Academic literature on the topic 'THE BACTERIA STAPHYLOCOCCUS AUREUS'

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Journal articles on the topic "THE BACTERIA STAPHYLOCOCCUS AUREUS"

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Charoenca, Naowarut, and Roger S. Fujioka. "Assessment of Staphylococcus Bacteria in Hawaii's Marine Recreational Waters." Water Science and Technology 27, no. 3-4 (February 1, 1993): 283–89. http://dx.doi.org/10.2166/wst.1993.0361.

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Several media to specifically recover staphylococcus from marine recreational waters in Hawaii were evaluated. The membrane filtration method using Vogel-Johnson and Tellurite Glycine Agar media supplemented with 0.005% sodium azide were shown to quantitatively recover total staphylococcus and S. aureus from marine recreational waters. Beaches in Hawaii were determined to contain high or low concentrations of staphylococci based on Favero's proposed standard of 100 staphylococci/100 ml as the maximum allowable concentration. Statistical analysis of the data showed that levels of total staphylococci were significantly correlated with that of S. aureus and with swimmer density. Staphylococci were recovered from beach waters throughout the day, including evening hours, indicating the stability of these bacteria in marine waters. Moreover, the antibiotic sensitivity patterns and phage types of S. aureus isolates from marine waters and from clinical human skin cultures were similar. These results support the hypothesis that recreational waters can serve as vehicles for the transmission of staphylococcal skin infections.
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HONG, JISOO, JONGUK KIM, LIN-HU QUAN, SUNGGI HEU, and EUNJUNG ROH. "Purification and Characterization of Pasteuricin Produced by Staphylococcus pasteuri RSP-1 and Active against Multidrug-Resistant Staphylococcus aureus." Journal of Food Protection 81, no. 11 (October 3, 2018): 1768–75. http://dx.doi.org/10.4315/0362-028x.jfp-18-111.

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ABSTRACT Staphylococcus aureus is an important pathogen implicated in various diseases, including staphylococcal food poisoning. Bacteriocins are considered safe and effective antimicrobial substances for the prevention of the growth of pathogenic bacteria. In this article, we describe the purification and characterization of pasteuricin, a novel bacteriocin produced by Staphylococcus pasteuri RSP-1. A cell-free supernatant of S. pasteuri RSP-1 exerted strong antimicrobial activity against staphylococci, including methicillin-resistant S. aureus (MRSA), and gram-positive bacteria. The loss of antimicrobial activity upon treatment with proteolytic enzymes confirmed the proteinaceous nature of pasteuricin. A rapid and pronounced bactericidal effect of pasteuricin was confirmed by a live-dead bacterial viability assay. To our knowledge, pasteuricin is the first reported S. pasteuri bacteriocin that inhibits S. aureus. Because pasteuricin is characterized by strong antimicrobial activity and high stability, it has potential as an alternative antimicrobial agent to antibiotics.
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Capparelli, Rosanna, Marianna Parlato, Giorgia Borriello, Paola Salvatore, and Domenico Iannelli. "Experimental Phage Therapy against Staphylococcus aureus in Mice." Antimicrobial Agents and Chemotherapy 51, no. 8 (May 21, 2007): 2765–73. http://dx.doi.org/10.1128/aac.01513-06.

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ABSTRACT The present study describes a bacteriophage (MSa) active against Staphylococcus aureus, including methicillin-resistant staphylococcal strains. When inoculated into mice simultaneously with S. aureus A170 (108 CFU/mouse), phage (109 PFU) rescued 97% of the mice; when applied to nonlethal (5 × 106 CFU/mouse) 10-day infections, the phage also fully cleared the bacteria. The phage MSa, delivered inside macrophages by S. aureus, kills the intracellular staphylococci in vivo and in vitro. The phage can also prevent abscess formation and reduce the bacterial load and weight of abscesses. These results suggest a potential use of the phage for the control of both local and systemic human S. aureus infections.
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Lutpiatina, Leka, and Nisa Nur Agistni Eriana. "Staphylococcus aureus and Methicillin resistant Staphylococcus aureus From The Diabetic Ulcer." Medical Laboratory Technology Journal 4, no. 1 (June 30, 2018): 30. http://dx.doi.org/10.31964/mltj.v4i1.182.

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Infections of diabetic ulcers may occur because patients with diabetes mellitus have a weak immune system and the presence of high blood sugar into a strategic place for bacterial growth one of them is Staphylococcus aureus. Increased resistance of Staphylococcus aureus to various antibiotics, causing problems for the treatment of this infection. As by Methicillin-resistant Staphylococcus aureus (MRSA) This study aims to assess the presence of Staphylococcus aureus and Methicillin-resistant Staphylococcus aureus (MRSA) at diabetic ulcer patient at Banjarmasin city hospital. This research uses descriptive observational research type. The study sample was patients with grade 2 and three diabetic ulcers with a total of 30 samples. The result of the research found 14 Staphylococcus aureus bacteria, Staphylococcus epidermidis nine, Staphylococcus saprophyticus two and 5 Gram-negative bacteria. Of the 14 samples of Staphylococcus aureus, there were eight samples of cefoxitin-resistant samples. The conclusion was 47% of Staphylococcus aureus in patients with a diabetic ulcer at Banjarmasin City Hospital. There was a 57% Methicillin-resistant Staphylococcus aureus in the isolated Staphylococcus aureus found. Need further research on the resistance of antibiotics oxacillin and cefoxitin to other Staphylococcus sp bacteria derived from patient diabetic ulcers.
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Goncagul, Gulsen. "Antimicrobialsusceptibilityof bacteria isolated from goats with subclinical mastitis in the Southern Marmara region of Turkey." Medycyna Weterynaryjna 77, no. 05 (2021): 6527–2021. http://dx.doi.org/10.21521/mw.6527.

