Relevant bibliographies by topics / Therapeutic Responses against Inflammatory Diseases

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Therapeutic Responses against Inflammatory Diseases'

Author: Grafiati
Published: 3 June 2025
Last updated: 31 July 2025

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Journal articles on the topic "Therapeutic Responses against Inflammatory Diseases"

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La Manna, Sara, Concetta Di Natale, Daniele Florio, and Daniela Marasco. "Peptides as Therapeutic Agents for Inflammatory-Related Diseases." International Journal of Molecular Sciences 19, no. 9 (2018): 2714. http://dx.doi.org/10.3390/ijms19092714.

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Inflammation is a physiological mechanism used by organisms to defend themselves against infection, restoring homeostasis in damaged tissues. It represents the starting point of several chronic diseases such as asthma, skin disorders, cancer, cardiovascular syndrome, arthritis, and neurological diseases. An increasing number of studies highlight the over-expression of inflammatory molecules such as oxidants, cytokines, chemokines, matrix metalloproteinases, and transcription factors into damaged tissues. The treatment of inflammatory disorders is usually linked to the use of unspecific small m
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D'Elia, Riccardo V., Kate Harrison, Petra C. Oyston, Roman A. Lukaszewski, and Graeme C. Clark. "Targeting the “Cytokine Storm” for Therapeutic Benefit." Clinical and Vaccine Immunology 20, no. 3 (2013): 319–27. http://dx.doi.org/10.1128/cvi.00636-12.

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ABSTRACTInflammation is the body's first line of defense against infection or injury, responding to challenges by activating innate and adaptive responses. Microbes have evolved a diverse range of strategies to avoid triggering inflammatory responses. However, some pathogens, such as the influenza virus and the Gram-negative bacteriumFrancisella tularensis, do trigger life-threatening “cytokine storms” in the host which can result in significant pathology and ultimately death. For these diseases, it has been proposed that downregulating inflammatory immune responses may improve outcome. We rev
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Cho, Minkyoung, and Hak Sun Yu. "Therapeutic potentials of Trichinella spiralis in immune disorders: From allergy to autoimmunity." Parasites, Hosts and Diseases 63, no. 2 (2025): 123–34. https://doi.org/10.3347/phd.24086.

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The incidence of immune system diseases is increasing globally, particularly in developed countries. The hygiene and old friend hypotheses suggest that the decreased incidence of helminth infections in these countries may underlie the rising prevalence of autoimmune, allergic, and inflammatory diseases. The preventive and therapeutic potential of Trichinella spiralis, a helminthic parasite, has been well demonstrated in animal models of immune dysregulation-mediated diseases. This review comprehensively analyze how T. spiralis modulates immune responses across a spectrum of immune dysregulatio
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Das, Palamou, and Oishee Chakrabarti. "Mitochondrial DNA in innate immune responses against infectious diseases." Biochemical Society Transactions 48, no. 6 (2020): 2823–38. http://dx.doi.org/10.1042/bst20200687.

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Mitochondrial DNA (mtDNA) can initiate an innate immune response when mislocalized in a compartment other than the mitochondrial matrix. mtDNA plays significant roles in regulating mitochondrial dynamics as well as mitochondrial unfolded protein response (UPR). The mislocalized extra-mtDNA can elicit innate immune response via cGAS–STING (cyclic GMP-AMP synthase–stimulator of interferon genes) pathway, inducing the expression of the interferon-stimulated genes (ISGs). Also, cytosolic damaged mtDNA is cleared up by various pathways which are responsible for participating in the activation of in
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Jung, Hi Eun, and Heung Kyu Lee. "Current Understanding of the Innate Control of Toll-like Receptors in Response to SARS-CoV-2 Infection." Viruses 13, no. 11 (2021): 2132. http://dx.doi.org/10.3390/v13112132.

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The global coronavirus disease 2019 (COVID-19) pandemic, caused by severe acute respiratory syndrome-coronavirus-2 (SARS-CoV-2) infection, threatens the entire world. It has affected every aspect of life and increased the burden on both healthcare and socioeconomic systems. Current studies have revealed that excessive inflammatory immune responses are responsible for the severity of COVID-19, which suggests that anti-inflammatory drugs may be promising therapeutic treatments. However, there are currently a limited number of approved therapeutics for COVID-19. Toll-like receptors (TLRs), which
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Rashwan, Hager K., Shahenda Mahgoub, Nermeen Z. Abuelezz, and Hatem K. Amin. "Black Cumin Seed (Nigella sativa) in Inflammatory Disorders: Therapeutic Potential and Promising Molecular Mechanisms." Drugs and Drug Candidates 2, no. 2 (2023): 516–37. http://dx.doi.org/10.3390/ddc2020027.

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Inflammation is an essential defense mechanism against harmful stimuli. However, uncontrolled inflammatory mechanisms culminate in disturbed responses that contribute to multiple serious diseases. Besides common synthetic drugs, there is a growing interest in optimizing the use of natural products as therapeutic or protective supplements against inflammatory disorders. Black cumin seed (BCS), or Nigella sativa (Family Ranunculaceae), is widely used as a health-supportive herb in the Middle East, Far East and West Asia. BCS is a rich source of phytochemicals, and studies have reported its promi
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Zheng, Miao-Yan, and Lian-Zhong Luo. "The Role of IL-17A in Mediating Inflammatory Responses and Progression of Neurodegenerative Diseases." International Journal of Molecular Sciences 26, no. 6 (2025): 2505. https://doi.org/10.3390/ijms26062505.

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IL-17A has been implicated as a critical pro-inflammatory cytokine in the pathogenesis of autoimmune and neurodegenerative disorders. Emerging evidence indicates its capacity to activate microglial cells and astrocytes, subsequently inducing the production of inflammatory mediators that exacerbate neuronal injury and functional impairment. Clinical observations have revealed a demonstrated association between IL-17A concentrations and blood–brain barrier (BBB) dysfunction, creating a pathological feedback loop that amplifies neuro-inflammatory responses. Recent advances highlight the cytokine’
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Rafe, Tanzir, Parvez Ahmed Shawon, Liyad Salem, et al. "Preventive Role of Resveratrol Against Inflammatory Cytokines and Related Diseases." Current Pharmaceutical Design 25, no. 12 (2019): 1345–71. http://dx.doi.org/10.2174/1381612825666190410153307.

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Background:Immunity is the ultimate barrier between foreign stimuli and a host cell. Unwanted immune responses can threaten the host cells and may eventually damage a vital organ. Overproduction of inflammatory cytokines may also lead to autoimmune diseases. Inflammatory cells and pro-inflammatory cytokines can eventually progress to renal, cardiac, brain, hepatic, pancreatic and ocular inflammation that can result in severe damage in the long run. Evidence also suggests that inflammation may lead to atherosclerosis, Alzheimer’s, hypertension, stroke, cysts and cancers.Methods:This study was d
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Yi, Young-Su. "Functional Role of Milk Fat Globule-Epidermal Growth Factor VIII in Macrophage-Mediated Inflammatory Responses and Inflammatory/Autoimmune Diseases." Mediators of Inflammation 2016 (2016): 1–12. http://dx.doi.org/10.1155/2016/5628486.

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Inflammation involves a series of complex biological processes mediated by innate immunity for host defense against pathogen infection. Chronic inflammation is considered to be one of the major causes of serious diseases, including a number of autoimmune/inflammatory diseases, cancers, cardiovascular diseases, and neurological diseases. Milk fat globule-epidermal growth factor 8 (MFG-E8) is a secreted protein found in vertebrates and was initially discovered as a critical component of the milk fat globule. Previously, a number of studies have reported that MFG-E8 contributes to various biologi
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Das, Kaushik, Subhojit Paul, Tanmoy Mukherjee, et al. "Beyond Macromolecules: Extracellular Vesicles as Regulators of Inflammatory Diseases." Cells 12, no. 15 (2023): 1963. http://dx.doi.org/10.3390/cells12151963.

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Inflammation is the defense mechanism of the immune system against harmful stimuli such as pathogens, toxic compounds, damaged cells, radiation, etc., and is characterized by tissue redness, swelling, heat generation, pain, and loss of tissue functions. Inflammation is essential in the recruitment of immune cells at the site of infection, which not only aids in the elimination of the cause, but also initiates the healing process. However, prolonged inflammation often brings about several chronic inflammatory disorders; hence, a balance between the pro- and anti-inflammatory responses is essent
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Dissertations / Theses on the topic "Therapeutic Responses against Inflammatory Diseases"

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Phalane, Khutso Gemina. "Evaluation of multiple cytokine levels to improve our understanding of protective immune responses against Tuberculosis and to develop novel diagnostic methods." Thesis, Stellenbosch : Stellenbosch University, 2013. http://hdl.handle.net/10019.1/79848.

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Thesis (MScMedSc)--Stellenbosch University, 2013.<br>ENGLISH ABSTRACT: Important steps towards the global control of Tuberculosis include the improvement of diagnosis, the development of effective vaccines and the identification of correlates of protection/protective immunity to Mycobacterium tuberculosis. This study has of three objectives: 1. To validate the findings of a previous study that showed increased levels of IL-1β and decreased levels of IL-17 in children who are exposed to tuberculosis but remain uninfected compared to those who are exposed/infected and unexposed/uninfected. 2.
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Books on the topic "Therapeutic Responses against Inflammatory Diseases"

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Eljaafari, Assia, and Pierre Miossec. Cellular side of acquired immunity (T cells). Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0049.

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The adaptive T-cell response represents the most sophisticated component of the immune response. Foreign invaders are recognized first by cells of the innate immune system. This leads to a rapid and non-specific inflammatory response, followed by induction of the adaptive and specific immune response. Different adaptive responses can be promoted, depending on the predominant effector cells that are involved, which themselves depend on the microbial/antigen stimuli. As examples, Th1 cells contribute to cell-mediated immunity against intracellular pathogens, Th2 cells protect against parasites,
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Kuete, Victor. Medicinal Spices and Vegetables from Africa: Therapeutic Potential Against Metabolic, Inflammatory, Infectious and Systemic Diseases. Elsevier Science & Technology Books, 2017.

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Kuete, Victor. Medicinal Spices and Vegetables from Africa: Therapeutic Potential Against Metabolic, Inflammatory, Infectious and Systemic Diseases. Elsevier Science & Technology Books, 2017.

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van der Vlag, Johan, and Jo H. M. Berden. The patient with systemic lupus erythematosus. Edited by Giuseppe Remuzzi. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0161.

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Systemic lupus erythematosus (SLE) is a systemic autoimmune disease with various clinical manifestations. The hallmark of SLE is the presence of antibodies against nuclear constituents, such as double-stranded (ds)DNA, histones, and nucleosomes. Local deposition of antinuclear antibodies in complex with nuclear autoantigens induces serious inflammatory conditions that can affect several tissues and organs, including the kidney.The levels of antinucleosome and anti-dsDNA antibodies seem to correlate with glomerulonephritis and these autoantibodies can often be detected years before the patient
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Santos, Dilvani Oliveira, and Paulo Renato Zuquim Antas, eds. Mitochondrial DNA and the Immuno-inflammatory Response: New Frontiers to Control Specific Microbial Diseases. BENTHAM SCIENCE PUBLISHERS, 2022. http://dx.doi.org/10.2174/97898150516981220301.

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Mitochondria are multifunctional organelles that actively participate in the immune-inflammatory response in various pathologies. This volume updates readers on knowledge about mitochondria function. The editors have compiled six chapters about inflammation in its broadest sense, with contributions from active groups of cell biologists, infectologists and pathologists. The chapters in this volume focus on research related to five notable diseases: (1) two diseases (one bacterial and one viral) in which the exacerbation of the inflammatory response can lead to neuropathies: leprosy (one of the
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Geri, Guillaume, and Jean-Paul Mira. Host–pathogen interactions in the critically ill. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0306.

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Infection by a pathogenic micro-organism triggers a coordinated activation of both innate and adaptive immune responses. The innate immune response quickly triggers an antimicrobial response that will initiate development of a pathogen-specific, long-lasting adaptive immune response. Accurate recognition of microbial-associated molecular patterns by pattern-recognition receptors (PRRs) is the cornerstone of this immediate response. Most studied PRRs are Toll-like receptors (TLRs) and their kinase signalling cascades that activate nuclear transcription factors, and induce gene expression and cy
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Wiersinga, W. Joost, and Tom van der Poll. The host response to infection in the critically ill. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0303.

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Infection continues to be a leading cause of intensive care unit death. The host response to infection can be seen as a pattern recognition receptor (PRR)-mediated dysregulation of the immune system following pathogen invasion in which a careful balance between inflammatory and anti-inflammatory responses is vital. A measured and rapid response to microbial invasion is essential to health. The same immunological and coagulation systems that protect against localized infection can act to our disadvantage when these systems are activated systemically during generalized microbial infection. Toll-
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van Assen, Sander, and Marc Bijl. Vaccination in immunocompromised adults. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0094.

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This chapter addresses all important questions regarding vaccination of patients with autoimmune inflammatory rheumatic diseases (AIIRD). First, the incidence of vaccine-preventable infections in these patients is discussed. Pulmonary infections, including influenza and pneumococcal infection, occur more often in AIIRD patients; herpes zoster and human papillomavirus are also more frequent. The efficacy of vaccination for all European registered vaccines is discussed. Treatment with disease-modifying anti-rheumatic drugs (DMARDs) and biologicals (in particular TNFα‎-blocking agents) do not ham
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Book chapters on the topic "Therapeutic Responses against Inflammatory Diseases"

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Gater, P. R., and L. M. Renzetti. "Ro 45-2081, a TNF receptor fusion protein, prevents inflammatory responses in the airways." In Therapeutic Strategies for Modulating the Inflammatory Diseases. Birkhäuser Basel, 1998. http://dx.doi.org/10.1007/978-3-0348-8857-8_10.

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Deroissart, Justine, Florentina Porsch, Thomas Koller, and Christoph J. Binder. "Anti-inflammatory and Immunomodulatory Therapies in Atherosclerosis." In Prevention and Treatment of Atherosclerosis. Springer International Publishing, 2021. http://dx.doi.org/10.1007/164_2021_505.

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AbstractHypercholesterolemia is a major risk factor in atherosclerosis development and lipid-lowering drugs (i.e., statins) remain the treatment of choice. Despite effective reduction of LDL cholesterol in patients, a residual cardiovascular risk persists in some individuals, highlighting the need for further therapeutic intervention. Recently, the CANTOS trial paved the way toward the development of specific therapies targeting inflammation, a key feature in atherosclerosis progression. The pre-existence of multiple drugs modulating both innate and adaptive immune responses has significantly
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Cakir, Oguz, and Mustafa Abdullah Yilmaz. "A Promising Bioactive Phytochemical; Rosmarinic Acid, Targets Nervous System Disorders." In Neurological Diseases and Treatments in Terms of Biochemistry. Nobel Tip Kitabevleri, 2024. http://dx.doi.org/10.69860/nobel.9786053359357.8.

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Rosmarinic acid (RA), a phenolic molecule found in culinary herbs, has shown potential therapeutic benefits for various neurodegenerative disorders due to its antiapoptotic, antioxidant, and anti-inflammatory properties. RA can reduce oxidative stress, prevent cytotoxicity, and protect against neurotoxicity caused by various substances. It also mitigates the effects of metal ions and prevents iron-induced neurotoxicity in neuroblastoma cells. RA’s potential to improve central nervous system disorders is growing worldwide. It has been shown to suppress GABA transaminase activity and reduce seiz
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Agolti, Mariela, and Lucrecia Solari. "Review of F-18 FDG PET/CT in Evaluating Response to Immunotherapy Treatment." In Beyond Becquerel and Biology to Precision Radiomolecular Oncology: Festschrift in Honor of Richard P. Baum. Springer International Publishing, 2024. http://dx.doi.org/10.1007/978-3-031-33533-4_2.

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AbstractIntroduction: Immunotherapy is a wide-spreading therapeutic resource in oncology. The therapy is guided to improve the patient’s immune response to cancer cells, on the basis of the concept of immune surveillance by activating both cell-mediated and humoral immunity to fight cancer. Immunomodulatory monoclonal antibody therapy utilizes preformed monoclonal antibodies directed against molecular targets to regulate T-cell activation. There are three mechanisms involved in this kind of therapy: antibodies directed against the programmed death protein 1 (PD-1)/programmed death receptor lig
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Randall, Sarah, John Schomberg, Andreina Giron, Alice Martino, and Zoe Flyer. "Identification of Molecular Leads for Treatment of Secondary TBI via Analysis of Gene Expression in Models of Traumatic Brain Injury in Combination with Two-Dimensional and Three-Dimensional Drug Library Analysis." In Communications in Computer and Information Science. Springer Nature Switzerland, 2025. https://doi.org/10.1007/978-3-031-88346-0_4.

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Abstract Traumatic brain injury (TBI) is a common and serious clinical problem with high variability in injury severity. Current clinical therapies generally only manage downstream effects of TBI. Thus, there is an increasing interest in finding treatments that can directly address underlying physiological responses such as edema, cell apoptosis, and tissue oxidation. We applied open-source, clinician-accessible informatics methods that identified differentially expressed genes (DEGs) in curated TBI rodent models, found associated hub genes and genetic pathways, and determined novel chemical l
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Pushpalatha, Deepta Shirin S. P. Sundararajan, Srinivasan Kumaraswamy, Ganesh Kumar Selvaraj, et al. "Plant-Derived Bioactive Compounds." In Advances in Medical Diagnosis, Treatment, and Care. IGI Global, 2023. http://dx.doi.org/10.4018/979-8-3693-0703-8.ch011.

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Inflammation is a robust immune response against exogenous stimulation processes like microbial invasion and physical injury. Despite inflammation being an essential host-defence mechanism, their chronic stage is highly associated with severe inflammatory disorders such as cancer and heart diseases. Natural bioactive compounds have been contributing a solid base for improving contemporary therapeutic agents against various inflammatory diseases. Due to their natural source and pharmacological benefits, medicinal plants have been utilized as a significant source of anti-inflammatory drugs. The
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AW. Almorish, Mohammed, and Ali A. Alyahawi. "Toll-Like Receptors as a New Era in Cancer Therapy." In Innate Immunity - New Perspectives and Therapeutic Opportunities. IntechOpen, 2025. https://doi.org/10.5772/intechopen.1007600.

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Toll-like receptors (TLRs) are crucial in immune response by detecting pathogen and host molecules, connecting innate and adaptive immunity. They trigger the expression of various proteins and kinases, leading to the production of inflammatory mediators. Research suggests TLRs are potential targets for treating inflammation-related diseases, autoimmune diseases, infections, and cancers, with TLR antagonists showing efficacy in pre-clinical and clinical settings. The modulation of immune responses by TLRs has led to their consideration as targets for cancer therapy, showing protective effects a
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R. Jothi Lakshmi, Mrs, R. Selvakumar, K. Pushparaj, et al. "HERBAL MEDICINES ON RESPIRATORY INFLAMMATION-RELATED DISEASES." In Futuristic Trends in Pharmacy & Nursing Volume 3 Book 18. Iterative International Publishers, Selfypage Developers Pvt Ltd, 2024. http://dx.doi.org/10.58532/v3bipn18p10ch2.

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Respiratory inflammation-related diseases pose significant health challenges globally, necessitating effective therapeutic interventions. Herbal medicines have emerged as promising candidates for managing these conditions due to their diverse bioactive compounds with anti-inflammatory properties. This abstract reviews the role of herbal medicines in alleviating respiratory inflammation and associated disorders. Herbal medicines offer a rich source of phytochemicals such as flavonoids, terpenoids, and polyphenols, known for their anti-inflammatory, antioxidant, and immunomodulatory effects. The
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Matter, Christian M., Stephan Winnik, and Gerhard Rogler. "Inflammatory bowel disease and the heart." In ESC CardioMed. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0275.

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Inflammatory bowel disease comprising ulcerative colitis and Crohn’s disease are chronic inflammatory diseases of the intestinal tract. Inflammatory bowel disease is characterized by a disturbed intestinal homeostasis caused, at least partially, by an impaired intestinal barrier function associated with alterations in the gut microbiota and decreased immune tolerance against the commensal bacteria. These changes result in an exaggerated response of initially mucosal innate and, later on, adaptive immune cells against bacteria and bacterial products. Eventually, these events may lead to chronif
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Lanong, Aquisha Suklin, and Annu Kumari. "INTERLEUKIN-6 AND ITS IMPERATIVE ROLE IN VARIOUS DISORDERS AND DISEASES." In Futuristic Trends in Biotechnology Volume 3 Book 21. Iterative International Publishers, Selfypage Developers Pvt Ltd, 2024. http://dx.doi.org/10.58532/v3bkbt21p1ch3.

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The cytokine Interleukin-6 (IL-6),plays an important role in both the pathological process of inflammatory disorders and the maintenance of physiological balance in various tissues across living organisms. Numerous diseases are accompanied with significant increase in IL-6 expression in different tissues. Through this study we tried to study the significance of IL-6 in various disease pathogenesis. For infectious diseases, we highlighted how IL-6 directs the immune response against pathogens, promoting protective immunity or contributing to immunopathology. In the context of autoimmune disorde
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Conference papers on the topic "Therapeutic Responses against Inflammatory Diseases"

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Borshchev, Yury, Inessa Burovenko, Olga Borshcheva, Egor Protsak, Victor Borshchev, and Michael Galagudza. "Probiotic strains as systemic biological anti-inflammatory agents." In "Fiziologia şi sănătatea", congresul fiziologilor. Bulletin of the Academy of Sciences of Moldova. Medical Sciences, 2025. https://doi.org/10.52692/cfzl2024.03.

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Therapeutic control of the intensity, duration, and quality of immune-inflammatory processes is fundamental to the successful treatment of most infectious and non-infectious diseases, including autoimmune disorders. The aim of this study was to investigate the anti-inflammatory potential of probiotic strains. In an experiment on male Wistar rats using a model of systemic inflammatory response syndrome (SIRS), significant reductions in key inflammatory markers such as TNF-α, IL-1, IL-12, IFN-γ, and MCP-1 were observed following the administration of a probiotic mix of L. acidophilus (LA-5) and
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Sieiro Santos, C., C. Moriano Morales, and E. Díez Alvarez. "PO.8.169 Immune responses to mrna vaccines against SARS-COV-2 in patients with immune-mediated inflammatory rheumatic diseases." In 13th European Lupus Meeting, Stockholm (October 5–8, 2022). Lupus Foundation of America, 2022. http://dx.doi.org/10.1136/lupus-2022-elm2022.188.

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JAWAD, Israa, Adian Abd Alrazak DAKL, and Hussein Jabar JASIM. "CHARACTERIZATION, MECHANISM OF ACTION, SOURCES TYPES AND USES OF THE ANTIMICROBIAL PEPTIDES IN DOMESTIC ANIMALS, REVIEW." In VII. INTERNATIONAL SCIENTIFIC CONGRESSOF PURE,APPLIEDANDTECHNOLOGICAL SCIENCES. Rimar Academy, 2023. http://dx.doi.org/10.47832/minarcongress7-13.

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This review aimed to identify the general characteristics of , mechanism of action, types and uses of antimicrobial peptides in animals, antimicrobial peptides were lass of small peptides that widely exist naturally, they varied greatly in structure, composition are found in the animal's species, and were standard structural features, twenty to sixty residue long, cationic and amphipathic peptides, have a positive charge that interacted with negatively charged molecules on the bacterial cell surfaces, a have an expansive field of inhibitory effects and were made as the first line of protection
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Gray, Justin, Baochuan Guo, and Rebecca Bearden. "The Development and Validation of a GC-MS Method to Quantify Short and Branched Chain Fatty Acids in Human Stool and Applied to Patients with Inflammatory Bowel Disease and Healthy Controls." In 5th World Conference on Chemistry and Chemical Engineering and 5th World Conference on Advanced Materials, Nanoscience and Nanotechnology. Eurasia Conferences, 2024. https://doi.org/10.62422/978-81-970328-7-5-028.

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The study of short (SCFAs) and branched chain fatty acids (BCFAs) in human stool related to gastrointestinal diseases, gut microbiota, metabolism and diet has dramatically increased. As a result, a fast, reliable method with minimal pretreatment is needed for quantification of these metabolites (acetic, propionic, isobutyric, butyric, isovaleric, valeric and caproic acid; μg/g stool) in stool. Therefore, a GC-MS method meeting this criterion was developed. Stool samples were homogenized, diluted with 80:20 water:methanol (v/v) and adjusted to a pH of 1.5 - 2.5. Samples were vortexed, centrifug
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Adil Mansoor, AL-Husnah. "Role of Interleukin-35 In The Pathogenesis of Hashimoto’s Thyroiditis Disease." In X INTERNATIONAL CONGRESS OF PURE AND APPLIED TECHNOLOGICAL SCIENCES. Rimar Academy, 2023. http://dx.doi.org/10.47832/minarcongress10-1.

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The immune system has the ability to distinguish between self and non-self. Also, Regulatory T-cells are a portion of Tcells that are in charge of preserving regulation of the immune system and immune tolerance through active immunerepression. Immune tolerance is very important to prevent the immune system from interacting against itself and thus to avoid the development of autoimmune diseasesInterleukin-35 (IL-35), which is a member of the interleukin-12 (IL12) cytokine family, is an anti-inflammatory immune response that plays the important function in the obstruction of the development of a
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Zhuang, Ran, Jiawei Tian, Apostolos Tassiopoulos, Chandramouli Sadasivan, Xianfeng David Gu, and Shikui Chen. "Designing Programmable Ferromagnetic Soft Metastructures for Minimally Invasive Endovascular Therapy." In ASME 2023 International Design Engineering Technical Conferences and Computers and Information in Engineering Conference. American Society of Mechanical Engineers, 2023. http://dx.doi.org/10.1115/detc2023-116342.

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Abstract Minimally invasive endovascular therapy (MIET) is an innovative technique that utilizes percutaneous access and transcatheter implantation of medical devices to treat vascular diseases. However, conventional devices often face limitations such as incomplete or suboptimal treatment, leading to issues like recanalization in brain aneurysms, endoleaks in aortic aneurysms, and paravalvular leaks in cardiac valves. In this study, we introduce a new metastructure design for MIET employing re-entrant honeycomb structures with negative Poisson’s ratio (NPR), which are initially designed throu
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