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Books on the topic 'The diathesis-stress model of depression'

Author: Grafiati

Published: 4 June 2021

Last updated: 8 February 2022

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1

Amster, Hagit. The effect of dramatherapy on elements of depression in the case o method model to strategies for coping with stress among nursingf a hospitalised adolescent girl. University of Surrey Roehampton, 2001.

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2

Krivoyekov, Syergyey, and Roman Ayzman. Psychophysiology. INFRA-M Academic Publishing LLC., 2015. http://dx.doi.org/10.12737/10884.

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Psychophysiology — the science studying interrelation of mentality of the person and physiological processes. Fundamental knowledge of work of a brain, first of all, of nervous regulation of functions of an organism, the general and specific features of the highest falls within the scope of its interests nervous activity, the defining character and behavior of the person, psychophysiological mechanisms of regulation of functional states. In the book neurophysiological bases of coding and information processing in nervous system, neural mechanisms of feelings, perceptions, mem

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3

Tacchi, Mary Jane, and Jan Scott. 4. Models of depression. Oxford University Press, 2017. http://dx.doi.org/10.1093/actrade/9780199558650.003.0004.

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Epidemiological studies of depression give important insights into populations at higher risk of depression, but not everyone in certain high-risk circumstances will develop a clinical depression. ‘Models of depression’ highlights some of the most well-known biological, psychological, and social models ranging from the monoamine hypothesis to Beck’s cognitive model, and then discusses attempts to integrate these into a multi-dimensional psycho-bio-social theory. It highlights the interaction between stress and vulnerability factors, and the importance of considering the origins of the vulnerab

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4

Stevenson, Jerry G., Robert F. Muzenrider, and Robert T. Golembiewski. Stress in Organizations: Toward A Phase Model of Burnout. Praeger Publishers, 1985.

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5

Stoddard, Frederick J., and Robert L. Sheridan. Wound Healing and Depression. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190603342.003.0009.

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Depression and wound healing are bidirectional processes for adults and children consistent with the conception of depression as systemic. This systemic interaction is similar to the “bidirectional impact of mood disorder on risk for development, progression, treatment, and outcomes of medical illness” generally. And, evidence is growing that the bidirectional impact of mood disorder may be true for injuries and for trauma surgery. Animal models have provided some support that treatment of depression may improve wound healing. An established biological model for a mechanism delaying wound heal

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6

Keel, Pamela K., and Lauren A. Holland. Eating Disorders. Edited by C. Steven Richards and Michael W. O'Hara. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780199797004.013.017.

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This chapter examines patterns of comorbidity between eating disorders and mood, anxiety, and substance use disorders along with evidence regarding support for different theoretical models that may account for these patterns. Although comorbidity estimates may be inflated by reliance on treatment-seeking samples and double counting of symptoms that overlap between syndromes, evidence supports elevated risk of mood, anxiety, and substance use disorders in anorexia nervosa, bulimia nervosa, and binge eating disorder. Data from family and twin studies support that eating and anxiety disorders may

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7

Watson, David, and Sara M. Stasik. Examining the Comorbidity Between Depression and the Anxiety Disorders From the Perspective of the Quadripartite Model. Edited by C. Steven Richards and Michael W. O'Hara. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780199797004.013.026.

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Major depression and posttraumatic stress disorder (PTSD) represent heterogeneous combinations of symptoms. Analyses focusing on these distinctive symptom dimensions can play an important role in explicating key diagnostic phenomena such as comorbidity. We review depression and PTSD from the perspective of the quadripartite model, which posits that it is important to consider two quantitative elements when analyzing the properties of symptoms: (a) the magnitude of their general distress component and (b) their level of specificity. Within both disorders, we identified certain symptoms—insomnia

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8

Swift, Adrienne. Anxiety and college women: Exploring a diathesis-stress model. 1996.

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9

Swift, Adrienne. Anxiety and college women: Exploring a diathesis-stress model. 1996.

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10

Alcantara, Lyonna F., Eric M. Parise, and Carlos A. Bolaños-Guzmán. Animal Models of Mood Disorders. Edited by Dennis S. Charney, Eric J. Nestler, Pamela Sklar, and Joseph D. Buxbaum. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190681425.003.0026.

