Journal articles: 'Therapeutic diet'

1

Hager, Mary H. "Therapeutic Diet Order Writing." Topics in Clinical Nutrition 22, no. 1 (January 2007): 28–36. http://dx.doi.org/10.1097/00008486-200701000-00005.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

Ždrale, Zdravko, Ana Kaličanin, Slaviša Đurđević, Nikola Bogunović, Svjetlana Ždrale, Momir Janjić, Hristo Anđelski, and Željko Kerkez. "Therapeutic and diet aspects of milk." Zdravstvena zastita 43, no. 6 (2014): 37–44. http://dx.doi.org/10.5937/zz1405037z.

Full text

APA, Harvard, Vancouver, ISO, and other styles

3

Ghosh, Amrita. "Therapeutic Diet: What We Need to Consider!" Bengal Physician Journal 5, no. 2 (2018): 25–27. http://dx.doi.org/10.5005/jp-journals-10070-5209.

Full text

Abstract:

ABSTRACT Therapeutic diet is needed so that nutritional factors may not affect individuals in diseases during convalescence and rehabilitation. Different research groups have observed a correlation between diseases and diets with diagnosis, prognosis, clinical outcomes and complications. In spite of considerable progress in healthcare, there is scarce evidence-based research excellence on community-level compliance on dietary advice. We need to change our mindset to find user-friendly dietary guidelines for the improvement in clinical approach and treatment in patients with diverse diseases in daily medical practice.

APA, Harvard, Vancouver, ISO, and other styles

4

Dubenko, S. E., and Tatyana V. Mazhaeva. "THE COMPARATIVE ASSESSMENT OF THE EFFECTIVENESS OF THE THERAPEUTIC DIET." Hygiene and sanitation 96, no. 12 (March 27, 2019): 1176–78. http://dx.doi.org/10.18821/0016-9900-2017-96-12-1176-1178.

Full text

Abstract:

In order to evaluate the effectiveness of therapeutic diet depending on background diet, eating behavior and occupational exposure, authors assessed food intake, nutritional adequacy of the daily food consumption in 2000 workers of similar social and economic background, working in similar conditions. Therapeutic diets No. 2 and No. 3 used at 6 metallurgical facilities were analyzed. The effectiveness of therapeutic diets developed by the authors was evaluated in workers of relevant occupations at two enterprise-based prevention and rehabilitation centers. The authors assessed the nutritional status and health of the workers with unbalanced background diet but on-the-job therapeutic diet and the workers who are on a balanced background diet and on-the-job therapeutic diet. The results show workers with unbalanced diet in terms of fat, carbohydrate, vitamin and trace element consumption as well as ill-conceived therapeutic diet to have impaired fat and carbohydrate metabolism, obesity of various degrees, premorbidities that exacerbate the effects of occupational exposure and increase the likelihood of occupational and work-related diseases. The comprehensive preventive approach, that involves reducing occupational exposure and improving the daily diet including the therapeutic diet, yields the most effective results. The use of therapeutic diets in workers maintaining a balanced background diet results in significant health gain compared to those who are treated in the same enterprise-based prevention and rehabilitation centers but whose background diet is unbalanced. They exhibit weight loss, lower arterial blood pressure, improved carbohydrate and fat metabolism, antioxidant status and functional indicators as well as reduced metal levels in biological media.

APA, Harvard, Vancouver, ISO, and other styles

5

Stănescu, Ana Maria Alexandra, Ioana Veronica Grăjdeanu, Mihaela Adela Iancu, Andreea Alexandra Hlescu, Ovidiu Gabriel Bratu, Andrei Kozma, and Camelia Cristina Diaconu. "Gluten-free diet – possible therapeutic approach to vitiligo." Romanian Journal of Medical Practice 13, no. 4 (December 31, 2018): 255–59. http://dx.doi.org/10.37897/rjmp.2018.4.4.

Full text

APA, Harvard, Vancouver, ISO, and other styles

6

Halliwell, Barry, Irwin K. Cheah, and Richard M. Y. Tang. "Ergothioneine – a diet‐derived antioxidant with therapeutic potential." FEBS Letters 592, no. 20 (June 15, 2018): 3357–66. http://dx.doi.org/10.1002/1873-3468.13123.

Full text

APA, Harvard, Vancouver, ISO, and other styles

7

Chentouf, Amina. "Ketogenic diet: a therapeutic alternative in pediatric refractory epilepsies." North African Journal of Food and Nutrition Research 6, no. 13 (February 27, 2022): 39–45. http://dx.doi.org/10.51745/najfnr.6.13.39-45.

Full text

Abstract:

Several studies have demonstrated the effectiveness of ketogenic diet (KD) on refractory epilepsies, especially in children. In this category of patients, a strong collaboration between the healthcare providers and the full engagement of parents is required. The regimen is significantly effective for different types of epilepsy that are resistant to medical treatment. It can also be used as a first-line treatment, given its well tolerance. Treatment has been rolled out for children but research into its use in adults is promising. There is no international or national protocol, the implementation of this diet follows a rigor being specific to every ketogenic center. The understanding of the underlying mechanisms is not well elucidated, it would make it possible to optimize the clinical use of the ketogenic diet, but also to develop novel antiepileptic treatments. This article aims to review the different variants of KD and their prescription terms in children with intractable epilepsy. Keywords: drug-resistant epilepsy, ketogenic diet, children, Modified Atkins Diet.

APA, Harvard, Vancouver, ISO, and other styles

8

TRUDEAU, ELYSE, and LAURETTE DUBÉ. "Moderators and Determinants of Satisfaction with Diet Counseling for Patients Consuming a Therapeutic Diet." Journal of the American Dietetic Association 95, no. 1 (January 1995): 34–39. http://dx.doi.org/10.1016/s0002-8223(95)00010-0.

