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1

Seif, Gamal I., Joshua C. Teichman, Kesava Reddy, Charmaine Martin, and Amadeo R. Rodriguez. "Incidence, Morbidity, and Mortality of Terson Syndrome in Hamilton, Ontario." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 41, no. 5 (September 2014): 572–76. http://dx.doi.org/10.1017/cjn.2014.7.

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AbstractObjectiveEvaluate the incidence, neurologic morbidity, and mortality of patients with Terson syndrome.MethodsConsecutive patients admitted to the Hamilton General Hospital from May 2012 to May 2013 with a diagnosis of spontaneous subarachnoid hemorrhage (SAH) were recruited. Funduscopic examinations were performed under pharmacological mydriasis. Outcome measures included: (1) the presence or absence of Terson syndrome; (2) The Glasgow Coma Scale (GCS), Hunt and Hess scale (H&H), and SAH Fisher score upon admission to the hospital; (3) the modified Rankin score upon discharge; and (4) and all-cause mortality.ResultsForty-six patients were included and 10 had Terson syndrome (21%). The median H&H, GCS, and Fisher scores were 4, 6.5, and 4.0 for patients with Terson syndrome vs. 2, 14, and 3 for patients without Terson syndrome (p=0.0032, 0.0052, and 0.031), respectively. The median Rankin score was 6 for patients with Terson syndrome vs. 3.5 for patients without Terson syndrome (p=0.0019). The odds of all-cause mortality with Terson syndrome vs. no Terson syndrome was 12: 1 (95% confidence interval 2.33-61.7), p =0.003. Only four of the 10 patients with Terson syndrome survived.ConclusionsBased on this study, approximately one-fifth of patients admitted to the hospital with a spontaneous SAH could have Terson syndrome. Patients with Terson syndrome have significantly worse GCS and H&H scores upon admission to the hospital, lower modified Rankin scores upon discharge, and greater mortality. Thus, Terson syndrome is not rare among patients with SAH and carries a worse prognosis.
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2

Ogawa, Tsukihiko, Takashi Kitaoka, Yoshinori Dake, and Tsugio Amemiya. "Terson syndrome." Ophthalmology 108, no. 9 (September 2001): 1654–56. http://dx.doi.org/10.1016/s0161-6420(01)00673-x.

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3

Mills, Monte D. "Terson syndrome." Ophthalmology 105, no. 12 (December 1998): 2161–62. http://dx.doi.org/10.1016/s0161-6420(98)91200-3.

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4

Kuhn, Ferenc, Robert Morris, C. Douglas Witherspoon, and Viktória Mester. "Terson syndrome." Ophthalmology 105, no. 3 (March 1998): 472–77. http://dx.doi.org/10.1016/s0161-6420(98)93030-5.

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5

Middleton, Kim, Peter Esselman, and P. Chuwn Lim. "Terson Syndrome." American Journal of Physical Medicine & Rehabilitation 91, no. 3 (March 2012): 271–74. http://dx.doi.org/10.1097/phm.0b013e3182328792.

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6

Troumani, Y., L. Beral, F. Glatre, E. Finke, and T. David. "Syndrome de Terson." Journal Français d'Ophtalmologie 37, no. 6 (June 2014): 501. http://dx.doi.org/10.1016/j.jfo.2013.12.011.

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7

Elmi Sadr, Navid, Bijan Samavat, Payam Mehrian, and Alireza Hedayatfar. "Intraocular Silicone Oil Masquerading as Terson Syndrome." Case Reports in Ophthalmological Medicine 2016 (2016): 1–3. http://dx.doi.org/10.1155/2016/4942109.