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The mastitis of dairy goats is a disease of the economic importance worldwide and is mostly associated with bacterial infections. The aim of this study was to isolate the bacteriae causing subclinical mastitis, and determine to the susceptibility of some clinical isolates against several antimicrobial agents frequently used to control bacterial subclinical mastitis in dairy goats in the Southern Marmara Region. A total of 68 Saanen goats were used for this investigation and subclinical mastitis was determined by using California Mastitis Test. As a result of bacteriological analysis of milk samples, 30 different bacteria species have been identified and non-aureus staphylococci found to be the predominant bacteria species with the rate of 22.1%. The species with the highest isolation rate among the isolates were Escherichia coli (18.9%), Staphylococcus aureus (15.7%), Streptococcus agalactiae (14.2%) and Staphylococcus epidermidis (7.9%). The antimicrobial susceptibility of the high isolation rate species including Escherichia coli, Staphylococcus aureus, Streptococcus agalactiae, Staphylococcus epidermidis, Streptococcus uberis, Enterococcus faecalis, Staphylococcus haemolyticus and Mannheimia haemolytica to twelve antibiotics were determined by disc diffusion method. Bacterial strains analyzed showed highest sensitivity to ofloxacin (87.9%) and followed by cefuroxime (85.8%) and cefazolin (83.6%). In conclusion, subclinical mastitis still remains a problem in dairy goats, and for the elimination of subclinical mastitis, besides protective measures, determination of the bacteriae causing mastitis and their antibiotic sensitivities should be priority.
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Pasachova Garzón, Jennifer, Sara Ramirez Martinez, and L. Muñoz Molina. "Staphylococcus aureus." Nova 17, no. 32 (November 15, 2019): 25–38. http://dx.doi.org/10.22490/24629448.3631.

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Staphylococcus aureus se caracteriza por ser la principal causa de bacteriemia nosocomial en el mundo, debido al incremento en la resistencia, a los diferentes factores de patogenicidad y virulencia y la expresión de una gran variedad de roteínas las cuales pertenecen a las moléculas de la matriz adhesiva (MSCRAMM), presentes en la superficie de la bacteria cuya función es la colonización e invasión celular al hospedero y favorecer la formación de biopelícula, El conjunto de estos mecanismos de patogenicidad y virulencia, le permiten a la bacteria persistir en el huésped y en el ambiente, sobreviviendo a factores adversos, al sistema inmune y a los antimicrobianos.
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Winias, Saka. "EFFECT OF CYNAMMYLDEHYDE FROM CINNAMON EXTRACT AS A NATURAL PRESERVATIVE ALTERNATIVE TO THE GROWTH OF Staphylococcus aureus BACTERIA." Indonesian Journal of Tropical and Infectious Disease 2, no. 1 (July 27, 2015): 38. http://dx.doi.org/10.20473/ijtid.v2i1.188.

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Food is one of the best media for the microorganism to live and grow. Therefore, food is often broken because it has been contaminated by the microorganism. In industry country, approximately 30% of population infected by food borne disease. Food borne disease is caused of phatogen bacteria food borne. Staphylococus aureus is a kind of bacteria that can make food rotten and also it is a phatogen bacteria cause food born disease, no forming spora, positive gram bacteria and the food substance which is contaminated by Staphylococus aureus will cause poisoned becaused of enterotoxin which is heat resisting. Essential oil is antimicrobial and anti bacterial that the most effective, it can inhibit the growing of microba and bacteria. One of the example of essential oil is Cinnamon.sp oil. Cinnamon oil is antimcroba agent for bacteri and fungi because it contain cynammyldehyde and cynammyl alcohol and also eugenol. The aim of this study is to understand the antimcrobacterial potential of cynammyldehyde from cinnamon extract to Staphylococus aureus. This study is laboratory experimantal research. Essential oil from Cinnamon by destilation, then redistilation was done to get cynammyldehyde from cinnamon. Cynammyldehyde was tested to Staphylococus aureus. Test method was done as dilution in the form. From this result, it show that cynammyldehide from cinnamon extract has ability in inhibit the Staphylococus aureus growth. We can conclude that Cynammaldehyde from cinnamon extract has antibacterial effect especially for positive gram bacteria that is Staphylococcus aureus. The optimum inhibiting effort is 0.09%.
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DeLeo, Frank R., and Michael Otto. "An antidote for Staphylococcus aureus pneumonia?" Journal of Experimental Medicine 205, no. 2 (February 11, 2008): 271–74. http://dx.doi.org/10.1084/jem.20080167.

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Methicillin-resistant Staphylococcus aureus (MRSA) is the leading cause of bacterial infections in the United States. Severe invasive MRSA infections, which include pneumonia, are difficult to treat because the bacteria are resistant to antibiotics. A new report now shows that immunization against α-hemolysin (Hla), a cytolytic toxin secreted by most S. aureus strains, protects mice against lethal pneumonia. This finding represents the first successful vaccine strategy for the treatment of staphylococcal pneumonia.
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Edslev, Sofie Marie, Caroline Meyer Olesen, Line Brok Nørreslet, Anna Cäcilia Ingham, Søren Iversen, Berit Lilje, Maja-Lisa Clausen, et al. "Staphylococcal Communities on Skin Are Associated with Atopic Dermatitis and Disease Severity." Microorganisms 9, no. 2 (February 19, 2021): 432. http://dx.doi.org/10.3390/microorganisms9020432.