Full text

APA, Harvard, Vancouver, ISO, and other styles

9

Pesce, Marcella, Martina Cargiolli, Sara Cassarano, Barbara Polese, Barbara De Conno, Laura Aurino, Nicola Mancino, and Giovanni Sarnelli. "Diet and functional dyspepsia: Clinical correlates and therapeutic perspectives." World Journal of Gastroenterology 26, no. 5 (February 7, 2020): 456–65. http://dx.doi.org/10.3748/wjg.v26.i5.456.

Full text

APA, Harvard, Vancouver, ISO, and other styles

10

Park, K. A., W. I. Cho, K. J. Song, Y. S. Lee, I. S. Sung, and S. M. Choi-Kwon. "Assessment of nurses' nutritional knowledge regarding therapeutic diet regimens." Nurse Education Today 31, no. 2 (February 2011): 192–97. http://dx.doi.org/10.1016/j.nedt.2010.05.017.

Full text

APA, Harvard, Vancouver, ISO, and other styles

11

Al-Khalifa, Alaa, Thazhumpal Chacko Mathew, Naji S. Al-Zaid, Elizabeth Mathew, and Hussein M. Dashti. "Therapeutic role of low-carbohydrate ketogenic diet in diabetes." Nutrition 25, no. 11-12 (November 2009): 1177–85. http://dx.doi.org/10.1016/j.nut.2009.04.004.

Full text

APA, Harvard, Vancouver, ISO, and other styles

12

Reid, Christopher A., Saul Mullen, Tae Hwan Kim, and Steven Petrou. "Epilepsy, energy deficiency and new therapeutic approaches including diet." Pharmacology & Therapeutics 144, no. 2 (November 2014): 192–201. http://dx.doi.org/10.1016/j.pharmthera.2014.06.001.

Full text

APA, Harvard, Vancouver, ISO, and other styles

13

Haas, Richard H., Marylynne A. Rice, Doris A. Trauner, T. Allen Merritt, John M. Opitz, and James F. Reynolds. "Therapeutic effects of a ketogenic diet in rett syndrome." American Journal of Medical Genetics 25, S1 (1986): 225–46. http://dx.doi.org/10.1002/ajmg.1320250525.

Full text

APA, Harvard, Vancouver, ISO, and other styles

14

Gorria Redondo, Nerea, María Luz Angulo García, María Montesclaros Hortigüela Saeta, and David Conejo Moreno. "Ketogenic diet as a therapeutic option in refractory epilepsy." Anales de Pediatría (English Edition) 84, no. 6 (June 2016): 341–43. http://dx.doi.org/10.1016/j.anpede.2015.10.020.

Full text

APA, Harvard, Vancouver, ISO, and other styles

15

Roe, Charles R., and Fanny Mochel. "Anaplerotic diet therapy in inherited metabolic disease: Therapeutic potential." Journal of Inherited Metabolic Disease 29, no. 2-3 (April 2006): 332–40. http://dx.doi.org/10.1007/s10545-006-0290-3.

Full text

APA, Harvard, Vancouver, ISO, and other styles

16

Kalra, Sanjay, Rajiv Singla, Rahul Rosha, Munish Dhawan, Deepak Khandelwal, and Bharti Kalra. "The Ketogenic Diet." US Endocrinology 14, no. 2 (2018): 62. http://dx.doi.org/10.17925/use.2018.14.2.62.

Full text

Abstract:

The ketogenic diet (KD), a well-established treatment for childhood epilepsy, is gradually gaining acceptance as a therapeutic modality for obesity and type 2 diabetes. The perception of ketone bodies as an unhealthy or “sinful” entity has led to concerns and doubts regarding the efficacy and safety of KD in physicians. This article describes the mechanism of action of KD and shares a pragmatic approach to its usage. It highlights the importance of predietary counseling, screening for indications/contraindications, and clinico-nutritional monitoring during therapy. Robust indications for KD are mentioned, to help place KD’s utility in the management of obesity and type 2 diabetes.

APA, Harvard, Vancouver, ISO, and other styles

17

Mank, Madeleine M., Leah F. Reed, Camille J. Walton, Madison L. T. Barup, Jennifer L. Ather, and Matthew E. Poynter. "Therapeutic ketosis decreases methacholine hyperresponsiveness in mouse models of inherent obese asthma." American Journal of Physiology-Lung Cellular and Molecular Physiology 322, no. 2 (February 1, 2022): L243—L257. http://dx.doi.org/10.1152/ajplung.00309.2021.

Full text

Abstract:

Obese asthmatics tend to have severe, poorly controlled disease and exhibit methacholine hyperresponsiveness manifesting in proximal airway narrowing and distal lung tissue collapsibility. Substantial weight loss in obese asthmatics or in mouse models of the condition decreases methacholine hyperresponsiveness. Ketone bodies are rapidly elevated during weight loss, coinciding with or preceding relief from asthma-related comorbidities. As ketone bodies may exert numerous potentially therapeutic effects, augmenting their systemic concentrations is being targeted for the treatment of several conditions. Circulating ketone body levels can be increased by feeding a ketogenic diet or by providing a ketone ester dietary supplement, which we hypothesized would exert protective effects in mouse models of inherent obese asthma. Weight loss induced by feeding a low-fat diet to mice previously fed a high-fat diet was preceded by increased urine and blood levels of the ketone body β-hydroxybutyrate (BHB). Feeding a ketogenic diet for 3 wk to high-fat diet-fed obese mice or genetically obese db/db mice increased BHB concentrations and decreased methacholine hyperresponsiveness without substantially decreasing body weight. Acute ketone ester administration decreased methacholine responsiveness of normal mice, and dietary ketone ester supplementation of high-fat diet-fed mice decreased methacholine hyperresponsiveness. Ketone ester supplementation also transiently induced an “antiobesogenic” gut microbiome with a decreased Fermicutes/Bacteroidetes ratio. Dietary interventions to increase systemic BHB concentrations could provide symptom relief for obese asthmatics without the need for the substantial weight loss required of patients to elicit benefits to their asthma through bariatric surgery or other diet or lifestyle alterations.