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Animal modeling has advanced our understanding of the underlying pathophysiology of human neuropsychiatric disorders and facilitated development of safer, more efficient medications. Similar to humans with depression, rodents exposed to various stress paradigms exhibit aberrant responses to rewarding stimuli, along with hormonal and immunological dysregulation. Development of more complex models, such as social defeat, has led to a firmer grasp of the mechanisms mediating resilience and susceptibility to stress; and adapted versions of social defeat have yielded insights into how emotional str

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11

Nestler, Eric J. The Biological Basis of Depression. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190603342.003.0001.

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Most major advances in biomedical research have relied on the use of animal and cell models of disease. This is a particularly difficult challenge in psychiatry, because many core symptoms of mental illnesses are inherently inaccessible in animals. Moreover, because still today there are no bona fide molecular-cellular abnormalities that are pathogenomic for these illnesses, cell models are even more far afield. This chapter reviews efforts to overcome these obstacles and use animal and cell studies to better understand the biological basis of depression and to develop improved treatments. An

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12

Frydenberg, Erica, ed. Beyond Coping. Oxford University Press, 2015. http://dx.doi.org/10.1093/med:psych/9780198508144.001.0001.

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There are many challenges to be faced in contemporary society including the stresses of everyday living in the technological age and changes in patterns of employment and family life. Depression is being experienced in ‘epidemic’ proportions in many Western communities, and in particular amongst young people. The search for effective ways to reverse this trend has resulted in a significant shift in psychological approach from a focus on helplessness and pathology to a more positive orientation that emphasises health and well-being. This volume brings together leading researchers in the field o

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13

McCarty, Richard. Stress and Mental Disorders: Insights from Animal Models. Oxford University Press, 2020. http://dx.doi.org/10.1093/med-psych/9780190697266.001.0001.

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Stress has now been recognized as an important factor in the development or recurrence of various mental disorders, from major depressive disorder to bipolar disorder to anxiety disorders. Stressful stimuli appear to exert their effects by acting upon individuals with susceptible genotypes. Over the past 50 years, animal models have been developed to study these dynamic interactions between stressful stimuli and genetically susceptible individuals during prenatal and postnatal development and into adulthood. This book begins with a discussion of the history of psychiatric diagnosis and the rec

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14

Duman, Ronald S. Neurotrophic Mechanisms of Depression. Edited by Dennis S. Charney, Eric J. Nestler, Pamela Sklar, and Joseph D. Buxbaum. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190681425.003.0027.

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Early theories of depression and treatment response were centered on the monoamine neurotransmitters, but more recent work has focused on functional and structural synaptic plasticity and the role of neurotrophic factors, particularly brain derived neurotrophic factor (BDNF). Neurotrophic factors regulate all aspects of neuronal function, including adaptive plasticity, synapse formation, and neuronal survival. Chronic stress and depression cause reductions in levels of BDNF and other key factors, including vascular endothelial growth factor (VEGF) and fibroblast growth factor 2 (FGF2), in cort

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15

Landsbergis, Paul A., Marnie Dobson, Anthony D. LaMontagne, BongKyoo Choi, Peter Schnall, and Dean B. Baker. Occupational Stress. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190662677.003.0017.

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This chapter describes sources of stress in the work environment, their adverse effects on the health of workers, and how they are influenced by economic globalization, political systems, laws, government policies, and the changing labor market. Models of occupational stress, in particular job strain and effort-reward imbalance, are presented. Additional occupational stressors are described, including long work hours, shift work, precarious work and job insecurity, work-family conflict and organizational injustice, including discrimination, harassment, and bullying. The health and safety conse

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16

Ménard, Caroline, Madeline L. Pfau, Georgia E. Hodes, and Scott J. Russo. Immune Mechanisms of Depression. Edited by Dennis S. Charney, Eric J. Nestler, Pamela Sklar, and Joseph D. Buxbaum. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190681425.003.0028.

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Mood disorders such as major depressive disorder (MDD) are diagnosed largely based upon behavioral symptoms rather than biological factors. Some have argued that a lack of rigorous biomarker-based diagnosis is the reason why 30%–50% of MDD patients are unresponsive to traditional antidepressant medications. Over the past few decades, MDD has been shown to be highly prevalent in patients suffering from chronic inflammatory conditions, such as lupus erythematosus, multiple sclerosis, etc. Moreover, subgroups of MDD patients have shown consistently higher levels of circulating pro-inflammatory cy

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17

Nestler, Eric J. New Approaches for Treating Depression. Edited by Dennis S. Charney, Eric J. Nestler, Pamela Sklar, and Joseph D. Buxbaum. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190681425.003.0030.

