACS and BDENF via VHL; MEDLINE via Pubmed, and the Scielo

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43

Clowse, M., J. LI, M. Birru Talabi, A. Eudy, and G. Schmajuk. "FRI0510 THE FREQUENCY OF CONTRACEPTION DOCUMENTATION IN WOMEN ON AND OFF TERATOGENIC ANTI-RHEUMATIC MEDICATIONS IN THE RISE REGISTRY." Annals of the Rheumatic Diseases 79, Suppl 1 (2020): 853.2–854. http://dx.doi.org/10.1136/annrheumdis-2020-eular.5762.

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Background:Several of the most commonly prescribed medications for women with rheumatic disease are teratogens, posing a risk for pregnancy loss and birth defects if taken in pregnancy. To prevent these life-altering complications, it is important that all women taking teratogenic medications avoid pregnancy through abstinence or contraception.Objectives:We sought to understand the accessibility to contraceptive data within the RISE Registry and to test whether, compared to other women, those prescribed a teratogen would be more likely to have documentation of contraceptive.Methods:Data were d

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44

DASTON, GEORGE P., BLAIR F. REHNBERG, BRENDA CARVER, ELLEN H. ROGERS, and ROBERT J. KAVLOCK. "Functional Teratogens of the Rat Kidney." Toxicological Sciences 11, no. 1 (1988): 381–400. http://dx.doi.org/10.1093/toxsci/11.1.381.

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45

Schardein, James L. "Teratogens. Chemicals which cause birth defects." Reproductive Toxicology 10, no. 1 (1996): 88. http://dx.doi.org/10.1016/s0890-6238(96)90002-7.

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46

Scialli, Anthony R. "Identifying teratogens: The tyranny of lists." Reproductive Toxicology 11, no. 4 (1997): 555–59. http://dx.doi.org/10.1016/s0890-6238(97)89174-5.

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47

Scialli, Anthony R. "Old teratogens: Are they still important?" Reproductive Toxicology 1, no. 4 (1987): 241. http://dx.doi.org/10.1016/0890-6238(87)90014-1.

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48

Scialli, Anthony R. "Teratogens, chemicals which cause birth defects." Reproductive Toxicology 5, no. 5 (1991): 459. http://dx.doi.org/10.1016/0890-6238(91)90012-5.

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49

Short, Angela L. "Teratogens, chemicals which cause birth defects." Toxicology Letters 78, no. 2 (1995): 173. http://dx.doi.org/10.1016/0378-4274(95)90243-0.

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50

Selig, Brady P., James R. Furr, Ryan W. Huey, et al. "Cancer chemotherapeutic agents as human teratogens." Birth Defects Research Part A: Clinical and Molecular Teratology 94, no. 8 (2012): 626–50. http://dx.doi.org/10.1002/bdra.23063.

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