APA, Harvard, Vancouver, ISO, and other styles

18

Trandafir, Laura Mihaela, Ginel Baciu, Maria Magdalena Leon Constantin, Alexandra Mastaleru, Oana Raluca Temneanu, Bogdan Mihai, Otilia Novac, et al. "Predictive Biological Markers in Post-therapeutic Evolution in Obese Patients." Revista de Chimie 69, no. 11 (December 15, 2018): 3048–51. http://dx.doi.org/10.37358/rc.18.11.6679.

Full text

Abstract:

Childhood obesity is a leading public health concern because it represents a risk factor for many comorbid conditions in youth, such as cardiovascular disease, metabolic syndrome and sleep apnea. The purposes of the study were to evaluate the effect of the program at 6 months after the first visit and determine the predictive factors. We realised a retrospective study that included 69 obese children and adolescent, boys and girls, followed-up at Saint Mary Children�s Hospital and Regional Center of Diagnosis, Counselling and Monitoring of Overweight and Obese Children from �Grigore T. Popa� University of Medicine and Pharmacy Iasi, Romania, aged 12 to 18 years. The patients were included in two groups: group 1 included 38 patients receveived a hypocaloric diet only and group 2 included 31 patients received a hypocaloric diet associated with kinetotherapy and psychoterapy. We evaluated the body mass index, total cholesterol and tryglicerides before and after treatment. Our results confirm that diet and physical activity affects significantly the serum lipid profile. In this context, decreasing obesity in children through diet and exercise should be an important strategy for preventing cardio-metabolical disease in adult.

APA, Harvard, Vancouver, ISO, and other styles

19

Liyanage, S. Imindu, Prachi Vilekar, and Donald F. Weaver. "Nutrients in Alzheimer’s Disease: The Interaction of Diet, Drugs and Disease." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 46, no. 1 (January 2019): 23–34. http://dx.doi.org/10.1017/cjn.2018.353.

Full text

Abstract:

AbstractIn recent decades, clinical trials in Alzheimer’s disease (AD) have failed at an unprecedented rate. The etiology of AD has since come under renewed scrutiny, both to elucidate the underlying pathologies and to identify novel therapeutic strategies. Here, diet has emerged as a potential causative/protective agent. A variety of nutrients, including lipids, minerals, vitamins, antioxidants and sugars as well as broader dietary patterns and microbiotal interactions have demonstrated associations with AD. Although clinical trials have yet to definitively implicate any singular dietary element as therapeutic or causative, it is apparent that dietary preferences, likely in complex synergies, may influence the risk, onset and course of AD. This review catalogs the impact of major dietary elements on AD. It further examines an unexplored reciprocal association where AD may modulate diet, as well as how potential therapeutics may complicate these interactions. In doing so, we observe diet may have profound effects on the outcome of a clinical trial, either as a confounder of a drug/disease interaction or as a generally disruptive covariate. We therefore conclude that future clinical trials in AD should endeavor to control for diet, either in study design or subsequent analyses.

APA, Harvard, Vancouver, ISO, and other styles

20

Sinay, Isaac R., Pablo Arias, Marta A. Schnitman, Sergio A. Damilano, Maria C. Faingold, and Jaime A. Moguilevsky. "Diet only or diet and sulfonylureas in mild type II diabetes (niddm)? Pathophysiologic and therapeutic implications." Acta Diabetologica Latina 25, no. 4 (October 1988): 289–97. http://dx.doi.org/10.1007/bf02581127.

Full text

APA, Harvard, Vancouver, ISO, and other styles

21

Afifi, Tarek, Aml El-Ashmawy, and Nadia El-Fakharany. "Evaluation of the therapeutic diet for enhancement of acne status." Bulletin of the National Nutrition Institute of the Arab Republic of Egypt 51, no. 1 (June 1, 2018): 19–38. http://dx.doi.org/10.21608/bnni.2018.14160.

Full text

APA, Harvard, Vancouver, ISO, and other styles

22

Fabbri, Andrea, Giorgio Gambale, Ruggero Maria Corso, and Giulio Marchesini. "Therapeutic hypothermia after cardiac arrest: The Mediterranean-diet Italian style." Resuscitation 82, no. 5 (May 2011): 637–38. http://dx.doi.org/10.1016/j.resuscitation.2010.12.021.

Full text

APA, Harvard, Vancouver, ISO, and other styles

23

Sharma, V. K. "2.219 KETOGENIC DIET: A POSSIBLE THERAPEUTIC TARGET FOR PARKINSON'S DISEASE?" Parkinsonism & Related Disorders 18 (January 2012): S119. http://dx.doi.org/10.1016/s1353-8020(11)70543-0.

Full text

APA, Harvard, Vancouver, ISO, and other styles

24

Lascelles, B. D. X., V. DePuy, A. Thomson, B. Hansen, D. J. Marcellin-Little, V. Biourge, and J. E. Bauer. "Evaluation of a Therapeutic Diet for Feline Degenerative Joint Disease." Journal of Veterinary Internal Medicine 24, no. 3 (March 22, 2010): 487–95. http://dx.doi.org/10.1111/j.1939-1676.2010.0495.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

25

Tan, Ying, Jin-Hu Chen, Jia-Ni Zhang, Li-Juan Wang, and Hui-Xia Liu. "Therapeutic effects of diet change on nonalcoholic fatty liver disease." World Chinese Journal of Digestology 17, no. 33 (2009): 3394. http://dx.doi.org/10.11569/wcjd.v17.i33.3394.