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Several obstacles have impeded the introduction of new antidepressant medications over the past six decades. These obstacles include our still rudimentary knowledge of the biological basis of depression, as well as difficulties in evaluating the therapeutic efficacy of new putative antidepressant mechanisms in pathophysiologically distinct subtypes of the syndrome. Despite these obstacles, several tangible steps can be taken to advance depression treatment moving forward. The field needs to continue to take advantage of serendipitous discoveries in humans, such as the demonstration of rapid an

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18

Watson, David, and Michael W. O'Hara. Understanding the Emotional Disorders. Oxford University Press, 2017. http://dx.doi.org/10.1093/med:psych/9780199301096.001.0001.

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Understanding the Emotional Disorders: A Symptom-Based Approach examines replicable symptom dimensions contained within five adjacent diagnostic classes in the fifth edition of the Diagnostic and Statistical Manual of Mental Disorders: depressive disorders, bipolar and related disorders, anxiety disorders, obsessive-compulsive and related disorders, and trauma- and stressor-related disorders. It reviews several problems and limitations associated with traditional, diagnosis-based approaches to studying psychopathology, and it establishes the theoretical and clinical value of analyzing specific

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19

Richards, C. Steven, and Michael W. O'Hara, eds. The Oxford Handbook of Depression and Comorbidity. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780199797004.001.0001.

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Depression is frequently associated with other psychiatric disorders, chronic health problems, and distressed close relationships. This comorbidity between depression and other disorders and problems is important. Furthermore, there has been a large increase in research on depressive comorbidity. Therefore, a book of 37 state-of-the-art reviews by experts will be helpful to teachers, researchers, practitioners, developers of relevant policies, and students in these areas. The comorbidity of depression with other psychiatric disorders is addressed in chapters focusing on panic disorder, post-tr

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20

Belsher, Bradley E., Daniel P. Evatt, Michael C. Freed, and Charles C. Engel. Internet and Computer-Based Treatments for the Management of PTSD. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190205959.003.0014.

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A rapid expansion in the development of telehealth treatments has occurred during the past several decades, with a growing body of evidence supporting online therapies for behavioral health disorders. These online interventions have focused primarily on the treatment of depression, panic disorder, social phobia, and generalized anxiety disorder. More recently, and with the relative success of the previous Web-based treatments, several online treatments for posttraumatic stress disorder (PTSD) have emerged. An overview of Internet and computer-based treatments (ICTs) for PTSD is presented, incl

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21

Ayers, Susan, and Elizabeth Ford. Posttraumatic Stress During Pregnancy and the Postpartum Period. Edited by Amy Wenzel. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780199778072.013.18.

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Research on post-traumatic stress disorder (PTSD) in pregnancy and postpartum is relatively new but clearly demonstrates the importance of recognizing and treating women with PTSD at this time. Women with PTSD in pregnancy are at greater risk of pregnancy complications and health behaviors that have a negative impact on the woman and fetus. Approximately –3% of women develop PTSD after giving birth, and rates increase for women who have preterm or stillborn infants or life-threatening complications during pregnancy or labor. Models of the etiology of postpartum PTSD focus on the interaction am

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22

Barrett, Catherine E., and Larry J. Young. Molecular Neurobiology of Social Bonding. Edited by Turhan Canli. Oxford University Press, 2013. http://dx.doi.org/10.1093/oxfordhb/9780199753888.013.001.

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Many psychiatric illnesses, including autism spectrum disorders (ASD), schizophrenia, and depression, are characterized by impaired social cognition and a compromised ability to form social relationships. Although drugs are currently available to treat other symptoms of these disorders, none specifically target the social deficits. In order to develop pharmacotherapies to enhance social functioning, particularly for ASD where social impairment is a core symptom, we must first understand the basic neurobiology underlying complex social behaviors. The socially monogamous prairie vole (Microtus o

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23

Mizock, Lauren, and Erika Carr. Women with Serious Mental Illness. Oxford University Press, 2020. http://dx.doi.org/10.1093/med-psych/9780190922351.001.0001.

