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Relevant bibliographies by topics / Therapy induced

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Academic literature on the topic 'Therapy induced'

Author: Grafiati

Published: 4 June 2021

Last updated: 10 February 2022

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Journal articles on the topic "Therapy induced"

1

Bobrova, Bobrova E. I., Vinogradskaya O. I. Vinogradskaya, Gubernatorova E. E. Gubernatorova, and Pavlova M. G. Pavlova. "Agranulocytosis induced by thyrostatics intake." Therapy 8_2020 (December 22, 2020): 162–67. http://dx.doi.org/10.18565/therapy.2020.8.162-167.

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Kumar, Niranjan. "Compare The Effectiveness of Modified Constraint Induced Movement Therapy (mCIMT) and Mirror Therapy (MT) in Stroke Patients Based on Severity." Physiotherapy and Occupational Therapy Journal 13, no. 1 (March 1, 2020): 29–40. http://dx.doi.org/10.21088/potj.0974.5777.13120.4.

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Kim, Min-Wook. "Constraint-induced movement therapy." Journal of the Korean Medical Association 56, no. 1 (2013): 38. http://dx.doi.org/10.5124/jkma.2013.56.1.38.

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Atay, Ahmet Engin, Elif Yorulmaz, Emel Gokmen, Bennur Esen Gullu, and Dede Sit. "Antihypertensive therapy-induced pancreatitis." Medical Journal of Goztepe Training and Research Hospital 28, no. 3 (May 16, 2014): 136–40. http://dx.doi.org/10.5222/j.goztepetrh.2013.136.

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Hayashida, Marina Zoega, Jhonatan Rafael Siqueira Pinheiro, Milvia Maria Simões e. Silva Enokihara, and Mônica Ribeiro de Azevedo Vasconcellos. "Biologic therapy-induced pemphigus." Anais Brasileiros de Dermatologia 92, no. 4 (August 2017): 591–93. http://dx.doi.org/10.1590/abd1806-4841.20176481.

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Zablotska, Lydia B., Anne H. Angevine, and Alfred I. Neugut. "Therapy-induced thoracic malignancies." Clinics in Chest Medicine 25, no. 1 (March 2004): 217–24. http://dx.doi.org/10.1016/s0272-5231(03)00124-2.

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van der Lee, Johanna H., Heleen Beckerman, Gustaaf J. Lankhorst, and Lex M. Bouter. "Constraint-induced movement therapy." Archives of Physical Medicine and Rehabilitation 80, no. 12 (December 1999): 1606. http://dx.doi.org/10.1016/s0003-9993(99)90339-8.

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Singanamala, Bhanudeep, Lokesh Saini, Priyanka Madaan, Paramjeet Singh, Pankaj C. Vaidya, and Jitendra Kumar Sahu. "Antitubercular therapy-induced psychosis." Neurology 93, no. 23 (December 2, 2019): 1012–13. http://dx.doi.org/10.1212/wnl.0000000000008578.

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Nair, Devi. "Antiretroviral therapy-induced hyperlipidaemia." International Journal of STD & AIDS 16, no. 1_suppl (October 2005): 2–13. http://dx.doi.org/10.1258/095646205774462805.

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Grotta, J. C. "Constraint-Induced Movement Therapy." Stroke 35, no. 11_suppl_1 (September 16, 2004): 2699–701. http://dx.doi.org/10.1161/01.str.0000143320.64953.c4.

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Dissertations / Theses on the topic "Therapy induced"

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Souvlis, Tina. "Characteristics of spinal manual therapy induced hypoalgesia /." St. Lucia, Qld, 2003. http://www.library.uq.edu.au/pdfserve.php?image=thesisabs/absthe17338.pdf.

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Lövang, Maria, and Ögren Eva-Lena. "Constriant Induced Movement Therapy : ur ett aktivitetsperspektiv." Thesis, Örebro University, Department of Health Sciences, 2006. http://urn.kb.se/resolve?urn=urn:nbn:se:oru:diva-632.

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Syftet med denna studie var att utvärdera CI-terapi för strokedrabbade. Metoden som användes var att analysera redan insamlad data från journaler. Bedömningsinstrumenten som användes i studien var självskattningsinstrumentet UE/MAL, handfunktionstestet GAT samt den klientcentrerade intervjun COPM. Undersökningsgruppen bestod av 23 personer. Data behandlades med statistiska metoder.