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Introduction. Terson syndrome is described as intraocular hemorrhage in association with any type of intracranial hemorrhage and is associated with higher mortality rate and vision loss. Intraocular hemorrhage in Terson syndrome may be diagnosed using computed tomography but there are false positive results. Silicone oil which is widely used for internal tamponade of complicated retinal detachments has high attenuation on computed tomography and hyperintensity on T1-weighted magnetic resonance imaging that can mimic intraocular hemorrhage. This report shows that silicone oil is another origin of false positive results in interpreting CT findings for detecting Terson syndrome.Case Report. A 71-year-old diabetic woman presented with loss of consciousness. Brain computed tomography revealed right cerebellar hemorrhage and ventricular hemorrhage and hyperdensity in vitreous cavity of the left eye that was initially interpreted as vitreous hemorrhage. Terson syndrome was the initial diagnosis but ophthalmoscopic examination and brain MRI showed that the left eye had silicone oil tamponade.Conclusion. Without knowing the history of previous vitreoretinal surgery, CT scan findings of intraocular silicone oil may be interpreted as vitreous hemorrhage. In patients with concomitant intracranial hemorrhage, it can masquerade as Terson syndrome.
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8

Hoving, Eelco W., Mehrnoush Rahmani, Leonie I. Los, and Victor W. Renardel de Lavalette. "Bilateral retinal hemorrhage after endoscopic third ventriculostomy: iatrogenic Terson syndrome." Journal of Neurosurgery 110, no. 5 (May 2009): 858–60. http://dx.doi.org/10.3171/2008.6.17610.

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A serious ophthalmological complication of an endoscopic third ventriculostomy that created an iatrogenic Terson syndrome is described. A patient with an obstructive hydrocephalus was treated endoscopically, but due to the inadvertent use of a pressure bag during rinsing, in combination with a blocked outflow channel, a steep rise in intracranial pressure occurred. Postoperatively the patient experienced disturbed vision caused by bilateral retinal hemorrhages, and an iatrogenic Terson syndrome was diagnosed. The pathogenesis of Terson syndrome is discussed based on this illustrative case.
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9

Karadzic, Jelena, Igor Kovacevic, Ivan Stefanovic, and Dijana Risimic. "Terson’s syndrome: A report of two cases." Srpski arhiv za celokupno lekarstvo 143, no. 9-10 (2015): 595–98. http://dx.doi.org/10.2298/sarh1510595k.

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Introduction. Vitreous or retinal hemorrhage occurring in association with subarachnoid hemorrhage is known as Terson?s syndrome. In Terson?s syndrome, intracranial hemorrhages are followed by intraocular hemorrhage, classically in the subhyaloid space, but may also include subretinal, retinal, preretinal, and vitreal collections. Vitreous hemorrhage recovery is usually spontaneous in six to 12 months, otherwise vitrectomy is considered. Outline of Cases. We report of two cases of Terson?s syndrome. The first was in a hypertensive middleaged female, following anterior communicating artery aneurismal subarachnoid hemorrhage, after postneurosurgical interventions. The second case report was of a young male who suffered from the bilateral vitreous hemorrhage after a severe traumatic brain injury. Conclusion. Terson?s syndrome should be considered in patients who had previous cerebral hemorrhage and are referred to eye specialist because of loss of vision. However, this phenomenon has only rarely been described in association with subdural and epidural hematomas or traumatic subarachnoid hemorrhage.
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10

El Kettani, M., A. Moussair, L. El Maaloum, D. Lahbil, H. Lamari, A. El Kettani, L. Rais, A. Amraoui, and K. Zaghloul. "736 Syndrome de Terson." Journal Français d'Ophtalmologie 31 (April 2008): 220. http://dx.doi.org/10.1016/s0181-5512(08)71335-4.

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11

Hedges, Thomas R. "Mechanism of Terson Syndrome." Ophthalmology 99, no. 5 (May 1992): 647. http://dx.doi.org/10.1016/s0161-6420(13)30972-5.

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12

Kuhn, Ferenc, Robert Morris, Viktória Mester, and C. Douglas Witherspoon. "Terson syndrome: Authors’ reply." Ophthalmology 105, no. 12 (December 1998): 2162–63. http://dx.doi.org/10.1016/s0161-6420(98)91201-5.