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The skin microbiota of atopic dermatitis (AD) patients is characterized by increased Staphylococcus aureus colonization, which exacerbates disease symptoms and has been linked to reduced bacterial diversity. Skin bacterial communities in AD patients have mostly been described at family and genus levels, while species-level characterization has been limited. In this study, we investigated the role of the bacteria belonging to the Staphylococcus genus using targeted sequencing of the tuf gene with genus-specific primers. We compared staphylococcal communities on lesional and non-lesional skin of AD patients, as well as AD patients with healthy controls, and determined the absolute abundance of bacteria present at each site. We observed that the staphylococcal community, bacterial alpha diversity, and bacterial densities were similar on lesional and non-lesional skin, whereas AD severity was associated with significant changes in staphylococcal composition. Increased S. aureus, Staphylococcus capitis, and Staphylococcus lugdunensis abundances were correlated with increased severity. Conversely, Staphylococcus hominis abundance was negatively correlated with severity. Furthermore, S. hominis relative abundance was reduced on AD skin compared to healthy skin. In conclusion, various staphylococcal species appear to be important for skin health.
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Berti, Andrew D., Lauren T. Harven, and Victoria Bingley. "Distinct Effectiveness of Oritavancin against Tolerance-Induced Staphylococcus aureus." Antibiotics 9, no. 11 (November 8, 2020): 789. http://dx.doi.org/10.3390/antibiotics9110789.

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Within a sufficiently large bacterial population, some members will naturally adopt an alternate, metabolically-active state that favors small molecule synthesis over cell division. These isogenic “tolerant” subpopulations have variable responses during antibiotic exposure and can remain viable in the presence of typically bactericidal concentrations. In this study, we determine the ability of typical and atypical antistaphylococcal therapies to reduce the viability of mupirocin-induced tolerant Staphylococcus aureus bacteria. Overall, tolerance-induced staphylococci exhibited a markedly decreased rate and extent of killing following antibiotic exposure. However, oritavancin remained effective at maintaining a similar extent of killing. Further studies to investigate the role of oritavancin against recurrent or relapse staphylococcal infection are warranted.
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Dissertations / Theses on the topic "THE BACTERIA STAPHYLOCOCCUS AUREUS"

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Jones, Eleanor. "Osmotic adaptations of Staphylococcus aureus." Thesis, University of Nottingham, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.310928.

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Fox, Paige McCarthy. "Characterization of Vancomycin Resistance in Staphylococcus Aureus." VCU Scholars Compass, 2006. http://scholarscompass.vcu.edu/etd/1243.

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In the past decade, Staphylococcus aureus has developed two distinct vancomycin resistance mechanisms. First, the bacterium is capable of generating a thickened, poorly crosslinked cell wall that creates false targets. These targets cause vancomycin to bind at the periphery of the thickened peptidoglycan, allowing normal cell wall formation to continue at the cell membrane. Second, S. aureus has acquired genes from Enterococcus that encode an alternative stem peptide. The genes, known as van genes, alter the target of vancomycin, rendering vancomycin treatment ineffectual.In this work, we attempted to further characterize both mechanisms of vancomycin resistance. First, a potential link between up-regulated purine biosynthesis and increased vancomycin resistance due to a thickened cell wall was examined. Despite exploration of multiple mechanisms to increase purine levels within the cell, increased purine synthesis did not provide S. aureus with any advantage in the presence of vancomycin. However, during the investigation, purine biosynthesis in S. aureus was further characterized by confirming purr as the repressor of the purine pathway and demonstrating its sensitivity to mutation.Next, the relationship between homotypic oxacillin resistance and increased vancomycin resistance in the absence of the van genes was investigated. Vancomycin passage of two heterotypic methicillin resistant S. aureus (MRSA) caused these strains to convert to homotypic oxacillin resistance in the absence of oxacillin exposure. Additionally, conversion of heterotypic oxacillin resistant strains to homotypy by oxacillin passage increased strain survival in vancomycin. The SOS response was examined as the possible link between conversion to homotypic oxacillin resistance and increasing vancomycin resistance due to a thickened cell wall. The current study, however, detected no induction of the SOS response during vancomycin exposure.Lastly, the relationship between oxacillin resistance and vancomycin resistance due to the acquisition of the van genes was examined. In vitro and in vivo methods were utilized to determine the effectiveness of a combination of β-lactam antibiotics and vancomycin to treat vancomycin resistant S. aureus (VRSA) infections. Combination therapy provided a significant advantage over untreated control or either antibiotic alone in the rabbit model of endocarditis.
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Vance, Lindsey. "The Inhibitory Effects of a Novel Gel on Staphylococcus aureus Biofilms." Digital Commons @ East Tennessee State University, 2018. https://dc.etsu.edu/honors/435.

