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1

da Fontoura Costa, Luciano, and Olaf Sporns. "Correlating thalamocortical connectivity and activity." Applied Physics Letters 89, no. 1 (2006): 013903. http://dx.doi.org/10.1063/1.2219736.

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2

Tu, Yiheng, Zening Fu, Fang Zeng, et al. "Abnormal thalamocortical network dynamics in migraine." Neurology 92, no. 23 (2019): e2706-e2716. http://dx.doi.org/10.1212/wnl.0000000000007607.

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ObjectiveTo investigate the dynamic functional connectivity of thalamocortical networks in interictal migraine patients and whether clinical features are associated with abnormal connectivity.MethodsWe investigated dynamic functional network connectivity (dFNC) of the migraine brain in 89 interictal migraine patients and 70 healthy controls. We focused on the temporal properties of thalamocortical connectivity using sliding window cross-correlation, clustering state analysis, and graph-theory methods. Relationships between clinical symptoms and abnormal dFNC were evaluated using a multivariate
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3

Sheffield, Julia M., Anna S. Huang, Baxter P. Rogers, et al. "Thalamocortical Anatomical Connectivity in Schizophrenia and Psychotic Bipolar Disorder." Schizophrenia Bulletin 46, no. 5 (2020): 1062–71. http://dx.doi.org/10.1093/schbul/sbaa022.

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Abstract Background Anatomical connectivity between the thalamus and cortex, including the prefrontal cortex (PFC), is abnormal in schizophrenia. Overlapping phenotypes, including deficits in executive cognitive abilities dependent on PFC-thalamic circuitry, suggest dysrupted thalamocortical anatomical connectivity may extend to psychotic bipolar disorder. We tested this hypothesis and examined the impact of illness stage to inform when in the illness course thalamocortical dysconnectivity emerges. Methods Diffusion-weighted imaging data were collected on 70 healthy individuals and 124 people
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4

Brown, Elliot C., Darren L. Clark, Stefanie Hassel, Glenda MacQueen, and Rajamannar Ramasubbu. "Thalamocortical connectivity in major depressive disorder." Journal of Affective Disorders 217 (August 2017): 125–31. http://dx.doi.org/10.1016/j.jad.2017.04.004.

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5

Reislev, N. H., T. B. Dyrby, H. R. Siebner, H. Lundell, M. Ptito, and R. Kupers. "Thalamocortical Connectivity and Microstructural Changes in Congenital and Late Blindness." Neural Plasticity 2017 (2017): 1–11. http://dx.doi.org/10.1155/2017/9807512.

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There is ample evidence that the occipital cortex of congenitally blind individuals processes nonvisual information. It remains a debate whether the cross-modal activation of the occipital cortex is mediated through the modulation of preexisting corticocortical projections or the reorganisation of thalamocortical connectivity. Current knowledge on this topic largely stems from anatomical studies in animal models. The aim of this study was to test whether purported changes in thalamocortical connectivity in blindness can be revealed by tractography based on diffusion-weighted magnetic resonance
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6

Qin, Yun, Nan Zhang, Yan Chen, et al. "Rhythmic Network Modulation to Thalamocortical Couplings in Epilepsy." International Journal of Neural Systems 30, no. 11 (2020): 2050014. http://dx.doi.org/10.1142/s0129065720500148.

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Thalamus interacts with cortical areas, generating oscillations characterized by their rhythm and levels of synchrony. However, little is known of what function the rhythmic dynamic may serve in thalamocortical couplings. This work introduced a general approach to investigate the modulatory contribution of rhythmic scalp network to the thalamo-frontal couplings in juvenile myoclonic epilepsy (JME) and frontal lobe epilepsy (FLE). Here, time-varying rhythmic network was constructed using the adapted directed transfer function between EEG electrodes, and then was applied as a modulator in fMRI-b
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7

Theyel, Brian B., Charles C. Lee, and S. Murray Sherman. "Specific and nonspecific thalamocortical connectivity in the auditory and somatosensory thalamocortical slices." NeuroReport 21, no. 13 (2010): 861–64. http://dx.doi.org/10.1097/wnr.0b013e32833d7cec.

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8

Liao, Yanhui, Jinsong Tang, Jianbin Liu, et al. "Decreased Thalamocortical Connectivity in Chronic Ketamine Users." PLOS ONE 11, no. 12 (2016): e0167381. http://dx.doi.org/10.1371/journal.pone.0167381.