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Khalil, Dayekh. "Novel Combination Therapy: Monensin Potentiates Erlotinib-Induced Cytotoxicity." Thèse, Université d'Ottawa / University of Ottawa, 2013. http://hdl.handle.net/10393/24405.

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Receptor Tyrosine Kinase (RTK) inhibitors, such as erlotinib/tarceva, have been introduced in the past decade as a promising therapeutic option in Head and Neck Squamous Cell Carcinoma (HNSCC), however, they lack significant efficacy as single agents. As a result, RTK inhibitors require a combination based therapeutic approach with other treatment modalities. To uncover such a combination of agents, we performed a high throughput Prestwick library screen that included 1200 compounds approved by the FDA on HNSCC cell lines and found that monensin, a coccidial antibiotic, synergistically enhanced the cytotoxicity of erlotinib. RT-PCR revealed that monensin induced the expression of Activation of Transcription Factor (ATF) 3 and its downstream target C/EBP homologous protein (CHOP) which are key regulators of apoptosis. Furthermore, RNA-Seq analysis suggests that monensin augments erlotinib cytotoxicity by disturbing lipid and sterol biosynthesis. Therefore, identifying the mechanism of action exerted by monensin may open alternative avenues of cancer treatment.

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DiPeri, Timothy P. "Neuromodulation Therapy Mitigates Heart Failure Induced Hippocampal Damage." Digital Commons @ East Tennessee State University, 2014. https://dc.etsu.edu/honors/208.

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Cardiovascular disease (CVD) is the leading cause of death in the United States. Nearly half of the people diagnosed with heart failure (HF) die within 5 years of diagnosis. Brain abnormalities secondary to CVD have been observed in many discrete regions, including the hippocampus. Nearly 25% of patients with CVD also have major depressive disorder (MDD), and hippocampal dysfunction is a characteristic of both diseases. In this study, the hippocampus and an area of the hippocampal formation, the dentate gyrus (DG), were studied in a canine model of HF. Using this canine HF model previously, we have determined that myocardial infarction with mitral valve regurgitation (MI/MR) + spinal cord stimulation (SCS) can preserve cardiac function. The goal of this study was to determine if the SCS can also protect the brain in a similar fashion. Both the entire hippocampus and the DG tissues were dissected from canine brains and analyzed. These findings provide strong evidence that, in addition to the cardioprotective effects observed previously, SCS following MI/MR induces neuroprotective effects in the brain.

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Nwokeoha, Sandra. "Lithotripter shock wave induced RNA-based gene therapy." Thesis, University of Oxford, 2017. https://ora.ox.ac.uk/objects/uuid:89b67c7d-4b1c-4254-8e10-86a8e6c6c79d.

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Gene therapy is the process of introducing genes to augment or minimise the expression of absent or defective genes, respectively. Non-viral gene delivery systems for the treatment of genetic diseases, have evolved into highly appreciable nucleic acid-based therapies due to considerably less risk of host immunogenicity and induction of inflammatory responses. However, they are challenged by limited delivery. Thus, more efficient strategies are continually being sought. Lithotripter shock waves (LSW) are powerful acoustic waves that are an attractive choice of delivery system, as they offer a non-invasive, targeted and safe approach. Furthermore, the delivery of messenger ribonucleic acid (mRNA) possesses several advantages over commonly delivered plasmid deoxyribonucleic acids (pDNA), because it does not require opening of the nuclear envelope, thereby reducing the level of cell injury necessary for transfection. This work presents the first investigation on the efficacy of LSW mediated mRNA delivery, based on optimised SW parameters that balance the desired enhanced permeability of cell membranes against undesired cytotoxicity, and maintain the structural and biological stability of the RNA. A transfectability measure that defines the ability of SWs to permeabilise a cell whilst keeping it alive was established for dissimilar cell types, as a function of the acoustic pressure and number of SWs. Statistically significant RNA uptake was recorded in a tissue mimicking system, and using RNA analogues at various concentrations, the SW induced bio-distribution was characterised. In addition to LSW induced gene augmentation using mRNA, it was shown that LSWs could be used to effect gene inhibition through the delivery of siRNA. Kinetic experiments were carried out to measure mRNA uptake during shock wave exposure and indicated that rate of delivery was highest at the start of the SW dose and decreased during treatment. The results also suggested that the enhancement of cell permeability was significantly transient, and that mRNA was highly susceptible to degradation in its naked state. Furthermore, mRNA-based gene expression was shown to be predictive but quantal. The in vitro tissue model was improved from a gel-based system, to one that incorporated multi-cellular spheroids which capture aspects of 3-D tumours. Static overpressure was applied during SW exposure in order to suppress cavitation effects and isolate effects that could be attributed to shear due to cell-to-cell coupling. The results showed that mild overpressure improved RNA uptake the most, but that at higher overpressure, the level of increase in RNA uptake relative to controls, was dependent on the type of RNA nucleotide being delivered. This suggested that a complex interaction between LSW cavitation and direct stress dominates delivery. A final report was on the significant improvement of gene delivery when mRNA was encapsulated within a lipid nanoparticle vector, and SW exposure was assisted by cavitation agents. Also, by exposure to another acoustic stimulus - focused ultrasound (US), direct comparisons were made between SWs and US on the efficiency of delivery and tissue penetration. In conclusion, this thesis has shown that by choosing parameters appropriately, shock waves can be a promising strategy for the delivery of genes to cells.