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Antibiotic resistance is an ever-growing topic of concern within the medical field causing researchers to examine the mechanisms of resistance to develop new antimicrobials. Bacteria’s ability to form biofilms is one mechanism which aids in antimicrobial resistance. Staphylococcus aureus is of special interest as it is one of the most frequent biofilm-forming bacteria found on medical devices causing infections and posing dangerous threats in a clinical setting. A recently developed antimicrobial gel has been shown to have profound effects on treating bacterial infections and wound healing. This research is centered upon examining the antimicrobial effects of this gel on the three different stages of biofilm formation in clinical and laboratory strains of S. aureus. Through a series of experiments examining the effects this gel has on S. aureus at the stages of biofilm attachment, maturation, and dispersion, the gel has shown significant levels of inhibition. These findings indicate that the novel gel disrupts biofilm forming processes of S. aureus, which provides useful information for fighting infections in the medical field. Further research on the uses and effects of this new gel could lead possibility using the antimicrobial compound for a variety of clinical purposes.
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Johnson, Adam L. "Characterization of a Novel Protease in Staphylococcus aureus." VCU Scholars Compass, 2015. http://scholarscompass.vcu.edu/etd/3943.

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A newly discovered cysteine protease, Prp, has been shown to perform an essential, site-specific cleavage of ribosomal protein L27 in Staphylococcus aureus. In Firmicutes and related bacteria, ribosomal protein L27 is encoded with a conserved N-terminal extension that must be removed to expose residues critical for ribosome function. Uncleavable and pre-cleaved variants were unable to complement an L27 deletion in S. aureus, indicating that this N-terminal processing event is essential and likely plays an important regulatory role. The gene encoding the responsible protease (prp) has been shown to be essential, and is found in all organisms encoding the N-terminal extension of L27. Cleavage of L27 by Prp represents a new target for potential antibiotic therapy. In order to characterize this protease, Prp has been overexpressed and purified. Using an assay we have developed, based on cleavage of a fluorogenic peptide derived from the conserved L27 cleavage sequence, we have undertaken an analysis of the enzyme kinetics and substrate specificity for Prp cleavage and tested predictions made based on a structural model using active-site mutants.
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Evans, Jane E. "The conjugation system of Staphylococcus aureus." Thesis, University of Oxford, 1986. https://ora.ox.ac.uk/objects/uuid:1c1f5c11-f854-4af5-b9cf-34fdf279fb28.

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A conjugation system in Staphylococcus aureus has been investigated and shown to be determined, at least in part, by genes carried on plasmids. Conjugation required cell-to-cell contact but not calcium ions. The frequency of conjugation depended on the recipient used and on the incubation conditions. Two conjugative plasmids were mapped by restriction enzyme analysis but experiments to clone the conjugation-determining region were unsuccessful although separate regions specifying gentamicin resistance, ethidium bromide resistance and cadmium resistance were cloned. The gentamicin resistance determinant was probably part of Tn4001. Deletion of various sized pieces of DNA from one of the plasmids resulted in reduction of its ability to specify conjugation but no specific part of this plasmid could be implicated in the process. Further experiments led to the conclusion that this particular plasmid (p8325-4) is probably not self-transmissible but transferred by a phage-mediated system. Strains of Staphylococcus aureus produced a pheromone-like substance that elicited a clumping response in Streptococcus faecalis but no evidence was found for the involvement of staphylococcal conjugative plasmids in this. The conjugative plasmid, p8325-2, mobilized a small plasmid (pT181) but not a chromosomal gene. Insertion of transposon Tn551 was used to produce mutants of the conjugative plasmid p8325-2. Some twenty-six mutants were studied and the position of Tn551 in them mapped. There were preferred regions of insertion for Tn551 and twenty out of the twenty-six mutants had altered ability to conjugate. One showed a significantly higher frequency of conjugation and the other nineteen, all with substantially lower frequencies of conjugation, were mapped to two well-separated regions of the plasmid. Similarity between the locations of these putative regions and those reported for some other conjugative plasmids from staphylococci is striking and suggests a common origin.
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Piemont, Yves. "Les Exfoliatines de Staphylococcus aureus." Grenoble 2 : ANRT, 1987. http://catalogue.bnf.fr/ark:/12148/cb37608872c.

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Griffin, Blakeley. "A Study of the Polymicrobial Inhibitory Interactions Between Alcaligenes faecalis and Staphylococcus aureus." Digital Commons @ East Tennessee State University, 2020. https://dc.etsu.edu/honors/579.

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Members of the Staphylococcus genus are found as a part of normal microflora in humans and can commonly be found on the skin or in the nasal cavity. However, these microorganisms can cause serious and life-threatening opportunistic infections when there is a break in the physical barrier of skin. These infections have become difficult to treat as resistant strains emerge, particularly Methicillin Resistant Staphylococcus aureus (MRSA). MRSA has become a commonly acquired nosocomial infection which is difficult to treat with conventional antibiotics of the blactam class. Even Vancomycin, a last resort antibiotic, has been ineffective on some infections. Furthermore, S. aureus readily forms biofilms on implanted medical devices which establishes a hardy and difficult to treat infection. These biofilms serve as a point of infection to the bloodstream. Research involving polymicrobial interactions and the inhibitory effects of bacterial-bacterial interactions could be a starting point for the discovery of a new therapeutic treatment for infections. It has been shown in our lab that Alcaligenes faecalishas inhibitory effects on Staphylococcus aureusplanktonic growth. Therefore, in this study, we wanted to examine 1) The mechanism by which A. faecalisinhibitsS. aureus growth and 2) how A. faecalisimpacts the various phases of S. aureusbiofilm growth. It was found that A. faecalislikely inhibits S. aureususing a physical mechanism that requires close contact, rather than using a secreted molecule. However, a Type VI secretion system could also produce similar results. Further research involving the formation of mutants to find the gene allowing A. faecalisto inhibit S. aureuswas started, but no viable mutants were created during the course of this research. A. faecaliswas found to inhibit the formation of S. aureus biofilm growth, but when added to a mature S. aureusbiofilm, the slow growth rate of A. faecaliscould not overtake the quickly replicating S. aureus. Further research in the polymicrobial interactions between S. aureus and A. faecaliscould lead to a finding of a new therapeutic target for antibiotics or the use of A. faecalisin infections.
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Moore, Catrin Elisabeth. "Case-control study of invasive Staphylococcus aureus disease-host genetic susceptibility and bacterial population structure." Thesis, University of Oxford, 2002. https://ora.ox.ac.uk/objects/uuid:85a3a6b4-b0cc-48ed-b584-5037ea884250.