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9

Cheng Chen, Anil Maybhate, David Israel, Nitish V. Thakor, and Xiaofeng Jia. "Assessing Thalamocortical Functional Connectivity With Granger Causality." IEEE Transactions on Neural Systems and Rehabilitation Engineering 21, no. 5 (2013): 725–33. http://dx.doi.org/10.1109/tnsre.2013.2271246.

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10

Barkat, Tania Rinaldi, Daniel B. Polley, and Takao K. Hensch. "A critical period for auditory thalamocortical connectivity." Nature Neuroscience 14, no. 9 (2011): 1189–94. http://dx.doi.org/10.1038/nn.2882.

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11

Jiang, S., C. Luo, J. Gong, et al. "Aberrant Thalamocortical Connectivity in Juvenile Myoclonic Epilepsy." International Journal of Neural Systems 28, no. 01 (2017): 1750034. http://dx.doi.org/10.1142/s0129065717500344.

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The purpose of this study was to investigate the functional connectivity (FC) of thalamic subdivisions in patients with juvenile myoclonic epilepsy (JME). Resting state functional magnetic resonance imaging (fMRI) and diffusion tensor imaging (DTI) data were acquired from 22 JME and 25 healthy controls. We first divided the thalamus into eight subdivisions by performing independent component analysis on tracking fibers and clustering thalamus-related FC maps. We then analyzed abnormal FC in each subdivision in JME compared with healthy controls, and we investigated their associations with clin
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12

Liao, Yanhui, and Jinsong Tang. "Decreased thalamocortical connectivity in chronic ketamine users." Drug and Alcohol Dependence 156 (November 2015): e128. http://dx.doi.org/10.1016/j.drugalcdep.2015.07.353.

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13

Mizuno, Akiko, Michele E. Villalobos, Molly M. Davies, Branelle C. Dahl, and Ralph-Axel Müller. "Partially enhanced thalamocortical functional connectivity in autism." Brain Research 1104, no. 1 (2006): 160–74. http://dx.doi.org/10.1016/j.brainres.2006.05.064.

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14

Klingner, Carsten M., Kerstin Langbein, Maren Dietzek, et al. "Thalamocortical connectivity during resting state in schizophrenia." European Archives of Psychiatry and Clinical Neuroscience 264, no. 2 (2013): 111–19. http://dx.doi.org/10.1007/s00406-013-0417-0.

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15

Scannell, Jack W., Simon Grant, Bertram R. Payne, and Roland Baddeley. "On variability in the density of corticocortical and thalamocortical connections." Philosophical Transactions of the Royal Society of London. Series B: Biological Sciences 355, no. 1393 (2000): 21–35. http://dx.doi.org/10.1098/rstb.2000.0547.

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Variability is an important but neglected aspect of connectional neuroanatomy. The quantitative density of the ‘same’ corticocortical or thalamocortical connection may vary by over two orders of magnitude between different injections of the same tracer. At present, however, the frequency distribution of connection densities is unknown. Therefore, it is unclear what kind of sampling strategies or statistical methods are appropriate for quantitative studies of connectivity. Nor is it clear if the measured variability represents differences between subjects, or if it is simply a consequence of in
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16

Choi, Soonwook, Eunah Yu, Seongwon Lee, and Rodolfo R. Llinás. "Altered thalamocortical rhythmicity and connectivity in mice lacking CaV3.1 T-type Ca2+ channels in unconsciousness." Proceedings of the National Academy of Sciences 112, no. 25 (2015): 7839–44. http://dx.doi.org/10.1073/pnas.1420983112.

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In unconscious status (e.g., deep sleep and anesthetic unconsciousness) where cognitive functions are not generated there is still a significant level of brain activity present. Indeed, the electrophysiology of the unconscious brain is characterized by well-defined thalamocortical rhythmicity. Here we address the ionic basis for such thalamocortical rhythms during unconsciousness. In particular, we address the role of CaV3.1 T-type Ca2+ channels, which are richly expressed in thalamic neurons. Toward this aim, we examined the electrophysiological and behavioral phenotypes of mice lacking CaV3.
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17

Ligot, Noémie, Pierre Krystkowiak, Clémence Simonin, et al. "External Globus Pallidus Stimulation Modulates Brain Connectivity in Huntington's Disease." Journal of Cerebral Blood Flow & Metabolism 31, no. 1 (2010): 41–46. http://dx.doi.org/10.1038/jcbfm.2010.186.