Full text

APA, Harvard, Vancouver, ISO, and other styles

26

LARSEN, P. MUNKHOLM, D. RASMUSSEN, B. RØNN, O. MUNCK, J. ELMGREEN, and V. BINDER. "Elemental diet: a therapeutic approach in chronic inflammatory bowel disease." Journal of Internal Medicine 225, no. 5 (May 1989): 325–31. http://dx.doi.org/10.1111/j.1365-2796.1989.tb00089.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

27

Kakodkar, Samir, and Ece A. Mutlu. "Diet as a Therapeutic Option for Adult Inflammatory Bowel Disease." Gastroenterology Clinics of North America 46, no. 4 (December 2017): 745–67. http://dx.doi.org/10.1016/j.gtc.2017.08.016.

Full text

APA, Harvard, Vancouver, ISO, and other styles

28

Lu, Ping-Hsun, Min-Chien Yu, Meng-Jiun Wei, and Ko-Lin Kuo. "The Therapeutic Strategies for Uremic Toxins Control in Chronic Kidney Disease." Toxins 13, no. 8 (August 17, 2021): 573. http://dx.doi.org/10.3390/toxins13080573.

Full text

Abstract:

Uremic toxins (UTs) are mainly produced by protein metabolized by the intestinal microbiota and converted in the liver or by mitochondria or other enzymes. The accumulation of UTs can damage the intestinal barrier integrity and cause vascular damage and progressive kidney damage. Together, these factors lead to metabolic imbalances, which in turn increase oxidative stress and inflammation and then produce uremia that affects many organs and causes diseases including renal fibrosis, vascular disease, and renal osteodystrophy. This article is based on the theory of the intestinal–renal axis, from bench to bedside, and it discusses nonextracorporeal therapies for UTs, which are classified into three categories: medication, diet and supplement therapy, and complementary and alternative medicine (CAM) and other therapies. The effects of medications such as AST-120 and meclofenamate are described. Diet and supplement therapies include plant-based diet, very low-protein diet, probiotics, prebiotics, synbiotics, and nutraceuticals. The research status of Chinese herbal medicine is discussed for CAM and other therapies. This review can provide some treatment recommendations for the reduction of UTs in patients with chronic kidney disease.

APA, Harvard, Vancouver, ISO, and other styles

29

Krishnan, Dharini, Chetan Mehndiratta, and Tanmay Agrawal. "Ketogenic Diet as Medical Nutrition Therapy." Journal of Social Health and Diabetes 7, no. 02 (December 2019): 73–76. http://dx.doi.org/10.1055/s-0039-3402528.

Full text

Abstract:

AbstractMedical nutrition therapy is a therapeutic approach to treat medical conditions and their associated symptoms via using a specifically tailored diet devised under the supervision of a doctor and a registered dietitian or nutrition professional. Ketogenic diet primarily consists of the high amount of fats, a moderate amount of proteins, and very low carbohydrates. It is known to stimulate the metabolic effects of starvation by forcing the body to use primarily fat as a fuel source. Ketogenic diet was developed in the 1920s. Nowadays, it is gaining considerable attention as a potential weight-loss strategy because of the low-carb diet. However, it is being considered for use in several diseases/disorders also because of the beneficial effects on the metabolic health and nervous system. This review revisits the therapeutic potential of ketogenic diets in many pathological conditions and its role as a medical nutrition therapy. It also talks about the ill effects that the keto diet can have in case of self-usage and monitoring.

APA, Harvard, Vancouver, ISO, and other styles

30

Ramalingum, Nelvana, and M. Fawzi Mahomoodally. "The Therapeutic Potential of Medicinal Foods." Advances in Pharmacological Sciences 2014 (2014): 1–18. http://dx.doi.org/10.1155/2014/354264.

Full text

Abstract:

Pharmaceutical and nutritional sciences have recently witnessed a bloom in the scientific literature geared towards the use of food plants for their diversified health benefits and potential clinical applications. Health professionals now recognize that a synergism of drug therapy and nutrition might confer optimum outcomes in the fight against diseases. The prophylactic benefits of food plants are being investigated for potential use as novel medicinal remedies due to the presence of pharmacologically active compounds. Although the availability of scientific data is rapidly growing, there is still a paucity of updated compilation of data and concerns about the rationale of these health-foods still persist in the literature. This paper attempts to congregate the nutritional value, phytochemical composition, traditional uses,in vitroandin vivostudies of 10 common medicinal food plants used against chronic noncommunicable and infectious diseases. Food plants included were based on the criteria that they are consumed as a common food in a typical diet as either fruit or vegetable for their nutritive value but have also other parts which are in common use in folk medicine. The potential challenges of incorporating these medicinal foods in the diet which offers prospective opportunities for future drug development are also discussed.

APA, Harvard, Vancouver, ISO, and other styles

31

Ivanova, Stefka, Liliya Vasileva, Stanislava Ivanova, Lily Peikova, and Danka Obreshkova. "Osteoporosis: Therapeutic Options." Folia Medica 57, no. 3-4 (April 1, 2016): 181–90. http://dx.doi.org/10.1515/folmed-2015-0037.