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Staphylococcus aureus is a major pathogen associated with serious community-acquired and nosocomial disease. It is carried nasally at some time by 70% of the population, yet severe disease is relatively uncommon. Two case-control studies were conducted in Oxford, UK and Thailand to examine bacterial and host genetic determinants of severe S. aureus disease. Over 800 cases and 1,600 healthy control individuals were recruited into the two studies. The genetic population structures of S. aureus disease and carriage isolate populations from the Oxford study were studied and compared using phage typing, pulsed-field gel electrophoresis, and multilocus sequence typing. Natural populations of S. aureus have a well-defined clonal population structure, but there was no evidence for the existence of hypervirulent clones. The presence or absence of 33 putative bacterial virulence determinants was examined. After adjusting for the effect of clonality, seven determinants (fhbA, cna, sdrE, sej, eta, hlg and ica) were significantly more common in invasive isolates; all contributed independently to virulence. In the host genetic studies a functional single nucleotide polymorphism at amino acid position 131 of the Fc gamma receptor IIA (FcyRIIA) gene was significantly associated with severe disease. In addition an additive x additive epistatic interaction was found between this FcyRIIA polymorphism and the functional FcyRIIIB NA1/NA2 polymorphism. These significant associations were present when community-acquired disease cases were considered alone, but absent in hospital-acquired disease cases. The putatively functional polymorphisms in genes coding for mannose binding lectin and Tolllike receptor 2 were not associated with disease. Both bacterial and host factors are important in determining the occurrence of severe S. aureus disease. In hospital-acquired infection it is likely that acquired host, bacterial and environmental factors predominate, lessening the importance of any host genetic component.
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Lindemann, Claudia. "Understanding early transcriptional events in Staphylococcus aureus infection." Thesis, University of Oxford, 2017. https://ora.ox.ac.uk/objects/uuid:037d5fff-14f7-4587-baf1-39634ec8b9ab.

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Staphylococcus aureus remains an important pathogen, which, due to its capability to develop antimicrobial resistance, imposes an increasing threat to human health. Developing preventive means to decrease disease burden is a major aim. However, the development of an S. aureus vaccine, which would be one strategy to achieve such goals, has been complicated through limited understanding of the bacterium's pathogenic mechanisms. This work uses four approaches to address these limitations: Firstly, a reproducible RNA sequencing based method for the determination of gene transcription by S. aureus in vivo during mammalian infection. Secondly, examination of the impact of the bacterial transcription regulator 'Rsp' on the bacterium, which shows that mutations in this gene have profound functional and transcriptional impacts. Thirdly, by examining the in vivo transcription of multiple S. aureus strains during infection, proposing a 'core in vivo transcriptome' of induced genes under the conditions tested. Some of these genes are known to be involved in pathogenesis, others are not completely characterised and may represent suitable vaccine antigens. Finally, this work addresses limited understanding of S. aureus pathogenesis through defining transcriptional changes in vivo, which are induced by an altered immune response in immunised hosts. Together, this body of work contributes to the understanding of S. aureus pathogenesis and provides candidate antigens for future vaccine development.
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Najar, A. G. "Protein exported by Staphylococcus aureus and the effect of some membrane modifying agents." Thesis, University of Kent, 1988. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.383048.

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Books on the topic "THE BACTERIA STAPHYLOCOCCUS AUREUS"

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Baddour, Manal M. MRSA (methicillin resistant Staphylococcus Aureus) infections and treatment. New York: Nova Science Publishers, 2010.

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Adlam, C. Structure and function of selected antigens from the pathogenic bacteria Propionibacterium acnes, Staphylococcus aureus and Pasteurella haemolytica. Birmingham: University of Birmingham, 1987.

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Emami, Saeed. New quinolones with potential anti-MRSA activity. Hauppauge, N.Y: Nova Science, 2009.

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Bjerketorp, Joakim. Novel adhesive proteins of pathogenic Staphylococci and their interaction with host proteins. Uppsala: Swedish University of Agricultural Sciences, 2004.

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Bagnoli, Fabio, Rino Rappuoli, and Guido Grandi, eds. Staphylococcus aureus. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-72063-0.

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Rice, Kelly C., ed. Staphylococcus aureus. New York, NY: Springer US, 2021. http://dx.doi.org/10.1007/978-1-0716-1550-8.

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Freeman-Cook, Lisa. Staphylococcus aureus infections. Edited by Freeman-Cook Kevin D, Alcamo I. Edward, and Heymann David L. Philadelphia: Chelsea House Publishers, 2006.