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Positron emission tomography with O-15-labeled water was used to study at rest the neurophysiological effects of bilateral external globus pallidus (GPe) deep brain stimulation in patients with Huntington's disease (HD). Five patients were compared with a control group in the on and off states of the stimulator. External globus pallidus stimulation decreased neuronal activity and modulated cerebral connectivity within the basal ganglia-thalamocortical circuitry, the sensorimotor, and the default-mode networks. These data indicate that GPe stimulation modulates functional integration in HD pati
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18

Zhang, Jing, King-Wai Chu, Ella B. Teague, Randall E. Newmark, and Monte S. Buchsbaum. "fMRI assessment of thalamocortical connectivity during attentional performance." Magnetic Resonance Imaging 31, no. 7 (2013): 1112–18. http://dx.doi.org/10.1016/j.mri.2013.01.008.

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19

Vo, An, Wataru Sako, Martin Niethammer, et al. "Thalamocortical Connectivity Correlates with Phenotypic Variability in Dystonia." Cerebral Cortex 25, no. 9 (2014): 3086–94. http://dx.doi.org/10.1093/cercor/bhu104.

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20

Ball, Gareth, Libuse Pazderova, Andrew Chew, et al. "Thalamocortical Connectivity Predicts Cognition in Children Born Preterm." Cerebral Cortex 25, no. 11 (2015): 4310–18. http://dx.doi.org/10.1093/cercor/bhu331.

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21

Nair, Aarti, Ruth A. Carper, Angela E. Abbott, et al. "Regional specificity of aberrant thalamocortical connectivity in autism." Human Brain Mapping 36, no. 11 (2015): 4497–511. http://dx.doi.org/10.1002/hbm.22938.

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22

Kim, Jung Bin, Sang-il Suh, Woo-Keun Seo, Kyungmi Oh, Seong-Beom Koh, and Ji Hyun Kim. "Altered thalamocortical functional connectivity in idiopathic generalized epilepsy." Epilepsia 55, no. 4 (2014): 592–600. http://dx.doi.org/10.1111/epi.12580.

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23

He, Xiaosong, Gaelle E. Doucet, Michael Sperling, Ashwini Sharan, and Joseph I. Tracy. "Reduced thalamocortical functional connectivity in temporal lobe epilepsy." Epilepsia 56, no. 10 (2015): 1571–79. http://dx.doi.org/10.1111/epi.13085.

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24

Lopez-Bendito, Guillermina. "Wiring thalamocortical connectivity: From axon guidance to plasticity." IBRO Reports 6 (September 2019): S53. http://dx.doi.org/10.1016/j.ibror.2019.07.165.

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25

O’Reilly, Christian, Elisabetta Iavarone, Jane Yi, and Sean L. Hill. "Rodent somatosensory thalamocortical circuitry: Neurons, synapses, and connectivity." Neuroscience & Biobehavioral Reviews 126 (July 2021): 213–35. http://dx.doi.org/10.1016/j.neubiorev.2021.03.015.

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26

Guller, Yelena, Giulio Tononi, and Bradley R. Postle. "Conserved Functional Connectivity but Impaired Effective Connectivity of Thalamocortical Circuitry in Schizophrenia." Brain Connectivity 2, no. 6 (2012): 311–19. http://dx.doi.org/10.1089/brain.2012.0100.

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27

Boveroux, Pierre, Audrey Vanhaudenhuyse, Marie-Aurélie Bruno, et al. "Breakdown of within- and between-network Resting State Functional Magnetic Resonance Imaging Connectivity during Propofol-induced Loss of Consciousness." Anesthesiology 113, no. 5 (2010): 1038–53. http://dx.doi.org/10.1097/aln.0b013e3181f697f5.

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Background Mechanisms of anesthesia-induced loss of consciousness remain poorly understood. Resting-state functional magnetic resonance imaging allows investigating whole-brain connectivity changes during pharmacological modulation of the level of consciousness. Methods Low-frequency spontaneous blood oxygen level-dependent fluctuations were measured in 19 healthy volunteers during wakefulness, mild sedation, deep sedation with clinical unconsciousness, and subsequent recovery of consciousness. Results Propofol-induced decrease in consciousness linearly correlates with decreased corticocortica
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28

Cho, Kang Ik K., Minah Kim, Youngwoo Bryan Yoon, Junhee Lee, Tae Young Lee, and Jun Soo Kwon. "Disturbed thalamocortical connectivity in unaffected relatives of schizophrenia patients with a high genetic loading." Australian & New Zealand Journal of Psychiatry 53, no. 9 (2019): 889–95. http://dx.doi.org/10.1177/0004867418824020.