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Women with Serious Mental Illness: Gender-Sensitive and Recovery-Oriented Care calls attention to a topic and a population that have been overlooked in research and psychotherapy—women with serious mental illnesses (schizophrenia, severe depression, bipolar disorder, and complex post-traumatic stress disorder). The book focuses on the history of mistreatment, marginalization, and oppression women with serious mental illness have encountered, not only from the general public but within the mental health system as well. This book provides an overview of recovery-oriented care for women with seri

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24

Kreitzer, Mary Jo, and Mary Koithan, eds. Integrative Nursing. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199860739.001.0001.

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Integrative medicine is defined as healing-oriented medicine that takes account of the whole person (body, mind, and spirit) as well as all aspects of lifestyle; it emphasizes the therapeutic relationship and makes use of appropriate therapies, both conventional and alternative. This resource is the first complete roadmap to integrative nursing, providing a step-by-step guide to assess and clinically treat conditions through a variety of combined methodologies including wellness, lifestyle enhancement, and nutrition. This resource puts forth both the skills and theoretical frameworks for multi

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25

Onoye, Jane M., Deborah Goebert, and Leslie Morland. Cross-Cultural Differences in Adjustment to Pregnancy and the Postpartum Period. Edited by Amy Wenzel. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780199778072.013.31.

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Cultural context is important to understanding cross-cultural difference in adjustment to pregnancy and the postpartum period. Culture is complex, with interrelated variables posing challenges for research. Highlighted with examples of research with women from Western, Eastern, Native, and Other cultures, the chapter discusses variables such as acculturation and acculturative stress, social support, religious and spiritual beliefs and practices, and help-seeking and utilization of services in perinatal mental health and adjustment. Although rates of psychiatric symptoms and disorders vary acro

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26

Vögele, Claus, Annika P. C. Lutz, and E. Leigh Gibson. Mood, Emotions, and Eating Disorders. Edited by W. Stewart Agras and Athena Robinson. Oxford University Press, 2017. http://dx.doi.org/10.1093/oxfordhb/9780190620998.013.8.

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Mood and emotions are intrinsically involved with eating. This chapter discusses basic mechanisms, findings, and models that help our understanding of the interactions between eating and emotions, in both clinical and nonclinical populations. The finding that negative affect predicts EDs transdiagnostically, and that comorbidity with depressive disorders and anxiety disorders is the norm among patients with EDs suggests that EDs may not necessarily be restricted to domains of eating behavior and body image but may also be associated with significant difficulties in affective functioning. This

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27

Bagby, R. Michael, Amanda Uliaszek, Tara M. Gralnick, and Nadia Al-Dajani. Axis I Disorders. Edited by Thomas A. Widiger. Oxford University Press, 2016. http://dx.doi.org/10.1093/oxfordhb/9780199352487.013.5.

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The purpose of this chapter is to summarize and discuss the complex relationship between Five Factor Model (FFM) personality traits and clinical (Axis I) psychopathology, including depressive, bipolar, anxiety, obsessive–compulsive, eating, schizophrenia and psychotic, trauma and stress-related, and substance use disorders. Considered herein will be the alternative forms of relationship, including vulnerability, common cause, pathoplasty, complication/scar, and spectrum. This chapter will highlight the necessity for well-designed, longitudinal studies aimed at elucidating the complex relations

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28

Feather, N. T. Historical Background to Research on Job Loss, Unemployment, and Job Search. Edited by Ute-Christine Klehe and Edwin van Hooft. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780199764921.013.001.

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This chapter provides a selective review of past research on job loss, unemployment, and job search up to the beginning of the 1990s. The Great Depression studies in the 1930s at Marienthal by Jahoda and colleagues and by Bakke at Greenwich and New Haven are described, along with other research at the time. These early studies sowed the seeds for subsequent research programs in England, Europe, and Australia; the theories that emerged from this early and later research are described. They include stage theory, deprivation theory, agency theory, and vitamin theory. Other more general approaches

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29

Arango, Victoria, and Mark Underwood. The Neurobiology of Suicide and Implications for Treatment and Prevention. Edited by Phillip M. Kleespies. Oxford University Press, 2015. http://dx.doi.org/10.1093/oxfordhb/9780199352722.013.23.

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Suicide has a biological component. It is the result not only of the necessary biological vulnerability, but also of multiple factors that must converge to elicit the behavior. These factors are discussed in this chapter and include genetic and epigenetic mechanisms, as well as psychopathologic, environmental, and stressful considerations, such as exposure to early adversity. We present a mathematical model with suicide as the outcome. The equation is based on a conventional stress-diathesis model, but it underscores the complexity of suicide behavior. In addition to discussing the contributor

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