Full text

Abstract:

Abstract The definition of osteoporosis was originally formulated at a conference of the World Health Organization (WHO) in 1993 as ‘a systemic skeletal disease characterized by decreased bone mass and altered micro-architecture of bone tissue, leading to enhanced bone fragility and risk of fractures’. Osteoporosis is characterized by low bone mineral density (BMD) and loss of the structural and bio-mechanical properties that are required to maintain bone homeostasis. This review aims to address the currently available options in prevention and treatment of osteoporosis. Management of osteoporosis includes non-pharmacological treatment - diet rich of calcium and vitamin D, healthy lifestyle, proper exercise plan, and pharmacological therapy. Combination of non-pharmacological and pharmacological treatment options have to be considered for prevention of osteoporosis and minimization of the risk of fractures. Given the heterogeneity of osteoporosis syndrome and lack of significant number of comparative studies, the choice of a pharmacological agents should be individualized.

APA, Harvard, Vancouver, ISO, and other styles

32

Fasano, Alessio. "Novel Therapeutic/Integrative Approaches for Celiac Disease and Dermatitis Herpetiformis." Clinical and Developmental Immunology 2012 (2012): 1–7. http://dx.doi.org/10.1155/2012/959061.

Full text

Abstract:

Celiac disease (CD) is an immune-mediated enteropathy triggered by the ingestion of gluten in genetically susceptible individuals. Gluten is a protein component in wheat and other cereals like rye and barley. At present, the only available treatment is a strict gluten-free diet. Recent advances have increased our understanding of the molecular basis for this disorder. Last decade has seen new scientific developments in this disease and led to the formulation of new concepts of pathophysiology that offer possible targets for new treatments or interventions integrative to the gluten-free diet.

APA, Harvard, Vancouver, ISO, and other styles

33

Bülow, M., R. Olsson, and O. Ekberg. "Do dysphagic patients with an absent pharyngeal swallow have a shorter survival than dysphagic patients with pharyngeal swallow? prognostic importance of a therapeutic videoradiographic swallowing study (TVSS)." Acta Radiologica 46, no. 2 (April 2005): 126–31. http://dx.doi.org/10.1080/02841850510015965.

Full text

Abstract:

Purpose:To study survival in two groups of dysphagic patients – one group unable to elicit the pharyngeal stage of swallow (APS) and another group with pharyngeal swallow (WPS) – and to compare recommendations regarding nutrition and therapeutic strategies based on the therapeutic swallowing study. Material and Methods: In this retrospective study, the records of dysphagic patients who have undergone a therapeutic videoradiographic swallowing study (TVSS) were reviewed. Forty patients without pharyngeal swallow were matched for age and gender with 40 patients with pharyngeal swallow; altogether 80 patients were included in the study. Survival was registered at 3, 12, and 72 months after the TVSS. Results: In this study, the APS group had a significantly shorter survival time ( P = 0.0030) compared to the WPS group when followed‐up at 12 months. In the APS group, most patients (37.5% (15/40)) died within the 3 months after TVSS. At 72 months, 62.5% (25/40) of the patients in the APS group had died. In the WPS group, 5% (2/40) had died within 3 months and 47.4% (19/40) after 12 months. At 72 months, 52.5% (21/40) of the patients in the WPS group had died. Regarding nutritional and therapeutic recommendations based on TVSS, 34/40 in the APS group were recommended no oral intake. Eighteen naso‐gastric tubes were placed directly after TVSS. The therapeutic strategies recommended were head‐positioning, thermal tactile stimulation, and tongue exercises (in 8 patients). In the WPS group, all patients were recommended oral intake. Diet modification was recommended in 29 patients. The therapeutic strategies recommended were head‐positioning, thermal tactile stimulation, tongue exercises, supraglottic swallow, and effortful swallow (in 24 patients). Conclusion: Patients unable to elicit the pharyngeal stage of swallow had a shorter survival time than patients with pharyngeal swallow, probably due to a more severe underlying disease. Tube feeding was more frequent in the APS group. Fewer therapeutic strategies were recommended compared to the WPS group. In the WPS group, diet modification was frequent. Several patients had different therapeutic strategies. At the end of the study, 8/40 patients (20%) in the APS group had recovered and regained the ability to elicit the pharyngeal stage of swallow. All eight had achieved active swallowing rehabilitation.

APA, Harvard, Vancouver, ISO, and other styles

34

Líška, Dávid, and Jozef Záhumenský. "Conservative possibilities influencing PCOS syndrome – the importance of nutrition." Česká gynekologie 86, no. 5 (October 22, 2021): 343–48. http://dx.doi.org/10.48095/cccg2021343.

Full text

Abstract:

Polycystic ovary syndrome (PCOS) is a common pathological condition in women. Conservative treatment is used in the treatment of polycystic ovary syndrome. Conservative treatment options include increased physical activity and diet. The main aim of the article is to discuss the therapeutic treatment of infl uencing PCOS from a nutritional point of view. PCOS is associated with several comorbidities, including infertility, metabolic syndrome, obesity, impaired glucose tolerance, diabetes mellitus II, and increased cardiovascular risk. Several therapeutic diets can be used in the treatment of PCOS, such as the DASH diet, the low-carbohydrate diet, and a diet based on a low glycemic index. A change in eating habits is associated with improvement in PCOS symptoms. Key words: polycystic ovary syndrome – nutrition – diet – metabolic syndrome

APA, Harvard, Vancouver, ISO, and other styles

35

Archer, Zoë A., and Julian G. Mercer. "Brain responses to obesogenic diets and diet-induced obesity." Proceedings of the Nutrition Society 66, no. 1 (February 2007): 124–30. http://dx.doi.org/10.1017/s0029665107005356.