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Objectives: Alterations in thalamocortical anatomical connectivity, specifically the connection between the orbitofrontal cortex and thalamus, have been frequently reported in schizophrenia and are suggested to contribute to the pathophysiology of schizophrenia. The connectivity of the thalamocortical white matter in unaffected relatives of schizophrenia patients was compared to that of healthy controls. Methods: The unaffected relative group was defined as asymptomatic family members who had at least one first-degree relative with schizophrenia and one or more other affected first- to third-d
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29

Tang, Shiyu, Elizabeth M. Powell, Wenjun Zhu, Fu-Sun Lo, Reha S. Erzurumlu, and Su Xu. "Altered Forebrain Functional Connectivity and Neurotransmission in a Kinase-Inactive Met Mouse Model of Autism." Molecular Imaging 18 (January 1, 2019): 153601211882103. http://dx.doi.org/10.1177/1536012118821034.

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MET, the gene encoding the tyrosine kinase receptor for hepatocyte growth factor, is a susceptibility gene for autism spectrum disorder (ASD). Genetically altered mice with a kinase-inactive Met offer a potential model for understanding neural circuit organization changes in autism. Here, we focus on the somatosensory thalamocortical circuitry because distinct somatosensory sensitivity phenotypes accompany ASD, and this system plays a major role in sensorimotor and social behaviors in mice. We employed resting-state functional magnetic resonance imaging and in vivo high-resolution proton MR sp
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30

Tu, Pei-Chi, Ya Mei Bai, Cheng-Ta Li, et al. "Identification of Common Thalamocortical Dysconnectivity in Four Major Psychiatric Disorders." Schizophrenia Bulletin 45, no. 5 (2018): 1143–51. http://dx.doi.org/10.1093/schbul/sby166.

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Abstract Background Recent genetic and imaging analyses of large datasets suggested that common biological substrates exist across psychiatric diagnoses. Functional connectivity (FC) abnormalities of thalamocortical circuits were consistently found in patients with schizophrenia but have been less studied in other major psychiatric disorders. This study aimed to examine thalamocortical FC in 4 major psychiatric disorders to identify the common connectivity abnormalities across major psychiatric disorders. Methods This study recruited 100 patients with schizophrenia, 100 patients with bipolar I
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31

Llinás, Rodolfo R., and Mircea Steriade. "Bursting of Thalamic Neurons and States of Vigilance." Journal of Neurophysiology 95, no. 6 (2006): 3297–308. http://dx.doi.org/10.1152/jn.00166.2006.

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This article addresses the functional significance of the electrophysiological properties of thalamic neurons. We propose that thalamocortical activity, is the product of the intrinsic electrical properties of the thalamocortical (TC) neurons and the connectivity their axons weave. We begin with an overview of the electrophysiological properties of single neurons in different functional states, followed by a review of the phylogeny of the electrical properties of thalamic neurons, in several vertebrate species. The similarity in electrophysiological properties unambiguously indicates that the
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32

Doyle, Daniel Z., Mandy M. Lam, Adel Qalieh, et al. "Chromatin remodeler Arid1a regulates subplate neuron identity and wiring of cortical connectivity." Proceedings of the National Academy of Sciences 118, no. 21 (2021): e2100686118. http://dx.doi.org/10.1073/pnas.2100686118.

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Loss-of-function mutations in chromatin remodeler gene ARID1A are a cause of Coffin-Siris syndrome, a developmental disorder characterized by dysgenesis of corpus callosum. Here, we characterize Arid1a function during cortical development and find unexpectedly selective roles for Arid1a in subplate neurons (SPNs). SPNs, strategically positioned at the interface of cortical gray and white matter, orchestrate multiple developmental processes indispensable for neural circuit wiring. We find that pancortical deletion of Arid1a leads to extensive mistargeting of intracortical axons and agenesis of
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33

Barre, Alexander, Coralie Berthoux, Dimitri De Bundel, et al. "Presynaptic serotonin 2A receptors modulate thalamocortical plasticity and associative learning." Proceedings of the National Academy of Sciences 113, no. 10 (2016): E1382—E1391. http://dx.doi.org/10.1073/pnas.1525586113.