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D, Freeman-Cook Kevin, ed. Staphylococcus aureus infections. Philadelphia: Chelsea House Publishers, 2005.

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Honeyman, Allen L., Herman Friedman, and Mauro Bendinelli, eds. Staphylococcus aureus Infection and Disease. Boston, MA: Springer US, 2001. http://dx.doi.org/10.1007/b111097.

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Ji, Yinduo, ed. Methicillin-Resistant Staphylococcus Aureus (MRSA) Protocols. New York, NY: Springer US, 2020. http://dx.doi.org/10.1007/978-1-4939-9849-4.

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Book chapters on the topic "THE BACTERIA STAPHYLOCOCCUS AUREUS"

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Iandolo, John J., and George C. Stewart. "Staphylococcus aureus." In Bacterial Genomes, 743–53. Boston, MA: Springer US, 1998. http://dx.doi.org/10.1007/978-1-4615-6369-3_80.

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Lee, Jean C. "Staphylococcus aureus Vaccine." In New Bacterial Vaccines, 283–93. Boston, MA: Springer US, 2003. http://dx.doi.org/10.1007/978-1-4615-0053-7_18.

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Doškař, Jiří, Roman Pantůček, Vladislava Růžičková, and Ivo Sedláček. "Molecular Diagnostics of Staphylococcus aureus." In Detection of Bacteria, Viruses, Parasites and Fungi, 139–84. Dordrecht: Springer Netherlands, 2010. http://dx.doi.org/10.1007/978-90-481-8544-3_7.

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Pattee, Peter A. "The Genetic Map of Staphylococcus aureus." In Bacillus subtilis and Other Gram-Positive Bacteria, 489–96. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555818388.ch34.

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Sapula, Sylvia A., and Melissa H. Brown. "Antimicrobial Drug Efflux Pumps in Staphylococcus aureus." In Efflux-Mediated Antimicrobial Resistance in Bacteria, 165–95. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-39658-3_7.

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Geisinger, Edward, and Richard P. Novick. "Signal Integration and Virulence Gene Regulation in Staphylococcus aureus." In Chemical Communication among Bacteria, 161–84. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555815578.ch11.

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Kalia, Nitin Pal. "Efflux-Mediated Drug Resistance in Staphylococcus aureus." In Drug Resistance in Bacteria, Fungi, Malaria, and Cancer, 307–23. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-48683-3_13.

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Torres, Victor J., Meredith A. Benson, and Jovanka M. Voyich. "Staphylococcus aureus Pathogenesis and Virulence Factor Regulation." In Regulation of Bacterial Virulence, 58–78. Washington, DC, USA: ASM Press, 2016. http://dx.doi.org/10.1128/9781555818524.ch4.

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Valisena, S., C. Pruzzo, P. E. Varaldo, and G. Satta. "Interference of a Staphylococcus Aureus Bacteriolytic Enzyme with Polymorphonuclear Leucocyte Functions." In Bacteria, Complement and the Phagocytic Cell, 255–67. Berlin, Heidelberg: Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-85718-8_21.

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Schneewind, Olaf, and Dominique Missiakas. "Sortases, Surface Proteins, and Their Roles in Staphylococcus aureus Disease and Vaccine Development." In Protein Secretion in Bacteria, 173–88. Washington, DC, USA: ASM Press, 2019. http://dx.doi.org/10.1128/9781683670285.ch15.

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Conference papers on the topic "THE BACTERIA STAPHYLOCOCCUS AUREUS"

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Baikova, T. V., P. A. Danilov, S. A. Gonchukov, V. M. Yermachenko, A. A. Ionin, R. A. Khmelnitskii, S. I. Kudryashov, et al. "Microstructures as IR-sensors with Staphylococcus aureus bacteria." In ADVANCES IN ELECTRICAL AND ELECTRONIC ENGINEERING: FROM THEORY TO APPLICATIONS: Proceedings of the International Conference on Electrical and Electronic Engineering (IC3E 2017). Author(s), 2017. http://dx.doi.org/10.1063/1.4998125.

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Braiek, M., A. Maaref, N. Jaffrezic-Renault, K. Bekir Rokbani, B. Mrabet, and A. Bakhrouf. "5.1.1 Detection of pathogenic Staphylococcus aureus bacteria by Electrochemical Impedance." In 14th International Meeting on Chemical Sensors - IMCS 2012. AMA Service GmbH, Von-Münchhausen-Str. 49, 31515 Wunstorf, Germany, 2012. http://dx.doi.org/10.5162/imcs2012/5.1.1.

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Khair, Nedaa Kamalalden. "Activity of Antibiotic Producing Bacteria Isolated from Rhizosphere Soil Region of Different Medicinal Plants." In Qatar University Annual Research Forum & Exhibition. Qatar University Press, 2020. http://dx.doi.org/10.29117/quarfe.2020.0093.