Full text

Abstract:

Rodent models of diet-induced obesity (DIO) mimic common human obesity more accurately than obese single-gene mutation lines, such as the ob/ob mouse. Sprague-Dawley rats sourced in the UK develop obesity when fed a high-energy diet, but susceptibility to DIO is normally distributed, as might be anticipated for a polygenic trait in an outbred population, in contrast to reports in the literature using ostensibly the same strain of rats sourced in the USA. Nevertheless, the responses of these rats to solid and liquid obesogenic diets are very similar to those reported elsewhere, and this model of DIO has much to commend it as a vehicle for the mechanistic study of susceptibility to DIO, development and reversal of obesity on solid and liquid diets and the response of peripheral and central energy balance systems to the development of obesity and to the obesogenic diets themselves. In general, hypothalamic energy-balance-related systems respond to obesogenic diets and developing obesity with activity changes that appear designed to counter the further development of the obese state. However, these hypothalamic changes are apparently unable to maintain body weight and composition within normal limits, suggesting that attributes of the obesogenic diets either evade the normal regulatory systems and/or engage with reward pathways that override the homeostatic systems. Since diets are a risk factor in the development of obesity, it will be important to establish how obesogenic diets interact with energy-balance pathways and whether there is potential for diets to be manipulated with therapeutic benefit.

APA, Harvard, Vancouver, ISO, and other styles

36

Magrone, Thea, Fatima Perez de Heredia, Emilio Jirillo, Giuseppa Morabito, Ascension Marcos, and Mauro Serafini. "Functional foods and nutraceuticals as therapeutic tools for the treatment of diet-related diseases." Canadian Journal of Physiology and Pharmacology 91, no. 6 (June 2013): 387–96. http://dx.doi.org/10.1139/cjpp-2012-0307.

Full text

Abstract:

In Western societies, the incidence of diet-related diseases is progressively increasing due to greater availability of hypercaloric food and a sedentary lifestyle. Obesity, diabetes, atherosclerosis, and neurodegeneration are major diet-related pathologies that share a common pathogenic denominator of low-grade inflammation. Functional foods and nutraceuticals may represent a novel therapeutic approach to prevent or attenuate diet-related disease in view of their ability to exert anti-inflammatory responses. In particular, activation of intestinal T regulatory cells and homeostatic regulation of the gut microbiota have the potential to reduce low-grade inflammation in diet-related diseases. In this review, clinical applications of polyphenol-rich functional foods and nutraceuticals in postprandial inflammation, obesity, and ageing will be discussed. We have placed special emphasis on polyphenols since they are broadly distributed in plants.

APA, Harvard, Vancouver, ISO, and other styles

37

Power, Susan E., Paul W. O'Toole, Catherine Stanton, R. Paul Ross, and Gerald F. Fitzgerald. "Intestinal microbiota, diet and health." British Journal of Nutrition 111, no. 3 (August 12, 2013): 387–402. http://dx.doi.org/10.1017/s0007114513002560.

Full text

Abstract:

The human intestine is colonised by 1013to 1014micro-organisms, the vast majority of which belong to the phyla Firmicutes and Bacteroidetes. Although highly stable over time, the composition and activities of the microbiota may be influenced by a number of factors including age, diet and antibiotic treatment. Although perturbations in the composition or functions of the microbiota are linked to inflammatory and metabolic disorders (e.g. inflammatory bowel diseases, irritable bowel syndrome and obesity), it is unclear at this point whether these changes are a symptom of the disease or a contributing factor. A better knowledge of the mechanisms through which changes in microbiota composition (dysbiosis) promote disease states is needed to improve our understanding of the causal relationship between the gut microbiota and disease. While evidence of the preventive and therapeutic effects of probiotic strains on diarrhoeal illness and other intestinal conditions is promising, the exact mechanisms of the beneficial effects are not fully understood. Recent studies have raised the question of whether non-viable probiotic strains can confer health benefits on the host by influencing the immune system. As the potential health effect of these non-viable bacteria depends on whether the mechanism of this effect is dependent on viability, future research needs to consider each probiotic strain on a case-by-case basis. The present review provides a comprehensive, updated overview of the human gut microbiota, the factors influencing its composition and the role of probiotics as a therapeutic modality in the treatment and prevention of diseases and/or restoration of human health.

APA, Harvard, Vancouver, ISO, and other styles

38

Głąbska, Dominika, Dominika Guzek, Karolina Gałązka, and Gustaw Lech. "Therapeutic Potential of Proanthocyanidins in Ulcerative Colitis in Remission." Journal of Clinical Medicine 9, no. 3 (March 12, 2020): 771. http://dx.doi.org/10.3390/jcm9030771.

Full text

Abstract:

A number of bioactive components of diet are indicated as potential dietary factors for the management of ulcerative colitis, while the recent study conducted in an animal model revealed that proanthocyanidins from grape seeds exert a broadly positive impact. The aim of the study was to verify the influence of dietary proanthocyanidins intake on the symptoms of ulcerative colitis in remission in human subjects. The study was conducted in a group of 55 participants (19 males, 36 females) in remission of ulcerative colitis confirmed based on both the Mayo Scoring system and Rachmilewitz index. Their habitual dietary intake of proanthocyanidins and intake recalculated per 1000 kcal of diet was assessed and it was verified whether they are associated with symptoms of ulcerative colitis. The energy value of diet and intake of other nutrients were analyzed as potential interfering factors. Participants declaring the presence of mucus in their stool compared with other participants were characterized by higher proanthocyanidins intake (142 vs. 75 mg; p = 0.0441) and intake per 1000 kcal (91 vs. 37 mg/1000 kcal; p = 0.0092), while for no other nutrient such association was stated. Participants declaring constipation compared with other participants were characterized by higher proanthocyanidins intake (214 vs. 82 mg; p = 0.0289) and intake per 1000 kcal (118 vs. 41 mg/1000 kcal; p = 0.0194). Similar association for constipation was observed in the case of energy value of diet and protein intake, but only for proanthocyanidins intake, it was confirmed in the logistic regression model (p = 0.0183; OR = 1.01; 95% CI 1.00–1.02). The positive influence of habitual dietary intake of proanthocyanidins was confirmed in the studied group of patients with ulcerative colitis in remission, as this intake may have increased the production of mucus, which is beneficial for intestinal healing, and may have reduced the frequency of bowel movements. However, further experimental human studies are necessary to confirm the potential influence of proanthocyanidins intake in patients with ulcerative colitis in remission.