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Higher-level cognitive processes strongly depend on a complex interplay between mediodorsal thalamus nuclei and the prefrontal cortex (PFC). Alteration of thalamofrontal connectivity has been involved in cognitive deficits of schizophrenia. Prefrontal serotonin (5-HT)2A receptors play an essential role in cortical network activity, but the mechanism underlying their modulation of glutamatergic transmission and plasticity at thalamocortical synapses remains largely unexplored. Here, we show that 5-HT2A receptor activation enhances NMDA transmission and gates the induction of temporal-dependent
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34

Millichap, J. Gordon. "Thalamocortical Structural and Functional Connectivity in Juvenile Myoclonic Epilepsy." Pediatric Neurology Briefs 27, no. 1 (2013): 3. http://dx.doi.org/10.15844/pedneurbriefs-27-1-3.

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35

Krishnan, Giri P., Burke Q. Rosen, Jen-Yung Chen, et al. "Thalamocortical and intracortical laminar connectivity determines sleep spindle properties." PLOS Computational Biology 14, no. 6 (2018): e1006171. http://dx.doi.org/10.1371/journal.pcbi.1006171.

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36

Killgore, William D. S., John R. Vanuk, Sara A. Knight, et al. "Daytime sleepiness is associated with altered resting thalamocortical connectivity." NeuroReport 26, no. 13 (2015): 779–84. http://dx.doi.org/10.1097/wnr.0000000000000418.

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37

Crocker-Buque, Alex, Sarah M. Brown, Peter C. Kind, John T. R. Isaac, and Michael I. Daw. "Experience-Dependent, Layer-Specific Development of Divergent Thalamocortical Connectivity." Cerebral Cortex 25, no. 8 (2014): 2255–66. http://dx.doi.org/10.1093/cercor/bhu031.

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38

Tu, Ye, Tian Yu, Xiao-Yun Fu, et al. "Altered Thalamocortical Functional Connectivity by Propofol Anesthesia in Rats." Pharmacology 88, no. 5-6 (2011): 322–26. http://dx.doi.org/10.1159/000334168.

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39

Wei, Qiang, Tongjian Bai, Elliot C. Brown, et al. "Thalamocortical connectivity in electroconvulsive therapy for major depressive disorder." Journal of Affective Disorders 264 (March 2020): 163–71. http://dx.doi.org/10.1016/j.jad.2019.11.120.

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40

Sheffield, Julia, Anna Huang, Baxter Rogers, et al. "Thalamocortical Anatomical Connectivity in Schizophrenia and Psychotic Bipolar Disorder." Biological Psychiatry 87, no. 9 (2020): S447—S448. http://dx.doi.org/10.1016/j.biopsych.2020.02.1139.

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41

Gezelius, Henrik, and Guillermina López‐Bendito. "ISDN2012_0261: Wiring thalamocortical connectivity: From axon guidance to plasticity." International Journal of Developmental Neuroscience 30, no. 8 (2012): 633. http://dx.doi.org/10.1016/j.ijdevneu.2012.10.084.

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42

Ramsay, Ian S., and Angus W. MacDonald. "The Ups and Downs of Thalamocortical Connectivity in Schizophrenia." Biological Psychiatry 83, no. 6 (2018): 473–74. http://dx.doi.org/10.1016/j.biopsych.2018.01.005.

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43

Llano, D. A., B. B. Theyel, A. K. Mallik, S. M. Sherman, and N. P. Issa. "Rapid and Sensitive Mapping of Long-Range Connections In Vitro Using Flavoprotein Autofluorescence Imaging Combined With Laser Photostimulation." Journal of Neurophysiology 101, no. 6 (2009): 3325–40. http://dx.doi.org/10.1152/jn.91291.2008.

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We investigated the use of flavoprotein autofluorescence (FA) as a tool to map long-range neural connections and combined FA with laser-uncaging of glutamate to facilitate rapid long-range mapping in vitro. Using the somatosensory thalamocortical slice, we determined that the spatial resolution of FA is ≥100–200 μm and that the sensitivity for detecting thalamocortical synaptic activity approximates that of whole cell recording. Blockade of ionotropic glutamate receptors with DNQX and AP5 abolished cortical responses to electrical thalamic stimulation. The combination of FA with photostimulati
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44

Nair, Aarti, Jeffrey M. Treiber, Dinesh K. Shukla, Patricia Shih, and Ralph-Axel Müller. "Impaired thalamocortical connectivity in autism spectrum disorder: a study of functional and anatomical connectivity." Brain 136, no. 6 (2013): 1942–55. http://dx.doi.org/10.1093/brain/awt079.