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The rhizosphere soil of medicinal plants is rich in microorganisms that develop antibiotics as natural mechanism of protection against other microbes that live in their vicinity. The present study aims to explore the production of antibacterial agents from rhizosphere soil bacteria of 11 medicinal plants and determine their activity against Gram-negative (Pseudomonas aeruginosa, Escherichia coli) and Gram-positive (Bacillus cereus, Staphylococcus aureus) bacteria. Soil samples were collected and used to isolate antibiotic producing bacteria (APB). Those isolates (108) were first tested using Cross-streak method against test bacteria. Then, isolates that showed a positive antibacterial effect (12) were tested by antibiotic susceptibility test (AST) of their cell free supernatant (CFS) and their extracellular and intracellular secondary metabolites extraction which gave positive results. Staphylococcus aureus found to be the most sensitive test bacteria with inhibitory zones ranging from 13.5 - 19 mm. Moreover, combinatorial effect of isolates CFS with two organic acids (3% Acetic acid and 0.4 mg/ml Acetylsalicylic acid), two commercial antibiotics (0.016 mg/ml Augmentin and 0.128 mg/ml Doxycycline), and two pure antibiotics (10 mcg/disk Penicillin and 25mcg/disk Carbenicillin) was in vitro evaluated using AST. The combinations of CFS-carbenicillin showed a marked synergistic activity against all test bacteria. The presence of possible antibacterial agents as acetic acid, lactic acid and citric acid in CFS of APB was confirmed by HPLC analysis. Ultimately, in vitro antibacterial study for rhizosphere soil bacteria in this work suggests the possibility of using these bacterial metabolites in clinical infections caused by selected test bacteria, especially when they combine with antibiotics or organic acids.
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Cordero-Samortin, Angelica, Ramon G. Garcia, and Jennifer C. Dela Cruz. "The Utilization of Electromagnetic Radiation from a Bowtie Antenna for Eradicating Staphylococcus Aureus Bacteria." In ICBET 2020: 2020 10th International Conference on Biomedical Engineering and Technology. New York, NY, USA: ACM, 2020. http://dx.doi.org/10.1145/3397391.3397418.

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Ade Rizky, Vincentia, Mei Rositasari, Visensius Krisdianilo, Julia Damayanti, and Sa'adah Siregar. "Antibiotic Sensitivity Test Gentamicin in Bacteria Staphylococcus aureus with Incubation Temperature 33ºC and 35ºC." In International Conference on Health Informatics and Medical Application Technology. SCITEPRESS - Science and Technology Publications, 2019. http://dx.doi.org/10.5220/0009974305280535.

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Domingues, Lohraine Talia, Mariana Kely Diniz Gomes De Lima, Paulo Schumann Neto, Isabela Reis Manzoli, and Diego Bezerra Soares. "MECANISMOS DE RESISTÊNCIA AOS ANTIBIÓTICOS BETALACTÂMICOS PELOS STAPHYLOCOCCUS AUREUS." In II Congresso Brasileiro de Saúde On-line. Revista Multidisciplinar em Saúde, 2021. http://dx.doi.org/10.51161/rems/1446.

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Introdução: A bactéria Staphylococcus aureus é uma das mais perigosas infecções estafilocócicas resistentes a antibióticos, sobretudo porque possui alta capacidade de virulência, liberação de toxinas e conseguem se disseminar rapidamente nas regiões cutâneas, pulmonares, válvulas cardíacas e ósseas. Ademais, essas bactérias são classificadas como gram-positivas, em forma de esferas (cocos) que podem provocar espinhas e furúnculos, inclusive acometimentos mais graves, como pneumonia, meningite, endocardite, síndrome do choque tóxico e septicemia. Objetivos: Devido a elevada prevalência no número de casos e a resistência aos antibióticos betalactâmicos essa doença pode ser considerada de grande relevância ao estudo médico. Nesse contexto, são necessários mais estudos que esclareçam a padronização dessa patologia. Com isso, foi levantado a seguinte problemática: “Quais seriam os mecanismos de resistência aos antibióticos betalactâmicos pelos Staphylococcus aureus?”. Material e Métodos: A pesquisa consiste em uma revisão de literatura retrospectiva, com o objetivo de entender os aspectos de resistência aos antibióticos betalactâmicos pelos Staphylococcus aureus. Resultados: A partir desse estudo foi possível observar que as bactérias por meio da alta capacidade de replicação, conseguiram produzir algumas enzimas e estruturas que conferem um fator de resistência aos antibióticos, tais como: aderência, a cápsula de ácido hialurônico, a proteína M e outras enzimas. Nesse sentido, a enzima que mais se destaca é a betalactamase que é capaz de inibir a ação dos antibióticos betalactâmicos favorecendo ainda mais a patogenicidade durante a infecção. Conclusão: Por meio desse estudo, observou-se acentuada resistência bacteriana do Staphylococcus aureus através da capacidade de produzir enzimas principalmente a betalactamase responsável pelo fator de inviabilização dos antibióticos betalactâmicos.
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Rastogi, Vivek, Shilpi Agarwal, Rahul Gadkari, and Chandra Shakher. "Holographic optical element based digital holographic interferometer for label-free imaging of staphylococcus aureus bacteria." In Holography, Diffractive Optics, and Applications IX, edited by Changhe Zhou, Yunlong Sheng, and Liangcai Cao. SPIE, 2019. http://dx.doi.org/10.1117/12.2537317.

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Cordero-Samortin, Angelica, Jennifer C. Dela Cruz, Ramon Garcia, and Zoren Mabunga. "The Analysis of Staphylococcus aureus Bacteria after Exposure to Electromagnetic Radiation via Support Vector Machine." In 2020 IEEE 12th International Conference on Humanoid, Nanotechnology, Information Technology, Communication and Control, Environment, and Management (HNICEM). IEEE, 2020. http://dx.doi.org/10.1109/hnicem51456.2020.9400030.