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Wagner, Nina. "Neuromodulation durch Constraint-induced movement therapy bei kongenitalem Hirninfarkt." Diss., lmu, 2008. http://nbn-resolving.de/urn:nbn:de:bvb:19-96728.

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Skiöld, Sara. "Radiation induced biomarkers of individual sensitivity to radiation therapy." Doctoral thesis, Stockholms universitet, Institutionen för molekylär biovetenskap, Wenner-Grens institut, 2014. http://urn.kb.se/resolve?urn=urn:nbn:se:su:diva-97123.

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Fifty percent of solid cancers are treated with radiation therapy (RT). The dose used in RT is adjusted to the most sensitive individuals so that not more than 5% of the patients will have severe adverse healthy tissue effects. As a consequence, the majority of the patients will receive a suboptimal dose, as they would have tolerated a higher total dose and received a better tumor control. Thus, if RT could be individualized based on radiation sensitivity (RS), more patients would be cured and the most severe adverse reactions could be avoided. At present the mechanisms behind RS are not known. The long term aim of this thesis was to develop diagnostic tools to assess the individual RS of breast cancer patients and to better understand the mechanisms behind the RS and radiation effects after low dose exposures. The approach was based on the hypothesis that biomarkers of individual RS, in terms of acute adverse skin reactions after breast cancer RT, can be found in whole blood that has been stressed by low doses of ionizing radiation (IR). To reach this goal two different approaches to identify biomarkers of RS have been investigated. A protocol for the analysis of differential protein expression in response to low dose in vitro irradiated whole blood was developed (paper I). This protocol was then used to investigate the proteomic profile of radiation sensitive and normo-sensitive patients, using isotope-coded protein labeled proteomics (ICPL). The results from the ICPL study (paper III) show that the two patient groups have different protein expression profiles both at the basal level and after IR. In paper II the potential biomarker 8-oxo-dG was investigated in serum after IR. The relative levels of IR induced 8-oxo-dG from radiation sensitive patients differ significantly from normo-sensitive patients. This indicates that the sensitive patients differ in their cellular response to IR and that 8-oxo-dG is a potential biomarker for RS.

At the time of the doctoral defense, the following paper was unpublished and had a status as follows: Paper 3: Manuscript.

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Yager, Nicole Leanne. "Natural and therapy-induced immune control of HIV-1." Thesis, University of Oxford, 2011. http://ora.ox.ac.uk/objects/uuid:e07f3022-4e14-4844-90ac-8d6f52a40a5a.

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The human immunodeficiency virus (HIV)-specific cytotoxic T-lymphocyte (CTL) response is important in the control of HIV-1 infection. Due to the virus having a high rate of mutation, immune pressure can select for variants that are no longer recognised by CTLs to dominate the viral quasispecies. This is similar to how antiretroviral resistance emerges. HIV-1 is therefore adapting to both human leukocyte antigen (HLA)-restricted immune responses and antiretroviral therapy. This thesis initially focused on the natural CTL response to an HLA-B*51-restricted epitope in integrase. This HLA class I allele is associated with slow progression to AIDS; however, as no CTL-driven escape mutation has been fully defined within this integrase epitope, we cannot determine what contributes to the association of HLA-B*51 and natural control of infection. By longitudinally studying a cohort of early HIV-infected individuals, we observed the emergence of polymorphisms that abrogate a CTL response to this epitope. CTL escape may also prove to be the downfall of current immunotherapy strategies attempting to combat HIV infection. T cell receptors (TCRs) have been genetically modified to enhance their binding affinity to an HLA-A*02-peptide complex and transduced into CD8+ cells to create an HIV adoptive therapy. We demonstrate through in vitro selection pressure assays that escape from these cells may be a difficult task for the virus given that the TCR is able to recognise the majority of variants of this epitope. Antigen processing mutations may represent the only option for escape. How this may translate clinically will only be determined through in vivo studies, which must be meticulously monitored. Finally, when this high affinity TCR was fused to an anti-CD3 single chain variable fragment to create proteins capable of redirecting non-HIV-specific CTLs to HIV-infected cells, we found that the result was specific lysis. These proteins may supersede the use of TCR-transduced cells when used in synergy with antiretroviral therapy.

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Griffith, Julie. "Post-Stroke Language Remediation Through Constraint-Induced Aphasia Therapy." University of Cincinnati / OhioLINK, 2014. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1415615475.

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Germain, Geneviève. "Effect of hyperbaric oxygen therapy on exercise-induced muscle injury." Thesis, McGill University, 2002. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=29504.

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The purpose of this study was to examine the effects of HBO2 therapy on exercise-induced muscle damage. Subjects (n = 16 university student volunteers) were randomly divided into an experimental group that received HBO2 therapy and a control group that did not receive any treatments. HBO2 treatments consisted of 5 sessions of breathing 95% oxygen at 2.5 atm abs for 100 min. Temporary muscle soreness was created using a single-leg eccentric exercise task involving the quadriceps femoris. Over the next 14 days, measurements were obtained on muscle soreness, leg circumference, quadriceps peak torque, quadriceps average power, fatigue and plasma creative kinase. After eccentric exercise, plasma CK levels and perceived muscle soreness were elevated but were not different between HBO2 and control groups. HBO2 therapy did not alter leg circumference, quadriceps peak torque, average power or fatigue compared to the control group. The data indicated that five HBO2 treatments did not speed recovery following eccentric exercise that induced temporary muscle damage.

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Books on the topic "Therapy induced"

1

Drug-induced hepatic injury. 2nd ed. Amsterdam: Elsevier, 1992.

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DeLuca, Stephanie. ACQUIREc therapy: A training manual for effective application of pediatric constraint-induced movement therapy. Hillsborough, NC: Mindnurture, 2007.

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Chemotherapy induced neuropathic pain. Boca Raton: CRC Press, 2012.

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Drug-induced nutritional deficiencies. 2nd ed. Westport, Conn: AVI Pub. Co., 1985.

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Fraunfelder, Frederick T. Drug-induced ocular side effects. Edited by Randall Joan A. 4th ed. Baltimore: Williams & Wilkins, 1996.

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Baroody, Theodore A. Alkalize or die: Superior health through proper alkaline-acid balance. Waynesville, NC: Eclectic, 1991.

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Alkalize or die: Superior health through proper alkaline-acid balance. 7th ed. Waynesville, NC: Eclectic, 1991.

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Drug-induced immune diseases. Amsterdam: Elsevier, 1990.

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Martha, Meyer S., ed. Drug-induced ocular side effects and drug interactions. 3rd ed. Philadelphia: Lea & Febiger, 1989.

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Marx, Robert E. Oral & intravenous bisphosphonate-induced osteonecrosis of the jaws: History, etiology, prevention, and treatment. Chicago: Quintessence Pub. Co., 2006.

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Book chapters on the topic "Therapy induced"

1

Downs, Emily, Svetlana Goldman, Surabhi Palkimas, and Aditya M. Sharma. "Heparin-Induced Thrombocytopenia." In Anticoagulation Therapy, 359–90. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-73709-6_17.

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Borza, Carley, Martin Mrazik, and Marianne Hrabok. "Constraint Induced Therapy." In Encyclopedia of Clinical Neuropsychology, 944–48. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-57111-9_1087.

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Hrabok, Marianne, and Kimberly A. Kerns. "Constraint Induced Therapy." In Encyclopedia of Clinical Neuropsychology, 692–95. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-0-387-79948-3_1087.

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Borza, Carley, Martin Mrazik, and Marianne Hrabok. "Constraint Induced Therapy." In Encyclopedia of Clinical Neuropsychology, 1–5. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-56782-2_1087-2.

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Weis, Serge, Michael Sonnberger, Andreas Dunzinger, Eva Voglmayr, Martin Aichholzer, Raimund Kleiser, and Peter Strasser. "Therapy-Induced Lesions." In Imaging Brain Diseases, 2107–18. Vienna: Springer Vienna, 2019. http://dx.doi.org/10.1007/978-3-7091-1544-2_82.

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Maranzano, E. "Prophylaxis of Radiation-Induced Emesis." In Antiemetic Therapy, 179–91. Basel: KARGER, 2003. http://dx.doi.org/10.1159/000071417.

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Kaiser, H. E. "Radiation Therapy-Induced Neoplasms." In Etiology of Cancer in Man, 122–31. Dordrecht: Springer Netherlands, 1989. http://dx.doi.org/10.1007/978-94-009-2532-8_14.

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Willey, Jeffrey S., Shane A. J. Lloyd, and Ted A. Bateman. "Radiation Therapy-Induced Osteoporosis." In Primer on the Metabolic Bone Diseases and Disorders of Mineral Metabolism, 728–33. Ames, USA: John Wiley & Sons, Inc., 2013. http://dx.doi.org/10.1002/9781118453926.ch88.

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Bartl, Reiner, and Christoph Bartl. "Tumour Therapy-Induced Osteoporosis." In The Osteoporosis Manual, 413–16. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-00731-7_66.

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Gupta, Rajesh. "Therapy-Induced Marrow Changes." In PET/MR Imaging, 21–23. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-65106-4_9.

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Conference papers on the topic "Therapy induced"

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Makki, I., and B. Bangash. "Immunologic Therapy Induced Asthma." In American Thoracic Society 2021 International Conference, May 14-19, 2021 - San Diego, CA. American Thoracic Society, 2021. http://dx.doi.org/10.1164/ajrccm-conference.2021.203.1_meetingabstracts.a1377.

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Oztekin, Erman K., and David W. Hahn. "Differential Laser-Induced Perturbation Spectroscopy Method for Biological Material Classification." In Cancer Imaging and Therapy. Washington, D.C.: OSA, 2016. http://dx.doi.org/10.1364/cancer.2016.jtu3a.50.

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Streckyte, Giedre. "Photomodification of ALA-induced protoporphyrin IX in cells in vitro." In Photodynamic Therapy of Cancer II. SPIE, 1995. http://dx.doi.org/10.1117/12.199173.

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Ottaviani, Giulia, Valentina Martinelli, Katia Rupel, Nicoletta Caronni, Asma Naseem, Lorenzo Zandonà, Giuseppe Perinetti, et al. "Laser-induced immune modulation inhibits tumor growth in vivo (Conference Presentation)." In Mechanisms of Photobiomodulation Therapy XII, edited by Michael R. Hamblin, James D. Carroll, and Praveen Arany. SPIE, 2017. http://dx.doi.org/10.1117/12.2257477.

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Maslennikova, Anna, Mikhail Pavlov, Anna Orlova, German Golubiatnikov, Pavel Suboshev, Sergey Kuznetsov, Natalia Shakhova, and Ilya Turchin. "Optical and ultrasound methods for detection of chemotherapy-induced changes of breast tumors blood supply." In Cancer Imaging and Therapy. Washington, D.C.: OSA, 2016. http://dx.doi.org/10.1364/cancer.2016.jm4a.3.

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Bensadoun, Rene-Jean. "Low-level laser therapy in chemo- and radiation-induced mucositis: results of multicenter phase III studies." In Low-Level Laser Therapy, edited by Tatiana I. Solovieva. SPIE, 2001. http://dx.doi.org/10.1117/12.425525.

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Lindner, Claus, Ivette Chochrón da Prat, Á. ngela Sánchez-Guerrero, Udo M. Weigel, Miriam de Nadal, Juan Sahuquillo, and Turgut Durduran. "Cerebral metabolism and blood flow during bispectral index-controlled, propofol-induced anesthesia assessed by hybrid diffuse optics." In Cancer Imaging and Therapy. Washington, D.C.: OSA, 2016. http://dx.doi.org/10.1364/cancer.2016.jw3a.29.

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Deng, Zhi-De, Angel V. Peterchev, Andrew D. Krystal, Bruce Luber, Shawn M. McClintock, Mustafa M. Husain, and Sarah H. Lisanby. "Topography of seizures induced by electroconvulsive therapy and magnetic seizure therapy." In 2013 6th International IEEE/EMBS Conference on Neural Engineering (NER). IEEE, 2013. http://dx.doi.org/10.1109/ner.2013.6696000.

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Choe, Regine, Gabriel Ramirez, Ashley R. Proctor, Songfeng Han, Turgut Durduran, and Edward B. Brown. "Diffuse correlation spectroscopy and tomography for longitudinal monitoring of blood flow changes induced by chemotherapy in breast cancer xenografts." In Cancer Imaging and Therapy. Washington, D.C.: OSA, 2016. http://dx.doi.org/10.1364/cancer.2016.ctu4a.4.

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Singh, Bhawana, Bernhard Zimmerman, Bin Deng, Qianqian Fang, David Boas, Jayne Cormier, Richard Moore, Daniel Kopans, Mansi Saksena, and Stefan Carp. "Integrated Near-infrared Diffuse Optical Imaging and Digital Breast Tomography for monitoring compression induced hemodynamics in breast cancer patients." In Cancer Imaging and Therapy. Washington, D.C.: OSA, 2016. http://dx.doi.org/10.1364/cancer.2016.jtu3a.38.

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Reports on the topic "Therapy induced"

1

Lu, Xiaojun, and David Rodman. TRAIL-Induced Apoptosis - A Prostate Cancer Therapy. Fort Belvoir, VA: Defense Technical Information Center, July 2001. http://dx.doi.org/10.21236/ada396752.

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Xiaojun, Lu, and David Rodman. TRAIL-Induced Apoptosis - A Prostate Cancer Therapy. Fort Belvoir, VA: Defense Technical Information Center, July 2002. http://dx.doi.org/10.21236/ada407291.

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Lu, Xiaojun, and David M. Rodman. TRAIL Induced Apoptosis - A Prostate Cancer Therapy. Fort Belvoir, VA: Defense Technical Information Center, July 2000. http://dx.doi.org/10.21236/ada392378.

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Pelizzari, Charles A>. Image Guidance and Assessment of Radiation Induced Gene Therapy. Fort Belvoir, VA: Defense Technical Information Center, February 2004. http://dx.doi.org/10.21236/ada424653.

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Fowlkes, Jeffrey B. Ultrasonically-Induced Vaporization of Perfluorocarbon Droplets for Occlusion Therapy of Breast Cancer. Fort Belvoir, VA: Defense Technical Information Center, June 2004. http://dx.doi.org/10.21236/ada471775.

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Beebe-Wang J., P. Vaska, F. A. Dilmanian, S. G. Peggs, and D. J. Schlyer. Simulation of Proton Therapy Treatment Verification via PET imaging of Induced Positron-Emitters. Office of Scientific and Technical Information (OSTI), November 2003. http://dx.doi.org/10.2172/1061715.

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Walczak, Piotr. Demyelination as a Target for Cell-Based Therapy of Chronic Blast-Induced Traumatic Brain Injury. Fort Belvoir, VA: Defense Technical Information Center, October 2014. http://dx.doi.org/10.21236/ada613175.

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Janowski, Miroslaw. Demyelination as a Target for Cell-Based Therapy of Chronic Blast-Induced Traumatic Brain Injury. Fort Belvoir, VA: Defense Technical Information Center, October 2014. http://dx.doi.org/10.21236/ada613176.

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Wu, Tong, Chengwei Fu, Yiran Deng, Wanping Huang, Yang Jiao, and Xiaoxiao Li. Acupuncture therapy for Radiotherapy-induced adverse effects: A protocol for systematic review and Bayesian network meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, July 2020. http://dx.doi.org/10.37766/inplasy2020.7.0054.

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Harch, Paul G. Hyperbaric Oxygen Therapy in the Treatment of Chronic Mild-Moderate Blast-Induced Traumatic Brain Injury PCS and PTSD. Fort Belvoir, VA: Defense Technical Information Center, October 2014. http://dx.doi.org/10.21236/ada611613.

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