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45

Manoach, Dara S., and Robert Stickgold. "Abnormal Sleep Spindles, Memory Consolidation, and Schizophrenia." Annual Review of Clinical Psychology 15, no. 1 (2019): 451–79. http://dx.doi.org/10.1146/annurev-clinpsy-050718-095754.

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There is overwhelming evidence that sleep is crucial for memory consolidation. Patients with schizophrenia and their unaffected relatives have a specific deficit in sleep spindles, a defining oscillation of non-rapid eye movement (NREM) Stage 2 sleep that, in coordination with other NREM oscillations, mediate memory consolidation. In schizophrenia, the spindle deficit correlates with impaired sleep-dependent memory consolidation, positive symptoms, and abnormal thalamocortical connectivity. These relations point to dysfunction of the thalamic reticular nucleus (TRN), which generates spindles,
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46

Andrade, Pablo, Petra Heiden, Moritz Hoevels, et al. "Modulation of Fibers to Motor Cortex during Thalamic DBS in Tourette Patients Correlates with Tic Reduction." Brain Sciences 10, no. 5 (2020): 302. http://dx.doi.org/10.3390/brainsci10050302.

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Probabilistic tractography in Tourette syndrome (TS) patients have shown an alteration in the connectivity of the primary motor cortex and supplementary motor area with the striatum and thalamus, suggesting an abnormal connectivity of the cortico-striatum-thalamocortical-pathways in TS. Deep brain stimulation (DBS) of the centromedian nucleus–nucleus ventrooralis internus (CM-Voi complex) in the thalamus is an effective treatment for refractory TS patients. We investigated the connectivity of activated fibers from CM-Voi to the motor cortex and its correlation between these projections and the
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47

Sumner, Rachael L., Meg J. Spriggs, and Alexander D. Shaw. "Modelling thalamocortical circuitry shows that visually induced LTP changes laminar connectivity in human visual cortex." PLOS Computational Biology 17, no. 1 (2021): e1008414. http://dx.doi.org/10.1371/journal.pcbi.1008414.

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Neuroplasticity is essential to learning and memory in the brain; it has therefore also been implicated in numerous neurological and psychiatric disorders, making measuring the state of neuroplasticity of foremost importance to clinical neuroscience. Long-term potentiation (LTP) is a key mechanism of neuroplasticity and has been studied extensively, and invasively in non-human animals. Translation to human application largely relies on the validation of non-invasive measures of LTP. The current study presents a generative thalamocortical computational model of visual cortex for investigating a
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48

Ranft, Andreas, Daniel Golkowski, Tobias Kiel, et al. "Neural Correlates of Sevoflurane-induced Unconsciousness Identified by Simultaneous Functional Magnetic Resonance Imaging and Electroencephalography." Anesthesiology 125, no. 5 (2016): 861–72. http://dx.doi.org/10.1097/aln.0000000000001322.

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Abstract Background The neural correlates of anesthetic-induced unconsciousness have yet to be fully elucidated. Sedative and anesthetic states induced by propofol have been studied extensively, consistently revealing a decrease of frontoparietal and thalamocortical connectivity. There is, however, less understanding of the effects of halogenated ethers on functional brain networks. Methods The authors recorded simultaneous resting-state functional magnetic resonance imaging and electroencephalography in 16 artificially ventilated volunteers during sevoflurane anesthesia at burst suppression a
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49

Iwabuchi, S., and L. Palaniyappan. "Abnormalities in the effective connectivity of thalamocortical circuitry in schizophrenia." European Neuropsychopharmacology 26 (October 2016): S308—S309. http://dx.doi.org/10.1016/s0924-977x(16)31214-7.

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50

White, Nathan S., and Michael T. Alkire. "Impaired thalamocortical connectivity in humans during general-anesthetic-induced unconsciousness." NeuroImage 19, no. 2 (2003): 402–11. http://dx.doi.org/10.1016/s1053-8119(03)00103-4.

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