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13

Tripathy, Koushik. "Dissociated optic nerve fiber layer in a case of Terson syndrome." European Journal of Ophthalmology 30, no. 5 (June 3, 2019): NP11—NP14. http://dx.doi.org/10.1177/1120672119853465.

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Purpose: To report a case of Terson syndrome who developed dissociated optic nerve fiber layer appearance after pars plana vitrectomy. Case report: The author reports a young male patient with Terson syndrome who developed bilateral vitreous hemorrhage and sub-internal limiting membrane hemorrhage after road traffic accident. He underwent pars plana vitrectomy and removal of the already detached internal limiting membrane and the sub-internal limiting membrane hemorrhage in both eyes. In both eyes, dissociated optic nerve fiber layer was appreciated and left eye had a peculiar resemblance to the appearance of cystoid macular edema. Conclusions: Dissociated optic nerve fiber layer appearance may be noted following vitrectomy and removal of detached internal limiting membrane in patients with vitreous hemorrhage and sub-internal limiting membrane bleed due to Terson syndrome.
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14

Souza, Rodrigo, Guilherme Aguiar, Sarah Weber, Juan Flores, Mario Conti, and José Veiga. "Vitreous Hemorrhage after Aneurismal Rupture: Terson Syndrome." Arquivos Brasileiros de Neurocirurgia: Brazilian Neurosurgery 37, no. 03 (April 14, 2016): 163–66. http://dx.doi.org/10.1055/s-0036-1581085.

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Introduction Terson syndrome is described as an intraocular hemorrhage consequent to a spontaneous subarachnoid hemorrhage (SSAH). In the present article, we describe cases of patients who underwent neurosurgical treatment of ruptured cerebral aneurysm at our institution over a period of one year, and who were diagnosed with Terson syndrome. Methods The present study included patients with a diagnosis of SSAH by rupture of a cerebral aneurysm who underwent treatment in our neurosurgical service from December 2009 to December 2010. The patients were followed-up for a minimum of 20 months. We have also performed a literature review and compared the data with those available in the current literature. Results The present study included 34 patients, 18 (53%) of which underwent endovascular treatment, and 16 (47%) who underwent microsurgical clipping. In the sample, the mortality was 14.7% (5 patients), the same percentage of patients who were diagnosed with Terson Syndrome, which is an incidence of 14.7%. Regarding the ophthalmologic evaluation, all patients had vitreous hemorrhage detected by an ultrasound examination, which was unilateral in only two patients. Visual acuity improved in all patients, being incomplete in only one of them. Conclusion Terson syndrome is relatively common and is associated with higher mortality. With the existence of an effective treatment, it should be investigated in all patients with SSAH.
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15

Monteagudo, M., C. Doménech, and T. Segura. "Terson syndrome and ocular ultrasound." Neurología (English Edition) 30, no. 2 (March 2015): 132–33. http://dx.doi.org/10.1016/j.nrleng.2014.12.004.

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16

Keithahn, Mari A. Z., Steven R. Bennett, Douglas Cameron, and William F. Mieler. "Retinal Folds in Terson Syndrome." Ophthalmology 100, no. 8 (August 1993): 1187–90. http://dx.doi.org/10.1016/s0161-6420(93)31507-1.

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17

Augsten, R., E. Königsdörffer, and J. Strobel. "Surgical Approach in Terson Syndrome: Vitreous and Retinal Findings." European Journal of Ophthalmology 10, no. 4 (January 2000): 293–96. http://dx.doi.org/10.1177/112067210001000404.