APA, Harvard, Vancouver, ISO, and other styles

39

Fung, Eva Lai-wah, Sandy Ka-yin Chang, Karen Kwan-ming Yam, and Phyllis Yin-ping Yau. "Ketogenic Diet as a Therapeutic Option in Super-refractory Status Epilepticus." Pediatrics & Neonatology 56, no. 6 (December 2015): 429–31. http://dx.doi.org/10.1016/j.pedneo.2015.01.010.

Full text

APA, Harvard, Vancouver, ISO, and other styles

40

Barnard, Neal, Anthony R. Scialli, Patricia Bertron, Donna Hurlock, and Kalia Edmonds. "Acceptability of a Therapeutic Low-Fat, Vegan Diet in Premenopausal Women." Journal of Nutrition Education 32, no. 6 (November 2000): 314–19. http://dx.doi.org/10.1016/s0022-3182(00)70590-5.

Full text

APA, Harvard, Vancouver, ISO, and other styles

41

Caio, Giacomo, Rachele Ciccocioppo, Giorgio Zoli, Roberto De Giorgio, and Umberto Volta. "Therapeutic options for coeliac disease: What else beyond gluten-free diet?" Digestive and Liver Disease 52, no. 2 (February 2020): 130–37. http://dx.doi.org/10.1016/j.dld.2019.11.010.

Full text

APA, Harvard, Vancouver, ISO, and other styles

42

Takumi, Akiko, Etsuko Muraki, Yasuyuki Oshima, Yuki Nakano, Junji Shono, Shohei Hoshino, Nobuaki Tsuge, et al. "Therapeutic effects of FRB on diet-induced metabolic disorders in rats." Chemistry and Physics of Lipids 160 (August 2009): S46. http://dx.doi.org/10.1016/j.chemphyslip.2009.06.082.

Full text

APA, Harvard, Vancouver, ISO, and other styles

43

Qu, Changbo, Jaap Keijer, Merel J. W. Adjobo-Hermans, Melissa van de Wal, Tom Schirris, Clara van Karnebeek, Yihang Pan, and Werner J. H. Koopman. "The ketogenic diet as a therapeutic intervention strategy in mitochondrial disease." International Journal of Biochemistry & Cell Biology 138 (September 2021): 106050. http://dx.doi.org/10.1016/j.biocel.2021.106050.

Full text

APA, Harvard, Vancouver, ISO, and other styles

44

Dumitrache (Nicolescu), Cristina-Elena, and Magdalena Mititelu. "Mediterranean diet – from food and lifestyle, to inclusion in therapeutic guidelines." Farmacist.ro 5, no. 202 (2021): 28. http://dx.doi.org/10.26416/farm.202.5.2021.5582.

Full text

APA, Harvard, Vancouver, ISO, and other styles

45

Willis-Mahn, Christine, Rebecca Remillard, and Kathy Tater. "ELISA Testing for Soy Antigens in Dry Dog Foods Used in Dietary Elimination Trials." Journal of the American Animal Hospital Association 50, no. 6 (November 1, 2014): 383–89. http://dx.doi.org/10.5326/jaaha-ms-6063.

Full text

Abstract:

The use of elimination diet trials is necessary in the diagnosis of food allergies and intolerances. The objective of this study was to determine in vitro if four over-the-counter (OTC) dry dog foods carrying a “no soy” claim and seven veterinary therapeutic dry dog foods designed for food elimination trials were suitable for a soybean elimination trial. A 100 g sample of each diet plus one soy positive and one soy negative control diet were submitted for enzyme-linked immunosorbent assay testing to an independent food laboratory. The positive control diet contained >25 ppm soy protein antigens and the negative control contained <2.5 ppm. Three of the four OTC “no soy” claiming diets were positive for soy antigen. Two of the three soy-containing diets had >25 ppm. Three veterinary therapeutic diets had less than the lowest detectable limit of soy protein and four were positive (>2.5 ppm). OTC dog food diets that claim to contain “no soy” may contain high concentrations of soy protein and, therefore, should not be used in soy elimination trials in suspect food allergic dogs. The veterinary therapeutic diet selected for a soy elimination trial needs to be carefully chosen based on diet history.

APA, Harvard, Vancouver, ISO, and other styles

46

Zangara, Megan T., and Christine McDonald. "How diet and the microbiome shape health or contribute to disease: A mini-review of current models and clinical studies." Experimental Biology and Medicine 244, no. 6 (January 31, 2019): 484–93. http://dx.doi.org/10.1177/1535370219826070.

Full text

Abstract:

The human gut microbiome is a complex ecosystem of commensal microorganisms that help to shape host development and health. Host diet directly influences not only the composition, but also the function of the microbiome. In turn, the microbiome impacts the health of the host through production of metabolites from ingested food. Studies of this host-microbe relationship have recently enhanced our understanding of disease development and pathogenesis, and are now being targeted to combat disease symptoms and promote health. For example, dietary shifts to a high-fat, high-sugar “Western diet” drives microbiome dysbiosis in several disease models, which correlates with disease severity. Reintroduction of fiber, increased antioxidants, or decreases in fat and sugar intake helps to restore microbial balance to a healthier composition and often improves disease parameters. Analyses of microbiome-produced metabolites emphasize that understanding how diet shifts the function of the microbiome may be of critical importance for therapeutic targeting of the host-microbe relationship to promote health. In this mini-review, we discuss what is known about the three-way relationship between diet, the microbiome, and disease utilizing animal models, as well as the findings from human clinical trials of dietary therapeutics. Impact statement The studies reviewed in this article combine diet in the context of disease progression or treatment with analysis of the microbiome. First, we present findings on how diet manipulation impacts the microbiome and disease pathogenesis in a broad variety of rodent models of disease. Then, we describe results from clinical trials that are using diet therapies to attempt to shift the microbiome and treat disease symptoms. Finally, we discuss what these studies have taught us about the influence of the microbiome of disease and health states and highlight the evidence suggesting that dietary modulation of the microbiome is an emerging therapeutic option for a variety of different diseases.