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Chirila, Laura, Marcela Corina Rosu, Sabina Olaru, Cristian Tudoran, Dragos-Viorel Cosma, Alexandra Urda, Alice-Ortansa Mateescu, Gheorghe Mateescu, and Georgiana Vasile. "Cotton fabrics coated with Ag-TiO2 and Ag-TiO2/reduced graphene oxide nanocomposites." In The 8th International Conference on Advanced Materials and Systems. INCDTP - Leather and Footwear Research Institute (ICPI), Bucharest, Romania, 2020. http://dx.doi.org/10.24264/icams-2020.i.6.

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Ag-TiO2 and Ag-TiO2/reduced graphene oxide nanopowders were deposited onto 100% cotton fabrics via electrostatic spraying method. The surface of cotton fabrics was pre-treated by plasma at atmospheric pressure using argon and nitrogen mixture. The as-prepared cotton fabrics were characterized in terms of structural and optical properties by X-ray diffraction (XRD) and optical reflectance measurements. The photocatalytic self-cleaning ability of Ag-TiO2 and Ag-TiO2/reduced graphene oxide coated cotton fabrics was evaluated by the photo-discoloration of methylene blue and berries juice stains, under 6 h simulated visible light irradiation. The combined functionalized coating on cotton fabrics demonstrated an improved photocatalytic effect compared with untreated cotton fabrics. The antimicrobial activity of Ag-TiO2 and Ag-TiO2/reduced graphene oxide coated cotton fabrics was tested against the Staphylococcus aureus and Candida albicans test strains as model microorganism of skin bacteria and fungi, respectively. An antimicrobial effect against the Staphylococcus aureus is observed even if the inhibition zone is not present. Untreated fabrics showed no antibacterial activity. No inhibitory effect on fungi colony growth was observed.
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Dichtelmuller, H., and W. Stephan. "IN VIVO AND IN VITRO NEUTRALIZATION OF BACTERIAL TOXINES BY IGM ENRICHED AND CONVENTIONAL I. V. IMMUNOGLOBULINS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644255.

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Severe septic phenomena are caused bybacterial toxins. We therefore investigated the neutralization of toxins derived from Staphylococcus aureus and Pseudcmonas aeruginosa by different i.v. irtmunoglobulin preparations using hemolysis inhibition tests and mouse protection tests. The efficacy of conventional i.v. immunoglobulin containing preparations were compared with an IgM enriched i.v. immunoglobulin (Pentaglobin).For hemolysis inhibition tests sterile filtered supernatant of Staphylococcusaureus was prepared and given to human erythrocytes. When IgM enriched immunoglobulin was added, toxin depended hemolysis was inhibited. By addition of three different i.v. immunoglobulin preparationsno inhibition of hemolysis was observed. In order to confirm these results in vivo, mice were exposed to the toxic supernatant of Staphylococcus aureus intraperitoneally and treated with i.v. immunoglobulins (3.1 mg/animal) 30 min after toxin exposure. Significant protection of toxin exposed animals was achieved by IgM enriched i.v. immunoglobulin (92 % protection) but not by conventional i.v. immunoglobulin (17 % protection). Similar results were obtained when mice were exposed to toxic supernatant of Pseudcmonas aeruginosa instead of Staphylococcus aureus. We therefore conclude, that IgM is essential for neutralization of bacterial toxins and IgM enriched i.v. immunoglobulins are more effective in therapy of severe septic phenomena, compared to conventional i.v. immunoglobulins.
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Reports on the topic "THE BACTERIA STAPHYLOCOCCUS AUREUS"

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Spencer, Jessica, and Uzo Chukwuma. Methicillin-Resistant Staphylococcus aureus (MRSA) Infections in the Department of Defense (DOD): Annual Summary 2013. Fort Belvoir, VA: Defense Technical Information Center, January 2015. http://dx.doi.org/10.21236/ada612614.

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Timms, Leo L., and Phil Sears. Field Trial Evaluation of Extended Pirlimycin Therapy With or Without Vaccination for Staphylococcus Aureus Mastitis. Ames (Iowa): Iowa State University, January 2004. http://dx.doi.org/10.31274/ans_air-180814-981.

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Ju, Mohan, Yueying Huang, Xiaofeng Xu, Yiyi Qian, Yingmin Bi, Shuang Liu, Xiangyu Li, Shuaiyue Dong, Jinyi Yuan, and Dongfang Lin. Predictors of mortality in patients with Methicillin-resistant Staphylococcus aureus bloodstream infection: A meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, February 2021. http://dx.doi.org/10.37766/inplasy2021.2.0082.

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Neyra, Joan M. Nasal Colonization with Methicillin-Resistant Staphylococcus aureus in Military Personnel in a Developing Country - Development of a Skin and Soft Tissue Infection Surveillance System in the Peruvian Air Force. Fort Belvoir, VA: Defense Technical Information Center, March 2015. http://dx.doi.org/10.21236/ad1012737.

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Health hazard evaluation report: HETA-2009-0098-3103, evaluation of methicillin-resistant Staphylococcus aureus (MRSA) cases among employees at a workholding manufacturing facility, Positrol Inc., Cincinnati, Ohio. U.S. Department of Health and Human Services, Public Health Service, Centers for Disease Control and Prevention, National Institute for Occupational Safety and Health, March 2010. http://dx.doi.org/10.26616/nioshheta200900983103.

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