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Purpose To report some interesting findings in patients with bilateral Terson syndrome. Methods We describe six eyes from three patients with Terson syndrome. Pars plana vitrectomy was performed in one eye twelve weeks, and in four eyes six months after the acute event. In one eye blood was suddenly spontaneously absorbed after four months. Results The four eyes operated six months after injury showed severe complications and final visual acuity was between light perception and 0.6. The eye with surgical attendance twelve weeks after the acute injury had an uneventful course, and final visual acuity was 0.7. Conclusions Because of severe ocular complications and with a view to early rehabilitation, vitrectomy has been recommended for eyes with bilateral Terson syndrome, without spontaneous blood resorption. Surgery should be performed in at least one eye not later than four to eight weeks after the acute injury.
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18

Iuliano, Lorenzo, Giovanni Fogliato, and Marco Codenotti. "Intrasurgical Imaging of Subinternal Limiting Membrane Blood Diffusion in Terson Syndrome." Case Reports in Ophthalmological Medicine 2014 (2014): 1–3. http://dx.doi.org/10.1155/2014/689793.

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We report a case of Terson syndrome, providing the first intrasurgical imaging of subinternal limiting membrane blood diffusion in Terson syndrome. We highlight some remarkable in vivo anatomical findings that may give a contribution to the debate about its pathogenesis. Here we hypothesize that the subretinal space might be unlikely to be a primary source of intraocular hemorrhage, and we support the two generally accepted theories about blood diffusion from the retinal vasculature or from the perivascular spaces.
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19

Severo Bem Junior, Luiz, Gustavo De Souza Andrade, Joao Ribeiro Memória Júnior, and Hildo Rocha Cirne de Azevedo Filho. "Terson sign:." Jornal Memorial da Medicina 2, no. 1 (November 30, 2020): 38–43. http://dx.doi.org/10.37085/jmmv2.n1.2020.pp.38-43.

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Terson's sign (TS) is classically defined as vitreous hemorrhage associated with subarachnoid hemorrhage of aneurysmal origin, being an important predictor of severity, indicating greater morbidity and mortality when compared to patients without the sign. The objective of this study is to review the relationship of Terson syndrome/Terson sign with the prognosis of aneurysmal subarachnoid hemorrhage. A search for original articles, research and case reports was performed on the PubMed, Scielo, Cochrane and ScienceDirect platform, with the following descriptors: Terson sign and subarachnoid hemorrhage. Retrospective, prospective articles and case reports published in the last 5 years and which were in accordance with the established objective and inclusion criteria were selected. Ten (10) articles were selected, in which the available results show an unfavorable prognostic relationship of TS and subarachnoid hemorrhage, because these patients had a worse clinical status assessed on the Glasgow scales ≤ 8, Hunt & Hess > III, Fisher > 3, in addition to intracranial hypertension and location of the aneurysm in the anterior communicating artery complex. The early recognition of this condition described by Albert Terson in 1900 brought an important contribution to neurosurgery, being recognized until nowadays.
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20

Burgos-Blasco, B., F. J. Moreno-Morillo, S. Hernández-Ruiz, A. Valverde-Megías, F. Sáenz-Francés, and E. Santos-Bueso. "Terson Syndrome: Vitrectomy vs Nd:YAG hyaloidotomy." Journal Français d'Ophtalmologie 42, no. 6 (June 2019): e263-e266. http://dx.doi.org/10.1016/j.jfo.2018.11.017.

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21

Rosenvald, Olga R., and Sashank Prasad. "Computed Tomography Diagnosis of Terson Syndrome." Neurohospitalist 7, no. 2 (June 22, 2016): 100–101. http://dx.doi.org/10.1177/1941874416648199.

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22

Yokoi, M. "Epiretinal membrane formation in Terson syndrome." Japanese Journal of Ophthalmology 41, no. 3 (June 1997): 168–73. http://dx.doi.org/10.1016/s0021-5155(97)00025-7.

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23

Castrén, J. A. "PATHOGENESES AND TREATMENT OF TERSON-SYNDROME." Acta Ophthalmologica 41, no. 4 (May 27, 2009): 430–34. http://dx.doi.org/10.1111/j.1755-3768.1963.tb03552.x.