APA, Harvard, Vancouver, ISO, and other styles

47

Howard, Elizabeth J., Tony K. T. Lam, and Frank A. Duca. "The Gut Microbiome: Connecting Diet, Glucose Homeostasis, and Disease." Annual Review of Medicine 73, no. 1 (January 27, 2022): 469–81. http://dx.doi.org/10.1146/annurev-med-042220-012821.

Full text

Abstract:

Type 2 diabetes rates continue to rise unabated, underscoring the need to better understand the etiology and potential therapeutic options available for this disease. The gut microbiome plays a role in glucose homeostasis, and diabetes is associated with alterations in the gut microbiome. Given that consumption of a Western diet is associated with increased metabolic disease, and that a Western diet alters the gut microbiome, it is plausible that changes in the gut microbiota mediate the dysregulation in glucose homeostasis. In this review, we highlight a few of the most significant mechanisms by which the gut microbiome can influence glucose regulation, including changes in gut permeability, gut–brain signaling, and production of bacteria-derived metabolites like short-chain fatty acids and bile acids. A better understanding of these pathways could lead to the development of novel therapeutics to target the gut microbiome in order to restore glucose homeostasis in metabolic disease.

APA, Harvard, Vancouver, ISO, and other styles

48

Durrani, Samir, Jane Irvine, and Robert P. Nolan. "Psychosocial Determinants of Health Behaviour Change in an E-Counseling Intervention for Hypertension." International Journal of Hypertension 2012 (2012): 1–5. http://dx.doi.org/10.1155/2012/191789.

Full text

Abstract:

We evaluated the influence of psychological stress and depression on motivation to adhere to recommended guidelines for exercise and diet. This study was conducted within a larger e-counseling trial. Subjects diagnosed with hypertension (n=387, age = 44–74 years, 59% female) completed assessments at baseline and within 2 weeks after a 4-month intervention period. Outcomes included mean level of readiness to change diet and exercise and symptoms of depression and stress. Per protocol analysis defined e-counseling support as follows: ≥8 e-mails = therapeutic dose, 1–7 e-mails = subtherapeutic dose, and 0 e-mails = Controls. Baseline adjusted symptoms of depression and stress were inversely correlated with improvement in exercise (partialR=-.14,P=.01, and partialR=-.17,P=.01, resp.) but not diet or e-counseling. Subjects who received a therapeutic dose of e-counseling demonstrated greater readiness for diet adherence versus Controls (P=.02). Similarly, subjects receiving a therapeutic level of e-counseling demonstrated significantly greater readiness for exercise adherence versus Controls (P=.04). In sum, e-counseling is associated with improved motivation to adhere to exercise and diet among patients with hypertension, independent of symptoms of psychological stress and depression.

APA, Harvard, Vancouver, ISO, and other styles

49

Abbas, Sabiha, Mian Kamran Sharif, Muhammad Sibt-e-Abbas, Tadesse Fikre Teferra, Muhammad Tauseef Sultan, and Muhammad Junaid Anwar. "Nutritional and Therapeutic Potential of Sesame Seeds." Journal of Food Quality 2022 (April 22, 2022): 1–9. http://dx.doi.org/10.1155/2022/6163753.

Full text

Abstract:

The major issue of the current era is an unbalanced and poor diet like unhealthy fast foods, the main cause of various diseases. Most nutraceutical and pharma industries formulating the medicines from artificial sources are expensive and have several side effects. However, scientists are making efforts to find out the natural sources of medicines for the betterment of human health and treatment of diseases. Simultaneously, the worldwide preferences have shifted from artificial to natural resources and unconventional crops (i.e., oilseeds as protein source) and foods are becoming part of regular diet in most of the community, nutraceutical, and pharma industries. Sesame (SesamumindicumL.) is one of the unconventional crops providing multiple benefits due to its special bioactive components, such as sesamin, sesaminol, and gamma-tocopherol, and fatty acids composition like unsaturated fatty acids (i.e., oleic acid, linoleic acid, stearidonic acid, palmitoleic acid, and traces of linolenic acid). Sesame seed oil supplementation not only improves the quality of snack frying oils but also plays a key role in the formation of good quality healthy snack foods. Moreover, its seeds and oil play imperative role in the formulation of medicines utilized for different ailments. The current review highlights the importance and utilization of sesame seed and oil in pharmaceuticals, nutraceuticals, and food (especially snacks) industries.

APA, Harvard, Vancouver, ISO, and other styles

50

Tuokkola, J., M. Kaila, C. Kronberg-Kippilä, H. K. Sinkko, T. Klaukka, P. Pietinen, R. Veijola, et al. "Cow's milk allergy in children: adherence to a therapeutic elimination diet and reintroduction of milk into the diet." European Journal of Clinical Nutrition 64, no. 10 (August 4, 2010): 1080–85. http://dx.doi.org/10.1038/ejcn.2010.132.

Full text

APA, Harvard, Vancouver, ISO, and other styles

Journal articles on the topic 'Therapeutic diet'

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles