Relevant bibliographies by topics / Therapeutic ends

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Therapeutic ends'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Therapeutic ends"

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Close, Henry. "Book Review: Narrative Means to Therapeutic Ends." Journal of Pastoral Care 46, no. 1 (1992): 84. http://dx.doi.org/10.1177/002234099204600115.

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Akinyela, Makungu M. "Testimony of Hope: African Centered Praxis for Therapeutic Ends." Journal of Systemic Therapies 24, no. 1 (2005): 5–18. http://dx.doi.org/10.1521/jsyt.2005.24.1.5.

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Floh�, Leopold. "Superoxide dismutase for therapeutic use: Clinical experience, dead ends and hopes." Molecular and Cellular Biochemistry 84, no. 2 (1988): 123–31. http://dx.doi.org/10.1007/bf00421046.

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Aziz, Miriam, and Murray Earle. "Legalising Cannabis for Therapeutic Use: A Comparative Assessment." Medical Law International 3, no. 4 (1998): 273–85. http://dx.doi.org/10.1177/096853329800300402.

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Therapeutic use of cannabis as a palliative treatment for certain medical conditions has been hotly debated in Britain in recent months. The weight of public opinion is behind a reform of the law; reform would legalise the possession and use of cannabis for therapeutic ends. This paper will endorse that argument by concentrating on a central contradiction within the law: while the law meticulously categorises the drugs which it regulates and prohibits, it is unwilling divide users of particular drugs into recreational and therapeutic groupings. This paper argues that the traditional arguments against making such a change in the law are unable to hold water, particularly when viewed in comparison with developments in Germany. We will also propose that the medical practitioner fulfil an existing traditional function as gate keeper in order to ensure supply and quality are both kept to a certain minimum standard. This policy will serve the ends of patients for whom this treatment is effective and cheap and which is justified in the public interest.
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Relitti, Nicola, Akella P. Saraswati, Stefano Federico, et al. "Telomerase-based Cancer Therapeutics: A Review on their Clinical Trials." Current Topics in Medicinal Chemistry 20, no. 6 (2020): 433–57. http://dx.doi.org/10.2174/1568026620666200102104930.

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Telomeres are protective chromosomal ends that shield the chromosomes from DNA damage, exonucleolytic degradation, recombination, and end-to-end fusion. Telomerase is a ribonucleoprotein that adds TTAGGG tandem repeats to the telomeric ends. It has been observed that 85 to 90% of human tumors express high levels of telomerase, playing a crucial role in the development of cancers. Interestingly, the telomerase activity is generally absent in normal somatic cells. This selective telomerase expression has driven scientists to develop novel anti-cancer therapeutics with high specificity and potency. Several advancements have been made in this area, which is reflected by the enormous success of the anticancer agent Imetelstat. Since the discovery of Imetelstat, several research groups have contributed to enrich the therapeutic arsenal against cancer. Such contributions include the application of new classes of small molecules, peptides, and hTERT-based immunotherapeutic agents (p540, GV1001, GRNVAC1 or combinations of these such as Vx-001). Many of these therapeutic tools are under different stages of clinical trials and have shown promising outcomes. In this review, we highlight the current status of telomerase-based cancer therapeutics and the outcome of these investigations.
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Khalipaev, M. G., I. M. Azizov, and Z. M. Zukhrabova. "ETIOPATHOGENESIS, DIAGNOSIS AND THERAPEUTIC AND PREVENTIVE MEASURES OF POSTPARTUM CATARRHAL-PURULENT ENDOMETRITIS OF COWS." Scientific Notes Kazan Bauman State Academy of Veterinary Medicine 245, no. 1 (2021): 204–10. http://dx.doi.org/10.31588/2413-4201-1883-245-1-204-210.

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The article presents the results of studies on the etiopathogenesis, clinical signs of postpartum catarrhal-purulent endometritis in cows, pathomorphological and histostructural changes in the geni-tals at the beginning of the disease and the results of treatment with intrauterine use of non-antibiotic, foaming and iodine-containing liquid drug "Metrasil". We believe that the restoration of the body mucosa and uterine horns as a result of treatment is difficult and long-lasting and ends by 30-35 days after the start of therapy and ends simultane-ously with positive processes and in the macroscopic picture of the organ.
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Brorson, Kurt, and Audrey Y. Jia. "Therapeutic monoclonal antibodies and consistent ends: terminal heterogeneity, detection, and impact on quality." Current Opinion in Biotechnology 30 (December 2014): 140–46. http://dx.doi.org/10.1016/j.copbio.2014.06.012.

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Landreth, Gary. "The Immunology of Alzheimer’s Disease: Prospects Towards Harnessing Disease Mechanisms for Therapeutic Ends." Journal of Neuroimmune Pharmacology 2, no. 2 (2007): 131–33. http://dx.doi.org/10.1007/s11481-007-9067-1.

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Carrasco, Patricia, Iratxe Zuazo-Gaztelu, and Oriol Casanovas. "Sprouting strategies and dead ends in anti-angiogenic targeting of NETs." Journal of Molecular Endocrinology 59, no. 1 (2017): R77—R91. http://dx.doi.org/10.1530/jme-17-0029.

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Neuroendocrine tumors (NETs) are a heterogeneous group of neoplasms that arise from cells of the neuroendocrine system. NETs are characterized by being highly vascularized tumors that produce large amounts of proangiogenic factors. Due to their complexity and heterogeneity, progress in the development of successful therapeutic approaches has been limited. For instance, standard chemotherapy-based therapies have proven to be poorly selective for tumor cells and toxic for normal tissues. Considering the urge to develop an efficient therapy to treat NET patients, vascular targeting has been proposed as a new approach to block tumor growth. This review provides an update of the mechanisms regulating different components of vessels and their contribution to tumor progression in order to develop new therapeutic drugs. Following the description of classical anti-angiogenic therapies that target VEGF pathway, new angiogenic targets such as PDGFs, EGFs, FGFs and semaphorins are further explored. Based on recent research in the field, the combination of therapies that target multiple and different components of vessel formation would be the best approach to specifically target NETs and inhibit tumor growth.
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Rutten, Eric Andreas, and Christophe Badie. "Radiation Biomarkers: Silver Bullet, or Wild Goose Chase?" Journal of Personalized Medicine 11, no. 7 (2021): 603. http://dx.doi.org/10.3390/jpm11070603.