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24

Wong, Agnes M. F., Wai-Ching Lam, and James A. Sharpe. "Terson??s Syndrome in Subarachnoid Hemorrhage." Journal of Neuro-Ophthalmology 18, no. 2 (June 1998): 148???149. http://dx.doi.org/10.1097/00041327-199806000-00014.

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25

Nazarali, Samir, Irfan Kherani, Bernard Hurley, Geoff Williams, Michael Fielden, Feisal Adatia, and Amin Kherani. "OUTCOMES OF VITRECTOMY IN TERSON SYNDROME." Retina 40, no. 7 (July 2020): 1325–30. http://dx.doi.org/10.1097/iae.0000000000002570.

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26

McArdle, David J. T., and Sumit J. Karia. "Computed Tomography Diagnosis of Terson Syndrome." Journal of Emergency Medicine 53, no. 3 (September 2017): e45-e46. http://dx.doi.org/10.1016/j.jemermed.2017.04.022.

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27

Lüke, Julia, Olcay Tatar, Matthias Lüeke, Karl-Ulrich Bartz-Schmidt, and Salvatore Grisanti. "Papillary capillary activation in Terson syndrome." International Ophthalmology 30, no. 4 (January 28, 2010): 439–41. http://dx.doi.org/10.1007/s10792-010-9343-6.

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28

Sharma, Tarun, Lingam Gopal, Jyotirmay Biswas, Mahesh P. Shanmugam, Pramod S. Bhende, Rajat Agrawal, Nitin S. Shetty, and Neeraj Sanduja. "Results of Vitrectomy in Terson Syndrome." Ophthalmic Surgery, Lasers and Imaging Retina 33, no. 3 (May 2002): 195–99. http://dx.doi.org/10.3928/1542-8877-20020501-05.

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29

Raevis, Joseph, and Valerie I. Elmalem. "Pseudotumor cerebri syndrome causing a terson like syndrome." American Journal of Ophthalmology Case Reports 20 (December 2020): 100993. http://dx.doi.org/10.1016/j.ajoc.2020.100993.

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30

Asahi, Masumi G., Stephanie J. Weiss, Krishi Peddada, and Deepika Malik. "A Case of Terson-Like Syndrome in a Patient with Viral Meningoencephalitis." Case Reports in Ophthalmological Medicine 2019 (April 24, 2019): 1–5. http://dx.doi.org/10.1155/2019/9650675.

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The proposed mechanism of Terson’s syndrome is increased intracranial pressure that leads to dilation of the retrobulbar optic nerve and compression of the central retinal vein. Terson’s syndrome has been associated with many conditions that increase intracranial pressure such as venous sinus thrombosis, Moyamoya disease, leukemia, direct head trauma, and intraocular hemorrhage related to shaken baby syndrome. We present a novel case of a patient with recent viral prodrome found to have papilledema and multilayered retinal hemorrhages consistent with Terson syndrome. Computed tomography and magnetic resonance venography of the brain did not reveal any subdural, subarachnoid, or intracranial hemorrhages. However, cerebrospinal fluid analyses were significant for increased opening pressure and elevated protein levels, which were suggestive of viral meningoencephalitis. We describe this case as a Terson-like syndrome because the etiology of intraocular hemorrhage is increased intracranial pressure. However, this case does not fit the traditional presentation of Terson’s syndrome as the intracranial pressure is secondary to meningeal inflammation instead of subdural, subarachnoid, or intracranial hemorrhage. We strongly feel that it is important for physicians to be aware of the link between viral meningoencephalitis and retinal conditions such as Terson-like syndrome because it can facilitate rapid diagnosis and treatment.
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31

Chowdhury, Sanjoy, Madhumita Srivastava, and Nilanjan Chowdhury. "Traumatic “TERSON SYNDROME PLUS”: Pneumocephalocele with optic atrophy." European Journal of Clinical and Experimental Medicine 18, no. 2 (2020): 116–20. http://dx.doi.org/10.15584/ejcem.2020.2.6.

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Introduction. Terson Syndrome is subarachnoid hemorrhage (SAH) with sub retinal hemorrhage flowing through channel. Reduced vision in such fresh case is due to hemorrhage itself, blocking macula/other photo receptors in the long run macular cellophane retinopathy which causes profound visual loss. SAH causes neurological problems which can become a risk factor for evacuating blood from vitreous. Hypertension is commonest cause to cause Terson Syndrome, but trauma is also devastating cause as it can lead to irreversible visual consequences like total loss of perception of light or blindness. Aim. Here we describe a case of Terson Syndrome plus disease features SAH in frontal lobe. Description of the case. When there is traumatic pneumocephalocele, it gives space to blood to imbibe towards bony optic canal and form hematoma around nerve sheath which causes compression around the same and leads to optic atrophy. Optic nerve can be injured by direct traumatic dissection during road traffic accidents (RTA), but even without that blood may accumulate around optic nerve and in turn leads to formation of hematoma and subsequently pressure induced optic atrophy. Moreover, blood can slowly travel to sub hyaloid space/sub retinal space (beneath internal limiting membrane or sub ILM) with probable gliosis covering typical boat shaped blood as seen in this case. This sub ILM hemorrhage or gliosis may have resolved through three injections of Triamcinolone in the orbital floor (OFTA) near apex, but optic atrophy snatches vision. This protocol was followed to treat traumatic compressive (peri optic hematoma) optic neuropathy and traumatic retinopathy associated with sub hyaloid hemorrhage. Conclusion. Diagnosis of Terson syndrome plus disease was established by addressing all features on computed tomography (CT) scan and magnetic resonance imaging (MRI). Plus, features include pneumocephalus, optic nerve sheath hematoma, optic atrophy and gliosis over sub-hyaloid hemorrhage, typical boat shaped. The part of hemorrhage still endured as seen on optical coherence topography, but vision was lost by virtue of optic atrophy. OCT shows clot in sub hyaloid space.
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32

Benbouzid, A., N. Benfdil, L. Gaboune, K. Moussaoui, T. Baha, and A. Moutaouakil. "695 Complication exceptionnelle du syndrome de Terson." Journal Français d'Ophtalmologie 32 (April 2009): 1S206. http://dx.doi.org/10.1016/s0181-5512(09)73820-3.

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33

Sánchez Ferreiro, A. V., and L. Muñoz Bellido. "Reply to “Terson syndrome and ocular ultrasound”." Neurología (English Edition) 30, no. 2 (March 2015): 133–34. http://dx.doi.org/10.1016/j.nrleng.2013.01.011.

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34

李, 林. "Clinical Observation of Treatment in Terson Syndrome." Hans Journal of Ophthalmology 05, no. 01 (2016): 5–9. http://dx.doi.org/10.12677/hjo.2016.51002.

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35

Schultz, Paul N., Warren M. Sobol, and Thomas A. Weingeist. "Long-term Visual Outcome in Terson Syndrome." Ophthalmology 98, no. 12 (December 1991): 1814–19. http://dx.doi.org/10.1016/s0161-6420(91)32045-1.

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36

George, Jithin S., and John S. Elston. "Mystery Case: Terson syndrome on CT head." Neurology 87, no. 13 (September 26, 2016): e133-e134. http://dx.doi.org/10.1212/wnl.0000000000003143.

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37

Kralt, Peter, Ayeshea Shenton, and Julian Lindsay Burton. "Terson syndrome on post-mortem computed tomography." Forensic Imaging 22 (September 2020): 200394. http://dx.doi.org/10.1016/j.fri.2020.200394.

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38

RUBOWITZ, ALEXANDER, and UDAY DESAI. "NONTRAUMATIC MACULAR HOLES ASSOCIATED WITH TERSON SYNDROME." Retina 26, no. 2 (February 2006): 229–32. http://dx.doi.org/10.1097/00006982-200602000-00022.

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39

DE LA MATA, G., M. SATUE, M. BAMBO, M. ARA, MC EGEA, S. FERNANDEZ-PEREZ, R. HERRERO, and E. GARCIA-MARTIN. "Unilateral Terson syndrome. Outcome after early vitrectomy." Acta Ophthalmologica 90 (August 6, 2012): 0. http://dx.doi.org/10.1111/j.1755-3768.2012.s080.x.

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40

Garweg, Justus G., and Fritz Koerner. "Outcome indicators for vitrectomy in Terson syndrome." Acta Ophthalmologica 87, no. 2 (March 2009): 222–26. http://dx.doi.org/10.1111/j.1755-3768.2008.01200.x.

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Milea, Dan, Anne-Laure Boch, Jacques Philippon, and Phuc LeHoang. "Terson syndrome caused by penetrating head injury." Annals of Ophthalmology 32, no. 1 (March 2000): 24–27. http://dx.doi.org/10.1007/s12009-000-0007-4.

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42

Michalewska, Zofia, Janusz Michalewski, and Jerzy Nawrocki. "Possible Methods of Blood Entrance in Terson Syndrome." Ophthalmic Surgery, Lasers, and Imaging 41, no. 6 (November 1, 2010): S42—S49. http://dx.doi.org/10.3928/15428877-20101031-15.

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43

Koman, Edyta, Beata Gajda, Agnieszka Kiszka, Agnieszka Cisek, Katarzyna Nowomiejska, and Robert Rejdak. "Effectiveness of vitrectomy in Terson syndrome — case series." Ophthalmology Journal 1, no. 2 (July 7, 2016): 73–77. http://dx.doi.org/10.5603/oj.2016.0013.

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44

Jamaleddine, Z., S. El Haddad, M. M. Cherkaoui, and A. J. El Quessar. "Syndrome de Terson : à propos de trois cas." Journal of Neuroradiology 39, no. 1 (March 2012): 42–43. http://dx.doi.org/10.1016/j.neurad.2012.01.128.

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45

Parsons, Shaun R., Mary Qiu, and Ian C. Han. "Terson Syndrome from Subarachnoid Hemorrhage in Aplastic Anemia." Ophthalmology 123, no. 5 (May 2016): 1035. http://dx.doi.org/10.1016/j.ophtha.2016.02.031.

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46

Gauntt, Chiaki D., Richard G. Sherry, and Chithra Kannan. "Terson Syndrome With Bilateral Optic Nerve Sheath Hemorrhage." Journal of Neuro-Ophthalmology 27, no. 3 (September 2007): 193–94. http://dx.doi.org/10.1097/wno.0b013e31814b22dc.

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47

Friedman, Scott M., and Curtis E. Margo. "Bilateral Subinternal Limiting Membrane Hemorrhage With Terson Syndrome." American Journal of Ophthalmology 124, no. 6 (December 1997): 850–51. http://dx.doi.org/10.1016/s0002-9394(14)71709-x.

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48

Lee, Sung Bok, Sang Hyuk Kim, and Jung Yeul Kim. "Full Thickness Macular Holes Associated with Terson Syndrome." Journal of the Korean Ophthalmological Society 49, no. 7 (2008): 1194. http://dx.doi.org/10.3341/jkos.2008.49.7.1194.

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49

Ritland, Jan Ståle, Per Syrdalen, Nils Eide, Harald O. Vatne, and Ralf Øvergaard. "Outcome of vitrectomy in patients with Terson syndrome." Acta Ophthalmologica Scandinavica 80, no. 2 (April 2002): 172–75. http://dx.doi.org/10.1034/j.1600-0420.2002.800210.x.

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50

Ashrafi, Akbar N., Rahul Chakrabarti, and John Laidlaw. "Terson syndrome: the need for fundoscopy in subarachnoid haemorrhage." Medical Journal of Australia 197, no. 3 (August 2012): 152. http://dx.doi.org/10.5694/mja12.10642.

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