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Dissertations / Theses on the topic "Therapeutic ends"

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Vassalos, Tony. "End organ effects of paediatric cardiopulmonary bypass." Thesis, University of Glasgow, 2011. http://theses.gla.ac.uk/2385/.

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Despite the scientific, technological and surgical improvements of the past 50 years organ dysfunction following elective paediatric cardiac surgery utilising cardiopulmonary bypass continues to account for increased complications, often leading to a protracted course in hospital with a longer stay in intensive care and the potential for irreversible organ damage long term. Furthermore, paediatric cardiac surgeons are routinely undertaking more complex operations with a shift from palliation to early correction. This has resulted in younger children being subjected to longer periods on the bypass machine with increased effects on vital organs. This thesis describes two clinical studies designed to further assess and characterise peri-operative cardiac, renal and pulmonary function in children undergoing elective cardiac repair at a tertiary referral centre in Scotland, UK. In the first instance a prospective, observational study was undertaken in forty-five children to examine the use of tissue Doppler imaging in the assessment of peri-operative cardiac function, its relationship to myocardial injury and clinical outcome. Tissue Doppler parameters were obtained using a Vivid 7 ultrasound scanner with a 7-MHz probe pre-operatively, on admission to paediatric intensive care and on day one. Myocardial injury was assessed using Troponin-I on the first post-operative day by a commercially available chemiluminescent immunoassay. In twenty children within this group peri-operative renal function was also investigated using standard estimates of glomerular filtration rate, namely creatinine clearance measured by the kinetic Jaffe method during the first and second twelve hour post-operative periods, in comparison to serum creatinine and the novel biomarker cystatin C. Routine plasma retained pre-operatively and on days 0, 1, 2 and 3 post-operatively was used to measure serum cystatin C and creatinine using a particle-enhanced nephelometric immunoassay and the Roche Creatinine Plus enzymatic assay respectively. The association between cystatin C and recorded perfusion parameters including bypass duration, pump flow, haematocrit, oxygen delivery and Troponin-I was investigated. Peri-operative pulmonary function was evaluated through a phase IV, randomised, double-blind, placebo controlled trial. In total, twenty four children were randomised to receive oral sildenafil or equivalent volume placebo four times the day before surgery. Blood samples were collected peri-operatively to measure serum cyclic guanosine monophosphate with a commercially available competitive enzyme immunoassay. Haemodynamic data and echocardiography were acquired at two and twenty four hours post-operatively including pulmonary vascular resistance index and bi-ventricular contractility. Post-operative oxygenation was also determined at the same time by oxygen delivery and oxygenation index. In Chapter 2, peri-operative cardiac function as assessed by tissue Doppler imaging was examined. The results of this study demonstrated that pre-operatively, bi-ventricular systolic function in the study group was reduced compared with normal controls, displaying a significant step-wise decrease with increasing complexity of lesion. This picture persisted post-operatively predominantly in the right ventricle and was significantly associated with the extent of myocardial injury. Impaired peri-operative left ventricular function correlated with clinical outcomes. In Chapter 3, peri-operative renal function as assessed by cystatin C and its association with parameters of perfusion was examined. The results of this study demonstrated that in comparison to serum creatinine, cystatin C had a superior correlation with glomerular filtration rate in the early post-operative period. An elevated level of this biomarker was significantly associated with bypass duration, minimum pump flow and post-operative myocardial injury. Haematocrit was not directly linked to renal dysfunction in this study although evidence of a critical dysoxic threshold within the kidney was suggested indirectly through oxygen delivery calculations. In Chapter 4, peri-operative pulmonary function and vascular reactivity in association with the pre-operative administration of oral sildenafil (0.5mg/kg, six hourly) was examined. The results of this trial demonstrated that compared to placebo, pre-operative sildenafil resulted in modest elevations of serum cyclic guanosine monophosphate, limited effects on pulmonary vascular resistance index, significant reductions in peri-operative bi-ventricular contractility, significant reductions in post-operative oxygen delivery and a trend for increasing ventilatory support. In summary, the current thesis has demonstrated that in children undergoing corrective cardiac surgery peri-operative bi-ventricular function can be accurately assessed by tissue Doppler imaging which to date has had limited use in this patient group. With regards to renal function, cystatin C was shown to be a better estimate of glomerular filtration rate and a more sensitive marker of early renal dysfunction in children after surgery. Furthermore, cystatin C identified a transient post-operative renal impairment, the magnitude of which was associated with duration of bypass, pump flow and myocardial injury. In relation to pulmonary function, this research identified that pre-operative administration of oral sildenafil to children undergoing cardiac surgery produced limited effects on pulmonary vascular resistance but was associated with reduced ventricular contractility and post-operative oxygenation raising significant concerns over its routine clinical use.
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Broek, Stanislaus Antonius Jacobus van den. "Angiotensin-converting enzyme inhibitors in heart failure clinical end points to assess therapeutic efficacy /." [S.l. : [Groningen : s.n.] ; University Library Groningen] [Host], 1994. http://irs.ub.rug.nl/ppn/293017212.

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Farmer, David George Stephen. "The receptor for advanced glycation end-products in pulmonary hypertension." Thesis, University of Glasgow, 2012. http://theses.gla.ac.uk/3730/.

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The receptor for advanced glycation endproducts (RAGE) is a 35-kDa polypeptide of the immunogloblin superfamily that has been implicated as a mediator of both acute and chronic vascular inflammation. RAGE has also recently been implicated in the pathology of pulmonary hypertension (PH): a rare, progressive disease of the small pulmonary arteries characterised by pulmonary vascular remodelling, thrombosis, vasoconstriction and increased pulmonary vascular resistance. A ligand for RAGE, the calcium binding protein MTS1/S100A4, is expressed in occlusive vascular lesions of patients with advanced PH. MTS1/S100A4 is upregulated and secreted by pulmonary arterial smooth muscle cells (PASMCs) in vitro on activation of the 5HT1b receptor and 5HT transporter (5HTT). Additionally, the proliferative effect of 5HT on these cells, which is mediated by 5HT1b and 5HTT, may be inhibited by antagonism of RAGE or reduced bioavailability of MTS1/S100A4. These data suggest that MTS1/S100A4, through its action at RAGE, is a key mediator of 5HT-induced hPASMC proliferation. Transgenic mice overexpressing MTS1/S100A4 are observed to develop obliterative pulmonary vascular disease and possess increased right ventricular pressure at baseline and after hypoxia when compared to wildtype mice (WT). These increases occur in the absence of an increase in pulmonary vascular remodelling suggesting that MTS1/S100A4 overexpression is associated with some other structural or functional change in the pulmonary circulation. We sought to further our understanding of the role of RAGE in pulmonary hypertension through treatment with a small molecule inhibitor of monocyte chemoattractant protein 1(MCP-1), a marker of downstream of RAGE rage activation; through further characterisation of the MTS1/S100A4 mouse in a chronic hypoxic model of PAH; and through treatment with soluble RAGE (sRAGE) to reduce RAGE ligand bioavailability in vivo. In each case systolic right ventricular pressure (sRVP), right ventricular hypertrophy (RVH) and pulmonary vascular remodelling were measured in normoxic conditions or after a two week chronic hypoxia challenge to induce PH. These in vivo experiments were supplemented with functional studies in isolated intrapulmonary arteries to assess vascular reactivity and vascular elastance as well as studies of pulmonary fibroblast proliferation in vitro. Treatment with the MCP-1 synthesis inhibitor Bindarit produced no detectable effects upon the pulmonary response of mice to chronic hypoxia, though this study may have been hampered by difficulties with the methylcellulose vehicle. MCP-1 produced no degree of proliferation in pulmonary fibroblasts and neither augmented nor inhibited proliferation induced by 5HT. We found little evidence for the exacerbation of PH in MTS1/S100A4 mice in normoxia, hypoxia or after 4 weeks of normoxic recovery. Mean RVP was elevated above that in WT mice exposed to hypoxia. However, MTS1/S100A4 mice appeared protected against hypoxia-induced vascular remodelling and decreases in vascular elastance. No other significant differences in sRVP, RVH or remodelling were observed between strains. Vessels isolated from MTS1/S100A4 mice tended towards an enhanced contractile response to 5HT in normoxia compared with vessels in WT mice but were also more sensitive to the nitric oxide donor SNP. These differences in vasoreactivity were largely abolished by exposure to hypoxia. Treatment with soluble RAGE (sRAGE) to reduce RAGE ligand bioavailability produced a significant reduction in sRVP after hypoxia in comparison to vehicle-dosed mice -possibly associated with the prevention of a hypoxia-induced decrease in proximal vascular elastance. However, no benefit upon the development of remodelling or the extent of RVH was observed. Vessels isolated from mice treated with sRAGE and challenged with hypoxia showed a marked increase in contractility. Further work demonstrated that sRAGE produces a small, slowly developing contraction in isolated vessels and that the maximal force of contraction to 5HT was markedly augmented in the presence of sRAGE. Finally, treatment with sRAGE did not inhibit fibroblast proliferation in vitro as induced by 5HT but was observed to cause a small degree of proliferation alone and to augment hypoxia-induced proliferation. In summary, we have reported a number of seemingly contradictory findings associated with RAGE in pulmonary hypertension. Treatment with sRAGE produced a beneficial reduction in hypoxia-induced PH associated with protection against decreased proximal vascular elastance but produced no change in hypoxia-induced RVH or remodelling as well as greatly increasing vascular contractility. MTS1/S100A4 mice show some evidence of deleterious changes to the pulmonary circulation, but these may be offset by beneficial compensatory mechanisms such as increased sensitivity to nitric oxide and protection against vascular remodelling. MTS1/S100A4 stimulates smooth muscle cell proliferation suggesting that it may involved pulmonary vascular remodelling. However, inhibition of RAGE was observed to enhance fibroblast proliferation in response to hypoxia here. Fibroblasts are important regulators of SMC proliferation in vivo. These findings therefore suggest a more complicated relationship between RAGE, its ligands and the remodelling process. Since both MTS1/S100A4 overexpression and sRAGE treatment in vivo produced findings which are difficult to reconcile using the currently employed techniques, it is clear that furthering our understanding of RAGE will require study with greater focus upon the interaction of different cell types in the pulmonary vasculature and the manner in which the disturbance of this may lead to alterations in the physical and physiological properties of the pulmonary circulation.
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Yi, Xiaoqin. "Total ginsenosides of Asian ginseng increase coronary artery perfusion flow of the ischemia-reperfusion injury rat heart in Langendorff system through activation of Akt-eNOS signaling and cardiac energy-associate protein expression." HKBU Institutional Repository, 2010. http://repository.hkbu.edu.hk/etd_ra/1195.

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Touati, Sabeur. "Obésité, risque athérogène et effet thérapeutique direct de l’exercice physique : étude sur la contribution des voies signalétiques Akt/eNOS et NADPH oxydase pour expliquer les mécanismes vasculo-protecteurs de l’exercice physique chez le rat rendu obèse par une alimentation enrichie en graisse." Thesis, Avignon, 2010. http://www.theses.fr/2010AVIG0704/document.

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La prévalence de l’obésité est en constante augmentation dans les pays occidentaux, en raison d’une sédentarisation accompagnée d’une alimentation malsaine. L’obésité est souvent associée à une dysfonction endothéliale et à un risque athérogène élevé. Plusieurs observations cliniques ont montré que la modification du mode de vie, incluant la pratique régulière d’une activité physique et l’adoption d’un mode alimentaire sain, représente une stratégie efficace pour combattre l’obésité et ses complications cardiovasculaires. Cependant, de nombreux mécanismes précisant les effets thérapeutiques directs de l’exercice physique sur le risque athérogène lié à l’obésité sont encore largement inconnus. Le but principal de ce travail a donc été d’identifier, en utilisant un modèle de rat rendu obèse par un régime enrichi en graisse, les mécanismes athéro-protecteurs de l’exercice physique seul et/ou associé à une modification du régime alimentaire (du régime riche en graisse au régime standard). Nos résultats montrent que l’exercice physique, indépendamment de la diète utilisée, corrige la dysfonction endothéliale installée au cours de l’obésité. Cet effet bénéfique a été associé à une diminution du stress oxydatif au niveau vasculaire. En effet, nos résultats indiquent que l’exercice diminue l’activité de la NADPH oxydase au niveau aortique. De plus, nous montrons pour la première fois que l’exercice physique seul, indépendamment de la diète utilisée, est capable de moduler la translocation de la sous-unité de la NADPH oxydase p47phox (principal acteur dans l’activation de ce complexe enzymatique) vers la membrane. Nos résultats indiquent également que l’exercice physique, avec ou sans modification du régime, améliore la voie Akt/eNOS phosphorylée, suggérant une augmentation de la production du NO. Ainsi, l’exercice physique, même sans l’associer à un changement du mode alimentaire, peut être considéré comme une stratégie non-pharmacologique efficace pour le traitement du risque athérogène généré par l’obésité<br>The prevalence of obesity is increasing at an alarming rate in the western countries. It has been attributed to sedentariness and abundance of unhealthy food. Obesity is often associated with endothelial dysfunction and a high atherogenic risk. Several clinical investigations have reported that life style modification included physical exercise and the adoption of healthydiet was an efficient strategy to combat cardiovascular complications linked to obesity. However, numerous mechanisms by which exercise exerts the direct therapeutic effect on atherogenic risk linked to obesity are still unknown. Using the experimental model of high fat diet-induced obesity rat, the general aim of this study, was to identify the possible molecularmechanisms through which exercise with or without diet modification (high fat to standard diet) exerts an antiatherogenic action. Our results show that exercise independently of diet used, corrected the endothelial dysfunction induced by obesity. This benefit effect was associated with the decreased vascular oxidative stress. In effect, our results show that exercise alone was able to decrease NADPH oxidase activity in aortic tissue. Furthermore, we show for the first time that exercise, independently diet used, was able to modulate the translocation of p47phox subunit to membrane (which plays a pivotal role in NADPH oxidase activation). Ours results show also, that exercise with or without diet modification improves the Akt/eNOS phosphorylation pathway, suggesting that exercise increases NO production. In summary, exercise training even without diet modification, may be a non-pharmacological strategy treatment for atherogenic risk linked to obesity
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Sirois, Cherilyn M. "Nucleic Acid Sensing by the Immune System: Roles For the Receptor For Advanced Glycation End Products (RAGE) and Intracellular Receptor Proteins: A Dissertation." eScholarship@UMMS, 2011. https://escholarship.umassmed.edu/gsbs_diss/551.

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As humans, we inhabit an environment shared with many microorganisms, some of which are harmless or beneficial, and others which represent a threat to our health. A complex network of organs, cells and their protein products form our bodies’ immune system, tasked with detecting these potentially harmful agents and eliminating them. This same system also serves to detect changes in the healthy balance of normal functions in the body, and for repairing tissue damage caused by injury. Immune recognition of nucleic acids, DNA and RNA, is one way that the body detects invading pathogens and initiates tissue repair. A number of specialized receptor proteins have evolved to distinguish nucleic acids that represent “threats” from those involved in normal physiology. These proteins include members of the Toll-like receptor family and diverse types of cytosolic proteins, all of which reside within the confines of the cell. Few proteins on the cell surface have been clearly characterized to interact with nucleic acids in the extracellular environment. In this dissertation, I present collaborative work that identifies the receptor for advanced glycation end products (RAGE) as a cell surface receptor for nucleic acids and positions it as an important modulator of immune responses. Molecular dimers of RAGE interact with the sugar-phosphate backbones of nucleic acid ligands, allowing this receptor to recognize a variety of DNA and RNA molecules regardless of their nucleotide sequence. Expression of RAGE on cells promotes uptake of DNA and enhances subsequent responses that are dependent on the nucleic acid sensor Toll-like receptor 9. When mice deficient in RAGE are exposed to DNA in the lung, the predominant site of RAGE expression, they do not mount a typical early inflammatory response, suggesting that RAGE is important in generating immune responses to DNA in mammalian organisms. Further evidence suggests that RAGE interacts preferentially with multimolecular complexes that contain nucleic acids, and that these complexes may induce clustering of receptor dimers into larger multimeric structures. Taken together, the data reported here identify RAGE as an important cell surface receptor protein for nucleic acids, which is capable of modulating the intensity of immune responses to DNA and RNA. Understanding of and intervention in this recognition pathway hold therapeutic promise for diseases characterized by excessive responses to self nucleic acids, such as systemic lupus erythematosus, and for the pathology caused by chronic inflammatory responses to self and foreign nucleic acids.
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Brown, Sarah-Jane. "Autonomy, the law, and ante-mortem interventions to facilitate organ donation." Thesis, University of Manchester, 2018. https://www.research.manchester.ac.uk/portal/en/theses/autonomy-the-law-and-antemortem-interventions-to-facilitate-organ-donation(1d8877ef-fa3c-4639-b2ec-6eacb923fe7a).html.

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Over the last few years, policies have been introduced in the UK which aim to improve organ transplantation rates by changing the way that potential organ donors are treated before death. Patients incapacitated due to catastrophic brain injury may now undergo ante-mortem donor optimisation procedures to facilitate deceased organ donation. As I identify in this thesis, the most significant ethical and legal problem with these policies is that they are not based on what the patient would have chosen for themselves in the specific circumstances. The policies identify and treat patients meeting certain clinical criteria as a group rather than the individuals, with their own viewpoints, that the law on best interests requires. They equate registration on the Organ Donation Register with ante-mortem donor optimisation procedures being in their best interests, despite registrants having neither been informed about nor given consent to ante-mortem interventions. The overarching claim I make in this thesis is that a system of specific advance consent is needed to provide a clear and unequivocal legal justification for ante-mortem donor optimisation procedures. The ethical foundation for this claim is autonomy, and this is the central theme running through all six chapters. I argue that autonomy should be incorporated into donor optimisation policy to promote the dignity and integrity of potential organ donors and to safeguard trust in the organ donation programme. I argue that a system of specific advance consent is needed as part of the duty of care owed to registrants on the Organ Donor Register and to facilitate the determination of the best interests of the potential organ donor. I argue that the state has not established the necessity of the current policy of non-consensual donor optimisation procedures and that they are under an ethical and legal obligation to introduce an autonomy-based framework for ante-mortem interventions to facilitate organ donation.
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Zoerner, Frank. "Novel Interventions in Cardiac Arrest : Targeted Temperature Management, Methylene Blue, S-PBN, Amiodarone, Milrinone and Esmolol, Endothelin and Nitric Oxide In Porcine Resuscitation Models." Doctoral thesis, Uppsala universitet, Anestesiologi och intensivvård, 2015. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-236312.

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It is a major clinical problem that survival rates after out-of-hospital cardiac arrest have not markedly improved during the last decades, despite extensive research and the introduction of new interventions. However, recent studies have demonstrated promising treatments such as targeted temperature management (TTM) and methylene blue (MB). In our first study, we investigated the effect of MB administered during experi-mental cardiopulmonary resuscitation (CPR) in the setting of postponed hypother-mia in piglets. We set out to study if MB could compensate for a delay to establish targeted TTM. The study demonstrated that MB more than compensated for 30 min delay in induction of TTM. The effect of MB added to that of TTM. The second study examined the effects of TTM and S-PBN on the endothelin system and nitric oxide synthases (NOS) after prolonged CA in a porcine CPR mod-el. The study was designed to understand the cardioprotective mechanism of S-PBN and TTM by their influence on the endothelin system and NOS regulation. We veri-fied for the first time, that these two cardioprotective postresuscitative interventions activate endothelin-1 and its receptors concomitantly with eNOS and nNOS in the myocardium. We concluded that nitric oxide and endothelin pathways are implicated in the postresuscitative cardioprotective effects of TTM. The third study compared survival and hemodynamic effects of low-dose amio-darone and vasopressin to vasopressin in a porcine hypovolemic CA model. The study was designed to evaluate whether resuscitation with amiodarone and vasopressin compared to vasopressin alone would have an impact on resuscitation success, survival, and hemodynamic parameters after hemorrhagic CA. We found that combined resuscitation with amiodarone and vasopressin after hemorrhagic circulatory arrest resulted in greater 3-hour survival, better preserved hemodynamic parameters and smaller myocardial injury compared to resuscitation with vasopressin only. In our fourth study we planned to compare hemodynamic parameters between the treatment group (milrinone, esmolol and vasopressin; MEV) and control group (vasopressin only) during resuscitation from prolonged cardiac arrest in piglets. The study was designed to demonstrate if MEV treatment improved hemodynamics or cardiac damage compared to controls. We demonstrated that MEV treatment reduced cardiac injury compared with vasopressin alone.
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Drouin, Érika Véronique. "L'utilisation des cellules souches embryonnaires à des fins thérapeutiques." Thèse, 2003. http://hdl.handle.net/1866/2381.

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La découverte des cellules souches embryonnaires et de leur immense potentiel thérapeutique a fait naître de grands espoirs. De nouvelles thérapies révolutionnaires pour traiter certaines des maladies les plus graves dont souffre l'humanité sont désormais envisageables. Le traitement de la vie à son stade le plus précoce est mis en cause. Le statut juridique reconnu au foetus et à l'embryon humain a des répercussions directes sur le domaine de la recherche et sur leur utilisation à des fins thérapeutiques. Nous avons examiné l'état du droit canadien quant au statut juridique du foetus et de l'embryon. De cette étude, nous avons constaté l'incertitude qui prévaut au Canada quant à leur statut. Par la suite, nous avons étudié les différentes normes canadiennes établies pour encadrer l'utilisation des cellules souches embryonnaires à des fins thérapeutiques et nous les avons analysées et comparées pour faire ressortir leurs similitudes et leurs différences. II est ressorti de notre analyse que les textes canadiens se rejoignent généralement sur l'essentiel et qu'il y a eu peu de changements de 1993 à aujourd'hui, en regard des activités de recherche interdites au Canada. Puis, nous avons examiné les systèmes normatifs applicables à ces recherches à l'étranger, soit aux États-Unis et en GrandeBretagne. Nous avons effectué une analyse comparative des trois systèmes normatifs étudiés, en évaluant différents paramètres communs à ces systèmes. Il est ressorti de cette analyse, que la Grande-Bretagne est le pays le plus libéral relativement à ces domaines de recherche, que les États-Unis sont les plus conservateurs sur ces questions et que le Canada se situe entre les deux.<br>The embryonic stem cells discovery and the immense therapeutic potential glven to them has created big hopes in the world of today. The appearance of new revolutionary therapies to treat sorne of the most serious known diseases are now conceivable. However, the treatment of life to its earliest stage is questionned. The legal status recognized to the foetus and the embryo has, in fact, a direct effect to the research area and industry as weil as to its therapeutic use. Therefore, we have examined and studied the CUITent canadian law with respect to the legal status of the foetus and embryo. Following this study, we have noticed the uncertainty that prevails in Canada concerning the said legal status. Afierwards, we have examined ail the different canadian norms and regulations already established regarding the use of embryonic stem cells for therapeutic ends. We also did the comparaison between those norms and regulations so as to see their differences and similarities. It appears from our analysis that ail the canadian litterature generally treat the subject in the same way and that there have been few changes from 1993 up until now with respect to the forbidden researchs activities in Canada. We also have analysed the foreign law standards and regulations in United States and Great Britain concerning those forbidden researchs activities. We did the exercise of comparing the state of the law in these three countries with different parameters. It emerges from that that Great Britain is the most liberal country, United States being the most conservative and Canada being in between them.<br>"Mémoire présenté à la Faculté des études supérieures en vue de l'obtention du grade de Maîtrise en droit (L.L.M.) Option recherche"
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Smith, Kylie Sheree. "Vitamins E and C in patients with end-stage renal disease undergoing hemodialysis." Thesis, 2002. http://hdl.handle.net/1957/27051.

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Patients with end-stage renal disease undergoing hemodialysis have a high incidence of oxidative stress-related diseases. This study evaluated oxidative stress and inflammatory markers in patients undergoing hemodialysis before and during vitamin E supplementation. Blood samples were obtained before and after dialysis during two separate dialysis sessions to establish baseline measurements. For the next two months, subjects consumed 400 IU RRR-α-tocopherol daily. At one month and two months of supplementation, blood samples were also obtained before and after dialysis. Circulating concentrations of α- and γ-tocopherols and their metabolites (carboxyethyl-hydroxychromans, α- and γ-CEHCs), vitamin C, and uric acid were determined by HPLC with electrochemical detection. C-reactive protein (CRP), tumor necrosis factor-α (TNF-α), and interleukin-6 (IL-6) were measured using standard clinical assays. F₂-isoprostanes were evaluated using an enzyme immunoassay. Dietary vitamins E and C were assessed using two 24-hour recalls. In response to vitamin E supplementation, plasma α-tocopherol concentrations increased from 18 ± 1.7 μM to 31 ± 5.4 μM (p<0.0001), while γ-tocopherol concentrations decreased from 2.8 ±1.0 μM to 1.7 ± 0.6 μM (p=0.001). Additionally, serum vitamin E metabolites increased, α-CEHCs from 68 ± 20 pmol/ml to 771 ± 161 (p<0.0001) and γ-CEHC from 837 ± 161.8 pmol/ml to 1136 ± 225.9 (p=0.0083). Both CEHCs are well above reported normal values (p<0.0001). Dietary antioxidants (vitamins E and C) were low in most subjects; thus, plasma ascorbic acid levels were low in most subjects, but high in a few, resulting a wide range of responses (88 ± 84 μM). Nonetheless, ascorbic acid concentrations decreased significantly after dialysis to 33 ± 34 μM (p=0.0124), but were unaffected by vitamin E supplementation. Indeed, many parameters decreased significantly by dialysis but were unchanged by vitamin E supplementation, including plasma concentrations of uric acid and TNF-α. Both IL-6 and F₂-isoprostane concentrations were elevated in the subjects but were unaffected by either vitamin E supplementation or dialysis. CRP increased significantly after dialysis (p=0.0161, ANOVA main effect), but in the vitamin E supplemented subjects CRP concentrations were slightly lower before dialysis , but increased following dialysis (p=0.0041, ANOVA interaction). Taken together, the data suggest that there is a complex relationship between chronic inflammation and oxidative stress. Longer supplementation with vitamin E might be necessary in order to observe beneficial effects.<br>Graduation date: 2003
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Books on the topic "Therapeutic ends"

1

David, Epston, ed. Narrative means to therapeutic ends. Norton, 1990.

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The therapeutic state: Justifying government at century's end. New York University, 1998.

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Kessler, Andy. The End of Medicine. HarperCollins, 2006.

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Kessler, Andy. The end of medicine: How Silicon Valley (and naked mice) will reboot your doctor. Collins, 2006.

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Kessler, Andy. The end of medicine: How Silicon Valley (and naked mice) will reboot your doctor. Collins, 2006.

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Fox, Arnold. DLPA to end chronic pain and depression. Pocket Books, 1985.

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Fox, Arnold. DLPA to end chronic pain and depression. Pocket Books, 1985.

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Goldschmidt, Barbara. Comforting touch in dementia and at end of life: Take my hand. Singing Dragon, 2012.

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Mauskop, Alexander. What your doctor may not tell you about migraines: The breakthrough program that can help end your pain. Warner Books, 2001.

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(Foreword), Karl Tomm, ed. Literate Means to Therapeutic Ends. Dulwich Centre Publications, 1989.

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Book chapters on the topic "Therapeutic ends"

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Watts, Jay. "Systemic Means to Subversive Ends: Maintaining the Therapeutic Space as a Unique Encounter." In Critical Psychotherapy, Psychoanalysis and Counselling. Palgrave Macmillan UK, 2015. http://dx.doi.org/10.1057/9781137460585_16.

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Gordon, Evelyn, and Maeve Kenny. "Group Work in Psychiatric/Mental Health Nursing: The Case for Psychoeducation as a Means to Therapeutic Ends." In Principles of Specialty Nursing. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-31772-4_21.

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Sato, Yukihito. "Multidisciplinary Management of End-Stage Heart Failure." In Therapeutic Strategies for Heart Failure. Springer Japan, 2017. http://dx.doi.org/10.1007/978-4-431-56065-4_5.

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Esposito, C., D. Stern, S. Ogawa, and J. Brett. "Advanced Glycosylation End Products: Interactions with Cells." In New Therapeutic Strategies in Nephrology. Springer US, 1991. http://dx.doi.org/10.1007/978-1-4615-3884-4_49.

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Bambauer, R., U. Weber, R. Walter, and H. E. Keller. "Nitrendipine in Patients with End-Stage Renal Disease and Hypertension." In New Therapeutic Strategies in Nephrology. Springer US, 1991. http://dx.doi.org/10.1007/978-1-4615-3884-4_68.

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Ziegler, Christian G., Martin Ullrich, Andrew V. Schally, et al. "Novel therapeutic strategies for the treatment of pheochromocytoma." In The Endocrine Society's 95th Annual Meeting and Expo, June 15–18, 2013 - San Francisco. The Endocrine Society, 2013. http://dx.doi.org/10.1210/endo-meetings.2013.ahpaa.11.mon-60.

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Galeas, Th, G. Papadakis, E. Papanicolaou, and C. Apostolakis. "Geographical Distribution of the Unknown Aetiology End-Stage Renal Failure in Greece." In New Therapeutic Strategies in Nephrology. Springer US, 1991. http://dx.doi.org/10.1007/978-1-4615-3884-4_39.

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Goldberg, Katherine. "Palliative Medicine and End of Life Care for Pets." In Pet Loss, Grief, and Therapeutic Interventions. Routledge, 2019. http://dx.doi.org/10.4324/9780429505201-18.

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Segoloni, G. P., G. Triolo, A. Pacitti, et al. "Dialytic Treatment of Diabetic Patients with End-Stage Renal Failure: 17 Years of Experience." In New Therapeutic Strategies in Nephrology. Springer US, 1991. http://dx.doi.org/10.1007/978-1-4615-3884-4_127.

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Giorcelli, G., and G. M. Vacha. "Prevention of Thrombosis of Vascular Prosthesis in Hemodialysis: Effect of Long-Term Treatment with Urokinase at the End of the Dialytic Session." In New Therapeutic Strategies in Nephrology. Springer US, 1991. http://dx.doi.org/10.1007/978-1-4615-3884-4_87.

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Conference papers on the topic "Therapeutic ends"

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Kleinstreuer, C., P. W. Longest, and Z. Zhang. "Theory of Two-Phase Biofluid Flow Dynamics and Selected Applications." In ASME 2004 Heat Transfer/Fluids Engineering Summer Conference. ASMEDC, 2004. http://dx.doi.org/10.1115/ht-fed2004-56560.

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Examples of two-phase flows in the human body include particle-hemodynamics in branching arteries and toxic/therapeutic air-particle mixtures in the respiratory system. In this review, the fundamentals of modeling dilute particle suspensions are presented with computer applications to the geometric design of bypass graft-ends and the prediction of local aerosol depositions in the human upper airways. For the latter project, aerosols in the nano- and micro-size ranges, solid and liquid particles as well as evaporating droplets are considered. Specifically, the particle-hemodynamics project deals with the prediction of aggravating two-phase flow events leading to arterial diseases, such as atherosclerosis and hyperplasia, and subsequently the design of bypass grafts mitigating post-operative complications. The lung-aerosol project requires accurate and realistic computations of laminar-to-turbulent airflows and toxic (or therapeutic) particle depositions in the human airways for two applications: dosimetry-and-health-effect assessments of toxic particles and optimal drug aerosol delivery by inhalation. Two-phase flow results from different case studies are presented.
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Barrientos, Katharine S., Benjamin L. Lampson, Stephan D. Kendall, and Christopher M. Counter. "Abstract 2584: eNOS as a therapeutic target in pancreatic cancer." In Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-2584.

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Viana, Beatriz, Ricardo Machado, William B. Stiles, João Salgado, Patrícia Pinheiro, and Isabel Basto. "THE ASSIMILATION PROCESS OF PROBLEMATIC EXPERIENCES AND LONG-TERM OUTCOMES IN PSYCHOTHERAPY FOR DEPRESSION: COMPARING A RELAPSED AND A NON-RELAPSED CASE." In International Psychological Applications Conference and Trends. inScience Press, 2021. http://dx.doi.org/10.36315/2021inpact007.

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"Over the years, research has demonstrated that psychotherapy is an effective treatment in different psychopathological conditions. However, which are the mechanisms or processes involved in therapeutic change that could explain its efficacy are not yet clear. The Assimilation of Problematic Experiences Model describes change in therapy as a process that occurs through the gradual assimilation of problematic experiences in the self – higher levels of assimilation seem to be associated with a better outcome at the end of therapy. However, little is known about the contribution of this process to the maintenance of therapeutic gains after the end of therapy. In the current study we aimed to explore how the level of assimilation achieved throughout therapy is associated with relapse prevention after treatment. We analyzed two good outcome cases of Emotion-Focused Therapy, previously diagnosed with depression: one case that remained asymptomatic and another that relapsed one year and a half after the end of therapy. The Assimilation of Problematic Experiences (APES) was used to assess the assimilation levels achieved and the Beck Depression Inventory-II (BDI-II) was used to assess the intensity of depressive symptoms. Five therapeutic sessions and three follow-up sessions were rated using the APES. The results showed that higher APES levels were associated with lower intensity of symptoms at the end and after therapy termination, being associated with relapse prevention in depression. These results suggest that a complete assimilation of the problematic experiences may help clients to maintain therapeutic gains reducing the probability of relapsing in depression."
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Pichardo, Samuel, Kullervo Hynynen, and Emad S. Ebbini. "New design for an endo-esophageal probe intended for the ablation of cardiac muscle in the left-atrium: A parametric simulation study." In 8TH INTERNATIONAL SYMPOSIUM ON THERAPEUTIC ULTRASOUND. AIP, 2009. http://dx.doi.org/10.1063/1.3131446.

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Elshiekh, Duaa Ibnomer, Hadeel Hendawi, Aya Goul, et al. "Effect of Hyperglycemia on eNOS function in EPCs." In Qatar University Annual Research Forum & Exhibition. Qatar University Press, 2020. http://dx.doi.org/10.29117/quarfe.2020.0215.

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Type 2 diabetes mullites (T2DM) results in different cardiovascular complications. The main cause of these complications is endothelial dysfunction, which affects the endothelium physiologically and pathologically. The chronic hyperglycemia introduced by T2DM impacts the pivotal enzyme endothelial nitric oxide synthase (eNOS) in terms of phosphorylation and dimerization, which initiates oxidative stress and reduces the bioavailability of the vasodilator nitric oxide. To overcome endothelial dysfunction, endothelial progenitor cells (EPCs) contribute to vascular repair due to their regenerative characteristics. The effects of hyperglycemia on EPCs are understudied. Thus, this study aims to investigate the effects of hyperglycemia on the eNOS/Akt signaling pathway and reactive oxygen species (ROS) formation. Cells were treated with normal glucose (NG, 5.5mM) and high glucose (HG, 25mM) media for 3 &amp; 6 days, and the effect on eNOS and Akt phosphorylation were assessed using western blot. ROS was assessed using CellROX stain following 1 and 3 days of treatment. Results showed that both acute and chronic hyperglycemia showed a trend towards decrease in phosphorylation of eNOS and Akt. In addition, ROS formation was increased following 24hr compared to NG. Further investigations are needed to enhance the capability of BOECs to serve as therapeutic tools in T2DM.
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Lin, Chih-Ju, Jeon Woong Kang, Peter T. C. So, and Chen-Yuan Dong. "Analysis diffusion and glycation rate of artery in high concentration sugar condition via autofluorescence of advanced glycation end productions." In Diagnostic and Therapeutic Applications of Light in Cardiology 2020, edited by Kenton W. Gregory and Laura Marcu. SPIE, 2020. http://dx.doi.org/10.1117/12.2545589.

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Caracciolo, Daniele, Martina Montesano, Emanuela Altomare, et al. "Abstract 2827: Alternative non-homologous end joining DNA repair as therapeutic target in multiple myeloma." In Proceedings: AACR Annual Meeting 2018; April 14-18, 2018; Chicago, IL. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.am2018-2827.

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Serkova, Natalie J., Erica L. Bradshaw-Pierce, Kendra M. Hasebroock, Andrea L. Merz, Todd M. Pitts, and Gail S. Eckhardt. "Imaging end-points to predict response to IGF1R/IR inhibition in CRC models." In AACR International Conference: Molecular Diagnostics in Cancer Therapeutic Development– Sep 27-30, 2010; Denver, CO. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/diag-10-a14.

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Koehler-McNicholas, Sara R., Alana Cataldo, Elizabeth Koch, et al. "Evaluation of a Novel Gait Training Device Using a Pressure Suit to Support Body Weight." In 2018 Design of Medical Devices Conference. American Society of Mechanical Engineers, 2018. http://dx.doi.org/10.1115/dmd2018-6845.

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Supporting body weight and balance control are foundations of our ability to move and function independently. However, neurological disease, injury, and aging often threaten these prerequisites of functional independence, leading to a decrease in quality of life. In the United States alone, 7.5 million individuals have survived stroke, traumatic brain injury (TBI), or spinal cord injury (SCI), and over a million new patients are diagnosed every year [1–2]. To improve gait function in these patient populations, partial body weight-supported gait training is a widely-used rehabilitation therapy. In general, the therapeutic quality of partial body weight-supported gait training is directly proportional to the amount of time patients are able to tolerate an upright posture (either standing or walking). To achieve an upright posture, therapists must first attach a support system (e.g., gait belt, harness lift system, exoskeleton), then several therapists must assist the patient into a standing position. Depending on the patient’s level of impairment, several therapists may also be needed to support and assist the patient while standing and walking, then again to remove the support system at the end of therapy. Accordingly, multiple therapists are often needed to provide a small quantity of upright physical therapy time with standard support systems. Furthermore, use of standard support systems can be uncomfortable and fatiguing for the patient, further reducing their actual therapeutic treatment time [3].
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Kemp, Regina, Kevin Fraser, Kyoko Fujita, Douglas MacFarlane, and Gloria Elliott. "Biocompatible Ionic Liquids: A New Approach for Stabilizing Proteins in Liquid Formulation." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192986.

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The stabilization of proteins is a priority for several important fields, most notably the pharmaceutical industry. Protein-based therapeutic drugs have demonstrated significant efficacy in controlling and curing disease. Unlike traditional small molecule-based drug therapies, a major hurdle in the development of protein drugs is the challenge of maintaining the protein in the folded state throughout processing and also during storage at the end point-of-use. When a protein is taken from its native environment, it is often unstable and unfolds. Because the protein’s 3-dimensional structure is responsible for its functional activity, much work has been dedicated to finding excipients that will stabilize proteins outside of their native environment.
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Reports on the topic "Therapeutic ends"

1

Basu, Sayani. Organ Transplantation: A New Lease of Life. Science Repository, 2021. http://dx.doi.org/10.31487/sr.blog.24.

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There is a growing debate over organ transplantation which is a successive therapeutic option for the treatment of end-stage organ diseases but the ethical issues associated with the shortage of transplantable organs must also be taken into account.
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